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Showing 1 to 12 of 171 entries
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Repetitive head impact exposure and later-life plasma total tau in former National Football League players.

Alzheimer's & dementia (Amsterdam, Netherlands)

Alosco ML, Tripodis Y, Jarnagin J, Baugh CM, Martin B, Chaisson CE, Estochen N, Song L, Cantu RC, Jeromin A, Stern RA.
PMID: 28229128
Alzheimers Dement (Amst). 2016 Dec 10;7:33-40. doi: 10.1016/j.dadm.2016.11.003. eCollection 2017.

INTRODUCTION: Blood protein analysis of total tau (t-tau) may be a practical screening biomarker for chronic traumatic encephalopathy (CTE), a neurodegenerative tauopathy associated with repetitive head impact (RHI) exposure. We examined plasma t-tau in symptomatic former National Football League...

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Alosco ML, Tripodis Y, Jarnagin J, et al. Repetitive head impact exposure and later-life plasma total tau in former National Football League players. Alzheimers Dement (Amst). 2016;7:33-40doi: 10.1016/j.dadm.2016.11.003.
Alosco, M. L., Tripodis, Y., Jarnagin, J., Baugh, C. M., Martin, B., Chaisson, C. E., Estochen, N., Song, L., Cantu, R. C., Jeromin, A., & Stern, R. A. (2017). Repetitive head impact exposure and later-life plasma total tau in former National Football League players. Alzheimer's & dementia (Amsterdam, Netherlands), 733-40. https://doi.org/10.1016/j.dadm.2016.11.003
Alosco, Michael L, et al. "Repetitive head impact exposure and later-life plasma total tau in former National Football League players." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 7 (2017): 33-40. doi: https://doi.org/10.1016/j.dadm.2016.11.003
Alosco ML, Tripodis Y, Jarnagin J, Baugh CM, Martin B, Chaisson CE, Estochen N, Song L, Cantu RC, Jeromin A, Stern RA. Repetitive head impact exposure and later-life plasma total tau in former National Football League players. Alzheimers Dement (Amst). 2016 Dec 10;7:33-40. doi: 10.1016/j.dadm.2016.11.003. eCollection 2017. PMID: 28229128; PMCID: PMC5312499.
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The Effect of Statins on Rate of Cognitive Decline in Mild Cognitive Impairment.

Alzheimer's & dementia (New York, N. Y.)

Smith KB, Kang P, Sabbagh MN.
PMID: 28480324
Alzheimers Dement (N Y). 2017 Jun;3(2):149-156. doi: 10.1016/j.trci.2017.01.001.

OBJECTIVE: This study's aims are to identify whether or not a relationship between statin use and rate of cognitive decline exists.BACKGROUND: The relationship between statins and MCI has been investigated in the past with the evidence showing mixed results.METHODS:...

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Smith KB, Kang P, Sabbagh MN. The Effect of Statins on Rate of Cognitive Decline in Mild Cognitive Impairment. Alzheimers Dement (N Y). 2017;3(2):149-156doi: 10.1016/j.trci.2017.01.001.
Smith, K. B., Kang, P., Sabbagh, M. N. (2017). The Effect of Statins on Rate of Cognitive Decline in Mild Cognitive Impairment. Alzheimer's & dementia (New York, N. Y.), 3(2), 149-156. https://doi.org/10.1016/j.trci.2017.01.001
Smith, Kyle B, et al. "The Effect of Statins on Rate of Cognitive Decline in Mild Cognitive Impairment." Alzheimer's & dementia (New York, N. Y.) vol. 3,2 (2017): 149-156. doi: https://doi.org/10.1016/j.trci.2017.01.001
Smith KB, Kang P, Sabbagh MN. The Effect of Statins on Rate of Cognitive Decline in Mild Cognitive Impairment. Alzheimers Dement (N Y). 2017 Jun;3(2):149-156. doi: 10.1016/j.trci.2017.01.001. PMID: 28480324; PMCID: PMC5412714.
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Structural Brain Network Changes across the Adult Lifespan.

Frontiers in aging neuroscience

Liu K, Yao S, Chen K, Zhang J, Yao L, Li K, Jin Z, Guo X.
PMID: 28860988
Front Aging Neurosci. 2017 Aug 17;9:275. doi: 10.3389/fnagi.2017.00275. eCollection 2017.

A number of magnetic resonance imaging (MRI) studies have shown age-related alterations in brain structural networks in different age groups. However, the specific age-associated changes in brain structural networks across the adult lifespan is underexplored. In the current study,...

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Liu K, Yao S, Chen K, et al. Structural Brain Network Changes across the Adult Lifespan. Front Aging Neurosci. 2017;9:275doi: 10.3389/fnagi.2017.00275.
Liu, K., Yao, S., Chen, K., Zhang, J., Yao, L., Li, K., Jin, Z., & Guo, X. (2017). Structural Brain Network Changes across the Adult Lifespan. Frontiers in aging neuroscience, 9275. https://doi.org/10.3389/fnagi.2017.00275
Liu, Ke, et al. "Structural Brain Network Changes across the Adult Lifespan." Frontiers in aging neuroscience vol. 9 (2017): 275. doi: https://doi.org/10.3389/fnagi.2017.00275
Liu K, Yao S, Chen K, Zhang J, Yao L, Li K, Jin Z, Guo X. Structural Brain Network Changes across the Adult Lifespan. Front Aging Neurosci. 2017 Aug 17;9:275. doi: 10.3389/fnagi.2017.00275. eCollection 2017. PMID: 28860988; PMCID: PMC5562685.
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Recent Perspectives on APP, Secretases, Endosomal Pathways and How they Influence Alzheimer's Related Pathological Changes in Down Syndrome.

Journal of Alzheimer's disease & Parkinsonism

Decourt B, Mobley W, Reiman E, Shah RJ, Sabbagh MN.
PMID: 24782952
J Alzheimers Dis Parkinsonism. 2013 Mar 20;002. doi: 10.4172/2161-0460.S7-002.

Down syndrome is one of the most common genetic conditions occurring in one in 700 live births. The trisomy of chromosome 21 causes over-expression of APP which in turn is indicated in the increased production of Aβ associated with...

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Decourt B, Mobley W, Reiman E, et al. Recent Perspectives on APP, Secretases, Endosomal Pathways and How they Influence Alzheimer's Related Pathological Changes in Down Syndrome. J Alzheimers Dis Parkinsonism. 2013;002doi: 10.4172/2161-0460.S7-002.
Decourt, B., Mobley, W., Reiman, E., Shah, R. J., & Sabbagh, M. N. (2013). Recent Perspectives on APP, Secretases, Endosomal Pathways and How they Influence Alzheimer's Related Pathological Changes in Down Syndrome. Journal of Alzheimer's disease & Parkinsonism, 002. https://doi.org/10.4172/2161-0460.S7-002
Decourt, Boris, et al. "Recent Perspectives on APP, Secretases, Endosomal Pathways and How they Influence Alzheimer's Related Pathological Changes in Down Syndrome." Journal of Alzheimer's disease & Parkinsonism vol. (2013): 002. doi: https://doi.org/10.4172/2161-0460.S7-002
Decourt B, Mobley W, Reiman E, Shah RJ, Sabbagh MN. Recent Perspectives on APP, Secretases, Endosomal Pathways and How they Influence Alzheimer's Related Pathological Changes in Down Syndrome. J Alzheimers Dis Parkinsonism. 2013 Mar 20;002. doi: 10.4172/2161-0460.S7-002. PMID: 24782952; PMCID: PMC4000700.
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Automatic gene prioritization in support of the inflammatory contribution to Alzheimer's disease.

AMIA Joint Summits on Translational Science proceedings. AMIA Joint Summits on Translational Science

Furniss SK, Yao R, Gonzalez G.
PMID: 25717399
AMIA Jt Summits Transl Sci Proc. 2014 Apr 07;2014:42-7. eCollection 2014.

This research seeks to extend the process of novel therapeutic gene target discovery for the treatment of Alzheimer's disease (AD). Gene-gene and gene-pathway annotation tools as well as human analysis are used to explore likely connections between potential gene...

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Furniss SK, Yao R, Gonzalez G. Automatic gene prioritization in support of the inflammatory contribution to Alzheimer's disease. AMIA Jt Summits Transl Sci Proc. 2014;2014:42-7
Furniss, S. K., Yao, R., & Gonzalez, G. (2014). Automatic gene prioritization in support of the inflammatory contribution to Alzheimer's disease. AMIA Joint Summits on Translational Science proceedings. AMIA Joint Summits on Translational Science, 201442-7.
Furniss, Stephanie K, et al. "Automatic gene prioritization in support of the inflammatory contribution to Alzheimer's disease." AMIA Joint Summits on Translational Science proceedings. AMIA Joint Summits on Translational Science vol. 2014 (2014): 42-7.
Furniss SK, Yao R, Gonzalez G. Automatic gene prioritization in support of the inflammatory contribution to Alzheimer's disease. AMIA Jt Summits Transl Sci Proc. 2014 Apr 07;2014:42-7. eCollection 2014. PMID: 25717399; PMCID: PMC4333706.
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Secretory PLA2-IIA: a new inflammatory factor for Alzheimer's disease.

Journal of neuroinflammation

Moses GS, Jensen MD, Lue LF, Walker DG, Sun AY, Simonyi A, Sun GY.
PMID: 17026770
J Neuroinflammation. 2006 Oct 07;3:28. doi: 10.1186/1742-2094-3-28.

Secretory phospholipase A2-IIA (sPLA2-IIA) is an inflammatory protein known to play a role in the pathogenesis of many inflammatory diseases. Although this enzyme has also been implicated in the pathogenesis of neurodegenerative diseases, there has not been a direct...

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Moses GS, Jensen MD, Lue LF, et al. Secretory PLA2-IIA: a new inflammatory factor for Alzheimer's disease. J Neuroinflammation. 2006;3:28doi: 10.1186/1742-2094-3-28.
Moses, G. S., Jensen, M. D., Lue, L. F., Walker, D. G., Sun, A. Y., Simonyi, A., & Sun, G. Y. (2006). Secretory PLA2-IIA: a new inflammatory factor for Alzheimer's disease. Journal of neuroinflammation, 328. https://doi.org/10.1186/1742-2094-3-28
Moses, Guna S D, et al. "Secretory PLA2-IIA: a new inflammatory factor for Alzheimer's disease." Journal of neuroinflammation vol. 3 (2006): 28. doi: https://doi.org/10.1186/1742-2094-3-28
Moses GS, Jensen MD, Lue LF, Walker DG, Sun AY, Simonyi A, Sun GY. Secretory PLA2-IIA: a new inflammatory factor for Alzheimer's disease. J Neuroinflammation. 2006 Oct 07;3:28. doi: 10.1186/1742-2094-3-28. PMID: 17026770; PMCID: PMC1613236.
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Multiple cytokines are involved in the early events leading to the Alzheimer's disease pathology.

Tottori Rinsho Kagaku Kenkyukai shi

Wilberding A, Morimoto K, Satoh H, Harano K, Harano T, Arita S, Tooyama I, Konishi Y.
PMID: 22586434
Tottori Rinsho Kagaku Kenkyukai Shi. 2008;1(2):359-373.

It is likely that neuroinflammation begins well before detectable cognitive impairment in Alzheimer's disease (AD) occurs. Clarifying the alterations occurring prior to the clinical manifestation of overt AD dementia may provide valuable insight into the early diagnosis and management...

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Wilberding A, Morimoto K, Satoh H, et al. Multiple cytokines are involved in the early events leading to the Alzheimer's disease pathology. Tottori Rinsho Kagaku Kenkyukai Shi. 2008;1(2):359-373
Wilberding, A., Morimoto, K., Satoh, H., Harano, K., Harano, T., Arita, S., Tooyama, I., & Konishi, Y. (2008). Multiple cytokines are involved in the early events leading to the Alzheimer's disease pathology. Tottori Rinsho Kagaku Kenkyukai shi, 1(2), 359-373.
Wilberding, Akiko, et al. "Multiple cytokines are involved in the early events leading to the Alzheimer's disease pathology." Tottori Rinsho Kagaku Kenkyukai shi vol. 1,2 (2008): 359-373.
Wilberding A, Morimoto K, Satoh H, Harano K, Harano T, Arita S, Tooyama I, Konishi Y. Multiple cytokines are involved in the early events leading to the Alzheimer's disease pathology. Tottori Rinsho Kagaku Kenkyukai Shi. 2008;1(2):359-373. PMID: 22586434; PMCID: PMC3350749.
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KIF6 719Arg Carrier Status Association with Homocysteine and C-Reactive Protein in Amnestic Mild Cognitive Impairment and Alzheimer's Disease Patients.

International journal of Alzheimer's disease

Malek-Ahmadi M, Patel A, Sabbagh MN.
PMID: 24455405
Int J Alzheimers Dis. 2013;2013:242303. doi: 10.1155/2013/242303. Epub 2013 Dec 24.

Recent research has demonstrated associations between statin use, KIF6 719Arg carrier status, and cholesterol levels and amnestic mild cognitive impairment (aMCI) and Alzheimer's disease (AD) patients. The association between 719Arg carrier status with homocysteine (tHcy) and c-reactive protein (CRP)...

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Malek-Ahmadi M, Patel A, Sabbagh MN. KIF6 719Arg Carrier Status Association with Homocysteine and C-Reactive Protein in Amnestic Mild Cognitive Impairment and Alzheimer's Disease Patients. Int J Alzheimers Dis. 2013;2013:242303doi: 10.1155/2013/242303.
Malek-Ahmadi, M., Patel, A., & Sabbagh, M. N. (2013). KIF6 719Arg Carrier Status Association with Homocysteine and C-Reactive Protein in Amnestic Mild Cognitive Impairment and Alzheimer's Disease Patients. International journal of Alzheimer's disease, 2013242303. https://doi.org/10.1155/2013/242303
Malek-Ahmadi, Michael, et al. "KIF6 719Arg Carrier Status Association with Homocysteine and C-Reactive Protein in Amnestic Mild Cognitive Impairment and Alzheimer's Disease Patients." International journal of Alzheimer's disease vol. 2013 (2013): 242303. doi: https://doi.org/10.1155/2013/242303
Malek-Ahmadi M, Patel A, Sabbagh MN. KIF6 719Arg Carrier Status Association with Homocysteine and C-Reactive Protein in Amnestic Mild Cognitive Impairment and Alzheimer's Disease Patients. Int J Alzheimers Dis. 2013;2013:242303. doi: 10.1155/2013/242303. Epub 2013 Dec 24. PMID: 24455405; PMCID: PMC3884607.
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Static and Dynamic Cognitive Reserve Proxy Measures: Interactions with Alzheimer's Disease Neuropathology and Cognition.

Journal of Alzheimer's disease & Parkinsonism

Malek-Ahmadi M, Lu S, Chan Y, Perez SE, Chen K, Mufson EJ.
PMID: 29423338
J Alzheimers Dis Parkinsonism. 2017;7(6). doi: 10.4172/2161-0460.1000390. Epub 2017 Oct 25.

OBJECTIVE: Years of education are the most common proxy for measuring cognitive reserve (CR) when assessing the relationship between Alzheimer's disease (AD) neuropathology and cognition. However, years of education may be limited as a CR proxy given that it...

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Malek-Ahmadi M, Lu S, Chan Y, et al. Static and Dynamic Cognitive Reserve Proxy Measures: Interactions with Alzheimer's Disease Neuropathology and Cognition. J Alzheimers Dis Parkinsonism. 2017;7(6)doi: 10.4172/2161-0460.1000390.
Malek-Ahmadi, M., Lu, S., Chan, Y., Perez, S. E., Chen, K., & Mufson, E. J. (2017). Static and Dynamic Cognitive Reserve Proxy Measures: Interactions with Alzheimer's Disease Neuropathology and Cognition. Journal of Alzheimer's disease & Parkinsonism, 7(6), . https://doi.org/10.4172/2161-0460.1000390
Malek-Ahmadi, Michael, et al. "Static and Dynamic Cognitive Reserve Proxy Measures: Interactions with Alzheimer's Disease Neuropathology and Cognition." Journal of Alzheimer's disease & Parkinsonism vol. 7,6 (2017). doi: https://doi.org/10.4172/2161-0460.1000390
Malek-Ahmadi M, Lu S, Chan Y, Perez SE, Chen K, Mufson EJ. Static and Dynamic Cognitive Reserve Proxy Measures: Interactions with Alzheimer's Disease Neuropathology and Cognition. J Alzheimers Dis Parkinsonism. 2017;7(6). doi: 10.4172/2161-0460.1000390. Epub 2017 Oct 25. PMID: 29423338; PMCID: PMC5800515.
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Deep Learning based Classification of FDG-PET Data for Alzheimers Disease Categories.

Proceedings of SPIE--the International Society for Optical Engineering

Singh S, Srivastava A, Mi L, Caselli RJ, Chen K, Goradia D, Reiman EM, Wang Y.
PMID: 29263566
Proc SPIE Int Soc Opt Eng. 2017 Oct;10572. doi: 10.1117/12.2294537. Epub 2017 Nov 17.

Fluorodeoxyglucose (FDG) positron emission tomography (PET) measures the decline in the regional cerebral metabolic rate for glucose, offering a reliable metabolic biomarker even on presymptomatic Alzheimer's disease (AD) patients. PET scans provide functional information that is unique and unavailable...

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Singh S, Srivastava A, Mi L, et al. Deep Learning based Classification of FDG-PET Data for Alzheimers Disease Categories. Proc SPIE Int Soc Opt Eng. 2017;10572doi: 10.1117/12.2294537.
Singh, S., Srivastava, A., Mi, L., Caselli, R. J., Chen, K., Goradia, D., Reiman, E. M., & Wang, Y. (2017). Deep Learning based Classification of FDG-PET Data for Alzheimers Disease Categories. Proceedings of SPIE--the International Society for Optical Engineering, 10572. https://doi.org/10.1117/12.2294537
Singh, Shibani, et al. "Deep Learning based Classification of FDG-PET Data for Alzheimers Disease Categories." Proceedings of SPIE--the International Society for Optical Engineering vol. 10572 (2017). doi: https://doi.org/10.1117/12.2294537
Singh S, Srivastava A, Mi L, Caselli RJ, Chen K, Goradia D, Reiman EM, Wang Y. Deep Learning based Classification of FDG-PET Data for Alzheimers Disease Categories. Proc SPIE Int Soc Opt Eng. 2017 Oct;10572. doi: 10.1117/12.2294537. Epub 2017 Nov 17. PMID: 29263566; PMCID: PMC5733797.
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An Optimal Transportation based Univariate Neuroimaging Index.

Proceedings. IEEE International Conference on Computer Vision

Mi L, Zhang W, Zhang J, Fan Y, Goradia D, Chen K, Reiman EM, Gu X, Wang Y.
PMID: 29225556
Proc IEEE Int Conf Comput Vis. 2017;2017:182-191.

The alterations of brain structures and functions have been considered closely correlated to the change of cognitive performance due to neurodegenerative diseases such as Alzheimer's disease. In this paper, we introduce a variational framework to compute the optimal transformation...

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Mi L, Zhang W, Zhang J, et al. An Optimal Transportation based Univariate Neuroimaging Index. Proc IEEE Int Conf Comput Vis. 2017;2017:182-191
Mi, L., Zhang, W., Zhang, J., Fan, Y., Goradia, D., Chen, K., Reiman, E. M., Gu, X., & Wang, Y. (2017). An Optimal Transportation based Univariate Neuroimaging Index. Proceedings. IEEE International Conference on Computer Vision, 2017182-191.
Mi, Liang, et al. "An Optimal Transportation based Univariate Neuroimaging Index." Proceedings. IEEE International Conference on Computer Vision vol. 2017 (2017): 182-191.
Mi L, Zhang W, Zhang J, Fan Y, Goradia D, Chen K, Reiman EM, Gu X, Wang Y. An Optimal Transportation based Univariate Neuroimaging Index. Proc IEEE Int Conf Comput Vis. 2017;2017:182-191. PMID: 29225556; PMCID: PMC5719504.
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Essential tremor is not a neurodegenerative disease.

Neurodegenerative disease management

Rajput AH, Adler CH, Shill HA, Rajput A.
PMID: 23105950
Neurodegener Dis Manag. 2012 Jun;2(3):259-268. doi: 10.2217/nmt.12.23.

The pathophysiology of essential tremor (ET) remains unknown. Standard neuropathological studies have reported no consistent changes but a detailed study found neurodegeneration in all ET cases - 24% demonstrated lower brainstem Lewy body (LB) inclusions and 76% experienced a...

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Rajput AH, Adler CH, Shill HA, et al. Essential tremor is not a neurodegenerative disease. Neurodegener Dis Manag. 2012;2(3):259-268doi: 10.2217/nmt.12.23.
Rajput, A. H., Adler, C. H., Shill, H. A., & Rajput, A. (2012). Essential tremor is not a neurodegenerative disease. Neurodegenerative disease management, 2(3), 259-268. https://doi.org/10.2217/nmt.12.23
Rajput, Ali H, et al. "Essential tremor is not a neurodegenerative disease." Neurodegenerative disease management vol. 2,3 (2012): 259-268. doi: https://doi.org/10.2217/nmt.12.23
Rajput AH, Adler CH, Shill HA, Rajput A. Essential tremor is not a neurodegenerative disease. Neurodegener Dis Manag. 2012 Jun;2(3):259-268. doi: 10.2217/nmt.12.23. PMID: 23105950; PMCID: PMC3478956.
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Showing 1 to 12 of 171 entries