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Data on carotid intima-media thickness and lipoprotein subclasses in type 1 diabetes from the Diabetes Control and Complications Trial and the Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC).

Data in brief

Basu A, Jenkins AJ, Zhang Y, Stoner JA, Klein RL, Lopes-Virella MF, Timothy Garvey W, Lyons TJ.
PMID: 26759826
Data Brief. 2015 Nov 22;6:33-8. doi: 10.1016/j.dib.2015.11.036. eCollection 2016 Mar.

Type 1 diabetes (T1DM) is associated with increased risk of macrovascular complications. We examined longitudinal associations of serum conventional lipids and nuclear magnetic resonance (NMR)-determined lipoprotein subclasses with carotid intima-media thickness (IMT) in adults with T1DM (n=455) enrolled in...

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Basu A, Jenkins AJ, Zhang Y, et al. Data on carotid intima-media thickness and lipoprotein subclasses in type 1 diabetes from the Diabetes Control and Complications Trial and the Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC). Data Brief. 2015;6:33-8doi: 10.1016/j.dib.2015.11.036.
Basu, A., Jenkins, A. J., Zhang, Y., Stoner, J. A., Klein, R. L., Lopes-Virella, M. F., Timothy Garvey, W., Lyons, T. J. (2016). Data on carotid intima-media thickness and lipoprotein subclasses in type 1 diabetes from the Diabetes Control and Complications Trial and the Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC). Data in brief, 633-8. https://doi.org/10.1016/j.dib.2015.11.036
Basu, Arpita, et al. "Data on carotid intima-media thickness and lipoprotein subclasses in type 1 diabetes from the Diabetes Control and Complications Trial and the Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC)." Data in brief vol. 6 (2016): 33-8. doi: https://doi.org/10.1016/j.dib.2015.11.036
Basu A, Jenkins AJ, Zhang Y, Stoner JA, Klein RL, Lopes-Virella MF, Timothy Garvey W, Lyons TJ. Data on carotid intima-media thickness and lipoprotein subclasses in type 1 diabetes from the Diabetes Control and Complications Trial and the Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC). Data Brief. 2015 Nov 22;6:33-8. doi: 10.1016/j.dib.2015.11.036. eCollection 2016 Mar. PMID: 26759826; PMCID: PMC4683326.
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The multifaceted nature of retinoid transport and metabolism.

Hepatobiliary surgery and nutrition

Li Y, Wongsiriroj N, Blaner WS.
PMID: 25019074
Hepatobiliary Surg Nutr. 2014 Jun;3(3):126-39. doi: 10.3978/j.issn.2304-3881.2014.05.04.

Since their discovery over a century ago, retinoids have been the most studied of the fat-soluble vitamins. Unlike most vitamins, retinoids are stored at relatively high concentrations in the body to buffer against nutritional insufficiency. Until recently, it was...

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Li Y, Wongsiriroj N, Blaner WS. The multifaceted nature of retinoid transport and metabolism. Hepatobiliary Surg Nutr. 2014;3(3):126-39doi: 10.3978/j.issn.2304-3881.2014.05.04.
Li, Y., Wongsiriroj, N., & Blaner, W. S. (2014). The multifaceted nature of retinoid transport and metabolism. Hepatobiliary surgery and nutrition, 3(3), 126-39. https://doi.org/10.3978/j.issn.2304-3881.2014.05.04
Li, Yang, et al. "The multifaceted nature of retinoid transport and metabolism." Hepatobiliary surgery and nutrition vol. 3,3 (2014): 126-39. doi: https://doi.org/10.3978/j.issn.2304-3881.2014.05.04
Li Y, Wongsiriroj N, Blaner WS. The multifaceted nature of retinoid transport and metabolism. Hepatobiliary Surg Nutr. 2014 Jun;3(3):126-39. doi: 10.3978/j.issn.2304-3881.2014.05.04. PMID: 25019074; PMCID: PMC4073323.
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Plasma triglyceride and high density lipoprotein cholesterol are poor surrogate markers of pro-atherogenic chylomicron remnant homeostasis in subjects with the metabolic syndrome.

Lipids in health and disease

Irawati D, Mamo JC, Dhaliwal SS, Soares MJ, Slivkoff-Clark KM, James AP.
PMID: 27686975
Lipids Health Dis. 2016 Sep 29;15(1):169. doi: 10.1186/s12944-016-0330-4.

BACKGROUND: Subjects with metabolic syndrome (MetS) exhibit impaired lipoprotein metabolism and have an increased risk of cardiovascular disease. Although the risk is attributed primarily to the risk associated with individual components, it is also likely affected by other associated...

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Irawati D, Mamo JC, Dhaliwal SS, et al. Plasma triglyceride and high density lipoprotein cholesterol are poor surrogate markers of pro-atherogenic chylomicron remnant homeostasis in subjects with the metabolic syndrome. Lipids Health Dis. 2016;15(1):169doi: 10.1186/s12944-016-0330-4.
Irawati, D., Mamo, J. C., Dhaliwal, S. S., Soares, M. J., Slivkoff-Clark, K. M., & James, A. P. (2016). Plasma triglyceride and high density lipoprotein cholesterol are poor surrogate markers of pro-atherogenic chylomicron remnant homeostasis in subjects with the metabolic syndrome. Lipids in health and disease, 15(1), 169. https://doi.org/10.1186/s12944-016-0330-4
Irawati, Deasy, et al. "Plasma triglyceride and high density lipoprotein cholesterol are poor surrogate markers of pro-atherogenic chylomicron remnant homeostasis in subjects with the metabolic syndrome." Lipids in health and disease vol. 15,1 (2016): 169. doi: https://doi.org/10.1186/s12944-016-0330-4
Irawati D, Mamo JC, Dhaliwal SS, Soares MJ, Slivkoff-Clark KM, James AP. Plasma triglyceride and high density lipoprotein cholesterol are poor surrogate markers of pro-atherogenic chylomicron remnant homeostasis in subjects with the metabolic syndrome. Lipids Health Dis. 2016 Sep 29;15(1):169. doi: 10.1186/s12944-016-0330-4. PMID: 27686975; PMCID: PMC5043522.
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A Cause of Bilateral Chylothorax: A Case of Mesothelioma without Pleural Involvement during Initial Diagnosis.

Case reports in pulmonology

Kurtipek E, Eren Karanis Mİ, Düzgün N, Esme H, Çaycı M.
PMID: 26451268
Case Rep Pulmonol. 2015;2015:962504. doi: 10.1155/2015/962504. Epub 2015 Sep 16.

Chylothorax is characterized by fluid accumulation in the pleural cavity containing chylomicrons due to disruption of lymphatic drainage in the thoracic ductus and development of chylothorax. A 60-year-old male patient presented to our clinic with shortness of breath and...

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Kurtipek E, Eren Karanis Mİ, Düzgün N, et al. A Cause of Bilateral Chylothorax: A Case of Mesothelioma without Pleural Involvement during Initial Diagnosis. Case Rep Pulmonol. 2015;2015:962504doi: 10.1155/2015/962504.
Kurtipek, E., Eren Karanis, M. �. �., Düzgün, N., Esme, H., & Çaycı, M. (2015). A Cause of Bilateral Chylothorax: A Case of Mesothelioma without Pleural Involvement during Initial Diagnosis. Case reports in pulmonology, 2015962504. https://doi.org/10.1155/2015/962504
Kurtipek, Ercan, et al. "A Cause of Bilateral Chylothorax: A Case of Mesothelioma without Pleural Involvement during Initial Diagnosis." Case reports in pulmonology vol. 2015 (2015): 962504. doi: https://doi.org/10.1155/2015/962504
Kurtipek E, Eren Karanis Mİ, Düzgün N, Esme H, Çaycı M. A Cause of Bilateral Chylothorax: A Case of Mesothelioma without Pleural Involvement during Initial Diagnosis. Case Rep Pulmonol. 2015;2015:962504. doi: 10.1155/2015/962504. Epub 2015 Sep 16. PMID: 26451268; PMCID: PMC4588346.
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Obesity and Insulin Resistance Are the Main Determinants of Postprandial Lipoprotein Dysmetabolism in Polycystic Ovary Syndrome.

International journal of endocrinology

Kyaw Tun T, McGowan A, Phelan N, Correia N, Boran G, O'Connor AL, Roche HM, Gibney J.
PMID: 26989412
Int J Endocrinol. 2016;2016:9545239. doi: 10.1155/2016/9545239. Epub 2016 Feb 18.

Postprandial dyslipidaemia may be a plausible mechanism by which polycystic ovary syndrome (PCOS) increases cardiovascular risk. We sought to investigate whether the postprandial glucose and insulin and lipid and lipoprotein responses, including that of apolipoprotein B-48 (apoB-48) containing chylomicrons,...

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Kyaw Tun T, McGowan A, Phelan N, et al. Obesity and Insulin Resistance Are the Main Determinants of Postprandial Lipoprotein Dysmetabolism in Polycystic Ovary Syndrome. Int J Endocrinol. 2016;2016:9545239doi: 10.1155/2016/9545239.
Kyaw Tun, T., McGowan, A., Phelan, N., Correia, N., Boran, G., O'Connor, A. L., Roche, H. M., & Gibney, J. (2016). Obesity and Insulin Resistance Are the Main Determinants of Postprandial Lipoprotein Dysmetabolism in Polycystic Ovary Syndrome. International journal of endocrinology, 20169545239. https://doi.org/10.1155/2016/9545239
Kyaw Tun, Tommy, et al. "Obesity and Insulin Resistance Are the Main Determinants of Postprandial Lipoprotein Dysmetabolism in Polycystic Ovary Syndrome." International journal of endocrinology vol. 2016 (2016): 9545239. doi: https://doi.org/10.1155/2016/9545239
Kyaw Tun T, McGowan A, Phelan N, Correia N, Boran G, O'Connor AL, Roche HM, Gibney J. Obesity and Insulin Resistance Are the Main Determinants of Postprandial Lipoprotein Dysmetabolism in Polycystic Ovary Syndrome. Int J Endocrinol. 2016;2016:9545239. doi: 10.1155/2016/9545239. Epub 2016 Feb 18. PMID: 26989412; PMCID: PMC4775795.
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The pathophysiology of intestinal lipoprotein production.

Frontiers in physiology

Giammanco A, Cefalù AB, Noto D, Averna MR.
PMID: 25852563
Front Physiol. 2015 Mar 20;6:61. doi: 10.3389/fphys.2015.00061. eCollection 2015.

Intestinal lipoprotein production is a multistep process, essential for the absorption of dietary fats and fat-soluble vitamins. Chylomicron assembly begins in the endoplasmic reticulum with the formation of primordial, phospholipids-rich particles that are then transported to the Golgi for...

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Giammanco A, Cefalù AB, Noto D, et al. The pathophysiology of intestinal lipoprotein production. Front Physiol. 2015;6:61doi: 10.3389/fphys.2015.00061.
Giammanco, A., Cefalù, A. B., Noto, D., & Averna, M. R. (2015). The pathophysiology of intestinal lipoprotein production. Frontiers in physiology, 661. https://doi.org/10.3389/fphys.2015.00061
Giammanco, Antonina, et al. "The pathophysiology of intestinal lipoprotein production." Frontiers in physiology vol. 6 (2015): 61. doi: https://doi.org/10.3389/fphys.2015.00061
Giammanco A, Cefalù AB, Noto D, Averna MR. The pathophysiology of intestinal lipoprotein production. Front Physiol. 2015 Mar 20;6:61. doi: 10.3389/fphys.2015.00061. eCollection 2015. PMID: 25852563; PMCID: PMC4367171.
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Mining the genome for lipid genes.

Biochimica et biophysica acta

Kuivenhoven JA, Hegele RA.
PMID: 24798233
Biochim Biophys Acta. 2014 Oct;1842(10):1993-2009. doi: 10.1016/j.bbadis.2014.04.028. Epub 2014 May 02.

Mining of the genome for lipid genes has since the early 1970s helped to shape our understanding of how triglycerides are packaged (in chylomicrons), repackaged (in very low density lipoproteins; VLDL), and hydrolyzed, and also how remnant and low-density...

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Kuivenhoven JA, Hegele RA. Mining the genome for lipid genes. Biochim Biophys Acta. 2014;1842(10):1993-2009doi: 10.1016/j.bbadis.2014.04.028.
Kuivenhoven, J. A., & Hegele, R. A. (2014). Mining the genome for lipid genes. Biochimica et biophysica acta, 1842(10), 1993-2009. https://doi.org/10.1016/j.bbadis.2014.04.028
Kuivenhoven, Jan Albert, and Hegele, Robert A. "Mining the genome for lipid genes." Biochimica et biophysica acta vol. 1842,10 (2014): 1993-2009. doi: https://doi.org/10.1016/j.bbadis.2014.04.028
Kuivenhoven JA, Hegele RA. Mining the genome for lipid genes. Biochim Biophys Acta. 2014 Oct;1842(10):1993-2009. doi: 10.1016/j.bbadis.2014.04.028. Epub 2014 May 02. PMID: 24798233.
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Unleashing the untold and misunderstood observations on vitamin E.

Genes & nutrition

Gee PT.
PMID: 21437026
Genes Nutr. 2011 Feb;6(1):5-16. doi: 10.1007/s12263-010-0180-z. Epub 2010 Jul 20.

Paradoxically, meta-analysis of human randomized controlled trials revealed that natural but not synthetic α-tocopherol supplementation significantly increases all-cause mortality at 95% confidence interval. The root cause was that natural α-tocopherol supplementation significantly depressed bioavailability of other forms of vitamin...

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Gee PT. Unleashing the untold and misunderstood observations on vitamin E. Genes Nutr. 2010;6(1):5-16doi: 10.1007/s12263-010-0180-z.
Gee, P. T. (2011). Unleashing the untold and misunderstood observations on vitamin E. Genes & nutrition, 6(1), 5-16. https://doi.org/10.1007/s12263-010-0180-z
Gee, Ping Tou. "Unleashing the untold and misunderstood observations on vitamin E." Genes & nutrition vol. 6,1 (2011): 5-16. doi: https://doi.org/10.1007/s12263-010-0180-z
Gee PT. Unleashing the untold and misunderstood observations on vitamin E. Genes Nutr. 2011 Feb;6(1):5-16. doi: 10.1007/s12263-010-0180-z. Epub 2010 Jul 20. PMID: 21437026; PMCID: PMC3040795.
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Removal of Chylomicron Remnants from the Bloodstream is Delayed in Aged Subjects.

Aging and disease

Vinagre CG, Freitas FR, de Mesquita CH, Vinagre JC, Mariani AC, Kalil-Filho R, Maranhão RC.
PMID: 30090662
Aging Dis. 2018 Aug 01;9(4):748-754. doi: 10.14336/AD.2017.1003. eCollection 2018 Aug.

Dietary fats absorbed in the intestine are transported in the circulation as chylomicrons and remnants that have atherogenic potential. Although postprandial lipidemia is increased in older subjects, the specific chylomicron metabolism has not been explored in older subjects nor...

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Vinagre CG, Freitas FR, de Mesquita CH, et al. Removal of Chylomicron Remnants from the Bloodstream is Delayed in Aged Subjects. Aging Dis. 2018;9(4):748-754doi: 10.14336/AD.2017.1003.
Vinagre, C. G., Freitas, F. R., de Mesquita, C. H., Vinagre, J. C., Mariani, A. C., Kalil-Filho, R., & Maranhão, R. C. (2018). Removal of Chylomicron Remnants from the Bloodstream is Delayed in Aged Subjects. Aging and disease, 9(4), 748-754. https://doi.org/10.14336/AD.2017.1003
Vinagre, Carmen G, et al. "Removal of Chylomicron Remnants from the Bloodstream is Delayed in Aged Subjects." Aging and disease vol. 9,4 (2018): 748-754. doi: https://doi.org/10.14336/AD.2017.1003
Vinagre CG, Freitas FR, de Mesquita CH, Vinagre JC, Mariani AC, Kalil-Filho R, Maranhão RC. Removal of Chylomicron Remnants from the Bloodstream is Delayed in Aged Subjects. Aging Dis. 2018 Aug 01;9(4):748-754. doi: 10.14336/AD.2017.1003. eCollection 2018 Aug. PMID: 30090662; PMCID: PMC6065288.
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[The cognitive biological function, difference in etiology and generality of pathogenesis of metabolic pandemics. The metabolism regulation in carnivorous and herbivorous animals under effect of environment factors.].

Klinicheskaia laboratornaia diagnostika

Titov VN.
PMID: 31509653
Klin Lab Diagn. 2017;62(5):260-271. doi: 10.18821/0869-2084-2017-62-5-260-271.

In phylogenesis, carnivorous animals initially and later herbivorous animals formed separate transfer of exogenous fatty acids in blood, predominantly palmitic acid and later - endogenous oleic acid pool, synthesized in hepatocytes from glucose. In vivo, exogenous fatty acids transfer...

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Titov VN. [The cognitive biological function, difference in etiology and generality of pathogenesis of metabolic pandemics. The metabolism regulation in carnivorous and herbivorous animals under effect of environment factors.]. Klin Lab Diagn. 2017;62(5):260-271doi: 10.18821/0869-2084-2017-62-5-260-271.
Titov, V. N. (2017). [The cognitive biological function, difference in etiology and generality of pathogenesis of metabolic pandemics. The metabolism regulation in carnivorous and herbivorous animals under effect of environment factors.]. Klinicheskaia laboratornaia diagnostika, 62(5), 260-271. https://doi.org/10.18821/0869-2084-2017-62-5-260-271
Titov, V N. "[The cognitive biological function, difference in etiology and generality of pathogenesis of metabolic pandemics. The metabolism regulation in carnivorous and herbivorous animals under effect of environment factors.]." Klinicheskaia laboratornaia diagnostika vol. 62,5 (2017): 260-271. doi: https://doi.org/10.18821/0869-2084-2017-62-5-260-271
Titov VN. [The cognitive biological function, difference in etiology and generality of pathogenesis of metabolic pandemics. The metabolism regulation in carnivorous and herbivorous animals under effect of environment factors.]. Klin Lab Diagn. 2017;62(5):260-271. doi: 10.18821/0869-2084-2017-62-5-260-271. Russian. PMID: 31509653.
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The Effect of Partly Replacing Vegetable Fat with Bovine Milk Fat in Infant Formula on Postprandial Lipid and Energy Metabolism: A Proof-of-principle Study in Healthy Young Male Adults.

Molecular nutrition & food research

Hageman JHJ, Erdõs B, Keijer J, Adriaens M, de Wit B, Stañková B, Tvrzická E, Arts ICW, Nieuwenhuizen AG.
PMID: 33682997
Mol Nutr Food Res. 2021 May;65(9):e2000848. doi: 10.1002/mnfr.202000848. Epub 2021 Apr 01.

SCOPE: Infant formula (IF) uses besides vegetable fats also bovine milk fat, which differs in triacylglycerol (TAG) structure. Furthermore, it differs in fatty acid (FA) composition. Whether changing fat source in IF affects postprandial energy metabolism, lipemic response, and...

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Hageman JHJ, Erdõs B, Keijer J, et al. The Effect of Partly Replacing Vegetable Fat with Bovine Milk Fat in Infant Formula on Postprandial Lipid and Energy Metabolism: A Proof-of-principle Study in Healthy Young Male Adults. Mol Nutr Food Res. 2021;65(9):e2000848doi: 10.1002/mnfr.202000848.
Hageman, J. H. J., Erdõs, B., Keijer, J., Adriaens, M., de Wit, B., Stañková, B., Tvrzická, E., Arts, I. C. W., & Nieuwenhuizen, A. G. (2021). The Effect of Partly Replacing Vegetable Fat with Bovine Milk Fat in Infant Formula on Postprandial Lipid and Energy Metabolism: A Proof-of-principle Study in Healthy Young Male Adults. Molecular nutrition & food research, 65(9), e2000848. https://doi.org/10.1002/mnfr.202000848
Hageman, Jeske H J, et al. "The Effect of Partly Replacing Vegetable Fat with Bovine Milk Fat in Infant Formula on Postprandial Lipid and Energy Metabolism: A Proof-of-principle Study in Healthy Young Male Adults." Molecular nutrition & food research vol. 65,9 (2021): e2000848. doi: https://doi.org/10.1002/mnfr.202000848
Hageman JHJ, Erdõs B, Keijer J, Adriaens M, de Wit B, Stañková B, Tvrzická E, Arts ICW, Nieuwenhuizen AG. The Effect of Partly Replacing Vegetable Fat with Bovine Milk Fat in Infant Formula on Postprandial Lipid and Energy Metabolism: A Proof-of-principle Study in Healthy Young Male Adults. Mol Nutr Food Res. 2021 May;65(9):e2000848. doi: 10.1002/mnfr.202000848. Epub 2021 Apr 01. PMID: 33682997; PMCID: PMC8243939.
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Insulin-stimulated adipocytes secrete lactate to promote endothelial fatty acid uptake and transport.

Journal of cell science

Ibrahim A, Neinast MD, Li K, Noji M, Kim B, Bornstein MR, Mohammed R, Wellen KE, Arany Z.
PMID: 34779480
J Cell Sci. 2022 Mar 01;135(5). doi: 10.1242/jcs.258964. Epub 2021 Dec 16.

Insulin stimulates adipose tissue to extract fatty acids from circulation and sequester them inside adipose cells. How fatty acids are transported across the capillary endothelial barrier, and how this process is regulated, remains unclear. We modeled the relationship of...

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Ibrahim A, Neinast MD, Li K, et al. Insulin-stimulated adipocytes secrete lactate to promote endothelial fatty acid uptake and transport. J Cell Sci. 2021;135(5)doi: 10.1242/jcs.258964.
Ibrahim, A., Neinast, M. D., Li, K., Noji, M., Kim, B., Bornstein, M. R., Mohammed, R., Wellen, K. E., & Arany, Z. (2022). Insulin-stimulated adipocytes secrete lactate to promote endothelial fatty acid uptake and transport. Journal of cell science, 135(5), . https://doi.org/10.1242/jcs.258964
Ibrahim, Ayon, et al. "Insulin-stimulated adipocytes secrete lactate to promote endothelial fatty acid uptake and transport." Journal of cell science vol. 135,5 (2022). doi: https://doi.org/10.1242/jcs.258964
Ibrahim A, Neinast MD, Li K, Noji M, Kim B, Bornstein MR, Mohammed R, Wellen KE, Arany Z. Insulin-stimulated adipocytes secrete lactate to promote endothelial fatty acid uptake and transport. J Cell Sci. 2022 Mar 01;135(5). doi: 10.1242/jcs.258964. Epub 2021 Dec 16. PMID: 34779480; PMCID: PMC8729779.
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Showing 1 to 12 of 151 entries