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Showing 1 to 8 of 8 entries
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Resveratrol Prevented Lipopolysaccharide-Induced Endothelial Dysfunction in Rat Thoracic Aorta Through Increased eNOS Expression.

Balkan medical journal

Uğurel SS, Kuşçu N, Özenci ÇÇ, Dalaklıoğlu S, Taşatargil A.
PMID: 27403381
Balkan Med J. 2016 Mar;33(2):138-43. doi: 10.5152/balkanmedj.2016.16879. Epub 2016 Mar 01.

BACKGROUND: The cardiovascular benefits of Resveratrol (RVT) have been well established by previous experimental and clinical studies.AIMS: The goal of this study was to test the effectiveness of RVT administration on the impaired endothelial function induced by lipopolysaccharide (LPS),...

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Uğurel SS, Kuşçu N, Özenci ÇÇ, et al. Resveratrol Prevented Lipopolysaccharide-Induced Endothelial Dysfunction in Rat Thoracic Aorta Through Increased eNOS Expression. Balkan Med J. 2016;33(2):138-43doi: 10.5152/balkanmedj.2016.16879.
Uğurel, S. S., Kuşçu, N., Özenci, �. �. �. �., Dalaklıoğlu, S., & Taşatargil, A. (2016). Resveratrol Prevented Lipopolysaccharide-Induced Endothelial Dysfunction in Rat Thoracic Aorta Through Increased eNOS Expression. Balkan medical journal, 33(2), 138-43. https://doi.org/10.5152/balkanmedj.2016.16879
Uğurel, Seda Sultan, et al. "Resveratrol Prevented Lipopolysaccharide-Induced Endothelial Dysfunction in Rat Thoracic Aorta Through Increased eNOS Expression." Balkan medical journal vol. 33,2 (2016): 138-43. doi: https://doi.org/10.5152/balkanmedj.2016.16879
Uğurel SS, Kuşçu N, Özenci ÇÇ, Dalaklıoğlu S, Taşatargil A. Resveratrol Prevented Lipopolysaccharide-Induced Endothelial Dysfunction in Rat Thoracic Aorta Through Increased eNOS Expression. Balkan Med J. 2016 Mar;33(2):138-43. doi: 10.5152/balkanmedj.2016.16879. Epub 2016 Mar 01. PMID: 27403381; PMCID: PMC4924956.
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Expressions and relationship of Krüppel-like factor 15 and endothelial nitric oxide synthase in experimental deep venous thrombosis.

Annals of translational medicine

Yang X, Xiang Y, Wang F, Cai G, Li Y, Zhong L, Pu L, Yang Y, Song E.
PMID: 33145309
Ann Transl Med. 2020 Sep;8(17):1090. doi: 10.21037/atm-20-5828.

BACKGROUND: Deep vein thrombosis (DVT) is an early postoperative complication. Thrombosis formation, which is potentially life-threatening, seriously affects the rehabilitation of patients after surgery. We aimed to establish a C57 mouse model of DVT and to examine the changes...

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Yang X, Xiang Y, Wang F, et al. Expressions and relationship of Krüppel-like factor 15 and endothelial nitric oxide synthase in experimental deep venous thrombosis. Ann Transl Med. 2020;8(17):1090doi: 10.21037/atm-20-5828.
Yang, X., Xiang, Y., Wang, F., Cai, G., Li, Y., Zhong, L., Pu, L., Yang, Y., & Song, E. (2020). Expressions and relationship of Krüppel-like factor 15 and endothelial nitric oxide synthase in experimental deep venous thrombosis. Annals of translational medicine, 8(17), 1090. https://doi.org/10.21037/atm-20-5828
Yang, Xianguang, et al. "Expressions and relationship of Krüppel-like factor 15 and endothelial nitric oxide synthase in experimental deep venous thrombosis." Annals of translational medicine vol. 8,17 (2020): 1090. doi: https://doi.org/10.21037/atm-20-5828
Yang X, Xiang Y, Wang F, Cai G, Li Y, Zhong L, Pu L, Yang Y, Song E. Expressions and relationship of Krüppel-like factor 15 and endothelial nitric oxide synthase in experimental deep venous thrombosis. Ann Transl Med. 2020 Sep;8(17):1090. doi: 10.21037/atm-20-5828. PMID: 33145309; PMCID: PMC7575959.
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In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms.

Circulation

Ku KH, Dubinsky MK, Sukumar AN, Subramaniam N, Feasson MYM, Nair R, Tran E, Steer BM, Knight BJ, Marsden PA.
PMID: 33910388
Circulation. 2021 Aug 03;144(5):365-381. doi: 10.1161/CIRCULATIONAHA.120.051078. Epub 2021 Apr 29.

BACKGROUND: eNOS (endothelial nitric oxide synthase) is an endothelial cell (EC)-specific gene predominantly expressed in medium- to large-sized arteries where ECs experience atheroprotective laminar flow with high shear stress. Disturbed flow with lower average shear stress decreases eNOS transcription,...

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Ku KH, Dubinsky MK, Sukumar AN, et al. In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms. Circulation. 2021;144(5):365-381doi: 10.1161/CIRCULATIONAHA.120.051078.
Ku, K. H., Dubinsky, M. K., Sukumar, A. N., Subramaniam, N., Feasson, M. Y. M., Nair, R., Tran, E., Steer, B. M., Knight, B. J., & Marsden, P. A. (2021). In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms. Circulation, 144(5), 365-381. https://doi.org/10.1161/CIRCULATIONAHA.120.051078
Ku, Kyung Ha, et al. "In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms." Circulation vol. 144,5 (2021): 365-381. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.051078
Ku KH, Dubinsky MK, Sukumar AN, Subramaniam N, Feasson MYM, Nair R, Tran E, Steer BM, Knight BJ, Marsden PA. In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms. Circulation. 2021 Aug 03;144(5):365-381. doi: 10.1161/CIRCULATIONAHA.120.051078. Epub 2021 Apr 29. PMID: 33910388.
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Anti-hypertensive and endothelia protective effects of Fufang Qima capsule on primary hypertension via adiponectin/adenosine monophosphate activated protein kinase pathway.

Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan

Zhao ZY, Shi ZY, Zhang ZZ, Li YH, Zeng XH, Chen YX, Yao N, Zhou M, Su H, Wang QH, Jin LL.
PMID: 34939388
J Tradit Chin Med. 2021 Dec;41(6):919-926. doi: 10.19852/j.cnki.jtcm.20210618.002.

OBJECTIVE: To investigate the potential mechanism of the vascular remodeling effect and provide additional information about anti-hypertension activity of Fufang Qima capsule.METHODS: Spontaneous hypertensive rats (SHRs) were used to study the underlying mechanism of the anti-hypertension activity of QM....

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Zhao ZY, Shi ZY, Zhang ZZ, et al. Anti-hypertensive and endothelia protective effects of Fufang Qima capsule on primary hypertension via adiponectin/adenosine monophosphate activated protein kinase pathway. J Tradit Chin Med. 2021;41(6):919-926doi: 10.19852/j.cnki.jtcm.20210618.002.
Zhao, Z. Y., Shi, Z. Y., Zhang, Z. Z., Li, Y. H., Zeng, X. H., Chen, Y. X., Yao, N., Zhou, M., Su, H., Wang, Q. H., & Jin, L. L. (2021). Anti-hypertensive and endothelia protective effects of Fufang Qima capsule on primary hypertension via adiponectin/adenosine monophosphate activated protein kinase pathway. Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan, 41(6), 919-926. https://doi.org/10.19852/j.cnki.jtcm.20210618.002
Zhao, Zhi-Yue, et al. "Anti-hypertensive and endothelia protective effects of Fufang Qima capsule on primary hypertension via adiponectin/adenosine monophosphate activated protein kinase pathway." Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan vol. 41,6 (2021): 919-926. doi: https://doi.org/10.19852/j.cnki.jtcm.20210618.002
Zhao ZY, Shi ZY, Zhang ZZ, Li YH, Zeng XH, Chen YX, Yao N, Zhou M, Su H, Wang QH, Jin LL. Anti-hypertensive and endothelia protective effects of Fufang Qima capsule on primary hypertension via adiponectin/adenosine monophosphate activated protein kinase pathway. J Tradit Chin Med. 2021 Dec;41(6):919-926. doi: 10.19852/j.cnki.jtcm.20210618.002. PMID: 34939388.
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Association of Nitric Oxide Levels and Endothelial Nitric Oxide Synthase G894T Polymorphism with Coronary Artery Disease in the Iranian Population.

Vascular specialist international

Mahmoodi K, Nasehi L, Karami E, Soltanpour MS.
PMID: 27699157
Vasc Specialist Int. 2016 Sep;32(3):105-112. doi: 10.5758/vsi.2016.32.3.105. Epub 2016 Sep 30.

PURPOSE: The endothelial nitric oxide synthase (eNOS) G894T polymorphism has been reported to cause endothelial dysfunction and may have a role in the development of coronary artery disease (CAD). The aim of the present study was to investigate the...

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Mahmoodi K, Nasehi L, Karami E, et al. Association of Nitric Oxide Levels and Endothelial Nitric Oxide Synthase G894T Polymorphism with Coronary Artery Disease in the Iranian Population. Vasc Specialist Int. 2016;32(3):105-112doi: 10.5758/vsi.2016.32.3.105.
Mahmoodi, K., Nasehi, L., Karami, E., & Soltanpour, M. S. (2016). Association of Nitric Oxide Levels and Endothelial Nitric Oxide Synthase G894T Polymorphism with Coronary Artery Disease in the Iranian Population. Vascular specialist international, 32(3), 105-112. https://doi.org/10.5758/vsi.2016.32.3.105
Mahmoodi, Khalil, et al. "Association of Nitric Oxide Levels and Endothelial Nitric Oxide Synthase G894T Polymorphism with Coronary Artery Disease in the Iranian Population." Vascular specialist international vol. 32,3 (2016): 105-112. doi: https://doi.org/10.5758/vsi.2016.32.3.105
Mahmoodi K, Nasehi L, Karami E, Soltanpour MS. Association of Nitric Oxide Levels and Endothelial Nitric Oxide Synthase G894T Polymorphism with Coronary Artery Disease in the Iranian Population. Vasc Specialist Int. 2016 Sep;32(3):105-112. doi: 10.5758/vsi.2016.32.3.105. Epub 2016 Sep 30. PMID: 27699157; PMCID: PMC5045252.
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Endothelial nitric oxide synthase (eNOS) 4b/a gene polymorphisms and coronary artery disease: evidence from a meta-analysis.

International journal of molecular sciences

Yang Y, Du K, Liu Z, Lu X.
PMID: 24810690
Int J Mol Sci. 2014 May 07;15(5):7987-8003. doi: 10.3390/ijms15057987.

A variety of studies have suggested that the 4b/a polymorphism in the endothelial nitric oxide synthase (eNOS) was associated with coronary artery disease (CAD) risk. However, the data remain conflicting. The aim of the present meta-analysis was to estimate...

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Yang Y, Du K, Liu Z, et al. Endothelial nitric oxide synthase (eNOS) 4b/a gene polymorphisms and coronary artery disease: evidence from a meta-analysis. Int J Mol Sci. 2014;15(5):7987-8003doi: 10.3390/ijms15057987.
Yang, Y., Du, K., Liu, Z., & Lu, X. (2014). Endothelial nitric oxide synthase (eNOS) 4b/a gene polymorphisms and coronary artery disease: evidence from a meta-analysis. International journal of molecular sciences, 15(5), 7987-8003. https://doi.org/10.3390/ijms15057987
Yang, Yujiao, et al. "Endothelial nitric oxide synthase (eNOS) 4b/a gene polymorphisms and coronary artery disease: evidence from a meta-analysis." International journal of molecular sciences vol. 15,5 (2014): 7987-8003. doi: https://doi.org/10.3390/ijms15057987
Yang Y, Du K, Liu Z, Lu X. Endothelial nitric oxide synthase (eNOS) 4b/a gene polymorphisms and coronary artery disease: evidence from a meta-analysis. Int J Mol Sci. 2014 May 07;15(5):7987-8003. doi: 10.3390/ijms15057987. PMID: 24810690; PMCID: PMC4057714.
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A mathematical model of endothelial nitric oxide synthase activation with time delay exhibiting Hopf bifurcation and oscillations.

Mathematical biosciences

Ritter LR, Chrestensen CA, Salerno JC.
PMID: 27614021
Math Biosci. 2016 Nov;281:62-73. doi: 10.1016/j.mbs.2016.09.003. Epub 2016 Sep 07.

Nitric oxide (NO) is a gaseous compound that serves as a signaling molecule in cellular interactions. In the vasculature, NO is synthesized from endogenous agents by endothelial nitric oxide synthase (eNOS) where it plays key roles in several functions...

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Ritter LR, Chrestensen CA, Salerno JC. A mathematical model of endothelial nitric oxide synthase activation with time delay exhibiting Hopf bifurcation and oscillations. Math Biosci. 2016;281:62-73doi: 10.1016/j.mbs.2016.09.003.
Ritter, L. R., Chrestensen, C. A., & Salerno, J. C. (2016). A mathematical model of endothelial nitric oxide synthase activation with time delay exhibiting Hopf bifurcation and oscillations. Mathematical biosciences, 28162-73. https://doi.org/10.1016/j.mbs.2016.09.003
Ritter, L R, et al. "A mathematical model of endothelial nitric oxide synthase activation with time delay exhibiting Hopf bifurcation and oscillations." Mathematical biosciences vol. 281 (2016): 62-73. doi: https://doi.org/10.1016/j.mbs.2016.09.003
Ritter LR, Chrestensen CA, Salerno JC. A mathematical model of endothelial nitric oxide synthase activation with time delay exhibiting Hopf bifurcation and oscillations. Math Biosci. 2016 Nov;281:62-73. doi: 10.1016/j.mbs.2016.09.003. Epub 2016 Sep 07. PMID: 27614021; PMCID: PMC5067240.
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In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms.

Circulation

Ku KH, Dubinsky MK, Sukumar AN, Subramaniam N, Feasson MYM, Nair R, Tran E, Steer BM, Knight BJ, Marsden PA.
PMID: 33910388
Circulation. 2021 Aug 03;144(5):365-381. doi: 10.1161/CIRCULATIONAHA.120.051078. Epub 2021 Apr 29.

BACKGROUND: eNOS (endothelial nitric oxide synthase) is an endothelial cell (EC)-specific gene predominantly expressed in medium- to large-sized arteries where ECs experience atheroprotective laminar flow with high shear stress. Disturbed flow with lower average shear stress decreases eNOS transcription,...

Cite
Ku KH, Dubinsky MK, Sukumar AN, et al. In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms. Circulation. 2021;144(5):365-381doi: 10.1161/CIRCULATIONAHA.120.051078.
Ku, K. H., Dubinsky, M. K., Sukumar, A. N., Subramaniam, N., Feasson, M. Y. M., Nair, R., Tran, E., Steer, B. M., Knight, B. J., & Marsden, P. A. (2021). In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms. Circulation, 144(5), 365-381. https://doi.org/10.1161/CIRCULATIONAHA.120.051078
Ku, Kyung Ha, et al. "In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms." Circulation vol. 144,5 (2021): 365-381. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.051078
Ku KH, Dubinsky MK, Sukumar AN, Subramaniam N, Feasson MYM, Nair R, Tran E, Steer BM, Knight BJ, Marsden PA. In Vivo Function of Flow-Responsive Cis-DNA Elements of eNOS Gene: A Role for Chromatin-Based Mechanisms. Circulation. 2021 Aug 03;144(5):365-381. doi: 10.1161/CIRCULATIONAHA.120.051078. Epub 2021 Apr 29. PMID: 33910388.
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Showing 1 to 8 of 8 entries