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Showing 1 to 12 of 46 entries
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From Locus Association to Mechanism of Gene Causality: The Devil Is in the Details.

Arteriosclerosis, thrombosis, and vascular biology

Miller CL, Pjanic M, Quertermous T.
PMID: 26399919
Arterioscler Thromb Vasc Biol. 2015 Oct;35(10):2079-2080. doi: 10.1161/ATVBAHA.115.306366.

No abstract available.

Cite
Miller CL, Pjanic M, Quertermous T. From Locus Association to Mechanism of Gene Causality: The Devil Is in the Details. Arterioscler Thromb Vasc Biol. 2015;35(10):2079-2080doi: 10.1161/ATVBAHA.115.306366.
Miller, C. L., Pjanic, M., & Quertermous, T. (2015). From Locus Association to Mechanism of Gene Causality: The Devil Is in the Details. Arteriosclerosis, thrombosis, and vascular biology, 35(10), 2079-2080. https://doi.org/10.1161/ATVBAHA.115.306366
Miller, Clint L, et al. "From Locus Association to Mechanism of Gene Causality: The Devil Is in the Details." Arteriosclerosis, thrombosis, and vascular biology vol. 35,10 (2015): 2079-2080. doi: https://doi.org/10.1161/ATVBAHA.115.306366
Miller CL, Pjanic M, Quertermous T. From Locus Association to Mechanism of Gene Causality: The Devil Is in the Details. Arterioscler Thromb Vasc Biol. 2015 Oct;35(10):2079-2080. doi: 10.1161/ATVBAHA.115.306366. PMID: 26399919; PMCID: PMC4594207.
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Genetic Risk, Lifestyle, and Coronary Artery Disease.

The New England journal of medicine

Khera AV, Emdin CA, Kathiresan S.
PMID: 28328341
N Engl J Med. 2017 Mar 23;376(12):1194-5. doi: 10.1056/NEJMc1700362.

No abstract available.

Cite
Khera AV, Emdin CA, Kathiresan S. Genetic Risk, Lifestyle, and Coronary Artery Disease. N Engl J Med. 2017;376(12):1194-5doi: 10.1056/NEJMc1700362.
Khera, A. V., Emdin, C. A., & Kathiresan, S. (2017). Genetic Risk, Lifestyle, and Coronary Artery Disease. The New England journal of medicine, 376(12), 1194-5. https://doi.org/10.1056/NEJMc1700362
Khera, Amit V, et al. "Genetic Risk, Lifestyle, and Coronary Artery Disease." The New England journal of medicine vol. 376,12 (2017): 1194-5. doi: https://doi.org/10.1056/NEJMc1700362
Khera AV, Emdin CA, Kathiresan S. Genetic Risk, Lifestyle, and Coronary Artery Disease. N Engl J Med. 2017 Mar 23;376(12):1194-5. doi: 10.1056/NEJMc1700362. PMID: 28328341.
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Patient-Friendly Summary of the ACR Appropriateness Criteria: Asymptomatic Patient at Risk for Coronary Artery Disease.

Journal of the American College of Radiology : JACR

Quinlan C.
PMID: 29519637
J Am Coll Radiol. 2018 Jul;15(7):e9. doi: 10.1016/j.jacr.2017.09.032. Epub 2018 Mar 05.

No abstract available.

Cite
Quinlan C. Patient-Friendly Summary of the ACR Appropriateness Criteria: Asymptomatic Patient at Risk for Coronary Artery Disease. J Am Coll Radiol. 2018;15(7):e9doi: 10.1016/j.jacr.2017.09.032.
Quinlan, C. (2018). Patient-Friendly Summary of the ACR Appropriateness Criteria: Asymptomatic Patient at Risk for Coronary Artery Disease. Journal of the American College of Radiology : JACR, 15(7), e9. https://doi.org/10.1016/j.jacr.2017.09.032
Quinlan, Casey. "Patient-Friendly Summary of the ACR Appropriateness Criteria: Asymptomatic Patient at Risk for Coronary Artery Disease." Journal of the American College of Radiology : JACR vol. 15,7 (2018): e9. doi: https://doi.org/10.1016/j.jacr.2017.09.032
Quinlan C. Patient-Friendly Summary of the ACR Appropriateness Criteria: Asymptomatic Patient at Risk for Coronary Artery Disease. J Am Coll Radiol. 2018 Jul;15(7):e9. doi: 10.1016/j.jacr.2017.09.032. Epub 2018 Mar 05. PMID: 29519637.
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Systems Genetics Analysis of Genome-Wide Association Study Reveals Novel Associations Between Key Biological Processes and Coronary Artery Disease.

Arteriosclerosis, thrombosis, and vascular biology

Ghosh S, Vivar J, Nelson CP, Willenborg C, Segrè AV, Mäkinen VP, Nikpay M, Erdmann J, Blankenberg S, O'Donnell C, März W, Laaksonen R, Stewart AF, Epstein SE, Shah SH, Granger CB, Hazen SL, Kathiresan S, Reilly MP, Yang X, Quertermous T, Samani NJ, Schunkert H, Assimes TL, McPherson R.
PMID: 25977570
Arterioscler Thromb Vasc Biol. 2015 Jul;35(7):1712-22. doi: 10.1161/ATVBAHA.115.305513. Epub 2015 May 14.

OBJECTIVE: Genome-wide association studies have identified multiple genetic variants affecting the risk of coronary artery disease (CAD). However, individually these explain only a small fraction of the heritability of CAD and for most, the causal biological mechanisms remain unclear....

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Ghosh S, Vivar J, Nelson CP, et al. Systems Genetics Analysis of Genome-Wide Association Study Reveals Novel Associations Between Key Biological Processes and Coronary Artery Disease. Arterioscler Thromb Vasc Biol. 2015;35(7):1712-22doi: 10.1161/ATVBAHA.115.305513.
Ghosh, S., Vivar, J., Nelson, C. P., Willenborg, C., Segrè, A. V., Mäkinen, V. P., Nikpay, M., Erdmann, J., Blankenberg, S., O'Donnell, C., März, W., Laaksonen, R., Stewart, A. F., Epstein, S. E., Shah, S. H., Granger, C. B., Hazen, S. L., Kathiresan, S., Reilly, M. P., Yang, X., Quertermous, T., Samani, N. J., Schunkert, H., Assimes, T. L., & McPherson, R. (2015). Systems Genetics Analysis of Genome-Wide Association Study Reveals Novel Associations Between Key Biological Processes and Coronary Artery Disease. Arteriosclerosis, thrombosis, and vascular biology, 35(7), 1712-22. https://doi.org/10.1161/ATVBAHA.115.305513
Ghosh, Sujoy, et al. "Systems Genetics Analysis of Genome-Wide Association Study Reveals Novel Associations Between Key Biological Processes and Coronary Artery Disease." Arteriosclerosis, thrombosis, and vascular biology vol. 35,7 (2015): 1712-22. doi: https://doi.org/10.1161/ATVBAHA.115.305513
Ghosh S, Vivar J, Nelson CP, Willenborg C, Segrè AV, Mäkinen VP, Nikpay M, Erdmann J, Blankenberg S, O'Donnell C, März W, Laaksonen R, Stewart AF, Epstein SE, Shah SH, Granger CB, Hazen SL, Kathiresan S, Reilly MP, Yang X, Quertermous T, Samani NJ, Schunkert H, Assimes TL, McPherson R. Systems Genetics Analysis of Genome-Wide Association Study Reveals Novel Associations Between Key Biological Processes and Coronary Artery Disease. Arterioscler Thromb Vasc Biol. 2015 Jul;35(7):1712-22. doi: 10.1161/ATVBAHA.115.305513. Epub 2015 May 14. PMID: 25977570; PMCID: PMC4841833.
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Type 1 diabetes and coronary artery disease.

Diabetes care

Orchard TJ, Costacou T, Kretowski A, Nesto RW.
PMID: 17065698
Diabetes Care. 2006 Nov;29(11):2528-38. doi: 10.2337/dc06-1161.

No abstract available.

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Orchard TJ, Costacou T, Kretowski A, et al. Type 1 diabetes and coronary artery disease. Diabetes Care. 2006;29(11):2528-38doi: 10.2337/dc06-1161.
Orchard, T. J., Costacou, T., Kretowski, A., & Nesto, R. W. (2006). Type 1 diabetes and coronary artery disease. Diabetes care, 29(11), 2528-38. https://doi.org/10.2337/dc06-1161
Orchard, Trevor J, et al. "Type 1 diabetes and coronary artery disease." Diabetes care vol. 29,11 (2006): 2528-38. doi: https://doi.org/10.2337/dc06-1161
Orchard TJ, Costacou T, Kretowski A, Nesto RW. Type 1 diabetes and coronary artery disease. Diabetes Care. 2006 Nov;29(11):2528-38. doi: 10.2337/dc06-1161. PMID: 17065698.
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Translating genomic analyses into improved management of coronary artery disease.

Future cardiology

Johansen CT, Lanktree MB, Hegele RA.
PMID: 20608823
Future Cardiol. 2010 Jul;6(4):507-21. doi: 10.2217/fca.10.28.

Human genetic variation can modulate pathophysiologic processes that alter susceptibility to complex disease. Recent genomic analyses have sought to identify how common genetic variation alters susceptibility to coronary artery disease (CAD). From genome-wide association studies (GWAS), common genetic variants...

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Johansen CT, Lanktree MB, Hegele RA. Translating genomic analyses into improved management of coronary artery disease. Future Cardiol. 2010;6(4):507-21doi: 10.2217/fca.10.28.
Johansen, C. T., Lanktree, M. B., & Hegele, R. A. (2010). Translating genomic analyses into improved management of coronary artery disease. Future cardiology, 6(4), 507-21. https://doi.org/10.2217/fca.10.28
Johansen, Christopher T, et al. "Translating genomic analyses into improved management of coronary artery disease." Future cardiology vol. 6,4 (2010): 507-21. doi: https://doi.org/10.2217/fca.10.28
Johansen CT, Lanktree MB, Hegele RA. Translating genomic analyses into improved management of coronary artery disease. Future Cardiol. 2010 Jul;6(4):507-21. doi: 10.2217/fca.10.28. PMID: 20608823.
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Considering a possible application to prevent or to treat arterial stiffness.

Heart (British Cardiac Society)

Spiegel R.
PMID: 23086967
Heart. 2013 Jan;99(1):66. doi: 10.1136/heartjnl-2012-302439. Epub 2012 Oct 19.

No abstract available.

Cite
Spiegel R. Considering a possible application to prevent or to treat arterial stiffness. Heart. 2012;99(1):66doi: 10.1136/heartjnl-2012-302439.
Spiegel, R. (2013). Considering a possible application to prevent or to treat arterial stiffness. Heart (British Cardiac Society), 99(1), 66. https://doi.org/10.1136/heartjnl-2012-302439
Spiegel, Rainer. "Considering a possible application to prevent or to treat arterial stiffness." Heart (British Cardiac Society) vol. 99,1 (2013): 66. doi: https://doi.org/10.1136/heartjnl-2012-302439
Spiegel R. Considering a possible application to prevent or to treat arterial stiffness. Heart. 2013 Jan;99(1):66. doi: 10.1136/heartjnl-2012-302439. Epub 2012 Oct 19. PMID: 23086967.
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Genetic Risk Stratification and Prevention of CAD: An Idea Whose Time Is Now.

Clinical chemistry

Roberts R.
PMID: 28931517
Clin Chem. 2017 Dec;63(12):1821-1823. doi: 10.1373/clinchem.2017.278895. Epub 2017 Sep 20.

No abstract available.

Cite
Roberts R. Genetic Risk Stratification and Prevention of CAD: An Idea Whose Time Is Now. Clin Chem. 2017;63(12):1821-1823doi: 10.1373/clinchem.2017.278895.
Roberts, R. (2017). Genetic Risk Stratification and Prevention of CAD: An Idea Whose Time Is Now. Clinical chemistry, 63(12), 1821-1823. https://doi.org/10.1373/clinchem.2017.278895
Roberts, Robert. "Genetic Risk Stratification and Prevention of CAD: An Idea Whose Time Is Now." Clinical chemistry vol. 63,12 (2017): 1821-1823. doi: https://doi.org/10.1373/clinchem.2017.278895
Roberts R. Genetic Risk Stratification and Prevention of CAD: An Idea Whose Time Is Now. Clin Chem. 2017 Dec;63(12):1821-1823. doi: 10.1373/clinchem.2017.278895. Epub 2017 Sep 20. PMID: 28931517.
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The author's reply.

Heart (British Cardiac Society)

Mulders TA, Pinto-Sietsma SJ.
PMID: 23250783
Heart. 2013 Jan;99(1):66-7. doi: 10.1136/heartjnl-2012-302440.

No abstract available.

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Mulders TA, Pinto-Sietsma SJ. The author's reply. Heart. 2013;99(1):66-7doi: 10.1136/heartjnl-2012-302440.
Mulders, T. A., & Pinto-Sietsma, S. J. (2013). The author's reply. Heart (British Cardiac Society), 99(1), 66-7. https://doi.org/10.1136/heartjnl-2012-302440
Mulders, Ties A, and Pinto-Sietsma, Sara-Joan. "The author's reply." Heart (British Cardiac Society) vol. 99,1 (2013): 66-7. doi: https://doi.org/10.1136/heartjnl-2012-302440
Mulders TA, Pinto-Sietsma SJ. The author's reply. Heart. 2013 Jan;99(1):66-7. doi: 10.1136/heartjnl-2012-302440. PMID: 23250783.
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Tissue-specific Network Analysis of Genetic Variants Associated with Coronary Artery Disease.

Scientific reports

Miao X, Chen X, Xie Z, Lin H.
PMID: 30065343
Sci Rep. 2018 Jul 31;8(1):11492. doi: 10.1038/s41598-018-29904-7.

Coronary artery disease (CAD) is a leading cause of death worldwide. Recent genome-wide association studies have identified more than one hundred susceptibility loci associated with CAD. However, the underlying mechanism of these genetic loci to CAD susceptibility is still...

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Miao X, Chen X, Xie Z, et al. Tissue-specific Network Analysis of Genetic Variants Associated with Coronary Artery Disease. Sci Rep. 2018;8(1):11492doi: 10.1038/s41598-018-29904-7.
Miao, X., Chen, X., Xie, Z., & Lin, H. (2018). Tissue-specific Network Analysis of Genetic Variants Associated with Coronary Artery Disease. Scientific reports, 8(1), 11492. https://doi.org/10.1038/s41598-018-29904-7
Miao, Xiao, et al. "Tissue-specific Network Analysis of Genetic Variants Associated with Coronary Artery Disease." Scientific reports vol. 8,1 (2018): 11492. doi: https://doi.org/10.1038/s41598-018-29904-7
Miao X, Chen X, Xie Z, Lin H. Tissue-specific Network Analysis of Genetic Variants Associated with Coronary Artery Disease. Sci Rep. 2018 Jul 31;8(1):11492. doi: 10.1038/s41598-018-29904-7. PMID: 30065343; PMCID: PMC6068195.
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Genome-wide association study of metabolites in patients with coronary artery disease identified novel metabolite quantitative trait loci.

Clinical and translational medicine

Wang Z, Zhu Q, Liu Y, Chen S, Zhang Y, Ma Q, Chen X, Liu C, Lei H, Chen H, Wang J, Zheng S, Li Z, Xiong L, Lai W, Zhong S.
PMID: 33634981
Clin Transl Med. 2021 Feb;11(2):e290. doi: 10.1002/ctm2.290.

No abstract available.

Cite
Wang Z, Zhu Q, Liu Y, et al. Genome-wide association study of metabolites in patients with coronary artery disease identified novel metabolite quantitative trait loci. Clin Transl Med. 2021;11(2):e290doi: 10.1002/ctm2.290.
Wang, Z., Zhu, Q., Liu, Y., Chen, S., Zhang, Y., Ma, Q., Chen, X., Liu, C., Lei, H., Chen, H., Wang, J., Zheng, S., Li, Z., Xiong, L., Lai, W., & Zhong, S. (2021). Genome-wide association study of metabolites in patients with coronary artery disease identified novel metabolite quantitative trait loci. Clinical and translational medicine, 11(2), e290. https://doi.org/10.1002/ctm2.290
Wang, Zixian, et al. "Genome-wide association study of metabolites in patients with coronary artery disease identified novel metabolite quantitative trait loci." Clinical and translational medicine vol. 11,2 (2021): e290. doi: https://doi.org/10.1002/ctm2.290
Wang Z, Zhu Q, Liu Y, Chen S, Zhang Y, Ma Q, Chen X, Liu C, Lei H, Chen H, Wang J, Zheng S, Li Z, Xiong L, Lai W, Zhong S. Genome-wide association study of metabolites in patients with coronary artery disease identified novel metabolite quantitative trait loci. Clin Transl Med. 2021 Feb;11(2):e290. doi: 10.1002/ctm2.290. PMID: 33634981; PMCID: PMC7839954.
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Construction of the gene expression subgroups of patients with coronary artery disease through bioinformatics approach.

Mathematical biosciences and engineering : MBE

Zhang B, Zeng K, Li R, Jiang H, Gao M, Zhang L, Li J, Guan R, Liu Y, Qiang Y, Yang Y.
PMID: 34814316
Math Biosci Eng. 2021 Oct 09;18(6):8622-8640. doi: 10.3934/mbe.2021427.

Coronary artery disease (CAD) is a heterogeneous disease that has placed a heavy burden on public health due to its considerable morbidity, mortality and high costs. Better understanding of the genetic drivers and gene expression clustering behind CAD will...

Cite
Zhang B, Zeng K, Li R, et al. Construction of the gene expression subgroups of patients with coronary artery disease through bioinformatics approach. Math Biosci Eng. 2021;18(6):8622-8640doi: 10.3934/mbe.2021427.
Zhang, B., Zeng, K., Li, R., Jiang, H., Gao, M., Zhang, L., Li, J., Guan, R., Liu, Y., Qiang, Y., & Yang, Y. (2021). Construction of the gene expression subgroups of patients with coronary artery disease through bioinformatics approach. Mathematical biosciences and engineering : MBE, 18(6), 8622-8640. https://doi.org/10.3934/mbe.2021427
Zhang, Bin, et al. "Construction of the gene expression subgroups of patients with coronary artery disease through bioinformatics approach." Mathematical biosciences and engineering : MBE vol. 18,6 (2021): 8622-8640. doi: https://doi.org/10.3934/mbe.2021427
Zhang B, Zeng K, Li R, Jiang H, Gao M, Zhang L, Li J, Guan R, Liu Y, Qiang Y, Yang Y. Construction of the gene expression subgroups of patients with coronary artery disease through bioinformatics approach. Math Biosci Eng. 2021 Oct 09;18(6):8622-8640. doi: 10.3934/mbe.2021427. PMID: 34814316.
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Showing 1 to 12 of 46 entries