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Cardiovascular effects of nitric oxide in the brain stem nuclei of rats

Stem education interventions

[No authors listed]
GSID: C-lr0sDhVUkJ
CJ Tseng, HY Liu, HC Lin, LP Ger, CS Tung… - Hypertension, 1996 - Am Heart Assoc

Nitric oxide, synthesized from the semiessential amino acid l-arginine by nitric oxide synthase, is a remarkable regulatory molecule and plays an important role in physiological functions. However, the physiological role of nitric oxide in cardiovascular regulation by the central...

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Tseng, C. J., Liu, H. Y., Lin, H. C., Ger, L. P., Tung, C. S., & Yen, M. H. (1996). Cardiovascular effects of nitric oxide in the brain stem nuclei of rats. Hypertension, 27(1), 36-42.
Tseng, Ching-Jiunn, et al. "Cardiovascular effects of nitric oxide in the brain stem nuclei of rats." Hypertension 27.1 (1996): 36-42.
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Evaluation of selected nuts and their proteins functional properties

discipline specific-education (e.g., chemistry education, biology education, economics education)

[No authors listed]
GSID: TDsTQs1bUWoJ
MAA Awad-Allah - Journal of Applied Sciences Research, 2013 - cabdirect.org

… Subject browse uses CABICODES which are CABI’s own classification codes for broad subjects that … Click on a subject area to search for all records coded with that CABICODE. … of nut meals indicated that argininearginineSubject Category: Chemicals...

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Awad-Allah, M. A. (2013). Evaluation of selected nuts and their proteins functional properties. Journal of Applied Sciences Research, 9(1), 885-896.‏
Awad-Allah, Mostafa AA. "Evaluation of selected nuts and their proteins functional properties." Journal of Applied Sciences Research 9.1 (2013): 885-896.‏
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Evaluation of Deformation Mechanisms at Mineral-Protein Composite Interface Using Steered Molecular Dynamics Simulations

EPSCoR Research Infrastructure Improvement Program AND Program Evaluation

Ghosh P, Katti DR, Katti K.
GSID: -MaDalvdGNkJ
DR Katti, P Ghosh, K Katti - MRS Online Proceedings Library (OPL), 2004 - cambridge.org

In the area of clay-polymer nanocomposites, recently montmorillonite is extensively used because of its unique characteristics of swelling. In this work, steered molecular dynamics is used to evaluate the mechanical behavior of a new class of nanocomposites, using amino...

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Katti, D. R., Ghosh, P., & Katti, K. (2004). Evaluation of Deformation Mechanisms at Mineral-Protein Composite Interface Using Steered Molecular Dynamics Simulations. MRS Online Proceedings Library (OPL), 844.
Katti, Dinesh R., Pijush Ghosh, and Kalpana Katti. "Evaluation of Deformation Mechanisms at Mineral-Protein Composite Interface Using Steered Molecular Dynamics Simulations." MRS Online Proceedings Library (OPL) 844 (2004).
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The role of a periplasmic gluconolactonase (PpgL)-like protein in Pseudomonas syringae pv. syringae B728a.

World journal of microbiology & biotechnology

Tarighi S, Taheri P.
PMID: 25187129
World J Microbiol Biotechnol. 2011 Jun;27(6):1303-11. doi: 10.1007/s11274-010-0577-2. Epub 2010 Oct 09.

In Pseudomonas syringae pv. syringae B728a, the Psyr_1712 locus ID encodes a putative protein with a signal peptide and a COG2706 domain of the type present in 3-carboxy-cis,cis-muconate lactonizing enzymes. An amino acid sequence alignment of the P. aeruginosa...

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Tarighi S, Taheri P. The role of a periplasmic gluconolactonase (PpgL)-like protein in Pseudomonas syringae pv. syringae B728a. World J Microbiol Biotechnol. 2010;27(6):1303-11doi: 10.1007/s11274-010-0577-2.
Tarighi, S., & Taheri, P. (2011). The role of a periplasmic gluconolactonase (PpgL)-like protein in Pseudomonas syringae pv. syringae B728a. World journal of microbiology & biotechnology, 27(6), 1303-11. https://doi.org/10.1007/s11274-010-0577-2
Tarighi, Saeed, and Taheri, Parissa. "The role of a periplasmic gluconolactonase (PpgL)-like protein in Pseudomonas syringae pv. syringae B728a." World journal of microbiology & biotechnology vol. 27,6 (2011): 1303-11. doi: https://doi.org/10.1007/s11274-010-0577-2
Tarighi S, Taheri P. The role of a periplasmic gluconolactonase (PpgL)-like protein in Pseudomonas syringae pv. syringae B728a. World J Microbiol Biotechnol. 2011 Jun;27(6):1303-11. doi: 10.1007/s11274-010-0577-2. Epub 2010 Oct 09. PMID: 25187129.
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The effect of almond consumption on elements of endurance exercise performance in trained athletes.

Journal of the International Society of Sports Nutrition

Yi M, Fu J, Zhou L, Gao H, Fan C, Shao J, Xu B, Wang Q, Li J, Huang G, Lapsley K, Blumberg JB, Chen CY.
PMID: 24860277
J Int Soc Sports Nutr. 2014 May 11;11:18. doi: 10.1186/1550-2783-11-18. eCollection 2014.

BACKGROUND: Almonds are a healthy tree nut food with high nutrient density. Their consumption has been shown to ameliorate oxidative stress, inflammation, etc. The objective of the study was to examine the effect of almonds on elements of endurance...

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Yi M, Fu J, Zhou L, et al. The effect of almond consumption on elements of endurance exercise performance in trained athletes. J Int Soc Sports Nutr. 2014;11:18doi: 10.1186/1550-2783-11-18.
Yi, M., Fu, J., Zhou, L., Gao, H., Fan, C., Shao, J., Xu, B., Wang, Q., Li, J., Huang, G., Lapsley, K., Blumberg, J. B., & Chen, C. Y. (2014). The effect of almond consumption on elements of endurance exercise performance in trained athletes. Journal of the International Society of Sports Nutrition, 1118. https://doi.org/10.1186/1550-2783-11-18
Yi, Muqing, et al. "The effect of almond consumption on elements of endurance exercise performance in trained athletes." Journal of the International Society of Sports Nutrition vol. 11 (2014): 18. doi: https://doi.org/10.1186/1550-2783-11-18
Yi M, Fu J, Zhou L, Gao H, Fan C, Shao J, Xu B, Wang Q, Li J, Huang G, Lapsley K, Blumberg JB, Chen CY. The effect of almond consumption on elements of endurance exercise performance in trained athletes. J Int Soc Sports Nutr. 2014 May 11;11:18. doi: 10.1186/1550-2783-11-18. eCollection 2014. PMID: 24860277; PMCID: PMC4031978.
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Detection of Carbofuran-Protein Adducts in Serum of Occupationally Exposed Pesticide Factory Workers in Pakistan.

Chemical research in toxicology

Rehman T, Khan MM, Shad MA, Hussain M, Oyler BL, Goo YA, Goodlett DR.
PMID: 27657490
Chem Res Toxicol. 2016 Oct 17;29(10):1720-1728. doi: 10.1021/acs.chemrestox.6b00222. Epub 2016 Oct 06.

This study was conducted to investigate the protein adducts with pesticides in a cohort of 172 factory workers that were exposed to a mixture of pesticides. The 35 samples showing considerable variation in biochemical parameters, i.e., butyrylcholinestrase (BChE), serum...

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Rehman T, Khan MM, Shad MA, et al. Detection of Carbofuran-Protein Adducts in Serum of Occupationally Exposed Pesticide Factory Workers in Pakistan. Chem Res Toxicol. 2016;29(10):1720-1728doi: 10.1021/acs.chemrestox.6b00222.
Rehman, T., Khan, M. M., Shad, M. A., Hussain, M., Oyler, B. L., Goo, Y. A., & Goodlett, D. R. (2016). Detection of Carbofuran-Protein Adducts in Serum of Occupationally Exposed Pesticide Factory Workers in Pakistan. Chemical research in toxicology, 29(10), 1720-1728. https://doi.org/10.1021/acs.chemrestox.6b00222
Rehman, Tanzila, et al. "Detection of Carbofuran-Protein Adducts in Serum of Occupationally Exposed Pesticide Factory Workers in Pakistan." Chemical research in toxicology vol. 29,10 (2016): 1720-1728. doi: https://doi.org/10.1021/acs.chemrestox.6b00222
Rehman T, Khan MM, Shad MA, Hussain M, Oyler BL, Goo YA, Goodlett DR. Detection of Carbofuran-Protein Adducts in Serum of Occupationally Exposed Pesticide Factory Workers in Pakistan. Chem Res Toxicol. 2016 Oct 17;29(10):1720-1728. doi: 10.1021/acs.chemrestox.6b00222. Epub 2016 Oct 06. PMID: 27657490.
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Glioma targeting peptide in combination with the P53 C terminus inhibits glioma cell proliferation in vitro.

Cytotechnology

Wang D, Guo M, Yu J, Wang X, Zhang Q, Yang X, Li J, Zhao C, Feng B.
PMID: 28879517
Cytotechnology. 2018 Feb;70(1):153-161. doi: 10.1007/s10616-017-0122-3. Epub 2017 Sep 06.

Glioma is a prevalent malignant primary brain tumor in adults, the treatment for which remains a challenge due to its high infiltration and recurrence. Hence, treatments that lead to the suppression of glioma cell migration and invasion may be...

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Wang D, Guo M, Yu J, et al. Glioma targeting peptide in combination with the P53 C terminus inhibits glioma cell proliferation in vitro. Cytotechnology. 2017;70(1):153-161doi: 10.1007/s10616-017-0122-3.
Wang, D., Guo, M., Yu, J., Wang, X., Zhang, Q., Yang, X., Li, J., Zhao, C., & Feng, B. (2018). Glioma targeting peptide in combination with the P53 C terminus inhibits glioma cell proliferation in vitro. Cytotechnology, 70(1), 153-161. https://doi.org/10.1007/s10616-017-0122-3
Wang, Dan, et al. "Glioma targeting peptide in combination with the P53 C terminus inhibits glioma cell proliferation in vitro." Cytotechnology vol. 70,1 (2018): 153-161. doi: https://doi.org/10.1007/s10616-017-0122-3
Wang D, Guo M, Yu J, Wang X, Zhang Q, Yang X, Li J, Zhao C, Feng B. Glioma targeting peptide in combination with the P53 C terminus inhibits glioma cell proliferation in vitro. Cytotechnology. 2018 Feb;70(1):153-161. doi: 10.1007/s10616-017-0122-3. Epub 2017 Sep 06. PMID: 28879517; PMCID: PMC5809644.
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Comparative evaluation of copeptin and NT-proBNP in patients with severe acute decompensated heart failure, and prediction of adverse events in a 90-day follow-up period: A prospective clinical observation trial.

Experimental and therapeutic medicine

Jia J, Chang GL, Qin S, Chen J, He WY, Lu K, Li Y, Zhang DY.
PMID: 28413508
Exp Ther Med. 2017 Apr;13(4):1554-1560. doi: 10.3892/etm.2017.4111. Epub 2017 Feb 08.

The present study compared the prognostic value of a marker, the C-terminal section of the arginine vasopressin prohormone (copeptin), with N-terminal B-type natriuretic peptide (NT-proBNP) in patients with severe acute decompensated heart failure. A prospective, observational cohort study was...

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Jia J, Chang GL, Qin S, et al. Comparative evaluation of copeptin and NT-proBNP in patients with severe acute decompensated heart failure, and prediction of adverse events in a 90-day follow-up period: A prospective clinical observation trial. Exp Ther Med. 2017;13(4):1554-1560doi: 10.3892/etm.2017.4111.
Jia, J., Chang, G. L., Qin, S., Chen, J., He, W. Y., Lu, K., Li, Y., & Zhang, D. Y. (2017). Comparative evaluation of copeptin and NT-proBNP in patients with severe acute decompensated heart failure, and prediction of adverse events in a 90-day follow-up period: A prospective clinical observation trial. Experimental and therapeutic medicine, 13(4), 1554-1560. https://doi.org/10.3892/etm.2017.4111
Jia, Jun, et al. "Comparative evaluation of copeptin and NT-proBNP in patients with severe acute decompensated heart failure, and prediction of adverse events in a 90-day follow-up period: A prospective clinical observation trial." Experimental and therapeutic medicine vol. 13,4 (2017): 1554-1560. doi: https://doi.org/10.3892/etm.2017.4111
Jia J, Chang GL, Qin S, Chen J, He WY, Lu K, Li Y, Zhang DY. Comparative evaluation of copeptin and NT-proBNP in patients with severe acute decompensated heart failure, and prediction of adverse events in a 90-day follow-up period: A prospective clinical observation trial. Exp Ther Med. 2017 Apr;13(4):1554-1560. doi: 10.3892/etm.2017.4111. Epub 2017 Feb 08. PMID: 28413508; PMCID: PMC5377512.
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Methyl-Arginine Profile of Brain from Aged PINK1-KO+A53T-SNCA Mice Suggests Altered Mitochondrial Biogenesis.

Parkinson's disease

Auburger G, Gispert S, Brehm N.
PMID: 27034888
Parkinsons Dis. 2016;2016:4686185. doi: 10.1155/2016/4686185. Epub 2016 Mar 01.

Hereditary Parkinson's disease can be triggered by an autosomal dominant overdose of alpha-Synuclein (SNCA) or the autosomal recessive deficiency of PINK1. We recently showed that the combination of PINK1-knockout with overexpression of A53T-SNCA in double mutant (DM) mice potentiates...

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Auburger G, Gispert S, Brehm N. Methyl-Arginine Profile of Brain from Aged PINK1-KO+A53T-SNCA Mice Suggests Altered Mitochondrial Biogenesis. Parkinsons Dis. 2016;2016:4686185doi: 10.1155/2016/4686185.
Auburger, G., Gispert, S., & Brehm, N. (2016). Methyl-Arginine Profile of Brain from Aged PINK1-KO+A53T-SNCA Mice Suggests Altered Mitochondrial Biogenesis. Parkinson's disease, 20164686185. https://doi.org/10.1155/2016/4686185
Auburger, Georg, et al. "Methyl-Arginine Profile of Brain from Aged PINK1-KO+A53T-SNCA Mice Suggests Altered Mitochondrial Biogenesis." Parkinson's disease vol. 2016 (2016): 4686185. doi: https://doi.org/10.1155/2016/4686185
Auburger G, Gispert S, Brehm N. Methyl-Arginine Profile of Brain from Aged PINK1-KO+A53T-SNCA Mice Suggests Altered Mitochondrial Biogenesis. Parkinsons Dis. 2016;2016:4686185. doi: 10.1155/2016/4686185. Epub 2016 Mar 01. PMID: 27034888; PMCID: PMC4791501.
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Fetal and neonatal programming of postnatal growth and feed efficiency in swine.

Journal of animal science and biotechnology

Ji Y, Wu Z, Dai Z, Wang X, Li J, Wang B, Wu G.
PMID: 28484595
J Anim Sci Biotechnol. 2017 May 05;8:42. doi: 10.1186/s40104-017-0173-5. eCollection 2017.

Maternal undernutrition or overnutrition during pregnancy alters organ structure, impairs prenatal and neonatal growth and development, and reduces feed efficiency for lean tissue gains in pigs. These adverse effects may be carried over to the next generation or beyond....

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Ji Y, Wu Z, Dai Z, et al. Fetal and neonatal programming of postnatal growth and feed efficiency in swine. J Anim Sci Biotechnol. 2017;8:42doi: 10.1186/s40104-017-0173-5.
Ji, Y., Wu, Z., Dai, Z., Wang, X., Li, J., Wang, B., & Wu, G. (2017). Fetal and neonatal programming of postnatal growth and feed efficiency in swine. Journal of animal science and biotechnology, 842. https://doi.org/10.1186/s40104-017-0173-5
Ji, Yun, et al. "Fetal and neonatal programming of postnatal growth and feed efficiency in swine." Journal of animal science and biotechnology vol. 8 (2017): 42. doi: https://doi.org/10.1186/s40104-017-0173-5
Ji Y, Wu Z, Dai Z, Wang X, Li J, Wang B, Wu G. Fetal and neonatal programming of postnatal growth and feed efficiency in swine. J Anim Sci Biotechnol. 2017 May 05;8:42. doi: 10.1186/s40104-017-0173-5. eCollection 2017. PMID: 28484595; PMCID: PMC5420136.
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Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps.

JCI insight

Kienhöfer D, Hahn J, Stoof J, Csepregi JZ, Reinwald C, Urbonaviciute V, Johnsson C, Maueröder C, Podolska MJ, Biermann MH, Leppkes M, Harrer T, Hultqvist M, Olofsson P, Munoz LE, Mocsai A, Herrmann M, Schett G, Holmdahl R, Hoffmann MH.
PMID: 28515366
JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92920. eCollection 2017 May 18.

Many effector mechanisms of neutrophils have been implicated in the pathogenesis of systemic lupus erythematosus (SLE). Neutrophil extracellular traps (NETs) have been assigned a particularly detrimental role. Here we investigated the functional impact of neutrophils and NETs on a...

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Kienhöfer D, Hahn J, Stoof J, et al. Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI Insight. 2017;2(10)doi: 10.1172/jci.insight.92920.
Kienhöfer, D., Hahn, J., Stoof, J., Csepregi, J. Z., Reinwald, C., Urbonaviciute, V., Johnsson, C., Maueröder, C., Podolska, M. J., Biermann, M. H., Leppkes, M., Harrer, T., Hultqvist, M., Olofsson, P., Munoz, L. E., Mocsai, A., Herrmann, M., Schett, G., Holmdahl, R., & Hoffmann, M. H. (2017). Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI insight, 2(10), . https://doi.org/10.1172/jci.insight.92920
Kienhöfer, Deborah, et al. "Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps." JCI insight vol. 2,10 (2017). doi: https://doi.org/10.1172/jci.insight.92920
Kienhöfer D, Hahn J, Stoof J, Csepregi JZ, Reinwald C, Urbonaviciute V, Johnsson C, Maueröder C, Podolska MJ, Biermann MH, Leppkes M, Harrer T, Hultqvist M, Olofsson P, Munoz LE, Mocsai A, Herrmann M, Schett G, Holmdahl R, Hoffmann MH. Experimental lupus is aggravated in mouse strains with impaired induction of neutrophil extracellular traps. JCI Insight. 2017 May 18;2(10). doi: 10.1172/jci.insight.92920. eCollection 2017 May 18. PMID: 28515366; PMCID: PMC5436535.
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Role of Nitric Oxide in Salmonella Infection.

Indian journal of clinical biochemistry : IJCB

Ghosh J.
PMID: 26405393
Indian J Clin Biochem. 2012 Jul;27(3):306-8. doi: 10.1007/s12291-012-0187-x. Epub 2012 Mar 23.

Nitric oxide (NO) derivative of l-arginine is an important signaling molecule that mediates a variety of essential physiological processes including vasodilation neurotransmission, and host cell defense. Many types of cells produce NO e.g., smooth muscle cell, endothelial cell, and...

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Ghosh J. Role of Nitric Oxide in Salmonella Infection. Indian J Clin Biochem. 2012;27(3):306-8doi: 10.1007/s12291-012-0187-x.
Ghosh, J. (2012). Role of Nitric Oxide in Salmonella Infection. Indian journal of clinical biochemistry : IJCB, 27(3), 306-8. https://doi.org/10.1007/s12291-012-0187-x
Ghosh, Joya. "Role of Nitric Oxide in Salmonella Infection." Indian journal of clinical biochemistry : IJCB vol. 27,3 (2012): 306-8. doi: https://doi.org/10.1007/s12291-012-0187-x
Ghosh J. Role of Nitric Oxide in Salmonella Infection. Indian J Clin Biochem. 2012 Jul;27(3):306-8. doi: 10.1007/s12291-012-0187-x. Epub 2012 Mar 23. PMID: 26405393; PMCID: PMC4577513.
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Showing 1 to 12 of 6358 entries