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Effects of endogenous neurotoxin quinolinic acid on reactive oxygen species production by Fenton reaction catalyzed by iron or copper.

Journal of organometallic chemistry

Kubicova L, Hadacek F, Weckwerth W, Chobot V.
PMID: 25892824
J Organomet Chem. 2015 Apr 15;782:111-115. doi: 10.1016/j.jorganchem.2015.01.030.

The tryptophan metabolite, quinolinic (2,3-pyridinedicarboxylic) acid, is known as an endogenous neurotoxin. Quinolinic acid can form coordination complexes with iron or copper. The effects of quinolinic acid on reactive oxygen species production in the presence of iron or copper...

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Kubicova L, Hadacek F, Weckwerth W, et al. Effects of endogenous neurotoxin quinolinic acid on reactive oxygen species production by Fenton reaction catalyzed by iron or copper. J Organomet Chem. 2015;782:111-115doi: 10.1016/j.jorganchem.2015.01.030.
Kubicova, L., Hadacek, F., Weckwerth, W., & Chobot, V. (2015). Effects of endogenous neurotoxin quinolinic acid on reactive oxygen species production by Fenton reaction catalyzed by iron or copper. Journal of organometallic chemistry, 782111-115. https://doi.org/10.1016/j.jorganchem.2015.01.030
Kubicova, Lenka, et al. "Effects of endogenous neurotoxin quinolinic acid on reactive oxygen species production by Fenton reaction catalyzed by iron or copper." Journal of organometallic chemistry vol. 782 (2015): 111-115. doi: https://doi.org/10.1016/j.jorganchem.2015.01.030
Kubicova L, Hadacek F, Weckwerth W, Chobot V. Effects of endogenous neurotoxin quinolinic acid on reactive oxygen species production by Fenton reaction catalyzed by iron or copper. J Organomet Chem. 2015 Apr 15;782:111-115. doi: 10.1016/j.jorganchem.2015.01.030. PMID: 25892824; PMCID: PMC4396856.
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The Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System Diseases.

Frontiers in cellular neuroscience

Repetto IE, Monti R, Tropiano M, Tomasi S, Arbini A, Andrade-Moraes CH, Lent R, Vercelli A.
PMID: 27547177
Front Cell Neurosci. 2016 Aug 05;10:190. doi: 10.3389/fncel.2016.00190. eCollection 2016.

One major aim in quantitative and translational neuroscience is to achieve a precise and fast neuronal counting method to work on high throughput scale to obtain reliable results. Here, we tested the isotropic fractionator (IF) method for evaluating neuronal...

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Repetto IE, Monti R, Tropiano M, et al. The Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System Diseases. Front Cell Neurosci. 2016;10:190doi: 10.3389/fncel.2016.00190.
Repetto, I. E., Monti, R., Tropiano, M., Tomasi, S., Arbini, A., Andrade-Moraes, C. H., Lent, R., & Vercelli, A. (2016). The Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System Diseases. Frontiers in cellular neuroscience, 10190. https://doi.org/10.3389/fncel.2016.00190
Repetto, Ivan E, et al. "The Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System Diseases." Frontiers in cellular neuroscience vol. 10 (2016): 190. doi: https://doi.org/10.3389/fncel.2016.00190
Repetto IE, Monti R, Tropiano M, Tomasi S, Arbini A, Andrade-Moraes CH, Lent R, Vercelli A. The Isotropic Fractionator as a Tool for Quantitative Analysis in Central Nervous System Diseases. Front Cell Neurosci. 2016 Aug 05;10:190. doi: 10.3389/fncel.2016.00190. eCollection 2016. PMID: 27547177; PMCID: PMC4974250.
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Isoprenoid pathway dysfunction in chronic fatigue syndrome.

Acta neuropsychiatrica

Kurup RK, Kurup PA.
PMID: 26983655
Acta Neuropsychiatr. 2003 Oct;15(5):266-73. doi: 10.1034/j.1601-5215.2003.00045.x.

BACKGROUND AND AIMS: The isoprenoid pathway was assessed in 15 patients with chronic fatigue syndrome (CFS). The pathway was also assessed in individuals with differing hemispheric dominance to assess whether hemispheric dominance has any correlation with these disease states.METHODS:...

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Kurup RK, Kurup PA. Isoprenoid pathway dysfunction in chronic fatigue syndrome. Acta Neuropsychiatr. 2003;15(5):266-73doi: 10.1034/j.1601-5215.2003.00045.x.
Kurup, R. K., & Kurup, P. A. (2003). Isoprenoid pathway dysfunction in chronic fatigue syndrome. Acta neuropsychiatrica, 15(5), 266-73. https://doi.org/10.1034/j.1601-5215.2003.00045.x
Kurup, Ravi Kumar, and Kurup, Parameswara Achutha. "Isoprenoid pathway dysfunction in chronic fatigue syndrome." Acta neuropsychiatrica vol. 15,5 (2003): 266-73. doi: https://doi.org/10.1034/j.1601-5215.2003.00045.x
Kurup RK, Kurup PA. Isoprenoid pathway dysfunction in chronic fatigue syndrome. Acta Neuropsychiatr. 2003 Oct;15(5):266-73. doi: 10.1034/j.1601-5215.2003.00045.x. PMID: 26983655.
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Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24].

The Journal of nutritional biochemistry

Carmona-Ramírez I, Santamaría A, Tobón-Velasco JC, Orozco-Ibarra M, González-Herrera IG, Pedraza-Chaverrí J, Maldonado PD.
PMID: 28320533
J Nutr Biochem. 2017 Apr;42:203. doi: 10.1016/j.jnutbio.2017.03.001.

This article has been retracted: please see Elsevier Policy on Article Withdrawal (http://www.elsevier.com/locate/withdrawalpolicy). This article has been retracted at the request of the Editor-in-Chief as it contains inappropriately manipulated images in Figure 7B. As such this article represents a...

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Carmona-Ramírez I, Santamaría A, Tobón-Velasco JC, et al. Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]. J Nutr Biochem. 2017;42:203doi: 10.1016/j.jnutbio.2017.03.001.
Carmona-Ramírez, I., Santamaría, A., Tobón-Velasco, J. C., Orozco-Ibarra, M., González-Herrera, I. G., Pedraza-Chaverrí, J., & Maldonado, P. D. (2017). Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]. The Journal of nutritional biochemistry, 42203. https://doi.org/10.1016/j.jnutbio.2017.03.001
Carmona-Ramírez, Iván, et al. "Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]." The Journal of nutritional biochemistry vol. 42 (2017): 203. doi: https://doi.org/10.1016/j.jnutbio.2017.03.001
Carmona-Ramírez I, Santamaría A, Tobón-Velasco JC, Orozco-Ibarra M, González-Herrera IG, Pedraza-Chaverrí J, Maldonado PD. Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]. J Nutr Biochem. 2017 Apr;42:203. doi: 10.1016/j.jnutbio.2017.03.001. PMID: 28320533.
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Inhibitory effect of some tropical green leafy vegetables on key enzymes linked to Alzheimer's disease and some pro-oxidant induced lipid peroxidation in rats' brain.

Journal of food science and technology

Oboh G, Akinyemi AJ, Ademiluyi AO, Bello FO.
PMID: 24803694
J Food Sci Technol. 2014 May;51(5):884-91. doi: 10.1007/s13197-011-0572-0. Epub 2011 Nov 04.

This study sought to investigate the inhibitory effect of some commonly consumed Nigerian green leafy vegetables (raw and blanched) on acetylcholinesterase and butyrylcholinesterase (key enzyme linked to Alzheimer's disease) activities and some pro-oxidants (FeSO4, Sodium nitroprusside and Quinolinic acid)...

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Oboh G, Akinyemi AJ, Ademiluyi AO, et al. Inhibitory effect of some tropical green leafy vegetables on key enzymes linked to Alzheimer's disease and some pro-oxidant induced lipid peroxidation in rats' brain. J Food Sci Technol. 2011;51(5):884-91doi: 10.1007/s13197-011-0572-0.
Oboh, G., Akinyemi, A. J., Ademiluyi, A. O., & Bello, F. O. (2014). Inhibitory effect of some tropical green leafy vegetables on key enzymes linked to Alzheimer's disease and some pro-oxidant induced lipid peroxidation in rats' brain. Journal of food science and technology, 51(5), 884-91. https://doi.org/10.1007/s13197-011-0572-0
Oboh, Ganiyu, et al. "Inhibitory effect of some tropical green leafy vegetables on key enzymes linked to Alzheimer's disease and some pro-oxidant induced lipid peroxidation in rats' brain." Journal of food science and technology vol. 51,5 (2014): 884-91. doi: https://doi.org/10.1007/s13197-011-0572-0
Oboh G, Akinyemi AJ, Ademiluyi AO, Bello FO. Inhibitory effect of some tropical green leafy vegetables on key enzymes linked to Alzheimer's disease and some pro-oxidant induced lipid peroxidation in rats' brain. J Food Sci Technol. 2014 May;51(5):884-91. doi: 10.1007/s13197-011-0572-0. Epub 2011 Nov 04. PMID: 24803694; PMCID: PMC4008742.
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The role of kynurenines in the production of neuronal death, and the neuroprotective effect of purines.

Journal of Alzheimer's disease : JAD

Stone TW, Behan WM, Jones PA, Darlington LG, Smith RA.
PMID: 12214038
J Alzheimers Dis. 2001 Aug;3(4):355-366. doi: 10.3233/jad-2001-3401.

The kynurenine metabolic pathway from tryptophan accounts for a large proportion of the metabolism of this amino acid in the brain. Although a major route for the generation of the essential co-factor nicotinamide adenine dinucleotide (NAD), two components of...

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Stone TW, Behan WM, Jones PA, et al. The role of kynurenines in the production of neuronal death, and the neuroprotective effect of purines. J Alzheimers Dis. 2001;3(4):355-366doi: 10.3233/jad-2001-3401.
Stone, T. W., Behan, W. M., Jones, P. A., Darlington, L. G., & Smith, R. A. (2001). The role of kynurenines in the production of neuronal death, and the neuroprotective effect of purines. Journal of Alzheimer's disease : JAD, 3(4), 355-366. https://doi.org/10.3233/jad-2001-3401
Stone, T.W., et al. "The role of kynurenines in the production of neuronal death, and the neuroprotective effect of purines." Journal of Alzheimer's disease : JAD vol. 3,4 (2001): 355-366. doi: https://doi.org/10.3233/jad-2001-3401
Stone TW, Behan WM, Jones PA, Darlington LG, Smith RA. The role of kynurenines in the production of neuronal death, and the neuroprotective effect of purines. J Alzheimers Dis. 2001 Aug;3(4):355-366. doi: 10.3233/jad-2001-3401. PMID: 12214038.
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Photoinduced electron-transfer reactions with quinolinic and trimellitic acid imides: experiments and spin density calculations(1).

The Journal of organic chemistry

Griesbeck AG, Gudipati MS, Hirt J, Lex J, Oelgemoller M, Schmickler H, Schouren F.
PMID: 11031043
J Org Chem. 2000 Oct 20;65(21):7151-7. doi: 10.1021/jo001070r.

The regioselectivity of photoinduced electron-transfer (PET) reactions of unsymmetrical phthalimides is controlled by the spin density distribution of the intermediate radical anions. ROHF ab initio calculations were found to be most suitable for atomic spin density analysis. Intramolecular PET...

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Griesbeck AG, Gudipati MS, Hirt J, et al. Photoinduced electron-transfer reactions with quinolinic and trimellitic acid imides: experiments and spin density calculations(1). J Org Chem. 2000;65(21):7151-7doi: 10.1021/jo001070r.
Griesbeck, A. G., Gudipati, M. S., Hirt, J., Lex, J., Oelgemoller, M., Schmickler, H., & Schouren, F. (2000). Photoinduced electron-transfer reactions with quinolinic and trimellitic acid imides: experiments and spin density calculations(1). The Journal of organic chemistry, 65(21), 7151-7. https://doi.org/10.1021/jo001070r
Griesbeck, et al. "Photoinduced electron-transfer reactions with quinolinic and trimellitic acid imides: experiments and spin density calculations(1)." The Journal of organic chemistry vol. 65,21 (2000): 7151-7. doi: https://doi.org/10.1021/jo001070r
Griesbeck AG, Gudipati MS, Hirt J, Lex J, Oelgemoller M, Schmickler H, Schouren F. Photoinduced electron-transfer reactions with quinolinic and trimellitic acid imides: experiments and spin density calculations(1). J Org Chem. 2000 Oct 20;65(21):7151-7. doi: 10.1021/jo001070r. PMID: 11031043.
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Synthesis of the neurotoxin quinolinic acid in apoptotic tissue from Suberites domuncula: cell biological, molecular biological, and chemical analyses.

Marine biotechnology (New York, N.Y.)

Schröder HC, Sudek S, Caro S, Rosa S, Perovic S, Steffen R, Müller IM, Müller WE.
PMID: 14961229
Mar Biotechnol (NY). 2002 Dec;4(6):546-58. doi: 10.1007/s10126-002-0028-7.

Sessile marine animals, such as sponges, are prone to infection by prokaryotic as well as by eukaryotic attacking organisms. Using the sponge Suberites domuncula we document for the first time that in its apoptotic tissue a toxic compound is...

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Schröder HC, Sudek S, Caro S, et al. Synthesis of the neurotoxin quinolinic acid in apoptotic tissue from Suberites domuncula: cell biological, molecular biological, and chemical analyses. Mar Biotechnol (NY). 2002;4(6):546-58doi: 10.1007/s10126-002-0028-7.
Schröder, H. C., Sudek, S., Caro, S., Rosa, S., Perovic, S., Steffen, R., Müller, I. M., & Müller, W. E. (2002). Synthesis of the neurotoxin quinolinic acid in apoptotic tissue from Suberites domuncula: cell biological, molecular biological, and chemical analyses. Marine biotechnology (New York, N.Y.), 4(6), 546-58. https://doi.org/10.1007/s10126-002-0028-7
Schröder, Heinz-Christoph, et al. "Synthesis of the neurotoxin quinolinic acid in apoptotic tissue from Suberites domuncula: cell biological, molecular biological, and chemical analyses." Marine biotechnology (New York, N.Y.) vol. 4,6 (2002): 546-58. doi: https://doi.org/10.1007/s10126-002-0028-7
Schröder HC, Sudek S, Caro S, Rosa S, Perovic S, Steffen R, Müller IM, Müller WE. Synthesis of the neurotoxin quinolinic acid in apoptotic tissue from Suberites domuncula: cell biological, molecular biological, and chemical analyses. Mar Biotechnol (NY). 2002 Dec;4(6):546-58. doi: 10.1007/s10126-002-0028-7. PMID: 14961229.
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Quinolinic acid selectively induces apoptosis of human astrocytes: potential role in AIDS dementia complex.

Journal of neuroinflammation

Guillemin GJ, Wang L, Brew BJ.
PMID: 16042813
J Neuroinflammation. 2005 Jul 26;2:16. doi: 10.1186/1742-2094-2-16.

There is evidence that the kynurenine pathway (KP) and particularly one of its end products, quinolinic acid (QUIN) play a role in the pathogenesis of several major neuroinflammatory diseases, and more particularly AIDS dementia complex (ADC). We hypothesized that...

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Guillemin GJ, Wang L, Brew BJ. Quinolinic acid selectively induces apoptosis of human astrocytes: potential role in AIDS dementia complex. J Neuroinflammation. 2005;2:16doi: 10.1186/1742-2094-2-16.
Guillemin, G. J., Wang, L., & Brew, B. J. (2005). Quinolinic acid selectively induces apoptosis of human astrocytes: potential role in AIDS dementia complex. Journal of neuroinflammation, 216. https://doi.org/10.1186/1742-2094-2-16
Guillemin, Gilles J, et al. "Quinolinic acid selectively induces apoptosis of human astrocytes: potential role in AIDS dementia complex." Journal of neuroinflammation vol. 2 (2005): 16. doi: https://doi.org/10.1186/1742-2094-2-16
Guillemin GJ, Wang L, Brew BJ. Quinolinic acid selectively induces apoptosis of human astrocytes: potential role in AIDS dementia complex. J Neuroinflammation. 2005 Jul 26;2:16. doi: 10.1186/1742-2094-2-16. PMID: 16042813; PMCID: PMC1187916.
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Neuroinflammation, Oxidative Stress and the Pathogenesis of Parkinson's Disease.

Clinical neuroscience research

Mosley RL, Benner EJ, Kadiu I, Thomas M, Boska MD, Hasan K, Laurie C, Gendelman HE.
PMID: 18060039
Clin Neurosci Res. 2006 Dec 06;6(5):261-281. doi: 10.1016/j.cnr.2006.09.006.

Neuroinflammatory processes play a significant role in the pathogenesis of Parkinson's disease (PD). Epidemiologic, animal, human, and therapeutic studies all support the presence of an neuroinflammatory cascade in disease. This is highlighted by the neurotoxic potential of microglia ....

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Mosley RL, Benner EJ, Kadiu I, et al. Neuroinflammation, Oxidative Stress and the Pathogenesis of Parkinson's Disease. Clin Neurosci Res. 2006;6(5):261-281doi: 10.1016/j.cnr.2006.09.006.
Mosley, R. L., Benner, E. J., Kadiu, I., Thomas, M., Boska, M. D., Hasan, K., Laurie, C., & Gendelman, H. E. (2006). Neuroinflammation, Oxidative Stress and the Pathogenesis of Parkinson's Disease. Clinical neuroscience research, 6(5), 261-281. https://doi.org/10.1016/j.cnr.2006.09.006
Mosley, R Lee, et al. "Neuroinflammation, Oxidative Stress and the Pathogenesis of Parkinson's Disease." Clinical neuroscience research vol. 6,5 (2006): 261-281. doi: https://doi.org/10.1016/j.cnr.2006.09.006
Mosley RL, Benner EJ, Kadiu I, Thomas M, Boska MD, Hasan K, Laurie C, Gendelman HE. Neuroinflammation, Oxidative Stress and the Pathogenesis of Parkinson's Disease. Clin Neurosci Res. 2006 Dec 06;6(5):261-281. doi: 10.1016/j.cnr.2006.09.006. PMID: 18060039; PMCID: PMC1831679.
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Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test.

Restorative neurology and neuroscience

Borlongan CV, Cahill DW, Sanberg PR.
PMID: 21551830
Restor Neurol Neurosci. 1995 Jan 01;9(1):15-9. doi: 10.3233/RNN-1995-9103.

Animals with unilateral neurotoxic lesions in the striatum exhibit a stereotypical biased rotational behavior in response to dopamine agonists. We recently argued that the rotational test may be subject to sensitization effects of the drug. Accordingly, we proposed the...

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Borlongan CV, Cahill DW, Sanberg PR. Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test. Restor Neurol Neurosci. 1995;9(1):15-9doi: 10.3233/RNN-1995-9103.
Borlongan, C. V., Cahill, D. W., & Sanberg, P. R. (1995). Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test. Restorative neurology and neuroscience, 9(1), 15-9. https://doi.org/10.3233/RNN-1995-9103
Borlongan, C V, et al. "Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test." Restorative neurology and neuroscience vol. 9,1 (1995): 15-9. doi: https://doi.org/10.3233/RNN-1995-9103
Borlongan CV, Cahill DW, Sanberg PR. Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test. Restor Neurol Neurosci. 1995 Jan 01;9(1):15-9. doi: 10.3233/RNN-1995-9103. PMID: 21551830.
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Dissociation of p75 receptors and nerve growth factor neurotrophic effects: lack of p75 immunoreactivity in striatum following physical trauma, excitotoxicity and NGF administration.

Restorative neurology and neuroscience

Bartus RT, Dean RL, Abelleira S, Charles V, Kordower JH.
PMID: 21551853
Restor Neurol Neurosci. 1996 Jan 01;10(1):49-59. doi: 10.3233/RNN-1996-10107.

While the function and regulation of the low affinity (p75) nerve growth factor (NGF) receptor in the central nervous system (CNS) remains a mystery, one of the more intriguing observations involves its response to injury in the adult rat...

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Bartus RT, Dean RL, Abelleira S, et al. Dissociation of p75 receptors and nerve growth factor neurotrophic effects: lack of p75 immunoreactivity in striatum following physical trauma, excitotoxicity and NGF administration. Restor Neurol Neurosci. 1996;10(1):49-59doi: 10.3233/RNN-1996-10107.
Bartus, R. T., Dean, R. L., Abelleira, S., Charles, V., & Kordower, J. H. (1996). Dissociation of p75 receptors and nerve growth factor neurotrophic effects: lack of p75 immunoreactivity in striatum following physical trauma, excitotoxicity and NGF administration. Restorative neurology and neuroscience, 10(1), 49-59. https://doi.org/10.3233/RNN-1996-10107
Bartus, R T, et al. "Dissociation of p75 receptors and nerve growth factor neurotrophic effects: lack of p75 immunoreactivity in striatum following physical trauma, excitotoxicity and NGF administration." Restorative neurology and neuroscience vol. 10,1 (1996): 49-59. doi: https://doi.org/10.3233/RNN-1996-10107
Bartus RT, Dean RL, Abelleira S, Charles V, Kordower JH. Dissociation of p75 receptors and nerve growth factor neurotrophic effects: lack of p75 immunoreactivity in striatum following physical trauma, excitotoxicity and NGF administration. Restor Neurol Neurosci. 1996 Jan 01;10(1):49-59. doi: 10.3233/RNN-1996-10107. PMID: 21551853.
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Showing 1 to 12 of 183 entries