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Retraction. Parkinsonism: a clinical marker of exposure to neurotoxins.

Neurotoxicology and teratology

Bleecker ML.
PMID: 2255298
Neurotoxicol Teratol. 1990 Nov-Dec;12(6):569. doi: 10.1016/0892-0362(90)90063-i.

No abstract available.

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Bleecker ML. Retraction. Parkinsonism: a clinical marker of exposure to neurotoxins. Neurotoxicol Teratol. 1990;12(6):569doi: 10.1016/0892-0362(90)90063-i.
Bleecker, M. L. (1990). Retraction. Parkinsonism: a clinical marker of exposure to neurotoxins. Neurotoxicology and teratology, 12(6), 569. https://doi.org/10.1016/0892-0362(90)90063-i
Bleecker, M L. "Retraction. Parkinsonism: a clinical marker of exposure to neurotoxins." Neurotoxicology and teratology vol. 12,6 (1990): 569. doi: https://doi.org/10.1016/0892-0362(90)90063-i
Bleecker ML. Retraction. Parkinsonism: a clinical marker of exposure to neurotoxins. Neurotoxicol Teratol. 1990 Nov-Dec;12(6):569. doi: 10.1016/0892-0362(90)90063-i. PMID: 2255298.
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Protection by glia-conditioned medium in a cell model of Huntington disease.

PLoS currents

Ruiz C, Casarejos MJ, Gomez A, Solano R, de Yebenes JG, Mena MA.
PMID: 22919565
PLoS Curr. 2012 Jul 02;4:e4fbca54a2028b. doi: 10.1371/4fbca54a2028b.

The physiological role of huntingtin and the pathogenic mechanisms that produce the disease are unknown. Mutant huntingtin changes its normal localization and produces cytoplasmic and intranuclear inclusions, changes gene transcription, alters synaptic transmission, impairs mitochondrial activity and activates caspases...

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Ruiz C, Casarejos MJ, Gomez A, et al. Protection by glia-conditioned medium in a cell model of Huntington disease. PLoS Curr. 2012;4:e4fbca54a2028bdoi: 10.1371/4fbca54a2028b.
Ruiz, C., Casarejos, M. J., Gomez, A., Solano, R., de Yebenes, J. G., & Mena, M. A. (2012). Protection by glia-conditioned medium in a cell model of Huntington disease. PLoS currents, 4e4fbca54a2028b. https://doi.org/10.1371/4fbca54a2028b
Ruiz, Carolina, et al. "Protection by glia-conditioned medium in a cell model of Huntington disease." PLoS currents vol. 4 (2012): e4fbca54a2028b. doi: https://doi.org/10.1371/4fbca54a2028b
Ruiz C, Casarejos MJ, Gomez A, Solano R, de Yebenes JG, Mena MA. Protection by glia-conditioned medium in a cell model of Huntington disease. PLoS Curr. 2012 Jul 02;4:e4fbca54a2028b. doi: 10.1371/4fbca54a2028b. PMID: 22919565; PMCID: PMC3423315.
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Determination of the Median Lethal Dose and Electrophoretic Pattern of Hottentotta saulcyi (Scorpiones, Buthidae) Scorpion Venom.

Journal of arthropod-borne diseases

Yağmur EA, Özkan Ö, Karaer KZ.
PMID: 26623435
J Arthropod Borne Dis. 2015 Mar 11;9(2):238-45. eCollection 2015 Dec.

BACKGROUND: In this study, we investigated the lethal potency, electrophoretic protein pattern and in vivo effects of Hottentotta saulcyi scorpion venom in mice.METHODS: Scorpions were collected at night, by using a UV lamp from Mardin Province, Turkey. Venom was...

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Yağmur EA, Özkan Ö, Karaer KZ. Determination of the Median Lethal Dose and Electrophoretic Pattern of Hottentotta saulcyi (Scorpiones, Buthidae) Scorpion Venom. J Arthropod Borne Dis. 2015;9(2):238-45
Yağmur, E. A., Özkan, �. �., & Karaer, K. Z. (2015). Determination of the Median Lethal Dose and Electrophoretic Pattern of Hottentotta saulcyi (Scorpiones, Buthidae) Scorpion Venom. Journal of arthropod-borne diseases, 9(2), 238-45.
Yağmur, Ersen Aydın, et al. "Determination of the Median Lethal Dose and Electrophoretic Pattern of Hottentotta saulcyi (Scorpiones, Buthidae) Scorpion Venom." Journal of arthropod-borne diseases vol. 9,2 (2015): 238-45.
Yağmur EA, Özkan Ö, Karaer KZ. Determination of the Median Lethal Dose and Electrophoretic Pattern of Hottentotta saulcyi (Scorpiones, Buthidae) Scorpion Venom. J Arthropod Borne Dis. 2015 Mar 11;9(2):238-45. eCollection 2015 Dec. PMID: 26623435; PMCID: PMC4662795.
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Enzyme-luminescence method: Tool for real-time monitoring of natural neurotoxins in vitro and l-glutamate release from primary cortical neurons.

Biotechnology reports (Amsterdam, Netherlands)

Zakir Hossain SM.
PMID: 28352593
Biotechnol Rep (Amst). 2016 Jan 18;9:57-65. doi: 10.1016/j.btre.2015.12.002. eCollection 2016 Mar.

Novel enzyme-luminescence method is used for the rapid and sensitive in vitro detection of natural neurotoxins (e.g., shellfish and mushroom toxins) using model brain cells. Paralytic shellfish poisons gonyautoxins (e.g., GTX2,3 and GTX1,4) were detected at 1 nM level...

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Zakir Hossain SM. Enzyme-luminescence method: Tool for real-time monitoring of natural neurotoxins in vitro and l-glutamate release from primary cortical neurons. Biotechnol Rep (Amst). 2016;9:57-65doi: 10.1016/j.btre.2015.12.002.
Zakir Hossain, S. M. (2016). Enzyme-luminescence method: Tool for real-time monitoring of natural neurotoxins in vitro and l-glutamate release from primary cortical neurons. Biotechnology reports (Amsterdam, Netherlands), 957-65. https://doi.org/10.1016/j.btre.2015.12.002
Zakir Hossain, S M. "Enzyme-luminescence method: Tool for real-time monitoring of natural neurotoxins in vitro and l-glutamate release from primary cortical neurons." Biotechnology reports (Amsterdam, Netherlands) vol. 9 (2016): 57-65. doi: https://doi.org/10.1016/j.btre.2015.12.002
Zakir Hossain SM. Enzyme-luminescence method: Tool for real-time monitoring of natural neurotoxins in vitro and l-glutamate release from primary cortical neurons. Biotechnol Rep (Amst). 2016 Jan 18;9:57-65. doi: 10.1016/j.btre.2015.12.002. eCollection 2016 Mar. PMID: 28352593; PMCID: PMC5360985.
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Ganoderma Lucidum polysaccharides protect against MPP(+) and rotenone-induced apoptosis in primary dopaminergic cell cultures through inhibiting oxidative stress.

American journal of neurodegenerative disease

Guo SS, Cui XL, Rausch WD.
PMID: 27335703
Am J Neurodegener Dis. 2016 Jun 01;5(2):131-44. eCollection 2016.

Oxidative stress plays a pivotal role in the progressive neurodegeneration in Parkinson's disease (PD) which is responsible for disabling motor abnormalities in more than 6.5 million people worldwide. Polysaccharides are the main active constituents from Ganoderma lucidum which is...

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Guo SS, Cui XL, Rausch WD. Ganoderma Lucidum polysaccharides protect against MPP(+) and rotenone-induced apoptosis in primary dopaminergic cell cultures through inhibiting oxidative stress. Am J Neurodegener Dis. 2016;5(2):131-44
Guo, S. S., Cui, X. L., & Rausch, W. D. (2016). Ganoderma Lucidum polysaccharides protect against MPP(+) and rotenone-induced apoptosis in primary dopaminergic cell cultures through inhibiting oxidative stress. American journal of neurodegenerative disease, 5(2), 131-44.
Guo, Shan-Shan, et al. "Ganoderma Lucidum polysaccharides protect against MPP(+) and rotenone-induced apoptosis in primary dopaminergic cell cultures through inhibiting oxidative stress." American journal of neurodegenerative disease vol. 5,2 (2016): 131-44.
Guo SS, Cui XL, Rausch WD. Ganoderma Lucidum polysaccharides protect against MPP(+) and rotenone-induced apoptosis in primary dopaminergic cell cultures through inhibiting oxidative stress. Am J Neurodegener Dis. 2016 Jun 01;5(2):131-44. eCollection 2016. PMID: 27335703; PMCID: PMC4913221.
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Can mixtures of cyanotoxins represent a risk to the zooplankton? The case study of Daphnia magna Straus exposed to hepatotoxic and neurotoxic cyanobacterial extracts.

Harmful algae

Freitas EC, Pinheiro C, Rocha O, Loureiro S.
PMID: 28040103
Harmful Algae. 2014 Jan;31:143-152. doi: 10.1016/j.hal.2013.11.004. Epub 2013 Dec 01.

Worldwide, cyanobacterial blooms have been increasing in intensity and frequency, with toxic cyanobacteria sometimes dominant throughout the year in many freshwater bodies. Since the coexistence of more than one type of cyanotoxins in freshwater environments is a common phenomenon,...

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Freitas EC, Pinheiro C, Rocha O, et al. Can mixtures of cyanotoxins represent a risk to the zooplankton? The case study of Daphnia magna Straus exposed to hepatotoxic and neurotoxic cyanobacterial extracts. Harmful Algae. 2013;31:143-152doi: 10.1016/j.hal.2013.11.004.
Freitas, E. C., Pinheiro, C., Rocha, O., & Loureiro, S. (2014). Can mixtures of cyanotoxins represent a risk to the zooplankton? The case study of Daphnia magna Straus exposed to hepatotoxic and neurotoxic cyanobacterial extracts. Harmful algae, 31143-152. https://doi.org/10.1016/j.hal.2013.11.004
Freitas, Emanuela Cristina, et al. "Can mixtures of cyanotoxins represent a risk to the zooplankton? The case study of Daphnia magna Straus exposed to hepatotoxic and neurotoxic cyanobacterial extracts." Harmful algae vol. 31 (2014): 143-152. doi: https://doi.org/10.1016/j.hal.2013.11.004
Freitas EC, Pinheiro C, Rocha O, Loureiro S. Can mixtures of cyanotoxins represent a risk to the zooplankton? The case study of Daphnia magna Straus exposed to hepatotoxic and neurotoxic cyanobacterial extracts. Harmful Algae. 2014 Jan;31:143-152. doi: 10.1016/j.hal.2013.11.004. Epub 2013 Dec 01. PMID: 28040103.
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Using Tinbergen's Four Questions as the Framework for a Neuroscience Capstone Course.

Journal of undergraduate neuroscience education : JUNE : a publication of FUN, Faculty for Undergraduate Neuroscience

Meitzen J.
PMID: 26557795
J Undergrad Neurosci Educ. 2015 Oct 15;14(1):A46-55. eCollection 2015.

Capstone courses for upper-division students are a common feature of the undergraduate neuroscience curriculum. Here is described a method for adapting Nikolaas Tinbergen's four questions to use as a framework for a neuroscience capstone course, in this case with...

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Meitzen J. Using Tinbergen's Four Questions as the Framework for a Neuroscience Capstone Course. J Undergrad Neurosci Educ. 2015;14(1):A46-55
Meitzen, J. (2015). Using Tinbergen's Four Questions as the Framework for a Neuroscience Capstone Course. Journal of undergraduate neuroscience education : JUNE : a publication of FUN, Faculty for Undergraduate Neuroscience, 14(1), A46-55.
Meitzen, John. "Using Tinbergen's Four Questions as the Framework for a Neuroscience Capstone Course." Journal of undergraduate neuroscience education : JUNE : a publication of FUN, Faculty for Undergraduate Neuroscience vol. 14,1 (2015): A46-55.
Meitzen J. Using Tinbergen's Four Questions as the Framework for a Neuroscience Capstone Course. J Undergrad Neurosci Educ. 2015 Oct 15;14(1):A46-55. eCollection 2015. PMID: 26557795; PMCID: PMC4640482.
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Detection of Botulinum Toxins A, B, E, and F in Foods by Endopep-MS.

Journal of agricultural and food chemistry

Kalb SR, Krilich JC, Dykes JK, Lúquez C, Maslanka SE, Barr JR.
PMID: 25578960
J Agric Food Chem. 2015 Feb 04;63(4):1133-1141. doi: 10.1021/jf505482b. Epub 2015 Jan 22.

Botulism is caused by exposure to botulinum neurotoxins (BoNTs). BoNTs are proteins secreted by some species of clostridia; these neurotoxins are known to interfere with nerve impulse transmission, thus causing paralysis. Botulism may be contracted through consumption of food...

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Kalb SR, Krilich JC, Dykes JK, et al. Detection of Botulinum Toxins A, B, E, and F in Foods by Endopep-MS. J Agric Food Chem. 2015;63(4):1133-1141doi: 10.1021/jf505482b.
Kalb, S. R., Krilich, J. C., Dykes, J. K., Lúquez, C., Maslanka, S. E., & Barr, J. R. (2015). Detection of Botulinum Toxins A, B, E, and F in Foods by Endopep-MS. Journal of agricultural and food chemistry, 63(4), 1133-1141. https://doi.org/10.1021/jf505482b
Kalb, Suzanne R, et al. "Detection of Botulinum Toxins A, B, E, and F in Foods by Endopep-MS." Journal of agricultural and food chemistry vol. 63,4 (2015): 1133-1141. doi: https://doi.org/10.1021/jf505482b
Kalb SR, Krilich JC, Dykes JK, Lúquez C, Maslanka SE, Barr JR. Detection of Botulinum Toxins A, B, E, and F in Foods by Endopep-MS. J Agric Food Chem. 2015 Feb 04;63(4):1133-1141. doi: 10.1021/jf505482b. Epub 2015 Jan 22. PMID: 25578960; PMCID: PMC4523457.
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Explaining human recreational use of 'pesticides': The neurotoxin regulation model of substance use vs. the hijack model and implications for age and sex differences in drug consumption.

Frontiers in psychiatry

Hagen EH, Roulette CJ, Sullivan RJ.
PMID: 24204348
Front Psychiatry. 2013 Nov 05;4:142. doi: 10.3389/fpsyt.2013.00142.

Most globally popular drugs are plant neurotoxins or their close chemical analogs. These compounds evolved to deter, not reward or reinforce, consumption. Moreover, they reliably activate virtually all toxin defense mechanisms, and are thus correctly identified by human neurophysiology...

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Hagen EH, Roulette CJ, Sullivan RJ. Explaining human recreational use of 'pesticides': The neurotoxin regulation model of substance use vs. the hijack model and implications for age and sex differences in drug consumption. Front Psychiatry. 2013;4:142doi: 10.3389/fpsyt.2013.00142.
Hagen, E. H., Roulette, C. J., & Sullivan, R. J. (2013). Explaining human recreational use of 'pesticides': The neurotoxin regulation model of substance use vs. the hijack model and implications for age and sex differences in drug consumption. Frontiers in psychiatry, 4142. https://doi.org/10.3389/fpsyt.2013.00142
Hagen, Edward H, et al. "Explaining human recreational use of 'pesticides': The neurotoxin regulation model of substance use vs. the hijack model and implications for age and sex differences in drug consumption." Frontiers in psychiatry vol. 4 (2013): 142. doi: https://doi.org/10.3389/fpsyt.2013.00142
Hagen EH, Roulette CJ, Sullivan RJ. Explaining human recreational use of 'pesticides': The neurotoxin regulation model of substance use vs. the hijack model and implications for age and sex differences in drug consumption. Front Psychiatry. 2013 Nov 05;4:142. doi: 10.3389/fpsyt.2013.00142. PMID: 24204348; PMCID: PMC3817850.
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Modeling Parkinson's disease in the common marmoset (Callithrix jacchus): overview of models, methods, and animal care.

Laboratory animal research

Yun JW, Ahn JB, Kang BC.
PMID: 26755918
Lab Anim Res. 2015 Dec;31(4):155-65. doi: 10.5625/lar.2015.31.4.155. Epub 2015 Dec 22.

The common marmoset (Callithrix jacchus) is a small-bodied, popular New World monkey and is used widely in reproductive biology, neuroscience, and drug development, due to its comparative ease of handling, high reproductive efficiency, and its unique behavioral characters. In...

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Yun JW, Ahn JB, Kang BC. Modeling Parkinson's disease in the common marmoset (Callithrix jacchus): overview of models, methods, and animal care. Lab Anim Res. 2015;31(4):155-65doi: 10.5625/lar.2015.31.4.155.
Yun, J. W., Ahn, J. B., & Kang, B. C. (2015). Modeling Parkinson's disease in the common marmoset (Callithrix jacchus): overview of models, methods, and animal care. Laboratory animal research, 31(4), 155-65. https://doi.org/10.5625/lar.2015.31.4.155
Yun, Jun-Won, et al. "Modeling Parkinson's disease in the common marmoset (Callithrix jacchus): overview of models, methods, and animal care." Laboratory animal research vol. 31,4 (2015): 155-65. doi: https://doi.org/10.5625/lar.2015.31.4.155
Yun JW, Ahn JB, Kang BC. Modeling Parkinson's disease in the common marmoset (Callithrix jacchus): overview of models, methods, and animal care. Lab Anim Res. 2015 Dec;31(4):155-65. doi: 10.5625/lar.2015.31.4.155. Epub 2015 Dec 22. PMID: 26755918; PMCID: PMC4707143.
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New Thoughts on Pathogenesis and Diagnosis of Encephalitis.

Current infectious disease reports

Cassady KA, Whitley RJ.
PMID: 11095786
Curr Infect Dis Rep. 1999 Jun;1(2):178-186. doi: 10.1007/s11908-996-0027-3.

Knowledge regarding the pathogenesis of viral encephalitis, defined as inflammation and destruction of the central nervous system (CNS) from viral infection and the resulting immune response, has improved with advances in molecular biology techniques and recent advances in immunology...

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Cassady KA, Whitley RJ. New Thoughts on Pathogenesis and Diagnosis of Encephalitis. Curr Infect Dis Rep. 1999;1(2):178-186doi: 10.1007/s11908-996-0027-3.
Cassady, K. A., & Whitley, R. J. (1999). New Thoughts on Pathogenesis and Diagnosis of Encephalitis. Current infectious disease reports, 1(2), 178-186. https://doi.org/10.1007/s11908-996-0027-3
Cassady, and Whitley. "New Thoughts on Pathogenesis and Diagnosis of Encephalitis." Current infectious disease reports vol. 1,2 (1999): 178-186. doi: https://doi.org/10.1007/s11908-996-0027-3
Cassady KA, Whitley RJ. New Thoughts on Pathogenesis and Diagnosis of Encephalitis. Curr Infect Dis Rep. 1999 Jun;1(2):178-186. doi: 10.1007/s11908-996-0027-3. PMID: 11095786.
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[Development of an in vitro assay for the detection of Botulinum Neurotoxins].

ALTEX

Gessler F, Behrens S, Loch P, Böhnel H.
PMID: 11178545
ALTEX. 1998;15(5):62-64.

Botulism is a severe, often lethal intoxication, which affects man and animal. It is caused by the neurotoxins (BoNT) of Clostridium botulinum, an anaerobic spore-former. At present the toxin types A-G have been identified. The extreme toxicity of these...

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Gessler F, Behrens S, Loch P, et al. Entwicklung eines in vitro Tests zum Botulinum-Toxinnachweis - erste Ergebnisse. [Development of an in vitro assay for the detection of Botulinum Neurotoxins]. ALTEX. 1998;15(5):62-64
Gessler, F., Behrens, S., Loch, P., & Böhnel, H. (1998). Entwicklung eines in vitro Tests zum Botulinum-Toxinnachweis - erste Ergebnisse. [Development of an in vitro assay for the detection of Botulinum Neurotoxins]. ALTEX, 15(5), 62-64.
Gessler, Frank, et al. "Entwicklung eines in vitro Tests zum Botulinum-Toxinnachweis - erste Ergebnisse." [Development of an in vitro assay for the detection of Botulinum Neurotoxins]. ALTEX vol. 15,5 (1998): 62-64.
Gessler F, Behrens S, Loch P, Böhnel H. Entwicklung eines in vitro Tests zum Botulinum-Toxinnachweis - erste Ergebnisse. [Development of an in vitro assay for the detection of Botulinum Neurotoxins]. ALTEX. 1998;15(5):62-64. PMID: 11178545.
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Showing 1 to 12 of 505 entries