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Showing 1 to 12 of 72 entries
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Relationship between somatostatin and death receptor expression in the orbital frontal cortex in schizophrenia: a postmortem brain mRNA study.

NPJ schizophrenia

Joshi D, Catts VS, Olaya JC, Shannon Weickert C.
PMID: 27336026
NPJ Schizophr. 2015 Mar 04;1:14004. doi: 10.1038/npjschz.2014.4. eCollection 2015.

BACKGROUND: Recently, we provided evidence showing reductions in GAD67 and Dlx mRNAs in the orbital frontal cortex (OFC) in schizophrenia. It is unknown whether these reductions relate mainly to somatostatin (SST) or parvalbumin (PV) mRNA expression changes, and/or whether...

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Joshi D, Catts VS, Olaya JC, et al. Relationship between somatostatin and death receptor expression in the orbital frontal cortex in schizophrenia: a postmortem brain mRNA study. NPJ Schizophr. 2015;1:14004doi: 10.1038/npjschz.2014.4.
Joshi, D., Catts, V. S., Olaya, J. C., & Shannon Weickert, C. (2015). Relationship between somatostatin and death receptor expression in the orbital frontal cortex in schizophrenia: a postmortem brain mRNA study. NPJ schizophrenia, 114004. https://doi.org/10.1038/npjschz.2014.4
Joshi, Dipesh, et al. "Relationship between somatostatin and death receptor expression in the orbital frontal cortex in schizophrenia: a postmortem brain mRNA study." NPJ schizophrenia vol. 1 (2015): 14004. doi: https://doi.org/10.1038/npjschz.2014.4
Joshi D, Catts VS, Olaya JC, Shannon Weickert C. Relationship between somatostatin and death receptor expression in the orbital frontal cortex in schizophrenia: a postmortem brain mRNA study. NPJ Schizophr. 2015 Mar 04;1:14004. doi: 10.1038/npjschz.2014.4. eCollection 2015. PMID: 27336026; PMCID: PMC4849439.
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Preparation of a functional fluorescent human Fas ligand extracellular domain derivative using a three-dimensional structure guided site-specific fluorochrome conjugation.

SpringerPlus

Muraki M.
PMID: 27398274
Springerplus. 2016 Jul 07;5(1):997. doi: 10.1186/s40064-016-2673-8. eCollection 2016.

BACKGROUND: Human Fas ligand extracellular domain has been investigated as an important target protein in the field of medical biotechnology. In a recent study, the author developed an effective method to produce biologically active human Fas ligand extracellular domain...

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Muraki M. Preparation of a functional fluorescent human Fas ligand extracellular domain derivative using a three-dimensional structure guided site-specific fluorochrome conjugation. Springerplus. 2016;5(1):997doi: 10.1186/s40064-016-2673-8.
Muraki, M. (2016). Preparation of a functional fluorescent human Fas ligand extracellular domain derivative using a three-dimensional structure guided site-specific fluorochrome conjugation. SpringerPlus, 5(1), 997. https://doi.org/10.1186/s40064-016-2673-8
Muraki, Michiro. "Preparation of a functional fluorescent human Fas ligand extracellular domain derivative using a three-dimensional structure guided site-specific fluorochrome conjugation." SpringerPlus vol. 5,1 (2016): 997. doi: https://doi.org/10.1186/s40064-016-2673-8
Muraki M. Preparation of a functional fluorescent human Fas ligand extracellular domain derivative using a three-dimensional structure guided site-specific fluorochrome conjugation. Springerplus. 2016 Jul 07;5(1):997. doi: 10.1186/s40064-016-2673-8. eCollection 2016. PMID: 27398274; PMCID: PMC4936993.
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Monitoring of CD95 (APO-1/Fas) ligand expression in human T cells by quantitative RT-PCR.

Cell death and differentiation

Herr I, Balemans L, Böhler T, Walczak H, Debatin KM.
PMID: 17180098
Cell Death Differ. 1996 Jul;3(3):299-305.

CD95 (APO-1/Fas) receptor/ligand interaction is a key regulatory pathway for apoptosis in lymphoid cells. We developed a quantitative RT-PCR for the human CD95-L to determine expression levels in lymphoid cell lines and in lymphocytes derived from blood of healthy...

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Herr I, Balemans L, Böhler T, et al. Monitoring of CD95 (APO-1/Fas) ligand expression in human T cells by quantitative RT-PCR. Cell Death Differ. 1996;3(3):299-305
Herr, I., Balemans, L., Böhler, T., Walczak, H., & Debatin, K. M. (1996). Monitoring of CD95 (APO-1/Fas) ligand expression in human T cells by quantitative RT-PCR. Cell death and differentiation, 3(3), 299-305.
Herr, I, et al. "Monitoring of CD95 (APO-1/Fas) ligand expression in human T cells by quantitative RT-PCR." Cell death and differentiation vol. 3,3 (1996): 299-305.
Herr I, Balemans L, Böhler T, Walczak H, Debatin KM. Monitoring of CD95 (APO-1/Fas) ligand expression in human T cells by quantitative RT-PCR. Cell Death Differ. 1996 Jul;3(3):299-305. PMID: 17180098.
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Role of apoptosis resistance in immune evasion and metastasis of colorectal cancer.

World journal of gastrointestinal oncology

Liu K.
PMID: 21160903
World J Gastrointest Oncol. 2010 Nov 15;2(11):399-406. doi: 10.4251/wjgo.v2.i11.399.

The host immune system functions as a guardian against tumor development. It has been demonstrated that cytotoxic T lymphocyte (CTL)-mediated cytotoxic pathways function to inhibit or delay human colorectal cancer development. However, the host anti-tumor immune responses also 'edit'...

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Liu K. Role of apoptosis resistance in immune evasion and metastasis of colorectal cancer. World J Gastrointest Oncol. 2010;2(11):399-406doi: 10.4251/wjgo.v2.i11.399.
Liu, K. (2010). Role of apoptosis resistance in immune evasion and metastasis of colorectal cancer. World journal of gastrointestinal oncology, 2(11), 399-406. https://doi.org/10.4251/wjgo.v2.i11.399
Liu, Kebin. "Role of apoptosis resistance in immune evasion and metastasis of colorectal cancer." World journal of gastrointestinal oncology vol. 2,11 (2010): 399-406. doi: https://doi.org/10.4251/wjgo.v2.i11.399
Liu K. Role of apoptosis resistance in immune evasion and metastasis of colorectal cancer. World J Gastrointest Oncol. 2010 Nov 15;2(11):399-406. doi: 10.4251/wjgo.v2.i11.399. PMID: 21160903; PMCID: PMC3000453.
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A mathematical model of receptor-mediated apoptosis: dying to know why fasl is a trimer.

Mathematical biosciences and engineering : MBE

Lai R, Jackson TL.
PMID: 20369974
Math Biosci Eng. 2004 Sep;1(2):325-38. doi: 10.3934/mbe.2004.1.325.

The scientific importance of understanding programmed cell death is undeniable; however, the complexity of death signal propagation and the formerly incomplete knowledge of apoptotic pathways has left this topic virtually untouched by mathematical modeling. In this paper, we use...

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Lai R, Jackson TL. A mathematical model of receptor-mediated apoptosis: dying to know why fasl is a trimer. Math Biosci Eng. 2004;1(2):325-38doi: 10.3934/mbe.2004.1.325.
Lai, R., & Jackson, T. L. (2004). A mathematical model of receptor-mediated apoptosis: dying to know why fasl is a trimer. Mathematical biosciences and engineering : MBE, 1(2), 325-38. https://doi.org/10.3934/mbe.2004.1.325
Lai, Ronald, and Jackson, Trachette L. "A mathematical model of receptor-mediated apoptosis: dying to know why fasl is a trimer." Mathematical biosciences and engineering : MBE vol. 1,2 (2004): 325-38. doi: https://doi.org/10.3934/mbe.2004.1.325
Lai R, Jackson TL. A mathematical model of receptor-mediated apoptosis: dying to know why fasl is a trimer. Math Biosci Eng. 2004 Sep;1(2):325-38. doi: 10.3934/mbe.2004.1.325. PMID: 20369974.
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Phyllanthus urinaria Induces Apoptosis in Human Osteosarcoma 143B Cells via Activation of Fas/FasL- and Mitochondria-Mediated Pathways.

Evidence-based complementary and alternative medicine : eCAM

Wu HY, Lin TK, Kuo HM, Huang YL, Liou CW, Wang PW, Chuang JH, Huang ST.
PMID: 22454688
Evid Based Complement Alternat Med. 2012;2012:925824. doi: 10.1155/2012/925824. Epub 2012 Feb 20.

Phyllanthus urinaria (P. urinaria), in this study, was used for the treatment of human osteosarcoma cells, which is one of the tough malignancies with few therapeutic modalities. Herein, we demonstrated that P. urinaria inhibited human osteosarcoma 143B cells growth...

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Wu HY, Lin TK, Kuo HM, et al. Phyllanthus urinaria Induces Apoptosis in Human Osteosarcoma 143B Cells via Activation of Fas/FasL- and Mitochondria-Mediated Pathways. Evid Based Complement Alternat Med. 2012;2012:925824doi: 10.1155/2012/925824.
Wu, H. Y., Lin, T. K., Kuo, H. M., Huang, Y. L., Liou, C. W., Wang, P. W., Chuang, J. H., & Huang, S. T. (2012). Phyllanthus urinaria Induces Apoptosis in Human Osteosarcoma 143B Cells via Activation of Fas/FasL- and Mitochondria-Mediated Pathways. Evidence-based complementary and alternative medicine : eCAM, 2012925824. https://doi.org/10.1155/2012/925824
Wu, Hsin-Yi, et al. "Phyllanthus urinaria Induces Apoptosis in Human Osteosarcoma 143B Cells via Activation of Fas/FasL- and Mitochondria-Mediated Pathways." Evidence-based complementary and alternative medicine : eCAM vol. 2012 (2012): 925824. doi: https://doi.org/10.1155/2012/925824
Wu HY, Lin TK, Kuo HM, Huang YL, Liou CW, Wang PW, Chuang JH, Huang ST. Phyllanthus urinaria Induces Apoptosis in Human Osteosarcoma 143B Cells via Activation of Fas/FasL- and Mitochondria-Mediated Pathways. Evid Based Complement Alternat Med. 2012;2012:925824. doi: 10.1155/2012/925824. Epub 2012 Feb 20. PMID: 22454688; PMCID: PMC3291129.
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The role of fas/fas ligand system in the pathogenesis of liver cirrhosis and hepatocellular carcinoma.

Hepatitis monthly

Hammam O, Mahmoud O, Zahran M, Aly S, Hosny K, Helmy A, Anas A.
PMID: 23300494
Hepat Mon. 2012 Nov;12(11):e6132. doi: 10.5812/hepatmon.6132. Epub 2012 Nov 03.

BACKGROUND: The Fas receptor/ligand system including soluble forms is the most important apoptotic initiator in the liver. Dysregulation of this pathway may contribute to abnormal cell proliferation and cell death and is regarded as one of the mechanisms preventing...

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Hammam O, Mahmoud O, Zahran M, et al. The role of fas/fas ligand system in the pathogenesis of liver cirrhosis and hepatocellular carcinoma. Hepat Mon. 2012;12(11):e6132doi: 10.5812/hepatmon.6132.
Hammam, O., Mahmoud, O., Zahran, M., Aly, S., Hosny, K., Helmy, A., & Anas, A. (2012). The role of fas/fas ligand system in the pathogenesis of liver cirrhosis and hepatocellular carcinoma. Hepatitis monthly, 12(11), e6132. https://doi.org/10.5812/hepatmon.6132
Hammam, Olfat, et al. "The role of fas/fas ligand system in the pathogenesis of liver cirrhosis and hepatocellular carcinoma." Hepatitis monthly vol. 12,11 (2012): e6132. doi: https://doi.org/10.5812/hepatmon.6132
Hammam O, Mahmoud O, Zahran M, Aly S, Hosny K, Helmy A, Anas A. The role of fas/fas ligand system in the pathogenesis of liver cirrhosis and hepatocellular carcinoma. Hepat Mon. 2012 Nov;12(11):e6132. doi: 10.5812/hepatmon.6132. Epub 2012 Nov 03. PMID: 23300494; PMCID: PMC3539063.
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Decrease in Fas-induced apoptosis by the γ-secretase inhibitor is dependent on p75(NTR) in a glioblastoma cell line.

Experimental and therapeutic medicine

Bessette B, Durand K, Giraud S, Bégaud G, Mathonnet M, Lalloué F.
PMID: 22969985
Exp Ther Med. 2012 May;3(5):873-877. doi: 10.3892/etm.2012.480. Epub 2012 Feb 13.

p75(NTR), a member of the tumor necrosis factor superfamily, plays a key role in numerous physiological processes, including cell survival or apoptosis. Yet, the associated signaling pathways remain poorly understood. Similar to Notch, γ-secretase cleavage is implicated in the...

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Bessette B, Durand K, Giraud S, et al. Decrease in Fas-induced apoptosis by the γ-secretase inhibitor is dependent on p75(NTR) in a glioblastoma cell line. Exp Ther Med. 2012;3(5):873-877doi: 10.3892/etm.2012.480.
Bessette, B., Durand, K., Giraud, S., Bégaud, G., Mathonnet, M., & Lalloué, F. (2012). Decrease in Fas-induced apoptosis by the γ-secretase inhibitor is dependent on p75(NTR) in a glioblastoma cell line. Experimental and therapeutic medicine, 3(5), 873-877. https://doi.org/10.3892/etm.2012.480
Bessette, Barbara, et al. "Decrease in Fas-induced apoptosis by the γ-secretase inhibitor is dependent on p75(NTR) in a glioblastoma cell line." Experimental and therapeutic medicine vol. 3,5 (2012): 873-877. doi: https://doi.org/10.3892/etm.2012.480
Bessette B, Durand K, Giraud S, Bégaud G, Mathonnet M, Lalloué F. Decrease in Fas-induced apoptosis by the γ-secretase inhibitor is dependent on p75(NTR) in a glioblastoma cell line. Exp Ther Med. 2012 May;3(5):873-877. doi: 10.3892/etm.2012.480. Epub 2012 Feb 13. PMID: 22969985; PMCID: PMC3438831.
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Apoptosis induced by Fas signaling does not alter hepatic hepcidin expression.

World journal of biological chemistry

Lu S, Zmijewski E, Gollan J, Harrison-Findik DD.
PMID: 25225605
World J Biol Chem. 2014 Aug 26;5(3):387-97. doi: 10.4331/wjbc.v5.i3.387.

AIM: To determine the regulation of human hepcidin (HAMP) and mouse hepcidin (hepcidin-1 and hepcidin-2) gene expression in the liver by apoptosis using in vivo and in vitro experimental models.METHODS: For the induction of the extrinsic apoptotic pathway, HepG2...

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Lu S, Zmijewski E, Gollan J, et al. Apoptosis induced by Fas signaling does not alter hepatic hepcidin expression. World J Biol Chem. 2014;5(3):387-97doi: 10.4331/wjbc.v5.i3.387.
Lu, S., Zmijewski, E., Gollan, J., & Harrison-Findik, D. D. (2014). Apoptosis induced by Fas signaling does not alter hepatic hepcidin expression. World journal of biological chemistry, 5(3), 387-97. https://doi.org/10.4331/wjbc.v5.i3.387
Lu, Sizhao, et al. "Apoptosis induced by Fas signaling does not alter hepatic hepcidin expression." World journal of biological chemistry vol. 5,3 (2014): 387-97. doi: https://doi.org/10.4331/wjbc.v5.i3.387
Lu S, Zmijewski E, Gollan J, Harrison-Findik DD. Apoptosis induced by Fas signaling does not alter hepatic hepcidin expression. World J Biol Chem. 2014 Aug 26;5(3):387-97. doi: 10.4331/wjbc.v5.i3.387. PMID: 25225605; PMCID: PMC4160531.
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Molecular chaperones and hypoxic-ischemic encephalopathy.

Neural regeneration research

Hua C, Ju WN, Jin H, Sun X, Zhao G.
PMID: 28250763
Neural Regen Res. 2017 Jan;12(1):153-160. doi: 10.4103/1673-5374.199008.

Hypoxic-ischemic encephalopathy (HIE) is a disease that occurs when the brain is subjected to hypoxia, resulting in neuronal death and neurological deficits, with a poor prognosis. The mechanisms underlying hypoxic-ischemic brain injury include excitatory amino acid release, cellular proteolysis,...

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Hua C, Ju WN, Jin H, et al. Molecular chaperones and hypoxic-ischemic encephalopathy. Neural Regen Res. 2017;12(1):153-160doi: 10.4103/1673-5374.199008.
Hua, C., Ju, W. N., Jin, H., Sun, X., & Zhao, G. (2017). Molecular chaperones and hypoxic-ischemic encephalopathy. Neural regeneration research, 12(1), 153-160. https://doi.org/10.4103/1673-5374.199008
Hua, Cong, et al. "Molecular chaperones and hypoxic-ischemic encephalopathy." Neural regeneration research vol. 12,1 (2017): 153-160. doi: https://doi.org/10.4103/1673-5374.199008
Hua C, Ju WN, Jin H, Sun X, Zhao G. Molecular chaperones and hypoxic-ischemic encephalopathy. Neural Regen Res. 2017 Jan;12(1):153-160. doi: 10.4103/1673-5374.199008. PMID: 28250763; PMCID: PMC5319223.
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Maternal undernutrition upregulates apoptosis in offspring nephrogenesis.

Journal of developmental origins of health and disease

Tafti SA, Nast CC, Desai M, Amaya KE, Ross MG, Magee TR.
PMID: 25141167
J Dev Orig Health Dis. 2011 Aug;2(4):226-35. doi: 10.1017/S2040174411000262.

Maternal undernutrition (MUN) results in growth-restricted newborns with reduced nephron numbers that is associated with increased risk of hypertension and renal disease. The total adult complement of nephrons is set during nephrogenesis suggesting that MUN affects the staged development...

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Tafti SA, Nast CC, Desai M, et al. Maternal undernutrition upregulates apoptosis in offspring nephrogenesis. J Dev Orig Health Dis. 2011;2(4):226-35doi: 10.1017/S2040174411000262.
Tafti, S. A., Nast, C. C., Desai, M., Amaya, K. E., Ross, M. G., & Magee, T. R. (2011). Maternal undernutrition upregulates apoptosis in offspring nephrogenesis. Journal of developmental origins of health and disease, 2(4), 226-35. https://doi.org/10.1017/S2040174411000262
Tafti, S A, et al. "Maternal undernutrition upregulates apoptosis in offspring nephrogenesis." Journal of developmental origins of health and disease vol. 2,4 (2011): 226-35. doi: https://doi.org/10.1017/S2040174411000262
Tafti SA, Nast CC, Desai M, Amaya KE, Ross MG, Magee TR. Maternal undernutrition upregulates apoptosis in offspring nephrogenesis. J Dev Orig Health Dis. 2011 Aug;2(4):226-35. doi: 10.1017/S2040174411000262. PMID: 25141167.
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Association Between Systemic Lupus Erythematosus and Myasthenia Gravis: Coincidence or Sequelae?.

Cureus

Raut S, Reddy I, Sahi FM, Masood A, Malik BH.
PMID: 32642338
Cureus. 2020 Jun 03;12(6):e8422. doi: 10.7759/cureus.8422.

Systemic lupus erythematosus (SLE) is a systemic autoimmune disease and myasthenia gravis (MG) is an organ-specific autoimmune disease, both may exhibit positive anti-nuclear antibodies and a female preponderance. They may have similar features and can coexist in a patient...

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Raut S, Reddy I, Sahi FM, et al. Association Between Systemic Lupus Erythematosus and Myasthenia Gravis: Coincidence or Sequelae?. Cureus. 2020;12(6):e8422doi: 10.7759/cureus.8422.
Raut, S., Reddy, I., Sahi, F. M., Masood, A., & Malik, B. H. (2020). Association Between Systemic Lupus Erythematosus and Myasthenia Gravis: Coincidence or Sequelae?. Cureus, 12(6), e8422. https://doi.org/10.7759/cureus.8422
Raut, Sumit, et al. "Association Between Systemic Lupus Erythematosus and Myasthenia Gravis: Coincidence or Sequelae?." Cureus vol. 12,6 (2020): e8422. doi: https://doi.org/10.7759/cureus.8422
Raut S, Reddy I, Sahi FM, Masood A, Malik BH. Association Between Systemic Lupus Erythematosus and Myasthenia Gravis: Coincidence or Sequelae?. Cureus. 2020 Jun 03;12(6):e8422. doi: 10.7759/cureus.8422. PMID: 32642338; PMCID: PMC7336596.
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