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Protective effect of quercetin on chloroquine-induced oxidative stress and hepatotoxicity in mice.

Malaria research and treatment

Kumar Mishra S, Singh P, Rath SK.
PMID: 23607047
Malar Res Treat. 2013;2013:141734. doi: 10.1155/2013/141734. Epub 2013 Mar 27.

The present study was aimed to find out the protective effect of quercetin on hepatotoxicity resulting by commonly used antimalarial drug chloroquine (CQ). Swiss albino mice were administered with different amounts of CQ ranging from human therapeutic equivalent of...

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Kumar Mishra S, Singh P, Rath SK. Protective effect of quercetin on chloroquine-induced oxidative stress and hepatotoxicity in mice. Malar Res Treat. 2013;2013:141734doi: 10.1155/2013/141734.
Kumar Mishra, S., Singh, P., & Rath, S. K. (2013). Protective effect of quercetin on chloroquine-induced oxidative stress and hepatotoxicity in mice. Malaria research and treatment, 2013141734. https://doi.org/10.1155/2013/141734
Kumar Mishra, Shrawan, et al. "Protective effect of quercetin on chloroquine-induced oxidative stress and hepatotoxicity in mice." Malaria research and treatment vol. 2013 (2013): 141734. doi: https://doi.org/10.1155/2013/141734
Kumar Mishra S, Singh P, Rath SK. Protective effect of quercetin on chloroquine-induced oxidative stress and hepatotoxicity in mice. Malar Res Treat. 2013;2013:141734. doi: 10.1155/2013/141734. Epub 2013 Mar 27. PMID: 23607047; PMCID: PMC3625570.
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The threat of antimalarial drug resistance.

Tropical diseases, travel medicine and vaccines

Hanboonkunupakarn B, White NJ.
PMID: 28883954
Trop Dis Travel Med Vaccines. 2016 Jul 07;2:10. doi: 10.1186/s40794-016-0027-8. eCollection 2016.

The battle between man and malaria has continued for thousands of years. Antimalarial drugs are essential weapons to fight the disease, but their efficacy is threatened by drug resistance which continues to emerge creating a major obstacle to malaria...

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Hanboonkunupakarn B, White NJ. The threat of antimalarial drug resistance. Trop Dis Travel Med Vaccines. 2016;2:10doi: 10.1186/s40794-016-0027-8.
Hanboonkunupakarn, B., & White, N. J. (2016). The threat of antimalarial drug resistance. Tropical diseases, travel medicine and vaccines, 210. https://doi.org/10.1186/s40794-016-0027-8
Hanboonkunupakarn, Borimas, and White, Nicholas J. "The threat of antimalarial drug resistance." Tropical diseases, travel medicine and vaccines vol. 2 (2016): 10. doi: https://doi.org/10.1186/s40794-016-0027-8
Hanboonkunupakarn B, White NJ. The threat of antimalarial drug resistance. Trop Dis Travel Med Vaccines. 2016 Jul 07;2:10. doi: 10.1186/s40794-016-0027-8. eCollection 2016. PMID: 28883954; PMCID: PMC5530994.
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Anti-plasmodial activity of ethanolic extract of root and stem back of Cassia sieberiana DC on mice.

Journal of intercultural ethnopharmacology

Abdulrazak N, Asiya UI, Usman NS, Unata IM, Farida A.
PMID: 26401393
J Intercult Ethnopharmacol. 2015 Apr-Jun;4(2):96-101. doi: 10.5455/jice.20141231014333. Epub 2015 Jan 20.

AIM: This study assessed within 4 days of suppressive test in vivo antimalarial activity of Ethanolic extract of root and stem bark of Cassia sieberiana DC against chloroquine sensitive strain of Plasmodium berghei NK65 in mice.METHODOLOGY: Two sets, each...

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Abdulrazak N, Asiya UI, Usman NS, et al. Anti-plasmodial activity of ethanolic extract of root and stem back of Cassia sieberiana DC on mice. J Intercult Ethnopharmacol. 2015;4(2):96-101doi: 10.5455/jice.20141231014333.
Abdulrazak, N., Asiya, U. I., Usman, N. S., Unata, I. M., & Farida, A. (2015). Anti-plasmodial activity of ethanolic extract of root and stem back of Cassia sieberiana DC on mice. Journal of intercultural ethnopharmacology, 4(2), 96-101. https://doi.org/10.5455/jice.20141231014333
Abdulrazak, Nuhu, et al. "Anti-plasmodial activity of ethanolic extract of root and stem back of Cassia sieberiana DC on mice." Journal of intercultural ethnopharmacology vol. 4,2 (2015): 96-101. doi: https://doi.org/10.5455/jice.20141231014333
Abdulrazak N, Asiya UI, Usman NS, Unata IM, Farida A. Anti-plasmodial activity of ethanolic extract of root and stem back of Cassia sieberiana DC on mice. J Intercult Ethnopharmacol. 2015 Apr-Jun;4(2):96-101. doi: 10.5455/jice.20141231014333. Epub 2015 Jan 20. PMID: 26401393; PMCID: PMC4566778.
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Synthesis and biological evaluation of tricyclic guanidine analogues of batzelladine K for antimalarial, antileishmanial, antibacterial, antifungal and anti-HIV activities.

Chemical biology & drug design

Ahmed N, Brahmbhatt KG, Khan SI, Jacob M, Tekwani BL, Sabde S, Mitra D, Singh IP, Khan IA, Bhutani KK.
PMID: 26404724
Chem Biol Drug Des. 2012 Jun 15; doi: 10.1111/j.1747-0285.2012.01427.x. Epub 2012 Jun 15.

Fifty analogues of batzelladine K were synthesized and evaluated for in vitro antimalarial (Plasmodium falciparum), antileishmanial (Leishmania donovani), antimicrobial (panel of bacteria and fungi), antiviral (HIV-1) activities. Analogues 14h and 20l exhibited potential antimalarial activity against chloroquine-sensitive D6 strain...

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Ahmed N, Brahmbhatt KG, Khan SI, et al. Synthesis and biological evaluation of tricyclic guanidine analogues of batzelladine K for antimalarial, antileishmanial, antibacterial, antifungal and anti-HIV activities. Chem Biol Drug Des. 2012;doi: 10.1111/j.1747-0285.2012.01427.x.
Ahmed, N., Brahmbhatt, K. G., Khan, S. I., Jacob, M., Tekwani, B. L., Sabde, S., Mitra, D., Singh, I. P., Khan, I. A., & Bhutani, K. K. (2012). Synthesis and biological evaluation of tricyclic guanidine analogues of batzelladine K for antimalarial, antileishmanial, antibacterial, antifungal and anti-HIV activities. Chemical biology & drug design, . https://doi.org/10.1111/j.1747-0285.2012.01427.x
Ahmed, Nafees, et al. "Synthesis and biological evaluation of tricyclic guanidine analogues of batzelladine K for antimalarial, antileishmanial, antibacterial, antifungal and anti-HIV activities." Chemical biology & drug design vol. (2012). doi: https://doi.org/10.1111/j.1747-0285.2012.01427.x
Ahmed N, Brahmbhatt KG, Khan SI, Jacob M, Tekwani BL, Sabde S, Mitra D, Singh IP, Khan IA, Bhutani KK. Synthesis and biological evaluation of tricyclic guanidine analogues of batzelladine K for antimalarial, antileishmanial, antibacterial, antifungal and anti-HIV activities. Chem Biol Drug Des. 2012 Jun 15; doi: 10.1111/j.1747-0285.2012.01427.x. Epub 2012 Jun 15. PMID: 26404724.
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Serum levels of anti-CCP antibodies, anti-MCV antibodies and RF IgA in the follow-up of patients with rheumatoid arthritis treated with rituximab.

Auto- immunity highlights

Fabris M, De Vita S, Blasone N, Visentini D, Pezzarini E, Pontarini E, Fabro C, Quartuccio L, Mazzolini S, Curcio F, Tonutti E.
PMID: 26000112
Auto Immun Highlights. 2010 Nov 04;1(2):87-94. doi: 10.1007/s13317-010-0013-5. eCollection 2010 Nov.

Rheumatoid arthritis (RA) is characterized by the presence of circulating rheumatoid factor (RF) and anticitrullinated peptide antibodies (ACPA), which are positive in about 70-80% of patients. APCA have a higher specificity and therefore a higher diagnostic power than RF,...

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Fabris M, De Vita S, Blasone N, et al. Serum levels of anti-CCP antibodies, anti-MCV antibodies and RF IgA in the follow-up of patients with rheumatoid arthritis treated with rituximab. Auto Immun Highlights. 2010;1(2):87-94doi: 10.1007/s13317-010-0013-5.
Fabris, M., De Vita, S., Blasone, N., Visentini, D., Pezzarini, E., Pontarini, E., Fabro, C., Quartuccio, L., Mazzolini, S., Curcio, F., & Tonutti, E. (2010). Serum levels of anti-CCP antibodies, anti-MCV antibodies and RF IgA in the follow-up of patients with rheumatoid arthritis treated with rituximab. Auto- immunity highlights, 1(2), 87-94. https://doi.org/10.1007/s13317-010-0013-5
Fabris, Martina, et al. "Serum levels of anti-CCP antibodies, anti-MCV antibodies and RF IgA in the follow-up of patients with rheumatoid arthritis treated with rituximab." Auto- immunity highlights vol. 1,2 (2010): 87-94. doi: https://doi.org/10.1007/s13317-010-0013-5
Fabris M, De Vita S, Blasone N, Visentini D, Pezzarini E, Pontarini E, Fabro C, Quartuccio L, Mazzolini S, Curcio F, Tonutti E. Serum levels of anti-CCP antibodies, anti-MCV antibodies and RF IgA in the follow-up of patients with rheumatoid arthritis treated with rituximab. Auto Immun Highlights. 2010 Nov 04;1(2):87-94. doi: 10.1007/s13317-010-0013-5. eCollection 2010 Nov. PMID: 26000112; PMCID: PMC4389048.
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Design and Synthesis of Novel Hybrid Molecules against Malaria.

International journal of medicinal chemistry

Lödige M, Hiersch L.
PMID: 25734014
Int J Med Chem. 2015;2015:458319. doi: 10.1155/2015/458319. Epub 2015 Feb 05.

The effective treatment of malaria can be very complex: Plasmodium parasites develop in multiple stages within a complex life cycle between mosquitoes as vectors and vertebrates as hosts. For the full and effective elimination of parasites, an effective drug...

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Lödige M, Hiersch L. Design and Synthesis of Novel Hybrid Molecules against Malaria. Int J Med Chem. 2015;2015:458319doi: 10.1155/2015/458319.
Lödige, M., & Hiersch, L. (2015). Design and Synthesis of Novel Hybrid Molecules against Malaria. International journal of medicinal chemistry, 2015458319. https://doi.org/10.1155/2015/458319
Lödige, Melanie, and Hiersch, Luisa. "Design and Synthesis of Novel Hybrid Molecules against Malaria." International journal of medicinal chemistry vol. 2015 (2015): 458319. doi: https://doi.org/10.1155/2015/458319
Lödige M, Hiersch L. Design and Synthesis of Novel Hybrid Molecules against Malaria. Int J Med Chem. 2015;2015:458319. doi: 10.1155/2015/458319. Epub 2015 Feb 05. PMID: 25734014; PMCID: PMC4334980.
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Influence of quinacrine and chloroquine on the in vitro 3'-azido-3'-deoxythymidine antiretroviral effect.

AIDS research and therapy

Torres KJ, Reyes-Terán G, Sotelo J, Jung-Cook H, Aguirre-Cruz L.
PMID: 25788967
AIDS Res Ther. 2015 Mar 19;12:7. doi: 10.1186/s12981-015-0048-9. eCollection 2015.

BACKGROUND: Antimalarials quinacrine (Qc) and chloroquine (Cq) intercalate DNA, potentiate the activity of other drugs and have lysosomotropic, anti-inflammatory and antiviral activities that could increase the effect of the 3'-azido-3'-deoxythymidine (AZT) antiretroviral agent. The aim of the current study...

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Torres KJ, Reyes-Terán G, Sotelo J, et al. Influence of quinacrine and chloroquine on the in vitro 3'-azido-3'-deoxythymidine antiretroviral effect. AIDS Res Ther. 2015;12:7doi: 10.1186/s12981-015-0048-9.
Torres, K. J., Reyes-Terán, G., Sotelo, J., Jung-Cook, H., & Aguirre-Cruz, L. (2015). Influence of quinacrine and chloroquine on the in vitro 3'-azido-3'-deoxythymidine antiretroviral effect. AIDS research and therapy, 127. https://doi.org/10.1186/s12981-015-0048-9
Torres, Klintsy J, et al. "Influence of quinacrine and chloroquine on the in vitro 3'-azido-3'-deoxythymidine antiretroviral effect." AIDS research and therapy vol. 12 (2015): 7. doi: https://doi.org/10.1186/s12981-015-0048-9
Torres KJ, Reyes-Terán G, Sotelo J, Jung-Cook H, Aguirre-Cruz L. Influence of quinacrine and chloroquine on the in vitro 3'-azido-3'-deoxythymidine antiretroviral effect. AIDS Res Ther. 2015 Mar 19;12:7. doi: 10.1186/s12981-015-0048-9. eCollection 2015. PMID: 25788967; PMCID: PMC4364635.
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Frontal Fibrosing Alopecia Coexisting with Lupus Erythematosus: Poor Response to Hydroxychloroquine.

Skin appendage disorders

Contin LA, Martins da Costa Marques ER, Noriega L.
PMID: 28232926
Skin Appendage Disord. 2017 Jan;2(3):162-165. doi: 10.1159/000452925. Epub 2016 Nov 12.

Lupus erythematosus, especially the discoid form, and lichen planopilaris may be associated and can occur in different topographies (coexistence) or in the same lesion (lupus eythematosus/lichen planus overlap syndrome). Frontal fibrosing alopecia is considered a variant form of lichen...

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Contin LA, Martins da Costa Marques ER, Noriega L. Frontal Fibrosing Alopecia Coexisting with Lupus Erythematosus: Poor Response to Hydroxychloroquine. Skin Appendage Disord. 2016;2(3):162-165doi: 10.1159/000452925.
Contin, L. A., Martins da Costa Marques, E. R., & Noriega, L. (2017). Frontal Fibrosing Alopecia Coexisting with Lupus Erythematosus: Poor Response to Hydroxychloroquine. Skin appendage disorders, 2(3), 162-165. https://doi.org/10.1159/000452925
Contin, Letícia Arsie, et al. "Frontal Fibrosing Alopecia Coexisting with Lupus Erythematosus: Poor Response to Hydroxychloroquine." Skin appendage disorders vol. 2,3 (2017): 162-165. doi: https://doi.org/10.1159/000452925
Contin LA, Martins da Costa Marques ER, Noriega L. Frontal Fibrosing Alopecia Coexisting with Lupus Erythematosus: Poor Response to Hydroxychloroquine. Skin Appendage Disord. 2017 Jan;2(3):162-165. doi: 10.1159/000452925. Epub 2016 Nov 12. PMID: 28232926; PMCID: PMC5264358.
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The Potential of β Carbolin Alkaloids to Hinder Growth and Reverse Chloroquine Resistance in Plasmodium falciparum.

Iranian journal of parasitology

Ibraheem ZO, Abdul Majid R, Mohd Noor S, Mohd Sidek H, Basir R.
PMID: 26811724
Iran J Parasitol. 2015 Oct-Dec;10(4):577-83.

BACKGROUND: Nowadays, scourge of malaria as a fatalistic disease has increased due to emergence of drug resistance and tolerance among different strains of Plasmodium falciparum. Emergence of chloroquine (CQ) resistance has worsened the calamity as CQ is still considered...

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Ibraheem ZO, Abdul Majid R, Mohd Noor S, et al. The Potential of β Carbolin Alkaloids to Hinder Growth and Reverse Chloroquine Resistance in Plasmodium falciparum. Iran J Parasitol. 2015;10(4):577-83
Ibraheem, Z. O., Abdul Majid, R., Mohd Noor, S., Mohd Sidek, H., & Basir, R. (2015). The Potential of β Carbolin Alkaloids to Hinder Growth and Reverse Chloroquine Resistance in Plasmodium falciparum. Iranian journal of parasitology, 10(4), 577-83.
Ibraheem, Zaid O, et al. "The Potential of β Carbolin Alkaloids to Hinder Growth and Reverse Chloroquine Resistance in Plasmodium falciparum." Iranian journal of parasitology vol. 10,4 (2015): 577-83.
Ibraheem ZO, Abdul Majid R, Mohd Noor S, Mohd Sidek H, Basir R. The Potential of β Carbolin Alkaloids to Hinder Growth and Reverse Chloroquine Resistance in Plasmodium falciparum. Iran J Parasitol. 2015 Oct-Dec;10(4):577-83. PMID: 26811724; PMCID: PMC4724834.
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Autophagy inhibition impairs the epithelial-mesenchymal transition and enhances cisplatin sensitivity in nasopharyngeal carcinoma.

Oncology letters

Su Z, Li G, Liu C, Ren S, Deng T, Zhang S, Tian Y, Liu Y, Qiu Y.
PMID: 28599416
Oncol Lett. 2017 Jun;13(6):4147-4154. doi: 10.3892/ol.2017.5963. Epub 2017 Mar 31.

Drug resistance restricts the efficacy of cisplatin in the treatment of nasopharyngeal carcinoma (NPC). Increasing evidence indicates that autophagy and the epithelial-mesenchymal transition (EMT) participate in cancer progression and drug sensitivity. The aim of the present study was to...

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Su Z, Li G, Liu C, et al. Autophagy inhibition impairs the epithelial-mesenchymal transition and enhances cisplatin sensitivity in nasopharyngeal carcinoma. Oncol Lett. 2017;13(6):4147-4154doi: 10.3892/ol.2017.5963.
Su, Z., Li, G., Liu, C., Ren, S., Deng, T., Zhang, S., Tian, Y., Liu, Y., & Qiu, Y. (2017). Autophagy inhibition impairs the epithelial-mesenchymal transition and enhances cisplatin sensitivity in nasopharyngeal carcinoma. Oncology letters, 13(6), 4147-4154. https://doi.org/10.3892/ol.2017.5963
Su, Zhongwu, et al. "Autophagy inhibition impairs the epithelial-mesenchymal transition and enhances cisplatin sensitivity in nasopharyngeal carcinoma." Oncology letters vol. 13,6 (2017): 4147-4154. doi: https://doi.org/10.3892/ol.2017.5963
Su Z, Li G, Liu C, Ren S, Deng T, Zhang S, Tian Y, Liu Y, Qiu Y. Autophagy inhibition impairs the epithelial-mesenchymal transition and enhances cisplatin sensitivity in nasopharyngeal carcinoma. Oncol Lett. 2017 Jun;13(6):4147-4154. doi: 10.3892/ol.2017.5963. Epub 2017 Mar 31. PMID: 28599416; PMCID: PMC5453060.
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The .

PeerJ

Songsungthong W, Kulawonganunchai S, Wilantho A, Tongsima S, Koonyosying P, Uthaipibull C, Kamchonwongpaisan S, Shaw PJ.
PMID: 29018598
PeerJ. 2017 Oct 05;5:e3766. doi: 10.7717/peerj.3766. eCollection 2017.

BACKGROUND: The current first line drugs for treating uncomplicated malaria are artemisinin (ART) combination therapies. However, METHODS: The RESULTS: We found that DISCUSSION: The variants identified in

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Songsungthong W, Kulawonganunchai S, Wilantho A, et al. The . PeerJ. 2017;5:e3766doi: 10.7717/peerj.3766.
Songsungthong, W., Kulawonganunchai, S., Wilantho, A., Tongsima, S., Koonyosying, P., Uthaipibull, C., Kamchonwongpaisan, S., & Shaw, P. J. (2017). The . PeerJ, 5e3766. https://doi.org/10.7717/peerj.3766
Songsungthong, Warangkhana, et al. "The ." PeerJ vol. 5 (2017): e3766. doi: https://doi.org/10.7717/peerj.3766
Songsungthong W, Kulawonganunchai S, Wilantho A, Tongsima S, Koonyosying P, Uthaipibull C, Kamchonwongpaisan S, Shaw PJ. The . PeerJ. 2017 Oct 05;5:e3766. doi: 10.7717/peerj.3766. eCollection 2017. PMID: 29018598; PMCID: PMC5632537.
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Allicin activates autophagic cell death to alleviate the malignant development of thyroid cancer.

Experimental and therapeutic medicine

Xiang Y, Zhao J, Zhao M, Wang K.
PMID: 29545880
Exp Ther Med. 2018 Apr;15(4):3537-3543. doi: 10.3892/etm.2018.5828. Epub 2018 Feb 01.

Allicin has been reported to inhibit cancer cell proliferation, induce cell apoptosis and enhance the accumulation of reactive oxygen species. However, it has remained elusive whether allicin improves multidrug resistance in thyroid cancer cells through modulating autophagy. The present...

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Xiang Y, Zhao J, Zhao M, et al. Allicin activates autophagic cell death to alleviate the malignant development of thyroid cancer. Exp Ther Med. 2018;15(4):3537-3543doi: 10.3892/etm.2018.5828.
Xiang, Y., Zhao, J., Zhao, M., & Wang, K. (2018). Allicin activates autophagic cell death to alleviate the malignant development of thyroid cancer. Experimental and therapeutic medicine, 15(4), 3537-3543. https://doi.org/10.3892/etm.2018.5828
Xiang, Yangfeng, et al. "Allicin activates autophagic cell death to alleviate the malignant development of thyroid cancer." Experimental and therapeutic medicine vol. 15,4 (2018): 3537-3543. doi: https://doi.org/10.3892/etm.2018.5828
Xiang Y, Zhao J, Zhao M, Wang K. Allicin activates autophagic cell death to alleviate the malignant development of thyroid cancer. Exp Ther Med. 2018 Apr;15(4):3537-3543. doi: 10.3892/etm.2018.5828. Epub 2018 Feb 01. PMID: 29545880; PMCID: PMC5841025.
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Showing 1 to 12 of 1595 entries