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Studies on Liquefaction Time and Proteins Involved in the Improvement of Seminal Characteristics in Dromedary Camels (Camelus dromedarius).

Scientifica

Mal G, Vyas S, Srinivasan A, Patil NV, Pathak KM.
PMID: 27022505
Scientifica (Cairo). 2016;2016:4659358. doi: 10.1155/2016/4659358. Epub 2016 Feb 18.

Semen was collected from six dromedary camels using artificial vagina during rutting season. Liquefaction of the viscous semen occurred in 23.89 ± 1.49 h. During liquefaction, proteins with molecular masses of 24.55 kDa and 22.07 kDa appeared in conjunction...

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Mal G, Vyas S, Srinivasan A, et al. Studies on Liquefaction Time and Proteins Involved in the Improvement of Seminal Characteristics in Dromedary Camels (Camelus dromedarius). Scientifica (Cairo). 2016;2016:4659358doi: 10.1155/2016/4659358.
Mal, G., Vyas, S., Srinivasan, A., Patil, N. V., & Pathak, K. M. (2016). Studies on Liquefaction Time and Proteins Involved in the Improvement of Seminal Characteristics in Dromedary Camels (Camelus dromedarius). Scientifica, 20164659358. https://doi.org/10.1155/2016/4659358
Mal, Gorakh, et al. "Studies on Liquefaction Time and Proteins Involved in the Improvement of Seminal Characteristics in Dromedary Camels (Camelus dromedarius)." Scientifica vol. 2016 (2016): 4659358. doi: https://doi.org/10.1155/2016/4659358
Mal G, Vyas S, Srinivasan A, Patil NV, Pathak KM. Studies on Liquefaction Time and Proteins Involved in the Improvement of Seminal Characteristics in Dromedary Camels (Camelus dromedarius). Scientifica (Cairo). 2016;2016:4659358. doi: 10.1155/2016/4659358. Epub 2016 Feb 18. PMID: 27022505; PMCID: PMC4775800.
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Composite pheochromocytoma-ganglioneuroma of the adrenal gland: A case report with immunohistochemical study.

Urology annals

Rao RN, Singla N, Yadav K.
PMID: 23798871
Urol Ann. 2013 Apr;5(2):115-8. doi: 10.4103/0974-7796.110011.

Composite tumors of the adrenal medulla consisting of pheochromocytoma and ganglioneuroma are rare tumors accounting for less than 3% of all sympathoadrenal tumors. These tumors display more than one line of differentiation in which normal and neoplastic chromaffin cells...

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Rao RN, Singla N, Yadav K. Composite pheochromocytoma-ganglioneuroma of the adrenal gland: A case report with immunohistochemical study. Urol Ann. 2013;5(2):115-8doi: 10.4103/0974-7796.110011.
Rao, R. N., Singla, N., & Yadav, K. (2013). Composite pheochromocytoma-ganglioneuroma of the adrenal gland: A case report with immunohistochemical study. Urology annals, 5(2), 115-8. https://doi.org/10.4103/0974-7796.110011
Rao, Ram Nawal, et al. "Composite pheochromocytoma-ganglioneuroma of the adrenal gland: A case report with immunohistochemical study." Urology annals vol. 5,2 (2013): 115-8. doi: https://doi.org/10.4103/0974-7796.110011
Rao RN, Singla N, Yadav K. Composite pheochromocytoma-ganglioneuroma of the adrenal gland: A case report with immunohistochemical study. Urol Ann. 2013 Apr;5(2):115-8. doi: 10.4103/0974-7796.110011. PMID: 23798871; PMCID: PMC3685741.
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Folic acid contributes to peripheral nerve injury repair by promoting Schwann cell proliferation, migration, and secretion of nerve growth factor.

Neural regeneration research

Kang WB, Chen YJ, Lu DY, Yan JZ.
PMID: 30531087
Neural Regen Res. 2019 Jan;14(1):132-139. doi: 10.4103/1673-5374.243718.

After peripheral nerve injury, intraperitoneal injection of folic acid improves axon quantity, increases axon density and improves electromyography results. However, the mechanisms for this remain unclear. This study explored whether folic acid promotes peripheral nerve injury repair by affecting...

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Kang WB, Chen YJ, Lu DY, et al. Folic acid contributes to peripheral nerve injury repair by promoting Schwann cell proliferation, migration, and secretion of nerve growth factor. Neural Regen Res. 2019;14(1):132-139doi: 10.4103/1673-5374.243718.
Kang, W. B., Chen, Y. J., Lu, D. Y., & Yan, J. Z. (2019). Folic acid contributes to peripheral nerve injury repair by promoting Schwann cell proliferation, migration, and secretion of nerve growth factor. Neural regeneration research, 14(1), 132-139. https://doi.org/10.4103/1673-5374.243718
Kang, Wei-Bo, et al. "Folic acid contributes to peripheral nerve injury repair by promoting Schwann cell proliferation, migration, and secretion of nerve growth factor." Neural regeneration research vol. 14,1 (2019): 132-139. doi: https://doi.org/10.4103/1673-5374.243718
Kang WB, Chen YJ, Lu DY, Yan JZ. Folic acid contributes to peripheral nerve injury repair by promoting Schwann cell proliferation, migration, and secretion of nerve growth factor. Neural Regen Res. 2019 Jan;14(1):132-139. doi: 10.4103/1673-5374.243718. PMID: 30531087; PMCID: PMC6263007.
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Stem cell therapy in pain medicine.

The Korean journal of pain

Han YH, Kim KH, Abdi S, Kim TK.
PMID: 31569916
Korean J Pain. 2019 Oct 01;32(4):245-255. doi: 10.3344/kjp.2019.32.4.245.

Stem cells are attracting attention as a key element in future medicine, satisfying the desire to live a healthier life with the possibility that they can regenerate tissue damaged or degenerated by disease or aging. Stem cells are defined...

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Han YH, Kim KH, Abdi S, et al. Stem cell therapy in pain medicine. Korean J Pain. 2019;32(4):245-255doi: 10.3344/kjp.2019.32.4.245.
Han, Y. H., Kim, K. H., Abdi, S., & Kim, T. K. (2019). Stem cell therapy in pain medicine. The Korean journal of pain, 32(4), 245-255. https://doi.org/10.3344/kjp.2019.32.4.245
Han, Yong Hee, et al. "Stem cell therapy in pain medicine." The Korean journal of pain vol. 32,4 (2019): 245-255. doi: https://doi.org/10.3344/kjp.2019.32.4.245
Han YH, Kim KH, Abdi S, Kim TK. Stem cell therapy in pain medicine. Korean J Pain. 2019 Oct 01;32(4):245-255. doi: 10.3344/kjp.2019.32.4.245. PMID: 31569916; PMCID: PMC6813895.
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Role of Physical Activity in Parkinson's Disease.

Annals of Indian Academy of Neurology

Bhalsing KS, Abbas MM, Tan LCS.
PMID: 30532351
Ann Indian Acad Neurol. 2018 Oct-Dec;21(4):242-249. doi: 10.4103/aian.AIAN_169_18.

Parkinson's disease (PD) is common, age-dependent neurodegenerative disorder caused by a severe loss of the nigrostriatal dopaminergic neurons. Given the projected increase in the number of people with PD over the coming decades, interventions aimed at minimizing morbidity and...

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Bhalsing KS, Abbas MM, Tan LCS. Role of Physical Activity in Parkinson's Disease. Ann Indian Acad Neurol. 2018;21(4):242-249doi: 10.4103/aian.AIAN_169_18.
Bhalsing, K. S., Abbas, M. M., & Tan, L. C. S. (2018). Role of Physical Activity in Parkinson's Disease. Annals of Indian Academy of Neurology, 21(4), 242-249. https://doi.org/10.4103/aian.AIAN_169_18
Bhalsing, Ketaki S, et al. "Role of Physical Activity in Parkinson's Disease." Annals of Indian Academy of Neurology vol. 21,4 (2018): 242-249. doi: https://doi.org/10.4103/aian.AIAN_169_18
Bhalsing KS, Abbas MM, Tan LCS. Role of Physical Activity in Parkinson's Disease. Ann Indian Acad Neurol. 2018 Oct-Dec;21(4):242-249. doi: 10.4103/aian.AIAN_169_18. PMID: 30532351; PMCID: PMC6238554.
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The role of gel wound dressings loaded with stem cells in the treatment of diabetic foot ulcers.

American journal of translational research

Chen X, Wu J, Cao X, Jiang H, Wu Z, Zeng Z, Chen H, Zhang J.
PMID: 35035674
Am J Transl Res. 2021 Dec 15;13(12):13261-13272. eCollection 2021.

Diabetic foot ulcers (DFUs) are a serious complication of diabetes and the main cause of nontraumatic lower limb amputations, resulting in a serious economic burden on society. The main causes of DFUs include peripheral neuropathy, foot deformity, chronic inflammation,...

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Chen X, Wu J, Cao X, et al. The role of gel wound dressings loaded with stem cells in the treatment of diabetic foot ulcers. Am J Transl Res. 2021;13(12):13261-13272
Chen, X., Wu, J., Cao, X., Jiang, H., Wu, Z., Zeng, Z., Chen, H., & Zhang, J. (2021). The role of gel wound dressings loaded with stem cells in the treatment of diabetic foot ulcers. American journal of translational research, 13(12), 13261-13272.
Chen, Xionglin, et al. "The role of gel wound dressings loaded with stem cells in the treatment of diabetic foot ulcers." American journal of translational research vol. 13,12 (2021): 13261-13272.
Chen X, Wu J, Cao X, Jiang H, Wu Z, Zeng Z, Chen H, Zhang J. The role of gel wound dressings loaded with stem cells in the treatment of diabetic foot ulcers. Am J Transl Res. 2021 Dec 15;13(12):13261-13272. eCollection 2021. PMID: 35035674; PMCID: PMC8748097.
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Modern Trends for Peripheral Nerve Repair and Regeneration: Beyond the Hollow Nerve Guidance Conduit.

Frontiers in bioengineering and biotechnology

Carvalho CR, Oliveira JM, Reis RL.
PMID: 31824934
Front Bioeng Biotechnol. 2019 Nov 22;7:337. doi: 10.3389/fbioe.2019.00337. eCollection 2019.

Peripheral nerve repair and regeneration remains among the greatest challenges in tissue engineering and regenerative medicine. Even though peripheral nerve injuries (PNIs) are capable of some degree of regeneration, frail recovery is seen even when the best microsurgical technique...

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Carvalho CR, Oliveira JM, Reis RL. Modern Trends for Peripheral Nerve Repair and Regeneration: Beyond the Hollow Nerve Guidance Conduit. Front Bioeng Biotechnol. 2019;7:337doi: 10.3389/fbioe.2019.00337.
Carvalho, C. R., Oliveira, J. M., & Reis, R. L. (2019). Modern Trends for Peripheral Nerve Repair and Regeneration: Beyond the Hollow Nerve Guidance Conduit. Frontiers in bioengineering and biotechnology, 7337. https://doi.org/10.3389/fbioe.2019.00337
Carvalho, Cristiana R, et al. "Modern Trends for Peripheral Nerve Repair and Regeneration: Beyond the Hollow Nerve Guidance Conduit." Frontiers in bioengineering and biotechnology vol. 7 (2019): 337. doi: https://doi.org/10.3389/fbioe.2019.00337
Carvalho CR, Oliveira JM, Reis RL. Modern Trends for Peripheral Nerve Repair and Regeneration: Beyond the Hollow Nerve Guidance Conduit. Front Bioeng Biotechnol. 2019 Nov 22;7:337. doi: 10.3389/fbioe.2019.00337. eCollection 2019. PMID: 31824934; PMCID: PMC6882937.
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[Corrigendum] Murine and Chinese cobra venom‑derived nerve growth factor stimulate chondrogenic differentiation of BMSCs .

Molecular medicine reports

Miao Z, Lu Z, Luo S, Lei D, He Y, Wu H, Zhao J, Zheng L.
PMID: 34528695
Mol Med Rep. 2021 Nov;24(5). doi: 10.3892/mmr.2021.12444. Epub 2021 Sep 16.

Subsequently to the publication of this paper, an interested reader drew to the authors' attention that Figs. 2 and 3, showing the results from experiments designed to assess the viability of bone‑derived mesenchymal stem cells culture with or without...

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Miao Z, Lu Z, Luo S, et al. [Corrigendum] Murine and Chinese cobra venom‑derived nerve growth factor stimulate chondrogenic differentiation of BMSCs . Mol Med Rep. 2021;24(5)doi: 10.3892/mmr.2021.12444.
Miao, Z., Lu, Z., Luo, S., Lei, D., He, Y., Wu, H., Zhao, J., & Zheng, L. (2021). [Corrigendum] Murine and Chinese cobra venom‑derived nerve growth factor stimulate chondrogenic differentiation of BMSCs . Molecular medicine reports, 24(5), . https://doi.org/10.3892/mmr.2021.12444
Miao, Zhikang, et al. "[Corrigendum] Murine and Chinese cobra venom‑derived nerve growth factor stimulate chondrogenic differentiation of BMSCs ." Molecular medicine reports vol. 24,5 (2021). doi: https://doi.org/10.3892/mmr.2021.12444
Miao Z, Lu Z, Luo S, Lei D, He Y, Wu H, Zhao J, Zheng L. [Corrigendum] Murine and Chinese cobra venom‑derived nerve growth factor stimulate chondrogenic differentiation of BMSCs . Mol Med Rep. 2021 Nov;24(5). doi: 10.3892/mmr.2021.12444. Epub 2021 Sep 16. PMID: 34528695; PMCID: PMC8477180.
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Overexpression of RAPGEF3 enhances the therapeutic effect of dezocine in treatment of neuropathic pain.

Genetics and molecular biology

Liu X, Song L, Ma X, Liu Y, Huang H, Xu Y, Yan W.
PMID: 34807222
Genet Mol Biol. 2021 Nov 19;44(4):e20200463. doi: 10.1590/1678-4685-GMB-2020-0463. eCollection 2021.

Pain is a significant problem worldwide that affects the quality of life of patients. Dezocine is a non-addictive analgesic drug with kappa-opioid antagonist activity and has been successfully used to alleviate of postoperative pain. In addition, dezocine has an...

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Liu X, Song L, Ma X, et al. Overexpression of RAPGEF3 enhances the therapeutic effect of dezocine in treatment of neuropathic pain. Genet Mol Biol. 2021;44(4):e20200463doi: 10.1590/1678-4685-GMB-2020-0463.
Liu, X., Song, L., Ma, X., Liu, Y., Huang, H., Xu, Y., & Yan, W. (2021). Overexpression of RAPGEF3 enhances the therapeutic effect of dezocine in treatment of neuropathic pain. Genetics and molecular biology, 44(4), e20200463. https://doi.org/10.1590/1678-4685-GMB-2020-0463
Liu, Xue, et al. "Overexpression of RAPGEF3 enhances the therapeutic effect of dezocine in treatment of neuropathic pain." Genetics and molecular biology vol. 44,4 (2021): e20200463. doi: https://doi.org/10.1590/1678-4685-GMB-2020-0463
Liu X, Song L, Ma X, Liu Y, Huang H, Xu Y, Yan W. Overexpression of RAPGEF3 enhances the therapeutic effect of dezocine in treatment of neuropathic pain. Genet Mol Biol. 2021 Nov 19;44(4):e20200463. doi: 10.1590/1678-4685-GMB-2020-0463. eCollection 2021. PMID: 34807222; PMCID: PMC8607529.
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Amyloid-Beta 1-42 Cross-Reactive Antibody Prevalent in Human Sera May Contribute to Intraneuronal Deposition of A-Beta-P-42.

International journal of Alzheimer's disease

Vojdani A, Vojdani E.
PMID: 30034864
Int J Alzheimers Dis. 2018 Jun 21;2018:1672568. doi: 10.1155/2018/1672568. eCollection 2018.

Antibodies against many neural antigens are detected in the sera of both patients with Alzheimer's disease (AD) and some healthy individuals. Blood-brain barrier dysfunction could make it possible for brain-reactive autoantibodies to reach the brain, where they can react...

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Vojdani A, Vojdani E. Amyloid-Beta 1-42 Cross-Reactive Antibody Prevalent in Human Sera May Contribute to Intraneuronal Deposition of A-Beta-P-42. Int J Alzheimers Dis. 2018;2018:1672568doi: 10.1155/2018/1672568.
Vojdani, A., & Vojdani, E. (2018). Amyloid-Beta 1-42 Cross-Reactive Antibody Prevalent in Human Sera May Contribute to Intraneuronal Deposition of A-Beta-P-42. International journal of Alzheimer's disease, 20181672568. https://doi.org/10.1155/2018/1672568
Vojdani, Aristo, and Vojdani, Elroy. "Amyloid-Beta 1-42 Cross-Reactive Antibody Prevalent in Human Sera May Contribute to Intraneuronal Deposition of A-Beta-P-42." International journal of Alzheimer's disease vol. 2018 (2018): 1672568. doi: https://doi.org/10.1155/2018/1672568
Vojdani A, Vojdani E. Amyloid-Beta 1-42 Cross-Reactive Antibody Prevalent in Human Sera May Contribute to Intraneuronal Deposition of A-Beta-P-42. Int J Alzheimers Dis. 2018 Jun 21;2018:1672568. doi: 10.1155/2018/1672568. eCollection 2018. PMID: 30034864; PMCID: PMC6032666.
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Views and opinion on BDNF as a target for diabetic cognitive dysfunction.

Bioinformation

Rao AA.
PMID: 23888094
Bioinformation. 2013 Jun 29;9(11):551-4. doi: 10.6026/97320630009551. Print 2013.

Type 2 diabetes mellitus (T2DM) is a known cause of cognitive dysfunction and involves increased risk of dementia. Brain-derived neurotrophic factor (BDNF) is a member of neurotrophic family of nerve growth factors, a key protein in promoting memory, growth...

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Rao AA. Views and opinion on BDNF as a target for diabetic cognitive dysfunction. Bioinformation. 2013;9(11):551-4doi: 10.6026/97320630009551.
Rao, A. A. (2013). Views and opinion on BDNF as a target for diabetic cognitive dysfunction. Bioinformation, 9(11), 551-4. https://doi.org/10.6026/97320630009551
Rao, Allam Appa. "Views and opinion on BDNF as a target for diabetic cognitive dysfunction." Bioinformation vol. 9,11 (2013): 551-4. doi: https://doi.org/10.6026/97320630009551
Rao AA. Views and opinion on BDNF as a target for diabetic cognitive dysfunction. Bioinformation. 2013 Jun 29;9(11):551-4. doi: 10.6026/97320630009551. Print 2013. PMID: 23888094; PMCID: PMC3717181.
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Ethanol neurotoxicity in the developing cerebellum: underlying mechanisms and implications.

Brain sciences

Kumar A, LaVoie HA, DiPette DJ, Singh US.
PMID: 24961432
Brain Sci. 2013 Jun 14;3(2):941-63. doi: 10.3390/brainsci3020941.

Ethanol is the main constituent of alcoholic beverages that exerts toxicity to neuronal development. Ethanol affects synaptogenesis and prevents proper brain development. In humans, synaptogenesis takes place during the third trimester of pregnancy, and in rodents this period corresponds...

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Kumar A, LaVoie HA, DiPette DJ, et al. Ethanol neurotoxicity in the developing cerebellum: underlying mechanisms and implications. Brain Sci. 2013;3(2):941-63doi: 10.3390/brainsci3020941.
Kumar, A., LaVoie, H. A., DiPette, D. J., & Singh, U. S. (2013). Ethanol neurotoxicity in the developing cerebellum: underlying mechanisms and implications. Brain sciences, 3(2), 941-63. https://doi.org/10.3390/brainsci3020941
Kumar, Ambrish, et al. "Ethanol neurotoxicity in the developing cerebellum: underlying mechanisms and implications." Brain sciences vol. 3,2 (2013): 941-63. doi: https://doi.org/10.3390/brainsci3020941
Kumar A, LaVoie HA, DiPette DJ, Singh US. Ethanol neurotoxicity in the developing cerebellum: underlying mechanisms and implications. Brain Sci. 2013 Jun 14;3(2):941-63. doi: 10.3390/brainsci3020941. PMID: 24961432; PMCID: PMC4061865.
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Showing 1 to 12 of 49 entries