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Showing 1 to 12 of 190 entries
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Altered serous levels of monoamine neurotransmitter metabolites in patients with refractory and non-refractory depression.

Neural regeneration research

Zhang G, Zhang Y, Yang J, Hu M, Zhang Y, Liang X.
PMID: 25722703
Neural Regen Res. 2012 May 15;7(14):1113-8. doi: 10.3969/j.issn.1673-5374.2012.14.011.

The study examined plasma metabolite changes of monoamine neurotransmitters in patients with treatment-resistant depression (TRD) and non-TRD before and after therapy. All 30 TRD and 30 non-TRD patients met the diagnostic criteria for a depressive episode in accordance with...

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Zhang G, Zhang Y, Yang J, et al. Altered serous levels of monoamine neurotransmitter metabolites in patients with refractory and non-refractory depression. Neural Regen Res. 2012;7(14):1113-8doi: 10.3969/j.issn.1673-5374.2012.14.011.
Zhang, G., Zhang, Y., Yang, J., Hu, M., Zhang, Y., & Liang, X. (2012). Altered serous levels of monoamine neurotransmitter metabolites in patients with refractory and non-refractory depression. Neural regeneration research, 7(14), 1113-8. https://doi.org/10.3969/j.issn.1673-5374.2012.14.011
Zhang, Guiqing, et al. "Altered serous levels of monoamine neurotransmitter metabolites in patients with refractory and non-refractory depression." Neural regeneration research vol. 7,14 (2012): 1113-8. doi: https://doi.org/10.3969/j.issn.1673-5374.2012.14.011
Zhang G, Zhang Y, Yang J, Hu M, Zhang Y, Liang X. Altered serous levels of monoamine neurotransmitter metabolites in patients with refractory and non-refractory depression. Neural Regen Res. 2012 May 15;7(14):1113-8. doi: 10.3969/j.issn.1673-5374.2012.14.011. PMID: 25722703; PMCID: PMC4340026.
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[Management of treatment in patients with neuroendocrine neoplasmas of digestive tract].

Vnitrni lekarstvi

Kiňová S, Kováčová M, Čaprnda M, Koreň M.
PMID: 26800468
Vnitr Lek. 2015;61:12-20.

UNLABELLED: Neuroendocrine neoplasmas are a form of cancer arising from cells of diffuse neuroendocrine system. They produce peptides or amines that act as hormones or neurotransmitters. Incidence of NENs is relatively low. Diagnostic work-up and treatment requires a multidisciplinary...

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Kiňová S, Kováčová M, Čaprnda M, et al. Manažment liečby pacientov s neuroendokrinnými nádormi tráviaceho traktu. [Management of treatment in patients with neuroendocrine neoplasmas of digestive tract]. Vnitr Lek. 2015;61:12-20
Kiňová, S., Kováčová, M., Čaprnda, M., & Koreň, M. (2015). Manažment liečby pacientov s neuroendokrinnými nádormi tráviaceho traktu. [Management of treatment in patients with neuroendocrine neoplasmas of digestive tract]. Vnitrni lekarstvi, 6112-20.
Kiňová, Soňa, et al. "Manažment liečby pacientov s neuroendokrinnými nádormi tráviaceho traktu." [Management of treatment in patients with neuroendocrine neoplasmas of digestive tract]. Vnitrni lekarstvi vol. 61 (2015): 12-20.
Kiňová S, Kováčová M, Čaprnda M, Koreň M. Manažment liečby pacientov s neuroendokrinnými nádormi tráviaceho traktu. [Management of treatment in patients with neuroendocrine neoplasmas of digestive tract]. Vnitr Lek. 2015;61:12-20. Czech. PMID: 26800468.
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Parkinson's disease subtypes: clinical classification and ventricular cerebrospinal fluid analysis.

Parkinsonism & related disorders

Schiess MC, Zheng H, Soukup VM, Bonnen JG, Nauta HJ.
PMID: 10699387
Parkinsonism Relat Disord. 2000 Apr 01;6(2):69-76. doi: 10.1016/s1353-8020(99)00051-6.

The current study presents preliminary data regarding the development and validation of a rating system designed to classify PD patients into clinical subgroups. Using portions of the Unified Parkinson's Disease Rating Scale, a ratio value was derived, yielding three...

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Schiess MC, Zheng H, Soukup VM, et al. Parkinson's disease subtypes: clinical classification and ventricular cerebrospinal fluid analysis. Parkinsonism Relat Disord. 2000;6(2):69-76doi: 10.1016/s1353-8020(99)00051-6.
Schiess, M. C., Zheng, H., Soukup, V. M., Bonnen, J. G., & Nauta, H. J. (2000). Parkinson's disease subtypes: clinical classification and ventricular cerebrospinal fluid analysis. Parkinsonism & related disorders, 6(2), 69-76. https://doi.org/10.1016/s1353-8020(99)00051-6
Schiess, et al. "Parkinson's disease subtypes: clinical classification and ventricular cerebrospinal fluid analysis." Parkinsonism & related disorders vol. 6,2 (2000): 69-76. doi: https://doi.org/10.1016/s1353-8020(99)00051-6
Schiess MC, Zheng H, Soukup VM, Bonnen JG, Nauta HJ. Parkinson's disease subtypes: clinical classification and ventricular cerebrospinal fluid analysis. Parkinsonism Relat Disord. 2000 Apr 01;6(2):69-76. doi: 10.1016/s1353-8020(99)00051-6. PMID: 10699387.
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Tryptophan depletion selectively reduces CSF 5-HT metabolites in healthy young men: results from single lumbar puncture sampling technique.

The international journal of neuropsychopharmacology

Moreno FA, McGavin C, Malan TP, Gelenberg AJ, Heninger GR, Mathé AA, Delgado PL.
PMID: 11343605
Int J Neuropsychopharmacol. 2000 Dec;3(4):277-283. doi: 10.1017/S1461145700002133.

Plasma tryptophan (Trp) depletion is a commonly used tool for determining the role of brain serotonin (5-HT) function in a variety of psychiatric disorders. This study measured the cerebrospinal fluid (CSF) monoamine metabolite response to Trp depletion and control...

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Moreno FA, McGavin C, Malan TP, et al. Tryptophan depletion selectively reduces CSF 5-HT metabolites in healthy young men: results from single lumbar puncture sampling technique. Int J Neuropsychopharmacol. 2000;3(4):277-283doi: 10.1017/S1461145700002133.
Moreno, F. A., McGavin, C., Malan, T. P., Gelenberg, A. J., Heninger, G. R., Mathé, A. A., & Delgado, P. L. (2000). Tryptophan depletion selectively reduces CSF 5-HT metabolites in healthy young men: results from single lumbar puncture sampling technique. The international journal of neuropsychopharmacology, 3(4), 277-283. https://doi.org/10.1017/S1461145700002133
Moreno, Francisco A., et al. "Tryptophan depletion selectively reduces CSF 5-HT metabolites in healthy young men: results from single lumbar puncture sampling technique." The international journal of neuropsychopharmacology vol. 3,4 (2000): 277-283. doi: https://doi.org/10.1017/S1461145700002133
Moreno FA, McGavin C, Malan TP, Gelenberg AJ, Heninger GR, Mathé AA, Delgado PL. Tryptophan depletion selectively reduces CSF 5-HT metabolites in healthy young men: results from single lumbar puncture sampling technique. Int J Neuropsychopharmacol. 2000 Dec;3(4):277-283. doi: 10.1017/S1461145700002133. PMID: 11343605.
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Changes in plasma monoamine metabolites during acute lithium intoxication.

Human psychopharmacology

Yoshimura R, Yamada Y, Ueda N, Nakamura J.
PMID: 12404314
Hum Psychopharmacol. 2000 Jul;15(5):357-360. doi: 10.1002/1099-1077(200007)15:5<357::AID-HUP174>3.0.CO;2-K.

A case of acute lithium intoxication is presented in which plasma levels of 3-methyoxy-4-hydroxyphenylglycol (MHPG), 5-hydroxyindoleacetic acid (5-HIAA), and homovanillic acid (HVA) were longitudinally measured during the intoxication period. Plasma levels of MHPH and 5-HIAA, but not HVA were...

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Yoshimura R, Yamada Y, Ueda N, et al. Changes in plasma monoamine metabolites during acute lithium intoxication. Hum Psychopharmacol. 2000;15(5):357-360doi: 10.1002/1099-1077(200007)15:5<357::AID-HUP174>3.0.CO;2-K.
Yoshimura, R., Yamada, Y., Ueda, N., & Nakamura, J. (2000). Changes in plasma monoamine metabolites during acute lithium intoxication. Human psychopharmacology, 15(5), 357-360. https://doi.org/10.1002/1099-1077(200007)15:5<357::AID-HUP174>3.0.CO;2-K
Yoshimura, Reiji, et al. "Changes in plasma monoamine metabolites during acute lithium intoxication." Human psychopharmacology vol. 15,5 (2000): 357-360. doi: https://doi.org/10.1002/1099-1077(200007)15:5<357::AID-HUP174>3.0.CO;2-K
Yoshimura R, Yamada Y, Ueda N, Nakamura J. Changes in plasma monoamine metabolites during acute lithium intoxication. Hum Psychopharmacol. 2000 Jul;15(5):357-360. doi: 10.1002/1099-1077(200007)15:5<357::AID-HUP174>3.0.CO;2-K. PMID: 12404314.
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Tissue 5-hydroxytryptamine and urinary 5-hydroxyindoleacetic acid after partial or total removal of the gastro-intestinal tract in the rat.

The Journal of physiology

Bertaccini G.
PMID: 16992061
J Physiol. 1960 Sep;153(2):239-49. doi: 10.1113/jphysiol.1960.sp006532.

No abstract available.

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Bertaccini G. Tissue 5-hydroxytryptamine and urinary 5-hydroxyindoleacetic acid after partial or total removal of the gastro-intestinal tract in the rat. J Physiol. 1960;153(2):239-49doi: 10.1113/jphysiol.1960.sp006532.
Bertaccini, G. (1960). Tissue 5-hydroxytryptamine and urinary 5-hydroxyindoleacetic acid after partial or total removal of the gastro-intestinal tract in the rat. The Journal of physiology, 153(2), 239-49. https://doi.org/10.1113/jphysiol.1960.sp006532
Bertaccini, G. "Tissue 5-hydroxytryptamine and urinary 5-hydroxyindoleacetic acid after partial or total removal of the gastro-intestinal tract in the rat." The Journal of physiology vol. 153,2 (1960): 239-49. doi: https://doi.org/10.1113/jphysiol.1960.sp006532
Bertaccini G. Tissue 5-hydroxytryptamine and urinary 5-hydroxyindoleacetic acid after partial or total removal of the gastro-intestinal tract in the rat. J Physiol. 1960 Sep;153(2):239-49. doi: 10.1113/jphysiol.1960.sp006532. PMID: 16992061; PMCID: PMC1359746.
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The neurochemical profile of long term oral administration of moclobemide.

Pakistan journal of pharmaceutical sciences

Haleem DJ, Haider S, Yasmeen A, Parveen T.
PMID: 16414805
Pak J Pharm Sci. 1998 Jan;11(1):9-14.

Moclobemide, a benzamide derivative, predominantly inhibits the A form of monoamine oxidase (MAO) and its MAO binding is reversible. Acute administration of moclobemide has been shown to induce an increase in brain levels of monoamines and a concomitant decrease...

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Haleem DJ, Haider S, Yasmeen A, et al. The neurochemical profile of long term oral administration of moclobemide. Pak J Pharm Sci. 1998;11(1):9-14
Haleem, D. J., Haider, S., Yasmeen, A., & Parveen, T. (1998). The neurochemical profile of long term oral administration of moclobemide. Pakistan journal of pharmaceutical sciences, 11(1), 9-14.
Haleem, D J, et al. "The neurochemical profile of long term oral administration of moclobemide." Pakistan journal of pharmaceutical sciences vol. 11,1 (1998): 9-14.
Haleem DJ, Haider S, Yasmeen A, Parveen T. The neurochemical profile of long term oral administration of moclobemide. Pak J Pharm Sci. 1998 Jan;11(1):9-14. PMID: 16414805.
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Increased precursor availability did not increase food intake and 5-HT turnover rate in the hypothalamus of diazepam injected rats.

Pakistan journal of pharmaceutical sciences

Batool F, Haleem DJ.
PMID: 16414823
Pak J Pharm Sci. 1999 Jan;12(1):21-6.

In view of a possible role of serotonin (5-hydroxytryptamine; 5- HT) in regulation of appetite and anxiety, the effects of 1, 3 and 5 mg/kg doses of diazepam on brain serotonin precursor and effects of single and repeated diazepam...

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Batool F, Haleem DJ. Increased precursor availability did not increase food intake and 5-HT turnover rate in the hypothalamus of diazepam injected rats. Pak J Pharm Sci. 1999;12(1):21-6
Batool, F., & Haleem, D. J. (1999). Increased precursor availability did not increase food intake and 5-HT turnover rate in the hypothalamus of diazepam injected rats. Pakistan journal of pharmaceutical sciences, 12(1), 21-6.
Batool, F, and Haleem, D J. "Increased precursor availability did not increase food intake and 5-HT turnover rate in the hypothalamus of diazepam injected rats." Pakistan journal of pharmaceutical sciences vol. 12,1 (1999): 21-6.
Batool F, Haleem DJ. Increased precursor availability did not increase food intake and 5-HT turnover rate in the hypothalamus of diazepam injected rats. Pak J Pharm Sci. 1999 Jan;12(1):21-6. PMID: 16414823.
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Effects of high dose propranolol treatment on dopamine and norepinephrine metabolism in regions of rat brain.

Neuroscience letters

Wiesel FA.
PMID: 19604810
Neurosci Lett. 1976 Mar;2(1):35-8. doi: 10.1016/0304-3940(76)90042-2.

The acute effects of a high dose of d,l-propranolol on the levels of homovanillic acid (HVA), 5-hydroxyindoleacetic acid (5-HIAA) and 4-hydroxy-3-methoxyphenylethylene glycol (HMPG) in rat brain regions were determined by mass fragmentography. A significant elevation of HVA was obtained...

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Wiesel FA. Effects of high dose propranolol treatment on dopamine and norepinephrine metabolism in regions of rat brain. Neurosci Lett. 1976;2(1):35-8doi: 10.1016/0304-3940(76)90042-2.
Wiesel, F. A. (1976). Effects of high dose propranolol treatment on dopamine and norepinephrine metabolism in regions of rat brain. Neuroscience letters, 2(1), 35-8. https://doi.org/10.1016/0304-3940(76)90042-2
Wiesel, F A. "Effects of high dose propranolol treatment on dopamine and norepinephrine metabolism in regions of rat brain." Neuroscience letters vol. 2,1 (1976): 35-8. doi: https://doi.org/10.1016/0304-3940(76)90042-2
Wiesel FA. Effects of high dose propranolol treatment on dopamine and norepinephrine metabolism in regions of rat brain. Neurosci Lett. 1976 Mar;2(1):35-8. doi: 10.1016/0304-3940(76)90042-2. PMID: 19604810.
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Determination of isatin and monoamine neurotransmitters in rat brain with liquid chromatography using palladium hexacyanoferrate modified electrode.

Analytica chimica acta

Xu H, Wang D, Zhang W, Zhu W, Yamamoto K, Jin L.
PMID: 17723673
Anal Chim Acta. 2006 Sep 08;577(2):207-13. doi: 10.1016/j.aca.2006.06.042. Epub 2006 Jun 27.

The fabrication and application of a novel electrochemical detector (ED) with palladium hexacyanoferrate (PdHCF) chemically modified electrode (CME) for liquid chromatography (LC) were described. The electrochemical behaviors of isatin, monoamine neurotransmitters and their metabolites at this CME were investigated...

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Xu H, Wang D, Zhang W, et al. Determination of isatin and monoamine neurotransmitters in rat brain with liquid chromatography using palladium hexacyanoferrate modified electrode. Anal Chim Acta. 2006;577(2):207-13doi: 10.1016/j.aca.2006.06.042.
Xu, H., Wang, D., Zhang, W., Zhu, W., Yamamoto, K., & Jin, L. (2006). Determination of isatin and monoamine neurotransmitters in rat brain with liquid chromatography using palladium hexacyanoferrate modified electrode. Analytica chimica acta, 577(2), 207-13. https://doi.org/10.1016/j.aca.2006.06.042
Xu, Haihong, et al. "Determination of isatin and monoamine neurotransmitters in rat brain with liquid chromatography using palladium hexacyanoferrate modified electrode." Analytica chimica acta vol. 577,2 (2006): 207-13. doi: https://doi.org/10.1016/j.aca.2006.06.042
Xu H, Wang D, Zhang W, Zhu W, Yamamoto K, Jin L. Determination of isatin and monoamine neurotransmitters in rat brain with liquid chromatography using palladium hexacyanoferrate modified electrode. Anal Chim Acta. 2006 Sep 08;577(2):207-13. doi: 10.1016/j.aca.2006.06.042. Epub 2006 Jun 27. PMID: 17723673.
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Primary hepatic carcinoid; a diagnostic dilemma: a case report.

Cases journal

Touloumis Z, Delis SG, Triantopoulou C, Giannakou N, Avgerinos C, Dervenis C.
PMID: 19014620
Cases J. 2008 Nov 17;1(1):314. doi: 10.1186/1757-1626-1-314.

INTRODUCTION: Primary hepatic carcinoid tumours (PHCTs) are extremely rare neuroendocrine neoplasms. Only 58 cases have been reported in the literature and less than 10 cases were functional.CASE PRESENTATION: We present a case of a 65 years old, Caucasian female...

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Touloumis Z, Delis SG, Triantopoulou C, et al. Primary hepatic carcinoid; a diagnostic dilemma: a case report. Cases J. 2008;1(1):314doi: 10.1186/1757-1626-1-314.
Touloumis, Z., Delis, S. G., Triantopoulou, C., Giannakou, N., Avgerinos, C., & Dervenis, C. (2008). Primary hepatic carcinoid; a diagnostic dilemma: a case report. Cases journal, 1(1), 314. https://doi.org/10.1186/1757-1626-1-314
Touloumis, Zisis, et al. "Primary hepatic carcinoid; a diagnostic dilemma: a case report." Cases journal vol. 1,1 (2008): 314. doi: https://doi.org/10.1186/1757-1626-1-314
Touloumis Z, Delis SG, Triantopoulou C, Giannakou N, Avgerinos C, Dervenis C. Primary hepatic carcinoid; a diagnostic dilemma: a case report. Cases J. 2008 Nov 17;1(1):314. doi: 10.1186/1757-1626-1-314. PMID: 19014620; PMCID: PMC2596793.
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Tryptophan and serotonin turnover rate in the brain of genetically hyperammonemic mice.

Neurochemistry international

Chaouloff F, Laude D, Mignot E, Kamoun P, Elghozi JL.
PMID: 20492910
Neurochem Int. 1985;7(1):143-53. doi: 10.1016/0197-0186(85)90019-1.

An investigation was made into the effects of hyperammonemia on the metabolism of brain serotonin (5-HT). The animal model used was the sparse fur (spf) mouse, which possesses an inborn error of the urea cycle, i.e. an abnormal form...

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Chaouloff F, Laude D, Mignot E, et al. Tryptophan and serotonin turnover rate in the brain of genetically hyperammonemic mice. Neurochem Int. 1985;7(1):143-53doi: 10.1016/0197-0186(85)90019-1.
Chaouloff, F., Laude, D., Mignot, E., Kamoun, P., & Elghozi, J. L. (1985). Tryptophan and serotonin turnover rate in the brain of genetically hyperammonemic mice. Neurochemistry international, 7(1), 143-53. https://doi.org/10.1016/0197-0186(85)90019-1
Chaouloff, F, et al. "Tryptophan and serotonin turnover rate in the brain of genetically hyperammonemic mice." Neurochemistry international vol. 7,1 (1985): 143-53. doi: https://doi.org/10.1016/0197-0186(85)90019-1
Chaouloff F, Laude D, Mignot E, Kamoun P, Elghozi JL. Tryptophan and serotonin turnover rate in the brain of genetically hyperammonemic mice. Neurochem Int. 1985;7(1):143-53. doi: 10.1016/0197-0186(85)90019-1. PMID: 20492910.
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