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The effects of buthionine sulfoximine on the proliferation and apoptosis of biliary tract cancer cells induced by cisplatin and gemcitabine.

Oncology letters

Li Q, Yin X, Wang W, Zhan M, Zhao B, Hou Z, Wang J.
PMID: 26870236
Oncol Lett. 2016 Jan;11(1):474-480. doi: 10.3892/ol.2015.3879. Epub 2015 Nov 06.

Patients with biliary tract cancer (BTC) have a poor prognosis. Advanced BTC patients have been treated with cisplatin in combination with gemcitabine, however, the treatment has had little impact on survival rates, and more effective treatments are urgently required...

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Li Q, Yin X, Wang W, et al. The effects of buthionine sulfoximine on the proliferation and apoptosis of biliary tract cancer cells induced by cisplatin and gemcitabine. Oncol Lett. 2015;11(1):474-480doi: 10.3892/ol.2015.3879.
Li, Q., Yin, X., Wang, W., Zhan, M., Zhao, B., Hou, Z., & Wang, J. (2016). The effects of buthionine sulfoximine on the proliferation and apoptosis of biliary tract cancer cells induced by cisplatin and gemcitabine. Oncology letters, 11(1), 474-480. https://doi.org/10.3892/ol.2015.3879
Li, Qiwei, et al. "The effects of buthionine sulfoximine on the proliferation and apoptosis of biliary tract cancer cells induced by cisplatin and gemcitabine." Oncology letters vol. 11,1 (2016): 474-480. doi: https://doi.org/10.3892/ol.2015.3879
Li Q, Yin X, Wang W, Zhan M, Zhao B, Hou Z, Wang J. The effects of buthionine sulfoximine on the proliferation and apoptosis of biliary tract cancer cells induced by cisplatin and gemcitabine. Oncol Lett. 2016 Jan;11(1):474-480. doi: 10.3892/ol.2015.3879. Epub 2015 Nov 06. PMID: 26870236; PMCID: PMC4727028.
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Effects of Crocin Supplementation during In Vitro Maturation of Mouse Oocytes on Glutathione Synthesis and Cytoplasmic Maturation.

International journal of fertility & sterility

Mokhber Maleki E, Eimani H, Bigdeli MR, Golkar Narenji A, Abedi R.
PMID: 27123201
Int J Fertil Steril. 2016 Apr-Jun;10(1):53-61. doi: 10.22074/ijfs.2016.4769. Epub 2016 Apr 05.

BACKGROUND: Crocin is an active ingredient of saffron (Crocus sativus L.) and its antioxidant properties have been previously investigated. This carotenoid scavenges free radicals and stimulates glutathione (GSH) synthesis; consequently, it may protect cells against oxidative stress. The aim...

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Mokhber Maleki E, Eimani H, Bigdeli MR, et al. Effects of Crocin Supplementation during In Vitro Maturation of Mouse Oocytes on Glutathione Synthesis and Cytoplasmic Maturation. Int J Fertil Steril. 2016;10(1):53-61doi: 10.22074/ijfs.2016.4769.
Mokhber Maleki, E., Eimani, H., Bigdeli, M. R., Golkar Narenji, A., & Abedi, R. (2016). Effects of Crocin Supplementation during In Vitro Maturation of Mouse Oocytes on Glutathione Synthesis and Cytoplasmic Maturation. International journal of fertility & sterility, 10(1), 53-61. https://doi.org/10.22074/ijfs.2016.4769
Mokhber Maleki, Elham, et al. "Effects of Crocin Supplementation during In Vitro Maturation of Mouse Oocytes on Glutathione Synthesis and Cytoplasmic Maturation." International journal of fertility & sterility vol. 10,1 (2016): 53-61. doi: https://doi.org/10.22074/ijfs.2016.4769
Mokhber Maleki E, Eimani H, Bigdeli MR, Golkar Narenji A, Abedi R. Effects of Crocin Supplementation during In Vitro Maturation of Mouse Oocytes on Glutathione Synthesis and Cytoplasmic Maturation. Int J Fertil Steril. 2016 Apr-Jun;10(1):53-61. doi: 10.22074/ijfs.2016.4769. Epub 2016 Apr 05. PMID: 27123201; PMCID: PMC4845530.
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Cellular glutathione content in the organ of Corti and its role during ototoxicity.

Frontiers in cellular neuroscience

Majumder P, Duchen MR, Gale JE.
PMID: 25972783
Front Cell Neurosci. 2015 Apr 28;9:143. doi: 10.3389/fncel.2015.00143. eCollection 2015.

Glutathione (GSH) is the major scavenger of reactive oxygen species (ROS) inside cells. We used live confocal imaging in order to clarify the role of GSH in the biology of the organ of Corti, the sensory epithelium of the...

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Majumder P, Duchen MR, Gale JE. Cellular glutathione content in the organ of Corti and its role during ototoxicity. Front Cell Neurosci. 2015;9:143doi: 10.3389/fncel.2015.00143.
Majumder, P., Duchen, M. R., & Gale, J. E. (2015). Cellular glutathione content in the organ of Corti and its role during ototoxicity. Frontiers in cellular neuroscience, 9143. https://doi.org/10.3389/fncel.2015.00143
Majumder, Paromita, et al. "Cellular glutathione content in the organ of Corti and its role during ototoxicity." Frontiers in cellular neuroscience vol. 9 (2015): 143. doi: https://doi.org/10.3389/fncel.2015.00143
Majumder P, Duchen MR, Gale JE. Cellular glutathione content in the organ of Corti and its role during ototoxicity. Front Cell Neurosci. 2015 Apr 28;9:143. doi: 10.3389/fncel.2015.00143. eCollection 2015. PMID: 25972783; PMCID: PMC4412067.
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Demand-Driven Control of Root ATP Sulfurylase Activity and SO42- Uptake in Intact Canola (The Role of Phloem-Translocated Glutathione).

Plant physiology

Lappartient AG, Touraine B.
PMID: 12226281
Plant Physiol. 1996 May;111(1):147-157. doi: 10.1104/pp.111.1.147.

The activity of ATP sulfurylase extracted from roots of intact canola (Brassica napus L. cv Drakkar) increased after withdrawal of the S source from the nutrient solution and declined after refeeding SO42- to S-starved plants. The rate of SO42-...

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Lappartient AG, Touraine B. Demand-Driven Control of Root ATP Sulfurylase Activity and SO42- Uptake in Intact Canola (The Role of Phloem-Translocated Glutathione). Plant Physiol. 1996;111(1):147-157doi: 10.1104/pp.111.1.147.
Lappartient, A. G., & Touraine, B. (1996). Demand-Driven Control of Root ATP Sulfurylase Activity and SO42- Uptake in Intact Canola (The Role of Phloem-Translocated Glutathione). Plant physiology, 111(1), 147-157. https://doi.org/10.1104/pp.111.1.147
Lappartient, A. G., and Touraine, B.. "Demand-Driven Control of Root ATP Sulfurylase Activity and SO42- Uptake in Intact Canola (The Role of Phloem-Translocated Glutathione)." Plant physiology vol. 111,1 (1996): 147-157. doi: https://doi.org/10.1104/pp.111.1.147
Lappartient AG, Touraine B. Demand-Driven Control of Root ATP Sulfurylase Activity and SO42- Uptake in Intact Canola (The Role of Phloem-Translocated Glutathione). Plant Physiol. 1996 May;111(1):147-157. doi: 10.1104/pp.111.1.147. PMID: 12226281; PMCID: PMC157821.
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Signalling steps in apoptosis by ether lipids.

Apoptosis : an international journal on programmed cell death

Smets LA, Van Rooij H, Salomons GS.
PMID: 14634326
Apoptosis. 1999 Dec;4(6):419-27. doi: 10.1023/a:1009644208512.

We have investigated the mechanisms of induction of apoptosis by the antineoplastic ether lipid ET-18-OCH3 (ALP) in sensitive S49wt mouse lymphoma cells and ALP-resistant S49ar variants, both with wild-type p53, and in related L1210 cells with mutated p53. Ether...

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Smets LA, Van Rooij H, Salomons GS. Signalling steps in apoptosis by ether lipids. Apoptosis. 1999;4(6):419-27doi: 10.1023/a:1009644208512.
Smets, L. A., Van Rooij, H., & Salomons, G. S. (1999). Signalling steps in apoptosis by ether lipids. Apoptosis : an international journal on programmed cell death, 4(6), 419-27. https://doi.org/10.1023/a:1009644208512
Smets, L A, et al. "Signalling steps in apoptosis by ether lipids." Apoptosis : an international journal on programmed cell death vol. 4,6 (1999): 419-27. doi: https://doi.org/10.1023/a:1009644208512
Smets LA, Van Rooij H, Salomons GS. Signalling steps in apoptosis by ether lipids. Apoptosis. 1999 Dec;4(6):419-27. doi: 10.1023/a:1009644208512. PMID: 14634326.
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Over-expression of Bcl-2 protects against apoptosis induced by the bioreductive cytotoxic drug SR4233 (Tirapazamine).

Cell death and differentiation

Gilbert MS, Rupnow BA, Ramirez DA, Trisler KD, Knox SJ.
PMID: 17180085
Cell Death Differ. 1996 Apr;3(2):215-22.

The human B-cell lymphoma cell line PW undergoes radiation-induced programmed cell death (PCD). Bcl-2 transfected PW cells, that overexpressed Bcl-2, were significantly more radioresistant than parental or neomycin control transfected PW cells. The viability of Bcl-2 transfected cells was...

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Gilbert MS, Rupnow BA, Ramirez DA, et al. Over-expression of Bcl-2 protects against apoptosis induced by the bioreductive cytotoxic drug SR4233 (Tirapazamine). Cell Death Differ. 1996;3(2):215-22
Gilbert, M. S., Rupnow, B. A., Ramirez, D. A., Trisler, K. D., & Knox, S. J. (1996). Over-expression of Bcl-2 protects against apoptosis induced by the bioreductive cytotoxic drug SR4233 (Tirapazamine). Cell death and differentiation, 3(2), 215-22.
Gilbert, M S, et al. "Over-expression of Bcl-2 protects against apoptosis induced by the bioreductive cytotoxic drug SR4233 (Tirapazamine)." Cell death and differentiation vol. 3,2 (1996): 215-22.
Gilbert MS, Rupnow BA, Ramirez DA, Trisler KD, Knox SJ. Over-expression of Bcl-2 protects against apoptosis induced by the bioreductive cytotoxic drug SR4233 (Tirapazamine). Cell Death Differ. 1996 Apr;3(2):215-22. PMID: 17180085.
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Reduction of phenoxyl radicals of the antitumour agent etoposide (VP-16) by glutathione and protein sulfhydryls in human leukaemia cells: Implications for cytotoxicity.

Toxicology in vitro : an international journal published in association with BIBRA

Yalowich JC, Tyurina YY, Tyurin VA, Allan WP, Kagan VE.
PMID: 20650183
Toxicol In Vitro. 1996 Feb;10(1):59-68. doi: 10.1016/0887-2333(95)00106-9.

Phenoxyl radicals are inevitable intermediates in the oxidative enzymatic metabolism of a phenolic antitumour drug, etoposide (VP-16), by peroxidases, cytochrome P-450, prostaglandin synthetase and tyrosinase, as well as in its interactions with oxygen and peroxyl radicals. It has been...

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Yalowich JC, Tyurina YY, Tyurin VA, et al. Reduction of phenoxyl radicals of the antitumour agent etoposide (VP-16) by glutathione and protein sulfhydryls in human leukaemia cells: Implications for cytotoxicity. Toxicol In Vitro. 1996;10(1):59-68doi: 10.1016/0887-2333(95)00106-9.
Yalowich, J. C., Tyurina, Y. Y., Tyurin, V. A., Allan, W. P., & Kagan, V. E. (1996). Reduction of phenoxyl radicals of the antitumour agent etoposide (VP-16) by glutathione and protein sulfhydryls in human leukaemia cells: Implications for cytotoxicity. Toxicology in vitro : an international journal published in association with BIBRA, 10(1), 59-68. https://doi.org/10.1016/0887-2333(95)00106-9
Yalowich, J C, et al. "Reduction of phenoxyl radicals of the antitumour agent etoposide (VP-16) by glutathione and protein sulfhydryls in human leukaemia cells: Implications for cytotoxicity." Toxicology in vitro : an international journal published in association with BIBRA vol. 10,1 (1996): 59-68. doi: https://doi.org/10.1016/0887-2333(95)00106-9
Yalowich JC, Tyurina YY, Tyurin VA, Allan WP, Kagan VE. Reduction of phenoxyl radicals of the antitumour agent etoposide (VP-16) by glutathione and protein sulfhydryls in human leukaemia cells: Implications for cytotoxicity. Toxicol In Vitro. 1996 Feb;10(1):59-68. doi: 10.1016/0887-2333(95)00106-9. PMID: 20650183.
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The modulation of intracellular glutathione level modulates the mitochondrial response in proliferating rat splenocytes.

Archives of gerontology and geriatrics

Pieri C, Marra M, Moroni F, Marcheselli F, Benatti C.
PMID: 15374208
Arch Gerontol Geriatr. 1995 Sep-Oct;21(2):115-25. doi: 10.1016/0167-4943(95)00630-4.

We have investigated the effect of intracellular reduced glutathione (GSH) levels on the mitochondrial parameters of proliferating rat splenocytes. The intracellular GSH content of the cells was altered by continuous culture with buthionine-S, R-sulfoximine (BSO), a specific inhibitor of...

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Pieri C, Marra M, Moroni F, et al. The modulation of intracellular glutathione level modulates the mitochondrial response in proliferating rat splenocytes. Arch Gerontol Geriatr. 1995;21(2):115-25doi: 10.1016/0167-4943(95)00630-4.
Pieri, C., Marra, M., Moroni, F., Marcheselli, F., & Benatti, C. (1995). The modulation of intracellular glutathione level modulates the mitochondrial response in proliferating rat splenocytes. Archives of gerontology and geriatrics, 21(2), 115-25. https://doi.org/10.1016/0167-4943(95)00630-4
Pieri, C, et al. "The modulation of intracellular glutathione level modulates the mitochondrial response in proliferating rat splenocytes." Archives of gerontology and geriatrics vol. 21,2 (1995): 115-25. doi: https://doi.org/10.1016/0167-4943(95)00630-4
Pieri C, Marra M, Moroni F, Marcheselli F, Benatti C. The modulation of intracellular glutathione level modulates the mitochondrial response in proliferating rat splenocytes. Arch Gerontol Geriatr. 1995 Sep-Oct;21(2):115-25. doi: 10.1016/0167-4943(95)00630-4. PMID: 15374208.
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High Throughput Method for Assessment of Cellular Reduced Glutathione in Mammalian Cells.

Journal of environmental protection science

Cox DP, Cardozo-Pelaez F.
PMID: 20463849
J Environ Prot Sci. 2007 Jan 01;1:23-28.

Reduced glutathione (GSH) is an intracellular molecule essential for many aspects of cell physiology and defense. Determination of GSH has been used to identify potential anti-cancer drugs and for the assessment of drug toxicity via generation of oxidative stress....

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Cox DP, Cardozo-Pelaez F. High Throughput Method for Assessment of Cellular Reduced Glutathione in Mammalian Cells. J Environ Prot Sci. 2007;1:23-28
Cox, D. P., & Cardozo-Pelaez, F. (2007). High Throughput Method for Assessment of Cellular Reduced Glutathione in Mammalian Cells. Journal of environmental protection science, 123-28.
Cox, David P, and Cardozo-Pelaez, Fernando. "High Throughput Method for Assessment of Cellular Reduced Glutathione in Mammalian Cells." Journal of environmental protection science vol. 1 (2007): 23-28.
Cox DP, Cardozo-Pelaez F. High Throughput Method for Assessment of Cellular Reduced Glutathione in Mammalian Cells. J Environ Prot Sci. 2007 Jan 01;1:23-28. PMID: 20463849; PMCID: PMC2867479.
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RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors.

International journal of physiology, pathophysiology and pharmacology

Kim S, Dayani L, Rosenberg PA, Li J.
PMID: 20706550
Int J Physiol Pathophysiol Pharmacol. 2010;2(2):137-47. Epub 2010 Jun 12.

Oxidative damage is implicated in many neurological disorders including ischemic cerebral white matter injury. Oligodendrocyte precursors (preOLs) are intrinsically highly susceptible to various forms of oxidative stress. Here we report the identification of RIP1 kinase as a signaling molecule...

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Kim S, Dayani L, Rosenberg PA, et al. RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors. Int J Physiol Pathophysiol Pharmacol. 2010;2(2):137-47
Kim, S., Dayani, L., Rosenberg, P. A., & Li, J. (2010). RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors. International journal of physiology, pathophysiology and pharmacology, 2(2), 137-47.
Kim, Sunja, et al. "RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors." International journal of physiology, pathophysiology and pharmacology vol. 2,2 (2010): 137-47.
Kim S, Dayani L, Rosenberg PA, Li J. RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors. Int J Physiol Pathophysiol Pharmacol. 2010;2(2):137-47. Epub 2010 Jun 12. PMID: 20706550; PMCID: PMC2919773.
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Hydroxyapatite beads containing Doxorubicin hydrochloride (dox) and buthionine sulfoximine (bso) - a new anticancer drug design for local treatment with multiple-drugs.

Oncology reports

Imamura M, Seki T, Kunieda K, Nakatani S, Inoue K, Nakano T, Harada K.
PMID: 21597683
Oncol Rep. 1995 Jan;2(1):33-6. doi: 10.3892/or.2.1.33.

Doxorubicin hydrochloride (DOX) and buthionine sulfoximine (BSO) were adsorbed on to a drug-carrier, hydroxyapatite (HAP), to form a DOX and BSO-HAP complex. The time-course of the release of these drugs from the complex into phosphate-buffered saline (PBS) was measured...

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Imamura M, Seki T, Kunieda K, et al. Hydroxyapatite beads containing Doxorubicin hydrochloride (dox) and buthionine sulfoximine (bso) - a new anticancer drug design for local treatment with multiple-drugs. Oncol Rep. 1995;2(1):33-6doi: 10.3892/or.2.1.33.
Imamura, M., Seki, T., Kunieda, K., Nakatani, S., Inoue, K., Nakano, T., & Harada, K. (1995). Hydroxyapatite beads containing Doxorubicin hydrochloride (dox) and buthionine sulfoximine (bso) - a new anticancer drug design for local treatment with multiple-drugs. Oncology reports, 2(1), 33-6. https://doi.org/10.3892/or.2.1.33
Imamura, M, et al. "Hydroxyapatite beads containing Doxorubicin hydrochloride (dox) and buthionine sulfoximine (bso) - a new anticancer drug design for local treatment with multiple-drugs." Oncology reports vol. 2,1 (1995): 33-6. doi: https://doi.org/10.3892/or.2.1.33
Imamura M, Seki T, Kunieda K, Nakatani S, Inoue K, Nakano T, Harada K. Hydroxyapatite beads containing Doxorubicin hydrochloride (dox) and buthionine sulfoximine (bso) - a new anticancer drug design for local treatment with multiple-drugs. Oncol Rep. 1995 Jan;2(1):33-6. doi: 10.3892/or.2.1.33. PMID: 21597683.
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Changes in intracellular redox status influence multidrug resistance in gastric adenocarcinoma cells.

Experimental and therapeutic medicine

Tai DJ, Jin WS, Wu CS, Si HW, Cao XD, Guo AJ, Chang JC.
PMID: 23139717
Exp Ther Med. 2012 Aug;4(2):291-296. doi: 10.3892/etm.2012.591. Epub 2012 May 24.

Multidrug resistance (MDR) to chemotherapeutic agents is a major obstacle for the treatment of various types of cancers. The exact mechanism of MDR has not yet been fully clarified, although it has been frequently associated with the variation of...

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Tai DJ, Jin WS, Wu CS, et al. Changes in intracellular redox status influence multidrug resistance in gastric adenocarcinoma cells. Exp Ther Med. 2012;4(2):291-296doi: 10.3892/etm.2012.591.
Tai, D. J., Jin, W. S., Wu, C. S., Si, H. W., Cao, X. D., Guo, A. J., & Chang, J. C. (2012). Changes in intracellular redox status influence multidrug resistance in gastric adenocarcinoma cells. Experimental and therapeutic medicine, 4(2), 291-296. https://doi.org/10.3892/etm.2012.591
Tai, DE-Jun, et al. "Changes in intracellular redox status influence multidrug resistance in gastric adenocarcinoma cells." Experimental and therapeutic medicine vol. 4,2 (2012): 291-296. doi: https://doi.org/10.3892/etm.2012.591
Tai DJ, Jin WS, Wu CS, Si HW, Cao XD, Guo AJ, Chang JC. Changes in intracellular redox status influence multidrug resistance in gastric adenocarcinoma cells. Exp Ther Med. 2012 Aug;4(2):291-296. doi: 10.3892/etm.2012.591. Epub 2012 May 24. PMID: 23139717; PMCID: PMC3460287.
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Showing 1 to 12 of 150 entries