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Sodium Glucose Transporter, Type 2 (SGLT2) Inhibitors (SGLT2i) and Glucagon-Like Peptide 1-Receptor Agonists: Newer Therapies in Whole-Body Glucose Stabilization.

Seminars in nephrology

Shepard BD, Ecelbarger CM.
PMID: 34715963
Semin Nephrol. 2021 Jul;41(4):331-348. doi: 10.1016/j.semnephrol.2021.06.005.

Diabetes is a worldwide epidemic that is increasing rapidly to become the seventh leading cause of death in the world. The increased incidence of this disease mirrors a similar uptick in obesity and metabolic syndrome, and, collectively, these conditions...

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Shepard BD, Ecelbarger CM. Sodium Glucose Transporter, Type 2 (SGLT2) Inhibitors (SGLT2i) and Glucagon-Like Peptide 1-Receptor Agonists: Newer Therapies in Whole-Body Glucose Stabilization. Semin Nephrol. 2021;41(4):331-348doi: 10.1016/j.semnephrol.2021.06.005.
Shepard, B. D., & Ecelbarger, C. M. (2021). Sodium Glucose Transporter, Type 2 (SGLT2) Inhibitors (SGLT2i) and Glucagon-Like Peptide 1-Receptor Agonists: Newer Therapies in Whole-Body Glucose Stabilization. Seminars in nephrology, 41(4), 331-348. https://doi.org/10.1016/j.semnephrol.2021.06.005
Shepard, Blythe D, and Ecelbarger, Carolyn M. "Sodium Glucose Transporter, Type 2 (SGLT2) Inhibitors (SGLT2i) and Glucagon-Like Peptide 1-Receptor Agonists: Newer Therapies in Whole-Body Glucose Stabilization." Seminars in nephrology vol. 41,4 (2021): 331-348. doi: https://doi.org/10.1016/j.semnephrol.2021.06.005
Shepard BD, Ecelbarger CM. Sodium Glucose Transporter, Type 2 (SGLT2) Inhibitors (SGLT2i) and Glucagon-Like Peptide 1-Receptor Agonists: Newer Therapies in Whole-Body Glucose Stabilization. Semin Nephrol. 2021 Jul;41(4):331-348. doi: 10.1016/j.semnephrol.2021.06.005. PMID: 34715963.
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Nano chitosan-zinc complex improves the growth performance and antioxidant capacity of the small intestine in weaned piglets.

The British journal of nutrition

Zhang M, Hou G, Hu P, Feng D, Wang J, Zhu W.
PMID: 33256856
Br J Nutr. 2021 Sep 28;126(6):801-812. doi: 10.1017/S0007114520004766. Epub 2020 Dec 01.

The present study was conducted to test the hypothesis that dietary supplementation with a nano chitosan-zinc complex (CP-Zn, 100 mg/kg Zn) could alleviate weaning stress in piglets challenged with enterotoxigenic Escherichia coli K88 by improving growth performance and intestinal...

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Zhang M, Hou G, Hu P, et al. Nano chitosan-zinc complex improves the growth performance and antioxidant capacity of the small intestine in weaned piglets. Br J Nutr. 2020;126(6):801-812doi: 10.1017/S0007114520004766.
Zhang, M., Hou, G., Hu, P., Feng, D., Wang, J., & Zhu, W. (2021). Nano chitosan-zinc complex improves the growth performance and antioxidant capacity of the small intestine in weaned piglets. The British journal of nutrition, 126(6), 801-812. https://doi.org/10.1017/S0007114520004766
Zhang, Minyang, et al. "Nano chitosan-zinc complex improves the growth performance and antioxidant capacity of the small intestine in weaned piglets." The British journal of nutrition vol. 126,6 (2021): 801-812. doi: https://doi.org/10.1017/S0007114520004766
Zhang M, Hou G, Hu P, Feng D, Wang J, Zhu W. Nano chitosan-zinc complex improves the growth performance and antioxidant capacity of the small intestine in weaned piglets. Br J Nutr. 2021 Sep 28;126(6):801-812. doi: 10.1017/S0007114520004766. Epub 2020 Dec 01. PMID: 33256856.
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Computational Modeling of Glucose Uptake in the Enterocyte.

Frontiers in physiology

Afshar N, Safaei S, Nickerson DP, Hunter PJ, Suresh V.
PMID: 31031632
Front Physiol. 2019 Apr 12;10:380. doi: 10.3389/fphys.2019.00380. eCollection 2019.

Absorption of glucose across the epithelial cells of the small intestine is a key process in human nutrition and initiates signaling cascades that regulate metabolic homeostasis. Validated and predictive mathematical models of glucose transport in intestinal epithelial cells are...

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Afshar N, Safaei S, Nickerson DP, et al. Computational Modeling of Glucose Uptake in the Enterocyte. Front Physiol. 2019;10:380doi: 10.3389/fphys.2019.00380.
Afshar, N., Safaei, S., Nickerson, D. P., Hunter, P. J., & Suresh, V. (2019). Computational Modeling of Glucose Uptake in the Enterocyte. Frontiers in physiology, 10380. https://doi.org/10.3389/fphys.2019.00380
Afshar, Nima, et al. "Computational Modeling of Glucose Uptake in the Enterocyte." Frontiers in physiology vol. 10 (2019): 380. doi: https://doi.org/10.3389/fphys.2019.00380
Afshar N, Safaei S, Nickerson DP, Hunter PJ, Suresh V. Computational Modeling of Glucose Uptake in the Enterocyte. Front Physiol. 2019 Apr 12;10:380. doi: 10.3389/fphys.2019.00380. eCollection 2019. PMID: 31031632; PMCID: PMC6473069.
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A Journey to Understand Glucose Homeostasis: Starting from Rat Glucose Transporter Type 2 Promoter Cloning to Hyperglycemia.

Diabetes & metabolism journal

Ahn YH.
PMID: 30398040
Diabetes Metab J. 2018 Dec;42(6):465-471. doi: 10.4093/dmj.2018.0116. Epub 2018 Nov 02.

My professional journey to understand the glucose homeostasis began in the 1990s, starting from cloning of the promoter region of glucose transporter type 2 (

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Ahn YH. A Journey to Understand Glucose Homeostasis: Starting from Rat Glucose Transporter Type 2 Promoter Cloning to Hyperglycemia. Diabetes Metab J. 2018;42(6):465-471doi: 10.4093/dmj.2018.0116.
Ahn, Y. H. (2018). A Journey to Understand Glucose Homeostasis: Starting from Rat Glucose Transporter Type 2 Promoter Cloning to Hyperglycemia. Diabetes & metabolism journal, 42(6), 465-471. https://doi.org/10.4093/dmj.2018.0116
Ahn, Yong Ho. "A Journey to Understand Glucose Homeostasis: Starting from Rat Glucose Transporter Type 2 Promoter Cloning to Hyperglycemia." Diabetes & metabolism journal vol. 42,6 (2018): 465-471. doi: https://doi.org/10.4093/dmj.2018.0116
Ahn YH. A Journey to Understand Glucose Homeostasis: Starting from Rat Glucose Transporter Type 2 Promoter Cloning to Hyperglycemia. Diabetes Metab J. 2018 Dec;42(6):465-471. doi: 10.4093/dmj.2018.0116. Epub 2018 Nov 02. PMID: 30398040; PMCID: PMC6300444.
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Nutritional Effects of the Enteral Nutritional Formula on Regulation of Gut Microbiota and Metabolic Level in Type 2 Diabetes Mellitus Mice.

Diabetes, metabolic syndrome and obesity : targets and therapy

Wei T, Jia Y, Xue W, Ma M, Wu W.
PMID: 33953585
Diabetes Metab Syndr Obes. 2021 Apr 28;14:1855-1869. doi: 10.2147/DMSO.S301454. eCollection 2021.

PURPOSE: Due to the adverse effects of antidiabetic drugs, nowadays, nutraceuticals have been of much interest to investigators. Therefore, the present study aimed to explore the potential effects of enteral nutritional (EN) formulas on the gut microbiota and metabolic...

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Wei T, Jia Y, Xue W, et al. Nutritional Effects of the Enteral Nutritional Formula on Regulation of Gut Microbiota and Metabolic Level in Type 2 Diabetes Mellitus Mice. Diabetes Metab Syndr Obes. 2021;14:1855-1869doi: 10.2147/DMSO.S301454.
Wei, T., Jia, Y., Xue, W., Ma, M., & Wu, W. (2021). Nutritional Effects of the Enteral Nutritional Formula on Regulation of Gut Microbiota and Metabolic Level in Type 2 Diabetes Mellitus Mice. Diabetes, metabolic syndrome and obesity : targets and therapy, 141855-1869. https://doi.org/10.2147/DMSO.S301454
Wei, Ting, et al. "Nutritional Effects of the Enteral Nutritional Formula on Regulation of Gut Microbiota and Metabolic Level in Type 2 Diabetes Mellitus Mice." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 14 (2021): 1855-1869. doi: https://doi.org/10.2147/DMSO.S301454
Wei T, Jia Y, Xue W, Ma M, Wu W. Nutritional Effects of the Enteral Nutritional Formula on Regulation of Gut Microbiota and Metabolic Level in Type 2 Diabetes Mellitus Mice. Diabetes Metab Syndr Obes. 2021 Apr 28;14:1855-1869. doi: 10.2147/DMSO.S301454. eCollection 2021. PMID: 33953585; PMCID: PMC8089093.
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Identification and Functional Characterization of P159L Mutation in HNF1B in a Family with Maturity-Onset Diabetes of the Young 5 (MODY5).

Genomics & informatics

Kim EK, Lee JS, Cheong HI, Chung SS, Kwak SH, Park KS.
PMID: 25705165
Genomics Inform. 2014 Dec;12(4):240-6. doi: 10.5808/GI.2014.12.4.240. Epub 2014 Dec 31.

Mutation in HNF1B, the hepatocyte nuclear factor-1β (HNF-1β) gene, results in maturity-onset diabetes of the young (MODY) 5, which is characterized by gradual impairment of insulin secretion. However, the functional role of HNF-1β in insulin secretion and glucose metabolism...

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Kim EK, Lee JS, Cheong HI, et al. Identification and Functional Characterization of P159L Mutation in HNF1B in a Family with Maturity-Onset Diabetes of the Young 5 (MODY5). Genomics Inform. 2014;12(4):240-6doi: 10.5808/GI.2014.12.4.240.
Kim, E. K., Lee, J. S., Cheong, H. I., Chung, S. S., Kwak, S. H., & Park, K. S. (2014). Identification and Functional Characterization of P159L Mutation in HNF1B in a Family with Maturity-Onset Diabetes of the Young 5 (MODY5). Genomics & informatics, 12(4), 240-6. https://doi.org/10.5808/GI.2014.12.4.240
Kim, Eun Ky, et al. "Identification and Functional Characterization of P159L Mutation in HNF1B in a Family with Maturity-Onset Diabetes of the Young 5 (MODY5)." Genomics & informatics vol. 12,4 (2014): 240-6. doi: https://doi.org/10.5808/GI.2014.12.4.240
Kim EK, Lee JS, Cheong HI, Chung SS, Kwak SH, Park KS. Identification and Functional Characterization of P159L Mutation in HNF1B in a Family with Maturity-Onset Diabetes of the Young 5 (MODY5). Genomics Inform. 2014 Dec;12(4):240-6. doi: 10.5808/GI.2014.12.4.240. Epub 2014 Dec 31. PMID: 25705165; PMCID: PMC4330261.
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Red rice .

Food & nutrition research

Yagi T, Ataka K, Cheng KC, Suzuki H, Ogata K, Yoshizaki Y, Takamine K, Kato I, Miyawaki S, Inui A, Asakawa A.
PMID: 33240034
Food Nutr Res. 2020 Oct 08;64. doi: 10.29219/fnr.v64.4226. eCollection 2020.

BACKGROUND: Red rice OBJECTIVE: The objective of this study was to examine the mechanism underlying the hypoglycemic action of RRK extract in two diabetic animal models: the insulin-deficiency mice, where the insulin deficiency was induced by streptozotocin (STZ), and...

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Yagi T, Ataka K, Cheng KC, et al. Red rice . Food Nutr Res. 2020;64doi: 10.29219/fnr.v64.4226.
Yagi, T., Ataka, K., Cheng, K. C., Suzuki, H., Ogata, K., Yoshizaki, Y., Takamine, K., Kato, I., Miyawaki, S., Inui, A., & Asakawa, A. (2020). Red rice . Food & nutrition research, 64. https://doi.org/10.29219/fnr.v64.4226
Yagi, Takakazu, et al. "Red rice ." Food & nutrition research vol. 64 (2020). doi: https://doi.org/10.29219/fnr.v64.4226
Yagi T, Ataka K, Cheng KC, Suzuki H, Ogata K, Yoshizaki Y, Takamine K, Kato I, Miyawaki S, Inui A, Asakawa A. Red rice . Food Nutr Res. 2020 Oct 08;64. doi: 10.29219/fnr.v64.4226. eCollection 2020. PMID: 33240034; PMCID: PMC7672486.
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Hypoglycemic activity and constituents analysis of blueberry (.

Diabetes, metabolic syndrome and obesity : targets and therapy

Huang W, Yao L, He X, Wang L, Li M, Yang Y, Wan C.
PMID: 30046248
Diabetes Metab Syndr Obes. 2018 Jul 17;11:357-366. doi: 10.2147/DMSO.S166728. eCollection 2018.

BACKGROUND: To investigate hypoglycemic activity and elucidate the active composition of the fruit blueberry (METHODS: Methanol extracts of blueberry (MEB) were separated using a D101 macroporous resin column to yield quinic acid derivative (Fr.1)- and flavonoid (Fr.2)-rich fractions. The...

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Huang W, Yao L, He X, et al. Hypoglycemic activity and constituents analysis of blueberry (. Diabetes Metab Syndr Obes. 2018;11:357-366doi: 10.2147/DMSO.S166728.
Huang, W., Yao, L., He, X., Wang, L., Li, M., Yang, Y., & Wan, C. (2018). Hypoglycemic activity and constituents analysis of blueberry (. Diabetes, metabolic syndrome and obesity : targets and therapy, 11357-366. https://doi.org/10.2147/DMSO.S166728
Huang, Weifeng, et al. "Hypoglycemic activity and constituents analysis of blueberry (." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 11 (2018): 357-366. doi: https://doi.org/10.2147/DMSO.S166728
Huang W, Yao L, He X, Wang L, Li M, Yang Y, Wan C. Hypoglycemic activity and constituents analysis of blueberry (. Diabetes Metab Syndr Obes. 2018 Jul 17;11:357-366. doi: 10.2147/DMSO.S166728. eCollection 2018. PMID: 30046248; PMCID: PMC6054273.
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Highlights in heart failure.

ESC heart failure

Tomasoni D, Adamo M, Lombardi CM, Metra M.
PMID: 31997538
ESC Heart Fail. 2019 Dec;6(6):1105-1127. doi: 10.1002/ehf2.12555.

Heart failure (HF) remains a major cause of mortality, morbidity, and poor quality of life. It is an area of active research. This article is aimed to give an update on recent advances in all aspects of this syndrome....

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Tomasoni D, Adamo M, Lombardi CM, et al. Highlights in heart failure. ESC Heart Fail. 2019;6(6):1105-1127doi: 10.1002/ehf2.12555.
Tomasoni, D., Adamo, M., Lombardi, C. M., & Metra, M. (2019). Highlights in heart failure. ESC heart failure, 6(6), 1105-1127. https://doi.org/10.1002/ehf2.12555
Tomasoni, Daniela, et al. "Highlights in heart failure." ESC heart failure vol. 6,6 (2019): 1105-1127. doi: https://doi.org/10.1002/ehf2.12555
Tomasoni D, Adamo M, Lombardi CM, Metra M. Highlights in heart failure. ESC Heart Fail. 2019 Dec;6(6):1105-1127. doi: 10.1002/ehf2.12555. PMID: 31997538; PMCID: PMC6989277.
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Upregulation Of Renal GLUT2 And SGLT2 Is Involved In High-Fat Diet-Induced Gestational Diabetes In Mice.

Diabetes, metabolic syndrome and obesity : targets and therapy

Jiang YK, Xin KY, Ge HW, Kong FJ, Zhao G.
PMID: 31686881
Diabetes Metab Syndr Obes. 2019 Oct 14;12:2095-2105. doi: 10.2147/DMSO.S221396. eCollection 2019.

INTRODUCTION: Gestational diabetes mellitus (GDM) is a metabolic disorder during mid- to late-pregnancy characterized by hyperglycemia, insulin resistance and fetal mal-development. Glucose transporter type 2 (GLUT2) and sodium-coupled glucose cotransporters 2 (SGLT2) in the proximal tubules play a critical...

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Jiang YK, Xin KY, Ge HW, et al. Upregulation Of Renal GLUT2 And SGLT2 Is Involved In High-Fat Diet-Induced Gestational Diabetes In Mice. Diabetes Metab Syndr Obes. 2019;12:2095-2105doi: 10.2147/DMSO.S221396.
Jiang, Y. K., Xin, K. Y., Ge, H. W., Kong, F. J., & Zhao, G. (2019). Upregulation Of Renal GLUT2 And SGLT2 Is Involved In High-Fat Diet-Induced Gestational Diabetes In Mice. Diabetes, metabolic syndrome and obesity : targets and therapy, 122095-2105. https://doi.org/10.2147/DMSO.S221396
Jiang, Yong-Kuan, et al. "Upregulation Of Renal GLUT2 And SGLT2 Is Involved In High-Fat Diet-Induced Gestational Diabetes In Mice." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 12 (2019): 2095-2105. doi: https://doi.org/10.2147/DMSO.S221396
Jiang YK, Xin KY, Ge HW, Kong FJ, Zhao G. Upregulation Of Renal GLUT2 And SGLT2 Is Involved In High-Fat Diet-Induced Gestational Diabetes In Mice. Diabetes Metab Syndr Obes. 2019 Oct 14;12:2095-2105. doi: 10.2147/DMSO.S221396. eCollection 2019. PMID: 31686881; PMCID: PMC6800457.
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MafA, NeuroD1, and HNF1β synergistically activate the Slc2a2 (Glut2) gene in β-cells.

Journal of molecular endocrinology

Ono Y, Kataoka K.
PMID: 34223824
J Mol Endocrinol. 2021 Jul 26;67(3):71-82. doi: 10.1530/JME-20-0339.

Glucose transporter type 2 (GLUT2), encoded by the SLC2A2 gene, is an essential component of glucose-stimulated insulin secretion in pancreatic islet β-cells. Like that of the gene encoding insulin, expression of the SLC2A2 gene expression is closely linked to...

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Ono Y, Kataoka K. MafA, NeuroD1, and HNF1β synergistically activate the Slc2a2 (Glut2) gene in β-cells. J Mol Endocrinol. 2021;67(3):71-82doi: 10.1530/JME-20-0339.
Ono, Y., & Kataoka, K. (2021). MafA, NeuroD1, and HNF1β synergistically activate the Slc2a2 (Glut2) gene in β-cells. Journal of molecular endocrinology, 67(3), 71-82. https://doi.org/10.1530/JME-20-0339
Ono, Yuka, and Kataoka, Kohsuke. "MafA, NeuroD1, and HNF1β synergistically activate the Slc2a2 (Glut2) gene in β-cells." Journal of molecular endocrinology vol. 67,3 (2021): 71-82. doi: https://doi.org/10.1530/JME-20-0339
Ono Y, Kataoka K. MafA, NeuroD1, and HNF1β synergistically activate the Slc2a2 (Glut2) gene in β-cells. J Mol Endocrinol. 2021 Jul 26;67(3):71-82. doi: 10.1530/JME-20-0339. PMID: 34223824.
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