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Showing 1 to 12 of 1151 entries
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[Rehabilitation of essential hypertension].

Nihon rinsho. Japanese journal of clinical medicine

Kobayashi T, Kajihara T, Sasamoto S, Kikuchi T, Terasaki U.
PMID: 4863150
Nihon Rinsho. 1967 Apr;25(4):691-6.

No abstract available.

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Kobayashi T, Kajihara T, Sasamoto S, et al. [Rehabilitation of essential hypertension]. Nihon Rinsho. 1967;25(4):691-6
Kobayashi, T., Kajihara, T., Sasamoto, S., Kikuchi, T., & Terasaki, U. (1967). [Rehabilitation of essential hypertension]. Nihon rinsho. Japanese journal of clinical medicine, 25(4), 691-6.
Kobayashi, T, et al. "[Rehabilitation of essential hypertension]." Nihon rinsho. Japanese journal of clinical medicine vol. 25,4 (1967): 691-6.
Kobayashi T, Kajihara T, Sasamoto S, Kikuchi T, Terasaki U. [Rehabilitation of essential hypertension]. Nihon Rinsho. 1967 Apr;25(4):691-6. Japanese. PMID: 4863150.
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Erratum: Loss of miR-542-3p enhances IGFBP-1 expression in decidualizing human endometrial stromal cells.

Scientific reports

Tochigi H, Kajihara T, Mizuno Y, Mizuno Y, Tamaru S, Kamei Y, Okazaki Y, Brosens JJ, Ishihara O.
PMID: 28443632
Sci Rep. 2017 Apr 26;7:46591. doi: 10.1038/srep46591.

No abstract available.

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Tochigi H, Kajihara T, Mizuno Y, et al. Erratum: Loss of miR-542-3p enhances IGFBP-1 expression in decidualizing human endometrial stromal cells. Sci Rep. 2017;7:46591doi: 10.1038/srep46591.
Tochigi, H., Kajihara, T., Mizuno, Y., Mizuno, Y., Tamaru, S., Kamei, Y., Okazaki, Y., Brosens, J. J., & Ishihara, O. (2017). Erratum: Loss of miR-542-3p enhances IGFBP-1 expression in decidualizing human endometrial stromal cells. Scientific reports, 746591. https://doi.org/10.1038/srep46591
Tochigi, Hideno, et al. "Erratum: Loss of miR-542-3p enhances IGFBP-1 expression in decidualizing human endometrial stromal cells." Scientific reports vol. 7 (2017): 46591. doi: https://doi.org/10.1038/srep46591
Tochigi H, Kajihara T, Mizuno Y, Mizuno Y, Tamaru S, Kamei Y, Okazaki Y, Brosens JJ, Ishihara O. Erratum: Loss of miR-542-3p enhances IGFBP-1 expression in decidualizing human endometrial stromal cells. Sci Rep. 2017 Apr 26;7:46591. doi: 10.1038/srep46591. PMID: 28443632; PMCID: PMC5405451.
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Comparative analysis of transcriptomes in aerial stems and roots of Ephedra sinica based on high-throughput mRNA sequencing.

Genomics data

Okada T, Takahashi H, Suzuki Y, Sugano S, Noji M, Kenmoku H, Toyota M, Kanaya S, Kawahara N, Asakawa Y, Sekita S.
PMID: 27625990
Genom Data. 2016 Aug 04;10:4-11. doi: 10.1016/j.gdata.2016.08.003. eCollection 2016 Dec.

Ephedra plants are taxonomically classified as gymnosperms, and are medicinally important as the botanical origin of crude drugs and as bioresources that contain pharmacologically active chemicals. Here we show a comparative analysis of the transcriptomes of aerial stems and...

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Okada T, Takahashi H, Suzuki Y, et al. Comparative analysis of transcriptomes in aerial stems and roots of Ephedra sinica based on high-throughput mRNA sequencing. Genom Data. 2016;10:4-11doi: 10.1016/j.gdata.2016.08.003.
Okada, T., Takahashi, H., Suzuki, Y., Sugano, S., Noji, M., Kenmoku, H., Toyota, M., Kanaya, S., Kawahara, N., Asakawa, Y., & Sekita, S. (2016). Comparative analysis of transcriptomes in aerial stems and roots of Ephedra sinica based on high-throughput mRNA sequencing. Genomics data, 104-11. https://doi.org/10.1016/j.gdata.2016.08.003
Okada, Taketo, et al. "Comparative analysis of transcriptomes in aerial stems and roots of Ephedra sinica based on high-throughput mRNA sequencing." Genomics data vol. 10 (2016): 4-11. doi: https://doi.org/10.1016/j.gdata.2016.08.003
Okada T, Takahashi H, Suzuki Y, Sugano S, Noji M, Kenmoku H, Toyota M, Kanaya S, Kawahara N, Asakawa Y, Sekita S. Comparative analysis of transcriptomes in aerial stems and roots of Ephedra sinica based on high-throughput mRNA sequencing. Genom Data. 2016 Aug 04;10:4-11. doi: 10.1016/j.gdata.2016.08.003. eCollection 2016 Dec. PMID: 27625990; PMCID: PMC5011178.
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Programmed cell death-ligand 1 expression and immunoscore in stage II and III non-small cell lung cancer patients receiving adjuvant chemotherapy.

Oncotarget

Ishii H, Azuma K, Kawahara A, Matsuo N, Tokito T, Kinoshita T, Yamada K, Sasada T, Akiba J, Hoshino T.
PMID: 28977890
Oncotarget. 2017 Jun 27;8(37):61618-61625. doi: 10.18632/oncotarget.18651. eCollection 2017 Sep 22.

Programmed cell death 1 (PD-1) receptor-ligand interaction is a major pathway that is often hijacked by tumors to suppress immune control. Immunoscore (IS), a combinational index of CD3 and CD8 tumor-infiltrating lymphocyte (TIL) density in the tumor's center and...

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Ishii H, Azuma K, Kawahara A, et al. Programmed cell death-ligand 1 expression and immunoscore in stage II and III non-small cell lung cancer patients receiving adjuvant chemotherapy. Oncotarget. 2017;8(37):61618-61625doi: 10.18632/oncotarget.18651.
Ishii, H., Azuma, K., Kawahara, A., Matsuo, N., Tokito, T., Kinoshita, T., Yamada, K., Sasada, T., Akiba, J., & Hoshino, T. (2017). Programmed cell death-ligand 1 expression and immunoscore in stage II and III non-small cell lung cancer patients receiving adjuvant chemotherapy. Oncotarget, 8(37), 61618-61625. https://doi.org/10.18632/oncotarget.18651
Ishii, Hidenobu, et al. "Programmed cell death-ligand 1 expression and immunoscore in stage II and III non-small cell lung cancer patients receiving adjuvant chemotherapy." Oncotarget vol. 8,37 (2017): 61618-61625. doi: https://doi.org/10.18632/oncotarget.18651
Ishii H, Azuma K, Kawahara A, Matsuo N, Tokito T, Kinoshita T, Yamada K, Sasada T, Akiba J, Hoshino T. Programmed cell death-ligand 1 expression and immunoscore in stage II and III non-small cell lung cancer patients receiving adjuvant chemotherapy. Oncotarget. 2017 Jun 27;8(37):61618-61625. doi: 10.18632/oncotarget.18651. eCollection 2017 Sep 22. PMID: 28977890; PMCID: PMC5617450.
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The psychoactive drug 25B-NBOMe recapitulates rhabdomyolysis in zebrafish larvae.

Forensic toxicology

Kawahara G, Maeda H, Kikura-Hanajiri R, Yoshida KI, Hayashi YK.
PMID: 28890736
Forensic Toxicol. 2017;35(2):369-375. doi: 10.1007/s11419-017-0366-9. Epub 2017 Apr 18.

No abstract available.

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Kawahara G, Maeda H, Kikura-Hanajiri R, et al. The psychoactive drug 25B-NBOMe recapitulates rhabdomyolysis in zebrafish larvae. Forensic Toxicol. 2017;35(2):369-375doi: 10.1007/s11419-017-0366-9.
Kawahara, G., Maeda, H., Kikura-Hanajiri, R., Yoshida, K. I., & Hayashi, Y. K. (2017). The psychoactive drug 25B-NBOMe recapitulates rhabdomyolysis in zebrafish larvae. Forensic toxicology, 35(2), 369-375. https://doi.org/10.1007/s11419-017-0366-9
Kawahara, Genri, et al. "The psychoactive drug 25B-NBOMe recapitulates rhabdomyolysis in zebrafish larvae." Forensic toxicology vol. 35,2 (2017): 369-375. doi: https://doi.org/10.1007/s11419-017-0366-9
Kawahara G, Maeda H, Kikura-Hanajiri R, Yoshida KI, Hayashi YK. The psychoactive drug 25B-NBOMe recapitulates rhabdomyolysis in zebrafish larvae. Forensic Toxicol. 2017;35(2):369-375. doi: 10.1007/s11419-017-0366-9. Epub 2017 Apr 18. PMID: 28890736; PMCID: PMC5552826.
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Data on the effect of .

Data in brief

Miyamoto Y, Torii T, Kawahara K, Inoue M, Morimoto T, Yamamoto M, Yamauchi J.
PMID: 28480322
Data Brief. 2017 Apr 15;12:313-319. doi: 10.1016/j.dib.2017.04.014. eCollection 2017 Jun.

Mature Schwann cells, the peripheral nervous system (PNS) glial cells, have two major roles for neuronal axons (Bunge, 1993) [1]. For large diameter axons, Schwann cells form myelin sheaths with multiple layers. For small diameter axons, they form Remak...

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Miyamoto Y, Torii T, Kawahara K, et al. Data on the effect of . Data Brief. 2017;12:313-319doi: 10.1016/j.dib.2017.04.014.
Miyamoto, Y., Torii, T., Kawahara, K., Inoue, M., Morimoto, T., Yamamoto, M., & Yamauchi, J. (2017). Data on the effect of . Data in brief, 12313-319. https://doi.org/10.1016/j.dib.2017.04.014
Miyamoto, Yuki, et al. "Data on the effect of ." Data in brief vol. 12 (2017): 313-319. doi: https://doi.org/10.1016/j.dib.2017.04.014
Miyamoto Y, Torii T, Kawahara K, Inoue M, Morimoto T, Yamamoto M, Yamauchi J. Data on the effect of . Data Brief. 2017 Apr 15;12:313-319. doi: 10.1016/j.dib.2017.04.014. eCollection 2017 Jun. PMID: 28480322; PMCID: PMC5407571.
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Dystrophic muscle improvement in zebrafish via increased heme oxygenase signaling.

Human molecular genetics

Kawahara G, Gasperini MJ, Myers JA, Widrick JJ, Eran A, Serafini PR, Alexander MS, Pletcher MT, Morris CA, Kunkel LM.
PMID: 26034133
Hum Mol Genet. 2015 Aug 01;24(15):4480-1. doi: 10.1093/hmg/ddv169. Epub 2015 Jun 01.

No abstract available.

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Kawahara G, Gasperini MJ, Myers JA, et al. Dystrophic muscle improvement in zebrafish via increased heme oxygenase signaling. Hum Mol Genet. 2015;24(15):4480-1doi: 10.1093/hmg/ddv169.
Kawahara, G., Gasperini, M. J., Myers, J. A., Widrick, J. J., Eran, A., Serafini, P. R., Alexander, M. S., Pletcher, M. T., Morris, C. A., & Kunkel, L. M. (2015). Dystrophic muscle improvement in zebrafish via increased heme oxygenase signaling. Human molecular genetics, 24(15), 4480-1. https://doi.org/10.1093/hmg/ddv169
Kawahara, Genri, et al. "Dystrophic muscle improvement in zebrafish via increased heme oxygenase signaling." Human molecular genetics vol. 24,15 (2015): 4480-1. doi: https://doi.org/10.1093/hmg/ddv169
Kawahara G, Gasperini MJ, Myers JA, Widrick JJ, Eran A, Serafini PR, Alexander MS, Pletcher MT, Morris CA, Kunkel LM. Dystrophic muscle improvement in zebrafish via increased heme oxygenase signaling. Hum Mol Genet. 2015 Aug 01;24(15):4480-1. doi: 10.1093/hmg/ddv169. Epub 2015 Jun 01. PMID: 26034133; PMCID: PMC5266494.
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Effects of Okadaic Acid on Embryonic Development of the Starfish Asterina pectinifera.

Bioscience, biotechnology, and biochemistry

Ikegami S, Kajiyama N, Ozaki Y, Ooe Y, Shimizu T, Kasahara T, Uemura D, Shioda M.
PMID: 27286370
Biosci Biotechnol Biochem. 1992 Jan;56(7):1007-11. doi: 10.1271/bbb.56.1007.

The effects of okadaic acid, an inhibitor of protein phosphatases types 1 and 2A, from the sponge Halichondria okadai Kadota, on the embryonic development of the starfish Asterina pectinifera, were investigated. When cultured in okadaic acid from fertilization, the...

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Ikegami S, Kajiyama N, Ozaki Y, et al. Effects of Okadaic Acid on Embryonic Development of the Starfish Asterina pectinifera. Biosci Biotechnol Biochem. 1992;56(7):1007-11doi: 10.1271/bbb.56.1007.
Ikegami, S., Kajiyama, N., Ozaki, Y., Ooe, Y., Shimizu, T., Kasahara, T., Uemura, D., & Shioda, M. (1992). Effects of Okadaic Acid on Embryonic Development of the Starfish Asterina pectinifera. Bioscience, biotechnology, and biochemistry, 56(7), 1007-11. https://doi.org/10.1271/bbb.56.1007
Ikegami, S, et al. "Effects of Okadaic Acid on Embryonic Development of the Starfish Asterina pectinifera." Bioscience, biotechnology, and biochemistry vol. 56,7 (1992): 1007-11. doi: https://doi.org/10.1271/bbb.56.1007
Ikegami S, Kajiyama N, Ozaki Y, Ooe Y, Shimizu T, Kasahara T, Uemura D, Shioda M. Effects of Okadaic Acid on Embryonic Development of the Starfish Asterina pectinifera. Biosci Biotechnol Biochem. 1992 Jan;56(7):1007-11. doi: 10.1271/bbb.56.1007. PMID: 27286370.
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A functional mesoporous ionic crystal based on polyoxometalate.

Dalton transactions (Cambridge, England : 2003)

Kawahara R, Niinomi K, Kondo JN, Hibino M, Mizuno N, Uchida S.
PMID: 26804446
Dalton Trans. 2016 Feb 21;45(7):2805-9. doi: 10.1039/c5dt04556h. Epub 2016 Jan 25.

A mesoporous ionic crystal is synthesized with a polyoxometalate and a macrocation with polar cyano groups. The compound possesses one-dimensional mesopores with an opening of 3.0 × 2.0 nm. The compound shows high proton conductivity and catalytic activity, which...

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Kawahara R, Niinomi K, Kondo JN, et al. A functional mesoporous ionic crystal based on polyoxometalate. Dalton Trans. 2016;45(7):2805-9doi: 10.1039/c5dt04556h.
Kawahara, R., Niinomi, K., Kondo, J. N., Hibino, M., Mizuno, N., & Uchida, S. (2016). A functional mesoporous ionic crystal based on polyoxometalate. Dalton transactions (Cambridge, England : 2003), 45(7), 2805-9. https://doi.org/10.1039/c5dt04556h
Kawahara, Ryosuke, et al. "A functional mesoporous ionic crystal based on polyoxometalate." Dalton transactions (Cambridge, England : 2003) vol. 45,7 (2016): 2805-9. doi: https://doi.org/10.1039/c5dt04556h
Kawahara R, Niinomi K, Kondo JN, Hibino M, Mizuno N, Uchida S. A functional mesoporous ionic crystal based on polyoxometalate. Dalton Trans. 2016 Feb 21;45(7):2805-9. doi: 10.1039/c5dt04556h. Epub 2016 Jan 25. PMID: 26804446.
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Direct intrahepatic portocaval shunt for refractory hepatic hydrothorax: a case report.

Acute medicine & surgery

Kawahara Y, Tanaka Y, Isoi N, Hatanaka K, Yamada K, Yamamoto M, Okamura T, Kaji T, Sakamoto T, Saitoh D, Ikeuchi H.
PMID: 29123879
Acute Med Surg. 2016 Dec 09;4(3):306-310. doi: 10.1002/ams2.257. eCollection 2017 Jul.

CASE: A 64-year-old man with complaints of dyspnea and orthopnea secondary to a hepatic hydrothorax refractory to diuretic medication underwent the transjugular intrahepatic portosystemic shunt (TIPS) procedure to decrease the portal vein pressure. The TIPS procedure failed due to...

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Kawahara Y, Tanaka Y, Isoi N, et al. Direct intrahepatic portocaval shunt for refractory hepatic hydrothorax: a case report. Acute Med Surg. 2016;4(3):306-310doi: 10.1002/ams2.257.
Kawahara, Y., Tanaka, Y., Isoi, N., Hatanaka, K., Yamada, K., Yamamoto, M., Okamura, T., Kaji, T., Sakamoto, T., Saitoh, D., & Ikeuchi, H. (2017). Direct intrahepatic portocaval shunt for refractory hepatic hydrothorax: a case report. Acute medicine & surgery, 4(3), 306-310. https://doi.org/10.1002/ams2.257
Kawahara, Yo, et al. "Direct intrahepatic portocaval shunt for refractory hepatic hydrothorax: a case report." Acute medicine & surgery vol. 4,3 (2017): 306-310. doi: https://doi.org/10.1002/ams2.257
Kawahara Y, Tanaka Y, Isoi N, Hatanaka K, Yamada K, Yamamoto M, Okamura T, Kaji T, Sakamoto T, Saitoh D, Ikeuchi H. Direct intrahepatic portocaval shunt for refractory hepatic hydrothorax: a case report. Acute Med Surg. 2016 Dec 09;4(3):306-310. doi: 10.1002/ams2.257. eCollection 2017 Jul. PMID: 29123879; PMCID: PMC5674466.
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A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report.

Journal of thoracic disease

Shiroyama T, Tamiya M, Hayama M, Nishihara T, Nishida T, Tanaka A, Morishita N, Suzuki H, Okamoto N, Kawahara K, Hirashima T.
PMID: 27162697
J Thorac Dis. 2016 May;8(5):E345-8. doi: 10.21037/jtd.2016.03.43.

An 87-year-old man with postoperative recurrent lung adenocarcinoma was treated with gefitinib. At the beginning of treatment, surgically resected archived tumor tissue revealed a deletion in exon 19 of the EGFR gene, but no ALK gene rearrangement. After gefitinib...

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Shiroyama T, Tamiya M, Hayama M, et al. A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. J Thorac Dis. 2016;8(5):E345-8doi: 10.21037/jtd.2016.03.43.
Shiroyama, T., Tamiya, M., Hayama, M., Nishihara, T., Nishida, T., Tanaka, A., Morishita, N., Suzuki, H., Okamoto, N., Kawahara, K., & Hirashima, T. (2016). A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. Journal of thoracic disease, 8(5), E345-8. https://doi.org/10.21037/jtd.2016.03.43
Shiroyama, Takayuki, et al. "A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report." Journal of thoracic disease vol. 8,5 (2016): E345-8. doi: https://doi.org/10.21037/jtd.2016.03.43
Shiroyama T, Tamiya M, Hayama M, Nishihara T, Nishida T, Tanaka A, Morishita N, Suzuki H, Okamoto N, Kawahara K, Hirashima T. A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. J Thorac Dis. 2016 May;8(5):E345-8. doi: 10.21037/jtd.2016.03.43. PMID: 27162697; PMCID: PMC4842787.
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Allele-specific Gene Silencing of Mutant mRNA Restores Cellular Function in Ullrich Congenital Muscular Dystrophy Fibroblasts.

Molecular therapy. Nucleic acids

Noguchi S, Ogawa M, Kawahara G, Malicdan MC, Nishino I.
PMID: 24959844
Mol Ther Nucleic Acids. 2014 Jun 24;3:e171. doi: 10.1038/mtna.2014.22.

Ullrich congenital muscular dystrophy (UCMD) is an inherited muscle disorder characterized clinically by muscle weakness, distal joint hyperlaxity, and proximal joint contractures. Sporadic and recessive mutations in the three collagen VI genes, COL6A1, COL6A2, and COL6A3, are reported to...

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Noguchi S, Ogawa M, Kawahara G, et al. Allele-specific Gene Silencing of Mutant mRNA Restores Cellular Function in Ullrich Congenital Muscular Dystrophy Fibroblasts. Mol Ther Nucleic Acids. 2014;3:e171doi: 10.1038/mtna.2014.22.
Noguchi, S., Ogawa, M., Kawahara, G., Malicdan, M. C., & Nishino, I. (2014). Allele-specific Gene Silencing of Mutant mRNA Restores Cellular Function in Ullrich Congenital Muscular Dystrophy Fibroblasts. Molecular therapy. Nucleic acids, 3e171. https://doi.org/10.1038/mtna.2014.22
Noguchi, Satoru, et al. "Allele-specific Gene Silencing of Mutant mRNA Restores Cellular Function in Ullrich Congenital Muscular Dystrophy Fibroblasts." Molecular therapy. Nucleic acids vol. 3 (2014): e171. doi: https://doi.org/10.1038/mtna.2014.22
Noguchi S, Ogawa M, Kawahara G, Malicdan MC, Nishino I. Allele-specific Gene Silencing of Mutant mRNA Restores Cellular Function in Ullrich Congenital Muscular Dystrophy Fibroblasts. Mol Ther Nucleic Acids. 2014 Jun 24;3:e171. doi: 10.1038/mtna.2014.22. PMID: 24959844; PMCID: PMC4078762.
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Showing 1 to 12 of 1151 entries