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[On childbirth and child rearing. 9. The pregnant woman and her diet].

Josanpu zasshi = The Japanese journal for midwife

Miyasato K, Sakakura K, Suganuma H, Kamata H, Suemitsu H.
PMID: 3854880
Josanpu Zasshi. 1985 Dec;39(12):1065-8.

No abstract available.

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Miyasato K, Sakakura K, Suganuma H, et al. [On childbirth and child rearing. 9. The pregnant woman and her diet]. Josanpu Zasshi. 1985;39(12):1065-8
Miyasato, K., Sakakura, K., Suganuma, H., Kamata, H., & Suemitsu, H. (1985). [On childbirth and child rearing. 9. The pregnant woman and her diet]. Josanpu zasshi = The Japanese journal for midwife, 39(12), 1065-8.
Miyasato, K, et al. "[On childbirth and child rearing. 9. The pregnant woman and her diet]." Josanpu zasshi = The Japanese journal for midwife vol. 39,12 (1985): 1065-8.
Miyasato K, Sakakura K, Suganuma H, Kamata H, Suemitsu H. [On childbirth and child rearing. 9. The pregnant woman and her diet]. Josanpu Zasshi. 1985 Dec;39(12):1065-8. Japanese. PMID: 3854880.
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Draft Genome Sequence of Five Shiga Toxin-Producing .

Genome announcements

Asakura H, Ikeda T, Yamamoto S, Kabeya H, Sugiyama H, Takai S.
PMID: 28254967
Genome Announc. 2017 Mar 02;5(9). doi: 10.1128/genomeA.01455-16.

Shiga toxin-producing

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Asakura H, Ikeda T, Yamamoto S, et al. Draft Genome Sequence of Five Shiga Toxin-Producing . Genome Announc. 2017;5(9)doi: 10.1128/genomeA.01455-16.
Asakura, H., Ikeda, T., Yamamoto, S., Kabeya, H., Sugiyama, H., & Takai, S. (2017). Draft Genome Sequence of Five Shiga Toxin-Producing . Genome announcements, 5(9), . https://doi.org/10.1128/genomeA.01455-16
Asakura, Hiroshi, et al. "Draft Genome Sequence of Five Shiga Toxin-Producing ." Genome announcements vol. 5,9 (2017). doi: https://doi.org/10.1128/genomeA.01455-16
Asakura H, Ikeda T, Yamamoto S, Kabeya H, Sugiyama H, Takai S. Draft Genome Sequence of Five Shiga Toxin-Producing . Genome Announc. 2017 Mar 02;5(9). doi: 10.1128/genomeA.01455-16. PMID: 28254967; PMCID: PMC5334574.
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Case of ketoacidosis by a sodium-glucose cotransporter 2 inhibitor in a diabetic patient with a low-carbohydrate diet.

Journal of diabetes investigation

Hayami T, Kato Y, Kamiya H, Kondo M, Naito E, Sugiura Y, Kojima C, Sato S, Yamada Y, Kasagi R, Ando T, Noda S, Nakai H, Takada E, Asano E, Motegi M, Watarai A, Kato K, Nakamura J.
PMID: 26417418
J Diabetes Investig. 2015 Sep;6(5):587-90. doi: 10.1111/jdi.12330. Epub 2015 Feb 20.

We present a case of a 32-year-old diabetic woman with Prader-Willi syndrome who developed severe ketoacidosis caused by a sodium-glucose cotransporter 2 (SGLT2) inhibitor, a novel class of antihyperglycemic agents, during a strict low-carbohydrate diet. At admission, a serum...

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Hayami T, Kato Y, Kamiya H, et al. Case of ketoacidosis by a sodium-glucose cotransporter 2 inhibitor in a diabetic patient with a low-carbohydrate diet. J Diabetes Investig. 2015;6(5):587-90doi: 10.1111/jdi.12330.
Hayami, T., Kato, Y., Kamiya, H., Kondo, M., Naito, E., Sugiura, Y., Kojima, C., Sato, S., Yamada, Y., Kasagi, R., Ando, T., Noda, S., Nakai, H., Takada, E., Asano, E., Motegi, M., Watarai, A., Kato, K., & Nakamura, J. (2015). Case of ketoacidosis by a sodium-glucose cotransporter 2 inhibitor in a diabetic patient with a low-carbohydrate diet. Journal of diabetes investigation, 6(5), 587-90. https://doi.org/10.1111/jdi.12330
Hayami, Tomohide, et al. "Case of ketoacidosis by a sodium-glucose cotransporter 2 inhibitor in a diabetic patient with a low-carbohydrate diet." Journal of diabetes investigation vol. 6,5 (2015): 587-90. doi: https://doi.org/10.1111/jdi.12330
Hayami T, Kato Y, Kamiya H, Kondo M, Naito E, Sugiura Y, Kojima C, Sato S, Yamada Y, Kasagi R, Ando T, Noda S, Nakai H, Takada E, Asano E, Motegi M, Watarai A, Kato K, Nakamura J. Case of ketoacidosis by a sodium-glucose cotransporter 2 inhibitor in a diabetic patient with a low-carbohydrate diet. J Diabetes Investig. 2015 Sep;6(5):587-90. doi: 10.1111/jdi.12330. Epub 2015 Feb 20. PMID: 26417418; PMCID: PMC4578500.
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Practical Liposomal Formulation for Taxanes with Polyethoxylated Castor Oil and Ethanol with Complete Encapsulation Efficiency and High Loading Efficiency.

Nanomaterials (Basel, Switzerland)

Shigehiro T, Masuda J, Saito S, Khayrani AC, Jinno K, Seno A, Vaidyanath A, Mizutani A, Kasai T, Murakami H, Satoh A, Ito T, Hamada H, Seno Y, Mandai T, Seno M.
PMID: 28946623
Nanomaterials (Basel). 2017 Sep 23;7(10). doi: 10.3390/nano7100290.

Taxanes including paclitaxel and docetaxel are effective anticancer agents preferably sufficient for liposomal drug delivery. However, the encapsulation of these drugs with effective amounts into conventional liposomes is difficult due to their high hydrophobicity. Therefore, an effective encapsulation strategy...

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Shigehiro T, Masuda J, Saito S, et al. Practical Liposomal Formulation for Taxanes with Polyethoxylated Castor Oil and Ethanol with Complete Encapsulation Efficiency and High Loading Efficiency. Nanomaterials (Basel). 2017;7(10)doi: 10.3390/nano7100290.
Shigehiro, T., Masuda, J., Saito, S., Khayrani, A. C., Jinno, K., Seno, A., Vaidyanath, A., Mizutani, A., Kasai, T., Murakami, H., Satoh, A., Ito, T., Hamada, H., Seno, Y., Mandai, T., & Seno, M. (2017). Practical Liposomal Formulation for Taxanes with Polyethoxylated Castor Oil and Ethanol with Complete Encapsulation Efficiency and High Loading Efficiency. Nanomaterials (Basel, Switzerland), 7(10), . https://doi.org/10.3390/nano7100290
Shigehiro, Tsukasa, et al. "Practical Liposomal Formulation for Taxanes with Polyethoxylated Castor Oil and Ethanol with Complete Encapsulation Efficiency and High Loading Efficiency." Nanomaterials (Basel, Switzerland) vol. 7,10 (2017). doi: https://doi.org/10.3390/nano7100290
Shigehiro T, Masuda J, Saito S, Khayrani AC, Jinno K, Seno A, Vaidyanath A, Mizutani A, Kasai T, Murakami H, Satoh A, Ito T, Hamada H, Seno Y, Mandai T, Seno M. Practical Liposomal Formulation for Taxanes with Polyethoxylated Castor Oil and Ethanol with Complete Encapsulation Efficiency and High Loading Efficiency. Nanomaterials (Basel). 2017 Sep 23;7(10). doi: 10.3390/nano7100290. PMID: 28946623; PMCID: PMC5666455.
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Yellow Nail Syndrome With Dramatic Improvement of Nail Manifestations After Endoscopic Sinus Surgery.

Clinical medicine insights. Ear, nose and throat

Hosokawa Y, Kuboki A, Mori A, Kanaya H, Nakayama T, Haruna S.
PMID: 28979177
Clin Med Insights Ear Nose Throat. 2017 Jul 05;10:1179550617718184. doi: 10.1177/1179550617718184. eCollection 2017.

OBJECTIVES: Yellow nail syndrome (YNS) is a rare disease of unknown cause characterized by the triad of yellow nails, respiratory manifestations, and lymphedema. Although several therapies for YNS have been reported, there is no common consensus in the treatment....

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Hosokawa Y, Kuboki A, Mori A, et al. Yellow Nail Syndrome With Dramatic Improvement of Nail Manifestations After Endoscopic Sinus Surgery. Clin Med Insights Ear Nose Throat. 2017;10:1179550617718184doi: 10.1177/1179550617718184.
Hosokawa, Y., Kuboki, A., Mori, A., Kanaya, H., Nakayama, T., & Haruna, S. (2017). Yellow Nail Syndrome With Dramatic Improvement of Nail Manifestations After Endoscopic Sinus Surgery. Clinical medicine insights. Ear, nose and throat, 101179550617718184. https://doi.org/10.1177/1179550617718184
Hosokawa, Yu, et al. "Yellow Nail Syndrome With Dramatic Improvement of Nail Manifestations After Endoscopic Sinus Surgery." Clinical medicine insights. Ear, nose and throat vol. 10 (2017): 1179550617718184. doi: https://doi.org/10.1177/1179550617718184
Hosokawa Y, Kuboki A, Mori A, Kanaya H, Nakayama T, Haruna S. Yellow Nail Syndrome With Dramatic Improvement of Nail Manifestations After Endoscopic Sinus Surgery. Clin Med Insights Ear Nose Throat. 2017 Jul 05;10:1179550617718184. doi: 10.1177/1179550617718184. eCollection 2017. PMID: 28979177; PMCID: PMC5617082.
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Adult-onset diffuse nesidioblastosis causing hypoglycemia.

Clinical journal of gastroenterology

Maeda Y, Yokoyama K, Takeda K, Takada J, Hamada H, Hujioka Y, Kudo SE.
PMID: 26181405
Clin J Gastroenterol. 2013 Feb;6(1):50-4. doi: 10.1007/s12328-012-0335-0. Epub 2012 Dec 08.

We report the case of a 32-year-old male with adult-onset diffuse nesidioblastosis causing hypoglycemia. Under the tentative diagnosis of insulinoma, localization procedures were carried out but no tumor was found. The presence of an insulinoma in the tail of...

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Maeda Y, Yokoyama K, Takeda K, et al. Adult-onset diffuse nesidioblastosis causing hypoglycemia. Clin J Gastroenterol. 2012;6(1):50-4doi: 10.1007/s12328-012-0335-0.
Maeda, Y., Yokoyama, K., Takeda, K., Takada, J., Hamada, H., Hujioka, Y., & Kudo, S. E. (2013). Adult-onset diffuse nesidioblastosis causing hypoglycemia. Clinical journal of gastroenterology, 6(1), 50-4. https://doi.org/10.1007/s12328-012-0335-0
Maeda, Yasuharu, et al. "Adult-onset diffuse nesidioblastosis causing hypoglycemia." Clinical journal of gastroenterology vol. 6,1 (2013): 50-4. doi: https://doi.org/10.1007/s12328-012-0335-0
Maeda Y, Yokoyama K, Takeda K, Takada J, Hamada H, Hujioka Y, Kudo SE. Adult-onset diffuse nesidioblastosis causing hypoglycemia. Clin J Gastroenterol. 2013 Feb;6(1):50-4. doi: 10.1007/s12328-012-0335-0. Epub 2012 Dec 08. PMID: 26181405.
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Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes.

Journal of diabetes investigation

Kondo M, Kamiya H, Himeno T, Naruse K, Nakashima E, Watarai A, Shibata T, Tosaki T, Kato J, Okawa T, Hamada Y, Isobe K, Oiso Y, Nakamura J.
PMID: 25802721
J Diabetes Investig. 2015 Mar;6(2):140-9. doi: 10.1111/jdi.12272. Epub 2014 Sep 11.

AIMS/INTRODUCTION: Recent studies have shown that cell transplantation therapies, such as endothelial precursor cells, bone marrow-derived mononuclear cells (BM-MNCs) and mesenchymal stem cells, are effective on diabetic polyneuropathy through ameliorating impaired nerve blood flow in diabetic rats. Here, we...

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Kondo M, Kamiya H, Himeno T, et al. Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes. J Diabetes Investig. 2014;6(2):140-9doi: 10.1111/jdi.12272.
Kondo, M., Kamiya, H., Himeno, T., Naruse, K., Nakashima, E., Watarai, A., Shibata, T., Tosaki, T., Kato, J., Okawa, T., Hamada, Y., Isobe, K., Oiso, Y., & Nakamura, J. (2015). Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes. Journal of diabetes investigation, 6(2), 140-9. https://doi.org/10.1111/jdi.12272
Kondo, Masaki, et al. "Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes." Journal of diabetes investigation vol. 6,2 (2015): 140-9. doi: https://doi.org/10.1111/jdi.12272
Kondo M, Kamiya H, Himeno T, Naruse K, Nakashima E, Watarai A, Shibata T, Tosaki T, Kato J, Okawa T, Hamada Y, Isobe K, Oiso Y, Nakamura J. Therapeutic efficacy of bone marrow-derived mononuclear cells in diabetic polyneuropathy is impaired with aging or diabetes. J Diabetes Investig. 2015 Mar;6(2):140-9. doi: 10.1111/jdi.12272. Epub 2014 Sep 11. PMID: 25802721; PMCID: PMC4364848.
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[No title available]

Oncotarget

Suzuki S, Iwaizumi M, Yamada H, Sugiyama T, Hamaya Y, Furuta T, Kanaoka S, Sugimura H, Miyajima H, Osawa S, Carethers JM, Sugimoto K.
PMID: 29545913
Oncotarget. 2017 Nov 15;9(14):11477-11488. doi: 10.18632/oncotarget.22484. eCollection 2018 Feb 20.

BACKGROUNDS: Trifluridine is an active antitumor component of TAS-102 that resembles 5-fluorouracil. Although patients with advanced colorectal cancer (CRC) exhibiting a mismatch repair (MMR) deficiency reportedly do not benefit from 5-fluorouracil-based chemotherapy and we previously reported that truncated methyl-CpG...

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Suzuki S, Iwaizumi M, Yamada H, et al. . Oncotarget. 2017;9(14):11477-11488doi: 10.18632/oncotarget.22484.
Suzuki, S., Iwaizumi, M., Yamada, H., Sugiyama, T., Hamaya, Y., Furuta, T., Kanaoka, S., Sugimura, H., Miyajima, H., Osawa, S., Carethers, J. M., & Sugimoto, K. (2018). . Oncotarget, 9(14), 11477-11488. https://doi.org/10.18632/oncotarget.22484
Suzuki, Satoshi, et al. "." Oncotarget vol. 9,14 (2018): 11477-11488. doi: https://doi.org/10.18632/oncotarget.22484
Suzuki S, Iwaizumi M, Yamada H, Sugiyama T, Hamaya Y, Furuta T, Kanaoka S, Sugimura H, Miyajima H, Osawa S, Carethers JM, Sugimoto K. . Oncotarget. 2017 Nov 15;9(14):11477-11488. doi: 10.18632/oncotarget.22484. eCollection 2018 Feb 20. PMID: 29545913; PMCID: PMC5837757.
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Possible association of single nucleotide polymorphisms in the 3' untranslated region of HOXB9 with acetabular overcoverage.

Bone & joint research

Sekimoto T, Kurogi S, Funamoto T, Ota T, Watanabe S, Sakamoto T, Hamada H, Chosa E.
PMID: 25833894
Bone Joint Res. 2015 Apr;4(4):50-5. doi: 10.1302/2046-3758.44.2000349.

OBJECTIVES: Excessive acetabular coverage is the most common cause of pincer-type femoroacetabular impingement. To date, an association between acetabular over-coverage and genetic variations has not been studied. In this study we investigated the association between single nucleotide polymorphisms (SNPs)...

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Sekimoto T, Kurogi S, Funamoto T, et al. Possible association of single nucleotide polymorphisms in the 3' untranslated region of HOXB9 with acetabular overcoverage. Bone Joint Res. 2015;4(4):50-5doi: 10.1302/2046-3758.44.2000349.
Sekimoto, T., Kurogi, S., Funamoto, T., Ota, T., Watanabe, S., Sakamoto, T., Hamada, H., & Chosa, E. (2015). Possible association of single nucleotide polymorphisms in the 3' untranslated region of HOXB9 with acetabular overcoverage. Bone & joint research, 4(4), 50-5. https://doi.org/10.1302/2046-3758.44.2000349
Sekimoto, T, et al. "Possible association of single nucleotide polymorphisms in the 3' untranslated region of HOXB9 with acetabular overcoverage." Bone & joint research vol. 4,4 (2015): 50-5. doi: https://doi.org/10.1302/2046-3758.44.2000349
Sekimoto T, Kurogi S, Funamoto T, Ota T, Watanabe S, Sakamoto T, Hamada H, Chosa E. Possible association of single nucleotide polymorphisms in the 3' untranslated region of HOXB9 with acetabular overcoverage. Bone Joint Res. 2015 Apr;4(4):50-5. doi: 10.1302/2046-3758.44.2000349. PMID: 25833894; PMCID: PMC4413364.
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Decreased SIRT1 expression and LKB1 phosphorylation occur with long-term high-fat diet feeding, in addition to AMPK phosphorylation impairment in the early phase.

Obesity research & clinical practice

Yoneda M, Guo Y, Ono H, Nakatsu Y, Zhang J, Cui X, Iwashita M, Kumamoto S, Tsuchiya Y, Sakoda H, Fujishiro M, Kushiyama A, Koketsu Y, Kikuchi T, Kamata H, Nishimura F, Asano T.
PMID: 24345663
Obes Res Clin Pract. 2010 Jul-Sep;4(3):e163-246. doi: 10.1016/j.orcp.2010.02.002.

AIMS: Energy sensing systems including AMPK and SIRT1 play important roles in the regulation of hepatic gluconeogenesis and fatty acid oxidation. In this study, we investigated how hepatic LKB1-AMPK signaling and SIRT1 expression are altered after 2 or 8...

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Yoneda M, Guo Y, Ono H, et al. Decreased SIRT1 expression and LKB1 phosphorylation occur with long-term high-fat diet feeding, in addition to AMPK phosphorylation impairment in the early phase. Obes Res Clin Pract. 2010;4(3):e163-246doi: 10.1016/j.orcp.2010.02.002.
Yoneda, M., Guo, Y., Ono, H., Nakatsu, Y., Zhang, J., Cui, X., Iwashita, M., Kumamoto, S., Tsuchiya, Y., Sakoda, H., Fujishiro, M., Kushiyama, A., Koketsu, Y., Kikuchi, T., Kamata, H., Nishimura, F., & Asano, T. (2010). Decreased SIRT1 expression and LKB1 phosphorylation occur with long-term high-fat diet feeding, in addition to AMPK phosphorylation impairment in the early phase. Obesity research & clinical practice, 4(3), e163-246. https://doi.org/10.1016/j.orcp.2010.02.002
Yoneda, Masayasu, et al. "Decreased SIRT1 expression and LKB1 phosphorylation occur with long-term high-fat diet feeding, in addition to AMPK phosphorylation impairment in the early phase." Obesity research & clinical practice vol. 4,3 (2010): e163-246. doi: https://doi.org/10.1016/j.orcp.2010.02.002
Yoneda M, Guo Y, Ono H, Nakatsu Y, Zhang J, Cui X, Iwashita M, Kumamoto S, Tsuchiya Y, Sakoda H, Fujishiro M, Kushiyama A, Koketsu Y, Kikuchi T, Kamata H, Nishimura F, Asano T. Decreased SIRT1 expression and LKB1 phosphorylation occur with long-term high-fat diet feeding, in addition to AMPK phosphorylation impairment in the early phase. Obes Res Clin Pract. 2010 Jul-Sep;4(3):e163-246. doi: 10.1016/j.orcp.2010.02.002. PMID: 24345663.
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High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt.

Journal of diabetes investigation

Nakamura N, Naruse K, Kobayashi Y, Matsuki T, Hamada Y, Nakashima E, Kamiya H, Hata M, Nishikawa T, Enomoto A, Takahashi M, Murohara T, Matsubara T, Oiso Y, Nakamura J.
PMID: 24843496
J Diabetes Investig. 2011 Aug 02;2(4):262-70. doi: 10.1111/j.2040-1124.2010.00093.x.

UNLABELLED: Aims/Introduction:  Endothelial progenitor cells (EPC) play a critical role in adult vasculogenesis and vascular repair. Previous studies have described the dysfunction of EPC in diabetic patients, but the precise mechanism is still unclear. To elucidate the dysfunction of...

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Nakamura N, Naruse K, Kobayashi Y, et al. High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt. J Diabetes Investig. 2011;2(4):262-70doi: 10.1111/j.2040-1124.2010.00093.x.
Nakamura, N., Naruse, K., Kobayashi, Y., Matsuki, T., Hamada, Y., Nakashima, E., Kamiya, H., Hata, M., Nishikawa, T., Enomoto, A., Takahashi, M., Murohara, T., Matsubara, T., Oiso, Y., & Nakamura, J. (2011). High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt. Journal of diabetes investigation, 2(4), 262-70. https://doi.org/10.1111/j.2040-1124.2010.00093.x
Nakamura, Nobuhisa, et al. "High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt." Journal of diabetes investigation vol. 2,4 (2011): 262-70. doi: https://doi.org/10.1111/j.2040-1124.2010.00093.x
Nakamura N, Naruse K, Kobayashi Y, Matsuki T, Hamada Y, Nakashima E, Kamiya H, Hata M, Nishikawa T, Enomoto A, Takahashi M, Murohara T, Matsubara T, Oiso Y, Nakamura J. High glucose impairs the proliferation and increases the apoptosis of endothelial progenitor cells by suppression of Akt. J Diabetes Investig. 2011 Aug 02;2(4):262-70. doi: 10.1111/j.2040-1124.2010.00093.x. PMID: 24843496; PMCID: PMC4014965.
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Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion.

Journal of diabetes investigation

Sakamoto E, Seino Y, Fukami A, Mizutani N, Tsunekawa S, Ishikawa K, Ogata H, Uenishi E, Kamiya H, Hamada Y, Sato H, Harada N, Toyoda Y, Miwa I, Nakamura J, Inagaki N, Oiso Y, Ozaki N.
PMID: 24843603
J Diabetes Investig. 2012 Oct 18;3(5):432-40. doi: 10.1111/j.2040-1124.2012.00208.x.

UNLABELLED: Aims/Introduction:  Excessive intake of sucrose can cause severe health issues, such as diabetes mellitus. In animal studies, consumption of a high-sucrose diet (SUC) has been shown to cause obesity, insulin resistance and glucose intolerance. However, several in vivo...

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Sakamoto E, Seino Y, Fukami A, et al. Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion. J Diabetes Investig. 2012;3(5):432-40doi: 10.1111/j.2040-1124.2012.00208.x.
Sakamoto, E., Seino, Y., Fukami, A., Mizutani, N., Tsunekawa, S., Ishikawa, K., Ogata, H., Uenishi, E., Kamiya, H., Hamada, Y., Sato, H., Harada, N., Toyoda, Y., Miwa, I., Nakamura, J., Inagaki, N., Oiso, Y., & Ozaki, N. (2012). Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion. Journal of diabetes investigation, 3(5), 432-40. https://doi.org/10.1111/j.2040-1124.2012.00208.x
Sakamoto, Eriko, et al. "Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion." Journal of diabetes investigation vol. 3,5 (2012): 432-40. doi: https://doi.org/10.1111/j.2040-1124.2012.00208.x
Sakamoto E, Seino Y, Fukami A, Mizutani N, Tsunekawa S, Ishikawa K, Ogata H, Uenishi E, Kamiya H, Hamada Y, Sato H, Harada N, Toyoda Y, Miwa I, Nakamura J, Inagaki N, Oiso Y, Ozaki N. Ingestion of a moderate high-sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon-like peptide-1 secretion. J Diabetes Investig. 2012 Oct 18;3(5):432-40. doi: 10.1111/j.2040-1124.2012.00208.x. PMID: 24843603; PMCID: PMC4019243.
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