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Risk factors and potential preventive measures for nephropatia epidemica in Sweden 2011-2012: a case-control study.

Infection ecology & epidemiology

Gherasim A, Hjertqvist M, Lundkvist Å, Kühlmann-Berenzon S, Carlson JV, Stenmark S, Widerström M, Österlund A, Boman H, Ahlm C, Wallensten A.
PMID: 26134289
Infect Ecol Epidemiol. 2015 Jun 30;5:27698. doi: 10.3402/iee.v5.27698. eCollection 2015.

INTRODUCTION: Nephropatia epidemica (NE), a relatively mild form of hemorrhagic fever with renal syndrome caused by the Puumala virus (PUUV), is endemic in northern Sweden. We aim to study the risk factors associated with NE in this region.METHODS: We...

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Gherasim A, Hjertqvist M, Lundkvist Å, et al. Risk factors and potential preventive measures for nephropatia epidemica in Sweden 2011-2012: a case-control study. Infect Ecol Epidemiol. 2015;5:27698doi: 10.3402/iee.v5.27698.
Gherasim, A., Hjertqvist, M., Lundkvist, �. �., Kühlmann-Berenzon, S., Carlson, J. V., Stenmark, S., Widerström, M., Österlund, A., Boman, H., Ahlm, C., & Wallensten, A. (2015). Risk factors and potential preventive measures for nephropatia epidemica in Sweden 2011-2012: a case-control study. Infection ecology & epidemiology, 527698. https://doi.org/10.3402/iee.v5.27698
Gherasim, Alin, et al. "Risk factors and potential preventive measures for nephropatia epidemica in Sweden 2011-2012: a case-control study." Infection ecology & epidemiology vol. 5 (2015): 27698. doi: https://doi.org/10.3402/iee.v5.27698
Gherasim A, Hjertqvist M, Lundkvist Å, Kühlmann-Berenzon S, Carlson JV, Stenmark S, Widerström M, Österlund A, Boman H, Ahlm C, Wallensten A. Risk factors and potential preventive measures for nephropatia epidemica in Sweden 2011-2012: a case-control study. Infect Ecol Epidemiol. 2015 Jun 30;5:27698. doi: 10.3402/iee.v5.27698. eCollection 2015. PMID: 26134289; PMCID: PMC4488335.
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Correction: Hemoglobin induced cell trauma indirectly influences endothelial TLR9 activity resulting in pulmonary vascular smooth muscle cell activation.

PloS one

Loomis Z, Eigenberger P, Redinius K, Lisk C, Karoor V, Nozik-Grayck E, Ferguson SK, Hassell K, Nuss R, Stenmark KR, Buehler PW, Irwin DC.
PMID: 28264019
PLoS One. 2017 Mar 06;12(3):e0173652. doi: 10.1371/journal.pone.0173652. eCollection 2017.

[This corrects the article DOI: 10.1371/journal.pone.0171219.].

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Loomis Z, Eigenberger P, Redinius K, et al. Correction: Hemoglobin induced cell trauma indirectly influences endothelial TLR9 activity resulting in pulmonary vascular smooth muscle cell activation. PLoS One. 2017;12(3):e0173652doi: 10.1371/journal.pone.0173652.
Loomis, Z., Eigenberger, P., Redinius, K., Lisk, C., Karoor, V., Nozik-Grayck, E., Ferguson, S. K., Hassell, K., Nuss, R., Stenmark, K. R., Buehler, P. W., & Irwin, D. C. (2017). Correction: Hemoglobin induced cell trauma indirectly influences endothelial TLR9 activity resulting in pulmonary vascular smooth muscle cell activation. PloS one, 12(3), e0173652. https://doi.org/10.1371/journal.pone.0173652
Loomis, Zoe, et al. "Correction: Hemoglobin induced cell trauma indirectly influences endothelial TLR9 activity resulting in pulmonary vascular smooth muscle cell activation." PloS one vol. 12,3 (2017): e0173652. doi: https://doi.org/10.1371/journal.pone.0173652
Loomis Z, Eigenberger P, Redinius K, Lisk C, Karoor V, Nozik-Grayck E, Ferguson SK, Hassell K, Nuss R, Stenmark KR, Buehler PW, Irwin DC. Correction: Hemoglobin induced cell trauma indirectly influences endothelial TLR9 activity resulting in pulmonary vascular smooth muscle cell activation. PLoS One. 2017 Mar 06;12(3):e0173652. doi: 10.1371/journal.pone.0173652. eCollection 2017. PMID: 28264019; PMCID: PMC5338835.
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Competition and Sensemaking in Ethical Situations.

Journal of applied social psychology

Caughron JJ, Antes AL, Stenmark CK, Thiel CE, Wang X, Mumford MD.
PMID: 26778850
J Appl Soc Psychol. 2013 Jul 01;43(7):1491-1507. doi: 10.1111/jasp.12141. Epub 2013 Jun 16.

Intra-organizational competition was examined in relation to ethicality. The effect of a competitor being an in-group versus and out-group member, competitor offering uncorroborated or corroborated information, and the impact of the competitor expressing selfish, pro-group, or pro-organizational level goals...

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Caughron JJ, Antes AL, Stenmark CK, et al. Competition and Sensemaking in Ethical Situations. J Appl Soc Psychol. 2013;43(7):1491-1507doi: 10.1111/jasp.12141.
Caughron, J. J., Antes, A. L., Stenmark, C. K., Thiel, C. E., Wang, X., & Mumford, M. D. (2013). Competition and Sensemaking in Ethical Situations. Journal of applied social psychology, 43(7), 1491-1507. https://doi.org/10.1111/jasp.12141
Caughron, Jay J, et al. "Competition and Sensemaking in Ethical Situations." Journal of applied social psychology vol. 43,7 (2013): 1491-1507. doi: https://doi.org/10.1111/jasp.12141
Caughron JJ, Antes AL, Stenmark CK, Thiel CE, Wang X, Mumford MD. Competition and Sensemaking in Ethical Situations. J Appl Soc Psychol. 2013 Jul 01;43(7):1491-1507. doi: 10.1111/jasp.12141. Epub 2013 Jun 16. PMID: 26778850; PMCID: PMC4712706.
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Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series).

Pulmonary circulation

Cavasin MA, Stenmark KR, McKinsey TA.
PMID: 25992271
Pulm Circ. 2015 Mar;5(1):63-72. doi: 10.1086/679700.

Reversible lysine acetylation has emerged as a critical mechanism for controlling the function of nucleosomal histones as well as diverse nonhistone proteins. Acetyl groups are conjugated to lysine residues in proteins by histone acetyltransferases and removed by histone deacetylases...

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Cavasin MA, Stenmark KR, McKinsey TA. Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series). Pulm Circ. 2015;5(1):63-72doi: 10.1086/679700.
Cavasin, M. A., Stenmark, K. R., & McKinsey, T. A. (2015). Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series). Pulmonary circulation, 5(1), 63-72. https://doi.org/10.1086/679700
Cavasin, Maria A, et al. "Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series)." Pulmonary circulation vol. 5,1 (2015): 63-72. doi: https://doi.org/10.1086/679700
Cavasin MA, Stenmark KR, McKinsey TA. Emerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series). Pulm Circ. 2015 Mar;5(1):63-72. doi: 10.1086/679700. PMID: 25992271; PMCID: PMC4405717.
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Applying Cases to Solve Ethical Problems: The Significance of Positive and Process-Oriented Reflection.

Ethics & behavior

Antes AL, Thiel CE, Martin LE, Stenmark CK, Connelly S, Devenport LD, Mumford MD.
PMID: 26257506
Ethics Behav. 2012;22(2):113-130. doi: 10.1080/10508422.2012.655646.

This study examined the role of reflection on personal cases for making ethical decisions with regard to new ethical problems. Participants assumed the position of a business manager in a hypothetical organization and solved ethical problems that might be...

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Antes AL, Thiel CE, Martin LE, et al. Applying Cases to Solve Ethical Problems: The Significance of Positive and Process-Oriented Reflection. Ethics Behav. 2012;22(2):113-130doi: 10.1080/10508422.2012.655646.
Antes, A. L., Thiel, C. E., Martin, L. E., Stenmark, C. K., Connelly, S., Devenport, L. D., & Mumford, M. D. (2012). Applying Cases to Solve Ethical Problems: The Significance of Positive and Process-Oriented Reflection. Ethics & behavior, 22(2), 113-130. https://doi.org/10.1080/10508422.2012.655646
Antes, Alison L, et al. "Applying Cases to Solve Ethical Problems: The Significance of Positive and Process-Oriented Reflection." Ethics & behavior vol. 22,2 (2012): 113-130. doi: https://doi.org/10.1080/10508422.2012.655646
Antes AL, Thiel CE, Martin LE, Stenmark CK, Connelly S, Devenport LD, Mumford MD. Applying Cases to Solve Ethical Problems: The Significance of Positive and Process-Oriented Reflection. Ethics Behav. 2012;22(2):113-130. doi: 10.1080/10508422.2012.655646. PMID: 26257506; PMCID: PMC4527578.
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Low cloud properties influenced by cosmic rays.

Physical review letters

Marsh ND, Svensmark H.
PMID: 11102172
Phys Rev Lett. 2000 Dec 04;85(23):5004-7. doi: 10.1103/PhysRevLett.85.5004.

The influence of solar variability on climate is currently uncertain. Recent observations have indicated a possible mechanism via the influence of solar modulated cosmic rays on global cloud cover. Surprisingly the influence of solar variability is strongest in low...

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Marsh ND, Svensmark H. Low cloud properties influenced by cosmic rays. Phys Rev Lett. 2000;85(23):5004-7doi: 10.1103/PhysRevLett.85.5004.
Marsh, N. D., & Svensmark, H. (2000). Low cloud properties influenced by cosmic rays. Physical review letters, 85(23), 5004-7. https://doi.org/10.1103/PhysRevLett.85.5004
Marsh, and Svensmark. "Low cloud properties influenced by cosmic rays." Physical review letters vol. 85,23 (2000): 5004-7. doi: https://doi.org/10.1103/PhysRevLett.85.5004
Marsh ND, Svensmark H. Low cloud properties influenced by cosmic rays. Phys Rev Lett. 2000 Dec 04;85(23):5004-7. doi: 10.1103/PhysRevLett.85.5004. PMID: 11102172.
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Constitutive Modeling of Anisotropic Finite-Deformation Hyperelastic Behaviors of Soft Materials Reinforced by Tortuous Fibers.

The international journal of structural changes in solids : mechanics and applications

Kao PH, Lammers SR, Hunter K, Stenmark KR, Shandas R, Qi HJ.
PMID: 21822502
Int J Struct Changes Sol. 2010 Apr;2(1):19-29.

Many biological materials are composites composed of a soft matrix reinforced with stiffer fibers. These stiffer fibers may have a tortuous shape and wind through the soft matrix. At small material deformation, these fibers deform in a bending mode...

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Kao PH, Lammers SR, Hunter K, et al. Constitutive Modeling of Anisotropic Finite-Deformation Hyperelastic Behaviors of Soft Materials Reinforced by Tortuous Fibers. Int J Struct Changes Sol. 2010;2(1):19-29
Kao, P. H., Lammers, S. R., Hunter, K., Stenmark, K. R., Shandas, R., & Qi, H. J. (2010). Constitutive Modeling of Anisotropic Finite-Deformation Hyperelastic Behaviors of Soft Materials Reinforced by Tortuous Fibers. The international journal of structural changes in solids : mechanics and applications, 2(1), 19-29.
Kao, Philip H, et al. "Constitutive Modeling of Anisotropic Finite-Deformation Hyperelastic Behaviors of Soft Materials Reinforced by Tortuous Fibers." The international journal of structural changes in solids : mechanics and applications vol. 2,1 (2010): 19-29.
Kao PH, Lammers SR, Hunter K, Stenmark KR, Shandas R, Qi HJ. Constitutive Modeling of Anisotropic Finite-Deformation Hyperelastic Behaviors of Soft Materials Reinforced by Tortuous Fibers. Int J Struct Changes Sol. 2010 Apr;2(1):19-29. PMID: 21822502; PMCID: PMC3150848.
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High Pulsatility Flow Induces Acute Endothelial Inflammation through Overpolarizing Cells to Activate NF-κB.

Cardiovascular engineering and technology

Li M, Tan Y, Stenmark KR, Tan W.
PMID: 23667401
Cardiovasc Eng Technol. 2013 Mar;4(1):26-38. doi: 10.1007/s13239-012-0115-5.

Large artery stiffening and small artery inflammation are both well-known pathological features of pulmonary and systemic hypertension, but the relationship between them has been seldom explored. We previously demonstrated that stiffening-induced high pulsatility flow stimulated a pro-inflammatory response in...

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Li M, Tan Y, Stenmark KR, et al. High Pulsatility Flow Induces Acute Endothelial Inflammation through Overpolarizing Cells to Activate NF-κB. Cardiovasc Eng Technol. 2013;4(1):26-38doi: 10.1007/s13239-012-0115-5.
Li, M., Tan, Y., Stenmark, K. R., & Tan, W. (2013). High Pulsatility Flow Induces Acute Endothelial Inflammation through Overpolarizing Cells to Activate NF-κB. Cardiovascular engineering and technology, 4(1), 26-38. https://doi.org/10.1007/s13239-012-0115-5
Li, Min, et al. "High Pulsatility Flow Induces Acute Endothelial Inflammation through Overpolarizing Cells to Activate NF-κB." Cardiovascular engineering and technology vol. 4,1 (2013): 26-38. doi: https://doi.org/10.1007/s13239-012-0115-5
Li M, Tan Y, Stenmark KR, Tan W. High Pulsatility Flow Induces Acute Endothelial Inflammation through Overpolarizing Cells to Activate NF-κB. Cardiovasc Eng Technol. 2013 Mar;4(1):26-38. doi: 10.1007/s13239-012-0115-5. PMID: 23667401; PMCID: PMC3646301.
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Perturbed period-doubling bifurcation. I. Theory.

Physical review. B, Condensed matter

Samuelsen MR, Svensmark H.
PMID: 9994240
Phys Rev B Condens Matter. 1990 Mar 01;41(7):4181-4188. doi: 10.1103/physrevb.41.4181.

No abstract available.

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Samuelsen MR, Svensmark H. Perturbed period-doubling bifurcation. I. Theory. Phys Rev B Condens Matter. 1990;41(7):4181-4188doi: 10.1103/physrevb.41.4181.
Samuelsen, M. R., & Svensmark, H. (1990). Perturbed period-doubling bifurcation. I. Theory. Physical review. B, Condensed matter, 41(7), 4181-4188. https://doi.org/10.1103/physrevb.41.4181
Samuelsen, and Svensmark. "Perturbed period-doubling bifurcation. I. Theory." Physical review. B, Condensed matter vol. 41,7 (1990): 4181-4188. doi: https://doi.org/10.1103/physrevb.41.4181
Samuelsen MR, Svensmark H. Perturbed period-doubling bifurcation. I. Theory. Phys Rev B Condens Matter. 1990 Mar 01;41(7):4181-4188. doi: 10.1103/physrevb.41.4181. PMID: 9994240.
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Perturbation treatment of boundary conditions for fluxon motion in long Josephson junctions.

Physical review. B, Condensed matter

Olsen OH, Pedersen NF, Samuelsen MR, Svensmark H, Welner aD.
PMID: 9937895
Phys Rev B Condens Matter. 1986 Jan 01;33(1):168-173. doi: 10.1103/physrevb.33.168.

No abstract available.

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Olsen OH, Pedersen NF, Samuelsen MR, et al. Perturbation treatment of boundary conditions for fluxon motion in long Josephson junctions. Phys Rev B Condens Matter. 1986;33(1):168-173doi: 10.1103/physrevb.33.168.
Olsen, O. H., Pedersen, N. F., Samuelsen, M. R., Svensmark, H., & Welner, a. D. (1986). Perturbation treatment of boundary conditions for fluxon motion in long Josephson junctions. Physical review. B, Condensed matter, 33(1), 168-173. https://doi.org/10.1103/physrevb.33.168
Olsen, et al. "Perturbation treatment of boundary conditions for fluxon motion in long Josephson junctions." Physical review. B, Condensed matter vol. 33,1 (1986): 168-173. doi: https://doi.org/10.1103/physrevb.33.168
Olsen OH, Pedersen NF, Samuelsen MR, Svensmark H, Welner aD. Perturbation treatment of boundary conditions for fluxon motion in long Josephson junctions. Phys Rev B Condens Matter. 1986 Jan 01;33(1):168-173. doi: 10.1103/physrevb.33.168. PMID: 9937895.
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Hypoxia-induced pulmonary hypertension and chronic lung disease: caveolin-1 dysfunction an important underlying feature.

Pulmonary circulation

Huang J, Frid M, Gewitz MH, Fallon JT, Brown D, Krafsur G, Stenmark K, Mathew R.
PMID: 30806156
Pulm Circ. 2019 Jan-Mar;9(1):2045894019837876. doi: 10.1177/2045894019837876.

Caveolin-1 (cav-1) has been shown to play a significant role in the pathogenesis of pulmonary hypertension (PH). In the monocrotaline model of PH, the loss of endothelial cav-1 as well as reciprocal activation of proliferative and anti-apoptotic pathways initiate...

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Huang J, Frid M, Gewitz MH, et al. Hypoxia-induced pulmonary hypertension and chronic lung disease: caveolin-1 dysfunction an important underlying feature. Pulm Circ. 2019;9(1):2045894019837876doi: 10.1177/2045894019837876.
Huang, J., Frid, M., Gewitz, M. H., Fallon, J. T., Brown, D., Krafsur, G., Stenmark, K., & Mathew, R. (2019). Hypoxia-induced pulmonary hypertension and chronic lung disease: caveolin-1 dysfunction an important underlying feature. Pulmonary circulation, 9(1), 2045894019837876. https://doi.org/10.1177/2045894019837876
Huang, Jing, et al. "Hypoxia-induced pulmonary hypertension and chronic lung disease: caveolin-1 dysfunction an important underlying feature." Pulmonary circulation vol. 9,1 (2019): 2045894019837876. doi: https://doi.org/10.1177/2045894019837876
Huang J, Frid M, Gewitz MH, Fallon JT, Brown D, Krafsur G, Stenmark K, Mathew R. Hypoxia-induced pulmonary hypertension and chronic lung disease: caveolin-1 dysfunction an important underlying feature. Pulm Circ. 2019 Jan-Mar;9(1):2045894019837876. doi: 10.1177/2045894019837876. PMID: 30806156; PMCID: PMC6434444.
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Outcomes after slipped capital femoral epiphysis: a population-based study with three-year follow-up.

Journal of children's orthopaedics

Herngren B, Stenmarker M, Enskär K, Hägglund G.
PMID: 30294367
J Child Orthop. 2018 Oct 01;12(5):434-443. doi: 10.1302/1863-2548.12.180067.

PURPOSE: To evaluate outcomes three years after treatment for slipped capital femoral epiphysis (SCFE): development of avascular necrosis (AVN), subsequent surgery, hip function and the contralateral hip.METHODS: This prospective cohort study included a total national population of 379 children...

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Herngren B, Stenmarker M, Enskär K, et al. Outcomes after slipped capital femoral epiphysis: a population-based study with three-year follow-up. J Child Orthop. 2018;12(5):434-443doi: 10.1302/1863-2548.12.180067.
Herngren, B., Stenmarker, M., Enskär, K., & Hägglund, G. (2018). Outcomes after slipped capital femoral epiphysis: a population-based study with three-year follow-up. Journal of children's orthopaedics, 12(5), 434-443. https://doi.org/10.1302/1863-2548.12.180067
Herngren, B, et al. "Outcomes after slipped capital femoral epiphysis: a population-based study with three-year follow-up." Journal of children's orthopaedics vol. 12,5 (2018): 434-443. doi: https://doi.org/10.1302/1863-2548.12.180067
Herngren B, Stenmarker M, Enskär K, Hägglund G. Outcomes after slipped capital femoral epiphysis: a population-based study with three-year follow-up. J Child Orthop. 2018 Oct 01;12(5):434-443. doi: 10.1302/1863-2548.12.180067. PMID: 30294367; PMCID: PMC6169552.
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Showing 1 to 12 of 122 entries