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Hierarchical Analysis of Self-Assembled PEGylated Hexaphenylalanine Photoluminescent Nanostructures.

Chemistry (Weinheim an der Bergstrasse, Germany)

Diaferia C, Sibillano T, Balasco N, Giannini C, Roviello V, Vitagliano L, Morelli G, Accardo A.
PMID: 27706842
Chemistry. 2016 Nov 07;22(46):16586-16597. doi: 10.1002/chem.201604107. Epub 2016 Oct 05.

Despite the growing literature about diphenylalanine-based peptide materials, it still remains a challenge to delineate the theoretical insight into peptide nanostructure formation and the structural features that could permit materials with enhanced properties to be engineered. Herein, we report...

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Diaferia C, Sibillano T, Balasco N, et al. Hierarchical Analysis of Self-Assembled PEGylated Hexaphenylalanine Photoluminescent Nanostructures. Chemistry. 2016;22(46):16586-16597doi: 10.1002/chem.201604107.
Diaferia, C., Sibillano, T., Balasco, N., Giannini, C., Roviello, V., Vitagliano, L., Morelli, G., & Accardo, A. (2016). Hierarchical Analysis of Self-Assembled PEGylated Hexaphenylalanine Photoluminescent Nanostructures. Chemistry (Weinheim an der Bergstrasse, Germany), 22(46), 16586-16597. https://doi.org/10.1002/chem.201604107
Diaferia, Carlo, et al. "Hierarchical Analysis of Self-Assembled PEGylated Hexaphenylalanine Photoluminescent Nanostructures." Chemistry (Weinheim an der Bergstrasse, Germany) vol. 22,46 (2016): 16586-16597. doi: https://doi.org/10.1002/chem.201604107
Diaferia C, Sibillano T, Balasco N, Giannini C, Roviello V, Vitagliano L, Morelli G, Accardo A. Hierarchical Analysis of Self-Assembled PEGylated Hexaphenylalanine Photoluminescent Nanostructures. Chemistry. 2016 Nov 07;22(46):16586-16597. doi: 10.1002/chem.201604107. Epub 2016 Oct 05. PMID: 27706842.
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[No title available]

Journal of investigative medicine high impact case reports

Wallace JA, Hussain J, Unzueta A, Morelli G.
PMID: 28890902
J Investig Med High Impact Case Rep. 2017 Aug 30;5(3):2324709617726103. doi: 10.1177/2324709617726103. eCollection 2017.

A 58-year-old male with nonalcoholic steatohepatitis cirrhosis presents with right lower extremity cellulitis, abdominal tenderness, and severe sepsis after sustaining puncture injury from a cactus on a property with feral cats. Blood cultures and diagnostic paracentesis were consistent with...

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Wallace JA, Hussain J, Unzueta A, et al. . J Investig Med High Impact Case Rep. 2017;5(3):2324709617726103doi: 10.1177/2324709617726103.
Wallace, J. A., Hussain, J., Unzueta, A., & Morelli, G. (2017). . Journal of investigative medicine high impact case reports, 5(3), 2324709617726103. https://doi.org/10.1177/2324709617726103
Wallace, Jodi-Anne, et al. "." Journal of investigative medicine high impact case reports vol. 5,3 (2017): 2324709617726103. doi: https://doi.org/10.1177/2324709617726103
Wallace JA, Hussain J, Unzueta A, Morelli G. . J Investig Med High Impact Case Rep. 2017 Aug 30;5(3):2324709617726103. doi: 10.1177/2324709617726103. eCollection 2017. PMID: 28890902; PMCID: PMC5580853.
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Myositis autoantibodies and clinical phenotypes.

Auto- immunity highlights

Ghirardello A, Borella E, Beggio M, Franceschini F, Fredi M, Doria A.
PMID: 26000158
Auto Immun Highlights. 2014 Aug 23;5(3):69-75. doi: 10.1007/s13317-014-0060-4. eCollection 2014 Dec.

Autoantibodies are powerful diagnostic tools in idiopathic inflammatory myopathies, especially for confirming the diagnosis and contributing to the definition of disease subsets. They are present in over 80 % of patients with immuno-mediated myositis and directed towards ubiquitously expressed...

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Ghirardello A, Borella E, Beggio M, et al. Myositis autoantibodies and clinical phenotypes. Auto Immun Highlights. 2014;5(3):69-75doi: 10.1007/s13317-014-0060-4.
Ghirardello, A., Borella, E., Beggio, M., Franceschini, F., Fredi, M., & Doria, A. (2014). Myositis autoantibodies and clinical phenotypes. Auto- immunity highlights, 5(3), 69-75. https://doi.org/10.1007/s13317-014-0060-4
Ghirardello, Anna, et al. "Myositis autoantibodies and clinical phenotypes." Auto- immunity highlights vol. 5,3 (2014): 69-75. doi: https://doi.org/10.1007/s13317-014-0060-4
Ghirardello A, Borella E, Beggio M, Franceschini F, Fredi M, Doria A. Myositis autoantibodies and clinical phenotypes. Auto Immun Highlights. 2014 Aug 23;5(3):69-75. doi: 10.1007/s13317-014-0060-4. eCollection 2014 Dec. PMID: 26000158; PMCID: PMC4386580.
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Conservation of AtTZF1, AtTZF2, and AtTZF3 homolog gene regulation by salt stress in evolutionarily distant plant species.

Frontiers in plant science

D'Orso F, De Leonardis AM, Salvi S, Gadaleta A, Ruberti I, Cattivelli L, Morelli G, Mastrangelo AM.
PMID: 26136754
Front Plant Sci. 2015 Jun 16;6:394. doi: 10.3389/fpls.2015.00394. eCollection 2015.

Arginine-rich tandem zinc-finger proteins (RR-TZF) participate in a wide range of plant developmental processes and adaptive responses to abiotic stress, such as cold, salt, and drought. This study investigates the conservation of the genes AtTZF1-5 at the level of...

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D'Orso F, De Leonardis AM, Salvi S, et al. Conservation of AtTZF1, AtTZF2, and AtTZF3 homolog gene regulation by salt stress in evolutionarily distant plant species. Front Plant Sci. 2015;6:394doi: 10.3389/fpls.2015.00394.
D'Orso, F., De Leonardis, A. M., Salvi, S., Gadaleta, A., Ruberti, I., Cattivelli, L., Morelli, G., & Mastrangelo, A. M. (2015). Conservation of AtTZF1, AtTZF2, and AtTZF3 homolog gene regulation by salt stress in evolutionarily distant plant species. Frontiers in plant science, 6394. https://doi.org/10.3389/fpls.2015.00394
D'Orso, Fabio, et al. "Conservation of AtTZF1, AtTZF2, and AtTZF3 homolog gene regulation by salt stress in evolutionarily distant plant species." Frontiers in plant science vol. 6 (2015): 394. doi: https://doi.org/10.3389/fpls.2015.00394
D'Orso F, De Leonardis AM, Salvi S, Gadaleta A, Ruberti I, Cattivelli L, Morelli G, Mastrangelo AM. Conservation of AtTZF1, AtTZF2, and AtTZF3 homolog gene regulation by salt stress in evolutionarily distant plant species. Front Plant Sci. 2015 Jun 16;6:394. doi: 10.3389/fpls.2015.00394. eCollection 2015. PMID: 26136754; PMCID: PMC4468379.
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Genotoxic evaluation of the River Paranaíba hydrographic basin in Monte Carmelo, MG, Brazil, by the Tradescantia micronucleus.

Genetics and molecular biology

Campos CF, Pereira BB, Campos-Junior EO, Sousa EF, Souto HN, Morelli S.
PMID: 26692158
Genet Mol Biol. 2015 Dec;38(4):507-12. doi: 10.1590/S1415-475738420150117.

Pollutants have adverse effects on human health and on other organisms that inhabit or use water resources. The aim of the present study was to assess the environmental quality of three watercourses in Monte Carmelo, MG, Brazil, using the...

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Campos CF, Pereira BB, Campos-Junior EO, et al. Genotoxic evaluation of the River Paranaíba hydrographic basin in Monte Carmelo, MG, Brazil, by the Tradescantia micronucleus. Genet Mol Biol. 2015;38(4):507-12doi: 10.1590/S1415-475738420150117.
Campos, C. F., Pereira, B. B., Campos-Junior, E. O., Sousa, E. F., Souto, H. N., & Morelli, S. (2015). Genotoxic evaluation of the River Paranaíba hydrographic basin in Monte Carmelo, MG, Brazil, by the Tradescantia micronucleus. Genetics and molecular biology, 38(4), 507-12. https://doi.org/10.1590/S1415-475738420150117
Campos, Carlos F, et al. "Genotoxic evaluation of the River Paranaíba hydrographic basin in Monte Carmelo, MG, Brazil, by the Tradescantia micronucleus." Genetics and molecular biology vol. 38,4 (2015): 507-12. doi: https://doi.org/10.1590/S1415-475738420150117
Campos CF, Pereira BB, Campos-Junior EO, Sousa EF, Souto HN, Morelli S. Genotoxic evaluation of the River Paranaíba hydrographic basin in Monte Carmelo, MG, Brazil, by the Tradescantia micronucleus. Genet Mol Biol. 2015 Dec;38(4):507-12. doi: 10.1590/S1415-475738420150117. PMID: 26692158; PMCID: PMC4763326.
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Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72.

Frontiers in molecular neuroscience

Budini M, Buratti E, Morselli E, Criollo A.
PMID: 28611593
Front Mol Neurosci. 2017 May 30;10:170. doi: 10.3389/fnmol.2017.00170. eCollection 2017.

Autophagy is a catabolic mechanism where intracellular material is degraded by vesicular structures called autophagolysosomes. Autophagy is necessary to maintain the normal function of the central nervous system (CNS), avoiding the accumulation of misfolded and aggregated proteins. Consistently, impaired...

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Budini M, Buratti E, Morselli E, et al. Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72. Front Mol Neurosci. 2017;10:170doi: 10.3389/fnmol.2017.00170.
Budini, M., Buratti, E., Morselli, E., & Criollo, A. (2017). Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72. Frontiers in molecular neuroscience, 10170. https://doi.org/10.3389/fnmol.2017.00170
Budini, Mauricio, et al. "Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72." Frontiers in molecular neuroscience vol. 10 (2017): 170. doi: https://doi.org/10.3389/fnmol.2017.00170
Budini M, Buratti E, Morselli E, Criollo A. Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72. Front Mol Neurosci. 2017 May 30;10:170. doi: 10.3389/fnmol.2017.00170. eCollection 2017. PMID: 28611593; PMCID: PMC5447761.
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Immune cells subpopulations in cerebrospinal fluid and peripheral blood of patients with Aneurysmal Subarachnoid Hemorrhage.

SpringerPlus

Moraes L, Grille S, Morelli P, Mila R, Trias N, Brugnini A, LLuberas N, Biestro A, Lens D.
PMID: 25977890
Springerplus. 2015 Apr 23;4:195. doi: 10.1186/s40064-015-0970-2. eCollection 2015.

BACKGROUND: There is growing evidence supporting the role of inflammation in aneurysmal subarachnoid hemorrhage (aSAH) pathophysiology and it is of great interest to elucidate which immune mechanisms are involved.METHODS: 12 aSAH patients and 28 healthy controls were enrolled prospectively....

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Moraes L, Grille S, Morelli P, et al. Immune cells subpopulations in cerebrospinal fluid and peripheral blood of patients with Aneurysmal Subarachnoid Hemorrhage. Springerplus. 2015;4:195doi: 10.1186/s40064-015-0970-2.
Moraes, L., Grille, S., Morelli, P., Mila, R., Trias, N., Brugnini, A., LLuberas, N., Biestro, A., & Lens, D. (2015). Immune cells subpopulations in cerebrospinal fluid and peripheral blood of patients with Aneurysmal Subarachnoid Hemorrhage. SpringerPlus, 4195. https://doi.org/10.1186/s40064-015-0970-2
Moraes, Leandro, et al. "Immune cells subpopulations in cerebrospinal fluid and peripheral blood of patients with Aneurysmal Subarachnoid Hemorrhage." SpringerPlus vol. 4 (2015): 195. doi: https://doi.org/10.1186/s40064-015-0970-2
Moraes L, Grille S, Morelli P, Mila R, Trias N, Brugnini A, LLuberas N, Biestro A, Lens D. Immune cells subpopulations in cerebrospinal fluid and peripheral blood of patients with Aneurysmal Subarachnoid Hemorrhage. Springerplus. 2015 Apr 23;4:195. doi: 10.1186/s40064-015-0970-2. eCollection 2015. PMID: 25977890; PMCID: PMC4414856.
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A commentary on "Differentiation of pluripotent stem cells into striatal projection neurons: a pure MSN fate may not be sufficient".

Frontiers in cellular neuroscience

Porfirio B, Morelli A, Conti R, Vannelli GB, Gallina P.
PMID: 26029049
Front Cell Neurosci. 2015 May 12;9:177. doi: 10.3389/fncel.2015.00177. eCollection 2015.

No abstract available.

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Porfirio B, Morelli A, Conti R, et al. A commentary on "Differentiation of pluripotent stem cells into striatal projection neurons: a pure MSN fate may not be sufficient". Front Cell Neurosci. 2015;9:177doi: 10.3389/fncel.2015.00177.
Porfirio, B., Morelli, A., Conti, R., Vannelli, G. B., & Gallina, P. (2015). A commentary on "Differentiation of pluripotent stem cells into striatal projection neurons: a pure MSN fate may not be sufficient". Frontiers in cellular neuroscience, 9177. https://doi.org/10.3389/fncel.2015.00177
Porfirio, Berardino, et al. "A commentary on "Differentiation of pluripotent stem cells into striatal projection neurons: a pure MSN fate may not be sufficient"." Frontiers in cellular neuroscience vol. 9 (2015): 177. doi: https://doi.org/10.3389/fncel.2015.00177
Porfirio B, Morelli A, Conti R, Vannelli GB, Gallina P. A commentary on "Differentiation of pluripotent stem cells into striatal projection neurons: a pure MSN fate may not be sufficient". Front Cell Neurosci. 2015 May 12;9:177. doi: 10.3389/fncel.2015.00177. eCollection 2015. PMID: 26029049; PMCID: PMC4428124.
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Chronic High Fat Diet Consumption Impairs Metabolic Health of Male Mice.

Inflammation and cell signaling

Morselli E, Criollo A, Rodriguez-Navas C, Clegg DJ.
PMID: 26046098
Inflamm Cell Signal. 2014;1(6):e561. doi: 10.14800/ics.561.

We show that chronic high fat diet (HFD) feeding affects the hypothalamus of male but not female mice. In our study we demonstrate that palmitic acid and sphingolipids accumulate in the central nervous system of HFD-fed males. Additionally, we...

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Morselli E, Criollo A, Rodriguez-Navas C, et al. Chronic High Fat Diet Consumption Impairs Metabolic Health of Male Mice. Inflamm Cell Signal. 2014;1(6):e561doi: 10.14800/ics.561.
Morselli, E., Criollo, A., Rodriguez-Navas, C., & Clegg, D. J. (2014). Chronic High Fat Diet Consumption Impairs Metabolic Health of Male Mice. Inflammation and cell signaling, 1(6), e561. https://doi.org/10.14800/ics.561
Morselli, Eugenia, et al. "Chronic High Fat Diet Consumption Impairs Metabolic Health of Male Mice." Inflammation and cell signaling vol. 1,6 (2014): e561. doi: https://doi.org/10.14800/ics.561
Morselli E, Criollo A, Rodriguez-Navas C, Clegg DJ. Chronic High Fat Diet Consumption Impairs Metabolic Health of Male Mice. Inflamm Cell Signal. 2014;1(6):e561. doi: 10.14800/ics.561. PMID: 26046098; PMCID: PMC4451571.
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Hyaluronic Acid and Its Composites as a Local Antimicrobial/Antiadhesive Barrier.

Journal of bone and joint infection

Romanò CL, De Vecchi E, Bortolin M, Morelli I, Drago L.
PMID: 28529865
J Bone Jt Infect. 2017 Jan 01;2(1):63-72. doi: 10.7150/jbji.17705. eCollection 2017.

Living in biofilms is probably the most common condition for bacteria and fungi and biofilm-related infections account for the majority of bacterial infectious diseases worldwide. Among others biofilm-related infections, those associated with implanted biomaterials have an enormous and still...

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Romanò CL, De Vecchi E, Bortolin M, et al. Hyaluronic Acid and Its Composites as a Local Antimicrobial/Antiadhesive Barrier. J Bone Jt Infect. 2017;2(1):63-72doi: 10.7150/jbji.17705.
Romanò, C. L., De Vecchi, E., Bortolin, M., Morelli, I., & Drago, L. (2017). Hyaluronic Acid and Its Composites as a Local Antimicrobial/Antiadhesive Barrier. Journal of bone and joint infection, 2(1), 63-72. https://doi.org/10.7150/jbji.17705
Romanò, C L, et al. "Hyaluronic Acid and Its Composites as a Local Antimicrobial/Antiadhesive Barrier." Journal of bone and joint infection vol. 2,1 (2017): 63-72. doi: https://doi.org/10.7150/jbji.17705
Romanò CL, De Vecchi E, Bortolin M, Morelli I, Drago L. Hyaluronic Acid and Its Composites as a Local Antimicrobial/Antiadhesive Barrier. J Bone Jt Infect. 2017 Jan 01;2(1):63-72. doi: 10.7150/jbji.17705. eCollection 2017. PMID: 28529865; PMCID: PMC5423572.
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Iatrogenic Coagulopathy and the Development of Posterior Reversible Encephalopathy Syndrome after L-asparaginase Chemotherapy.

Journal of clinical imaging science

Rota E, Vallisa D, Morelli N, Scagnelli P.
PMID: 27014503
J Clin Imaging Sci. 2016 Feb 26;6:7. doi: 10.4103/2156-7514.177553. eCollection 2016.

Posterior reversible encephalopathy syndrome (PRES) is a clinical and radiological syndrome mostly related to hypertension, eclampsia, renal failure, or to chemotherapy and/or immunosuppressive drugs. Although the PRES pathophysiology is multifactorial, hypertension and endothelial dysfunction are hypothesized to be the...

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Rota E, Vallisa D, Morelli N, et al. Iatrogenic Coagulopathy and the Development of Posterior Reversible Encephalopathy Syndrome after L-asparaginase Chemotherapy. J Clin Imaging Sci. 2016;6:7doi: 10.4103/2156-7514.177553.
Rota, E., Vallisa, D., Morelli, N., & Scagnelli, P. (2016). Iatrogenic Coagulopathy and the Development of Posterior Reversible Encephalopathy Syndrome after L-asparaginase Chemotherapy. Journal of clinical imaging science, 67. https://doi.org/10.4103/2156-7514.177553
Rota, Eugenia, et al. "Iatrogenic Coagulopathy and the Development of Posterior Reversible Encephalopathy Syndrome after L-asparaginase Chemotherapy." Journal of clinical imaging science vol. 6 (2016): 7. doi: https://doi.org/10.4103/2156-7514.177553
Rota E, Vallisa D, Morelli N, Scagnelli P. Iatrogenic Coagulopathy and the Development of Posterior Reversible Encephalopathy Syndrome after L-asparaginase Chemotherapy. J Clin Imaging Sci. 2016 Feb 26;6:7. doi: 10.4103/2156-7514.177553. eCollection 2016. PMID: 27014503; PMCID: PMC4785783.
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Correction: DMT1 iron uptake in the PNS: bridging the gap between injury and regeneration.

Metallomics : integrated biometal science

Martinez-Vivot R, Copello G, Leal MC, Piñero G, Usach V, Rozenszajn M, Morelli L, Setton-Avruj CP.
PMID: 26542333
Metallomics. 2015 Dec;7(12):1612. doi: 10.1039/c5mt90048d.

No abstract available.

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Martinez-Vivot R, Copello G, Leal MC, et al. Correction: DMT1 iron uptake in the PNS: bridging the gap between injury and regeneration. Metallomics. 2015;7(12):1612doi: 10.1039/c5mt90048d.
Martinez-Vivot, R., Copello, G., Leal, M. C., Piñero, G., Usach, V., Rozenszajn, M., Morelli, L., & Setton-Avruj, C. P. (2015). Correction: DMT1 iron uptake in the PNS: bridging the gap between injury and regeneration. Metallomics : integrated biometal science, 7(12), 1612. https://doi.org/10.1039/c5mt90048d
Martinez-Vivot, Rocío, et al. "Correction: DMT1 iron uptake in the PNS: bridging the gap between injury and regeneration." Metallomics : integrated biometal science vol. 7,12 (2015): 1612. doi: https://doi.org/10.1039/c5mt90048d
Martinez-Vivot R, Copello G, Leal MC, Piñero G, Usach V, Rozenszajn M, Morelli L, Setton-Avruj CP. Correction: DMT1 iron uptake in the PNS: bridging the gap between injury and regeneration. Metallomics. 2015 Dec;7(12):1612. doi: 10.1039/c5mt90048d. PMID: 26542333.
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