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Obesity-induced hepatic steatosis is mediated by endoplasmic reticulum stress in the subfornical organ of the brain.

JCI insight

Horwath JA, Hurr C, Butler SD, Guruju M, Cassell MD, Mark AL, Davisson RL, Young CN.
PMID: 28422749
JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.90170. eCollection 2017 Apr 20.

Nonalcoholic fatty liver disease (NAFLD), characterized by an excess accumulation of hepatic triglycerides, is a growing health epidemic. While ER stress in the liver has been implicated in the development of NAFLD, the role of brain ER stress -...

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Horwath JA, Hurr C, Butler SD, et al. Obesity-induced hepatic steatosis is mediated by endoplasmic reticulum stress in the subfornical organ of the brain. JCI Insight. 2017;2(8)doi: 10.1172/jci.insight.90170.
Horwath, J. A., Hurr, C., Butler, S. D., Guruju, M., Cassell, M. D., Mark, A. L., Davisson, R. L., & Young, C. N. (2017). Obesity-induced hepatic steatosis is mediated by endoplasmic reticulum stress in the subfornical organ of the brain. JCI insight, 2(8), . https://doi.org/10.1172/jci.insight.90170
Horwath, Julie A, et al. "Obesity-induced hepatic steatosis is mediated by endoplasmic reticulum stress in the subfornical organ of the brain." JCI insight vol. 2,8 (2017). doi: https://doi.org/10.1172/jci.insight.90170
Horwath JA, Hurr C, Butler SD, Guruju M, Cassell MD, Mark AL, Davisson RL, Young CN. Obesity-induced hepatic steatosis is mediated by endoplasmic reticulum stress in the subfornical organ of the brain. JCI Insight. 2017 Apr 20;2(8). doi: 10.1172/jci.insight.90170. eCollection 2017 Apr 20. PMID: 28422749; PMCID: PMC5396512.
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Metabolic effects of a mitochondrial-targeted coenzyme Q analog in high fat fed obese mice.

Pharmacology research & perspectives

Fink BD, Guo DF, Kulkarni CA, Rahmouni K, Kerns RJ, Sivitz WI.
PMID: 28357127
Pharmacol Res Perspect. 2017 Mar 10;5(2):e00301. doi: 10.1002/prp2.301. eCollection 2017 Apr.

We recently reported that mitoquinone (mitoQ, 500 

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Fink BD, Guo DF, Kulkarni CA, et al. Metabolic effects of a mitochondrial-targeted coenzyme Q analog in high fat fed obese mice. Pharmacol Res Perspect. 2017;5(2):e00301doi: 10.1002/prp2.301.
Fink, B. D., Guo, D. F., Kulkarni, C. A., Rahmouni, K., Kerns, R. J., & Sivitz, W. I. (2017). Metabolic effects of a mitochondrial-targeted coenzyme Q analog in high fat fed obese mice. Pharmacology research & perspectives, 5(2), e00301. https://doi.org/10.1002/prp2.301
Fink, Brian D, et al. "Metabolic effects of a mitochondrial-targeted coenzyme Q analog in high fat fed obese mice." Pharmacology research & perspectives vol. 5,2 (2017): e00301. doi: https://doi.org/10.1002/prp2.301
Fink BD, Guo DF, Kulkarni CA, Rahmouni K, Kerns RJ, Sivitz WI. Metabolic effects of a mitochondrial-targeted coenzyme Q analog in high fat fed obese mice. Pharmacol Res Perspect. 2017 Mar 10;5(2):e00301. doi: 10.1002/prp2.301. eCollection 2017 Apr. PMID: 28357127; PMCID: PMC5368965.
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Decidual Cox2 inhibition improves fetal and maternal outcomes in a preeclampsia-like mouse model.

JCI insight

Sones JL, Cha J, Woods AK, Bartos A, Heyward CY, Lob HE, Isroff CE, Butler SD, Shapiro SE, Dey SK, Davisson RL.
PMID: 27159542
JCI Insight. 2016 Mar 17;1(3). doi: 10.1172/jci.insight.75351.

Preeclampsia (PE) is a disorder of pregnancy that manifests as late gestational maternal hypertension and proteinuria and can be life-threatening to both the mother and baby. It is believed that abnormal placentation is responsible for the cascade of events...

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Sones JL, Cha J, Woods AK, et al. Decidual Cox2 inhibition improves fetal and maternal outcomes in a preeclampsia-like mouse model. JCI Insight. 2016;1(3)doi: 10.1172/jci.insight.75351.
Sones, J. L., Cha, J., Woods, A. K., Bartos, A., Heyward, C. Y., Lob, H. E., Isroff, C. E., Butler, S. D., Shapiro, S. E., Dey, S. K., & Davisson, R. L. (2016). Decidual Cox2 inhibition improves fetal and maternal outcomes in a preeclampsia-like mouse model. JCI insight, 1(3), . https://doi.org/10.1172/jci.insight.75351
Sones, Jenny L, et al. "Decidual Cox2 inhibition improves fetal and maternal outcomes in a preeclampsia-like mouse model." JCI insight vol. 1,3 (2016). doi: https://doi.org/10.1172/jci.insight.75351
Sones JL, Cha J, Woods AK, Bartos A, Heyward CY, Lob HE, Isroff CE, Butler SD, Shapiro SE, Dey SK, Davisson RL. Decidual Cox2 inhibition improves fetal and maternal outcomes in a preeclampsia-like mouse model. JCI Insight. 2016 Mar 17;1(3). doi: 10.1172/jci.insight.75351. PMID: 27159542; PMCID: PMC4855694.
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Differential Control of the Sympathetic Nervous System by Leptin: Implications for Obesity.

Clinical and experimental pharmacology & physiology. Supplement

Rahmouni K.
PMID: 21546982
Clin Exp Pharmacol Physiol Suppl. 2007 Nov 01;34:S8-S10. doi: 10.1111/j.1440-1681.2007.04760.x.

1. Leptin is a hormone that is secreted by adipocytes and delivered to the brain to regulate appetite and energy expenditure. Other effects of leptin include activation of the sympathetic nervous system and an increase in arterial pressure.2. Mounting...

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Rahmouni K. Differential Control of the Sympathetic Nervous System by Leptin: Implications for Obesity. Clin Exp Pharmacol Physiol Suppl. 2007;34:S8-S10doi: 10.1111/j.1440-1681.2007.04760.x.
Rahmouni, K. (2007). Differential Control of the Sympathetic Nervous System by Leptin: Implications for Obesity. Clinical and experimental pharmacology & physiology. Supplement, 34S8-S10. https://doi.org/10.1111/j.1440-1681.2007.04760.x
Rahmouni, Kamal. "Differential Control of the Sympathetic Nervous System by Leptin: Implications for Obesity." Clinical and experimental pharmacology & physiology. Supplement vol. 34 (2007): S8-S10. doi: https://doi.org/10.1111/j.1440-1681.2007.04760.x
Rahmouni K. Differential Control of the Sympathetic Nervous System by Leptin: Implications for Obesity. Clin Exp Pharmacol Physiol Suppl. 2007 Nov 01;34:S8-S10. doi: 10.1111/j.1440-1681.2007.04760.x. PMID: 21546982; PMCID: PMC3086057.
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PPARγ: no SirT, no service.

Circulation research

Quelle FW, Sigmund CD.
PMID: 23371897
Circ Res. 2013 Feb 01;112(3):411-4. doi: 10.1161/CIRCRESAHA.113.300870.

No abstract available.

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Quelle FW, Sigmund CD. PPARγ: no SirT, no service. Circ Res. 2013;112(3):411-4doi: 10.1161/CIRCRESAHA.113.300870.
Quelle, F. W., & Sigmund, C. D. (2013). PPARγ: no SirT, no service. Circulation research, 112(3), 411-4. https://doi.org/10.1161/CIRCRESAHA.113.300870
Quelle, Frederick W, and Sigmund, Curt D. "PPARγ: no SirT, no service." Circulation research vol. 112,3 (2013): 411-4. doi: https://doi.org/10.1161/CIRCRESAHA.113.300870
Quelle FW, Sigmund CD. PPARγ: no SirT, no service. Circ Res. 2013 Feb 01;112(3):411-4. doi: 10.1161/CIRCRESAHA.113.300870. PMID: 23371897; PMCID: PMC3580855.
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Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations.

Molecular metabolism

Singh U, Jiang J, Saito K, Toth BA, Dickey JE, Rodeghiero SR, Deng Y, Deng G, Xue B, Zhu Z, Zingman LV, Geerling JC, Cui H.
PMID: 34823066
Mol Metab. 2021 Nov 22;55:101401. doi: 10.1016/j.molmet.2021.101401. Epub 2021 Nov 22.

OBJECTIVE: The paraventricular nucleus of hypothalamus (PVN), an integrative center in the brain, orchestrates a wide range of physiological and behavioral responses. While the PVN melanocortin 4 receptor (MC4R) signaling (PVNMETHODS: Using a combination of viral tools, we mapped...

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Singh U, Jiang J, Saito K, et al. Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations. Mol Metab. 2021;55:101401doi: 10.1016/j.molmet.2021.101401.
Singh, U., Jiang, J., Saito, K., Toth, B. A., Dickey, J. E., Rodeghiero, S. R., Deng, Y., Deng, G., Xue, B., Zhu, Z., Zingman, L. V., Geerling, J. C., & Cui, H. (2021). Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations. Molecular metabolism, 55101401. https://doi.org/10.1016/j.molmet.2021.101401
Singh, Uday, et al. "Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations." Molecular metabolism vol. 55 (2021): 101401. doi: https://doi.org/10.1016/j.molmet.2021.101401
Singh U, Jiang J, Saito K, Toth BA, Dickey JE, Rodeghiero SR, Deng Y, Deng G, Xue B, Zhu Z, Zingman LV, Geerling JC, Cui H. Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations. Mol Metab. 2021 Nov 22;55:101401. doi: 10.1016/j.molmet.2021.101401. Epub 2021 Nov 22. PMID: 34823066; PMCID: PMC8689242.
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BBSome: a New Player in Hypertension and Other Cardiovascular Risks.

Hypertension (Dallas, Tex. : 1979)

Zhao Y, Rahmouni K.
PMID: 34865504
Hypertension. 2022 Feb;79(2):303-313. doi: 10.1161/HYPERTENSIONAHA.121.17946. Epub 2021 Dec 06.

The BBSome is an octameric protein complex involved in Bardet-Biedl syndrome (BBS), a human pleiotropic, autosomal recessive condition. Patients with BBS display various clinical features including obesity, hypertension, and renal abnormalities. Association studies have also linked the

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Zhao Y, Rahmouni K. BBSome: a New Player in Hypertension and Other Cardiovascular Risks. Hypertension. 2021;79(2):303-313doi: 10.1161/HYPERTENSIONAHA.121.17946.
Zhao, Y., & Rahmouni, K. (2022). BBSome: a New Player in Hypertension and Other Cardiovascular Risks. Hypertension (Dallas, Tex. : 1979), 79(2), 303-313. https://doi.org/10.1161/HYPERTENSIONAHA.121.17946
Zhao, Yuying, and Rahmouni, Kamal. "BBSome: a New Player in Hypertension and Other Cardiovascular Risks." Hypertension (Dallas, Tex. : 1979) vol. 79,2 (2022): 303-313. doi: https://doi.org/10.1161/HYPERTENSIONAHA.121.17946
Zhao Y, Rahmouni K. BBSome: a New Player in Hypertension and Other Cardiovascular Risks. Hypertension. 2022 Feb;79(2):303-313. doi: 10.1161/HYPERTENSIONAHA.121.17946. Epub 2021 Dec 06. PMID: 34865504; PMCID: PMC8755612.
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Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations.

Molecular metabolism

Singh U, Jiang J, Saito K, Toth BA, Dickey JE, Rodeghiero SR, Deng Y, Deng G, Xue B, Zhu Z, Zingman LV, Geerling JC, Cui H.
PMID: 34823066
Mol Metab. 2022 Jan;55:101401. doi: 10.1016/j.molmet.2021.101401. Epub 2021 Nov 22.

OBJECTIVE: The paraventricular nucleus of hypothalamus (PVN), an integrative center in the brain, orchestrates a wide range of physiological and behavioral responses. While the PVN melanocortin 4 receptor (MC4R) signaling (PVNMETHODS: Using a combination of viral tools, we mapped...

Cite
Singh U, Jiang J, Saito K, et al. Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations. Mol Metab. 2021;55:101401doi: 10.1016/j.molmet.2021.101401.
Singh, U., Jiang, J., Saito, K., Toth, B. A., Dickey, J. E., Rodeghiero, S. R., Deng, Y., Deng, G., Xue, B., Zhu, Z., Zingman, L. V., Geerling, J. C., & Cui, H. (2022). Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations. Molecular metabolism, 55101401. https://doi.org/10.1016/j.molmet.2021.101401
Singh, Uday, et al. "Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations." Molecular metabolism vol. 55 (2022): 101401. doi: https://doi.org/10.1016/j.molmet.2021.101401
Singh U, Jiang J, Saito K, Toth BA, Dickey JE, Rodeghiero SR, Deng Y, Deng G, Xue B, Zhu Z, Zingman LV, Geerling JC, Cui H. Neuroanatomical organization and functional roles of PVN MC4R pathways in physiological and behavioral regulations. Mol Metab. 2022 Jan;55:101401. doi: 10.1016/j.molmet.2021.101401. Epub 2021 Nov 22. PMID: 34823066; PMCID: PMC8689242.
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Role of the Peroxisome Proliferator Activated Receptors in Hypertension.

Circulation research

Fang S, Livergood MC, Nakagawa P, Wu J, Sigmund CD.
PMID: 33793338
Circ Res. 2021 Apr 02;128(7):1021-1039. doi: 10.1161/CIRCRESAHA.120.318062. Epub 2021 Apr 01.

Nuclear receptors represent a large family of ligand-activated transcription factors which sense the physiological environment and make long-term adaptations by mediating changes in gene expression. In this review, we will first discuss the fundamental mechanisms by which nuclear receptors...

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Fang S, Livergood MC, Nakagawa P, et al. Role of the Peroxisome Proliferator Activated Receptors in Hypertension. Circ Res. 2021;128(7):1021-1039doi: 10.1161/CIRCRESAHA.120.318062.
Fang, S., Livergood, M. C., Nakagawa, P., Wu, J., & Sigmund, C. D. (2021). Role of the Peroxisome Proliferator Activated Receptors in Hypertension. Circulation research, 128(7), 1021-1039. https://doi.org/10.1161/CIRCRESAHA.120.318062
Fang, Shi, et al. "Role of the Peroxisome Proliferator Activated Receptors in Hypertension." Circulation research vol. 128,7 (2021): 1021-1039. doi: https://doi.org/10.1161/CIRCRESAHA.120.318062
Fang S, Livergood MC, Nakagawa P, Wu J, Sigmund CD. Role of the Peroxisome Proliferator Activated Receptors in Hypertension. Circ Res. 2021 Apr 02;128(7):1021-1039. doi: 10.1161/CIRCRESAHA.120.318062. Epub 2021 Apr 01. PMID: 33793338; PMCID: PMC8020861.
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Breaking a Mother's Heart: Circulating Antiangiogenic Factors and Hypertension During Pregnancy Correlate With Specific Cardiac Dysfunctions.

Hypertension (Dallas, Tex. : 1979)

Sandgren JA, Santillan MK, Grobe JL.
PMID: 27113050
Hypertension. 2016 Jun;67(6):1119-20. doi: 10.1161/HYPERTENSIONAHA.116.07380. Epub 2016 Apr 25.

No abstract available.

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Sandgren JA, Santillan MK, Grobe JL. Breaking a Mother's Heart: Circulating Antiangiogenic Factors and Hypertension During Pregnancy Correlate With Specific Cardiac Dysfunctions. Hypertension. 2016;67(6):1119-20doi: 10.1161/HYPERTENSIONAHA.116.07380.
Sandgren, J. A., Santillan, M. K., & Grobe, J. L. (2016). Breaking a Mother's Heart: Circulating Antiangiogenic Factors and Hypertension During Pregnancy Correlate With Specific Cardiac Dysfunctions. Hypertension (Dallas, Tex. : 1979), 67(6), 1119-20. https://doi.org/10.1161/HYPERTENSIONAHA.116.07380
Sandgren, Jeremy A, et al. "Breaking a Mother's Heart: Circulating Antiangiogenic Factors and Hypertension During Pregnancy Correlate With Specific Cardiac Dysfunctions." Hypertension (Dallas, Tex. : 1979) vol. 67,6 (2016): 1119-20. doi: https://doi.org/10.1161/HYPERTENSIONAHA.116.07380
Sandgren JA, Santillan MK, Grobe JL. Breaking a Mother's Heart: Circulating Antiangiogenic Factors and Hypertension During Pregnancy Correlate With Specific Cardiac Dysfunctions. Hypertension. 2016 Jun;67(6):1119-20. doi: 10.1161/HYPERTENSIONAHA.116.07380. Epub 2016 Apr 25. PMID: 27113050; PMCID: PMC4865412.
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Reply to Petersen et al.: An alternative hypothesis for why exposure to static magnetic and electric fields treats type 2 diabetes.

American journal of physiology. Endocrinology and metabolism

Carter CS, Huang SC, Searby CC, Cassaidy B, Miller MJ, Grzesik WJ, Piorczynski TB, Pak TK, Walsh SA, Acevedo M, Zhang Q, Mapuskar KA, Milne GL, Hinton AO, Guo DF, Weiss R, Bradberry K, Taylor EB, Rauckhorst AJ, Dick DW, Akurathi V, Falls-Hubert KC, Wagner BA, Carter WA, Wang K, Norris AW, Rahmouni K, Buettner GR, Hansen JM, Spitz DR, Abel ED, Sheffield VC.
PMID: 33843283
Am J Physiol Endocrinol Metab. 2021 May 01;320(5):E1004-E1005. doi: 10.1152/ajpendo.00119.2021. Epub 2021 Apr 12.

No abstract available.

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Carter CS, Huang SC, Searby CC, et al. Reply to Petersen et al.: An alternative hypothesis for why exposure to static magnetic and electric fields treats type 2 diabetes. Am J Physiol Endocrinol Metab. 2021;320(5):E1004-E1005doi: 10.1152/ajpendo.00119.2021.
Carter, C. S., Huang, S. C., Searby, C. C., Cassaidy, B., Miller, M. J., Grzesik, W. J., Piorczynski, T. B., Pak, T. K., Walsh, S. A., Acevedo, M., Zhang, Q., Mapuskar, K. A., Milne, G. L., Hinton, A. O., Guo, D. F., Weiss, R., Bradberry, K., Taylor, E. B., Rauckhorst, A. J., Dick, D. W., Akurathi, V., Falls-Hubert, K. C., Wagner, B. A., Carter, W. A., Wang, K., Norris, A. W., Rahmouni, K., Buettner, G. R., Hansen, J. M., Spitz, D. R., Abel, E. D., & Sheffield, V. C. (2021). Reply to Petersen et al.: An alternative hypothesis for why exposure to static magnetic and electric fields treats type 2 diabetes. American journal of physiology. Endocrinology and metabolism, 320(5), E1004-E1005. https://doi.org/10.1152/ajpendo.00119.2021
Carter, Calvin S, et al. "Reply to Petersen et al.: An alternative hypothesis for why exposure to static magnetic and electric fields treats type 2 diabetes." American journal of physiology. Endocrinology and metabolism vol. 320,5 (2021): E1004-E1005. doi: https://doi.org/10.1152/ajpendo.00119.2021
Carter CS, Huang SC, Searby CC, Cassaidy B, Miller MJ, Grzesik WJ, Piorczynski TB, Pak TK, Walsh SA, Acevedo M, Zhang Q, Mapuskar KA, Milne GL, Hinton AO, Guo DF, Weiss R, Bradberry K, Taylor EB, Rauckhorst AJ, Dick DW, Akurathi V, Falls-Hubert KC, Wagner BA, Carter WA, Wang K, Norris AW, Rahmouni K, Buettner GR, Hansen JM, Spitz DR, Abel ED, Sheffield VC. Reply to Petersen et al.: An alternative hypothesis for why exposure to static magnetic and electric fields treats type 2 diabetes. Am J Physiol Endocrinol Metab. 2021 May 01;320(5):E1004-E1005. doi: 10.1152/ajpendo.00119.2021. Epub 2021 Apr 12. PMID: 33843283; PMCID: PMC8238129.
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Intracranial Pressure Is a Determinant of Sympathetic Activity.

Frontiers in physiology

Schmidt EA, Despas F, Pavy-Le Traon A, Czosnyka Z, Pickard JD, Rahmouni K, Pathak A, Senard JM.
PMID: 29472865
Front Physiol. 2018 Feb 08;9:11. doi: 10.3389/fphys.2018.00011. eCollection 2018.

Intracranial pressure (ICP) is the pressure within the

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Schmidt EA, Despas F, Pavy-Le Traon A, et al. Intracranial Pressure Is a Determinant of Sympathetic Activity. Front Physiol. 2018;9:11doi: 10.3389/fphys.2018.00011.
Schmidt, E. A., Despas, F., Pavy-Le Traon, A., Czosnyka, Z., Pickard, J. D., Rahmouni, K., Pathak, A., & Senard, J. M. (2018). Intracranial Pressure Is a Determinant of Sympathetic Activity. Frontiers in physiology, 911. https://doi.org/10.3389/fphys.2018.00011
Schmidt, Eric A, et al. "Intracranial Pressure Is a Determinant of Sympathetic Activity." Frontiers in physiology vol. 9 (2018): 11. doi: https://doi.org/10.3389/fphys.2018.00011
Schmidt EA, Despas F, Pavy-Le Traon A, Czosnyka Z, Pickard JD, Rahmouni K, Pathak A, Senard JM. Intracranial Pressure Is a Determinant of Sympathetic Activity. Front Physiol. 2018 Feb 08;9:11. doi: 10.3389/fphys.2018.00011. eCollection 2018. PMID: 29472865; PMCID: PMC5809772.
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