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Calcitonin Gene-related Peptide Causes Intraspinal Spreading of Substance P Released by Peripheral Stimulation.

The European journal of neuroscience

Schaible HG, Hope PJ, Lang CW, Duggan AW.
PMID: 12106319
Eur J Neurosci. 1992;4(8):750-7. doi: 10.1111/j.1460-9568.1992.tb00184.x.

Experiments were performed in barbiturate-anaesthetized, spinalized cats to investigate the effect of calcitonin gene-related peptide (CGRP) on the spatial distribution of immunoreactive substance P (ir-SP) in the spinal cord released by electrical nerve stimulation and noxious mechanical stimuli. The...

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Schaible HG, Hope PJ, Lang CW, et al. Calcitonin Gene-related Peptide Causes Intraspinal Spreading of Substance P Released by Peripheral Stimulation. Eur J Neurosci. 1992;4(8):750-7doi: 10.1111/j.1460-9568.1992.tb00184.x.
Schaible, H. G., Hope, P. J., Lang, C. W., & Duggan, A. W. (1992). Calcitonin Gene-related Peptide Causes Intraspinal Spreading of Substance P Released by Peripheral Stimulation. The European journal of neuroscience, 4(8), 750-7. https://doi.org/10.1111/j.1460-9568.1992.tb00184.x
Schaible, H G, et al. "Calcitonin Gene-related Peptide Causes Intraspinal Spreading of Substance P Released by Peripheral Stimulation." The European journal of neuroscience vol. 4,8 (1992): 750-7. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00184.x
Schaible HG, Hope PJ, Lang CW, Duggan AW. Calcitonin Gene-related Peptide Causes Intraspinal Spreading of Substance P Released by Peripheral Stimulation. Eur J Neurosci. 1992;4(8):750-7. doi: 10.1111/j.1460-9568.1992.tb00184.x. PMID: 12106319.
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Reporting of sex as a variable in cardiovascular studies using cultured cells.

Biology of sex differences

Taylor KE, Vallejo-Giraldo C, Schaible NS, Zakeri R, Miller VM.
PMID: 22060014
Biol Sex Differ. 2011 Nov 07;2:11. doi: 10.1186/2042-6410-2-11.

BACKGROUND: Chromosomal complement, including that provided by the sex chromosomes, influences expression of proteins and molecular signaling in every cell. However, less than 50% of the scientific studies published in 2009 using experimental animals reported sex as a biological...

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Taylor KE, Vallejo-Giraldo C, Schaible NS, et al. Reporting of sex as a variable in cardiovascular studies using cultured cells. Biol Sex Differ. 2011;2:11doi: 10.1186/2042-6410-2-11.
Taylor, K. E., Vallejo-Giraldo, C., Schaible, N. S., Zakeri, R., & Miller, V. M. (2011). Reporting of sex as a variable in cardiovascular studies using cultured cells. Biology of sex differences, 211. https://doi.org/10.1186/2042-6410-2-11
Taylor, K Efua, et al. "Reporting of sex as a variable in cardiovascular studies using cultured cells." Biology of sex differences vol. 2 (2011): 11. doi: https://doi.org/10.1186/2042-6410-2-11
Taylor KE, Vallejo-Giraldo C, Schaible NS, Zakeri R, Miller VM. Reporting of sex as a variable in cardiovascular studies using cultured cells. Biol Sex Differ. 2011 Nov 07;2:11. doi: 10.1186/2042-6410-2-11. PMID: 22060014; PMCID: PMC3224776.
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[Der Schmerz - the leading German-language pain journal].

Schmerz (Berlin, Germany)

Radbruch L, Schaible HG.
PMID: 25465245
Schmerz. 2014 Dec;28(6):569-70. doi: 10.1007/s00482-014-1498-z.

No abstract available.

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Radbruch L, Schaible HG. Der Schmerz - die führende deutschsprachige Schmerzzeitschrift. [Der Schmerz - the leading German-language pain journal]. Schmerz. 2014;28(6):569-70doi: 10.1007/s00482-014-1498-z.
Radbruch, L., & Schaible, H. G. (2014). Der Schmerz - die führende deutschsprachige Schmerzzeitschrift. [Der Schmerz - the leading German-language pain journal]. Schmerz (Berlin, Germany), 28(6), 569-70. https://doi.org/10.1007/s00482-014-1498-z
Radbruch, L, and Schaible, H-G. "Der Schmerz - die führende deutschsprachige Schmerzzeitschrift." [Der Schmerz - the leading German-language pain journal]. Schmerz (Berlin, Germany) vol. 28,6 (2014): 569-70. doi: https://doi.org/10.1007/s00482-014-1498-z
Radbruch L, Schaible HG. Der Schmerz - die führende deutschsprachige Schmerzzeitschrift. [Der Schmerz - the leading German-language pain journal]. Schmerz. 2014 Dec;28(6):569-70. doi: 10.1007/s00482-014-1498-z. German. PMID: 25465245.
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Perspectives for personalized therapy for patients with multidrug-resistant tuberculosis.

Journal of internal medicine

Lange C, Alghamdi WA, Al-Shaer MH, Brighenti S, Diacon AH, DiNardo AR, Grobbel HP, Gröschel MI, von Groote-Bidlingmaier F, Hauptmann M, Heyckendorf J, Köhler N, Kohl TA, Merker M, Niemann S, Peloquin CA, Reimann M, Schaible UE, Schaub D, Schleusener V, Thye T, Schön T.
PMID: 29806961
J Intern Med. 2018 May 28; doi: 10.1111/joim.12780. Epub 2018 May 28.

According to the World Health Organization (WHO), tuberculosis is the leading cause of death attributed to a single microbial pathogen worldwide. In addition to the large number of patients affected by tuberculosis, the emergence of Mycobacterium tuberculosis drug-resistance is...

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Lange C, Alghamdi WA, Al-Shaer MH, et al. Perspectives for personalized therapy for patients with multidrug-resistant tuberculosis. J Intern Med. 2018;doi: 10.1111/joim.12780.
Lange, C., Alghamdi, W. A., Al-Shaer, M. H., Brighenti, S., Diacon, A. H., DiNardo, A. R., Grobbel, H. P., Gröschel, M. I., von Groote-Bidlingmaier, F., Hauptmann, M., Heyckendorf, J., Köhler, N., Kohl, T. A., Merker, M., Niemann, S., Peloquin, C. A., Reimann, M., Schaible, U. E., Schaub, D., Schleusener, V., Thye, T., & Schön, T. (2018). Perspectives for personalized therapy for patients with multidrug-resistant tuberculosis. Journal of internal medicine, . https://doi.org/10.1111/joim.12780
Lange, C, et al. "Perspectives for personalized therapy for patients with multidrug-resistant tuberculosis." Journal of internal medicine vol. (2018). doi: https://doi.org/10.1111/joim.12780
Lange C, Alghamdi WA, Al-Shaer MH, Brighenti S, Diacon AH, DiNardo AR, Grobbel HP, Gröschel MI, von Groote-Bidlingmaier F, Hauptmann M, Heyckendorf J, Köhler N, Kohl TA, Merker M, Niemann S, Peloquin CA, Reimann M, Schaible UE, Schaub D, Schleusener V, Thye T, Schön T. Perspectives for personalized therapy for patients with multidrug-resistant tuberculosis. J Intern Med. 2018 May 28; doi: 10.1111/joim.12780. Epub 2018 May 28. PMID: 29806961.
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[A new rubric in Der Schmerz: "PAIN CLINICAL UPDATES"].

Schmerz (Berlin, Germany)

Schaible HG, Radbruch L.
PMID: 29404684
Schmerz. 2018 Apr;32(2):89. doi: 10.1007/s00482-018-0274-x.

No abstract available.

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Schaible HG, Radbruch L. Neue Rubrik „PAIN CLINICAL UPDATES“ in Der Schmerz. [A new rubric in Der Schmerz: "PAIN CLINICAL UPDATES"]. Schmerz. 2018;32(2):89doi: 10.1007/s00482-018-0274-x.
Schaible, H. G., & Radbruch, L. (2018). Neue Rubrik „PAIN CLINICAL UPDATES“ in Der Schmerz. [A new rubric in Der Schmerz: "PAIN CLINICAL UPDATES"]. Schmerz (Berlin, Germany), 32(2), 89. https://doi.org/10.1007/s00482-018-0274-x
Schaible, H-G, and Radbruch, L. "Neue Rubrik „PAIN CLINICAL UPDATES“ in Der Schmerz." [A new rubric in Der Schmerz: "PAIN CLINICAL UPDATES"]. Schmerz (Berlin, Germany) vol. 32,2 (2018): 89. doi: https://doi.org/10.1007/s00482-018-0274-x
Schaible HG, Radbruch L. Neue Rubrik „PAIN CLINICAL UPDATES“ in Der Schmerz. [A new rubric in Der Schmerz: "PAIN CLINICAL UPDATES"]. Schmerz. 2018 Apr;32(2):89. doi: 10.1007/s00482-018-0274-x. German. PMID: 29404684.
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Macrophage Inducible C-Type Lectin As a Multifunctional Player in Immunity.

Frontiers in immunology

Patin EC, Orr SJ, Schaible UE.
PMID: 28791019
Front Immunol. 2017 Jul 25;8:861. doi: 10.3389/fimmu.2017.00861. eCollection 2017.

The macrophage-inducible C-type lectin (Mincle) is an innate immune receptor on myeloid cells sensing diverse entities including pathogens and damaged cells. Mincle was first described as a receptor for the mycobacterial cell wall glycolipid, trehalose-6,6'-dimycolate, or cord factor, and...

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Patin EC, Orr SJ, Schaible UE. Macrophage Inducible C-Type Lectin As a Multifunctional Player in Immunity. Front Immunol. 2017;8:861doi: 10.3389/fimmu.2017.00861.
Patin, E. C., Orr, S. J., & Schaible, U. E. (2017). Macrophage Inducible C-Type Lectin As a Multifunctional Player in Immunity. Frontiers in immunology, 8861. https://doi.org/10.3389/fimmu.2017.00861
Patin, Emmanuel C, et al. "Macrophage Inducible C-Type Lectin As a Multifunctional Player in Immunity." Frontiers in immunology vol. 8 (2017): 861. doi: https://doi.org/10.3389/fimmu.2017.00861
Patin EC, Orr SJ, Schaible UE. Macrophage Inducible C-Type Lectin As a Multifunctional Player in Immunity. Front Immunol. 2017 Jul 25;8:861. doi: 10.3389/fimmu.2017.00861. eCollection 2017. PMID: 28791019; PMCID: PMC5525440.
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Corrigendum to "Characterization of Bone Marrow Mononuclear Cells on Biomaterials for Bone Tissue Engineering In Vitro".

BioMed research international

Henrich D, Verboket R, Schaible A, Kontradowitz K, Oppermann E, Brune JC, Nau C, Meier S, Bonig H, Marzi I, Seebach C.
PMID: 26417593
Biomed Res Int. 2015;2015:325203. doi: 10.1155/2015/325203. Epub 2015 Aug 31.

No abstract available.

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Henrich D, Verboket R, Schaible A, et al. Corrigendum to "Characterization of Bone Marrow Mononuclear Cells on Biomaterials for Bone Tissue Engineering In Vitro". Biomed Res Int. 2015;2015:325203doi: 10.1155/2015/325203.
Henrich, D., Verboket, R., Schaible, A., Kontradowitz, K., Oppermann, E., Brune, J. C., Nau, C., Meier, S., Bonig, H., Marzi, I., & Seebach, C. (2015). Corrigendum to "Characterization of Bone Marrow Mononuclear Cells on Biomaterials for Bone Tissue Engineering In Vitro". BioMed research international, 2015325203. https://doi.org/10.1155/2015/325203
Henrich, Dirk, et al. "Corrigendum to "Characterization of Bone Marrow Mononuclear Cells on Biomaterials for Bone Tissue Engineering In Vitro"." BioMed research international vol. 2015 (2015): 325203. doi: https://doi.org/10.1155/2015/325203
Henrich D, Verboket R, Schaible A, Kontradowitz K, Oppermann E, Brune JC, Nau C, Meier S, Bonig H, Marzi I, Seebach C. Corrigendum to "Characterization of Bone Marrow Mononuclear Cells on Biomaterials for Bone Tissue Engineering In Vitro". Biomed Res Int. 2015;2015:325203. doi: 10.1155/2015/325203. Epub 2015 Aug 31. PMID: 26417593; PMCID: PMC4568334.
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[Tracing the neurobiological causes of chronic pain].

Schmerz (Berlin, Germany)

Schaible HG, Radbruch L.
PMID: 27028534
Schmerz. 2016 Apr;30(2):125-6. doi: 10.1007/s00482-016-0108-7.

No abstract available.

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Schaible HG, Radbruch L. Den neurobiologischen Grundlagen des chronischen Schmerzes auf der Spur. [Tracing the neurobiological causes of chronic pain]. Schmerz. 2016;30(2):125-6doi: 10.1007/s00482-016-0108-7.
Schaible, H. G., & Radbruch, L. (2016). Den neurobiologischen Grundlagen des chronischen Schmerzes auf der Spur. [Tracing the neurobiological causes of chronic pain]. Schmerz (Berlin, Germany), 30(2), 125-6. https://doi.org/10.1007/s00482-016-0108-7
Schaible, H-G, and Radbruch, L. "Den neurobiologischen Grundlagen des chronischen Schmerzes auf der Spur." [Tracing the neurobiological causes of chronic pain]. Schmerz (Berlin, Germany) vol. 30,2 (2016): 125-6. doi: https://doi.org/10.1007/s00482-016-0108-7
Schaible HG, Radbruch L. Den neurobiologischen Grundlagen des chronischen Schmerzes auf der Spur. [Tracing the neurobiological causes of chronic pain]. Schmerz. 2016 Apr;30(2):125-6. doi: 10.1007/s00482-016-0108-7. German. PMID: 27028534.
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Immediate tumor resection in patients with locally advanced gastroesophageal adenocarcinoma with nonresponse to chemotherapy after 4 weeks of treatment versus resection after completion of chemotherapy (OPTITREAT trial, DRKS00004668): study protocol for a randomized controlled pilot trial.

Pilot and feasibility studies

Blank S, Knebel P, Haag GM, Bruckner T, Klaiber U, Burian M, Schaible A, Sisic L, Schmidt T, Diener MK, Ott K.
PMID: 27965838
Pilot Feasibility Stud. 2016 Apr 04;2:18. doi: 10.1186/s40814-016-0059-x. eCollection 2016.

BACKGROUND: Neoadjuvant chemotherapy is a standard of care for patients with adenocarcinoma of the esophagus and stomach in Europe, but still only 20-40 % respond to therapy and the critical issue; how to treat nonresponding patients is still unclear....

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Blank S, Knebel P, Haag GM, et al. Immediate tumor resection in patients with locally advanced gastroesophageal adenocarcinoma with nonresponse to chemotherapy after 4 weeks of treatment versus resection after completion of chemotherapy (OPTITREAT trial, DRKS00004668): study protocol for a randomized controlled pilot trial. Pilot Feasibility Stud. 2016;2:18doi: 10.1186/s40814-016-0059-x.
Blank, S., Knebel, P., Haag, G. M., Bruckner, T., Klaiber, U., Burian, M., Schaible, A., Sisic, L., Schmidt, T., Diener, M. K., & Ott, K. (2016). Immediate tumor resection in patients with locally advanced gastroesophageal adenocarcinoma with nonresponse to chemotherapy after 4 weeks of treatment versus resection after completion of chemotherapy (OPTITREAT trial, DRKS00004668): study protocol for a randomized controlled pilot trial. Pilot and feasibility studies, 218. https://doi.org/10.1186/s40814-016-0059-x
Blank, Susanne, et al. "Immediate tumor resection in patients with locally advanced gastroesophageal adenocarcinoma with nonresponse to chemotherapy after 4 weeks of treatment versus resection after completion of chemotherapy (OPTITREAT trial, DRKS00004668): study protocol for a randomized controlled pilot trial." Pilot and feasibility studies vol. 2 (2016): 18. doi: https://doi.org/10.1186/s40814-016-0059-x
Blank S, Knebel P, Haag GM, Bruckner T, Klaiber U, Burian M, Schaible A, Sisic L, Schmidt T, Diener MK, Ott K. Immediate tumor resection in patients with locally advanced gastroesophageal adenocarcinoma with nonresponse to chemotherapy after 4 weeks of treatment versus resection after completion of chemotherapy (OPTITREAT trial, DRKS00004668): study protocol for a randomized controlled pilot trial. Pilot Feasibility Stud. 2016 Apr 04;2:18. doi: 10.1186/s40814-016-0059-x. eCollection 2016. PMID: 27965838; PMCID: PMC5153833.
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Mechanisms of reduced susceptibility and genotypic prediction of antibiotic resistance in Prevotella isolated from cystic fibrosis (CF) and non-CF patients.

The Journal of antimicrobial chemotherapy

Sherrard LJ, Schaible B, Graham KA, McGrath SJ, McIlreavey L, Hatch J, Wolfgang MC, Muhlebach MS, Gilpin DF, Schneiders T, Stuart Elborn J, Tunney MM.
PMID: 30351435
J Antimicrob Chemother. 2018 Nov 01;73(11):3218. doi: 10.1093/jac/dky313.

No abstract available.

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Sherrard LJ, Schaible B, Graham KA, et al. Mechanisms of reduced susceptibility and genotypic prediction of antibiotic resistance in Prevotella isolated from cystic fibrosis (CF) and non-CF patients. J Antimicrob Chemother. 2018;73(11):3218doi: 10.1093/jac/dky313.
Sherrard, L. J., Schaible, B., Graham, K. A., McGrath, S. J., McIlreavey, L., Hatch, J., Wolfgang, M. C., Muhlebach, M. S., Gilpin, D. F., Schneiders, T., Stuart Elborn, J., & Tunney, M. M. (2018). Mechanisms of reduced susceptibility and genotypic prediction of antibiotic resistance in Prevotella isolated from cystic fibrosis (CF) and non-CF patients. The Journal of antimicrobial chemotherapy, 73(11), 3218. https://doi.org/10.1093/jac/dky313
Sherrard, Laura J, et al. "Mechanisms of reduced susceptibility and genotypic prediction of antibiotic resistance in Prevotella isolated from cystic fibrosis (CF) and non-CF patients." The Journal of antimicrobial chemotherapy vol. 73,11 (2018): 3218. doi: https://doi.org/10.1093/jac/dky313
Sherrard LJ, Schaible B, Graham KA, McGrath SJ, McIlreavey L, Hatch J, Wolfgang MC, Muhlebach MS, Gilpin DF, Schneiders T, Stuart Elborn J, Tunney MM. Mechanisms of reduced susceptibility and genotypic prediction of antibiotic resistance in Prevotella isolated from cystic fibrosis (CF) and non-CF patients. J Antimicrob Chemother. 2018 Nov 01;73(11):3218. doi: 10.1093/jac/dky313. PMID: 30351435; PMCID: PMC6927872.
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Isolation of .

Medical mycology case reports

Newman R, Schaible M.
PMID: 31249771
Med Mycol Case Rep. 2019 Jun 12;25:19-21. doi: 10.1016/j.mmcr.2019.06.002. eCollection 2019 Sep.

We hereby report a unique manifestation of feline cryptococcosis in the form of extensively consolidated lipoid pneumonia resulting in an isolated space-occupying lung lobe mass. Lung lobectomy was performed and a diagnosis of severe lipoid pneumonia with intra-lesional

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Newman R, Schaible M. Isolation of . Med Mycol Case Rep. 2019;25:19-21doi: 10.1016/j.mmcr.2019.06.002.
Newman, R., & Schaible, M. (2019). Isolation of . Medical mycology case reports, 2519-21. https://doi.org/10.1016/j.mmcr.2019.06.002
Newman, Robert, and Schaible, Megan. "Isolation of ." Medical mycology case reports vol. 25 (2019): 19-21. doi: https://doi.org/10.1016/j.mmcr.2019.06.002
Newman R, Schaible M. Isolation of . Med Mycol Case Rep. 2019 Jun 12;25:19-21. doi: 10.1016/j.mmcr.2019.06.002. eCollection 2019 Sep. PMID: 31249771; PMCID: PMC6584600.
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Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19.

Autonomic neuroscience : basic & clinical

Fischer L, Barop H, Ludin SM, Schaible HG.
PMID: 34894589
Auton Neurosci. 2021 Nov 10;237:102903. doi: 10.1016/j.autneu.2021.102903. Epub 2021 Nov 10.

Whereas the autonomic nervous system (ANS) and the immune system used to be assigned separate functions, it has now become clear that the ANS and the immune system (and thereby inflammatory cascades) work closely together. During an acute immune...

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Fischer L, Barop H, Ludin SM, et al. Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19. Auton Neurosci. 2021;237:102903doi: 10.1016/j.autneu.2021.102903.
Fischer, L., Barop, H., Ludin, S. M., & Schaible, H. G. (2021). Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19. Autonomic neuroscience : basic & clinical, 237102903. https://doi.org/10.1016/j.autneu.2021.102903
Fischer, Lorenz, et al. "Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19." Autonomic neuroscience : basic & clinical vol. 237 (2021): 102903. doi: https://doi.org/10.1016/j.autneu.2021.102903
Fischer L, Barop H, Ludin SM, Schaible HG. Regulation of acute reflectory hyperinflammation in viral and other diseases by means of stellate ganglion block. A conceptual view with a focus on Covid-19. Auton Neurosci. 2021 Nov 10;237:102903. doi: 10.1016/j.autneu.2021.102903. Epub 2021 Nov 10. PMID: 34894589.
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Showing 1 to 12 of 100 entries