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Showing 1 to 12 of 37 entries
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Interferons and HIV Infection: The Good, the Bad, and the Ugly.

Pathogens & immunity

Utay NS, Douek DC.
PMID: 27500281
Pathog Immun. 2016;1(1):107-116. doi: 10.20411/pai.v1i1.125.

Whether type I interferons (IFNs) hinder or facilitate HIV disease progression is controversial. Type I IFNs induce the production of restriction factors that protect against mucosal HIV/SIV acquisition and limit virus replication once systemic infection is established. However, type...

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Utay NS, Douek DC. Interferons and HIV Infection: The Good, the Bad, and the Ugly. Pathog Immun. 2016;1(1):107-116doi: 10.20411/pai.v1i1.125.
Utay, N. S., & Douek, D. C. (2016). Interferons and HIV Infection: The Good, the Bad, and the Ugly. Pathogens & immunity, 1(1), 107-116. https://doi.org/10.20411/pai.v1i1.125
Utay, Netanya S, and Douek, Daniel C. "Interferons and HIV Infection: The Good, the Bad, and the Ugly." Pathogens & immunity vol. 1,1 (2016): 107-116. doi: https://doi.org/10.20411/pai.v1i1.125
Utay NS, Douek DC. Interferons and HIV Infection: The Good, the Bad, and the Ugly. Pathog Immun. 2016;1(1):107-116. doi: 10.20411/pai.v1i1.125. PMID: 27500281; PMCID: PMC4972494.
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Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV.

Journal of proteome research

Grossegesse M, Bourquain D, Neumann M, Schaade L, Schulze J, Mache C, Wolff T, Nitsche A, Doellinger J.
PMID: 34982558
J Proteome Res. 2022 Jan 04; doi: 10.1021/acs.jproteome.1c00783. Epub 2022 Jan 04.

Severe acute respiratory syndrome (SARS)-CoV and SARS-CoV-2 infections are characterized by remarkable differences, including infectivity and case fatality rate. The underlying mechanisms are not well understood, illustrating major knowledge gaps of coronavirus biology. In this study, protein expression of...

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Grossegesse M, Bourquain D, Neumann M, et al. Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV. J Proteome Res. 2022;doi: 10.1021/acs.jproteome.1c00783.
Grossegesse, M., Bourquain, D., Neumann, M., Schaade, L., Schulze, J., Mache, C., Wolff, T., Nitsche, A., & Doellinger, J. (2022). Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV. Journal of proteome research, . https://doi.org/10.1021/acs.jproteome.1c00783
Grossegesse, Marica, et al. "Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV." Journal of proteome research vol. (2022). doi: https://doi.org/10.1021/acs.jproteome.1c00783
Grossegesse M, Bourquain D, Neumann M, Schaade L, Schulze J, Mache C, Wolff T, Nitsche A, Doellinger J. Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV. J Proteome Res. 2022 Jan 04; doi: 10.1021/acs.jproteome.1c00783. Epub 2022 Jan 04. PMID: 34982558.
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The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells.

Experimental and molecular pathology

Granqvist V, Holmgren C, Larsson C.
PMID: 35007560
Exp Mol Pathol. 2022 Jan 07;104739. doi: 10.1016/j.yexmp.2021.104739. Epub 2022 Jan 07.

BACKGROUND: Breast cancer is the most common malignancy affecting women. Although the prognosis generally is good, a substantial number of patients still suffer from relapse, emphasizing the need for novel treatments. Smac mimetics were developed to facilitate cell death...

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Granqvist V, Holmgren C, Larsson C. The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells. Exp Mol Pathol. 2022;104739doi: 10.1016/j.yexmp.2021.104739.
Granqvist, V., Holmgren, C., & Larsson, C. (2022). The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells. Experimental and molecular pathology, 104739. https://doi.org/10.1016/j.yexmp.2021.104739
Granqvist, Victoria, et al. "The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells." Experimental and molecular pathology vol. (2022): 104739. doi: https://doi.org/10.1016/j.yexmp.2021.104739
Granqvist V, Holmgren C, Larsson C. The combination of TRAIL and the Smac mimetic LCL-161 induces an irreversible phenotypic change of MCF-7 breast cancer cells. Exp Mol Pathol. 2022 Jan 07;104739. doi: 10.1016/j.yexmp.2021.104739. Epub 2022 Jan 07. PMID: 35007560.
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COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm.

International journal of dermatology

Ionescu MA.
PMID: 34213786
Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02.

BACKGROUND: Several skin manifestations have been reported since the start of the COVID-19 pandemic: chilblains-like, livedoid lesions, urticaria-like, pseudo-Kawasaki disease, and others. Histopathologic images of these lesions most often show aspects of endothelitis, images similar to autoimmune vasculitis. Cutaneous...

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Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2021;61(1):3-6doi: 10.1111/ijd.15749.
Ionescu, M. A. (2022). COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. International journal of dermatology, 61(1), 3-6. https://doi.org/10.1111/ijd.15749
Ionescu, Marius-Anton. "COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm." International journal of dermatology vol. 61,1 (2022): 3-6. doi: https://doi.org/10.1111/ijd.15749
Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02. PMID: 34213786; PMCID: PMC8444652.
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COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm.

International journal of dermatology

Ionescu MA.
PMID: 34213786
Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02.

BACKGROUND: Several skin manifestations have been reported since the start of the COVID-19 pandemic: chilblains-like, livedoid lesions, urticaria-like, pseudo-Kawasaki disease, and others. Histopathologic images of these lesions most often show aspects of endothelitis, images similar to autoimmune vasculitis. Cutaneous...

Cite
Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2021;61(1):3-6doi: 10.1111/ijd.15749.
Ionescu, M. A. (2022). COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. International journal of dermatology, 61(1), 3-6. https://doi.org/10.1111/ijd.15749
Ionescu, Marius-Anton. "COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm." International journal of dermatology vol. 61,1 (2022): 3-6. doi: https://doi.org/10.1111/ijd.15749
Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02. PMID: 34213786; PMCID: PMC8444652.
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COVID-19 in Immunosuppressed Children.

Frontiers in pediatrics

Nicastro E, Verdoni L, Bettini LR, Zuin G, Balduzzi A, Montini G, Biondi A, D'Antiga L.
PMID: 33996683
Front Pediatr. 2021 Apr 29;9:629240. doi: 10.3389/fped.2021.629240. eCollection 2021.

Following the spread of the SARS-CoV-2 infection and coronavirus disease 2019 (COVID-19) to a global pandemic, concerns have arisen for the disease impact in at-risk populations, especially in immunocompromised hosts. On the other hand, clinical studies have clarified that...

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Nicastro E, Verdoni L, Bettini LR, et al. COVID-19 in Immunosuppressed Children. Front Pediatr. 2021;9:629240doi: 10.3389/fped.2021.629240.
Nicastro, E., Verdoni, L., Bettini, L. R., Zuin, G., Balduzzi, A., Montini, G., Biondi, A., & D'Antiga, L. (2021). COVID-19 in Immunosuppressed Children. Frontiers in pediatrics, 9629240. https://doi.org/10.3389/fped.2021.629240
Nicastro, Emanuele, et al. "COVID-19 in Immunosuppressed Children." Frontiers in pediatrics vol. 9 (2021): 629240. doi: https://doi.org/10.3389/fped.2021.629240
Nicastro E, Verdoni L, Bettini LR, Zuin G, Balduzzi A, Montini G, Biondi A, D'Antiga L. COVID-19 in Immunosuppressed Children. Front Pediatr. 2021 Apr 29;9:629240. doi: 10.3389/fped.2021.629240. eCollection 2021. PMID: 33996683; PMCID: PMC8116542.
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Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia.

Frontiers in immunology

Smith JN, Kanwar VS, MacNamara KC.
PMID: 27621733
Front Immunol. 2016 Aug 29;7:330. doi: 10.3389/fimmu.2016.00330. eCollection 2016.

Aplastic anemia (AA) occurs when the bone marrow fails to support production of all three lineages of blood cells, which are necessary for tissue oxygenation, infection control, and hemostasis. The etiology of acquired AA is elusive in the vast...

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Smith JN, Kanwar VS, MacNamara KC. Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia. Front Immunol. 2016;7:330doi: 10.3389/fimmu.2016.00330.
Smith, J. N., Kanwar, V. S., & MacNamara, K. C. (2016). Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia. Frontiers in immunology, 7330. https://doi.org/10.3389/fimmu.2016.00330
Smith, Julianne N P, et al. "Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia." Frontiers in immunology vol. 7 (2016): 330. doi: https://doi.org/10.3389/fimmu.2016.00330
Smith JN, Kanwar VS, MacNamara KC. Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia. Front Immunol. 2016 Aug 29;7:330. doi: 10.3389/fimmu.2016.00330. eCollection 2016. PMID: 27621733; PMCID: PMC5002897.
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Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV.

Journal of proteome research

Grossegesse M, Bourquain D, Neumann M, Schaade L, Schulze J, Mache C, Wolff T, Nitsche A, Doellinger J.
PMID: 34982558
J Proteome Res. 2022 Jan 04; doi: 10.1021/acs.jproteome.1c00783. Epub 2022 Jan 04.

Severe acute respiratory syndrome (SARS)-CoV and SARS-CoV-2 infections are characterized by remarkable differences, including infectivity and case fatality rate. The underlying mechanisms are not well understood, illustrating major knowledge gaps of coronavirus biology. In this study, protein expression of...

Cite
Grossegesse M, Bourquain D, Neumann M, et al. Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV. J Proteome Res. 2022;doi: 10.1021/acs.jproteome.1c00783.
Grossegesse, M., Bourquain, D., Neumann, M., Schaade, L., Schulze, J., Mache, C., Wolff, T., Nitsche, A., & Doellinger, J. (2022). Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV. Journal of proteome research, . https://doi.org/10.1021/acs.jproteome.1c00783
Grossegesse, Marica, et al. "Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV." Journal of proteome research vol. (2022). doi: https://doi.org/10.1021/acs.jproteome.1c00783
Grossegesse M, Bourquain D, Neumann M, Schaade L, Schulze J, Mache C, Wolff T, Nitsche A, Doellinger J. Deep Time Course Proteomics of SARS-CoV- and SARS-CoV-2-Infected Human Lung Epithelial Cells (Calu-3) Reveals Strong Induction of Interferon-Stimulated Gene Expression by SARS-CoV-2 in Contrast to SARS-CoV. J Proteome Res. 2022 Jan 04; doi: 10.1021/acs.jproteome.1c00783. Epub 2022 Jan 04. PMID: 34982558.
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The dUTPase of caprine arthritis-encephalitis virus negatively regulates interferon signaling pathway.

Iranian journal of veterinary research

Shi J, Li X, Zhu M, Chi H, Song Y, Wang J, Huang J.
PMID: 34777521
Iran J Vet Res. 2021;22(3):209-216. doi: 10.22099/ijvr.2021.38240.5568.

BACKGROUND: Deoxyuracil triphosphate nucleotide (dUTP) pyrophosphatase (dUTPase, DU) is an enzyme of caprine arthritis-encephalitis virus (CAEV) that minimizes incorporation of dUTP into the DNA. Caprine arthritis-encephalitis virus relies partly on its ability to escape from innate immunity to cause...

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Shi J, Li X, Zhu M, et al. The dUTPase of caprine arthritis-encephalitis virus negatively regulates interferon signaling pathway. Iran J Vet Res. 2021;22(3):209-216doi: 10.22099/ijvr.2021.38240.5568.
Shi, J., Li, X., Zhu, M., Chi, H., Song, Y., Wang, J., & Huang, J. (2021). The dUTPase of caprine arthritis-encephalitis virus negatively regulates interferon signaling pathway. Iranian journal of veterinary research, 22(3), 209-216. https://doi.org/10.22099/ijvr.2021.38240.5568
Shi, J, et al. "The dUTPase of caprine arthritis-encephalitis virus negatively regulates interferon signaling pathway." Iranian journal of veterinary research vol. 22,3 (2021): 209-216. doi: https://doi.org/10.22099/ijvr.2021.38240.5568
Shi J, Li X, Zhu M, Chi H, Song Y, Wang J, Huang J. The dUTPase of caprine arthritis-encephalitis virus negatively regulates interferon signaling pathway. Iran J Vet Res. 2021;22(3):209-216. doi: 10.22099/ijvr.2021.38240.5568. PMID: 34777521; PMCID: PMC8573393.
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Type I Interferon Impairs Specific Antibody Responses Early during Establishment of LCMV Infection.

Frontiers in immunology

Daugan M, Murira A, Mindt BC, Germain A, Tarrab E, Lapierre P, Fritz JH, Lamarre A.
PMID: 27994594
Front Immunol. 2016 Dec 05;7:564. doi: 10.3389/fimmu.2016.00564. eCollection 2016.

Elicitation of type I interferon (IFN-I) has been shown to both enhance and impair cell-mediated immune responses in acute and persistent viral infections, respectively. Here, we show that, in addition to its effect on T cells, IFN-I drives impairment...

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Daugan M, Murira A, Mindt BC, et al. Type I Interferon Impairs Specific Antibody Responses Early during Establishment of LCMV Infection. Front Immunol. 2016;7:564doi: 10.3389/fimmu.2016.00564.
Daugan, M., Murira, A., Mindt, B. C., Germain, A., Tarrab, E., Lapierre, P., Fritz, J. H., & Lamarre, A. (2016). Type I Interferon Impairs Specific Antibody Responses Early during Establishment of LCMV Infection. Frontiers in immunology, 7564. https://doi.org/10.3389/fimmu.2016.00564
Daugan, Matthieu, et al. "Type I Interferon Impairs Specific Antibody Responses Early during Establishment of LCMV Infection." Frontiers in immunology vol. 7 (2016): 564. doi: https://doi.org/10.3389/fimmu.2016.00564
Daugan M, Murira A, Mindt BC, Germain A, Tarrab E, Lapierre P, Fritz JH, Lamarre A. Type I Interferon Impairs Specific Antibody Responses Early during Establishment of LCMV Infection. Front Immunol. 2016 Dec 05;7:564. doi: 10.3389/fimmu.2016.00564. eCollection 2016. PMID: 27994594; PMCID: PMC5136549.
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Dendritic Cells in Innate and Adaptive Immune Responses against Influenza Virus.

Viruses

Summerfield A, McCullough KC.
PMID: 21994580
Viruses. 2009 Dec;1(3):1022-34. doi: 10.3390/v1031022. Epub 2009 Nov 24.

Dendritic cells (DC) are major players in both innate and adaptive immune responses against influenza virus. These immune responses, as well as the important interface between the innate and adaptive systems, are orchestrated by specialized subsets of DC, including...

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Summerfield A, McCullough KC. Dendritic Cells in Innate and Adaptive Immune Responses against Influenza Virus. Viruses. 2009;1(3):1022-34doi: 10.3390/v1031022.
Summerfield, A., & McCullough, K. C. (2009). Dendritic Cells in Innate and Adaptive Immune Responses against Influenza Virus. Viruses, 1(3), 1022-34. https://doi.org/10.3390/v1031022
Summerfield, Artur, and McCullough, Kenneth C. "Dendritic Cells in Innate and Adaptive Immune Responses against Influenza Virus." Viruses vol. 1,3 (2009): 1022-34. doi: https://doi.org/10.3390/v1031022
Summerfield A, McCullough KC. Dendritic Cells in Innate and Adaptive Immune Responses against Influenza Virus. Viruses. 2009 Dec;1(3):1022-34. doi: 10.3390/v1031022. Epub 2009 Nov 24. PMID: 21994580; PMCID: PMC3185519.
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New insights into an X-traordinary viral protein.

Frontiers in microbiology

Schaller T, Bauby H, Hué S, Malim MH, Goujon C.
PMID: 24782834
Front Microbiol. 2014 Apr 08;5:126. doi: 10.3389/fmicb.2014.00126. eCollection 2014.

Vpx is a protein encoded by members of the HIV-2/SIVsmm and SIVrcm/SIVmnd-2 lineages of primate lentiviruses, and is packaged into viral particles. Vpx plays a critical role during the early steps of the viral life cycle and has been...

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Schaller T, Bauby H, Hué S, et al. New insights into an X-traordinary viral protein. Front Microbiol. 2014;5:126doi: 10.3389/fmicb.2014.00126.
Schaller, T., Bauby, H., Hué, S., Malim, M. H., & Goujon, C. (2014). New insights into an X-traordinary viral protein. Frontiers in microbiology, 5126. https://doi.org/10.3389/fmicb.2014.00126
Schaller, Torsten, et al. "New insights into an X-traordinary viral protein." Frontiers in microbiology vol. 5 (2014): 126. doi: https://doi.org/10.3389/fmicb.2014.00126
Schaller T, Bauby H, Hué S, Malim MH, Goujon C. New insights into an X-traordinary viral protein. Front Microbiol. 2014 Apr 08;5:126. doi: 10.3389/fmicb.2014.00126. eCollection 2014. PMID: 24782834; PMCID: PMC3986551.
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Showing 1 to 12 of 37 entries