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Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells.

Cellular logistics

McRae R, Lapierre LA, Manning EH, Goldenring JR.
PMID: 28396819
Cell Logist. 2017 Jan 09;7(1):e1271498. doi: 10.1080/21592799.2016.1271498. eCollection 2017.

MARK2/Par1b/EMK1, a serine/threonine kinase, is required for correct apical/basolateral membrane polarization in epithelial cells. However, the specific substrates mediating MARK2 action are less well understood. We have now found that MARK2 phosphorylates Rab11-FIP1B/C at serine 234 in a consensus...

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McRae R, Lapierre LA, Manning EH, et al. Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells. Cell Logist. 2017;7(1):e1271498doi: 10.1080/21592799.2016.1271498.
McRae, R., Lapierre, L. A., Manning, E. H., & Goldenring, J. R. (2017). Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells. Cellular logistics, 7(1), e1271498. https://doi.org/10.1080/21592799.2016.1271498
McRae, Rebecca, et al. "Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells." Cellular logistics vol. 7,1 (2017): e1271498. doi: https://doi.org/10.1080/21592799.2016.1271498
McRae R, Lapierre LA, Manning EH, Goldenring JR. Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells. Cell Logist. 2017 Jan 09;7(1):e1271498. doi: 10.1080/21592799.2016.1271498. eCollection 2017. PMID: 28396819; PMCID: PMC5383218.
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Association of gene coding variation and resting metabolic rate in a multi-ethnic sample of children and adults.

BMC obesity

Hellwege JN, Velez Edwards DR, Acra S, Chen K, Buchowski MS, Edwards TL.
PMID: 28417008
BMC Obes. 2017 Apr 05;4:12. doi: 10.1186/s40608-017-0145-5. eCollection 2017.

BACKGROUND: Resting metabolic rates (RMR) vary across individuals. Understanding the determinants of RMR could provide biological insight into obesity and its metabolic consequences such as type 2 diabetes and cardiovascular diseases.METHODS: The present study measured RMR using reference standard...

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Hellwege JN, Velez Edwards DR, Acra S, et al. Association of gene coding variation and resting metabolic rate in a multi-ethnic sample of children and adults. BMC Obes. 2017;4:12doi: 10.1186/s40608-017-0145-5.
Hellwege, J. N., Velez Edwards, D. R., Acra, S., Chen, K., Buchowski, M. S., & Edwards, T. L. (2017). Association of gene coding variation and resting metabolic rate in a multi-ethnic sample of children and adults. BMC obesity, 412. https://doi.org/10.1186/s40608-017-0145-5
Hellwege, Jacklyn N, et al. "Association of gene coding variation and resting metabolic rate in a multi-ethnic sample of children and adults." BMC obesity vol. 4 (2017): 12. doi: https://doi.org/10.1186/s40608-017-0145-5
Hellwege JN, Velez Edwards DR, Acra S, Chen K, Buchowski MS, Edwards TL. Association of gene coding variation and resting metabolic rate in a multi-ethnic sample of children and adults. BMC Obes. 2017 Apr 05;4:12. doi: 10.1186/s40608-017-0145-5. eCollection 2017. PMID: 28417008; PMCID: PMC5381071.
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Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment.

Molecular metabolism

Hill AA, Anderson-Baucum EK, Kennedy AJ, Webb CD, Yull FE, Hasty AH.
PMID: 26779432
Mol Metab. 2015 Jul 28;4(10):665-77. doi: 10.1016/j.molmet.2015.07.005. eCollection 2015 Oct.

OBJECTIVE: Macrophage accumulation in adipose tissue (AT) during obesity contributes to inflammation and insulin resistance. Recruitment of monocytes to obese AT has been the most studied mechanism explaining this accumulation. However, recent evidence suggests that recruitment-independent mechanisms may also...

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Hill AA, Anderson-Baucum EK, Kennedy AJ, et al. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. Mol Metab. 2015;4(10):665-77doi: 10.1016/j.molmet.2015.07.005.
Hill, A. A., Anderson-Baucum, E. K., Kennedy, A. J., Webb, C. D., Yull, F. E., & Hasty, A. H. (2015). Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. Molecular metabolism, 4(10), 665-77. https://doi.org/10.1016/j.molmet.2015.07.005
Hill, Andrea A, et al. "Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment." Molecular metabolism vol. 4,10 (2015): 665-77. doi: https://doi.org/10.1016/j.molmet.2015.07.005
Hill AA, Anderson-Baucum EK, Kennedy AJ, Webb CD, Yull FE, Hasty AH. Activation of NF-κB drives the enhanced survival of adipose tissue macrophages in an obesogenic environment. Mol Metab. 2015 Jul 28;4(10):665-77. doi: 10.1016/j.molmet.2015.07.005. eCollection 2015 Oct. PMID: 26779432; PMCID: PMC4588436.
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High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones.

Frontiers in endocrinology

Palmisano BT, Stafford JM, Pendergast JS.
PMID: 28352249
Front Endocrinol (Lausanne). 2017 Mar 14;8:44. doi: 10.3389/fendo.2017.00044. eCollection 2017.

Obesity in women is increased by the loss of circulating estrogen after menopause. Shift work, which disrupts circadian rhythms, also increases the risk for obesity. It is not known whether ovarian hormones interact with the circadian system to protect...

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Palmisano BT, Stafford JM, Pendergast JS. High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones. Front Endocrinol (Lausanne). 2017;8:44doi: 10.3389/fendo.2017.00044.
Palmisano, B. T., Stafford, J. M., & Pendergast, J. S. (2017). High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones. Frontiers in endocrinology, 844. https://doi.org/10.3389/fendo.2017.00044
Palmisano, Brian T, et al. "High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones." Frontiers in endocrinology vol. 8 (2017): 44. doi: https://doi.org/10.3389/fendo.2017.00044
Palmisano BT, Stafford JM, Pendergast JS. High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones. Front Endocrinol (Lausanne). 2017 Mar 14;8:44. doi: 10.3389/fendo.2017.00044. eCollection 2017. PMID: 28352249; PMCID: PMC5348546.
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Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity.

JCI insight

McKinley ET, Sui Y, Al-Kofahi Y, Millis BA, Tyska MJ, Roland JT, Santamaria-Pang A, Ohland CL, Jobin C, Franklin JL, Lau KS, Gerdes MJ, Coffey RJ.
PMID: 28570279
JCI Insight. 2017 Jun 02;2(11). doi: 10.1172/jci.insight.93487. eCollection 2017 Jun 02.

Intestinal tuft cells are a rare, poorly understood cell type recently shown to be a critical mediator of type 2 immune response to helminth infection. Here, we present advances in segmentation algorithms and analytical tools for multiplex immunofluorescence (MxIF),...

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McKinley ET, Sui Y, Al-Kofahi Y, et al. Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity. JCI Insight. 2017;2(11)doi: 10.1172/jci.insight.93487.
McKinley, E. T., Sui, Y., Al-Kofahi, Y., Millis, B. A., Tyska, M. J., Roland, J. T., Santamaria-Pang, A., Ohland, C. L., Jobin, C., Franklin, J. L., Lau, K. S., Gerdes, M. J., & Coffey, R. J. (2017). Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity. JCI insight, 2(11), . https://doi.org/10.1172/jci.insight.93487
McKinley, Eliot T, et al. "Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity." JCI insight vol. 2,11 (2017). doi: https://doi.org/10.1172/jci.insight.93487
McKinley ET, Sui Y, Al-Kofahi Y, Millis BA, Tyska MJ, Roland JT, Santamaria-Pang A, Ohland CL, Jobin C, Franklin JL, Lau KS, Gerdes MJ, Coffey RJ. Optimized multiplex immunofluorescence single-cell analysis reveals tuft cell heterogeneity. JCI Insight. 2017 Jun 02;2(11). doi: 10.1172/jci.insight.93487. eCollection 2017 Jun 02. PMID: 28570279; PMCID: PMC5453701.
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Draft Genome Sequence of Gerbil-Adapted Carcinogenic Helicobacter pylori Strain 7.13.

Genome announcements

Asim M, Chikara SK, Ghosh A, Vudathala S, Romero-Gallo J, Krishna US, Wilson KT, Israel DA, Peek RM, Chaturvedi R.
PMID: 26067974
Genome Announc. 2015 Jun 11;3(3). doi: 10.1128/genomeA.00641-15.

We report here the draft genome sequence of Helicobacter pylori strain 7.13, a gerbil-adapted strain that causes gastric cancer in gerbils. Strain 7.13 is derived from clinical strain B128, isolated from a patient with a duodenal ulcer. This study...

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Asim M, Chikara SK, Ghosh A, et al. Draft Genome Sequence of Gerbil-Adapted Carcinogenic Helicobacter pylori Strain 7.13. Genome Announc. 2015;3(3)doi: 10.1128/genomeA.00641-15.
Asim, M., Chikara, S. K., Ghosh, A., Vudathala, S., Romero-Gallo, J., Krishna, U. S., Wilson, K. T., Israel, D. A., Peek, R. M., & Chaturvedi, R. (2015). Draft Genome Sequence of Gerbil-Adapted Carcinogenic Helicobacter pylori Strain 7.13. Genome announcements, 3(3), . https://doi.org/10.1128/genomeA.00641-15
Asim, Mohammad, et al. "Draft Genome Sequence of Gerbil-Adapted Carcinogenic Helicobacter pylori Strain 7.13." Genome announcements vol. 3,3 (2015). doi: https://doi.org/10.1128/genomeA.00641-15
Asim M, Chikara SK, Ghosh A, Vudathala S, Romero-Gallo J, Krishna US, Wilson KT, Israel DA, Peek RM, Chaturvedi R. Draft Genome Sequence of Gerbil-Adapted Carcinogenic Helicobacter pylori Strain 7.13. Genome Announc. 2015 Jun 11;3(3). doi: 10.1128/genomeA.00641-15. PMID: 26067974; PMCID: PMC4463538.
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Distinct patterns of corticogeniculate feedback to different layers of the lateral geniculate nucleus.

Eye and brain

Ichida JM, Mavity-Hudson JA, Casagrande VA.
PMID: 25892906
Eye Brain. 2014 Sep;2014(6):57-73. doi: 10.2147/EB.S64281.

In primates, feedforward visual pathways from retina to lateral geniculate nucleus (LGN) are segregated to different layers. These layers also receive strong reciprocal feedback pathways from cortex. The degree to which feedforward streams in primates are segregated from feedback...

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Ichida JM, Mavity-Hudson JA, Casagrande VA. Distinct patterns of corticogeniculate feedback to different layers of the lateral geniculate nucleus. Eye Brain. 2014;2014(6):57-73doi: 10.2147/EB.S64281.
Ichida, J. M., Mavity-Hudson, J. A., & Casagrande, V. A. (2014). Distinct patterns of corticogeniculate feedback to different layers of the lateral geniculate nucleus. Eye and brain, 2014(6), 57-73. https://doi.org/10.2147/EB.S64281
Ichida, Jennifer M, et al. "Distinct patterns of corticogeniculate feedback to different layers of the lateral geniculate nucleus." Eye and brain vol. 2014,6 (2014): 57-73. doi: https://doi.org/10.2147/EB.S64281
Ichida JM, Mavity-Hudson JA, Casagrande VA. Distinct patterns of corticogeniculate feedback to different layers of the lateral geniculate nucleus. Eye Brain. 2014 Sep;2014(6):57-73. doi: 10.2147/EB.S64281. PMID: 25892906; PMCID: PMC4399558.
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Biotinylation and Purification of Surface-exposed .

Bio-protocol

Voss BJ, Cover TL.
PMID: 26448963
Bio Protoc. 2015 Apr 20;5(8):e1455. doi: 10.21769/bioprotoc.1455.

Interactions between pathogenic bacteria and host cells are often mediated by proteins found on the surfaces of the bacteria. The Gram-negative bacterium

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Voss BJ, Cover TL. Biotinylation and Purification of Surface-exposed . Bio Protoc. 2015;5(8):e1455doi: 10.21769/bioprotoc.1455.
Voss, B. J., & Cover, T. L. (2015). Biotinylation and Purification of Surface-exposed . Bio-protocol, 5(8), e1455. https://doi.org/10.21769/bioprotoc.1455
Voss, Bradley J, and Cover, Timothy L. "Biotinylation and Purification of Surface-exposed ." Bio-protocol vol. 5,8 (2015): e1455. doi: https://doi.org/10.21769/bioprotoc.1455
Voss BJ, Cover TL. Biotinylation and Purification of Surface-exposed . Bio Protoc. 2015 Apr 20;5(8):e1455. doi: 10.21769/bioprotoc.1455. PMID: 26448963; PMCID: PMC4593060.
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Distinct metaplastic and inflammatory phenotypes in autoimmune and adenocarcinoma-associated chronic atrophic gastritis.

United European gastroenterology journal

Jeong S, Choi E, Petersen CP, Roland JT, Federico A, Ippolito R, D'Armiento FP, Nardone G, Nagano O, Saya H, Romano M, Goldenring JR.
PMID: 28405320
United European Gastroenterol J. 2017 Feb;5(1):37-44. doi: 10.1177/2050640616644142. Epub 2016 Jun 23.

BACKGROUND: Autoimmune gastritis (AIG) and adenocarcinoma-associated chronic atrophic gastritis (CAG) are both associated with oxyntic atrophy, but AIG patients demonstrate an increased risk of carcinoid tumors rather than the elevated risk of adenocarcinoma observed with CAG. We therefore sought...

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Jeong S, Choi E, Petersen CP, et al. Distinct metaplastic and inflammatory phenotypes in autoimmune and adenocarcinoma-associated chronic atrophic gastritis. United European Gastroenterol J. 2016;5(1):37-44doi: 10.1177/2050640616644142.
Jeong, S., Choi, E., Petersen, C. P., Roland, J. T., Federico, A., Ippolito, R., D'Armiento, F. P., Nardone, G., Nagano, O., Saya, H., Romano, M., & Goldenring, J. R. (2017). Distinct metaplastic and inflammatory phenotypes in autoimmune and adenocarcinoma-associated chronic atrophic gastritis. United European gastroenterology journal, 5(1), 37-44. https://doi.org/10.1177/2050640616644142
Jeong, Sangho, et al. "Distinct metaplastic and inflammatory phenotypes in autoimmune and adenocarcinoma-associated chronic atrophic gastritis." United European gastroenterology journal vol. 5,1 (2017): 37-44. doi: https://doi.org/10.1177/2050640616644142
Jeong S, Choi E, Petersen CP, Roland JT, Federico A, Ippolito R, D'Armiento FP, Nardone G, Nagano O, Saya H, Romano M, Goldenring JR. Distinct metaplastic and inflammatory phenotypes in autoimmune and adenocarcinoma-associated chronic atrophic gastritis. United European Gastroenterol J. 2017 Feb;5(1):37-44. doi: 10.1177/2050640616644142. Epub 2016 Jun 23. PMID: 28405320; PMCID: PMC5384548.
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Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH.

BMC endocrine disorders

Gunawardana SC, Rocheleau JV, Head WS, Piston DW.
PMID: 15193158
BMC Endocr Disord. 2004 Jun 11;4(1):1. doi: 10.1186/1472-6823-4-1.

BACKGROUND: Many mechanistic steps underlying nutrient-stimulated insulin secretion (NSIS) are poorly understood. The influence of intracellular pH (pHi) on insulin secretion is widely documented, and can be used as an investigative tool. This study demonstrates previously unknown effects of...

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Gunawardana SC, Rocheleau JV, Head WS, et al. Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH. BMC Endocr Disord. 2004;4(1):1doi: 10.1186/1472-6823-4-1.
Gunawardana, S. C., Rocheleau, J. V., Head, W. S., & Piston, D. W. (2004). Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH. BMC endocrine disorders, 4(1), 1. https://doi.org/10.1186/1472-6823-4-1
Gunawardana, Subhadra C, et al. "Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH." BMC endocrine disorders vol. 4,1 (2004): 1. doi: https://doi.org/10.1186/1472-6823-4-1
Gunawardana SC, Rocheleau JV, Head WS, Piston DW. Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH. BMC Endocr Disord. 2004 Jun 11;4(1):1. doi: 10.1186/1472-6823-4-1. PMID: 15193158; PMCID: PMC434517.
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Statistical strategies for microRNAseq batch effect reduction.

Translational cancer research

Guo Y, Zhao S, Su PF, Li CI, Ye F, Flynn CR, Shyr Y.
PMID: 25258704
Transl Cancer Res. 2014 Jun 01;3(3):260-265. doi: 10.3978/j.issn.2218-676X.2014.06.05.

RNAseq technology is replacing microarray technology as the tool of choice for gene expression profiling. While providing much richer data than microarray, analysis of RNAseq data has been much more challenging. Among the many difficulties of RNAseq analysis, correctly...

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Guo Y, Zhao S, Su PF, et al. Statistical strategies for microRNAseq batch effect reduction. Transl Cancer Res. 2014;3(3):260-265doi: 10.3978/j.issn.2218-676X.2014.06.05.
Guo, Y., Zhao, S., Su, P. F., Li, C. I., Ye, F., Flynn, C. R., & Shyr, Y. (2014). Statistical strategies for microRNAseq batch effect reduction. Translational cancer research, 3(3), 260-265. https://doi.org/10.3978/j.issn.2218-676X.2014.06.05
Guo, Yan, et al. "Statistical strategies for microRNAseq batch effect reduction." Translational cancer research vol. 3,3 (2014): 260-265. doi: https://doi.org/10.3978/j.issn.2218-676X.2014.06.05
Guo Y, Zhao S, Su PF, Li CI, Ye F, Flynn CR, Shyr Y. Statistical strategies for microRNAseq batch effect reduction. Transl Cancer Res. 2014 Jun 01;3(3):260-265. doi: 10.3978/j.issn.2218-676X.2014.06.05. PMID: 25258704; PMCID: PMC4171948.
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Draft Genome Sequences of Helicobacter pylori Strains Isolated from Regions of Low and High Gastric Cancer Risk in Colombia.

Genome announcements

Sheh A, Piazuelo MB, Wilson KT, Correa P, Fox JG.
PMID: 24051318
Genome Announc. 2013 Sep 19;1(5). doi: 10.1128/genomeA.00736-13.

The draft genome sequences of six Colombian Helicobacter pylori strains are presented. These strains were isolated from patients from regions of high and low gastric cancer risk in Colombia and were characterized by multilocus sequence typing. The data provide...

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Sheh A, Piazuelo MB, Wilson KT, et al. Draft Genome Sequences of Helicobacter pylori Strains Isolated from Regions of Low and High Gastric Cancer Risk in Colombia. Genome Announc. 2013;1(5)doi: 10.1128/genomeA.00736-13.
Sheh, A., Piazuelo, M. B., Wilson, K. T., Correa, P., & Fox, J. G. (2013). Draft Genome Sequences of Helicobacter pylori Strains Isolated from Regions of Low and High Gastric Cancer Risk in Colombia. Genome announcements, 1(5), . https://doi.org/10.1128/genomeA.00736-13
Sheh, Alexander, et al. "Draft Genome Sequences of Helicobacter pylori Strains Isolated from Regions of Low and High Gastric Cancer Risk in Colombia." Genome announcements vol. 1,5 (2013). doi: https://doi.org/10.1128/genomeA.00736-13
Sheh A, Piazuelo MB, Wilson KT, Correa P, Fox JG. Draft Genome Sequences of Helicobacter pylori Strains Isolated from Regions of Low and High Gastric Cancer Risk in Colombia. Genome Announc. 2013 Sep 19;1(5). doi: 10.1128/genomeA.00736-13. PMID: 24051318; PMCID: PMC3778201.
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Showing 1 to 12 of 268 entries