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Showing 1 to 9 of 9 entries
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Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice.

Frontiers in neuroscience

Sanna E, Talani G, Obili N, Mascia MP, Mostallino MC, Secci PP, Pisu MG, Biggio F, Utzeri C, Olla P, Biggio G, Follesa P.
PMID: 21347217
Front Neurosci. 2011 Feb 10;5:15. doi: 10.3389/fnins.2011.00015. eCollection 2011.

Post-weaning social isolation (SI) is a model of prolonged mild stress characterized by behavioral and neurochemical alterations. We used SI in C57BL/6J mice to investigate the effects of ethanol (EtOH) in the free-choice drinking paradigm on gene expression and...

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Sanna E, Talani G, Obili N, et al. Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice. Front Neurosci. 2011;5:15doi: 10.3389/fnins.2011.00015.
Sanna, E., Talani, G., Obili, N., Mascia, M. P., Mostallino, M. C., Secci, P. P., Pisu, M. G., Biggio, F., Utzeri, C., Olla, P., Biggio, G., & Follesa, P. (2011). Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice. Frontiers in neuroscience, 515. https://doi.org/10.3389/fnins.2011.00015
Sanna, Enrico, et al. "Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice." Frontiers in neuroscience vol. 5 (2011): 15. doi: https://doi.org/10.3389/fnins.2011.00015
Sanna E, Talani G, Obili N, Mascia MP, Mostallino MC, Secci PP, Pisu MG, Biggio F, Utzeri C, Olla P, Biggio G, Follesa P. Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice. Front Neurosci. 2011 Feb 10;5:15. doi: 10.3389/fnins.2011.00015. eCollection 2011. PMID: 21347217; PMCID: PMC3039156.
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Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation.

World journal of biological chemistry

Benke D, Zemoura K, Maier PJ.
PMID: 22558486
World J Biol Chem. 2012 Apr 26;3(4):61-72. doi: 10.4331/wjbc.v3.i4.61.

Inhibitory neurotransmission ensures normal brain function by counteracting and integrating excitatory activity. γ-Aminobutyric acid (GABA) is the main inhibitory neurotransmitter in the mammalian central nervous system, and mediates its effects via two classes of receptors: the GABA(A) and GABA(B)...

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Benke D, Zemoura K, Maier PJ. Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation. World J Biol Chem. 2012;3(4):61-72doi: 10.4331/wjbc.v3.i4.61.
Benke, D., Zemoura, K., & Maier, P. J. (2012). Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation. World journal of biological chemistry, 3(4), 61-72. https://doi.org/10.4331/wjbc.v3.i4.61
Benke, Dietmar, et al. "Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation." World journal of biological chemistry vol. 3,4 (2012): 61-72. doi: https://doi.org/10.4331/wjbc.v3.i4.61
Benke D, Zemoura K, Maier PJ. Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation. World J Biol Chem. 2012 Apr 26;3(4):61-72. doi: 10.4331/wjbc.v3.i4.61. PMID: 22558486; PMCID: PMC3342575.
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Are spinal GABAergic elements related to the manifestation of neuropathic pain in rat?.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Lee J, Back SK, Lim EJ, Cho GC, Kim MA, Kim HJ, Lee MH, Na HS.
PMID: 20473376
Korean J Physiol Pharmacol. 2010 Apr;14(2):59-69. doi: 10.4196/kjpp.2010.14.2.59. Epub 2010 Apr 30.

Impairment in spinal inhibition caused by quantitative alteration of GABAergic elements following peripheral nerve injury has been postulated to mediate neuropathic pain. In the present study, we tested whether neuropathic pain could be induced or reversed by pharmacologically modulating...

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Lee J, Back SK, Lim EJ, et al. Are spinal GABAergic elements related to the manifestation of neuropathic pain in rat?. Korean J Physiol Pharmacol. 2010;14(2):59-69doi: 10.4196/kjpp.2010.14.2.59.
Lee, J., Back, S. K., Lim, E. J., Cho, G. C., Kim, M. A., Kim, H. J., Lee, M. H., & Na, H. S. (2010). Are spinal GABAergic elements related to the manifestation of neuropathic pain in rat?. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 14(2), 59-69. https://doi.org/10.4196/kjpp.2010.14.2.59
Lee, Jaehee, et al. "Are spinal GABAergic elements related to the manifestation of neuropathic pain in rat?." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 14,2 (2010): 59-69. doi: https://doi.org/10.4196/kjpp.2010.14.2.59
Lee J, Back SK, Lim EJ, Cho GC, Kim MA, Kim HJ, Lee MH, Na HS. Are spinal GABAergic elements related to the manifestation of neuropathic pain in rat?. Korean J Physiol Pharmacol. 2010 Apr;14(2):59-69. doi: 10.4196/kjpp.2010.14.2.59. Epub 2010 Apr 30. PMID: 20473376; PMCID: PMC2869462.
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Ethanol Activation of Protein Kinase A Regulates GABA(A) Receptor Subunit Expression in the Cerebral Cortex and Contributes to Ethanol-Induced Hypnosis.

Frontiers in neuroscience

Kumar S, Ren Q, Beckley JH, O'Buckley TK, Gigante ED, Santerre JL, Werner DF, Morrow AL.
PMID: 22509146
Front Neurosci. 2012 Apr 09;6:44. doi: 10.3389/fnins.2012.00044. eCollection 2012.

Protein kinases are implicated in neuronal cell functions such as modulation of ion channel function, trafficking, and synaptic excitability. Both protein kinase C (PKC) and A (PKA) are involved in regulation of γ-aminobutyric acid type A (GABA(A)) receptors through...

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Kumar S, Ren Q, Beckley JH, et al. Ethanol Activation of Protein Kinase A Regulates GABA(A) Receptor Subunit Expression in the Cerebral Cortex and Contributes to Ethanol-Induced Hypnosis. Front Neurosci. 2012;6:44doi: 10.3389/fnins.2012.00044.
Kumar, S., Ren, Q., Beckley, J. H., O'Buckley, T. K., Gigante, E. D., Santerre, J. L., Werner, D. F., & Morrow, A. L. (2012). Ethanol Activation of Protein Kinase A Regulates GABA(A) Receptor Subunit Expression in the Cerebral Cortex and Contributes to Ethanol-Induced Hypnosis. Frontiers in neuroscience, 644. https://doi.org/10.3389/fnins.2012.00044
Kumar, Sandeep, et al. "Ethanol Activation of Protein Kinase A Regulates GABA(A) Receptor Subunit Expression in the Cerebral Cortex and Contributes to Ethanol-Induced Hypnosis." Frontiers in neuroscience vol. 6 (2012): 44. doi: https://doi.org/10.3389/fnins.2012.00044
Kumar S, Ren Q, Beckley JH, O'Buckley TK, Gigante ED, Santerre JL, Werner DF, Morrow AL. Ethanol Activation of Protein Kinase A Regulates GABA(A) Receptor Subunit Expression in the Cerebral Cortex and Contributes to Ethanol-Induced Hypnosis. Front Neurosci. 2012 Apr 09;6:44. doi: 10.3389/fnins.2012.00044. eCollection 2012. PMID: 22509146; PMCID: PMC3321501.
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The reciprocal regulation of stress hormones and GABA(A) receptors.

Frontiers in cellular neuroscience

Mody I, Maguire J.
PMID: 22319473
Front Cell Neurosci. 2012 Jan 30;6:4. doi: 10.3389/fncel.2012.00004. eCollection 2011 Jan.

Stress-derived steroid hormones regulate the expression and function of GABA(A) receptors (GABA(A)Rs). Changes in GABA(A)R subunit expression have been demonstrated under conditions of altered steroid hormone levels, such as stress, as well as following exogenous steroid hormone administration. In...

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Mody I, Maguire J. The reciprocal regulation of stress hormones and GABA(A) receptors. Front Cell Neurosci. 2012;6:4doi: 10.3389/fncel.2012.00004.
Mody, I., & Maguire, J. (2011). The reciprocal regulation of stress hormones and GABA(A) receptors. Frontiers in cellular neuroscience, 64. https://doi.org/10.3389/fncel.2012.00004
Mody, Istvan, and Maguire, Jamie. "The reciprocal regulation of stress hormones and GABA(A) receptors." Frontiers in cellular neuroscience vol. 6 (2011): 4. doi: https://doi.org/10.3389/fncel.2012.00004
Mody I, Maguire J. The reciprocal regulation of stress hormones and GABA(A) receptors. Front Cell Neurosci. 2012 Jan 30;6:4. doi: 10.3389/fncel.2012.00004. eCollection 2011 Jan. PMID: 22319473; PMCID: PMC3268361.
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GABA Not Only a Neurotransmitter: Osmotic Regulation by GABA(A)R Signaling.

Frontiers in cellular neuroscience

Cesetti T, Ciccolini F, Li Y.
PMID: 22319472
Front Cell Neurosci. 2012 Jan 30;6:3. doi: 10.3389/fncel.2012.00003. eCollection 2011 Jan.

Mature macroglia and almost all neural progenitor types express γ-aminobutyric (GABA) A receptors (GABA(A)Rs), whose activation by ambient or synaptic GABA, leads to influx or efflux of chloride (Cl(-)) depending on its electro-chemical gradient (E(Cl)). Since the flux of...

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Cesetti T, Ciccolini F, Li Y. GABA Not Only a Neurotransmitter: Osmotic Regulation by GABA(A)R Signaling. Front Cell Neurosci. 2012;6:3doi: 10.3389/fncel.2012.00003.
Cesetti, T., Ciccolini, F., & Li, Y. (2011). GABA Not Only a Neurotransmitter: Osmotic Regulation by GABA(A)R Signaling. Frontiers in cellular neuroscience, 63. https://doi.org/10.3389/fncel.2012.00003
Cesetti, Tiziana, et al. "GABA Not Only a Neurotransmitter: Osmotic Regulation by GABA(A)R Signaling." Frontiers in cellular neuroscience vol. 6 (2011): 3. doi: https://doi.org/10.3389/fncel.2012.00003
Cesetti T, Ciccolini F, Li Y. GABA Not Only a Neurotransmitter: Osmotic Regulation by GABA(A)R Signaling. Front Cell Neurosci. 2012 Jan 30;6:3. doi: 10.3389/fncel.2012.00003. eCollection 2011 Jan. PMID: 22319472; PMCID: PMC3268181.
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Neuronal integration and the depolarizing effects of axonal GABA(A) receptors.

Journal of neurophysiology

Younts TJ, Makani S.
PMID: 21813744
J Neurophysiol. 2011 Nov;106(5):2105-7. doi: 10.1152/jn.00328.2011. Epub 2011 Aug 03.

Despite their presence throughout the central nervous system, the impact of axonally expressed gamma-amino-butyric acid type-A receptors (GABA(A)Rs) on neuronal signaling is just beginning to be understood. A recently published article (Pugh JR and Jahr CE, J Neurosci 31:...

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Younts TJ, Makani S. Neuronal integration and the depolarizing effects of axonal GABA(A) receptors. J Neurophysiol. 2011;106(5):2105-7doi: 10.1152/jn.00328.2011.
Younts, T. J., & Makani, S. (2011). Neuronal integration and the depolarizing effects of axonal GABA(A) receptors. Journal of neurophysiology, 106(5), 2105-7. https://doi.org/10.1152/jn.00328.2011
Younts, Thomas J, and Makani, Sachin. "Neuronal integration and the depolarizing effects of axonal GABA(A) receptors." Journal of neurophysiology vol. 106,5 (2011): 2105-7. doi: https://doi.org/10.1152/jn.00328.2011
Younts TJ, Makani S. Neuronal integration and the depolarizing effects of axonal GABA(A) receptors. J Neurophysiol. 2011 Nov;106(5):2105-7. doi: 10.1152/jn.00328.2011. Epub 2011 Aug 03. PMID: 21813744.
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GABA(A) Receptors: Post-Synaptic Co-Localization and Cross-Talk with Other Receptors.

Frontiers in cellular neuroscience

Shrivastava AN, Triller A, Sieghart W.
PMID: 21734865
Front Cell Neurosci. 2011 Jun 22;5:7. doi: 10.3389/fncel.2011.00007. eCollection 2011.

γ-Aminobutyric acid type A receptors (GABA(A)Rs) are the major inhibitory neurotransmitter receptors in the central nervous system, and importantly contribute to the functional regulation of the nervous system. Several studies in the last few decades have convincingly shown that...

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Shrivastava AN, Triller A, Sieghart W. GABA(A) Receptors: Post-Synaptic Co-Localization and Cross-Talk with Other Receptors. Front Cell Neurosci. 2011;5:7doi: 10.3389/fncel.2011.00007.
Shrivastava, A. N., Triller, A., & Sieghart, W. (2011). GABA(A) Receptors: Post-Synaptic Co-Localization and Cross-Talk with Other Receptors. Frontiers in cellular neuroscience, 57. https://doi.org/10.3389/fncel.2011.00007
Shrivastava, Amulya Nidhi, et al. "GABA(A) Receptors: Post-Synaptic Co-Localization and Cross-Talk with Other Receptors." Frontiers in cellular neuroscience vol. 5 (2011): 7. doi: https://doi.org/10.3389/fncel.2011.00007
Shrivastava AN, Triller A, Sieghart W. GABA(A) Receptors: Post-Synaptic Co-Localization and Cross-Talk with Other Receptors. Front Cell Neurosci. 2011 Jun 22;5:7. doi: 10.3389/fncel.2011.00007. eCollection 2011. PMID: 21734865; PMCID: PMC3123775.
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Abuse Potential of Soma: the GABA(A) Receptor as a Target.

Molecular and cellular pharmacology

Gonzalez LA, Gatch MB, Forster MJ, Dillon GH.
PMID: 20419052
Mol Cell Pharmacol. 2009 Jan 01;1(4):180-186.

Soma(®) (carisoprodol) is an increasingly abused, centrally-acting muscle relaxant. Despite the prevalence of carisoprodol abuse, its mechanism of action remains unclear. Its sedative effects, which contribute to its therapeutic and recreational use, are generally attributed to the actions of...

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Gonzalez LA, Gatch MB, Forster MJ, et al. Abuse Potential of Soma: the GABA(A) Receptor as a Target. Mol Cell Pharmacol. 2009;1(4):180-186
Gonzalez, L. A., Gatch, M. B., Forster, M. J., & Dillon, G. H. (2009). Abuse Potential of Soma: the GABA(A) Receptor as a Target. Molecular and cellular pharmacology, 1(4), 180-186.
Gonzalez, Lorie A, et al. "Abuse Potential of Soma: the GABA(A) Receptor as a Target." Molecular and cellular pharmacology vol. 1,4 (2009): 180-186.
Gonzalez LA, Gatch MB, Forster MJ, Dillon GH. Abuse Potential of Soma: the GABA(A) Receptor as a Target. Mol Cell Pharmacol. 2009 Jan 01;1(4):180-186. PMID: 20419052; PMCID: PMC2858432.
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Showing 1 to 9 of 9 entries