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Showing 1 to 12 of 21 entries
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Quinapril overdose-induced renal failure.

The California journal of emergency medicine

Suchard JR, Bearie BJ.
PMID: 20852701
Cal J Emerg Med. 2002 Jan;3(1):9-10.

No abstract available.

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Suchard JR, Bearie BJ. Quinapril overdose-induced renal failure. Cal J Emerg Med. 2002;3(1):9-10
Suchard, J. R., & Bearie, B. J. (2002). Quinapril overdose-induced renal failure. The California journal of emergency medicine, 3(1), 9-10.
Suchard, Jeffrey R, and Bearie, Brian J. "Quinapril overdose-induced renal failure." The California journal of emergency medicine vol. 3,1 (2002): 9-10.
Suchard JR, Bearie BJ. Quinapril overdose-induced renal failure. Cal J Emerg Med. 2002 Jan;3(1):9-10. PMID: 20852701; PMCID: PMC2906930.
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The effect of antihypertensive treatment on arterial stiffness and serum concentration of selected matrix metalloproteinases.

Archives of medical science : AMS

Rajzer M, Wojciechowska W, Kameczura T, Olszanecka A, Fedak D, Terlecki M, Kawecka-Jaszcz K, Czarnecka D.
PMID: 28721143
Arch Med Sci. 2017 Jun;13(4):760-770. doi: 10.5114/aoms.2016.58825. Epub 2016 Mar 23.

INTRODUCTION: The aim of the study was to assess the arterial stiffness and serum levels of selected metalloproteinases (MMPs) in hypertensive patients and their changes following antihypertensive therapy.MATERIAL AND METHODS: The study group consisted of 95 patients with essential...

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Rajzer M, Wojciechowska W, Kameczura T, et al. The effect of antihypertensive treatment on arterial stiffness and serum concentration of selected matrix metalloproteinases. Arch Med Sci. 2016;13(4):760-770doi: 10.5114/aoms.2016.58825.
Rajzer, M., Wojciechowska, W., Kameczura, T., Olszanecka, A., Fedak, D., Terlecki, M., Kawecka-Jaszcz, K., & Czarnecka, D. (2017). The effect of antihypertensive treatment on arterial stiffness and serum concentration of selected matrix metalloproteinases. Archives of medical science : AMS, 13(4), 760-770. https://doi.org/10.5114/aoms.2016.58825
Rajzer, Marek, et al. "The effect of antihypertensive treatment on arterial stiffness and serum concentration of selected matrix metalloproteinases." Archives of medical science : AMS vol. 13,4 (2017): 760-770. doi: https://doi.org/10.5114/aoms.2016.58825
Rajzer M, Wojciechowska W, Kameczura T, Olszanecka A, Fedak D, Terlecki M, Kawecka-Jaszcz K, Czarnecka D. The effect of antihypertensive treatment on arterial stiffness and serum concentration of selected matrix metalloproteinases. Arch Med Sci. 2017 Jun;13(4):760-770. doi: 10.5114/aoms.2016.58825. Epub 2016 Mar 23. PMID: 28721143; PMCID: PMC5510502.
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Molecular modeling of the interaction of ligands with ACE2-SARS-CoV-2 spike protein complex.

In silico pharmacology

Isaac-Lam MF.
PMID: 34631362
In Silico Pharmacol. 2021 Oct 07;9(1):55. doi: 10.1007/s40203-021-00114-w. eCollection 2021.

COVID-19 is a new communicable disease with a widespread outbreak that affects all populations worldwide triggering a rush of scientific interest in coronavirus research globally. In silico molecular docking experiment was utilized to determine interactions of available compounds with...

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Isaac-Lam MF. Molecular modeling of the interaction of ligands with ACE2-SARS-CoV-2 spike protein complex. In Silico Pharmacol. 2021;9(1):55doi: 10.1007/s40203-021-00114-w.
Isaac-Lam, M. F. (2021). Molecular modeling of the interaction of ligands with ACE2-SARS-CoV-2 spike protein complex. In silico pharmacology, 9(1), 55. https://doi.org/10.1007/s40203-021-00114-w
Isaac-Lam, Meden F. "Molecular modeling of the interaction of ligands with ACE2-SARS-CoV-2 spike protein complex." In silico pharmacology vol. 9,1 (2021): 55. doi: https://doi.org/10.1007/s40203-021-00114-w
Isaac-Lam MF. Molecular modeling of the interaction of ligands with ACE2-SARS-CoV-2 spike protein complex. In Silico Pharmacol. 2021 Oct 07;9(1):55. doi: 10.1007/s40203-021-00114-w. eCollection 2021. PMID: 34631362; PMCID: PMC8495439.
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Investigative Concerns in Demonstrating Reduced Risk From Reversing Left Ventricular Hypertrophy.

Journal of cardiovascular pharmacology and therapeutics

Frohlich ED, Aristizabal D, Soria F, Messerli FH.
PMID: 10684395
J Cardiovasc Pharmacol Ther. 1996 Jan;1(1):17-22. doi: 10.1177/107424849600100104.

BACKGROUND: To demonstrate reduced risk from reversing left ventricular hypertrophy (LVH) in hypertension, one must show that it is independent of blood pressure reduction. METHODS AND RESULTS: A feasibility study was conducted with 15 patients. The study employed 48-hour...

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Frohlich ED, Aristizabal D, Soria F, et al. Investigative Concerns in Demonstrating Reduced Risk From Reversing Left Ventricular Hypertrophy. J Cardiovasc Pharmacol Ther. 1996;1(1):17-22doi: 10.1177/107424849600100104.
Frohlich, E. D., Aristizabal, D., Soria, F., & Messerli, F. H. (1996). Investigative Concerns in Demonstrating Reduced Risk From Reversing Left Ventricular Hypertrophy. Journal of cardiovascular pharmacology and therapeutics, 1(1), 17-22. https://doi.org/10.1177/107424849600100104
Frohlich, et al. "Investigative Concerns in Demonstrating Reduced Risk From Reversing Left Ventricular Hypertrophy." Journal of cardiovascular pharmacology and therapeutics vol. 1,1 (1996): 17-22. doi: https://doi.org/10.1177/107424849600100104
Frohlich ED, Aristizabal D, Soria F, Messerli FH. Investigative Concerns in Demonstrating Reduced Risk From Reversing Left Ventricular Hypertrophy. J Cardiovasc Pharmacol Ther. 1996 Jan;1(1):17-22. doi: 10.1177/107424849600100104. PMID: 10684395.
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Angiotensin II blockade causes acute renal failure in eNOS-deficient mice.

Journal of the renin-angiotensin-aldosterone system : JRAAS

Schnermann J, Huang YG, Briggs JP.
PMID: 28095225
J Renin Angiotensin Aldosterone Syst. 2001 Mar;2(1):S199-S203. doi: 10.1177/14703203010020013501.

Compared with wild-type mice, adult endothelial nitric oxide synthase (eNOS) knockout mice (eight months of age) have increased blood pressure (BP) (126±9 mmHg vs. 100±4 mmHg), and an increased renal vascular resistance (155±16 vs. 65±4 mmHg.min/ml). Renal vascular resistance...

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Schnermann J, Huang YG, Briggs JP. Angiotensin II blockade causes acute renal failure in eNOS-deficient mice. J Renin Angiotensin Aldosterone Syst. 2001;2(1):S199-S203doi: 10.1177/14703203010020013501.
Schnermann, J., Huang, Y. G., & Briggs, J. P. (2001). Angiotensin II blockade causes acute renal failure in eNOS-deficient mice. Journal of the renin-angiotensin-aldosterone system : JRAAS, 2(1), S199-S203. https://doi.org/10.1177/14703203010020013501
Schnermann, Jürgen, et al. "Angiotensin II blockade causes acute renal failure in eNOS-deficient mice." Journal of the renin-angiotensin-aldosterone system : JRAAS vol. 2,1 (2001): S199-S203. doi: https://doi.org/10.1177/14703203010020013501
Schnermann J, Huang YG, Briggs JP. Angiotensin II blockade causes acute renal failure in eNOS-deficient mice. J Renin Angiotensin Aldosterone Syst. 2001 Mar;2(1):S199-S203. doi: 10.1177/14703203010020013501. PMID: 28095225.
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Endothelial Vascular Function in Hypertensive Patients After Renin-Angiotensin System Blockad.

Journal of clinical hypertension (Greenwich, Conn.)

Souza-Barbosa LA, Ferreira-Melo SLE, Ubaid-Girioli S, Nogueira EA, Yugar-Toledo JC, Moreno H.
PMID: 29024440
J Clin Hypertens (Greenwich). 2006 Nov;8(11):803-811. doi: 10.1111/j.1524-6175.2006.05663.x.

It is unclear whether single and combined pharmacologic inhibition of the renin-angiotensin-aldosterone system have similar effects on endothelial function and blood pressure (BP). The authors evaluated 63 hypertensive patients divided into 4 groups (hydrochlorothiazide 25 mg/d; irbesartan [IRBE] 150...

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Souza-Barbosa LA, Ferreira-Melo SLE, Ubaid-Girioli S, et al. Endothelial Vascular Function in Hypertensive Patients After Renin-Angiotensin System Blockad. J Clin Hypertens (Greenwich). 2006;8(11):803-811doi: 10.1111/j.1524-6175.2006.05663.x.
Souza-Barbosa, L. A., Ferreira-Melo, S. L. E., Ubaid-Girioli, S., Nogueira, E. A., Yugar-Toledo, J. C., & Moreno, H. (2006). Endothelial Vascular Function in Hypertensive Patients After Renin-Angiotensin System Blockad. Journal of clinical hypertension (Greenwich, Conn.), 8(11), 803-811. https://doi.org/10.1111/j.1524-6175.2006.05663.x
Souza-Barbosa, Leon Adriana, et al. "Endothelial Vascular Function in Hypertensive Patients After Renin-Angiotensin System Blockad." Journal of clinical hypertension (Greenwich, Conn.) vol. 8,11 (2006): 803-811. doi: https://doi.org/10.1111/j.1524-6175.2006.05663.x
Souza-Barbosa LA, Ferreira-Melo SLE, Ubaid-Girioli S, Nogueira EA, Yugar-Toledo JC, Moreno H. Endothelial Vascular Function in Hypertensive Patients After Renin-Angiotensin System Blockad. J Clin Hypertens (Greenwich). 2006 Nov;8(11):803-811. doi: 10.1111/j.1524-6175.2006.05663.x. PMID: 29024440.
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Nebivolol and quinapril reduce p-wave duration and dispersion in hypertensive patients.

Indian pacing and electrophysiology journal

Korkmaz H, Onalan O, Akbulut M, Ozbay Y.
PMID: 19471593
Indian Pacing Electrophysiol J. 2009 May 15;9(3):158-66.

We aimed to investigate the effects of nebivolol and quinapril treatments on P-wave duration and dispersion in hypertensive patients. Hypertensive patients who were in sinus rhythm were assigned to the two treatment groups and received either 20 mg quinapril/day...

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Korkmaz H, Onalan O, Akbulut M, et al. Nebivolol and quinapril reduce p-wave duration and dispersion in hypertensive patients. Indian Pacing Electrophysiol J. 2009;9(3):158-66
Korkmaz, H., Onalan, O., Akbulut, M., & Ozbay, Y. (2009). Nebivolol and quinapril reduce p-wave duration and dispersion in hypertensive patients. Indian pacing and electrophysiology journal, 9(3), 158-66.
Korkmaz, Hasan, et al. "Nebivolol and quinapril reduce p-wave duration and dispersion in hypertensive patients." Indian pacing and electrophysiology journal vol. 9,3 (2009): 158-66.
Korkmaz H, Onalan O, Akbulut M, Ozbay Y. Nebivolol and quinapril reduce p-wave duration and dispersion in hypertensive patients. Indian Pacing Electrophysiol J. 2009 May 15;9(3):158-66. PMID: 19471593; PMCID: PMC2683636.
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Stabilization of quinapril by incorporating hydrogen bonding interactions.

Indian journal of pharmaceutical sciences

Roy BN, Singh GP, Godbole HM, Nehate SP.
PMID: 20502545
Indian J Pharm Sci. 2009 Jul;71(4):395-405. doi: 10.4103/0250-474X.57288.

In the present study stability of various known solvates of quinapril hydrochloride has been compared with nitromethane solvate. Nitromethane solvate was found to be more stable compared to other known solvates. Single crystal X-ray diffraction analysis of quinapril nitromethane...

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Roy BN, Singh GP, Godbole HM, et al. Stabilization of quinapril by incorporating hydrogen bonding interactions. Indian J Pharm Sci. 2009;71(4):395-405doi: 10.4103/0250-474X.57288.
Roy, B. N., Singh, G. P., Godbole, H. M., & Nehate, S. P. (2009). Stabilization of quinapril by incorporating hydrogen bonding interactions. Indian journal of pharmaceutical sciences, 71(4), 395-405. https://doi.org/10.4103/0250-474X.57288
Roy, B N, et al. "Stabilization of quinapril by incorporating hydrogen bonding interactions." Indian journal of pharmaceutical sciences vol. 71,4 (2009): 395-405. doi: https://doi.org/10.4103/0250-474X.57288
Roy BN, Singh GP, Godbole HM, Nehate SP. Stabilization of quinapril by incorporating hydrogen bonding interactions. Indian J Pharm Sci. 2009 Jul;71(4):395-405. doi: 10.4103/0250-474X.57288. PMID: 20502545; PMCID: PMC2865811.
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Lipophilicity Examination of Some ACE inhibitors andHydrochlorothiazide on Cellulose in RP Thin-Layer Chromatography.

Iranian journal of pharmaceutical research : IJPR

Odović J, Karljiković-Rajić K, Trbojević-Stanković J, Stojimirović B, Vladimirov S.
PMID: 24250503
Iran J Pharm Res. 2012;11(3):763-70.

In this assay, the evaluation of lipophilicity of four ACE-inhibitors and hydrochlorothiazide (HCTZ) with RP-TLC on cellulose layers was described using three binary solvent systems. The selected ACE inhibitors had sufficiently different structures which can indicate the method suitability...

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Odović J, Karljiković-Rajić K, Trbojević-Stanković J, et al. Lipophilicity Examination of Some ACE inhibitors andHydrochlorothiazide on Cellulose in RP Thin-Layer Chromatography. Iran J Pharm Res. 2012;11(3):763-70
Odović, J., Karljiković-Rajić, K., Trbojević-Stanković, J., Stojimirović, B., & Vladimirov, S. (2012). Lipophilicity Examination of Some ACE inhibitors andHydrochlorothiazide on Cellulose in RP Thin-Layer Chromatography. Iranian journal of pharmaceutical research : IJPR, 11(3), 763-70.
Odović, Jadranka, et al. "Lipophilicity Examination of Some ACE inhibitors andHydrochlorothiazide on Cellulose in RP Thin-Layer Chromatography." Iranian journal of pharmaceutical research : IJPR vol. 11,3 (2012): 763-70.
Odović J, Karljiković-Rajić K, Trbojević-Stanković J, Stojimirović B, Vladimirov S. Lipophilicity Examination of Some ACE inhibitors andHydrochlorothiazide on Cellulose in RP Thin-Layer Chromatography. Iran J Pharm Res. 2012;11(3):763-70. PMID: 24250503; PMCID: PMC3813140.
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In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs.

International journal of molecular sciences

Zurawska-Plaksej E, Wiglusz R, Piwowar A, Wiglusz K.
PMID: 35008711
Int J Mol Sci. 2021 Dec 28;23(1). doi: 10.3390/ijms23010286.

Type 2 diabetes management usually requires polytherapy, which increases the risk of drug-to-drug interactions. Among the multiple diabetes comorbidities, hypertension is the most prevalent. This study aimed to investigate the binding interactions between the model protein, bovine albumin, and...

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Zurawska-Plaksej E, Wiglusz R, Piwowar A, et al. In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs. Int J Mol Sci. 2021;23(1)doi: 10.3390/ijms23010286.
Zurawska-Plaksej, E., Wiglusz, R., Piwowar, A., & Wiglusz, K. (2021). In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010286
Zurawska-Plaksej, Ewa, et al. "In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs." International journal of molecular sciences vol. 23,1 (2021). doi: https://doi.org/10.3390/ijms23010286
Zurawska-Plaksej E, Wiglusz R, Piwowar A, Wiglusz K. In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs. Int J Mol Sci. 2021 Dec 28;23(1). doi: 10.3390/ijms23010286. PMID: 35008711.
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In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs.

International journal of molecular sciences

Zurawska-Plaksej E, Wiglusz R, Piwowar A, Wiglusz K.
PMID: 35008711
Int J Mol Sci. 2021 Dec 28;23(1). doi: 10.3390/ijms23010286.

Type 2 diabetes management usually requires polytherapy, which increases the risk of drug-to-drug interactions. Among the multiple diabetes comorbidities, hypertension is the most prevalent. This study aimed to investigate the binding interactions between the model protein, bovine albumin, and...

Cite
Zurawska-Plaksej E, Wiglusz R, Piwowar A, et al. In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs. Int J Mol Sci. 2021;23(1)doi: 10.3390/ijms23010286.
Zurawska-Plaksej, E., Wiglusz, R., Piwowar, A., & Wiglusz, K. (2021). In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010286
Zurawska-Plaksej, Ewa, et al. "In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs." International journal of molecular sciences vol. 23,1 (2021). doi: https://doi.org/10.3390/ijms23010286
Zurawska-Plaksej E, Wiglusz R, Piwowar A, Wiglusz K. In Vitro Investigation of Binding Interactions between Albumin-Gliclazide Model and Typical Hypotensive Drugs. Int J Mol Sci. 2021 Dec 28;23(1). doi: 10.3390/ijms23010286. PMID: 35008711; PMCID: PMC8745505.
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Lack of efficacy of quinapril on vascular damage in limited cutaneous systemic sclerosis.

Nature clinical practice. Rheumatology

Guiducci S, Cerinic MM.
PMID: 18461063
Nat Clin Pract Rheumatol. 2008 Jun;4(6):288-9. doi: 10.1038/ncprheum0803. Epub 2008 May 06.

Gliddon et al. conducted a randomized, double-blind, multicenter, placebo-controlled study to evaluate the efficacy of the angiotensin-converting enzyme inhibitor quinapril for the management of vascular damage in systemic sclerosis (SSc). The trial comprised 213 patients with limited cutaneous SSc...

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Guiducci S, Cerinic MM. Lack of efficacy of quinapril on vascular damage in limited cutaneous systemic sclerosis. Nat Clin Pract Rheumatol. 2008;4(6):288-9doi: 10.1038/ncprheum0803.
Guiducci, S., & Cerinic, M. M. (2008). Lack of efficacy of quinapril on vascular damage in limited cutaneous systemic sclerosis. Nature clinical practice. Rheumatology, 4(6), 288-9. https://doi.org/10.1038/ncprheum0803
Guiducci, Serena, and Cerinic, Marco Matucci. "Lack of efficacy of quinapril on vascular damage in limited cutaneous systemic sclerosis." Nature clinical practice. Rheumatology vol. 4,6 (2008): 288-9. doi: https://doi.org/10.1038/ncprheum0803
Guiducci S, Cerinic MM. Lack of efficacy of quinapril on vascular damage in limited cutaneous systemic sclerosis. Nat Clin Pract Rheumatol. 2008 Jun;4(6):288-9. doi: 10.1038/ncprheum0803. Epub 2008 May 06. PMID: 18461063.
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Showing 1 to 12 of 21 entries