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Identification of collaborative activities with oxidative phosphorylation in bipolar disorder.

Bioinformation

Sawai H, Takai-Igarashi T, Tanaka H.
PMID: 26124562
Bioinformation. 2015 Apr 30;11(4):207-16. doi: 10.6026/97320630011207. eCollection 2015.

UNLABELLED: Bipolar disorder (BD) is a psychiatric disease considered to polygenic with multiple factors in genetics, each of which is not dominant but collaborative during pathogenic progression. We describe a method that estimates the collaborative contribution to the disease...

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Sawai H, Takai-Igarashi T, Tanaka H. Identification of collaborative activities with oxidative phosphorylation in bipolar disorder. Bioinformation. 2015;11(4):207-16doi: 10.6026/97320630011207.
Sawai, H., Takai-Igarashi, T., & Tanaka, H. (2015). Identification of collaborative activities with oxidative phosphorylation in bipolar disorder. Bioinformation, 11(4), 207-16. https://doi.org/10.6026/97320630011207
Sawai, Hashime, et al. "Identification of collaborative activities with oxidative phosphorylation in bipolar disorder." Bioinformation vol. 11,4 (2015): 207-16. doi: https://doi.org/10.6026/97320630011207
Sawai H, Takai-Igarashi T, Tanaka H. Identification of collaborative activities with oxidative phosphorylation in bipolar disorder. Bioinformation. 2015 Apr 30;11(4):207-16. doi: 10.6026/97320630011207. eCollection 2015. PMID: 26124562; PMCID: PMC4479050.
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PP2A-mTOR-p70S6K/4E-BP1 axis regulates M1 polarization of pulmonary macrophages and promotes ambient particulate matter induced mouse lung injury.

Journal of hazardous materials

Chen S, Chen L, Ye L, Jiang Y, Li Q, Zhang H, Zhang R, Li H, Yu D, Zhang R, Niu Y, Zhao Q, Liu J, Ouyang G, Aschner M, Zheng Y, Zhang L, Chen W, Li D.
PMID: 34740159
J Hazard Mater. 2021 Oct 29;424:127624. doi: 10.1016/j.jhazmat.2021.127624. Epub 2021 Oct 29.

To identify key signaling pathways involved in ambient particulate matter (PM)-induced pulmonary injury, we generated a mouse model with myeloid-specific deletion of Ppp2r1a gene (encoding protein phosphatase 2 A (PP2A) A subunit), and conducted experiments in a real-ambient PM...

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Chen S, Chen L, Ye L, et al. PP2A-mTOR-p70S6K/4E-BP1 axis regulates M1 polarization of pulmonary macrophages and promotes ambient particulate matter induced mouse lung injury. J Hazard Mater. 2021;424:127624doi: 10.1016/j.jhazmat.2021.127624.
Chen, S., Chen, L., Ye, L., Jiang, Y., Li, Q., Zhang, H., Zhang, R., Li, H., Yu, D., Zhang, R., Niu, Y., Zhao, Q., Liu, J., Ouyang, G., Aschner, M., Zheng, Y., Zhang, L., Chen, W., & Li, D. (2021). PP2A-mTOR-p70S6K/4E-BP1 axis regulates M1 polarization of pulmonary macrophages and promotes ambient particulate matter induced mouse lung injury. Journal of hazardous materials, 424127624. https://doi.org/10.1016/j.jhazmat.2021.127624
Chen, Shen, et al. "PP2A-mTOR-p70S6K/4E-BP1 axis regulates M1 polarization of pulmonary macrophages and promotes ambient particulate matter induced mouse lung injury." Journal of hazardous materials vol. 424 (2021): 127624. doi: https://doi.org/10.1016/j.jhazmat.2021.127624
Chen S, Chen L, Ye L, Jiang Y, Li Q, Zhang H, Zhang R, Li H, Yu D, Zhang R, Niu Y, Zhao Q, Liu J, Ouyang G, Aschner M, Zheng Y, Zhang L, Chen W, Li D. PP2A-mTOR-p70S6K/4E-BP1 axis regulates M1 polarization of pulmonary macrophages and promotes ambient particulate matter induced mouse lung injury. J Hazard Mater. 2021 Oct 29;424:127624. doi: 10.1016/j.jhazmat.2021.127624. Epub 2021 Oct 29. PMID: 34740159.
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miR-762 Promotes Malignant Development of Head and Neck Squamous Cell Carcinoma by Targeting PHLPP2 and FOXO4.

OncoTargets and therapy

Chen S, Zhang JY, Sun LS, Li XF, Bai JY, Zhang HY, Li TJ.
PMID: 31920332
Onco Targets Ther. 2019 Dec 24;12:11425-11436. doi: 10.2147/OTT.S221442. eCollection 2019.

BACKGROUND: Head and neck squamous cell carcinoma (HNSCC) is among the most common malignant tumors worldwide. This study, investigated the role of microRNA (miR)-762 in regulating HNSCC progression.MATERIALS AND METHODS: The expression levels of miR-762 in HNSCC tissues were...

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Chen S, Zhang JY, Sun LS, et al. miR-762 Promotes Malignant Development of Head and Neck Squamous Cell Carcinoma by Targeting PHLPP2 and FOXO4. Onco Targets Ther. 2019;12:11425-11436doi: 10.2147/OTT.S221442.
Chen, S., Zhang, J. Y., Sun, L. S., Li, X. F., Bai, J. Y., Zhang, H. Y., & Li, T. J. (2019). miR-762 Promotes Malignant Development of Head and Neck Squamous Cell Carcinoma by Targeting PHLPP2 and FOXO4. OncoTargets and therapy, 1211425-11436. https://doi.org/10.2147/OTT.S221442
Chen, Shuai, et al. "miR-762 Promotes Malignant Development of Head and Neck Squamous Cell Carcinoma by Targeting PHLPP2 and FOXO4." OncoTargets and therapy vol. 12 (2019): 11425-11436. doi: https://doi.org/10.2147/OTT.S221442
Chen S, Zhang JY, Sun LS, Li XF, Bai JY, Zhang HY, Li TJ. miR-762 Promotes Malignant Development of Head and Neck Squamous Cell Carcinoma by Targeting PHLPP2 and FOXO4. Onco Targets Ther. 2019 Dec 24;12:11425-11436. doi: 10.2147/OTT.S221442. eCollection 2019. PMID: 31920332; PMCID: PMC6935361.
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High expression of protein phosphatase 2 regulatory subunit B'' alpha predicts poor outcome in hepatocellular carcinoma patients after liver transplantation.

World journal of gastrointestinal oncology

He JJ, Shang L, Yu QW, Jiao N, Qiu S, Zhu WX, Wu DF, Tian YE, Zhang Q.
PMID: 34322200
World J Gastrointest Oncol. 2021 Jul 15;13(7):716-731. doi: 10.4251/wjgo.v13.i7.716.

BACKGROUND: Protein phosphatase 2 regulatory subunit B'' alpha (AIM: To investigate whether the METHODS: Diseased liver tissues of HCC patients after LT were collected as well as their clinical data and follow-up information. The immunohistochemical method was used to...

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He JJ, Shang L, Yu QW, et al. High expression of protein phosphatase 2 regulatory subunit B'' alpha predicts poor outcome in hepatocellular carcinoma patients after liver transplantation. World J Gastrointest Oncol. 2021;13(7):716-731doi: 10.4251/wjgo.v13.i7.716.
He, J. J., Shang, L., Yu, Q. W., Jiao, N., Qiu, S., Zhu, W. X., Wu, D. F., Tian, Y. E., & Zhang, Q. (2021). High expression of protein phosphatase 2 regulatory subunit B'' alpha predicts poor outcome in hepatocellular carcinoma patients after liver transplantation. World journal of gastrointestinal oncology, 13(7), 716-731. https://doi.org/10.4251/wjgo.v13.i7.716
He, Jia-Jia, et al. "High expression of protein phosphatase 2 regulatory subunit B'' alpha predicts poor outcome in hepatocellular carcinoma patients after liver transplantation." World journal of gastrointestinal oncology vol. 13,7 (2021): 716-731. doi: https://doi.org/10.4251/wjgo.v13.i7.716
He JJ, Shang L, Yu QW, Jiao N, Qiu S, Zhu WX, Wu DF, Tian YE, Zhang Q. High expression of protein phosphatase 2 regulatory subunit B'' alpha predicts poor outcome in hepatocellular carcinoma patients after liver transplantation. World J Gastrointest Oncol. 2021 Jul 15;13(7):716-731. doi: 10.4251/wjgo.v13.i7.716. PMID: 34322200; PMCID: PMC8299934.
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Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring.

Brain research bulletin

Wu Q, Zhou D, Wu R, Shi R, Shen X, Jin N, Gu J, Gu JH, Liu F, Chu D.
PMID: 34808323
Brain Res Bull. 2022 Jan;178:133-143. doi: 10.1016/j.brainresbull.2021.11.008. Epub 2021 Nov 19.

Folic acid (FA) supplementation in early pregnancy is recommended to protect against birth defects. But excess FA has exhibited neurodevelopmental toxicity. We previously reported that the mice treated with 2.5-fold the dietary requirement of FA one week before mating...

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Wu Q, Zhou D, Wu R, et al. Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring. Brain Res Bull. 2021;178:133-143doi: 10.1016/j.brainresbull.2021.11.008.
Wu, Q., Zhou, D., Wu, R., Shi, R., Shen, X., Jin, N., Gu, J., Gu, J. H., Liu, F., & Chu, D. (2022). Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring. Brain research bulletin, 178133-143. https://doi.org/10.1016/j.brainresbull.2021.11.008
Wu, Qian, et al. "Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring." Brain research bulletin vol. 178 (2022): 133-143. doi: https://doi.org/10.1016/j.brainresbull.2021.11.008
Wu Q, Zhou D, Wu R, Shi R, Shen X, Jin N, Gu J, Gu JH, Liu F, Chu D. Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring. Brain Res Bull. 2022 Jan;178:133-143. doi: 10.1016/j.brainresbull.2021.11.008. Epub 2021 Nov 19. PMID: 34808323.
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Developmental and light regulation of tumor suppressor protein PP2A in the retina.

Oncotarget

Rajala A, Wang Y, Abcouwer SF, Gardner TW, Rajala RVS.
PMID: 29416710
Oncotarget. 2017 Dec 17;9(2):1505-1523. doi: 10.18632/oncotarget.23351. eCollection 2018 Jan 05.

Protein phosphatases are a group of universal enzymes that are responsible for the dephosphorylation of various proteins and enzymes in cells. Cellular signal transduction events are largely governed by the phosphorylation of key proteins. The length of cellular response...

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Rajala A, Wang Y, Abcouwer SF, et al. Developmental and light regulation of tumor suppressor protein PP2A in the retina. Oncotarget. 2017;9(2):1505-1523doi: 10.18632/oncotarget.23351.
Rajala, A., Wang, Y., Abcouwer, S. F., Gardner, T. W., & Rajala, R. V. S. (2018). Developmental and light regulation of tumor suppressor protein PP2A in the retina. Oncotarget, 9(2), 1505-1523. https://doi.org/10.18632/oncotarget.23351
Rajala, Ammaji, et al. "Developmental and light regulation of tumor suppressor protein PP2A in the retina." Oncotarget vol. 9,2 (2018): 1505-1523. doi: https://doi.org/10.18632/oncotarget.23351
Rajala A, Wang Y, Abcouwer SF, Gardner TW, Rajala RVS. Developmental and light regulation of tumor suppressor protein PP2A in the retina. Oncotarget. 2017 Dec 17;9(2):1505-1523. doi: 10.18632/oncotarget.23351. eCollection 2018 Jan 05. PMID: 29416710; PMCID: PMC5788578.
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Glycogen synthase kinase-3β activity plays a key role in the antitumor effect of nafamostat mesilate in pancreatic cancer cells.

Annals of gastroenterological surgery

Haruki K, Shiba H, Shimada Y, Shirai Y, Iwase R, Fujiwara Y, Uwagawa T, Ohashi T, Yanaga K.
PMID: 29863120
Ann Gastroenterol Surg. 2017 Aug 31;2(1):65-71. doi: 10.1002/ags3.12025. eCollection 2018 Jan.

Pancreatic cancer is often resistant to chemotherapy. We previously showed the efficacy of combination treatment using gemcitabine and nafamostat mesilate (FUT-175) for patients with unresectable pancreatic cancer. However, the mechanisms that affect the sensitivity of FUT-175 are not fully...

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Haruki K, Shiba H, Shimada Y, et al. Glycogen synthase kinase-3β activity plays a key role in the antitumor effect of nafamostat mesilate in pancreatic cancer cells. Ann Gastroenterol Surg. 2017;2(1):65-71doi: 10.1002/ags3.12025.
Haruki, K., Shiba, H., Shimada, Y., Shirai, Y., Iwase, R., Fujiwara, Y., Uwagawa, T., Ohashi, T., & Yanaga, K. (2018). Glycogen synthase kinase-3β activity plays a key role in the antitumor effect of nafamostat mesilate in pancreatic cancer cells. Annals of gastroenterological surgery, 2(1), 65-71. https://doi.org/10.1002/ags3.12025
Haruki, Koichiro, et al. "Glycogen synthase kinase-3β activity plays a key role in the antitumor effect of nafamostat mesilate in pancreatic cancer cells." Annals of gastroenterological surgery vol. 2,1 (2018): 65-71. doi: https://doi.org/10.1002/ags3.12025
Haruki K, Shiba H, Shimada Y, Shirai Y, Iwase R, Fujiwara Y, Uwagawa T, Ohashi T, Yanaga K. Glycogen synthase kinase-3β activity plays a key role in the antitumor effect of nafamostat mesilate in pancreatic cancer cells. Ann Gastroenterol Surg. 2017 Aug 31;2(1):65-71. doi: 10.1002/ags3.12025. eCollection 2018 Jan. PMID: 29863120; PMCID: PMC5868870.
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AMBRA1: When autophagy meets cell proliferation.

Autophagy

Cianfanelli V, Cecconi F.
PMID: 26101901
Autophagy. 2015;11(9):1705-7. doi: 10.1080/15548627.2015.1053681.

A growing amount of evidence reported in the literature in recent years strongly supports the relevance of the interplay between autophagy and other pathways. In this context, the study of the link between autophagy and cell proliferation regulation has...

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Cianfanelli V, Cecconi F. AMBRA1: When autophagy meets cell proliferation. Autophagy. 2015;11(9):1705-7doi: 10.1080/15548627.2015.1053681.
Cianfanelli, V., & Cecconi, F. (2015). AMBRA1: When autophagy meets cell proliferation. Autophagy, 11(9), 1705-7. https://doi.org/10.1080/15548627.2015.1053681
Cianfanelli, Valentina, and Cecconi, Francesco. "AMBRA1: When autophagy meets cell proliferation." Autophagy vol. 11,9 (2015): 1705-7. doi: https://doi.org/10.1080/15548627.2015.1053681
Cianfanelli V, Cecconi F. AMBRA1: When autophagy meets cell proliferation. Autophagy. 2015;11(9):1705-7. doi: 10.1080/15548627.2015.1053681. PMID: 26101901; PMCID: PMC4590640.
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Midline 1 controls polarization and migration of murine cytotoxic T cells.

Immunity, inflammation and disease

Boding L, Hansen AK, Nielsen MM, Meroni G, Braunstein TH, Woetmann A, Ødum N, Bonefeld CM, Geisler C.
PMID: 25866633
Immun Inflamm Dis. 2014 Dec;2(4):262-71. doi: 10.1002/iid3.44. Epub 2015 Jan 27.

Midline 1 (MID1) is a microtubule-associated ubiquitin ligase that regulates protein phosphatase 2 A levels. Loss-of-function mutations in MID1 lead to the human X-linked Opitz G/BBB (OS) syndrome characterized by defective midline development during embryogenesis. We have recently shown...

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Boding L, Hansen AK, Nielsen MM, et al. Midline 1 controls polarization and migration of murine cytotoxic T cells. Immun Inflamm Dis. 2015;2(4):262-71doi: 10.1002/iid3.44.
Boding, L., Hansen, A. K., Nielsen, M. M., Meroni, G., Braunstein, T. H., Woetmann, A., Ødum, N., Bonefeld, C. M., & Geisler, C. (2014). Midline 1 controls polarization and migration of murine cytotoxic T cells. Immunity, inflammation and disease, 2(4), 262-71. https://doi.org/10.1002/iid3.44
Boding, Lasse, et al. "Midline 1 controls polarization and migration of murine cytotoxic T cells." Immunity, inflammation and disease vol. 2,4 (2014): 262-71. doi: https://doi.org/10.1002/iid3.44
Boding L, Hansen AK, Nielsen MM, Meroni G, Braunstein TH, Woetmann A, Ødum N, Bonefeld CM, Geisler C. Midline 1 controls polarization and migration of murine cytotoxic T cells. Immun Inflamm Dis. 2014 Dec;2(4):262-71. doi: 10.1002/iid3.44. Epub 2015 Jan 27. PMID: 25866633; PMCID: PMC4386920.
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TRIM29 regulates the SETBP1/SET/PP2A axis via transcription factor VEZF1 to promote progression of ovarian cancer.

Cancer letters

Qiao HY, Zhang Q, Wang JM, Jiang JY, Huyan LY, Yan J, Li C, Wang HQ.
PMID: 34973391
Cancer Lett. 2021 Dec 29;529:85-99. doi: 10.1016/j.canlet.2021.12.029. Epub 2021 Dec 29.

Ovarian cancer (OC) is a common gynecological malignant tumor that seriously endangers the health of women worldwide. Tripartite motif containing 29 (TRIM29) is a TRIM family member that is frequently overexpressed in OC. However, the specific role of TRIM29...

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Qiao HY, Zhang Q, Wang JM, et al. TRIM29 regulates the SETBP1/SET/PP2A axis via transcription factor VEZF1 to promote progression of ovarian cancer. Cancer Lett. 2021;529:85-99doi: 10.1016/j.canlet.2021.12.029.
Qiao, H. Y., Zhang, Q., Wang, J. M., Jiang, J. Y., Huyan, L. Y., Yan, J., Li, C., & Wang, H. Q. (2021). TRIM29 regulates the SETBP1/SET/PP2A axis via transcription factor VEZF1 to promote progression of ovarian cancer. Cancer letters, 52985-99. https://doi.org/10.1016/j.canlet.2021.12.029
Qiao, Huai-Yu, et al. "TRIM29 regulates the SETBP1/SET/PP2A axis via transcription factor VEZF1 to promote progression of ovarian cancer." Cancer letters vol. 529 (2021): 85-99. doi: https://doi.org/10.1016/j.canlet.2021.12.029
Qiao HY, Zhang Q, Wang JM, Jiang JY, Huyan LY, Yan J, Li C, Wang HQ. TRIM29 regulates the SETBP1/SET/PP2A axis via transcription factor VEZF1 to promote progression of ovarian cancer. Cancer Lett. 2021 Dec 29;529:85-99. doi: 10.1016/j.canlet.2021.12.029. Epub 2021 Dec 29. PMID: 34973391.
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Associations between polymorphisms of SLC22A7, NGFR, ARNTL and PPP2R2B genes and Milk production traits in Chinese Holstein.

BMC genomic data

Jia R, Fu Y, Xu L, Li H, Li Y, Liu L, Ma Z, Sun D, Han B.
PMID: 34732138
BMC Genom Data. 2021 Nov 03;22(1):47. doi: 10.1186/s12863-021-01002-0.

BACKGROUND: Our preliminary work confirmed that, SLC22A7 (solute carrier family 22 member 7), NGFR (nerve growth factor receptor), ARNTL (aryl hydrocarbon receptor nuclear translocator like) and PPP2R2B (protein phosphatase 2 regulatory subunit Bβ) genes were differentially expressed in dairy...

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Jia R, Fu Y, Xu L, et al. Associations between polymorphisms of SLC22A7, NGFR, ARNTL and PPP2R2B genes and Milk production traits in Chinese Holstein. BMC Genom Data. 2021;22(1):47doi: 10.1186/s12863-021-01002-0.
Jia, R., Fu, Y., Xu, L., Li, H., Li, Y., Liu, L., Ma, Z., Sun, D., & Han, B. (2021). Associations between polymorphisms of SLC22A7, NGFR, ARNTL and PPP2R2B genes and Milk production traits in Chinese Holstein. BMC genomic data, 22(1), 47. https://doi.org/10.1186/s12863-021-01002-0
Jia, Ruike, et al. "Associations between polymorphisms of SLC22A7, NGFR, ARNTL and PPP2R2B genes and Milk production traits in Chinese Holstein." BMC genomic data vol. 22,1 (2021): 47. doi: https://doi.org/10.1186/s12863-021-01002-0
Jia R, Fu Y, Xu L, Li H, Li Y, Liu L, Ma Z, Sun D, Han B. Associations between polymorphisms of SLC22A7, NGFR, ARNTL and PPP2R2B genes and Milk production traits in Chinese Holstein. BMC Genom Data. 2021 Nov 03;22(1):47. doi: 10.1186/s12863-021-01002-0. PMID: 34732138; PMCID: PMC8567656.
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Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring.

Brain research bulletin

Wu Q, Zhou D, Wu R, Shi R, Shen X, Jin N, Gu J, Gu JH, Liu F, Chu D.
PMID: 34808323
Brain Res Bull. 2022 Jan;178:133-143. doi: 10.1016/j.brainresbull.2021.11.008. Epub 2021 Nov 19.

Folic acid (FA) supplementation in early pregnancy is recommended to protect against birth defects. But excess FA has exhibited neurodevelopmental toxicity. We previously reported that the mice treated with 2.5-fold the dietary requirement of FA one week before mating...

Cite
Wu Q, Zhou D, Wu R, et al. Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring. Brain Res Bull. 2021;178:133-143doi: 10.1016/j.brainresbull.2021.11.008.
Wu, Q., Zhou, D., Wu, R., Shi, R., Shen, X., Jin, N., Gu, J., Gu, J. H., Liu, F., & Chu, D. (2022). Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring. Brain research bulletin, 178133-143. https://doi.org/10.1016/j.brainresbull.2021.11.008
Wu, Qian, et al. "Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring." Brain research bulletin vol. 178 (2022): 133-143. doi: https://doi.org/10.1016/j.brainresbull.2021.11.008
Wu Q, Zhou D, Wu R, Shi R, Shen X, Jin N, Gu J, Gu JH, Liu F, Chu D. Excess folic acid supplementation before and during pregnancy and lactation activates β-catenin in the brain of male mouse offspring. Brain Res Bull. 2022 Jan;178:133-143. doi: 10.1016/j.brainresbull.2021.11.008. Epub 2021 Nov 19. PMID: 34808323.
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Showing 1 to 12 of 46 entries