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Carbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destruction.

Hepatobiliary surgery and nutrition

Basaranoglu M, Basaranoglu G, Bugianesi E.
PMID: 26005677
Hepatobiliary Surg Nutr. 2015 Apr;4(2):109-16. doi: 10.3978/j.issn.2304-3881.2014.11.05.

Excessive accumulation of triglycerides (TG) in liver, in the absence of significant alcohol consumption is nonalcoholic fatty liver disease (NAFLD). NAFLD is a significant risk factor for developing cirrhosis and an independent predictor of cardiovascular disease. High fructose corn...

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Basaranoglu M, Basaranoglu G, Bugianesi E. Carbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destruction. Hepatobiliary Surg Nutr. 2015;4(2):109-16doi: 10.3978/j.issn.2304-3881.2014.11.05.
Basaranoglu, M., Basaranoglu, G., & Bugianesi, E. (2015). Carbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destruction. Hepatobiliary surgery and nutrition, 4(2), 109-16. https://doi.org/10.3978/j.issn.2304-3881.2014.11.05
Basaranoglu, Metin, et al. "Carbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destruction." Hepatobiliary surgery and nutrition vol. 4,2 (2015): 109-16. doi: https://doi.org/10.3978/j.issn.2304-3881.2014.11.05
Basaranoglu M, Basaranoglu G, Bugianesi E. Carbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destruction. Hepatobiliary Surg Nutr. 2015 Apr;4(2):109-16. doi: 10.3978/j.issn.2304-3881.2014.11.05. PMID: 26005677; PMCID: PMC4405421.
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d-Allose is absorbed via sodium-dependent glucose cotransporter 1 (SGLT1) in the rat small intestine.

Metabolism open

Kishida K, Iida T, Yamada T, Toyoda Y.
PMID: 34381987
Metabol Open. 2021 Jul 22;11:100112. doi: 10.1016/j.metop.2021.100112. eCollection 2021 Sep.

d-Allose is the C3 epimer of d-glucose and has been reported to have beneficial health effects. The transporter mediating intestinal transport of d-allose is unknown. We examined whether d-allose is absorbed via sodium-dependent glucose cotransporter 1 (SGLT1) as well...

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Kishida K, Iida T, Yamada T, et al. d-Allose is absorbed via sodium-dependent glucose cotransporter 1 (SGLT1) in the rat small intestine. Metabol Open. 2021;11:100112doi: 10.1016/j.metop.2021.100112.
Kishida, K., Iida, T., Yamada, T., & Toyoda, Y. (2021). d-Allose is absorbed via sodium-dependent glucose cotransporter 1 (SGLT1) in the rat small intestine. Metabolism open, 11100112. https://doi.org/10.1016/j.metop.2021.100112
Kishida, Kunihiro, et al. "d-Allose is absorbed via sodium-dependent glucose cotransporter 1 (SGLT1) in the rat small intestine." Metabolism open vol. 11 (2021): 100112. doi: https://doi.org/10.1016/j.metop.2021.100112
Kishida K, Iida T, Yamada T, Toyoda Y. d-Allose is absorbed via sodium-dependent glucose cotransporter 1 (SGLT1) in the rat small intestine. Metabol Open. 2021 Jul 22;11:100112. doi: 10.1016/j.metop.2021.100112. eCollection 2021 Sep. PMID: 34381987; PMCID: PMC8339219.
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Impact of natural mutations on the riboflavin transporter 2 and their relevance to human riboflavin transporter deficiency 2.

IUBMB life

Console L, Tolomeo M, Cosco J, Massey K, Barile M, Indiveri C.
PMID: 34428344
IUBMB Life. 2021 Aug 24; doi: 10.1002/iub.2541. Epub 2021 Aug 24.

Riboflavin transporter deficiency 2 (RTD2) is a rare neurological disorder caused by mutations in the Solute carrier family 52 member 2 (Slc52a2) gene encoding human riboflavin transporter 2 (RFVT2). This transporter is ubiquitously expressed and mediates tissue distribution of...

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Console L, Tolomeo M, Cosco J, et al. Impact of natural mutations on the riboflavin transporter 2 and their relevance to human riboflavin transporter deficiency 2. IUBMB Life. 2021;doi: 10.1002/iub.2541.
Console, L., Tolomeo, M., Cosco, J., Massey, K., Barile, M., & Indiveri, C. (2021). Impact of natural mutations on the riboflavin transporter 2 and their relevance to human riboflavin transporter deficiency 2. IUBMB life, . https://doi.org/10.1002/iub.2541
Console, Lara, et al. "Impact of natural mutations on the riboflavin transporter 2 and their relevance to human riboflavin transporter deficiency 2." IUBMB life vol. (2021). doi: https://doi.org/10.1002/iub.2541
Console L, Tolomeo M, Cosco J, Massey K, Barile M, Indiveri C. Impact of natural mutations on the riboflavin transporter 2 and their relevance to human riboflavin transporter deficiency 2. IUBMB Life. 2021 Aug 24; doi: 10.1002/iub.2541. Epub 2021 Aug 24. PMID: 34428344.
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