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Panobinostat enhances olaparib efficacy by modifying expression of homologous recombination repair and immune transcripts in ovarian cancer.

Neoplasia (New York, N.Y.)

Wilson AJ, Gupta VG, Liu Q, Yull F, Crispens MA, Khabele D.
PMID: 34933276
Neoplasia. 2022 Feb;24(2):63-75. doi: 10.1016/j.neo.2021.12.002. Epub 2021 Dec 20.

Histone deacetylase inhibitors (HDACi) sensitize homologous recombination (HR)-proficient human ovarian cancer cells to PARP inhibitors (PARPi). To investigate mechanisms of anti-tumor effects of combined HDACi/PARPi treatment we performed transcriptome analysis in HR- proficient human ovarian cancer cells and tested...

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Wilson AJ, Gupta VG, Liu Q, et al. Panobinostat enhances olaparib efficacy by modifying expression of homologous recombination repair and immune transcripts in ovarian cancer. Neoplasia. 2021;24(2):63-75doi: 10.1016/j.neo.2021.12.002.
Wilson, A. J., Gupta, V. G., Liu, Q., Yull, F., Crispens, M. A., & Khabele, D. (2022). Panobinostat enhances olaparib efficacy by modifying expression of homologous recombination repair and immune transcripts in ovarian cancer. Neoplasia (New York, N.Y.), 24(2), 63-75. https://doi.org/10.1016/j.neo.2021.12.002
Wilson, Andrew J, et al. "Panobinostat enhances olaparib efficacy by modifying expression of homologous recombination repair and immune transcripts in ovarian cancer." Neoplasia (New York, N.Y.) vol. 24,2 (2022): 63-75. doi: https://doi.org/10.1016/j.neo.2021.12.002
Wilson AJ, Gupta VG, Liu Q, Yull F, Crispens MA, Khabele D. Panobinostat enhances olaparib efficacy by modifying expression of homologous recombination repair and immune transcripts in ovarian cancer. Neoplasia. 2022 Feb;24(2):63-75. doi: 10.1016/j.neo.2021.12.002. Epub 2021 Dec 20. PMID: 34933276; PMCID: PMC8702851.
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Loss of Corpus-Specific Lipids in Helicobacter pylori-Induced Atrophic Gastritis.

mSphere

Lin AS, Shuman JHB, Kotnala A, Shaw JA, Beckett AC, Harvey JL, Tuck M, Dixon BREA, Reyzer ML, Algood HMS, Schey KL, Piazuelo MB, Cover TL.
PMID: 34817238
mSphere. 2021 Nov 24;e0082621. doi: 10.1128/mSphere.00826-21. Epub 2021 Nov 24.

Helicobacter pylori colonization of the stomach is a strong risk factor for the development of stomach cancer and peptic ulcer disease. In this study, we tested the hypothesis that H. pylori infection triggers alterations in gastric lipid composition. Mongolian...

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Lin AS, Shuman JHB, Kotnala A, et al. Loss of Corpus-Specific Lipids in Helicobacter pylori-Induced Atrophic Gastritis. mSphere. 2021;e0082621doi: 10.1128/mSphere.00826-21.
Lin, A. S., Shuman, J. H. B., Kotnala, A., Shaw, J. A., Beckett, A. C., Harvey, J. L., Tuck, M., Dixon, B. R. E. A., Reyzer, M. L., Algood, H. M. S., Schey, K. L., Piazuelo, M. B., & Cover, T. L. (2021). Loss of Corpus-Specific Lipids in Helicobacter pylori-Induced Atrophic Gastritis. mSphere, e0082621. https://doi.org/10.1128/mSphere.00826-21
Lin, Aung Soe, et al. "Loss of Corpus-Specific Lipids in Helicobacter pylori-Induced Atrophic Gastritis." mSphere vol. (2021): e0082621. doi: https://doi.org/10.1128/mSphere.00826-21
Lin AS, Shuman JHB, Kotnala A, Shaw JA, Beckett AC, Harvey JL, Tuck M, Dixon BREA, Reyzer ML, Algood HMS, Schey KL, Piazuelo MB, Cover TL. Loss of Corpus-Specific Lipids in Helicobacter pylori-Induced Atrophic Gastritis. mSphere. 2021 Nov 24;e0082621. doi: 10.1128/mSphere.00826-21. Epub 2021 Nov 24. PMID: 34817238; PMCID: PMC8612251.
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High-throughput drug screening of fine-needle aspiration-derived cancer organoids.

STAR protocols

Bergdorf K, Phifer C, Bharti V, Westover D, Bauer J, Vilgelm A, Lee E, Weiss V.
PMID: 33377106
STAR Protoc. 2020 Dec 16;1(3):100212. doi: 10.1016/j.xpro.2020.100212. eCollection 2020 Dec 18.

Generation of fine-needle aspiration (FNA)-derived cancer organoids has allowed us to develop a number of downstream applications. In this protocol, we start with organoids cultured in a semi-solid format. We dissociate organoids into single cells and then plate in...

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Bergdorf K, Phifer C, Bharti V, et al. High-throughput drug screening of fine-needle aspiration-derived cancer organoids. STAR Protoc. 2020;1(3):100212doi: 10.1016/j.xpro.2020.100212.
Bergdorf, K., Phifer, C., Bharti, V., Westover, D., Bauer, J., Vilgelm, A., Lee, E., & Weiss, V. (2020). High-throughput drug screening of fine-needle aspiration-derived cancer organoids. STAR protocols, 1(3), 100212. https://doi.org/10.1016/j.xpro.2020.100212
Bergdorf, Kensey, et al. "High-throughput drug screening of fine-needle aspiration-derived cancer organoids." STAR protocols vol. 1,3 (2020): 100212. doi: https://doi.org/10.1016/j.xpro.2020.100212
Bergdorf K, Phifer C, Bharti V, Westover D, Bauer J, Vilgelm A, Lee E, Weiss V. High-throughput drug screening of fine-needle aspiration-derived cancer organoids. STAR Protoc. 2020 Dec 16;1(3):100212. doi: 10.1016/j.xpro.2020.100212. eCollection 2020 Dec 18. PMID: 33377106; PMCID: PMC7757655.
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Adverse Childhood Experiences and Chronic Disease Risk in the Southern Community Cohort Study.

Journal of health care for the poor and underserved

Sanderson M, Mouton CP, Cook M, Liu J, Blot WJ, Hargreaves MK.
PMID: 34421038
J Health Care Poor Underserved. 2021;32(3):1384-1402. doi: 10.1353/hpu.2021.0139.

We used the Southern Community Cohort Study of people residing in 12 states in the southeastern United States (n=38,200 participants) to examine associations between adverse childhood experiences (ACEs) and chronic disease risk. After adjustment for confounding, there were statistically...

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Sanderson M, Mouton CP, Cook M, et al. Adverse Childhood Experiences and Chronic Disease Risk in the Southern Community Cohort Study. J Health Care Poor Underserved. 2021;32(3):1384-1402doi: 10.1353/hpu.2021.0139.
Sanderson, M., Mouton, C. P., Cook, M., Liu, J., Blot, W. J., & Hargreaves, M. K. (2021). Adverse Childhood Experiences and Chronic Disease Risk in the Southern Community Cohort Study. Journal of health care for the poor and underserved, 32(3), 1384-1402. https://doi.org/10.1353/hpu.2021.0139
Sanderson, Maureen, et al. "Adverse Childhood Experiences and Chronic Disease Risk in the Southern Community Cohort Study." Journal of health care for the poor and underserved vol. 32,3 (2021): 1384-1402. doi: https://doi.org/10.1353/hpu.2021.0139
Sanderson M, Mouton CP, Cook M, Liu J, Blot WJ, Hargreaves MK. Adverse Childhood Experiences and Chronic Disease Risk in the Southern Community Cohort Study. J Health Care Poor Underserved. 2021;32(3):1384-1402. doi: 10.1353/hpu.2021.0139. PMID: 34421038; PMCID: PMC8462987.
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A head-to-head comparison of ribodepletion and polyA selection approaches for C. elegans low input RNA-sequencing libraries.

G3 (Bethesda, Md.)

Barrett A, McWhirter R, Taylor SR, Weinreb A, Miller DM, Hammarlund M.
PMID: 33856427
G3 (Bethesda). 2021 Apr 15; doi: 10.1093/g3journal/jkab121. Epub 2021 Apr 15.

A recent and powerful technique is to obtain transcriptomes from rare cell populations, such as single neurons in C. elegans, by enriching dissociated cells using fluorescent sorting. However, these cell samples often have low yields of RNA that present...

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Barrett A, McWhirter R, Taylor SR, et al. A head-to-head comparison of ribodepletion and polyA selection approaches for C. elegans low input RNA-sequencing libraries. G3 (Bethesda). 2021;doi: 10.1093/g3journal/jkab121.
Barrett, A., McWhirter, R., Taylor, S. R., Weinreb, A., Miller, D. M., & Hammarlund, M. (2021). A head-to-head comparison of ribodepletion and polyA selection approaches for C. elegans low input RNA-sequencing libraries. G3 (Bethesda, Md.), . https://doi.org/10.1093/g3journal/jkab121
Barrett, Alec, et al. "A head-to-head comparison of ribodepletion and polyA selection approaches for C. elegans low input RNA-sequencing libraries." G3 (Bethesda, Md.) vol. (2021). doi: https://doi.org/10.1093/g3journal/jkab121
Barrett A, McWhirter R, Taylor SR, Weinreb A, Miller DM, Hammarlund M. A head-to-head comparison of ribodepletion and polyA selection approaches for C. elegans low input RNA-sequencing libraries. G3 (Bethesda). 2021 Apr 15; doi: 10.1093/g3journal/jkab121. Epub 2021 Apr 15. PMID: 33856427; PMCID: PMC8495925.
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Metabolic modulation by CDK4/6 inhibitor promotes chemokine-mediated recruitment of T cells into mammary tumors.

Cell reports

Uzhachenko RV, Bharti V, Ouyang Z, Blevins A, Mont S, Saleh N, Lawrence HA, Shen C, Chen SC, Ayers GD, DeNardo DG, Arteaga C, Richmond A, Vilgelm AE.
PMID: 33826903
Cell Rep. 2021 Apr 06;35(1):108944. doi: 10.1016/j.celrep.2021.108944.

Inhibitors of cyclin-dependent kinases 4 and 6 (CDK4/6i) delay progression of metastatic breast cancer. However, complete responses are uncommon and tumors eventually relapse. Here, we show that CDK4/6i can enhance efficacy of T cell-based therapies, such as adoptive T...

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Uzhachenko RV, Bharti V, Ouyang Z, et al. Metabolic modulation by CDK4/6 inhibitor promotes chemokine-mediated recruitment of T cells into mammary tumors. Cell Rep. 2021;35(1):108944doi: 10.1016/j.celrep.2021.108944.
Uzhachenko, R. V., Bharti, V., Ouyang, Z., Blevins, A., Mont, S., Saleh, N., Lawrence, H. A., Shen, C., Chen, S. C., Ayers, G. D., DeNardo, D. G., Arteaga, C., Richmond, A., & Vilgelm, A. E. (2021). Metabolic modulation by CDK4/6 inhibitor promotes chemokine-mediated recruitment of T cells into mammary tumors. Cell reports, 35(1), 108944. https://doi.org/10.1016/j.celrep.2021.108944
Uzhachenko, Roman V, et al. "Metabolic modulation by CDK4/6 inhibitor promotes chemokine-mediated recruitment of T cells into mammary tumors." Cell reports vol. 35,1 (2021): 108944. doi: https://doi.org/10.1016/j.celrep.2021.108944
Uzhachenko RV, Bharti V, Ouyang Z, Blevins A, Mont S, Saleh N, Lawrence HA, Shen C, Chen SC, Ayers GD, DeNardo DG, Arteaga C, Richmond A, Vilgelm AE. Metabolic modulation by CDK4/6 inhibitor promotes chemokine-mediated recruitment of T cells into mammary tumors. Cell Rep. 2021 Apr 06;35(1):108944. doi: 10.1016/j.celrep.2021.108944. PMID: 33826903; PMCID: PMC8383195.
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Pik3c3 deficiency in myeloid cells imparts partial resistance to experimental autoimmune encephalomyelitis associated with reduced IL-1β production.

Cellular & molecular immunology

Yang G, Song W, Xu J, Postoak JL, Cheng F, Martinez J, Zhang J, Wu L, Van Kaer L.
PMID: 33235386
Cell Mol Immunol. 2021 Aug;18(8):2024-2039. doi: 10.1038/s41423-020-00589-1. Epub 2020 Nov 24.

The PIK3C3/VPS34 subunit of the class III phosphatidylinositol 3-kinase (PtdIns3K) complex plays a role in both canonical and noncanonical autophagy, key processes that control immune-cell responsiveness to a variety of stimuli. Our previous studies found that PIK3C3 is a...

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Yang G, Song W, Xu J, et al. Pik3c3 deficiency in myeloid cells imparts partial resistance to experimental autoimmune encephalomyelitis associated with reduced IL-1β production. Cell Mol Immunol. 2020;18(8):2024-2039doi: 10.1038/s41423-020-00589-1.
Yang, G., Song, W., Xu, J., Postoak, J. L., Cheng, F., Martinez, J., Zhang, J., Wu, L., & Van Kaer, L. (2021). Pik3c3 deficiency in myeloid cells imparts partial resistance to experimental autoimmune encephalomyelitis associated with reduced IL-1β production. Cellular & molecular immunology, 18(8), 2024-2039. https://doi.org/10.1038/s41423-020-00589-1
Yang, Guan, et al. "Pik3c3 deficiency in myeloid cells imparts partial resistance to experimental autoimmune encephalomyelitis associated with reduced IL-1β production." Cellular & molecular immunology vol. 18,8 (2021): 2024-2039. doi: https://doi.org/10.1038/s41423-020-00589-1
Yang G, Song W, Xu J, Postoak JL, Cheng F, Martinez J, Zhang J, Wu L, Van Kaer L. Pik3c3 deficiency in myeloid cells imparts partial resistance to experimental autoimmune encephalomyelitis associated with reduced IL-1β production. Cell Mol Immunol. 2021 Aug;18(8):2024-2039. doi: 10.1038/s41423-020-00589-1. Epub 2020 Nov 24. PMID: 33235386; PMCID: PMC8322046.
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Synthesis of tetranor-PGE.

Tetrahedron letters

Kimbrough JR, Jana S, Kim K, Allweil A, Oates JA, Milne GL, Sulikowski GA.
PMID: 32523235
Tetrahedron Lett. 2020 May 28;61(22). doi: 10.1016/j.tetlet.2020.151922. Epub 2020 Apr 10.

Prostaglandin E

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Kimbrough JR, Jana S, Kim K, et al. Synthesis of tetranor-PGE. Tetrahedron Lett. 2020;61(22)doi: 10.1016/j.tetlet.2020.151922.
Kimbrough, J. R., Jana, S., Kim, K., Allweil, A., Oates, J. A., Milne, G. L., & Sulikowski, G. A. (2020). Synthesis of tetranor-PGE. Tetrahedron letters, 61(22), . https://doi.org/10.1016/j.tetlet.2020.151922
Kimbrough, Jennifer R, et al. "Synthesis of tetranor-PGE." Tetrahedron letters vol. 61,22 (2020). doi: https://doi.org/10.1016/j.tetlet.2020.151922
Kimbrough JR, Jana S, Kim K, Allweil A, Oates JA, Milne GL, Sulikowski GA. Synthesis of tetranor-PGE. Tetrahedron Lett. 2020 May 28;61(22). doi: 10.1016/j.tetlet.2020.151922. Epub 2020 Apr 10. PMID: 32523235; PMCID: PMC7286543.
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Unsupervised machine learning reveals key immune cell subsets in COVID-19, rhinovirus infection, and cancer therapy.

bioRxiv : the preprint server for biology

Barone SM, Paul AGA, Muehling LM, Lannigan JA, Kwok WW, Turner RB, Woodfolk JA, Irish JM.
PMID: 32766581
bioRxiv. 2020 Nov 04; doi: 10.1101/2020.07.31.190454.

For an emerging disease like COVID-19, systems immunology tools may quickly identify and quantitatively characterize cells associated with disease progression or clinical response. With repeated sampling, immune monitoring creates a real-time portrait of the cells reacting to a novel...

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Barone SM, Paul AGA, Muehling LM, et al. Unsupervised machine learning reveals key immune cell subsets in COVID-19, rhinovirus infection, and cancer therapy. bioRxiv. 2020;doi: 10.1101/2020.07.31.190454.
Barone, S. M., Paul, A. G. A., Muehling, L. M., Lannigan, J. A., Kwok, W. W., Turner, R. B., Woodfolk, J. A., & Irish, J. M. (2020). Unsupervised machine learning reveals key immune cell subsets in COVID-19, rhinovirus infection, and cancer therapy. bioRxiv : the preprint server for biology, . https://doi.org/10.1101/2020.07.31.190454
Barone, Sierra M, et al. "Unsupervised machine learning reveals key immune cell subsets in COVID-19, rhinovirus infection, and cancer therapy." bioRxiv : the preprint server for biology vol. (2020). doi: https://doi.org/10.1101/2020.07.31.190454
Barone SM, Paul AGA, Muehling LM, Lannigan JA, Kwok WW, Turner RB, Woodfolk JA, Irish JM. Unsupervised machine learning reveals key immune cell subsets in COVID-19, rhinovirus infection, and cancer therapy. bioRxiv. 2020 Nov 04; doi: 10.1101/2020.07.31.190454. PMID: 32766581; PMCID: PMC7402038.
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Functional Properties of Oligomeric and Monomeric Forms of Helicobacter pylori VacA Toxin.

Infection and immunity

Caso GC, McClain MS, Erwin AL, Truelock MD, Campbell AM, Leasure CS, Nagel M, Schey KL, Lacy DB, Ohi MD, Cover TL.
PMID: 34543122
Infect Immun. 2021 Nov 16;89(12):e0034821. doi: 10.1128/IAI.00348-21. Epub 2021 Sep 20.

Helicobacter pylori VacA is a secreted toxin that assembles into water-soluble oligomeric structures and forms anion-selective membrane channels. Acidification of purified VacA enhances its activity in cell culture assays. Sites of protomer-protomer contact within VacA oligomers have been identified...

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Caso GC, McClain MS, Erwin AL, et al. Functional Properties of Oligomeric and Monomeric Forms of Helicobacter pylori VacA Toxin. Infect Immun. 2021;89(12):e0034821doi: 10.1128/IAI.00348-21.
Caso, G. C., McClain, M. S., Erwin, A. L., Truelock, M. D., Campbell, A. M., Leasure, C. S., Nagel, M., Schey, K. L., Lacy, D. B., Ohi, M. D., & Cover, T. L. (2021). Functional Properties of Oligomeric and Monomeric Forms of Helicobacter pylori VacA Toxin. Infection and immunity, 89(12), e0034821. https://doi.org/10.1128/IAI.00348-21
Caso, Georgia C, et al. "Functional Properties of Oligomeric and Monomeric Forms of Helicobacter pylori VacA Toxin." Infection and immunity vol. 89,12 (2021): e0034821. doi: https://doi.org/10.1128/IAI.00348-21
Caso GC, McClain MS, Erwin AL, Truelock MD, Campbell AM, Leasure CS, Nagel M, Schey KL, Lacy DB, Ohi MD, Cover TL. Functional Properties of Oligomeric and Monomeric Forms of Helicobacter pylori VacA Toxin. Infect Immun. 2021 Nov 16;89(12):e0034821. doi: 10.1128/IAI.00348-21. Epub 2021 Sep 20. PMID: 34543122; PMCID: PMC8594603.
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Lung Cancer and Severe Acute Respiratory Syndrome Coronavirus 2 Infection: Identifying Important Knowledge Gaps for Investigation.

Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer

Rolfo C, Meshulami N, Russo A, Krammer F, García-Sastre A, Mack PC, Gomez JE, Bhardwaj N, Benyounes A, Sirera R, Moore A, Rohs N, Henschke CI, Yankelevitz D, King J, Shyr Y, Bunn PA, Minna JD, Hirsch FR.
PMID: 34774792
J Thorac Oncol. 2021 Nov 10; doi: 10.1016/j.jtho.2021.11.001. Epub 2021 Nov 10.

Patients with lung cancer are especially vulnerable to coronavirus disease 2019 (COVID-19) with a greater than sevenfold higher rate of becoming infected with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) COVID-19, a greater than threefold higher hospitalization rate with...

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Rolfo C, Meshulami N, Russo A, et al. Lung Cancer and Severe Acute Respiratory Syndrome Coronavirus 2 Infection: Identifying Important Knowledge Gaps for Investigation. J Thorac Oncol. 2021;doi: 10.1016/j.jtho.2021.11.001.
Rolfo, C., Meshulami, N., Russo, A., Krammer, F., García-Sastre, A., Mack, P. C., Gomez, J. E., Bhardwaj, N., Benyounes, A., Sirera, R., Moore, A., Rohs, N., Henschke, C. I., Yankelevitz, D., King, J., Shyr, Y., Bunn, P. A., Minna, J. D., & Hirsch, F. R. (2021). Lung Cancer and Severe Acute Respiratory Syndrome Coronavirus 2 Infection: Identifying Important Knowledge Gaps for Investigation. Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer, . https://doi.org/10.1016/j.jtho.2021.11.001
Rolfo, Christian, et al. "Lung Cancer and Severe Acute Respiratory Syndrome Coronavirus 2 Infection: Identifying Important Knowledge Gaps for Investigation." Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer vol. (2021). doi: https://doi.org/10.1016/j.jtho.2021.11.001
Rolfo C, Meshulami N, Russo A, Krammer F, García-Sastre A, Mack PC, Gomez JE, Bhardwaj N, Benyounes A, Sirera R, Moore A, Rohs N, Henschke CI, Yankelevitz D, King J, Shyr Y, Bunn PA, Minna JD, Hirsch FR. Lung Cancer and Severe Acute Respiratory Syndrome Coronavirus 2 Infection: Identifying Important Knowledge Gaps for Investigation. J Thorac Oncol. 2021 Nov 10; doi: 10.1016/j.jtho.2021.11.001. Epub 2021 Nov 10. PMID: 34774792; PMCID: PMC8579698.
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Nuclear FGFR1 Regulates Gene Transcription and Promotes Antiestrogen Resistance in ER.

Clinical cancer research : an official journal of the American Association for Cancer Research

Servetto A, Kollipara R, Formisano L, Lin CC, Lee KM, Sudhan DR, Gonzalez-Ericsson PI, Chatterjee S, Guerrero-Zotano A, Mendiratta S, Akamatsu H, James N, Bianco R, Hanker AB, Kittler R, Arteaga CL.
PMID: 34011560
Clin Cancer Res. 2021 Aug 01;27(15):4379-4396. doi: 10.1158/1078-0432.CCR-20-3905. Epub 2021 May 19.

PURPOSE: FGFR1 overexpression has been associated with endocrine resistance in EREXPERIMENTAL DESIGN: Tumors from patients treated with letrozole were subjected to Ki67 and FGFR1 IHC. MCF7 cells were transduced with FGFR1(SP-)(NLS) to promote nuclear FGFR1 overexpression. FGFR1 genomic activity...

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Servetto A, Kollipara R, Formisano L, et al. Nuclear FGFR1 Regulates Gene Transcription and Promotes Antiestrogen Resistance in ER. Clin Cancer Res. 2021;27(15):4379-4396doi: 10.1158/1078-0432.CCR-20-3905.
Servetto, A., Kollipara, R., Formisano, L., Lin, C. C., Lee, K. M., Sudhan, D. R., Gonzalez-Ericsson, P. I., Chatterjee, S., Guerrero-Zotano, A., Mendiratta, S., Akamatsu, H., James, N., Bianco, R., Hanker, A. B., Kittler, R., & Arteaga, C. L. (2021). Nuclear FGFR1 Regulates Gene Transcription and Promotes Antiestrogen Resistance in ER. Clinical cancer research : an official journal of the American Association for Cancer Research, 27(15), 4379-4396. https://doi.org/10.1158/1078-0432.CCR-20-3905
Servetto, Alberto, et al. "Nuclear FGFR1 Regulates Gene Transcription and Promotes Antiestrogen Resistance in ER." Clinical cancer research : an official journal of the American Association for Cancer Research vol. 27,15 (2021): 4379-4396. doi: https://doi.org/10.1158/1078-0432.CCR-20-3905
Servetto A, Kollipara R, Formisano L, Lin CC, Lee KM, Sudhan DR, Gonzalez-Ericsson PI, Chatterjee S, Guerrero-Zotano A, Mendiratta S, Akamatsu H, James N, Bianco R, Hanker AB, Kittler R, Arteaga CL. Nuclear FGFR1 Regulates Gene Transcription and Promotes Antiestrogen Resistance in ER. Clin Cancer Res. 2021 Aug 01;27(15):4379-4396. doi: 10.1158/1078-0432.CCR-20-3905. Epub 2021 May 19. PMID: 34011560; PMCID: PMC8338892.
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