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Curcumin Attenuates Gentamicin-Induced Kidney Mitochondrial Alterations: Possible Role of a Mitochondrial Biogenesis Mechanism.

Evidence-based complementary and alternative medicine : eCAM

Negrette-Guzmán M, García-Niño WR, Tapia E, Zazueta C, Huerta-Yepez S, León-Contreras JC, Hernández-Pando R, Aparicio-Trejo OE, Madero M, Pedraza-Chaverri J.
PMID: 26345660
Evid Based Complement Alternat Med. 2015;2015:917435. doi: 10.1155/2015/917435. Epub 2015 Aug 05.

It has been shown that curcumin (CUR), a polyphenol derived from Curcuma longa, exerts a protective effect against gentamicin- (GM-) induced nephrotoxicity in rats, associated with a preservation of the antioxidant status. Although mitochondrial dysfunction is a hallmark in...

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Negrette-Guzmán M, García-Niño WR, Tapia E, et al. Curcumin Attenuates Gentamicin-Induced Kidney Mitochondrial Alterations: Possible Role of a Mitochondrial Biogenesis Mechanism. Evid Based Complement Alternat Med. 2015;2015:917435doi: 10.1155/2015/917435.
Negrette-Guzmán, M., García-Niño, W. R., Tapia, E., Zazueta, C., Huerta-Yepez, S., León-Contreras, J. C., Hernández-Pando, R., Aparicio-Trejo, O. E., Madero, M., & Pedraza-Chaverri, J. (2015). Curcumin Attenuates Gentamicin-Induced Kidney Mitochondrial Alterations: Possible Role of a Mitochondrial Biogenesis Mechanism. Evidence-based complementary and alternative medicine : eCAM, 2015917435. https://doi.org/10.1155/2015/917435
Negrette-Guzmán, Mario, et al. "Curcumin Attenuates Gentamicin-Induced Kidney Mitochondrial Alterations: Possible Role of a Mitochondrial Biogenesis Mechanism." Evidence-based complementary and alternative medicine : eCAM vol. 2015 (2015): 917435. doi: https://doi.org/10.1155/2015/917435
Negrette-Guzmán M, García-Niño WR, Tapia E, Zazueta C, Huerta-Yepez S, León-Contreras JC, Hernández-Pando R, Aparicio-Trejo OE, Madero M, Pedraza-Chaverri J. Curcumin Attenuates Gentamicin-Induced Kidney Mitochondrial Alterations: Possible Role of a Mitochondrial Biogenesis Mechanism. Evid Based Complement Alternat Med. 2015;2015:917435. doi: 10.1155/2015/917435. Epub 2015 Aug 05. PMID: 26345660; PMCID: PMC4541007.
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Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24].

The Journal of nutritional biochemistry

Carmona-Ramírez I, Santamaría A, Tobón-Velasco JC, Orozco-Ibarra M, González-Herrera IG, Pedraza-Chaverrí J, Maldonado PD.
PMID: 28320533
J Nutr Biochem. 2017 Apr;42:203. doi: 10.1016/j.jnutbio.2017.03.001.

This article has been retracted: please see Elsevier Policy on Article Withdrawal (http://www.elsevier.com/locate/withdrawalpolicy). This article has been retracted at the request of the Editor-in-Chief as it contains inappropriately manipulated images in Figure 7B. As such this article represents a...

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Carmona-Ramírez I, Santamaría A, Tobón-Velasco JC, et al. Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]. J Nutr Biochem. 2017;42:203doi: 10.1016/j.jnutbio.2017.03.001.
Carmona-Ramírez, I., Santamaría, A., Tobón-Velasco, J. C., Orozco-Ibarra, M., González-Herrera, I. G., Pedraza-Chaverrí, J., & Maldonado, P. D. (2017). Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]. The Journal of nutritional biochemistry, 42203. https://doi.org/10.1016/j.jnutbio.2017.03.001
Carmona-Ramírez, Iván, et al. "Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]." The Journal of nutritional biochemistry vol. 42 (2017): 203. doi: https://doi.org/10.1016/j.jnutbio.2017.03.001
Carmona-Ramírez I, Santamaría A, Tobón-Velasco JC, Orozco-Ibarra M, González-Herrera IG, Pedraza-Chaverrí J, Maldonado PD. Retraction notice to "Curcumin restores Nrf2 levels and prevents quinolinic acid-induced neurotoxicity " [JNB 24 (2013) 14-24]. J Nutr Biochem. 2017 Apr;42:203. doi: 10.1016/j.jnutbio.2017.03.001. PMID: 28320533.
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Garlic's ability to prevent in vitro Cu2+-induced lipoprotein oxidation in human serum is preserved in heated garlic: effect unrelated to Cu2+-chelation.

Nutrition journal

Pedraza-Chaverrí J, Gil-Ortiz M, Albarrán G, Barbachano-Esparza L, Menjívar M, Medina-Campos ON.
PMID: 15341661
Nutr J. 2004 Sep 01;3:10. doi: 10.1186/1475-2891-3-10.

BACKGROUND: It has been shown that several extracts and compounds derived from garlic are able to inhibit Cu2+-induced low density lipoprotein oxidation. In this work we explored if the ability of aqueous garlic extract to prevent in vitro Cu2+-induced...

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Pedraza-Chaverrí J, Gil-Ortiz M, Albarrán G, et al. Garlic's ability to prevent in vitro Cu2+-induced lipoprotein oxidation in human serum is preserved in heated garlic: effect unrelated to Cu2+-chelation. Nutr J. 2004;3:10doi: 10.1186/1475-2891-3-10.
Pedraza-Chaverrí, J., Gil-Ortiz, M., Albarrán, G., Barbachano-Esparza, L., Menjívar, M., & Medina-Campos, O. N. (2004). Garlic's ability to prevent in vitro Cu2+-induced lipoprotein oxidation in human serum is preserved in heated garlic: effect unrelated to Cu2+-chelation. Nutrition journal, 310. https://doi.org/10.1186/1475-2891-3-10
Pedraza-Chaverrí, José, et al. "Garlic's ability to prevent in vitro Cu2+-induced lipoprotein oxidation in human serum is preserved in heated garlic: effect unrelated to Cu2+-chelation." Nutrition journal vol. 3 (2004): 10. doi: https://doi.org/10.1186/1475-2891-3-10
Pedraza-Chaverrí J, Gil-Ortiz M, Albarrán G, Barbachano-Esparza L, Menjívar M, Medina-Campos ON. Garlic's ability to prevent in vitro Cu2+-induced lipoprotein oxidation in human serum is preserved in heated garlic: effect unrelated to Cu2+-chelation. Nutr J. 2004 Sep 01;3:10. doi: 10.1186/1475-2891-3-10. PMID: 15341661; PMCID: PMC519022.
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Ozone exposure induces iNOS expression and tyrosine nitration in rat aorta.

Environmental toxicology and pharmacology

Sánchez-González DJ, Moro MA, Castillo-Henkel C, Herrera-González N, Hernández-Pando R, Larios-Medina FJ, Cobilt R, Blanco JA, Pedraza-Chaverrí J, Villanueva C.
PMID: 21782706
Environ Toxicol Pharmacol. 2004 May;17(1):1-7. doi: 10.1016/j.etap.2004.01.002.

The aim was to study whether ozone affects vascular endothelium by causing inducible nitric oxide synthase (iNOS) expression and tyrosine nitration. We also studied biomarkers of endothelial function. Male Wistar rats were exposed to ozone (0.25ppm, 4h/day) or filtered...

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Sánchez-González DJ, Moro MA, Castillo-Henkel C, et al. Ozone exposure induces iNOS expression and tyrosine nitration in rat aorta. Environ Toxicol Pharmacol. 2004;17(1):1-7doi: 10.1016/j.etap.2004.01.002.
Sánchez-González, D. J., Moro, M. A., Castillo-Henkel, C., Herrera-González, N., Hernández-Pando, R., Larios-Medina, F. J., Cobilt, R., Blanco, J. A., Pedraza-Chaverrí, J., & Villanueva, C. (2004). Ozone exposure induces iNOS expression and tyrosine nitration in rat aorta. Environmental toxicology and pharmacology, 17(1), 1-7. https://doi.org/10.1016/j.etap.2004.01.002
Sánchez-González, Dolores J, et al. "Ozone exposure induces iNOS expression and tyrosine nitration in rat aorta." Environmental toxicology and pharmacology vol. 17,1 (2004): 1-7. doi: https://doi.org/10.1016/j.etap.2004.01.002
Sánchez-González DJ, Moro MA, Castillo-Henkel C, Herrera-González N, Hernández-Pando R, Larios-Medina FJ, Cobilt R, Blanco JA, Pedraza-Chaverrí J, Villanueva C. Ozone exposure induces iNOS expression and tyrosine nitration in rat aorta. Environ Toxicol Pharmacol. 2004 May;17(1):1-7. doi: 10.1016/j.etap.2004.01.002. PMID: 21782706.
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Corrigendum to "Association of Nuclear Factor-Erythroid 2-Related Factor 2, Thioredoxin Interacting Protein, and Heme Oxygenase-1 Gene Polymorphisms with Diabetes and Obesity in Mexican Patients".

Oxidative medicine and cellular longevity

Jiménez-Osorio AS, González-Reyes S, García-Niño WR, Moreno-Macías H, Rodríguez-Arellano ME, Vargas-Alarcón G, Zúñiga J, Barquera R, Pedraza-Chaverri J, Meza-Espinoza JP, Leal-Ugarte E, Peralta-Leal V.
PMID: 28642813
Oxid Med Cell Longev. 2017;2017:7543194. doi: 10.1155/2017/7543194. Epub 2017 May 30.

[This corrects the article DOI: 10.1155/2016/7367641.].

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Jiménez-Osorio AS, González-Reyes S, García-Niño WR, et al. Corrigendum to "Association of Nuclear Factor-Erythroid 2-Related Factor 2, Thioredoxin Interacting Protein, and Heme Oxygenase-1 Gene Polymorphisms with Diabetes and Obesity in Mexican Patients". Oxid Med Cell Longev. 2017;2017:7543194doi: 10.1155/2017/7543194.
Jiménez-Osorio, A. S., González-Reyes, S., García-Niño, W. R., Moreno-Macías, H., Rodríguez-Arellano, M. E., Vargas-Alarcón, G., Zúñiga, J., Barquera, R., Pedraza-Chaverri, J., Meza-Espinoza, J. P., Leal-Ugarte, E., & Peralta-Leal, V. (2017). Corrigendum to "Association of Nuclear Factor-Erythroid 2-Related Factor 2, Thioredoxin Interacting Protein, and Heme Oxygenase-1 Gene Polymorphisms with Diabetes and Obesity in Mexican Patients". Oxidative medicine and cellular longevity, 20177543194. https://doi.org/10.1155/2017/7543194
Jiménez-Osorio, Angélica Saraí, et al. "Corrigendum to "Association of Nuclear Factor-Erythroid 2-Related Factor 2, Thioredoxin Interacting Protein, and Heme Oxygenase-1 Gene Polymorphisms with Diabetes and Obesity in Mexican Patients"." Oxidative medicine and cellular longevity vol. 2017 (2017): 7543194. doi: https://doi.org/10.1155/2017/7543194
Jiménez-Osorio AS, González-Reyes S, García-Niño WR, Moreno-Macías H, Rodríguez-Arellano ME, Vargas-Alarcón G, Zúñiga J, Barquera R, Pedraza-Chaverri J, Meza-Espinoza JP, Leal-Ugarte E, Peralta-Leal V. Corrigendum to "Association of Nuclear Factor-Erythroid 2-Related Factor 2, Thioredoxin Interacting Protein, and Heme Oxygenase-1 Gene Polymorphisms with Diabetes and Obesity in Mexican Patients". Oxid Med Cell Longev. 2017;2017:7543194. doi: 10.1155/2017/7543194. Epub 2017 May 30. PMID: 28642813; PMCID: PMC5468567.
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Astrocytes Are More Vulnerable than Neurons to Silicon Dioxide Nanoparticle Toxicity in Vitro.

Toxics

Limón-Pacheco JH, Jiménez-Barrios N, Déciga-Alcaraz A, Martínez-Cuazitl A, Mata-Miranda MM, Vázquez-Zapién GJ, Pedraza-Chaverri J, Chirino YI, Orozco-Ibarra M.
PMID: 32751182
Toxics. 2020 Jul 29;8(3). doi: 10.3390/toxics8030051.

Some studies have shown that silicon dioxide nanoparticles (SiO

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Limón-Pacheco JH, Jiménez-Barrios N, Déciga-Alcaraz A, et al. Astrocytes Are More Vulnerable than Neurons to Silicon Dioxide Nanoparticle Toxicity in Vitro. Toxics. 2020;8(3)doi: 10.3390/toxics8030051.
Limón-Pacheco, J. H., Jiménez-Barrios, N., Déciga-Alcaraz, A., Martínez-Cuazitl, A., Mata-Miranda, M. M., Vázquez-Zapién, G. J., Pedraza-Chaverri, J., Chirino, Y. I., & Orozco-Ibarra, M. (2020). Astrocytes Are More Vulnerable than Neurons to Silicon Dioxide Nanoparticle Toxicity in Vitro. Toxics, 8(3), . https://doi.org/10.3390/toxics8030051
Limón-Pacheco, Jorge Humberto, et al. "Astrocytes Are More Vulnerable than Neurons to Silicon Dioxide Nanoparticle Toxicity in Vitro." Toxics vol. 8,3 (2020). doi: https://doi.org/10.3390/toxics8030051
Limón-Pacheco JH, Jiménez-Barrios N, Déciga-Alcaraz A, Martínez-Cuazitl A, Mata-Miranda MM, Vázquez-Zapién GJ, Pedraza-Chaverri J, Chirino YI, Orozco-Ibarra M. Astrocytes Are More Vulnerable than Neurons to Silicon Dioxide Nanoparticle Toxicity in Vitro. Toxics. 2020 Jul 29;8(3). doi: 10.3390/toxics8030051. PMID: 32751182; PMCID: PMC7560395.
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Cellular Senescence and ApoE4: Their Repercussions in Alzheimer's Disease.

CNS & neurological disorders drug targets

Tecalco-Cruz AC, Zepeda-Cervantes J, López-Canovas L, Ramírez-Jarquín JO, Pedraza-Chaverrí J, Briones-Herrera A.
PMID: 34182916
CNS Neurol Disord Drug Targets. 2021;20(9):778-785. doi: 10.2174/1871527320666210628102721.

Alzheimer's Disease (AD) is characterized by progressive memory loss due to neurodegeneration that occurs mainly during aging. The accumulation of senescent cells has been related to aging. Furthermore, the expression of the variant ApoE ε4 is a critical risk...

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Tecalco-Cruz AC, Zepeda-Cervantes J, López-Canovas L, et al. Cellular Senescence and ApoE4: Their Repercussions in Alzheimer's Disease. CNS Neurol Disord Drug Targets. 2021;20(9):778-785doi: 10.2174/1871527320666210628102721.
Tecalco-Cruz, A. C., Zepeda-Cervantes, J., López-Canovas, L., Ramírez-Jarquín, J. O., Pedraza-Chaverrí, J., & Briones-Herrera, A. (2021). Cellular Senescence and ApoE4: Their Repercussions in Alzheimer's Disease. CNS & neurological disorders drug targets, 20(9), 778-785. https://doi.org/10.2174/1871527320666210628102721
Tecalco-Cruz, Angeles C, et al. "Cellular Senescence and ApoE4: Their Repercussions in Alzheimer's Disease." CNS & neurological disorders drug targets vol. 20,9 (2021): 778-785. doi: https://doi.org/10.2174/1871527320666210628102721
Tecalco-Cruz AC, Zepeda-Cervantes J, López-Canovas L, Ramírez-Jarquín JO, Pedraza-Chaverrí J, Briones-Herrera A. Cellular Senescence and ApoE4: Their Repercussions in Alzheimer's Disease. CNS Neurol Disord Drug Targets. 2021;20(9):778-785. doi: 10.2174/1871527320666210628102721. PMID: 34182916.
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Exposure to Sub-Lethal Doses of Permethrin Is Associated with Neurotoxicity: Changes in Bioenergetics, Redox Markers, Neuroinflammation and Morphology.

Toxics

López-Aceves TG, Coballase-Urrutia E, Estrada-Rojo F, Vanoye-Carlo A, Carmona-Aparicio L, Hernández ME, Pedraza-Chaverri J, Navarro L, Aparicio-Trejo OE, Pérez-Torres A, Medina-Campos ON, Martínez-Fong D, Sánchez-Valle V, Cárdenas-Rodríguez N, Granados-Rojas L, Pulido-Camarillo E, Rodríguez-Mata V, León-Sicairos CDR.
PMID: 34941771
Toxics. 2021 Dec 06;9(12). doi: 10.3390/toxics9120337.

Permethrin (PERM) is a member of the class I family of synthetic pyrethroids. Human use has shown that it affects different systems, with wide health dysfunctions. Our aim was to determine bioenergetics, neuroinflammation and morphology changes, as redox markers...

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López-Aceves TG, Coballase-Urrutia E, Estrada-Rojo F, et al. Exposure to Sub-Lethal Doses of Permethrin Is Associated with Neurotoxicity: Changes in Bioenergetics, Redox Markers, Neuroinflammation and Morphology. Toxics. 2021;9(12)doi: 10.3390/toxics9120337.
López-Aceves, T. G., Coballase-Urrutia, E., Estrada-Rojo, F., Vanoye-Carlo, A., Carmona-Aparicio, L., Hernández, M. E., Pedraza-Chaverri, J., Navarro, L., Aparicio-Trejo, O. E., Pérez-Torres, A., Medina-Campos, O. N., Martínez-Fong, D., Sánchez-Valle, V., Cárdenas-Rodríguez, N., Granados-Rojas, L., Pulido-Camarillo, E., Rodríguez-Mata, V., & León-Sicairos, C. D. R. (2021). Exposure to Sub-Lethal Doses of Permethrin Is Associated with Neurotoxicity: Changes in Bioenergetics, Redox Markers, Neuroinflammation and Morphology. Toxics, 9(12), . https://doi.org/10.3390/toxics9120337
López-Aceves, Teresita Guadalupe, et al. "Exposure to Sub-Lethal Doses of Permethrin Is Associated with Neurotoxicity: Changes in Bioenergetics, Redox Markers, Neuroinflammation and Morphology." Toxics vol. 9,12 (2021). doi: https://doi.org/10.3390/toxics9120337
López-Aceves TG, Coballase-Urrutia E, Estrada-Rojo F, Vanoye-Carlo A, Carmona-Aparicio L, Hernández ME, Pedraza-Chaverri J, Navarro L, Aparicio-Trejo OE, Pérez-Torres A, Medina-Campos ON, Martínez-Fong D, Sánchez-Valle V, Cárdenas-Rodríguez N, Granados-Rojas L, Pulido-Camarillo E, Rodríguez-Mata V, León-Sicairos CDR. Exposure to Sub-Lethal Doses of Permethrin Is Associated with Neurotoxicity: Changes in Bioenergetics, Redox Markers, Neuroinflammation and Morphology. Toxics. 2021 Dec 06;9(12). doi: 10.3390/toxics9120337. PMID: 34941771.
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A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation.

Life sciences

Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E.
PMID: 34921866
Life Sci. 2021 Dec 16;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16.

BACKGROUND: Ischemic kidney injury is a common clinical condition resulting from transient interruption of the kidney's normal blood flow, leading to oxidative stress, inflammation, and kidney dysfunction. The ketogenic diet (KD), a low-carbohydrate, high-fat diet that stimulates endogenous ketone...

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Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, et al. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021;289:120227doi: 10.1016/j.lfs.2021.120227.
Rojas-Morales, P., León-Contreras, J. C., Sánchez-Tapia, M., Silva-Palacios, A., Cano-Martínez, A., González-Reyes, S., Jiménez-Osorio, A. S., Hernández-Pando, R., Osorio-Alonso, H., Sánchez-Lozada, L. G., Tovar, A. R., Pedraza-Chaverri, J., & Tapia, E. (2021). A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life sciences, 289120227. https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales, Pedro, et al. "A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation." Life sciences vol. 289 (2021): 120227. doi: https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021 Dec 16;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16. PMID: 34921866.
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S-allyl Cysteine, a Garlic Compound, Produces an Antidepressant-Like Effect and Exhibits Antioxidant Properties in Mice.

Brain sciences

Ruiz-Sánchez E, Pedraza-Chaverri J, Medina-Campos ON, Maldonado PD, Rojas P.
PMID: 32859119
Brain Sci. 2020 Aug 26;10(9). doi: 10.3390/brainsci10090592.

Depression is a psychiatric disorder, and oxidative stress is a significant mechanism of damage in this mood disorder. It is characterized by an enhancement of oxidative stress markers and low concentrations of endogenous antioxidants, or antioxidants enzymes. This suggests...

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Ruiz-Sánchez E, Pedraza-Chaverri J, Medina-Campos ON, et al. S-allyl Cysteine, a Garlic Compound, Produces an Antidepressant-Like Effect and Exhibits Antioxidant Properties in Mice. Brain Sci. 2020;10(9)doi: 10.3390/brainsci10090592.
Ruiz-Sánchez, E., Pedraza-Chaverri, J., Medina-Campos, O. N., Maldonado, P. D., & Rojas, P. (2020). S-allyl Cysteine, a Garlic Compound, Produces an Antidepressant-Like Effect and Exhibits Antioxidant Properties in Mice. Brain sciences, 10(9), . https://doi.org/10.3390/brainsci10090592
Ruiz-Sánchez, Elizabeth, et al. "S-allyl Cysteine, a Garlic Compound, Produces an Antidepressant-Like Effect and Exhibits Antioxidant Properties in Mice." Brain sciences vol. 10,9 (2020). doi: https://doi.org/10.3390/brainsci10090592
Ruiz-Sánchez E, Pedraza-Chaverri J, Medina-Campos ON, Maldonado PD, Rojas P. S-allyl Cysteine, a Garlic Compound, Produces an Antidepressant-Like Effect and Exhibits Antioxidant Properties in Mice. Brain Sci. 2020 Aug 26;10(9). doi: 10.3390/brainsci10090592. PMID: 32859119; PMCID: PMC7564461.
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The Protective Effect of Alpha-Mangostin against Cisplatin-Induced Cell Death in LLC-PK1 Cells is Associated to Mitochondrial Function Preservation.

Antioxidants (Basel, Switzerland)

Reyes-Fermín LM, Avila-Rojas SH, Aparicio-Trejo OE, Tapia E, Rivero I, Pedraza-Chaverri J.
PMID: 31096625
Antioxidants (Basel). 2019 May 15;8(5). doi: 10.3390/antiox8050133.

Cis-dichlorodiammineplatinum II (CDDP) is a chemotherapeutic agent that induces nephrotoxicity by different mechanisms, including oxidative stress, mitochondrial dysfunction, autophagy, and endoplasmic reticulum stress. This study aimed to evaluate if the protective effects of the antioxidant alpha-mangostin (αM) in CDDP-induced...

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Reyes-Fermín LM, Avila-Rojas SH, Aparicio-Trejo OE, et al. The Protective Effect of Alpha-Mangostin against Cisplatin-Induced Cell Death in LLC-PK1 Cells is Associated to Mitochondrial Function Preservation. Antioxidants (Basel). 2019;8(5)doi: 10.3390/antiox8050133.
Reyes-Fermín, L. M., Avila-Rojas, S. H., Aparicio-Trejo, O. E., Tapia, E., Rivero, I., & Pedraza-Chaverri, J. (2019). The Protective Effect of Alpha-Mangostin against Cisplatin-Induced Cell Death in LLC-PK1 Cells is Associated to Mitochondrial Function Preservation. Antioxidants (Basel, Switzerland), 8(5), . https://doi.org/10.3390/antiox8050133
Reyes-Fermín, Laura María, et al. "The Protective Effect of Alpha-Mangostin against Cisplatin-Induced Cell Death in LLC-PK1 Cells is Associated to Mitochondrial Function Preservation." Antioxidants (Basel, Switzerland) vol. 8,5 (2019). doi: https://doi.org/10.3390/antiox8050133
Reyes-Fermín LM, Avila-Rojas SH, Aparicio-Trejo OE, Tapia E, Rivero I, Pedraza-Chaverri J. The Protective Effect of Alpha-Mangostin against Cisplatin-Induced Cell Death in LLC-PK1 Cells is Associated to Mitochondrial Function Preservation. Antioxidants (Basel). 2019 May 15;8(5). doi: 10.3390/antiox8050133. PMID: 31096625; PMCID: PMC6562511.
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Nordihydroguaiaretic Acid: From Herbal Medicine to Clinical Development for Cancer and Chronic Diseases.

Frontiers in pharmacology

Manda G, Rojo AI, Martínez-Klimova E, Pedraza-Chaverri J, Cuadrado A.
PMID: 32184727
Front Pharmacol. 2020 Feb 28;11:151. doi: 10.3389/fphar.2020.00151. eCollection 2020.

Nordihydroguaiaretic acid (NDGA) is a phenolic lignan obtained from

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Manda G, Rojo AI, Martínez-Klimova E, et al. Nordihydroguaiaretic Acid: From Herbal Medicine to Clinical Development for Cancer and Chronic Diseases. Front Pharmacol. 2020;11:151doi: 10.3389/fphar.2020.00151.
Manda, G., Rojo, A. I., Martínez-Klimova, E., Pedraza-Chaverri, J., & Cuadrado, A. (2020). Nordihydroguaiaretic Acid: From Herbal Medicine to Clinical Development for Cancer and Chronic Diseases. Frontiers in pharmacology, 11151. https://doi.org/10.3389/fphar.2020.00151
Manda, Gina, et al. "Nordihydroguaiaretic Acid: From Herbal Medicine to Clinical Development for Cancer and Chronic Diseases." Frontiers in pharmacology vol. 11 (2020): 151. doi: https://doi.org/10.3389/fphar.2020.00151
Manda G, Rojo AI, Martínez-Klimova E, Pedraza-Chaverri J, Cuadrado A. Nordihydroguaiaretic Acid: From Herbal Medicine to Clinical Development for Cancer and Chronic Diseases. Front Pharmacol. 2020 Feb 28;11:151. doi: 10.3389/fphar.2020.00151. eCollection 2020. PMID: 32184727; PMCID: PMC7058590.
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