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Bindu S, Pal C, Dey S, et al. Translocation of heme oxygenase-1 to mitochondria is a novel cytoprotective mechanism against non-steroidal anti-inflammatory drug-induced mitochondrial oxidative stress, apoptosis, and gastric mucosal injury. J Biol Chem. 2015;290(22):13667-8doi: 10.1074/jbc.A111.279893.
Bindu, S., Pal, C., Dey, S., Goyal, M., Alam, A., Iqbal, M. S., Dutta, S., Sarkar, S., Kumar, R., Maity, P., & Bandyopadhyay, U. (2015). Translocation of heme oxygenase-1 to mitochondria is a novel cytoprotective mechanism against non-steroidal anti-inflammatory drug-induced mitochondrial oxidative stress, apoptosis, and gastric mucosal injury. The Journal of biological chemistry, 290(22), 13667-8. https://doi.org/10.1074/jbc.A111.279893
Bindu, Samik, et al. "Translocation of heme oxygenase-1 to mitochondria is a novel cytoprotective mechanism against non-steroidal anti-inflammatory drug-induced mitochondrial oxidative stress, apoptosis, and gastric mucosal injury." The Journal of biological chemistry vol. 290,22 (2015): 13667-8. doi: https://doi.org/10.1074/jbc.A111.279893
Bindu S, Pal C, Dey S, Goyal M, Alam A, Iqbal MS, Dutta S, Sarkar S, Kumar R, Maity P, Bandyopadhyay U. Translocation of heme oxygenase-1 to mitochondria is a novel cytoprotective mechanism against non-steroidal anti-inflammatory drug-induced mitochondrial oxidative stress, apoptosis, and gastric mucosal injury. J Biol Chem. 2015 May 29;290(22):13667-8. doi: 10.1074/jbc.A111.279893. PMID: 26025999; PMCID: PMC4447946.
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