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Aromatic L-Amino acid decarboxylase deficiency: A new case from Turkey with a novel mutation.

Annals of Indian Academy of Neurology

Gücüyener K, Kasapkara CS, Tümer L, Verbeek MM.
PMID: 25024584
Ann Indian Acad Neurol. 2014 Apr;17(2):234-6. doi: 10.4103/0972-2327.132652.

Aromatic L-amino acid decarboxylase (AADC), a vitamin B6-requiring enzyme that converts L-dopa to dopamine and 5-hydroxytryptophan to serotonin. Deficiency of this enzyme results in developmental delay, muscular hypotonia, dystonia, involuntary movements, autonomic dysfunction, and oculogyric crises. We now report...

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Gücüyener K, Kasapkara CS, Tümer L, et al. Aromatic L-Amino acid decarboxylase deficiency: A new case from Turkey with a novel mutation. Ann Indian Acad Neurol. 2014;17(2):234-6doi: 10.4103/0972-2327.132652.
Gücüyener, K., Kasapkara, C. S., Tümer, L., & Verbeek, M. M. (2014). Aromatic L-Amino acid decarboxylase deficiency: A new case from Turkey with a novel mutation. Annals of Indian Academy of Neurology, 17(2), 234-6. https://doi.org/10.4103/0972-2327.132652
Gücüyener, Kıvılcım, et al. "Aromatic L-Amino acid decarboxylase deficiency: A new case from Turkey with a novel mutation." Annals of Indian Academy of Neurology vol. 17,2 (2014): 234-6. doi: https://doi.org/10.4103/0972-2327.132652
Gücüyener K, Kasapkara CS, Tümer L, Verbeek MM. Aromatic L-Amino acid decarboxylase deficiency: A new case from Turkey with a novel mutation. Ann Indian Acad Neurol. 2014 Apr;17(2):234-6. doi: 10.4103/0972-2327.132652. PMID: 25024584; PMCID: PMC4090859.
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Aripiprazole for Treatment of Apathy.

Innovations in clinical neuroscience

Monga V, Padala PR.
PMID: 26634180
Innov Clin Neurosci. 2015 Sep-Oct;12(9):33-6.

OBJECTIVE: The objective of this article is to present a case of improved outcome of apathy syndrome with aripiprazole.CASE SUMMARY: A 42-year-old man with depression and seizure disorder had significant apathy that did not respond to carbamazepine, sertraline, and...

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Monga V, Padala PR. Aripiprazole for Treatment of Apathy. Innov Clin Neurosci. 2015;12(9):33-6
Monga, V., & Padala, P. R. (2015). Aripiprazole for Treatment of Apathy. Innovations in clinical neuroscience, 12(9), 33-6.
Monga, Varun, and Padala, Prasad R. "Aripiprazole for Treatment of Apathy." Innovations in clinical neuroscience vol. 12,9 (2015): 33-6.
Monga V, Padala PR. Aripiprazole for Treatment of Apathy. Innov Clin Neurosci. 2015 Sep-Oct;12(9):33-6. PMID: 26634180; PMCID: PMC4655898.
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The Unexpected Effects of Beneficial and Adverse Social Experiences during Adolescence on Anxiety and Aggression and Their Modulation by Genotype.

Frontiers in behavioral neuroscience

Meyer N, Richter SH, Schreiber RS, Kloke V, Kaiser S, Lesch KP, Sachser N.
PMID: 27303275
Front Behav Neurosci. 2016 May 26;10:97. doi: 10.3389/fnbeh.2016.00097. eCollection 2016.

Anxiety and aggression are part of the behavioral repertoire of humans and animals. However, in their exaggerated form both can become maladaptive and result in psychiatric disorders. On the one hand, genetic predisposition has been shown to play a...

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Meyer N, Richter SH, Schreiber RS, et al. The Unexpected Effects of Beneficial and Adverse Social Experiences during Adolescence on Anxiety and Aggression and Their Modulation by Genotype. Front Behav Neurosci. 2016;10:97doi: 10.3389/fnbeh.2016.00097.
Meyer, N., Richter, S. H., Schreiber, R. S., Kloke, V., Kaiser, S., Lesch, K. P., & Sachser, N. (2016). The Unexpected Effects of Beneficial and Adverse Social Experiences during Adolescence on Anxiety and Aggression and Their Modulation by Genotype. Frontiers in behavioral neuroscience, 1097. https://doi.org/10.3389/fnbeh.2016.00097
Meyer, Neele, et al. "The Unexpected Effects of Beneficial and Adverse Social Experiences during Adolescence on Anxiety and Aggression and Their Modulation by Genotype." Frontiers in behavioral neuroscience vol. 10 (2016): 97. doi: https://doi.org/10.3389/fnbeh.2016.00097
Meyer N, Richter SH, Schreiber RS, Kloke V, Kaiser S, Lesch KP, Sachser N. The Unexpected Effects of Beneficial and Adverse Social Experiences during Adolescence on Anxiety and Aggression and Their Modulation by Genotype. Front Behav Neurosci. 2016 May 26;10:97. doi: 10.3389/fnbeh.2016.00097. eCollection 2016. PMID: 27303275; PMCID: PMC4880570.
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Overactive Bladder Successfully Treated with Duloxetine in a Female Adolescent.

Clinical psychopharmacology and neuroscience : the official scientific journal of the Korean College of Neuropsychopharmacology

Wang SM, Lee HK, Kweon YS, Lee CT, Lee KU.
PMID: 26243851
Clin Psychopharmacol Neurosci. 2015 Aug 31;13(2):212-4. doi: 10.9758/cpn.2015.13.2.212.

Overactive bladder (OAB) is defined as urgency, usually with frequency and nocturia, and with or without urge incontinence. Duloxetine, an antidepressant that inhibits reuptake of serotonin and norepinephrine, is indicated for the treatment of stress urinary incontinence in Europe....

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Wang SM, Lee HK, Kweon YS, et al. Overactive Bladder Successfully Treated with Duloxetine in a Female Adolescent. Clin Psychopharmacol Neurosci. 2015;13(2):212-4doi: 10.9758/cpn.2015.13.2.212.
Wang, S. M., Lee, H. K., Kweon, Y. S., Lee, C. T., & Lee, K. U. (2015). Overactive Bladder Successfully Treated with Duloxetine in a Female Adolescent. Clinical psychopharmacology and neuroscience : the official scientific journal of the Korean College of Neuropsychopharmacology, 13(2), 212-4. https://doi.org/10.9758/cpn.2015.13.2.212
Wang, Sheng-Min, et al. "Overactive Bladder Successfully Treated with Duloxetine in a Female Adolescent." Clinical psychopharmacology and neuroscience : the official scientific journal of the Korean College of Neuropsychopharmacology vol. 13,2 (2015): 212-4. doi: https://doi.org/10.9758/cpn.2015.13.2.212
Wang SM, Lee HK, Kweon YS, Lee CT, Lee KU. Overactive Bladder Successfully Treated with Duloxetine in a Female Adolescent. Clin Psychopharmacol Neurosci. 2015 Aug 31;13(2):212-4. doi: 10.9758/cpn.2015.13.2.212. PMID: 26243851; PMCID: PMC4540032.
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Inhibition of Cytochrome P450 by Propolis in Human Liver Microsomes.

Toxicological research

Ryu CS, Oh SJ, Oh JM, Lee JY, Lee SY, Chae JW, Kwon KI, Kim SK.
PMID: 27437087
Toxicol Res. 2016 Jul;32(3):207-13. doi: 10.5487/TR.2016.32.3.207. Epub 2016 Jul 30.

Although propolis is one of the most popular functional foods for human health, there have been no comprehensive studies of herb-drug interactions through cytochrome P450 (CYP) inhibition. The purpose of this study was to determine the inhibitory effects of...

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Ryu CS, Oh SJ, Oh JM, et al. Inhibition of Cytochrome P450 by Propolis in Human Liver Microsomes. Toxicol Res. 2016;32(3):207-13doi: 10.5487/TR.2016.32.3.207.
Ryu, C. S., Oh, S. J., Oh, J. M., Lee, J. Y., Lee, S. Y., Chae, J. W., Kwon, K. I., & Kim, S. K. (2016). Inhibition of Cytochrome P450 by Propolis in Human Liver Microsomes. Toxicological research, 32(3), 207-13. https://doi.org/10.5487/TR.2016.32.3.207
Ryu, Chang Seon, et al. "Inhibition of Cytochrome P450 by Propolis in Human Liver Microsomes." Toxicological research vol. 32,3 (2016): 207-13. doi: https://doi.org/10.5487/TR.2016.32.3.207
Ryu CS, Oh SJ, Oh JM, Lee JY, Lee SY, Chae JW, Kwon KI, Kim SK. Inhibition of Cytochrome P450 by Propolis in Human Liver Microsomes. Toxicol Res. 2016 Jul;32(3):207-13. doi: 10.5487/TR.2016.32.3.207. Epub 2016 Jul 30. PMID: 27437087; PMCID: PMC4946414.
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Dendritic Spine Density of Pyramidal Neurons in Field CA1 of the Hippocampus Decreases due to Chronic Tryptophan Restriction.

Nutritional neuroscience

Pérez-Vega MI, Barajas-López G, Del Angel-Meza AR, González-Burgos I, Feria-Velasco A.
PMID: 27406202
Nutr Neurosci. 1998;1(3):237-42. doi: 10.1080/1028415X.1998.11747233.

The CA1 hippocampal region is involved in organizing several neuropsychological processes. Pyramidal cell dendritic spines in the CA1 field of rats subjected to chronic tryptophan diet restriction were quantified at 21, 40, and 60 days of age. At 40...

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Pérez-Vega MI, Barajas-López G, Del Angel-Meza AR, et al. Dendritic Spine Density of Pyramidal Neurons in Field CA1 of the Hippocampus Decreases due to Chronic Tryptophan Restriction. Nutr Neurosci. 1998;1(3):237-42doi: 10.1080/1028415X.1998.11747233.
Pérez-Vega, M. I., Barajas-López, G., Del Angel-Meza, A. R., González-Burgos, I., & Feria-Velasco, A. (1998). Dendritic Spine Density of Pyramidal Neurons in Field CA1 of the Hippocampus Decreases due to Chronic Tryptophan Restriction. Nutritional neuroscience, 1(3), 237-42. https://doi.org/10.1080/1028415X.1998.11747233
Pérez-Vega, M I, et al. "Dendritic Spine Density of Pyramidal Neurons in Field CA1 of the Hippocampus Decreases due to Chronic Tryptophan Restriction." Nutritional neuroscience vol. 1,3 (1998): 237-42. doi: https://doi.org/10.1080/1028415X.1998.11747233
Pérez-Vega MI, Barajas-López G, Del Angel-Meza AR, González-Burgos I, Feria-Velasco A. Dendritic Spine Density of Pyramidal Neurons in Field CA1 of the Hippocampus Decreases due to Chronic Tryptophan Restriction. Nutr Neurosci. 1998;1(3):237-42. doi: 10.1080/1028415X.1998.11747233. PMID: 27406202.
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Development of Non-C2-symmetric ProPhenol Ligands. The Asymmetric Vinylation of N-Boc Imines.

Organic letters

Trost BM, Hung CI, Koester DC, Miller Y.
PMID: 26200769
Org Lett. 2015 Aug 07;17(15):3778-81. doi: 10.1021/acs.orglett.5b01755. Epub 2015 Jul 22.

The development and application of a new generation of non-C2-symmetric ProPhenol ligands is reported herein. Rational design of the ProPhenol ligand paved the way to the first catalytic and asymmetric vinylation of N-Boc imines via hydrozirconation giving rise to...

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Trost BM, Hung CI, Koester DC, et al. Development of Non-C2-symmetric ProPhenol Ligands. The Asymmetric Vinylation of N-Boc Imines. Org Lett. 2015;17(15):3778-81doi: 10.1021/acs.orglett.5b01755.
Trost, B. M., Hung, C. I., Koester, D. C., & Miller, Y. (2015). Development of Non-C2-symmetric ProPhenol Ligands. The Asymmetric Vinylation of N-Boc Imines. Organic letters, 17(15), 3778-81. https://doi.org/10.1021/acs.orglett.5b01755
Trost, Barry M, et al. "Development of Non-C2-symmetric ProPhenol Ligands. The Asymmetric Vinylation of N-Boc Imines." Organic letters vol. 17,15 (2015): 3778-81. doi: https://doi.org/10.1021/acs.orglett.5b01755
Trost BM, Hung CI, Koester DC, Miller Y. Development of Non-C2-symmetric ProPhenol Ligands. The Asymmetric Vinylation of N-Boc Imines. Org Lett. 2015 Aug 07;17(15):3778-81. doi: 10.1021/acs.orglett.5b01755. Epub 2015 Jul 22. PMID: 26200769.
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Acupuncture for visceral pain: neural substrates and potential mechanisms.

Evidence-based complementary and alternative medicine : eCAM

Chen S, Wang S, Rong P, Wang J, Qiao L, Feng X, Liu J, Zhang J.
PMID: 25614752
Evid Based Complement Alternat Med. 2014;2014:609594. doi: 10.1155/2014/609594. Epub 2014 Dec 29.

Visceral pain is the most common form of pain caused by varied diseases and a major reason for patients to seek medical consultation. Despite much advances, the pathophysiological mechanism is still poorly understood comparing with its somatic counterpart and,...

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Chen S, Wang S, Rong P, et al. Acupuncture for visceral pain: neural substrates and potential mechanisms. Evid Based Complement Alternat Med. 2014;2014:609594doi: 10.1155/2014/609594.
Chen, S., Wang, S., Rong, P., Wang, J., Qiao, L., Feng, X., Liu, J., & Zhang, J. (2014). Acupuncture for visceral pain: neural substrates and potential mechanisms. Evidence-based complementary and alternative medicine : eCAM, 2014609594. https://doi.org/10.1155/2014/609594
Chen, Shuping, et al. "Acupuncture for visceral pain: neural substrates and potential mechanisms." Evidence-based complementary and alternative medicine : eCAM vol. 2014 (2014): 609594. doi: https://doi.org/10.1155/2014/609594
Chen S, Wang S, Rong P, Wang J, Qiao L, Feng X, Liu J, Zhang J. Acupuncture for visceral pain: neural substrates and potential mechanisms. Evid Based Complement Alternat Med. 2014;2014:609594. doi: 10.1155/2014/609594. Epub 2014 Dec 29. PMID: 25614752; PMCID: PMC4295157.
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Chronic Diarrhea in L-Amino Acid Decarboxylase (AADC) Deficiency: A Prominent Clinical Finding Among a Series of Ten French Patients.

JIMD reports

Spitz MA, Nguyen MA, Roche S, Heron B, Milh M, de Lonlay P, Lion-François L, Testard H, Napuri S, Barth M, Fournier-Favre S, Christa L, Vianey-Saban C, Corne C, Roubertie A.
PMID: 27147232
JIMD Rep. 2017;31:85-93. doi: 10.1007/8904_2016_550. Epub 2016 May 05.

Aromatic L-amino acid decarboxylase (AADC) deficiency is an autosomal recessive inborn error of metabolism, affecting catecholamines and serotonin biosynthesis. Cardinal signs consist in psychomotor delay, hypotonia, oculogyric crises, dystonia, and extraneurological symptoms.PATIENTS AND METHODS: We present a retrospective descriptive...

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Spitz MA, Nguyen MA, Roche S, et al. Chronic Diarrhea in L-Amino Acid Decarboxylase (AADC) Deficiency: A Prominent Clinical Finding Among a Series of Ten French Patients. JIMD Rep. 2016;31:85-93doi: 10.1007/8904_2016_550.
Spitz, M. A., Nguyen, M. A., Roche, S., Heron, B., Milh, M., de Lonlay, P., Lion-François, L., Testard, H., Napuri, S., Barth, M., Fournier-Favre, S., Christa, L., Vianey-Saban, C., Corne, C., & Roubertie, A. (2017). Chronic Diarrhea in L-Amino Acid Decarboxylase (AADC) Deficiency: A Prominent Clinical Finding Among a Series of Ten French Patients. JIMD reports, 3185-93. https://doi.org/10.1007/8904_2016_550
Spitz, M A, et al. "Chronic Diarrhea in L-Amino Acid Decarboxylase (AADC) Deficiency: A Prominent Clinical Finding Among a Series of Ten French Patients." JIMD reports vol. 31 (2017): 85-93. doi: https://doi.org/10.1007/8904_2016_550
Spitz MA, Nguyen MA, Roche S, Heron B, Milh M, de Lonlay P, Lion-François L, Testard H, Napuri S, Barth M, Fournier-Favre S, Christa L, Vianey-Saban C, Corne C, Roubertie A. Chronic Diarrhea in L-Amino Acid Decarboxylase (AADC) Deficiency: A Prominent Clinical Finding Among a Series of Ten French Patients. JIMD Rep. 2017;31:85-93. doi: 10.1007/8904_2016_550. Epub 2016 May 05. PMID: 27147232; PMCID: PMC5272843.
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Impact of pretreatment with antidepressants on the efficacy of duloxetine in terms of mood symptoms and functioning: an analysis of 15 pooled major depressive disorder studies.

The primary care companion for CNS disorders

Barros BR, Schacht A, Happich M, Televantou F, Berggren L, Walker DJ, Dueñas HJ.
PMID: 25667808
Prim Care Companion CNS Disord. 2014 Oct 02;16(5). doi: 10.4088/PCC.14m01661. eCollection 2014.

OBJECTIVE: This post hoc analysis aimed to determine whether patients with major depressive disorder (MDD) in duloxetine trials who were antidepressant naive or who were previously exposed to antidepressants exhibited differences in efficacy and functioning.METHOD: Data were pooled from...

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Barros BR, Schacht A, Happich M, et al. Impact of pretreatment with antidepressants on the efficacy of duloxetine in terms of mood symptoms and functioning: an analysis of 15 pooled major depressive disorder studies. Prim Care Companion CNS Disord. 2014;16(5)doi: 10.4088/PCC.14m01661.
Barros, B. R., Schacht, A., Happich, M., Televantou, F., Berggren, L., Walker, D. J., & Dueñas, H. J. (2014). Impact of pretreatment with antidepressants on the efficacy of duloxetine in terms of mood symptoms and functioning: an analysis of 15 pooled major depressive disorder studies. The primary care companion for CNS disorders, 16(5), . https://doi.org/10.4088/PCC.14m01661
Barros, Bruno R, et al. "Impact of pretreatment with antidepressants on the efficacy of duloxetine in terms of mood symptoms and functioning: an analysis of 15 pooled major depressive disorder studies." The primary care companion for CNS disorders vol. 16,5 (2014). doi: https://doi.org/10.4088/PCC.14m01661
Barros BR, Schacht A, Happich M, Televantou F, Berggren L, Walker DJ, Dueñas HJ. Impact of pretreatment with antidepressants on the efficacy of duloxetine in terms of mood symptoms and functioning: an analysis of 15 pooled major depressive disorder studies. Prim Care Companion CNS Disord. 2014 Oct 02;16(5). doi: 10.4088/PCC.14m01661. eCollection 2014. PMID: 25667808; PMCID: PMC4321013.
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Evaluation in monkey of two candidate PET radioligands, [.

Synapse (New York, N.Y.)

Lohith TG, Xu R, Tsujikawa T, Morse CL, Anderson KB, Gladding RL, Zoghbi SS, Fujita M, Innis RB, Pike VW.
PMID: 25088028
Synapse. 2014 Dec;68(12):613-623. doi: 10.1002/syn.21773. Epub 2014 Aug 11.

The serotonin subtype-4 (5-HT

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Lohith TG, Xu R, Tsujikawa T, et al. Evaluation in monkey of two candidate PET radioligands, [. Synapse. 2014;68(12):613-623doi: 10.1002/syn.21773.
Lohith, T. G., Xu, R., Tsujikawa, T., Morse, C. L., Anderson, K. B., Gladding, R. L., Zoghbi, S. S., Fujita, M., Innis, R. B., & Pike, V. W. (2014). Evaluation in monkey of two candidate PET radioligands, [. Synapse (New York, N.Y.), 68(12), 613-623. https://doi.org/10.1002/syn.21773
Lohith, Talakad G, et al. "Evaluation in monkey of two candidate PET radioligands, [." Synapse (New York, N.Y.) vol. 68,12 (2014): 613-623. doi: https://doi.org/10.1002/syn.21773
Lohith TG, Xu R, Tsujikawa T, Morse CL, Anderson KB, Gladding RL, Zoghbi SS, Fujita M, Innis RB, Pike VW. Evaluation in monkey of two candidate PET radioligands, [. Synapse. 2014 Dec;68(12):613-623. doi: 10.1002/syn.21773. Epub 2014 Aug 11. PMID: 25088028; PMCID: PMC4317381.
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Association of citalopram with congenital anomalies: A meta-analysis.

Obstetrics & gynecology science

Kang HH, Ahn KH, Hong SC, Kwon BY, Lee EH, Lee JS, Oh MJ, Kim HJ.
PMID: 28344955
Obstet Gynecol Sci. 2017 Mar;60(2):145-153. doi: 10.5468/ogs.2017.60.2.145. Epub 2017 Mar 16.

OBJECTIVE: The antenatal use of citalopram, a widely prescribed selective serotonin reuptake inhibitor, has been suspected to be associated with congenital, particularly cardiac, anomalies. This study aimed to prove the association between citalopram use and congenital anomalies.METHODS: We searched...

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Kang HH, Ahn KH, Hong SC, et al. Association of citalopram with congenital anomalies: A meta-analysis. Obstet Gynecol Sci. 2017;60(2):145-153doi: 10.5468/ogs.2017.60.2.145.
Kang, H. H., Ahn, K. H., Hong, S. C., Kwon, B. Y., Lee, E. H., Lee, J. S., Oh, M. J., & Kim, H. J. (2017). Association of citalopram with congenital anomalies: A meta-analysis. Obstetrics & gynecology science, 60(2), 145-153. https://doi.org/10.5468/ogs.2017.60.2.145
Kang, Hyun-Hye, et al. "Association of citalopram with congenital anomalies: A meta-analysis." Obstetrics & gynecology science vol. 60,2 (2017): 145-153. doi: https://doi.org/10.5468/ogs.2017.60.2.145
Kang HH, Ahn KH, Hong SC, Kwon BY, Lee EH, Lee JS, Oh MJ, Kim HJ. Association of citalopram with congenital anomalies: A meta-analysis. Obstet Gynecol Sci. 2017 Mar;60(2):145-153. doi: 10.5468/ogs.2017.60.2.145. Epub 2017 Mar 16. PMID: 28344955; PMCID: PMC5364096.
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Showing 25 to 36 of 7558 entries