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Showing 1 to 12 of 712 entries
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Maintenance of Mouse, Rat, and Pig Pancreatic Islet Functions by Coculture with Human Islet-Derived Fibroblasts.

Cell transplantation

Miki A, Narushima M, Okitsu T, Takeno Y, Soto-Gutierrez A, Rivas-Carrillo JD, Navarro-Alvarez N, Chen Y, Tanaka K, Noguchi H, Matsumoto S, Kohara M, Lakey JRT, Kobayashi E, Tanaka N, Kobayashi N.
PMID: 28863744
Cell Transplant. 2006 Apr;15(4):325-334. doi: 10.3727/000000006783981882.

Development of an efficient preculture system of islets is ideal. Toward that goal, we constructed a human pancreatic islet-derived fibroblast cell line MNNK-1 for a source as a coculture system for freshly isolated islets to maintain islet functions. Human...

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Miki A, Narushima M, Okitsu T, et al. Maintenance of Mouse, Rat, and Pig Pancreatic Islet Functions by Coculture with Human Islet-Derived Fibroblasts. Cell Transplant. 2006;15(4):325-334doi: 10.3727/000000006783981882.
Miki, A., Narushima, M., Okitsu, T., Takeno, Y., Soto-Gutierrez, A., Rivas-Carrillo, J. D., Navarro-Alvarez, N., Chen, Y., Tanaka, K., Noguchi, H., Matsumoto, S., Kohara, M., Lakey, J. R. T., Kobayashi, E., Tanaka, N., & Kobayashi, N. (2006). Maintenance of Mouse, Rat, and Pig Pancreatic Islet Functions by Coculture with Human Islet-Derived Fibroblasts. Cell transplantation, 15(4), 325-334. https://doi.org/10.3727/000000006783981882
Miki, Atsushi, et al. "Maintenance of Mouse, Rat, and Pig Pancreatic Islet Functions by Coculture with Human Islet-Derived Fibroblasts." Cell transplantation vol. 15,4 (2006): 325-334. doi: https://doi.org/10.3727/000000006783981882
Miki A, Narushima M, Okitsu T, Takeno Y, Soto-Gutierrez A, Rivas-Carrillo JD, Navarro-Alvarez N, Chen Y, Tanaka K, Noguchi H, Matsumoto S, Kohara M, Lakey JRT, Kobayashi E, Tanaka N, Kobayashi N. Maintenance of Mouse, Rat, and Pig Pancreatic Islet Functions by Coculture with Human Islet-Derived Fibroblasts. Cell Transplant. 2006 Apr;15(4):325-334. doi: 10.3727/000000006783981882. PMID: 28863744.
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Protective role for miR-9-5p in the fibrogenic transformation of human dermal fibroblasts.

Fibrogenesis & tissue repair

Miguel V, Busnadiego O, Fierro-Fernández M, Lamas S.
PMID: 27274768
Fibrogenesis Tissue Repair. 2016 May 10;9:7. doi: 10.1186/s13069-016-0044-2. eCollection 2016.

BACKGROUND: Excessive accumulation of extracellular matrix (ECM) proteins is the hallmark of fibrotic diseases, including skin fibrosis. This response relies on the activation of dermal fibroblasts that evolve into a pro-fibrogenic phenotype. One of the major players in this...

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Miguel V, Busnadiego O, Fierro-Fernández M, et al. Protective role for miR-9-5p in the fibrogenic transformation of human dermal fibroblasts. Fibrogenesis Tissue Repair. 2016;9:7doi: 10.1186/s13069-016-0044-2.
Miguel, V., Busnadiego, O., Fierro-Fernández, M., & Lamas, S. (2016). Protective role for miR-9-5p in the fibrogenic transformation of human dermal fibroblasts. Fibrogenesis & tissue repair, 97. https://doi.org/10.1186/s13069-016-0044-2
Miguel, Verónica, et al. "Protective role for miR-9-5p in the fibrogenic transformation of human dermal fibroblasts." Fibrogenesis & tissue repair vol. 9 (2016): 7. doi: https://doi.org/10.1186/s13069-016-0044-2
Miguel V, Busnadiego O, Fierro-Fernández M, Lamas S. Protective role for miR-9-5p in the fibrogenic transformation of human dermal fibroblasts. Fibrogenesis Tissue Repair. 2016 May 10;9:7. doi: 10.1186/s13069-016-0044-2. eCollection 2016. PMID: 27274768; PMCID: PMC4891847.
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Comparative Transcriptome Analysis Identifies CCDC80 as a Novel Gene Associated with Pulmonary Arterial Hypertension.

Frontiers in pharmacology

Sasagawa S, Nishimura Y, Sawada H, Zhang E, Okabe S, Murakami S, Ashikawa Y, Yuge M, Kawaguchi K, Kawase R, Mitani Y, Maruyama K, Tanaka T.
PMID: 27375481
Front Pharmacol. 2016 Jun 07;7:142. doi: 10.3389/fphar.2016.00142. eCollection 2016.

Pulmonary arterial hypertension (PAH) is a heterogeneous disorder associated with a progressive increase in pulmonary artery resistance and pressure. Although various therapies have been developed, the 5-year survival rate of PAH patients remains low. There is thus an important...

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Sasagawa S, Nishimura Y, Sawada H, et al. Comparative Transcriptome Analysis Identifies CCDC80 as a Novel Gene Associated with Pulmonary Arterial Hypertension. Front Pharmacol. 2016;7:142doi: 10.3389/fphar.2016.00142.
Sasagawa, S., Nishimura, Y., Sawada, H., Zhang, E., Okabe, S., Murakami, S., Ashikawa, Y., Yuge, M., Kawaguchi, K., Kawase, R., Mitani, Y., Maruyama, K., & Tanaka, T. (2016). Comparative Transcriptome Analysis Identifies CCDC80 as a Novel Gene Associated with Pulmonary Arterial Hypertension. Frontiers in pharmacology, 7142. https://doi.org/10.3389/fphar.2016.00142
Sasagawa, Shota, et al. "Comparative Transcriptome Analysis Identifies CCDC80 as a Novel Gene Associated with Pulmonary Arterial Hypertension." Frontiers in pharmacology vol. 7 (2016): 142. doi: https://doi.org/10.3389/fphar.2016.00142
Sasagawa S, Nishimura Y, Sawada H, Zhang E, Okabe S, Murakami S, Ashikawa Y, Yuge M, Kawaguchi K, Kawase R, Mitani Y, Maruyama K, Tanaka T. Comparative Transcriptome Analysis Identifies CCDC80 as a Novel Gene Associated with Pulmonary Arterial Hypertension. Front Pharmacol. 2016 Jun 07;7:142. doi: 10.3389/fphar.2016.00142. eCollection 2016. PMID: 27375481; PMCID: PMC4894905.
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Effects of Collagen Grafting on Cell Behaviors in BCP Scaffold with Interconnected Pore Structure.

Biomaterials research

Yang DJ, Jeon JH, Lee SY, An HW, Park KO, Park KB, Kim S.
PMID: 26779345
Biomater Res. 2016 Jan 15;20:3. doi: 10.1186/s40824-016-0049-3. eCollection 2016.

BACKGROUND: This study was to investigate the effect of collagen grafted porous biphasic calcium phosphate (BCP) on cell attachment, proliferation, and differentiation. Porous BCP scaffolds with interconnected micropore structure were prepared with were prepared and then grafted with a...

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Yang DJ, Jeon JH, Lee SY, et al. Effects of Collagen Grafting on Cell Behaviors in BCP Scaffold with Interconnected Pore Structure. Biomater Res. 2016;20:3doi: 10.1186/s40824-016-0049-3.
Yang, D. J., Jeon, J. H., Lee, S. Y., An, H. W., Park, K. O., Park, K. B., & Kim, S. (2016). Effects of Collagen Grafting on Cell Behaviors in BCP Scaffold with Interconnected Pore Structure. Biomaterials research, 203. https://doi.org/10.1186/s40824-016-0049-3
Yang, Dong-Jun, et al. "Effects of Collagen Grafting on Cell Behaviors in BCP Scaffold with Interconnected Pore Structure." Biomaterials research vol. 20 (2016): 3. doi: https://doi.org/10.1186/s40824-016-0049-3
Yang DJ, Jeon JH, Lee SY, An HW, Park KO, Park KB, Kim S. Effects of Collagen Grafting on Cell Behaviors in BCP Scaffold with Interconnected Pore Structure. Biomater Res. 2016 Jan 15;20:3. doi: 10.1186/s40824-016-0049-3. eCollection 2016. PMID: 26779345; PMCID: PMC4714428.
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International Cartilage Repair Society (ICRS) Recommended Guidelines for Histological Endpoints for Cartilage Repair Studies in Animal Models and Clinical Trials.

Cartilage

Hoemann C, Kandel R, Roberts S, Saris DB, Creemers L, Mainil-Varlet P, Méthot S, Hollander AP, Buschmann MD.
PMID: 26069577
Cartilage. 2011 Apr;2(2):153-72. doi: 10.1177/1947603510397535.

Cartilage repair strategies aim to resurface a lesion with osteochondral tissue resembling native cartilage, but a variety of repair tissues are usually observed. Histology is an important structural outcome that could serve as an interim measure of efficacy in...

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Hoemann C, Kandel R, Roberts S, et al. International Cartilage Repair Society (ICRS) Recommended Guidelines for Histological Endpoints for Cartilage Repair Studies in Animal Models and Clinical Trials. Cartilage. 2011;2(2):153-72doi: 10.1177/1947603510397535.
Hoemann, C., Kandel, R., Roberts, S., Saris, D. B., Creemers, L., Mainil-Varlet, P., Méthot, S., Hollander, A. P., & Buschmann, M. D. (2011). International Cartilage Repair Society (ICRS) Recommended Guidelines for Histological Endpoints for Cartilage Repair Studies in Animal Models and Clinical Trials. Cartilage, 2(2), 153-72. https://doi.org/10.1177/1947603510397535
Hoemann, Caroline, et al. "International Cartilage Repair Society (ICRS) Recommended Guidelines for Histological Endpoints for Cartilage Repair Studies in Animal Models and Clinical Trials." Cartilage vol. 2,2 (2011): 153-72. doi: https://doi.org/10.1177/1947603510397535
Hoemann C, Kandel R, Roberts S, Saris DB, Creemers L, Mainil-Varlet P, Méthot S, Hollander AP, Buschmann MD. International Cartilage Repair Society (ICRS) Recommended Guidelines for Histological Endpoints for Cartilage Repair Studies in Animal Models and Clinical Trials. Cartilage. 2011 Apr;2(2):153-72. doi: 10.1177/1947603510397535. PMID: 26069577; PMCID: PMC4300784.
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Developmental Competence of Buffalo (Bubalus bubalis) Pluripotent Embryonic Stem Cells Over Different Homologous Feeder Layers and the Comparative Evaluation with Various Extracellular Matrices.

International journal of stem cells

Sharma M, Dubey PK, Kumar R, Nath A, Kumar GS, Sharma GT.
PMID: 24298371
Int J Stem Cells. 2013 May;6(1):26-36. doi: 10.15283/ijsc.2013.6.1.26.

BACKGROUND AND OBJECTIVES: Use of somatic cells as a feeder layer to maintain the embryonic stem cells (ESCs) in undifferentiated state limits the stem cell research design, since experimental data may result from a combined ESCs and feeder cell...

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Sharma M, Dubey PK, Kumar R, et al. Developmental Competence of Buffalo (Bubalus bubalis) Pluripotent Embryonic Stem Cells Over Different Homologous Feeder Layers and the Comparative Evaluation with Various Extracellular Matrices. Int J Stem Cells. 2013;6(1):26-36doi: 10.15283/ijsc.2013.6.1.26.
Sharma, M., Dubey, P. K., Kumar, R., Nath, A., Kumar, G. S., & Sharma, G. T. (2013). Developmental Competence of Buffalo (Bubalus bubalis) Pluripotent Embryonic Stem Cells Over Different Homologous Feeder Layers and the Comparative Evaluation with Various Extracellular Matrices. International journal of stem cells, 6(1), 26-36. https://doi.org/10.15283/ijsc.2013.6.1.26
Sharma, Manjinder, et al. "Developmental Competence of Buffalo (Bubalus bubalis) Pluripotent Embryonic Stem Cells Over Different Homologous Feeder Layers and the Comparative Evaluation with Various Extracellular Matrices." International journal of stem cells vol. 6,1 (2013): 26-36. doi: https://doi.org/10.15283/ijsc.2013.6.1.26
Sharma M, Dubey PK, Kumar R, Nath A, Kumar GS, Sharma GT. Developmental Competence of Buffalo (Bubalus bubalis) Pluripotent Embryonic Stem Cells Over Different Homologous Feeder Layers and the Comparative Evaluation with Various Extracellular Matrices. Int J Stem Cells. 2013 May;6(1):26-36. doi: 10.15283/ijsc.2013.6.1.26. PMID: 24298371; PMCID: PMC3841000.
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Tissue Inhibitor of Metalloproteinase-2 Suppresses Collagen Synthesis in Cultured Keloid Fibroblasts.

Plastic and reconstructive surgery. Global open

Dohi T, Miyake K, Aoki M, Ogawa R, Akaishi S, Shimada T, Okada T, Hyakusoku H.
PMID: 26495233
Plast Reconstr Surg Glob Open. 2015 Sep 22;3(9):e520. doi: 10.1097/GOX.0000000000000503. eCollection 2015 Sep.

BACKGROUND: Keloids are defined as a kind of dermal fibroproliferative disorder resulting from the accumulation of collagen. In the remodeling of extracellular matrix, the balance between matrix metalloproteinases (MMPs) and the tissue inhibitors of metalloproteinases (TIMPs) is as critical...

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Dohi T, Miyake K, Aoki M, et al. Tissue Inhibitor of Metalloproteinase-2 Suppresses Collagen Synthesis in Cultured Keloid Fibroblasts. Plast Reconstr Surg Glob Open. 2015;3(9):e520doi: 10.1097/GOX.0000000000000503.
Dohi, T., Miyake, K., Aoki, M., Ogawa, R., Akaishi, S., Shimada, T., Okada, T., & Hyakusoku, H. (2015). Tissue Inhibitor of Metalloproteinase-2 Suppresses Collagen Synthesis in Cultured Keloid Fibroblasts. Plastic and reconstructive surgery. Global open, 3(9), e520. https://doi.org/10.1097/GOX.0000000000000503
Dohi, Teruyuki, et al. "Tissue Inhibitor of Metalloproteinase-2 Suppresses Collagen Synthesis in Cultured Keloid Fibroblasts." Plastic and reconstructive surgery. Global open vol. 3,9 (2015): e520. doi: https://doi.org/10.1097/GOX.0000000000000503
Dohi T, Miyake K, Aoki M, Ogawa R, Akaishi S, Shimada T, Okada T, Hyakusoku H. Tissue Inhibitor of Metalloproteinase-2 Suppresses Collagen Synthesis in Cultured Keloid Fibroblasts. Plast Reconstr Surg Glob Open. 2015 Sep 22;3(9):e520. doi: 10.1097/GOX.0000000000000503. eCollection 2015 Sep. PMID: 26495233; PMCID: PMC4596445.
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Cell-Laden and Cell-Free Matrix-Induced Chondrogenesis versus Microfracture for the Treatment of Articular Cartilage Defects: A Histological and Biomechanical Study in Sheep.

Cartilage

Gille J, Kunow J, Boisch L, Behrens P, Bos I, Hoffmann C, Köller W, Russlies M, Kurz B.
PMID: 26069534
Cartilage. 2010 Jan;1(1):29-42. doi: 10.1177/1947603509358721.

OBJECTIVE: The aim of this study was to evaluate the regenerative potential of cell-laden and cell-free collagen matrices in comparison to microfracture treatment applied to full-thickness chondral defects in an ovine model.METHODS: Animals (n = 30) were randomized into...

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Gille J, Kunow J, Boisch L, et al. Cell-Laden and Cell-Free Matrix-Induced Chondrogenesis versus Microfracture for the Treatment of Articular Cartilage Defects: A Histological and Biomechanical Study in Sheep. Cartilage. 2010;1(1):29-42doi: 10.1177/1947603509358721.
Gille, J., Kunow, J., Boisch, L., Behrens, P., Bos, I., Hoffmann, C., Köller, W., Russlies, M., & Kurz, B. (2010). Cell-Laden and Cell-Free Matrix-Induced Chondrogenesis versus Microfracture for the Treatment of Articular Cartilage Defects: A Histological and Biomechanical Study in Sheep. Cartilage, 1(1), 29-42. https://doi.org/10.1177/1947603509358721
Gille, Justus, et al. "Cell-Laden and Cell-Free Matrix-Induced Chondrogenesis versus Microfracture for the Treatment of Articular Cartilage Defects: A Histological and Biomechanical Study in Sheep." Cartilage vol. 1,1 (2010): 29-42. doi: https://doi.org/10.1177/1947603509358721
Gille J, Kunow J, Boisch L, Behrens P, Bos I, Hoffmann C, Köller W, Russlies M, Kurz B. Cell-Laden and Cell-Free Matrix-Induced Chondrogenesis versus Microfracture for the Treatment of Articular Cartilage Defects: A Histological and Biomechanical Study in Sheep. Cartilage. 2010 Jan;1(1):29-42. doi: 10.1177/1947603509358721. PMID: 26069534; PMCID: PMC4440609.
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Inhibitory Effect of Delphinidin on Extracellular Matrix Production via the MAPK/NF-κB Pathway in Nasal Polyp-Derived Fibroblasts.

Allergy, asthma & immunology research

Cho JS, Kang JH, Shin JM, Park IH, Lee HM.
PMID: 25749779
Allergy Asthma Immunol Res. 2015 May;7(3):276-82. doi: 10.4168/aair.2015.7.3.276. Epub 2015 Mar 05.

PURPOSE: Nasal polyps are associated with chronic inflammation of the mucous membranes in the nose and paranasal sinuses and involved in extracellular matrix (ECM) accumulation. Delphinidin promotes ECM degradation in hepatitis and cardiac fibrosis. The aims of this study...

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Cho JS, Kang JH, Shin JM, et al. Inhibitory Effect of Delphinidin on Extracellular Matrix Production via the MAPK/NF-κB Pathway in Nasal Polyp-Derived Fibroblasts. Allergy Asthma Immunol Res. 2015;7(3):276-82doi: 10.4168/aair.2015.7.3.276.
Cho, J. S., Kang, J. H., Shin, J. M., Park, I. H., & Lee, H. M. (2015). Inhibitory Effect of Delphinidin on Extracellular Matrix Production via the MAPK/NF-κB Pathway in Nasal Polyp-Derived Fibroblasts. Allergy, asthma & immunology research, 7(3), 276-82. https://doi.org/10.4168/aair.2015.7.3.276
Cho, Jung Sun, et al. "Inhibitory Effect of Delphinidin on Extracellular Matrix Production via the MAPK/NF-κB Pathway in Nasal Polyp-Derived Fibroblasts." Allergy, asthma & immunology research vol. 7,3 (2015): 276-82. doi: https://doi.org/10.4168/aair.2015.7.3.276
Cho JS, Kang JH, Shin JM, Park IH, Lee HM. Inhibitory Effect of Delphinidin on Extracellular Matrix Production via the MAPK/NF-κB Pathway in Nasal Polyp-Derived Fibroblasts. Allergy Asthma Immunol Res. 2015 May;7(3):276-82. doi: 10.4168/aair.2015.7.3.276. Epub 2015 Mar 05. PMID: 25749779; PMCID: PMC4397368.
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Heparan Sulfate: A Potential Candidate for the Development of Biomimetic Immunomodulatory Membranes.

Frontiers in bioengineering and biotechnology

Corradetti B, Taraballi F, Giretti I, Bauza G, Pistillo RS, Banche Niclot F, Pandolfi L, Demarchi D, Tasciotti E.
PMID: 28983481
Front Bioeng Biotechnol. 2017 Sep 21;5:54. doi: 10.3389/fbioe.2017.00054. eCollection 2017.

Clinical trials have demonstrated that heparan sulfate (HS) could be used as a therapeutic agent for the treatment of inflammatory diseases. Its anti-inflammatory effect makes it suitable for the development of biomimetic innovative strategies aiming at modulating stem cells...

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Corradetti B, Taraballi F, Giretti I, et al. Heparan Sulfate: A Potential Candidate for the Development of Biomimetic Immunomodulatory Membranes. Front Bioeng Biotechnol. 2017;5:54doi: 10.3389/fbioe.2017.00054.
Corradetti, B., Taraballi, F., Giretti, I., Bauza, G., Pistillo, R. S., Banche Niclot, F., Pandolfi, L., Demarchi, D., & Tasciotti, E. (2017). Heparan Sulfate: A Potential Candidate for the Development of Biomimetic Immunomodulatory Membranes. Frontiers in bioengineering and biotechnology, 554. https://doi.org/10.3389/fbioe.2017.00054
Corradetti, Bruna, et al. "Heparan Sulfate: A Potential Candidate for the Development of Biomimetic Immunomodulatory Membranes." Frontiers in bioengineering and biotechnology vol. 5 (2017): 54. doi: https://doi.org/10.3389/fbioe.2017.00054
Corradetti B, Taraballi F, Giretti I, Bauza G, Pistillo RS, Banche Niclot F, Pandolfi L, Demarchi D, Tasciotti E. Heparan Sulfate: A Potential Candidate for the Development of Biomimetic Immunomodulatory Membranes. Front Bioeng Biotechnol. 2017 Sep 21;5:54. doi: 10.3389/fbioe.2017.00054. eCollection 2017. PMID: 28983481; PMCID: PMC5613095.
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Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats.

SpringerPlus

Zhang CC, Gu WL, Wu XM, Li YM, Chen CX, Huang XY.
PMID: 27066371
Springerplus. 2016 Mar 22;5:358. doi: 10.1186/s40064-016-1985-z. eCollection 2016.

BACKGROUND: In the previous study, active extract of Radix Scrophularia (ACRS) demonstrated beneficial effects on ventricular remodeling induced by coronary artery ligation and lowered blood pressure in rats. And ACRS also exhibited the effect on lowering the blood pressure...

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Zhang CC, Gu WL, Wu XM, et al. Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats. Springerplus. 2016;5:358doi: 10.1186/s40064-016-1985-z.
Zhang, C. C., Gu, W. L., Wu, X. M., Li, Y. M., Chen, C. X., & Huang, X. Y. (2016). Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats. SpringerPlus, 5358. https://doi.org/10.1186/s40064-016-1985-z
Zhang, Chao Chao, et al. "Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats." SpringerPlus vol. 5 (2016): 358. doi: https://doi.org/10.1186/s40064-016-1985-z
Zhang CC, Gu WL, Wu XM, Li YM, Chen CX, Huang XY. Active components from Radix Scrophulariae inhibits the ventricular remodeling induced by hypertension in rats. Springerplus. 2016 Mar 22;5:358. doi: 10.1186/s40064-016-1985-z. eCollection 2016. PMID: 27066371; PMCID: PMC4801831.
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A cephalometric method to diagnosis the craniovertebral junction abnormalities in osteogenesis imperfecta patients.

Journal of clinical and experimental dentistry

Ríos-Rodenas M, de Nova J, Gutiérrez-Díez MP, Feijóo G, Mourelle MR, Garcilazo M, Ortega-Aranegui R.
PMID: 25810828
J Clin Exp Dent. 2015 Feb 01;7(1):e153-8. doi: 10.4317/jced.52126. eCollection 2015 Feb.

Osteogenesis imperfecta (OI) is a hereditary bone fragility disorder that in most patients is caused by mutations affecting collagen type I. Their typical oral and craneofacial characteristics (Dentinogenesis imperfecta type I and class III malocclusion), involve the dentist in...

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Ríos-Rodenas M, de Nova J, Gutiérrez-Díez MP, et al. A cephalometric method to diagnosis the craniovertebral junction abnormalities in osteogenesis imperfecta patients. J Clin Exp Dent. 2015;7(1):e153-8doi: 10.4317/jced.52126.
Ríos-Rodenas, M., de Nova, J., Gutiérrez-Díez, M. P., Feijóo, G., Mourelle, M. R., Garcilazo, M., & Ortega-Aranegui, R. (2015). A cephalometric method to diagnosis the craniovertebral junction abnormalities in osteogenesis imperfecta patients. Journal of clinical and experimental dentistry, 7(1), e153-8. https://doi.org/10.4317/jced.52126
Ríos-Rodenas, Mercedes, et al. "A cephalometric method to diagnosis the craniovertebral junction abnormalities in osteogenesis imperfecta patients." Journal of clinical and experimental dentistry vol. 7,1 (2015): e153-8. doi: https://doi.org/10.4317/jced.52126
Ríos-Rodenas M, de Nova J, Gutiérrez-Díez MP, Feijóo G, Mourelle MR, Garcilazo M, Ortega-Aranegui R. A cephalometric method to diagnosis the craniovertebral junction abnormalities in osteogenesis imperfecta patients. J Clin Exp Dent. 2015 Feb 01;7(1):e153-8. doi: 10.4317/jced.52126. eCollection 2015 Feb. PMID: 25810828; PMCID: PMC4368004.
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