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The yliA, -B, -C, and -D Genes of Escherichia coli K-12 Encode a Novel Glutathione Importer with an ATP-Binding Cassette

k-12

Koyanagi T.
GSID: k1EUIxd14uEJ
H Suzuki, T Koyanagi, S Izuka, A Onishi… - Journal of …, 2005 - Am Soc Microbiol

Glutathione protects cells and organisms from oxygen species and peroxides and is indispensable for aerobically living organisms. Moreover, it acts against xenobiotics and drugs by the formation and excretion of glutathione S conjugates. In this study, we show that...

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Suzuki, H., Koyanagi, T., Izuka, S., Onishi, A., & Kumagai, H. (2005). The yliA,-B,-C, and-D genes of Escherichia coli K-12 encode a novel glutathione importer with an ATP-binding cassette. Journal of bacteriology, 187(17), 5861-5867.
Suzuki, Hideyuki, et al. "The yliA,-B,-C, and-D genes of Escherichia coli K-12 encode a novel glutathione importer with an ATP-binding cassette." Journal of bacteriology 187.17 (2005): 5861-5867.
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Molecular biology of the blood-brain and the blood-cerebrospinal fluid barriers: similarities and differences.

Fluids and barriers of the CNS

Redzic Z.
PMID: 21349151
Fluids Barriers CNS. 2011 Jan 18;8(1):3. doi: 10.1186/2045-8118-8-3.

Efficient processing of information by the central nervous system (CNS) represents an important evolutionary advantage. Thus, homeostatic mechanisms have developed that provide appropriate circumstances for neuronal signaling, including a highly controlled and stable microenvironment. To provide such a milieu...

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Redzic Z. Molecular biology of the blood-brain and the blood-cerebrospinal fluid barriers: similarities and differences. Fluids Barriers CNS. 2011;8(1):3doi: 10.1186/2045-8118-8-3.
Redzic, Z. (2011). Molecular biology of the blood-brain and the blood-cerebrospinal fluid barriers: similarities and differences. Fluids and barriers of the CNS, 8(1), 3. https://doi.org/10.1186/2045-8118-8-3
Redzic, Zoran. "Molecular biology of the blood-brain and the blood-cerebrospinal fluid barriers: similarities and differences." Fluids and barriers of the CNS vol. 8,1 (2011): 3. doi: https://doi.org/10.1186/2045-8118-8-3
Redzic Z. Molecular biology of the blood-brain and the blood-cerebrospinal fluid barriers: similarities and differences. Fluids Barriers CNS. 2011 Jan 18;8(1):3. doi: 10.1186/2045-8118-8-3. PMID: 21349151; PMCID: PMC3045361.
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3,4-Dichloroaniline revisited: A study on the fate of the priority pollutant in a sediment-water system derived from a rice growing region in Italy.

The Science of the total environment

Yuan Y, Zhang P, Schäffer A, Schmidt B.
PMID: 27668853
Sci Total Environ. 2017 Jan 01;574:1012-1020. doi: 10.1016/j.scitotenv.2016.09.099. Epub 2016 Oct 14.

As ultimate sink for xenobiotics released into the environment, sediments play an important role concerning the evaluation of the fate of foreign compounds. 3,4-Dichloroaniline (3,4-DCA) is a degradation product of herbicide propanil and some urea herbicides. Propanil was extensively...

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Yuan Y, Zhang P, Schäffer A, et al. 3,4-Dichloroaniline revisited: A study on the fate of the priority pollutant in a sediment-water system derived from a rice growing region in Italy. Sci Total Environ. 2016;574:1012-1020doi: 10.1016/j.scitotenv.2016.09.099.
Yuan, Y., Zhang, P., Schäffer, A., & Schmidt, B. (2017). 3,4-Dichloroaniline revisited: A study on the fate of the priority pollutant in a sediment-water system derived from a rice growing region in Italy. The Science of the total environment, 5741012-1020. https://doi.org/10.1016/j.scitotenv.2016.09.099
Yuan, Ye, et al. "3,4-Dichloroaniline revisited: A study on the fate of the priority pollutant in a sediment-water system derived from a rice growing region in Italy." The Science of the total environment vol. 574 (2017): 1012-1020. doi: https://doi.org/10.1016/j.scitotenv.2016.09.099
Yuan Y, Zhang P, Schäffer A, Schmidt B. 3,4-Dichloroaniline revisited: A study on the fate of the priority pollutant in a sediment-water system derived from a rice growing region in Italy. Sci Total Environ. 2017 Jan 01;574:1012-1020. doi: 10.1016/j.scitotenv.2016.09.099. Epub 2016 Oct 14. PMID: 27668853.
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Analysis and Toxicity of Plain (PMP) and Blended (PMT) Indian Pan Masala (PM).

The Eurasian journal of medicine

Nigam SK, Venkatakrishna-Bhatt H.
PMID: 25610244
Eurasian J Med. 2013 Feb;45(1):21-33. doi: 10.5152/eajm.2013.04.

OBJECTIVE: Betel leaf combined with areca nut is known as betel quid pan masala (PM), and tobacco with areca nut, catechu and lime is pan masala (PMT) blended with gulkhand. These narcotics are popular among young and old individuals....

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Nigam SK, Venkatakrishna-Bhatt H. Analysis and Toxicity of Plain (PMP) and Blended (PMT) Indian Pan Masala (PM). Eurasian J Med. 2013;45(1):21-33doi: 10.5152/eajm.2013.04.
Nigam, S. K., & Venkatakrishna-Bhatt, H. (2013). Analysis and Toxicity of Plain (PMP) and Blended (PMT) Indian Pan Masala (PM). The Eurasian journal of medicine, 45(1), 21-33. https://doi.org/10.5152/eajm.2013.04
Nigam, Suresh Kumar, and Venkatakrishna-Bhatt, H. "Analysis and Toxicity of Plain (PMP) and Blended (PMT) Indian Pan Masala (PM)." The Eurasian journal of medicine vol. 45,1 (2013): 21-33. doi: https://doi.org/10.5152/eajm.2013.04
Nigam SK, Venkatakrishna-Bhatt H. Analysis and Toxicity of Plain (PMP) and Blended (PMT) Indian Pan Masala (PM). Eurasian J Med. 2013 Feb;45(1):21-33. doi: 10.5152/eajm.2013.04. PMID: 25610244; PMCID: PMC4261503.
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Role of the Aryl Hydrocarbon Receptor in Colon Neoplasia.

Cancers

Xie G, Raufman JP.
PMID: 26264025
Cancers (Basel). 2015 Jul 31;7(3):1436-46. doi: 10.3390/cancers7030847.

For both men and women, colorectal cancer (CRC) is the second leading cause of cancer death in the United States, primarily as a consequence of limited therapies for metastatic disease. The aryl hydrocarbon receptor (AhR) is a ligand-dependent transcription...

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Xie G, Raufman JP. Role of the Aryl Hydrocarbon Receptor in Colon Neoplasia. Cancers (Basel). 2015;7(3):1436-46doi: 10.3390/cancers7030847.
Xie, G., & Raufman, J. P. (2015). Role of the Aryl Hydrocarbon Receptor in Colon Neoplasia. Cancers, 7(3), 1436-46. https://doi.org/10.3390/cancers7030847
Xie, Guofeng, and Raufman, Jean-Pierre. "Role of the Aryl Hydrocarbon Receptor in Colon Neoplasia." Cancers vol. 7,3 (2015): 1436-46. doi: https://doi.org/10.3390/cancers7030847
Xie G, Raufman JP. Role of the Aryl Hydrocarbon Receptor in Colon Neoplasia. Cancers (Basel). 2015 Jul 31;7(3):1436-46. doi: 10.3390/cancers7030847. PMID: 26264025; PMCID: PMC4586780.
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Identification of deleterious nsSNPs in α, μ, π and θ class of GST family and their influence on protein structure.

Genomics data

Yadav P, Chatterjee A, Bhattacharjee A.
PMID: 26484073
Genom Data. 2014 May 09;2:66-72. doi: 10.1016/j.gdata.2014.03.004. eCollection 2014 Dec.

GST family genes have a critical role in xenobiotic metabolism and drug resistance. Among the GST family the GST-μ, GST-π, GST-α and GST-θ are the most abundant classes and have a major role in the carcinogen detoxification process. Nevertheless...

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Yadav P, Chatterjee A, Bhattacharjee A. Identification of deleterious nsSNPs in α, μ, π and θ class of GST family and their influence on protein structure. Genom Data. 2014;2:66-72doi: 10.1016/j.gdata.2014.03.004.
Yadav, P., Chatterjee, A., & Bhattacharjee, A. (2014). Identification of deleterious nsSNPs in α, μ, π and θ class of GST family and their influence on protein structure. Genomics data, 266-72. https://doi.org/10.1016/j.gdata.2014.03.004
Yadav, P, et al. "Identification of deleterious nsSNPs in α, μ, π and θ class of GST family and their influence on protein structure." Genomics data vol. 2 (2014): 66-72. doi: https://doi.org/10.1016/j.gdata.2014.03.004
Yadav P, Chatterjee A, Bhattacharjee A. Identification of deleterious nsSNPs in α, μ, π and θ class of GST family and their influence on protein structure. Genom Data. 2014 May 09;2:66-72. doi: 10.1016/j.gdata.2014.03.004. eCollection 2014 Dec. PMID: 26484073; PMCID: PMC4535831.
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Non-animal Tests for Evaluating the Toxicity of Solid Xenobiotics.

Alternatives to laboratory animals : ATLA

Fubini B, Aust AE, Bolton RE, Borm PJ, Bruch J, Ciapetti G, Donaldson K, Elias Z, Gold J, Jaurand MC, Kane AB, Lison D, Muhle H.
PMID: 26042488
Altern Lab Anim. 1998 Sep-Oct;26(5):579-617.

No abstract available.

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Fubini B, Aust AE, Bolton RE, et al. Non-animal Tests for Evaluating the Toxicity of Solid Xenobiotics. Altern Lab Anim. 1998;26(5):579-617
Fubini, B., Aust, A. E., Bolton, R. E., Borm, P. J., Bruch, J., Ciapetti, G., Donaldson, K., Elias, Z., Gold, J., Jaurand, M. C., Kane, A. B., Lison, D., & Muhle, H. (1998). Non-animal Tests for Evaluating the Toxicity of Solid Xenobiotics. Alternatives to laboratory animals : ATLA, 26(5), 579-617.
Fubini, B, et al. "Non-animal Tests for Evaluating the Toxicity of Solid Xenobiotics." Alternatives to laboratory animals : ATLA vol. 26,5 (1998): 579-617.
Fubini B, Aust AE, Bolton RE, Borm PJ, Bruch J, Ciapetti G, Donaldson K, Elias Z, Gold J, Jaurand MC, Kane AB, Lison D, Muhle H. Non-animal Tests for Evaluating the Toxicity of Solid Xenobiotics. Altern Lab Anim. 1998 Sep-Oct;26(5):579-617. PMID: 26042488.
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Association of glutathione S-transferase (GSTM1 and GSTT1) genes with chronic myeloid leukemia.

SpringerPlus

Kassogue Y, Dehbi H, Quachouh M, Quessar A, Benchekroun S, Nadifi S.
PMID: 25969820
Springerplus. 2015 May 01;4:210. doi: 10.1186/s40064-015-0966-y. eCollection 2015.

Chronic myeloid leukemia (CML), as most of cancers results from a complex interaction between genetic or non genetic factors. Exposures to xenobiotics endogenous or exogenous associated with a reduced individual ability in detoxifying activity, constitutes a risk of developing...

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Kassogue Y, Dehbi H, Quachouh M, et al. Association of glutathione S-transferase (GSTM1 and GSTT1) genes with chronic myeloid leukemia. Springerplus. 2015;4:210doi: 10.1186/s40064-015-0966-y.
Kassogue, Y., Dehbi, H., Quachouh, M., Quessar, A., Benchekroun, S., & Nadifi, S. (2015). Association of glutathione S-transferase (GSTM1 and GSTT1) genes with chronic myeloid leukemia. SpringerPlus, 4210. https://doi.org/10.1186/s40064-015-0966-y
Kassogue, Yaya, et al. "Association of glutathione S-transferase (GSTM1 and GSTT1) genes with chronic myeloid leukemia." SpringerPlus vol. 4 (2015): 210. doi: https://doi.org/10.1186/s40064-015-0966-y
Kassogue Y, Dehbi H, Quachouh M, Quessar A, Benchekroun S, Nadifi S. Association of glutathione S-transferase (GSTM1 and GSTT1) genes with chronic myeloid leukemia. Springerplus. 2015 May 01;4:210. doi: 10.1186/s40064-015-0966-y. eCollection 2015. PMID: 25969820; PMCID: PMC4417468.
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The effects of oxidative stress in in vitro cultured astroglial cells.

Alternatives to laboratory animals : ATLA

Frederiksen CM, Clausen J.
PMID: 25470673
Altern Lab Anim. 1999 May-Jun;27(3):351-7. doi: 10.1177/026119299902700307.

It has been suggested that glial cells in the central nervous system might function as a buffer and protect neurons and synapses. Associated with such a function, glial cells might be affected in degenerative diseases, for example, Alzheimer's disease...

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Frederiksen CM, Clausen J. The effects of oxidative stress in in vitro cultured astroglial cells. Altern Lab Anim. 1999;27(3):351-7doi: 10.1177/026119299902700307.
Frederiksen, C. M., & Clausen, J. (1999). The effects of oxidative stress in in vitro cultured astroglial cells. Alternatives to laboratory animals : ATLA, 27(3), 351-7. https://doi.org/10.1177/026119299902700307
Frederiksen, C M, and Clausen, J. "The effects of oxidative stress in in vitro cultured astroglial cells." Alternatives to laboratory animals : ATLA vol. 27,3 (1999): 351-7. doi: https://doi.org/10.1177/026119299902700307
Frederiksen CM, Clausen J. The effects of oxidative stress in in vitro cultured astroglial cells. Altern Lab Anim. 1999 May-Jun;27(3):351-7. doi: 10.1177/026119299902700307. PMID: 25470673.
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Glutathione S-transferase Omega 2 Genetic Polymorphism and Risk of Hepatic Failure that Lead to Liver Transplantation in Iranian Population.

International journal of organ transplantation medicine

Khosravi M, Saadat I, Karimi MH, Geramizadeh B, Saadat M.
PMID: 25013648
Int J Organ Transplant Med. 2013;4(1):16-20.

BACKGROUND: Liver transplantation is the treatment of choice for both acute and chronic hepatic failure. GSTs family is one of the most important genes in phase II detoxification interfering with the xenobiotics and free radical metabolism. GSTO2 (N142D) is...

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Khosravi M, Saadat I, Karimi MH, et al. Glutathione S-transferase Omega 2 Genetic Polymorphism and Risk of Hepatic Failure that Lead to Liver Transplantation in Iranian Population. Int J Organ Transplant Med. 2013;4(1):16-20
Khosravi, M., Saadat, I., Karimi, M. H., Geramizadeh, B., & Saadat, M. (2013). Glutathione S-transferase Omega 2 Genetic Polymorphism and Risk of Hepatic Failure that Lead to Liver Transplantation in Iranian Population. International journal of organ transplantation medicine, 4(1), 16-20.
Khosravi, M, et al. "Glutathione S-transferase Omega 2 Genetic Polymorphism and Risk of Hepatic Failure that Lead to Liver Transplantation in Iranian Population." International journal of organ transplantation medicine vol. 4,1 (2013): 16-20.
Khosravi M, Saadat I, Karimi MH, Geramizadeh B, Saadat M. Glutathione S-transferase Omega 2 Genetic Polymorphism and Risk of Hepatic Failure that Lead to Liver Transplantation in Iranian Population. Int J Organ Transplant Med. 2013;4(1):16-20. PMID: 25013648; PMCID: PMC4089303.
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Kynurenine aminotransferase 3/glutamine transaminase L/cysteine conjugate beta-lyase 2 is a major glutamine transaminase in the mouse kidney.

Biochemistry and biophysics reports

Yang C, Zhang L, Han Q, Liao C, Lan J, Ding H, Zhou H, Diao X, Li J.
PMID: 28955961
Biochem Biophys Rep. 2016 Sep 21;8:234-241. doi: 10.1016/j.bbrep.2016.09.008. eCollection 2016 Dec.

BACKGROUND: Kynurenine aminotransferase 3 (KAT3) catalyzes the transamination of Kynurenine to kynurenic acid, and is identical to cysteine conjugate beta-lyase 2 (CCBL2) and glutamine transaminase L (GTL). GTL was previously purified from the rat liver and considered as a...

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Yang C, Zhang L, Han Q, et al. Kynurenine aminotransferase 3/glutamine transaminase L/cysteine conjugate beta-lyase 2 is a major glutamine transaminase in the mouse kidney. Biochem Biophys Rep. 2016;8:234-241doi: 10.1016/j.bbrep.2016.09.008.
Yang, C., Zhang, L., Han, Q., Liao, C., Lan, J., Ding, H., Zhou, H., Diao, X., & Li, J. (2016). Kynurenine aminotransferase 3/glutamine transaminase L/cysteine conjugate beta-lyase 2 is a major glutamine transaminase in the mouse kidney. Biochemistry and biophysics reports, 8234-241. https://doi.org/10.1016/j.bbrep.2016.09.008
Yang, Cihan, et al. "Kynurenine aminotransferase 3/glutamine transaminase L/cysteine conjugate beta-lyase 2 is a major glutamine transaminase in the mouse kidney." Biochemistry and biophysics reports vol. 8 (2016): 234-241. doi: https://doi.org/10.1016/j.bbrep.2016.09.008
Yang C, Zhang L, Han Q, Liao C, Lan J, Ding H, Zhou H, Diao X, Li J. Kynurenine aminotransferase 3/glutamine transaminase L/cysteine conjugate beta-lyase 2 is a major glutamine transaminase in the mouse kidney. Biochem Biophys Rep. 2016 Sep 21;8:234-241. doi: 10.1016/j.bbrep.2016.09.008. eCollection 2016 Dec. PMID: 28955961; PMCID: PMC5613967.
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The Ginkgo biloba Extract Reverses the Renal Effects of Titanium Dioxide Nanoparticles in Adult Male Rats.

Biochemistry research international

Escárcega-González CE, Reynoso-Andeola IG, Jaramillo-Juárez F, Martínez-Ruvalcaba H, Posadas Del Rio FA.
PMID: 27042354
Biochem Res Int. 2016;2016:5781579. doi: 10.1155/2016/5781579. Epub 2016 Mar 03.

The Ginkgo biloba extract (GbE) is a commercial product used as a nutraceutic herbal remedy in Europe and US. It contains 27% of the polyphenols isorhamnetin, kaempferol, and quercetin, as antioxidants. We used male adult Wistar rats (200-300 g),...

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Escárcega-González CE, Reynoso-Andeola IG, Jaramillo-Juárez F, et al. The Ginkgo biloba Extract Reverses the Renal Effects of Titanium Dioxide Nanoparticles in Adult Male Rats. Biochem Res Int. 2016;2016:5781579doi: 10.1155/2016/5781579.
Escárcega-González, C. E., Reynoso-Andeola, I. G., Jaramillo-Juárez, F., Martínez-Ruvalcaba, H., & Posadas Del Rio, F. A. (2016). The Ginkgo biloba Extract Reverses the Renal Effects of Titanium Dioxide Nanoparticles in Adult Male Rats. Biochemistry research international, 20165781579. https://doi.org/10.1155/2016/5781579
Escárcega-González, Carlos Enrique, et al. "The Ginkgo biloba Extract Reverses the Renal Effects of Titanium Dioxide Nanoparticles in Adult Male Rats." Biochemistry research international vol. 2016 (2016): 5781579. doi: https://doi.org/10.1155/2016/5781579
Escárcega-González CE, Reynoso-Andeola IG, Jaramillo-Juárez F, Martínez-Ruvalcaba H, Posadas Del Rio FA. The Ginkgo biloba Extract Reverses the Renal Effects of Titanium Dioxide Nanoparticles in Adult Male Rats. Biochem Res Int. 2016;2016:5781579. doi: 10.1155/2016/5781579. Epub 2016 Mar 03. PMID: 27042354; PMCID: PMC4794584.
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Showing 1 to 12 of 1557 entries