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Correction: An increase in visceral fat is associated with a decrease in the taste and olfactory capacity.

PloS one

Fernandez-Garcia JC, Alcaide J, Santiago-Fernandez C, Roca-Rodriguez MM, Aguera Z, Baños R, Botella C, de la Torre R, Fernandez-Real JM, Fruhbeck G, Gomez-Ambrosi J, Jimenez-Murcia S, Menchon JM, Casanueva FF, Fernandez-Aranda F, Tinahones FJ, Garrido-Sanchez L.
PMID: 28257495
PLoS One. 2017 Mar 03;12(3):e0173588. doi: 10.1371/journal.pone.0173588. eCollection 2017.

[This corrects the article DOI: 10.1371/journal.pone.0171204.].

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Fernandez-Garcia JC, Alcaide J, Santiago-Fernandez C, et al. Correction: An increase in visceral fat is associated with a decrease in the taste and olfactory capacity. PLoS One. 2017;12(3):e0173588doi: 10.1371/journal.pone.0173588.
Fernandez-Garcia, J. C., Alcaide, J., Santiago-Fernandez, C., Roca-Rodriguez, M. M., Aguera, Z., Baños, R., Botella, C., de la Torre, R., Fernandez-Real, J. M., Fruhbeck, G., Gomez-Ambrosi, J., Jimenez-Murcia, S., Menchon, J. M., Casanueva, F. F., Fernandez-Aranda, F., Tinahones, F. J., & Garrido-Sanchez, L. (2017). Correction: An increase in visceral fat is associated with a decrease in the taste and olfactory capacity. PloS one, 12(3), e0173588. https://doi.org/10.1371/journal.pone.0173588
Fernandez-Garcia, Jose Carlos, et al. "Correction: An increase in visceral fat is associated with a decrease in the taste and olfactory capacity." PloS one vol. 12,3 (2017): e0173588. doi: https://doi.org/10.1371/journal.pone.0173588
Fernandez-Garcia JC, Alcaide J, Santiago-Fernandez C, Roca-Rodriguez MM, Aguera Z, Baños R, Botella C, de la Torre R, Fernandez-Real JM, Fruhbeck G, Gomez-Ambrosi J, Jimenez-Murcia S, Menchon JM, Casanueva FF, Fernandez-Aranda F, Tinahones FJ, Garrido-Sanchez L. Correction: An increase in visceral fat is associated with a decrease in the taste and olfactory capacity. PLoS One. 2017 Mar 03;12(3):e0173588. doi: 10.1371/journal.pone.0173588. eCollection 2017. PMID: 28257495; PMCID: PMC5336289.
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Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin.

Genes & nutrition

Catalán V, Gómez-Ambrosi J, Rodríguez A, Ramírez B, Valentí V, Moncada R, Silva C, Salvador J, Frühbeck G.
PMID: 25869413
Genes Nutr. 2015 May;10(3):460. doi: 10.1007/s12263-015-0460-8. Epub 2015 Apr 14.

Inflammation is a critical contributor to the pathogenesis of metabolic disorders with adipose tissue being crucial in the inflammatory response by releasing multiple adipokines with either pro- or anti-inflammatory activities with potential functions as metabolic regulators. Peripheral blood mononuclear...

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Catalán V, Gómez-Ambrosi J, Rodríguez A, et al. Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin. Genes Nutr. 2015;10(3):460doi: 10.1007/s12263-015-0460-8.
Catalán, V., Gómez-Ambrosi, J., Rodríguez, A., Ramírez, B., Valentí, V., Moncada, R., Silva, C., Salvador, J., & Frühbeck, G. (2015). Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin. Genes & nutrition, 10(3), 460. https://doi.org/10.1007/s12263-015-0460-8
Catalán, Victoria, et al. "Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin." Genes & nutrition vol. 10,3 (2015): 460. doi: https://doi.org/10.1007/s12263-015-0460-8
Catalán V, Gómez-Ambrosi J, Rodríguez A, Ramírez B, Valentí V, Moncada R, Silva C, Salvador J, Frühbeck G. Peripheral mononuclear blood cells contribute to the obesity-associated inflammatory state independently of glycemic status: involvement of the novel proinflammatory adipokines chemerin, chitinase-3-like protein 1, lipocalin-2 and osteopontin. Genes Nutr. 2015 May;10(3):460. doi: 10.1007/s12263-015-0460-8. Epub 2015 Apr 14. PMID: 25869413; PMCID: PMC4395559.
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Insulin resistance: the metabolic syndrome X.

European journal of clinical nutrition

Fruhbeck G.
PMID: 10713753
Eur J Clin Nutr. 2000 Mar;54(3):272. doi: 10.1038/sj.ejcn.1600929.

No abstract available.

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Fruhbeck G. Insulin resistance: the metabolic syndrome X. Eur J Clin Nutr. 2000;54(3):272doi: 10.1038/sj.ejcn.1600929.
Fruhbeck, G. (2000). Insulin resistance: the metabolic syndrome X. European journal of clinical nutrition, 54(3), 272. https://doi.org/10.1038/sj.ejcn.1600929
Fruhbeck. "Insulin resistance: the metabolic syndrome X." European journal of clinical nutrition vol. 54,3 (2000): 272. doi: https://doi.org/10.1038/sj.ejcn.1600929
Fruhbeck G. Insulin resistance: the metabolic syndrome X. Eur J Clin Nutr. 2000 Mar;54(3):272. doi: 10.1038/sj.ejcn.1600929. PMID: 10713753.
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Circulating omentin concentration increases after weight loss.

Nutrition & metabolism

Moreno-Navarrete JM, Catalán V, Ortega F, Gómez-Ambrosi J, Ricart W, Frühbeck G, Fernández-Real JM.
PMID: 20380714
Nutr Metab (Lond). 2010 Apr 09;7:27. doi: 10.1186/1743-7075-7-27.

BACKGROUND: Omentin-1 is a novel adipokine expressed in visceral adipose tissue and negatively associated with insulin resistance and obesity. We aimed to study the effects of weight loss-induced improved insulin sensitivity on circulating omentin concentrations.METHODS: Circulating omentin-1 (ELISA) concentration...

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Moreno-Navarrete JM, Catalán V, Ortega F, et al. Circulating omentin concentration increases after weight loss. Nutr Metab (Lond). 2010;7:27doi: 10.1186/1743-7075-7-27.
Moreno-Navarrete, J. M., Catalán, V., Ortega, F., Gómez-Ambrosi, J., Ricart, W., Frühbeck, G., & Fernández-Real, J. M. (2010). Circulating omentin concentration increases after weight loss. Nutrition & metabolism, 727. https://doi.org/10.1186/1743-7075-7-27
Moreno-Navarrete, José María, et al. "Circulating omentin concentration increases after weight loss." Nutrition & metabolism vol. 7 (2010): 27. doi: https://doi.org/10.1186/1743-7075-7-27
Moreno-Navarrete JM, Catalán V, Ortega F, Gómez-Ambrosi J, Ricart W, Frühbeck G, Fernández-Real JM. Circulating omentin concentration increases after weight loss. Nutr Metab (Lond). 2010 Apr 09;7:27. doi: 10.1186/1743-7075-7-27. PMID: 20380714; PMCID: PMC2859768.
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IL-32α-induced inflammation constitutes a link between obesity and colon cancer.

Oncoimmunology

Catalán V, Gómez-Ambrosi J, Rodríguez A, Ramírez B, Ortega VA, Hernández-Lizoain JL, Baixauli J, Becerril S, Rotellar F, Valentí V, Moncada R, Silva C, Salvador J, Frühbeck G.
PMID: 28811968
Oncoimmunology. 2017 May 16;6(7):e1328338. doi: 10.1080/2162402X.2017.1328338. eCollection 2017.

Growing evidence indicates that adipose tissue inflammation is an important mechanism whereby obesity promotes cancer risk and progression. Since IL-32 is an important inflammatory and remodeling factor in obesity and is also related to colon cancer (CC) development, the...

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Catalán V, Gómez-Ambrosi J, Rodríguez A, et al. IL-32α-induced inflammation constitutes a link between obesity and colon cancer. Oncoimmunology. 2017;6(7):e1328338doi: 10.1080/2162402X.2017.1328338.
Catalán, V., Gómez-Ambrosi, J., Rodríguez, A., Ramírez, B., Ortega, V. A., Hernández-Lizoain, J. L., Baixauli, J., Becerril, S., Rotellar, F., Valentí, V., Moncada, R., Silva, C., Salvador, J., & Frühbeck, G. (2017). IL-32α-induced inflammation constitutes a link between obesity and colon cancer. Oncoimmunology, 6(7), e1328338. https://doi.org/10.1080/2162402X.2017.1328338
Catalán, Victoria, et al. "IL-32α-induced inflammation constitutes a link between obesity and colon cancer." Oncoimmunology vol. 6,7 (2017): e1328338. doi: https://doi.org/10.1080/2162402X.2017.1328338
Catalán V, Gómez-Ambrosi J, Rodríguez A, Ramírez B, Ortega VA, Hernández-Lizoain JL, Baixauli J, Becerril S, Rotellar F, Valentí V, Moncada R, Silva C, Salvador J, Frühbeck G. IL-32α-induced inflammation constitutes a link between obesity and colon cancer. Oncoimmunology. 2017 May 16;6(7):e1328338. doi: 10.1080/2162402X.2017.1328338. eCollection 2017. PMID: 28811968; PMCID: PMC5543901.
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Non-alcoholic fatty liver disease: A patient guideline.

JHEP reports : innovation in hepatology

Francque SM, Marchesini G, Kautz A, Walmsley M, Dorner R, Lazarus JV, Zelber-Sagi S, Hallsworth K, Busetto L, Frühbeck G, Dicker D, Woodward E, Korenjak M, Willemse J, Koek GH, Vinker S, Ungan M, Mendive JM, Lionis C.
PMID: 34693236
JHEP Rep. 2021 Sep 17;3(5):100322. doi: 10.1016/j.jhepr.2021.100322. eCollection 2021 Oct.

This patient guideline is intended for all patients at risk of or living with non-alcoholic fatty liver disease (NAFLD). NAFLD is the most frequent chronic liver disease worldwide and comes with a high disease burden. Yet, there is a...

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Francque SM, Marchesini G, Kautz A, et al. Non-alcoholic fatty liver disease: A patient guideline. JHEP Rep. 2021;3(5):100322doi: 10.1016/j.jhepr.2021.100322.
Francque, S. M., Marchesini, G., Kautz, A., Walmsley, M., Dorner, R., Lazarus, J. V., Zelber-Sagi, S., Hallsworth, K., Busetto, L., Frühbeck, G., Dicker, D., Woodward, E., Korenjak, M., Willemse, J., Koek, G. H., Vinker, S., Ungan, M., Mendive, J. M., & Lionis, C. (2021). Non-alcoholic fatty liver disease: A patient guideline. JHEP reports : innovation in hepatology, 3(5), 100322. https://doi.org/10.1016/j.jhepr.2021.100322
Francque, Sven M, et al. "Non-alcoholic fatty liver disease: A patient guideline." JHEP reports : innovation in hepatology vol. 3,5 (2021): 100322. doi: https://doi.org/10.1016/j.jhepr.2021.100322
Francque SM, Marchesini G, Kautz A, Walmsley M, Dorner R, Lazarus JV, Zelber-Sagi S, Hallsworth K, Busetto L, Frühbeck G, Dicker D, Woodward E, Korenjak M, Willemse J, Koek GH, Vinker S, Ungan M, Mendive JM, Lionis C. Non-alcoholic fatty liver disease: A patient guideline. JHEP Rep. 2021 Sep 17;3(5):100322. doi: 10.1016/j.jhepr.2021.100322. eCollection 2021 Oct. PMID: 34693236; PMCID: PMC8514420.
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High plasma and lingual uroguanylin as potential contributors to changes in food preference after sleeve gastrectomy.

Metabolism: clinical and experimental

Frühbeck G, Becerril S, Martín M, Ramírez B, Valentí V, Moncada R, Catalán V, Gómez-Ambrosi J, Silva C, Burrell MA, Escalada J, Rodríguez A.
PMID: 34990711
Metabolism. 2022 Jan 04;128:155119. doi: 10.1016/j.metabol.2021.155119. Epub 2022 Jan 04.

BACKGROUND: The biological mediators supporting long-term weight loss and changes in dietary choice behaviour after sleeve gastrectomy remain unclear. Guanylin and uroguanylin are gut hormones involved in the regulation of satiety, food preference and adiposity. Thus, we sought to...

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Frühbeck G, Becerril S, Martín M, et al. High plasma and lingual uroguanylin as potential contributors to changes in food preference after sleeve gastrectomy. Metabolism. 2022;128:155119doi: 10.1016/j.metabol.2021.155119.
Frühbeck, G., Becerril, S., Martín, M., Ramírez, B., Valentí, V., Moncada, R., Catalán, V., Gómez-Ambrosi, J., Silva, C., Burrell, M. A., Escalada, J., & Rodríguez, A. (2022). High plasma and lingual uroguanylin as potential contributors to changes in food preference after sleeve gastrectomy. Metabolism: clinical and experimental, 128155119. https://doi.org/10.1016/j.metabol.2021.155119
Frühbeck, Gema, et al. "High plasma and lingual uroguanylin as potential contributors to changes in food preference after sleeve gastrectomy." Metabolism: clinical and experimental vol. 128 (2022): 155119. doi: https://doi.org/10.1016/j.metabol.2021.155119
Frühbeck G, Becerril S, Martín M, Ramírez B, Valentí V, Moncada R, Catalán V, Gómez-Ambrosi J, Silva C, Burrell MA, Escalada J, Rodríguez A. High plasma and lingual uroguanylin as potential contributors to changes in food preference after sleeve gastrectomy. Metabolism. 2022 Jan 04;128:155119. doi: 10.1016/j.metabol.2021.155119. Epub 2022 Jan 04. PMID: 34990711.
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Cagrilintide plus semaglutide for obesity management.

Lancet (London, England)

Becerril S, Frühbeck G.
PMID: 33894837
Lancet. 2021 May 08;397(10286):1687-1689. doi: 10.1016/S0140-6736(21)00944-2. Epub 2021 Apr 22.

No abstract available.

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Becerril S, Frühbeck G. Cagrilintide plus semaglutide for obesity management. Lancet. 2021;397(10286):1687-1689doi: 10.1016/S0140-6736(21)00944-2.
Becerril, S., & Frühbeck, G. (2021). Cagrilintide plus semaglutide for obesity management. Lancet (London, England), 397(10286), 1687-1689. https://doi.org/10.1016/S0140-6736(21)00944-2
Becerril, Sara, and Frühbeck, Gema. "Cagrilintide plus semaglutide for obesity management." Lancet (London, England) vol. 397,10286 (2021): 1687-1689. doi: https://doi.org/10.1016/S0140-6736(21)00944-2
Becerril S, Frühbeck G. Cagrilintide plus semaglutide for obesity management. Lancet. 2021 May 08;397(10286):1687-1689. doi: 10.1016/S0140-6736(21)00944-2. Epub 2021 Apr 22. PMID: 33894837.
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Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements.

Acta biomaterialia

Unamuno X, Gómez-Ambrosi J, Becerril S, Álvarez-Cienfuegos FJ, Ramírez B, Rodríguez A, Ezquerro S, Valentí V, Moncada R, Mentxaka A, Llorente M, Silva C, Elizalde MLR, Catalán V, Frühbeck G.
PMID: 35007786
Acta Biomater. 2022 Jan 07; doi: 10.1016/j.actbio.2022.01.007. Epub 2022 Jan 07.

Biomechanical properties of adipose tissue (AT) are closely involved in the development of obesity-associated comorbidities. Bariatric surgery (BS) constitutes the most effective option for a sustained weight loss in addition to improving obesity-associated metabolic diseases including type 2 diabetes...

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Unamuno X, Gómez-Ambrosi J, Becerril S, et al. Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements. Acta Biomater. 2022;doi: 10.1016/j.actbio.2022.01.007.
Unamuno, X., Gómez-Ambrosi, J., Becerril, S., Álvarez-Cienfuegos, F. J., Ramírez, B., Rodríguez, A., Ezquerro, S., Valentí, V., Moncada, R., Mentxaka, A., Llorente, M., Silva, C., Elizalde, M. L. R., Catalán, V., & Frühbeck, G. (2022). Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements. Acta biomaterialia, . https://doi.org/10.1016/j.actbio.2022.01.007
Unamuno, Xabier, et al. "Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements." Acta biomaterialia vol. (2022). doi: https://doi.org/10.1016/j.actbio.2022.01.007
Unamuno X, Gómez-Ambrosi J, Becerril S, Álvarez-Cienfuegos FJ, Ramírez B, Rodríguez A, Ezquerro S, Valentí V, Moncada R, Mentxaka A, Llorente M, Silva C, Elizalde MLR, Catalán V, Frühbeck G. Changes in mechanical properties of adipose tissue after bariatric surgery driven by extracellular matrix remodelling and neovascularization are associated with metabolic improvements. Acta Biomater. 2022 Jan 07; doi: 10.1016/j.actbio.2022.01.007. Epub 2022 Jan 07. PMID: 35007786.
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Role of ANGPTL8 in NAFLD Improvement after Bariatric Surgery in Experimental and Human Obesity.

International journal of molecular sciences

Perdomo CM, Gómez-Ambrosi J, Becerril S, Valentí V, Moncada R, Fernández-Sáez EM, Méndez-Giménez L, Ezquerro S, Catalán V, Silva C, Escalada J, Frühbeck G, Rodríguez A.
PMID: 34884755
Int J Mol Sci. 2021 Nov 30;22(23). doi: 10.3390/ijms222312945.

Angiopoietin-like protein 8 (ANGPTL8) is an hepatokine altered in several metabolic conditions, such as obesity, type 2 diabetes, dyslipidemia and nonalcoholic fatty liver disease (NAFLD). We sought to explore whether ANGPTL8 is involved in NAFLD amelioration after bariatric surgery...

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Perdomo CM, Gómez-Ambrosi J, Becerril S, et al. Role of ANGPTL8 in NAFLD Improvement after Bariatric Surgery in Experimental and Human Obesity. Int J Mol Sci. 2021;22(23)doi: 10.3390/ijms222312945.
Perdomo, C. M., Gómez-Ambrosi, J., Becerril, S., Valentí, V., Moncada, R., Fernández-Sáez, E. M., Méndez-Giménez, L., Ezquerro, S., Catalán, V., Silva, C., Escalada, J., Frühbeck, G., & Rodríguez, A. (2021). Role of ANGPTL8 in NAFLD Improvement after Bariatric Surgery in Experimental and Human Obesity. International journal of molecular sciences, 22(23), . https://doi.org/10.3390/ijms222312945
Perdomo, Carolina M, et al. "Role of ANGPTL8 in NAFLD Improvement after Bariatric Surgery in Experimental and Human Obesity." International journal of molecular sciences vol. 22,23 (2021). doi: https://doi.org/10.3390/ijms222312945
Perdomo CM, Gómez-Ambrosi J, Becerril S, Valentí V, Moncada R, Fernández-Sáez EM, Méndez-Giménez L, Ezquerro S, Catalán V, Silva C, Escalada J, Frühbeck G, Rodríguez A. Role of ANGPTL8 in NAFLD Improvement after Bariatric Surgery in Experimental and Human Obesity. Int J Mol Sci. 2021 Nov 30;22(23). doi: 10.3390/ijms222312945. PMID: 34884755.
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The Differential Expression of the Inflammasomes in Adipose Tissue and Colon Influences the Development of Colon Cancer in a Context of Obesity by Regulating Intestinal Inflammation.

Journal of inflammation research

Frühbeck G, Mentxaka A, Ahechu P, Gómez-Ambrosi J, Ramírez B, Becerril S, Rodríguez A, Unamuno X, Cienfuegos JA, Casado M, Burrell MA, Martín M, Baixauli J, Valentí V, Moncada R, Reina G, Silva C, Catalán V.
PMID: 34880645
J Inflamm Res. 2021 Dec 01;14:6431-6446. doi: 10.2147/JIR.S335882. eCollection 2021.

BACKGROUND: Inflammasomes maintain tissue homeostasis and their altered regulation in the colon, and the adipose tissue (AT) leads to chronic activation of inflammatory pathways promoting colon cancer (CC) development. We aimed to analyze the potential involvement of inflammasomes in...

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Frühbeck G, Mentxaka A, Ahechu P, et al. The Differential Expression of the Inflammasomes in Adipose Tissue and Colon Influences the Development of Colon Cancer in a Context of Obesity by Regulating Intestinal Inflammation. J Inflamm Res. 2021;14:6431-6446doi: 10.2147/JIR.S335882.
Frühbeck, G., Mentxaka, A., Ahechu, P., Gómez-Ambrosi, J., Ramírez, B., Becerril, S., Rodríguez, A., Unamuno, X., Cienfuegos, J. A., Casado, M., Burrell, M. A., Martín, M., Baixauli, J., Valentí, V., Moncada, R., Reina, G., Silva, C., & Catalán, V. (2021). The Differential Expression of the Inflammasomes in Adipose Tissue and Colon Influences the Development of Colon Cancer in a Context of Obesity by Regulating Intestinal Inflammation. Journal of inflammation research, 146431-6446. https://doi.org/10.2147/JIR.S335882
Frühbeck, Gema, et al. "The Differential Expression of the Inflammasomes in Adipose Tissue and Colon Influences the Development of Colon Cancer in a Context of Obesity by Regulating Intestinal Inflammation." Journal of inflammation research vol. 14 (2021): 6431-6446. doi: https://doi.org/10.2147/JIR.S335882
Frühbeck G, Mentxaka A, Ahechu P, Gómez-Ambrosi J, Ramírez B, Becerril S, Rodríguez A, Unamuno X, Cienfuegos JA, Casado M, Burrell MA, Martín M, Baixauli J, Valentí V, Moncada R, Reina G, Silva C, Catalán V. The Differential Expression of the Inflammasomes in Adipose Tissue and Colon Influences the Development of Colon Cancer in a Context of Obesity by Regulating Intestinal Inflammation. J Inflamm Res. 2021 Dec 01;14:6431-6446. doi: 10.2147/JIR.S335882. eCollection 2021. PMID: 34880645; PMCID: PMC8646233.
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Transdiagnostic Perspective of Impulsivity and Compulsivity in Obesity: From Cognitive Profile to Self-Reported Dimensions in Clinical Samples with and without Diabetes.

Nutrients

Testa G, Mora-Maltas B, Camacho-Barcia L, Granero R, Lucas I, Agüera Z, Jiménez-Murcia S, Baños R, Bertaina-Anglade V, Botella C, Bulló M, Casanueva FF, Dalsgaard S, Fernández-Real JM, Franke B, Frühbeck G, Fitó M, Gómez-Martínez C, Pintó X, Poelmans G, Tinahones FJ, Torre R, Salas-Salvadó J, Serra-Majem L, Vos S, Wimberley T, Fernández-Aranda F.
PMID: 34959979
Nutrients. 2021 Dec 10;13(12). doi: 10.3390/nu13124426.

Impulsive and compulsive behaviors have both been observed in individuals with obesity. The co-occurrence of obesity and type 2 diabetes (T2D) is more strongly associated with impulsivity, although there are no conclusive results yet. A multidimensional assessment of impulsivity...

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Testa G, Mora-Maltas B, Camacho-Barcia L, et al. Transdiagnostic Perspective of Impulsivity and Compulsivity in Obesity: From Cognitive Profile to Self-Reported Dimensions in Clinical Samples with and without Diabetes. Nutrients. 2021;13(12)doi: 10.3390/nu13124426.
Testa, G., Mora-Maltas, B., Camacho-Barcia, L., Granero, R., Lucas, I., Agüera, Z., Jiménez-Murcia, S., Baños, R., Bertaina-Anglade, V., Botella, C., Bulló, M., Casanueva, F. F., Dalsgaard, S., Fernández-Real, J. M., Franke, B., Frühbeck, G., Fitó, M., Gómez-Martínez, C., Pintó, X., Poelmans, G., Tinahones, F. J., Torre, R., Salas-Salvadó, J., Serra-Majem, L., Vos, S., Wimberley, T., & Fernández-Aranda, F. (2021). Transdiagnostic Perspective of Impulsivity and Compulsivity in Obesity: From Cognitive Profile to Self-Reported Dimensions in Clinical Samples with and without Diabetes. Nutrients, 13(12), . https://doi.org/10.3390/nu13124426
Testa, Giulia, et al. "Transdiagnostic Perspective of Impulsivity and Compulsivity in Obesity: From Cognitive Profile to Self-Reported Dimensions in Clinical Samples with and without Diabetes." Nutrients vol. 13,12 (2021). doi: https://doi.org/10.3390/nu13124426
Testa G, Mora-Maltas B, Camacho-Barcia L, Granero R, Lucas I, Agüera Z, Jiménez-Murcia S, Baños R, Bertaina-Anglade V, Botella C, Bulló M, Casanueva FF, Dalsgaard S, Fernández-Real JM, Franke B, Frühbeck G, Fitó M, Gómez-Martínez C, Pintó X, Poelmans G, Tinahones FJ, Torre R, Salas-Salvadó J, Serra-Majem L, Vos S, Wimberley T, Fernández-Aranda F. Transdiagnostic Perspective of Impulsivity and Compulsivity in Obesity: From Cognitive Profile to Self-Reported Dimensions in Clinical Samples with and without Diabetes. Nutrients. 2021 Dec 10;13(12). doi: 10.3390/nu13124426. PMID: 34959979; PMCID: PMC8707121.
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