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Bladder and rectal incontinence without paraplegia or paraparesis after endovascular aneurysm repair.

Surgical case reports

Nishioka N, Kurimoto Y, Maruyama R, Ujihira K, Iba Y, Hatta E, Yamada A, Nakanishi K.
PMID: 26943687
Surg Case Rep. 2016 Dec;2(1):11. doi: 10.1186/s40792-016-0140-y. Epub 2016 Feb 11.

Spinal cord ischemia is a well-known potential complication of endovascular aneurysm repair (EVAR), and it is usually manifested by paraplegia or paraparesis. We describe a case in which spinal cord ischemia after EVAR presented by isolated bladder and rectal...

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Nishioka N, Kurimoto Y, Maruyama R, et al. Bladder and rectal incontinence without paraplegia or paraparesis after endovascular aneurysm repair. Surg Case Rep. 2016;2(1):11doi: 10.1186/s40792-016-0140-y.
Nishioka, N., Kurimoto, Y., Maruyama, R., Ujihira, K., Iba, Y., Hatta, E., Yamada, A., & Nakanishi, K. (2016). Bladder and rectal incontinence without paraplegia or paraparesis after endovascular aneurysm repair. Surgical case reports, 2(1), 11. https://doi.org/10.1186/s40792-016-0140-y
Nishioka, Naritomo, et al. "Bladder and rectal incontinence without paraplegia or paraparesis after endovascular aneurysm repair." Surgical case reports vol. 2,1 (2016): 11. doi: https://doi.org/10.1186/s40792-016-0140-y
Nishioka N, Kurimoto Y, Maruyama R, Ujihira K, Iba Y, Hatta E, Yamada A, Nakanishi K. Bladder and rectal incontinence without paraplegia or paraparesis after endovascular aneurysm repair. Surg Case Rep. 2016 Dec;2(1):11. doi: 10.1186/s40792-016-0140-y. Epub 2016 Feb 11. PMID: 26943687; PMCID: PMC4751105.
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Nintedanib: evidence for its therapeutic potential in idiopathic pulmonary fibrosis.

Core evidence

Inomata M, Nishioka Y, Azuma A.
PMID: 26346347
Core Evid. 2015 Aug 27;10:89-98. doi: 10.2147/CE.S82905. eCollection 2015.

Idiopathic pulmonary fibrosis (IPF) is a progressive disease with poor prognosis. The molecular mechanisms involved in the progression of IPF are not fully understood; however, the platelet-derived growth factor (PDGF)/PDGF receptor pathway is thought to play a critical role...

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Inomata M, Nishioka Y, Azuma A. Nintedanib: evidence for its therapeutic potential in idiopathic pulmonary fibrosis. Core Evid. 2015;10:89-98doi: 10.2147/CE.S82905.
Inomata, M., Nishioka, Y., & Azuma, A. (2015). Nintedanib: evidence for its therapeutic potential in idiopathic pulmonary fibrosis. Core evidence, 1089-98. https://doi.org/10.2147/CE.S82905
Inomata, Minoru, et al. "Nintedanib: evidence for its therapeutic potential in idiopathic pulmonary fibrosis." Core evidence vol. 10 (2015): 89-98. doi: https://doi.org/10.2147/CE.S82905
Inomata M, Nishioka Y, Azuma A. Nintedanib: evidence for its therapeutic potential in idiopathic pulmonary fibrosis. Core Evid. 2015 Aug 27;10:89-98. doi: 10.2147/CE.S82905. eCollection 2015. PMID: 26346347; PMCID: PMC4555978.
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SU-E-J-31: Direct Point Dose Verification Using IGRT for Actual Beam Delivery.

Medical physics

Matsubara K, Kohno R, Nishioka S, Shibuya T, Ariji T, Akimoto T, Saitoh H.
PMID: 28517591
Med Phys. 2012 Jun;39(6):3659. doi: 10.1118/1.4734864.

PURPOSE: In radiation therapy, treatment planning for patients is performed using pre-acquired CT images. However, many patients with head-and-neck (H&N) cancer have tumor shrinkage and/or weight loss during their treatment course. Daily positional error of patients also causes unexpected...

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Matsubara K, Kohno R, Nishioka S, et al. SU-E-J-31: Direct Point Dose Verification Using IGRT for Actual Beam Delivery. Med Phys. 2012;39(6):3659doi: 10.1118/1.4734864.
Matsubara, K., Kohno, R., Nishioka, S., Shibuya, T., Ariji, T., Akimoto, T., & Saitoh, H. (2012). SU-E-J-31: Direct Point Dose Verification Using IGRT for Actual Beam Delivery. Medical physics, 39(6), 3659. https://doi.org/10.1118/1.4734864
Matsubara, K, et al. "SU-E-J-31: Direct Point Dose Verification Using IGRT for Actual Beam Delivery." Medical physics vol. 39,6 (2012): 3659. doi: https://doi.org/10.1118/1.4734864
Matsubara K, Kohno R, Nishioka S, Shibuya T, Ariji T, Akimoto T, Saitoh H. SU-E-J-31: Direct Point Dose Verification Using IGRT for Actual Beam Delivery. Med Phys. 2012 Jun;39(6):3659. doi: 10.1118/1.4734864. PMID: 28517591.
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A top-down methodology for ultrafast tuning of nanosized zeolites.

Chemical communications (Cambridge, England)

Liu Z, Nomura N, Nishioka D, Hotta Y, Matsuo T, Oshima K, Yanaba Y, Yoshikawa T, Ohara K, Kohara S, Takewaki T, Okubo T, Wakihara T.
PMID: 26154841
Chem Commun (Camb). 2015 Aug 14;51(63):12567-70. doi: 10.1039/c5cc04542h.

We herein present a top-down methodology to prepare nanosized zeolites with tunable size by combining post-synthesis milling and fast recrystallization of several minutes (10 min for SSZ-13 and 5 min for AlPO4-5). A continuous-flow recrystallization process is demonstrated to...

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Liu Z, Nomura N, Nishioka D, et al. A top-down methodology for ultrafast tuning of nanosized zeolites. Chem Commun (Camb). 2015;51(63):12567-70doi: 10.1039/c5cc04542h.
Liu, Z., Nomura, N., Nishioka, D., Hotta, Y., Matsuo, T., Oshima, K., Yanaba, Y., Yoshikawa, T., Ohara, K., Kohara, S., Takewaki, T., Okubo, T., & Wakihara, T. (2015). A top-down methodology for ultrafast tuning of nanosized zeolites. Chemical communications (Cambridge, England), 51(63), 12567-70. https://doi.org/10.1039/c5cc04542h
Liu, Zhendong, et al. "A top-down methodology for ultrafast tuning of nanosized zeolites." Chemical communications (Cambridge, England) vol. 51,63 (2015): 12567-70. doi: https://doi.org/10.1039/c5cc04542h
Liu Z, Nomura N, Nishioka D, Hotta Y, Matsuo T, Oshima K, Yanaba Y, Yoshikawa T, Ohara K, Kohara S, Takewaki T, Okubo T, Wakihara T. A top-down methodology for ultrafast tuning of nanosized zeolites. Chem Commun (Camb). 2015 Aug 14;51(63):12567-70. doi: 10.1039/c5cc04542h. PMID: 26154841.
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Involvement of UTR-dependent gene expression in the maintenance of cancer stem cell like phenotypes.

Oncology letters

Yasuda M, Hatanaka T, Shirato H, Nishioka T.
PMID: 26722307
Oncol Lett. 2015 Nov;10(5):3171-3176. doi: 10.3892/ol.2015.3688. Epub 2015 Sep 09.

The present study demonstrated the acquisition of additional malignant characteristics in irradiated mouse fibrosarcoma cells compared with the parent cells. Several reporter assays indicated that hypoxia-inducible factor (HIF)-1α, activator protein-1 and Ets-dependent transcription were activated in irradiated cells. The

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Yasuda M, Hatanaka T, Shirato H, et al. Involvement of UTR-dependent gene expression in the maintenance of cancer stem cell like phenotypes. Oncol Lett. 2015;10(5):3171-3176doi: 10.3892/ol.2015.3688.
Yasuda, M., Hatanaka, T., Shirato, H., & Nishioka, T. (2015). Involvement of UTR-dependent gene expression in the maintenance of cancer stem cell like phenotypes. Oncology letters, 10(5), 3171-3176. https://doi.org/10.3892/ol.2015.3688
Yasuda, Motoaki, et al. "Involvement of UTR-dependent gene expression in the maintenance of cancer stem cell like phenotypes." Oncology letters vol. 10,5 (2015): 3171-3176. doi: https://doi.org/10.3892/ol.2015.3688
Yasuda M, Hatanaka T, Shirato H, Nishioka T. Involvement of UTR-dependent gene expression in the maintenance of cancer stem cell like phenotypes. Oncol Lett. 2015 Nov;10(5):3171-3176. doi: 10.3892/ol.2015.3688. Epub 2015 Sep 09. PMID: 26722307; PMCID: PMC4665853.
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Generation of 300 nm bandwidth 0.5 mJ pulses near 1 μm in a single stage gas filled hollow core fiber.

Optics express

Guo X, Tokita S, Yoshii K, Nishioka H, Kawanaka J.
PMID: 29041523
Opt Express. 2017 Sep 04;25(18):21171-21179. doi: 10.1364/OE.25.021171.

A simple and compact spectral-broadening system is presented that is based on a single-stage statically pressurized Ar filled hollow core fiber. By optimizing the inner diameter of the hollow core fiber, a bandwidth of 300 nm is obtained. This...

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Guo X, Tokita S, Yoshii K, et al. Generation of 300 nm bandwidth 0.5 mJ pulses near 1 μm in a single stage gas filled hollow core fiber. Opt Express. 2017;25(18):21171-21179doi: 10.1364/OE.25.021171.
Guo, X., Tokita, S., Yoshii, K., Nishioka, H., & Kawanaka, J. (2017). Generation of 300 nm bandwidth 0.5 mJ pulses near 1 μm in a single stage gas filled hollow core fiber. Optics express, 25(18), 21171-21179. https://doi.org/10.1364/OE.25.021171
Guo, Xiaoyang, et al. "Generation of 300 nm bandwidth 0.5 mJ pulses near 1 μm in a single stage gas filled hollow core fiber." Optics express vol. 25,18 (2017): 21171-21179. doi: https://doi.org/10.1364/OE.25.021171
Guo X, Tokita S, Yoshii K, Nishioka H, Kawanaka J. Generation of 300 nm bandwidth 0.5 mJ pulses near 1 μm in a single stage gas filled hollow core fiber. Opt Express. 2017 Sep 04;25(18):21171-21179. doi: 10.1364/OE.25.021171. PMID: 29041523.
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The first Japanese case of central precocious puberty with a novel .

Human genome variation

Nishioka J, Shima H, Fukami M, Yatsuga S, Matsumoto T, Ushijima K, Kitamura M, Koga Y.
PMID: 28546864
Hum Genome Var. 2017 May 18;4:17017. doi: 10.1038/hgv.2017.17. eCollection 2017.

No abstract available.

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Nishioka J, Shima H, Fukami M, et al. The first Japanese case of central precocious puberty with a novel . Hum Genome Var. 2017;4:17017doi: 10.1038/hgv.2017.17.
Nishioka, J., Shima, H., Fukami, M., Yatsuga, S., Matsumoto, T., Ushijima, K., Kitamura, M., & Koga, Y. (2017). The first Japanese case of central precocious puberty with a novel . Human genome variation, 417017. https://doi.org/10.1038/hgv.2017.17
Nishioka, Junko, et al. "The first Japanese case of central precocious puberty with a novel ." Human genome variation vol. 4 (2017): 17017. doi: https://doi.org/10.1038/hgv.2017.17
Nishioka J, Shima H, Fukami M, Yatsuga S, Matsumoto T, Ushijima K, Kitamura M, Koga Y. The first Japanese case of central precocious puberty with a novel . Hum Genome Var. 2017 May 18;4:17017. doi: 10.1038/hgv.2017.17. eCollection 2017. PMID: 28546864; PMCID: PMC5435957.
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A case of severe ganciclovir-induced encephalopathy.

Case reports in neurology

Sakamoto H, Hirano M, Nose K, Ueno S, Oki T, Sugimoto K, Nishioka T, Kusunoki S, Nakamura Y.
PMID: 24403897
Case Rep Neurol. 2013 Oct 05;5(3):183-6. doi: 10.1159/000355638. eCollection 2013.

BACKGROUND: Ganciclovir, a drug against cytomegalovirus (CMV) infection, is generally well tolerated, but can cause neurotoxicity such as encephalopathy. Although ganciclovir-induced encephalopathy has been described in several reports, a literature search revealed that ganciclovir concentrations in the blood or...

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Sakamoto H, Hirano M, Nose K, et al. A case of severe ganciclovir-induced encephalopathy. Case Rep Neurol. 2013;5(3):183-6doi: 10.1159/000355638.
Sakamoto, H., Hirano, M., Nose, K., Ueno, S., Oki, T., Sugimoto, K., Nishioka, T., Kusunoki, S., & Nakamura, Y. (2013). A case of severe ganciclovir-induced encephalopathy. Case reports in neurology, 5(3), 183-6. https://doi.org/10.1159/000355638
Sakamoto, Hikaru, et al. "A case of severe ganciclovir-induced encephalopathy." Case reports in neurology vol. 5,3 (2013): 183-6. doi: https://doi.org/10.1159/000355638
Sakamoto H, Hirano M, Nose K, Ueno S, Oki T, Sugimoto K, Nishioka T, Kusunoki S, Nakamura Y. A case of severe ganciclovir-induced encephalopathy. Case Rep Neurol. 2013 Oct 05;5(3):183-6. doi: 10.1159/000355638. eCollection 2013. PMID: 24403897; PMCID: PMC3884185.
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Impact of aldosterone-producing adenoma on cardiac structures in echocardiography.

Journal of echocardiography

Hidaka T, Shiwa T, Fujii Y, Nishioka K, Utsunomiya H, Ishibashi K, Mitsuba N, Dohi Y, Oda N, Noma K, Kurisu S, Nakano Y, Yamamoto H, Iishida T, Higashi Y, Kihara Y.
PMID: 24319340
J Echocardiogr. 2013;11:123-9. doi: 10.1007/s12574-013-0168-y. Epub 2013 Feb 05.

BACKGROUND: Primary aldosteronism (PA) is a most common cause of secondary hypertension. In PA, left ventricular hypertrophy (LVH) is more progressive than in any other cause of hypertension. Aldosterone-producing adenoma (APA) and idiopathic hyperaldosteronism (IHA) are major subtypes of...

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Hidaka T, Shiwa T, Fujii Y, et al. Impact of aldosterone-producing adenoma on cardiac structures in echocardiography. J Echocardiogr. 2013;11:123-9doi: 10.1007/s12574-013-0168-y.
Hidaka, T., Shiwa, T., Fujii, Y., Nishioka, K., Utsunomiya, H., Ishibashi, K., Mitsuba, N., Dohi, Y., Oda, N., Noma, K., Kurisu, S., Nakano, Y., Yamamoto, H., Iishida, T., Higashi, Y., & Kihara, Y. (2013). Impact of aldosterone-producing adenoma on cardiac structures in echocardiography. Journal of echocardiography, 11123-9. https://doi.org/10.1007/s12574-013-0168-y
Hidaka, Takayuki, et al. "Impact of aldosterone-producing adenoma on cardiac structures in echocardiography." Journal of echocardiography vol. 11 (2013): 123-9. doi: https://doi.org/10.1007/s12574-013-0168-y
Hidaka T, Shiwa T, Fujii Y, Nishioka K, Utsunomiya H, Ishibashi K, Mitsuba N, Dohi Y, Oda N, Noma K, Kurisu S, Nakano Y, Yamamoto H, Iishida T, Higashi Y, Kihara Y. Impact of aldosterone-producing adenoma on cardiac structures in echocardiography. J Echocardiogr. 2013;11:123-9. doi: 10.1007/s12574-013-0168-y. Epub 2013 Feb 05. PMID: 24319340; PMCID: PMC3851697.
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Erratum: Precise genome-wide base editing by the CRISPR Nickase system in yeast.

Scientific reports

Satomura A, Nishioka R, Mori H, Sato K, Kuroda K, Ueda M.
PMID: 28955053
Sci Rep. 2017 Sep 27;7(1):12354. doi: 10.1038/s41598-017-09606-2.

A correction to this Article has been published and is linked from the HTML version of this paper. The error has been fixed in the paper.

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Satomura A, Nishioka R, Mori H, et al. Erratum: Precise genome-wide base editing by the CRISPR Nickase system in yeast. Sci Rep. 2017;7(1):12354doi: 10.1038/s41598-017-09606-2.
Satomura, A., Nishioka, R., Mori, H., Sato, K., Kuroda, K., & Ueda, M. (2017). Erratum: Precise genome-wide base editing by the CRISPR Nickase system in yeast. Scientific reports, 7(1), 12354. https://doi.org/10.1038/s41598-017-09606-2
Satomura, Atsushi, et al. "Erratum: Precise genome-wide base editing by the CRISPR Nickase system in yeast." Scientific reports vol. 7,1 (2017): 12354. doi: https://doi.org/10.1038/s41598-017-09606-2
Satomura A, Nishioka R, Mori H, Sato K, Kuroda K, Ueda M. Erratum: Precise genome-wide base editing by the CRISPR Nickase system in yeast. Sci Rep. 2017 Sep 27;7(1):12354. doi: 10.1038/s41598-017-09606-2. PMID: 28955053; PMCID: PMC5617842.
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Hydrophobicity of Residue 128 of the Stress-Inducible Sigma Factor RpoS Is Critical for Its Activity.

Frontiers in microbiology

Iwase T, Matsuo T, Nishioka S, Tajima A, Mizunoe Y.
PMID: 28491053
Front Microbiol. 2017 Apr 26;8:656. doi: 10.3389/fmicb.2017.00656. eCollection 2017.

RpoS is a key stress-inducible sigma factor that regulates stress resistance genes in

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Iwase T, Matsuo T, Nishioka S, et al. Hydrophobicity of Residue 128 of the Stress-Inducible Sigma Factor RpoS Is Critical for Its Activity. Front Microbiol. 2017;8:656doi: 10.3389/fmicb.2017.00656.
Iwase, T., Matsuo, T., Nishioka, S., Tajima, A., & Mizunoe, Y. (2017). Hydrophobicity of Residue 128 of the Stress-Inducible Sigma Factor RpoS Is Critical for Its Activity. Frontiers in microbiology, 8656. https://doi.org/10.3389/fmicb.2017.00656
Iwase, Tadayuki, et al. "Hydrophobicity of Residue 128 of the Stress-Inducible Sigma Factor RpoS Is Critical for Its Activity." Frontiers in microbiology vol. 8 (2017): 656. doi: https://doi.org/10.3389/fmicb.2017.00656
Iwase T, Matsuo T, Nishioka S, Tajima A, Mizunoe Y. Hydrophobicity of Residue 128 of the Stress-Inducible Sigma Factor RpoS Is Critical for Its Activity. Front Microbiol. 2017 Apr 26;8:656. doi: 10.3389/fmicb.2017.00656. eCollection 2017. PMID: 28491053; PMCID: PMC5405132.
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Macrophage ubiquitin-specific protease 2 modifies insulin sensitivity in obese mice.

Biochemistry and biophysics reports

Saito N, Kimura S, Miyamoto T, Fukushima S, Amagasa M, Shimamoto Y, Nishioka C, Okamoto S, Toda C, Washio K, Asano A, Miyoshi I, Takahashi E, Kitamura H.
PMID: 28956020
Biochem Biophys Rep. 2017 Jan 26;9:322-329. doi: 10.1016/j.bbrep.2017.01.009. eCollection 2017 Mar.

We previously reported that ubiquitin-specific protease (USP) 2 in macrophages down-regulates genes associated with metabolic diseases, suggesting a putative anti-diabetic role for USP2 in macrophages. In this study, we evaluate this role at both cellular and individual levels. Isolated...

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Saito N, Kimura S, Miyamoto T, et al. Macrophage ubiquitin-specific protease 2 modifies insulin sensitivity in obese mice. Biochem Biophys Rep. 2017;9:322-329doi: 10.1016/j.bbrep.2017.01.009.
Saito, N., Kimura, S., Miyamoto, T., Fukushima, S., Amagasa, M., Shimamoto, Y., Nishioka, C., Okamoto, S., Toda, C., Washio, K., Asano, A., Miyoshi, I., Takahashi, E., & Kitamura, H. (2017). Macrophage ubiquitin-specific protease 2 modifies insulin sensitivity in obese mice. Biochemistry and biophysics reports, 9322-329. https://doi.org/10.1016/j.bbrep.2017.01.009
Saito, Natsuko, et al. "Macrophage ubiquitin-specific protease 2 modifies insulin sensitivity in obese mice." Biochemistry and biophysics reports vol. 9 (2017): 322-329. doi: https://doi.org/10.1016/j.bbrep.2017.01.009
Saito N, Kimura S, Miyamoto T, Fukushima S, Amagasa M, Shimamoto Y, Nishioka C, Okamoto S, Toda C, Washio K, Asano A, Miyoshi I, Takahashi E, Kitamura H. Macrophage ubiquitin-specific protease 2 modifies insulin sensitivity in obese mice. Biochem Biophys Rep. 2017 Jan 26;9:322-329. doi: 10.1016/j.bbrep.2017.01.009. eCollection 2017 Mar. PMID: 28956020; PMCID: PMC5614627.
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