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Showing 1 to 12 of 137 entries
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UV-B damage and protection at the molecular level in plants.

Photosynthesis research

Strid A, Chow WS, Anderson JM.
PMID: 24311138
Photosynth Res. 1994 Mar;39(3):475-89. doi: 10.1007/BF00014600.

Influx of solar UV-B radiation (280-320 nm) will probably increase in the future due to depletion of stratospheric ozone. In plants, there are several targets for the deleterious UV-B radiation, especially the chloroplast. This review summarizes the early effects...

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Strid A, Chow WS, Anderson JM. UV-B damage and protection at the molecular level in plants. Photosynth Res. 1994;39(3):475-89doi: 10.1007/BF00014600.
Strid, A., Chow, W. S., & Anderson, J. M. (1994). UV-B damage and protection at the molecular level in plants. Photosynthesis research, 39(3), 475-89. https://doi.org/10.1007/BF00014600
Strid, A, et al. "UV-B damage and protection at the molecular level in plants." Photosynthesis research vol. 39,3 (1994): 475-89. doi: https://doi.org/10.1007/BF00014600
Strid A, Chow WS, Anderson JM. UV-B damage and protection at the molecular level in plants. Photosynth Res. 1994 Mar;39(3):475-89. doi: 10.1007/BF00014600. PMID: 24311138.
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The Roles of Several Residues of Escherichia coli DNA Photolyase in the Highly Efficient Photo-Repair of Cyclobutane Pyrimidine Dimers.

Journal of nucleic acids

Xu L, Zhu G.
PMID: 20871655
J Nucleic Acids. 2010 Aug 31;2010. doi: 10.4061/2010/794782.

Escherichia coli DNA photolyase is an enzyme that repairs the major kind of UV-induced lesions, cyclobutane pyrimidine dimer (CPD) in DNA utilizing 350-450 nm light as energy source. The enzyme has very high photo-repair efficiency (the quantum yield of...

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Xu L, Zhu G. The Roles of Several Residues of Escherichia coli DNA Photolyase in the Highly Efficient Photo-Repair of Cyclobutane Pyrimidine Dimers. J Nucleic Acids. 2010;2010doi: 10.4061/2010/794782.
Xu, L., & Zhu, G. (2010). The Roles of Several Residues of Escherichia coli DNA Photolyase in the Highly Efficient Photo-Repair of Cyclobutane Pyrimidine Dimers. Journal of nucleic acids, 2010. https://doi.org/10.4061/2010/794782
Xu, Lei, and Zhu, Guoping. "The Roles of Several Residues of Escherichia coli DNA Photolyase in the Highly Efficient Photo-Repair of Cyclobutane Pyrimidine Dimers." Journal of nucleic acids vol. 2010 (2010). doi: https://doi.org/10.4061/2010/794782
Xu L, Zhu G. The Roles of Several Residues of Escherichia coli DNA Photolyase in the Highly Efficient Photo-Repair of Cyclobutane Pyrimidine Dimers. J Nucleic Acids. 2010 Aug 31;2010. doi: 10.4061/2010/794782. PMID: 20871655; PMCID: PMC2939405.
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Bioinformatic prediction of ultraviolet light mutagenesis sensitivity of human genes and a method for genetically engineering UVB resistance.

Cancer informatics

Lease KA, Papageorgio C.
PMID: 21584265
Cancer Inform. 2011 Apr 18;10:121-31. doi: 10.4137/CIN.S6670.

Living on earth, we are exposed to ultraviolet (UV) light as part of the solar radiation. UVB spectrum light exposure contributes to the development of skin cancer by interacting with pyrimidine pairs to create lesions called cyclobutane pyrimidine dimers....

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Lease KA, Papageorgio C. Bioinformatic prediction of ultraviolet light mutagenesis sensitivity of human genes and a method for genetically engineering UVB resistance. Cancer Inform. 2011;10:121-31doi: 10.4137/CIN.S6670.
Lease, K. A., & Papageorgio, C. (2011). Bioinformatic prediction of ultraviolet light mutagenesis sensitivity of human genes and a method for genetically engineering UVB resistance. Cancer informatics, 10121-31. https://doi.org/10.4137/CIN.S6670
Lease, Kevin A, and Papageorgio, Chris. "Bioinformatic prediction of ultraviolet light mutagenesis sensitivity of human genes and a method for genetically engineering UVB resistance." Cancer informatics vol. 10 (2011): 121-31. doi: https://doi.org/10.4137/CIN.S6670
Lease KA, Papageorgio C. Bioinformatic prediction of ultraviolet light mutagenesis sensitivity of human genes and a method for genetically engineering UVB resistance. Cancer Inform. 2011 Apr 18;10:121-31. doi: 10.4137/CIN.S6670. PMID: 21584265; PMCID: PMC3091412.
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Molecular mechanisms of ultraviolet radiation-induced DNA damage and repair.

Journal of nucleic acids

Rastogi RP, Richa, Kumar A, Tyagi MB, Sinha RP.
PMID: 21209706
J Nucleic Acids. 2010 Dec 16;2010:592980. doi: 10.4061/2010/592980.

DNA is one of the prime molecules, and its stability is of utmost importance for proper functioning and existence of all living systems. Genotoxic chemicals and radiations exert adverse effects on genome stability. Ultraviolet radiation (UVR) (mainly UV-B: 280-315...

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Rastogi RP, Richa, Kumar A, et al. Molecular mechanisms of ultraviolet radiation-induced DNA damage and repair. J Nucleic Acids. 2010;2010:592980doi: 10.4061/2010/592980.
Rastogi, R. P., Richa, Kumar, A., Tyagi, M. B., & Sinha, R. P. (2010). Molecular mechanisms of ultraviolet radiation-induced DNA damage and repair. Journal of nucleic acids, 2010592980. https://doi.org/10.4061/2010/592980
Rastogi, Rajesh P, et al. "Molecular mechanisms of ultraviolet radiation-induced DNA damage and repair." Journal of nucleic acids vol. 2010 (2010): 592980. doi: https://doi.org/10.4061/2010/592980
Rastogi RP, Richa, Kumar A, Tyagi MB, Sinha RP. Molecular mechanisms of ultraviolet radiation-induced DNA damage and repair. J Nucleic Acids. 2010 Dec 16;2010:592980. doi: 10.4061/2010/592980. PMID: 21209706; PMCID: PMC3010660.
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Ursolic Acid-Regulated Energy Metabolism-Reliever or Propeller of Ultraviolet-Induced Oxidative Stress and DNA Damage?.

Proteomes

Lee YH, Sun Y, Glickman RD.
PMID: 28250388
Proteomes. 2014 Aug 06;2(3):399-425. doi: 10.3390/proteomes2030399.

Ultraviolet (UV) light is a leading cause of diseases, such as skin cancers and cataracts. A main process mediating UV-induced pathogenesis is the production of reactive oxygen species (ROS). Excessive ROS levels induce the formation of DNA adducts (e.g.,...

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Lee YH, Sun Y, Glickman RD. Ursolic Acid-Regulated Energy Metabolism-Reliever or Propeller of Ultraviolet-Induced Oxidative Stress and DNA Damage?. Proteomes. 2014;2(3):399-425doi: 10.3390/proteomes2030399.
Lee, Y. H., Sun, Y., & Glickman, R. D. (2014). Ursolic Acid-Regulated Energy Metabolism-Reliever or Propeller of Ultraviolet-Induced Oxidative Stress and DNA Damage?. Proteomes, 2(3), 399-425. https://doi.org/10.3390/proteomes2030399
Lee, Yuan-Hao, et al. "Ursolic Acid-Regulated Energy Metabolism-Reliever or Propeller of Ultraviolet-Induced Oxidative Stress and DNA Damage?." Proteomes vol. 2,3 (2014): 399-425. doi: https://doi.org/10.3390/proteomes2030399
Lee YH, Sun Y, Glickman RD. Ursolic Acid-Regulated Energy Metabolism-Reliever or Propeller of Ultraviolet-Induced Oxidative Stress and DNA Damage?. Proteomes. 2014 Aug 06;2(3):399-425. doi: 10.3390/proteomes2030399. PMID: 28250388; PMCID: PMC5302752.
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Correction: Role of charge transfer states into the formation of cyclobutane pyrimidine dimers in DNA.

Faraday discussions

[No authors listed]
PMID: 31290889
Faraday Discuss. 2019 Jul 11;216:574-579. doi: 10.1039/c9fd90047k.

No abstract available.

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Correction: Role of charge transfer states into the formation of cyclobutane pyrimidine dimers in DNA. Faraday Discuss. 2019;216:574-579doi: 10.1039/c9fd90047k.
(2019). Correction: Role of charge transfer states into the formation of cyclobutane pyrimidine dimers in DNA. Faraday discussions, 216574-579. https://doi.org/10.1039/c9fd90047k
"Correction: Role of charge transfer states into the formation of cyclobutane pyrimidine dimers in DNA." Faraday discussions vol. 216 (2019): 574-579. doi: https://doi.org/10.1039/c9fd90047k
Correction: Role of charge transfer states into the formation of cyclobutane pyrimidine dimers in DNA. Faraday Discuss. 2019 Jul 11;216:574-579. doi: 10.1039/c9fd90047k. PMID: 31290889.
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Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of .

Antioxidants (Basel, Switzerland)

Portillo-Esnaola M, Rodríguez-Luna A, Nicolás-Morala J, Gallego-Rentero M, Villalba M, Juarranz Á, González S.
PMID: 34943064
Antioxidants (Basel). 2021 Dec 07;10(12). doi: 10.3390/antiox10121961.

Exposure to sun and especially to ultraviolet radiation (UVR) exerts well known detrimental effects on skin which are implicated in malignancy. UVR induces production of cyclobutane pyrimidine dimers (CPDs), immediately during exposure and even hours after the exposure, these...

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Portillo-Esnaola M, Rodríguez-Luna A, Nicolás-Morala J, et al. Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of . Antioxidants (Basel). 2021;10(12)doi: 10.3390/antiox10121961.
Portillo-Esnaola, M., Rodríguez-Luna, A., Nicolás-Morala, J., Gallego-Rentero, M., Villalba, M., Juarranz, �. �., & González, S. (2021). Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of . Antioxidants (Basel, Switzerland), 10(12), . https://doi.org/10.3390/antiox10121961
Portillo-Esnaola, Mikel, et al. "Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of ." Antioxidants (Basel, Switzerland) vol. 10,12 (2021). doi: https://doi.org/10.3390/antiox10121961
Portillo-Esnaola M, Rodríguez-Luna A, Nicolás-Morala J, Gallego-Rentero M, Villalba M, Juarranz Á, González S. Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of . Antioxidants (Basel). 2021 Dec 07;10(12). doi: 10.3390/antiox10121961. PMID: 34943064.
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Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of .

Antioxidants (Basel, Switzerland)

Portillo-Esnaola M, Rodríguez-Luna A, Nicolás-Morala J, Gallego-Rentero M, Villalba M, Juarranz Á, González S.
PMID: 34943064
Antioxidants (Basel). 2021 Dec 07;10(12). doi: 10.3390/antiox10121961.

Exposure to sun and especially to ultraviolet radiation (UVR) exerts well known detrimental effects on skin which are implicated in malignancy. UVR induces production of cyclobutane pyrimidine dimers (CPDs), immediately during exposure and even hours after the exposure, these...

Cite
Portillo-Esnaola M, Rodríguez-Luna A, Nicolás-Morala J, et al. Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of . Antioxidants (Basel). 2021;10(12)doi: 10.3390/antiox10121961.
Portillo-Esnaola, M., Rodríguez-Luna, A., Nicolás-Morala, J., Gallego-Rentero, M., Villalba, M., Juarranz, �. �., & González, S. (2021). Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of . Antioxidants (Basel, Switzerland), 10(12), . https://doi.org/10.3390/antiox10121961
Portillo-Esnaola, Mikel, et al. "Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of ." Antioxidants (Basel, Switzerland) vol. 10,12 (2021). doi: https://doi.org/10.3390/antiox10121961
Portillo-Esnaola M, Rodríguez-Luna A, Nicolás-Morala J, Gallego-Rentero M, Villalba M, Juarranz Á, González S. Formation of Cyclobutane Pyrimidine Dimers after UVA Exposure (Dark-CPDs) Is Inhibited by an Hydrophilic Extract of . Antioxidants (Basel). 2021 Dec 07;10(12). doi: 10.3390/antiox10121961. PMID: 34943064.
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Downsizing in plants-UV light induces pronounced morphological changes in the absence of stress.

Plant physiology

Qian M, Rosenqvist E, Prinsen E, Pescheck F, Flygare AM, Kalbina I, Jansen MAK, Strid Å.
PMID: 34618138
Plant Physiol. 2021 Sep 04;187(1):378-395. doi: 10.1093/plphys/kiab262.

Ultraviolet (UV) light induces a stocky phenotype in many plant species. In this study, we investigate this effect with regard to specific UV wavebands (UV-A or UV-B) and the cause for this dwarfing. UV-A- or UV-B-enrichment of growth light...

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Qian M, Rosenqvist E, Prinsen E, et al. Downsizing in plants-UV light induces pronounced morphological changes in the absence of stress. Plant Physiol. 2021;187(1):378-395doi: 10.1093/plphys/kiab262.
Qian, M., Rosenqvist, E., Prinsen, E., Pescheck, F., Flygare, A. M., Kalbina, I., Jansen, M. A. K., & Strid, �. �. (2021). Downsizing in plants-UV light induces pronounced morphological changes in the absence of stress. Plant physiology, 187(1), 378-395. https://doi.org/10.1093/plphys/kiab262
Qian, Minjie, et al. "Downsizing in plants-UV light induces pronounced morphological changes in the absence of stress." Plant physiology vol. 187,1 (2021): 378-395. doi: https://doi.org/10.1093/plphys/kiab262
Qian M, Rosenqvist E, Prinsen E, Pescheck F, Flygare AM, Kalbina I, Jansen MAK, Strid Å. Downsizing in plants-UV light induces pronounced morphological changes in the absence of stress. Plant Physiol. 2021 Sep 04;187(1):378-395. doi: 10.1093/plphys/kiab262. PMID: 34618138; PMCID: PMC8418406.
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DNA Fiber Assay upon Treatment with Ultraviolet Radiations.

Bio-protocol

Luigi A, Giordano A, Pentimalli F.
PMID: 34541070
Bio Protoc. 2017 Jun 05;7(11):e2301. doi: 10.21769/BioProtoc.2301. eCollection 2017 Jun 05.

Genome stability is continuously challenged by a wide range of DNA damaging factors. To promote a correct DNA repair and cell survival, cells orchestrate a coordinated and finely tuned cascade of events collectively known as the DNA Damage Response...

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Luigi A, Giordano A, Pentimalli F. DNA Fiber Assay upon Treatment with Ultraviolet Radiations. Bio Protoc. 2017;7(11):e2301doi: 10.21769/BioProtoc.2301.
Luigi, A., Giordano, A., & Pentimalli, F. (2017). DNA Fiber Assay upon Treatment with Ultraviolet Radiations. Bio-protocol, 7(11), e2301. https://doi.org/10.21769/BioProtoc.2301
Luigi, Alfano, et al. "DNA Fiber Assay upon Treatment with Ultraviolet Radiations." Bio-protocol vol. 7,11 (2017): e2301. doi: https://doi.org/10.21769/BioProtoc.2301
Luigi A, Giordano A, Pentimalli F. DNA Fiber Assay upon Treatment with Ultraviolet Radiations. Bio Protoc. 2017 Jun 05;7(11):e2301. doi: 10.21769/BioProtoc.2301. eCollection 2017 Jun 05. PMID: 34541070; PMCID: PMC8410287.
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Mechanistic studies of dinucleotide and oligonucleotide model cyclobutane pyrimidine dimer (CPD) DNA lesions under alkaline conditions.

Bioorganic & medicinal chemistry

Chaturvedi R, Long EC.
PMID: 34922308
Bioorg Med Chem. 2021 Nov 10;54:116499. doi: 10.1016/j.bmc.2021.116499. Epub 2021 Nov 10.

Cyclobutane pyrimidine dimers (CPDs) are the most abundant mutagenic DNA lesions formed in mammalian cells upon exposure to UV-B radiation (280-315 nm) in sunlight. These lesions are thought to be chemically stable and to withstand high concentrations of acids...

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Chaturvedi R, Long EC. Mechanistic studies of dinucleotide and oligonucleotide model cyclobutane pyrimidine dimer (CPD) DNA lesions under alkaline conditions. Bioorg Med Chem. 2021;54:116499doi: 10.1016/j.bmc.2021.116499.
Chaturvedi, R., & Long, E. C. (2021). Mechanistic studies of dinucleotide and oligonucleotide model cyclobutane pyrimidine dimer (CPD) DNA lesions under alkaline conditions. Bioorganic & medicinal chemistry, 54116499. https://doi.org/10.1016/j.bmc.2021.116499
Chaturvedi, Ritu, and Long, Eric C. "Mechanistic studies of dinucleotide and oligonucleotide model cyclobutane pyrimidine dimer (CPD) DNA lesions under alkaline conditions." Bioorganic & medicinal chemistry vol. 54 (2021): 116499. doi: https://doi.org/10.1016/j.bmc.2021.116499
Chaturvedi R, Long EC. Mechanistic studies of dinucleotide and oligonucleotide model cyclobutane pyrimidine dimer (CPD) DNA lesions under alkaline conditions. Bioorg Med Chem. 2021 Nov 10;54:116499. doi: 10.1016/j.bmc.2021.116499. Epub 2021 Nov 10. PMID: 34922308.
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Mutant p53 reactivator SLMP53-2 hinders ultraviolet B radiation-induced skin carcinogenesis.

Pharmacological research

Loureiro JB, Ribeiro R, Nazareth N, Ferreira T, Lopes EA, Gama A, Machuqueiro M, Alves MG, Marabini L, Oliveira PA, Santos MMM, Saraiva L.
PMID: 34890775
Pharmacol Res. 2021 Dec 08;175:106026. doi: 10.1016/j.phrs.2021.106026. Epub 2021 Dec 08.

The growing incidence of skin cancer (SC) has prompted the search for additional preventive strategies to counteract this global health concern. Mutant p53 (mutp53), particularly with ultraviolet radiation (UVR) signature, has emerged as a promising target for SC prevention...

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Loureiro JB, Ribeiro R, Nazareth N, et al. Mutant p53 reactivator SLMP53-2 hinders ultraviolet B radiation-induced skin carcinogenesis. Pharmacol Res. 2021;175:106026doi: 10.1016/j.phrs.2021.106026.
Loureiro, J. B., Ribeiro, R., Nazareth, N., Ferreira, T., Lopes, E. A., Gama, A., Machuqueiro, M., Alves, M. G., Marabini, L., Oliveira, P. A., Santos, M. M. M., & Saraiva, L. (2021). Mutant p53 reactivator SLMP53-2 hinders ultraviolet B radiation-induced skin carcinogenesis. Pharmacological research, 175106026. https://doi.org/10.1016/j.phrs.2021.106026
Loureiro, Joana B, et al. "Mutant p53 reactivator SLMP53-2 hinders ultraviolet B radiation-induced skin carcinogenesis." Pharmacological research vol. 175 (2021): 106026. doi: https://doi.org/10.1016/j.phrs.2021.106026
Loureiro JB, Ribeiro R, Nazareth N, Ferreira T, Lopes EA, Gama A, Machuqueiro M, Alves MG, Marabini L, Oliveira PA, Santos MMM, Saraiva L. Mutant p53 reactivator SLMP53-2 hinders ultraviolet B radiation-induced skin carcinogenesis. Pharmacol Res. 2021 Dec 08;175:106026. doi: 10.1016/j.phrs.2021.106026. Epub 2021 Dec 08. PMID: 34890775.
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Showing 1 to 12 of 137 entries