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Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells.

BioResearch open access

Hachem LD, Mothe AJ, Tator CH.
PMID: 26309791
Biores Open Access. 2015 Feb 01;4(1):146-59. doi: 10.1089/biores.2014.0058. eCollection 2015.

Transplantation of neural stem/progenitor cells (NSPCs) is a promising strategy in spinal cord injury (SCI). However, poor survival of transplanted stem cells remains a major limitation of this therapy due to the hostile environment of the injured cord. Oxidative...

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Hachem LD, Mothe AJ, Tator CH. Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells. Biores Open Access. 2015;4(1):146-59doi: 10.1089/biores.2014.0058.
Hachem, L. D., Mothe, A. J., & Tator, C. H. (2015). Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells. BioResearch open access, 4(1), 146-59. https://doi.org/10.1089/biores.2014.0058
Hachem, Laureen D, et al. "Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells." BioResearch open access vol. 4,1 (2015): 146-59. doi: https://doi.org/10.1089/biores.2014.0058
Hachem LD, Mothe AJ, Tator CH. Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells. Biores Open Access. 2015 Feb 01;4(1):146-59. doi: 10.1089/biores.2014.0058. eCollection 2015. PMID: 26309791; PMCID: PMC4497651.
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Thyrotropin Receptor Antibody (TRAb)-IgM Levels Are Markedly Higher Than TRAb-IgG Levels in Graves' Disease Patients and Controls, and TRAb-IgM Production Is Related to Epstein-Barr Virus Reactivation.

Viral immunology

Kumata K, Nagata K, Matsushita M, Kuwamoto S, Kato M, Murakami I, Fukata S, Hayashi K.
PMID: 27529807
Viral Immunol. 2016 Oct;29(8):459-463. doi: 10.1089/vim.2016.0043. Epub 2016 Aug 16.

Graves' disease is an autoimmune thyroid disorder that mainly presents as hyperthyroidism and is caused by thyrotropin receptor antibodies (TRAbs) that stimulate thyroid-stimulating hormone receptors. We previously reported that Graves' disease patients and healthy controls both had Epstein-Barr virus...

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Kumata K, Nagata K, Matsushita M, et al. Thyrotropin Receptor Antibody (TRAb)-IgM Levels Are Markedly Higher Than TRAb-IgG Levels in Graves' Disease Patients and Controls, and TRAb-IgM Production Is Related to Epstein-Barr Virus Reactivation. Viral Immunol. 2016;29(8):459-463doi: 10.1089/vim.2016.0043.
Kumata, K., Nagata, K., Matsushita, M., Kuwamoto, S., Kato, M., Murakami, I., Fukata, S., & Hayashi, K. (2016). Thyrotropin Receptor Antibody (TRAb)-IgM Levels Are Markedly Higher Than TRAb-IgG Levels in Graves' Disease Patients and Controls, and TRAb-IgM Production Is Related to Epstein-Barr Virus Reactivation. Viral immunology, 29(8), 459-463. https://doi.org/10.1089/vim.2016.0043
Kumata, Keisuke, et al. "Thyrotropin Receptor Antibody (TRAb)-IgM Levels Are Markedly Higher Than TRAb-IgG Levels in Graves' Disease Patients and Controls, and TRAb-IgM Production Is Related to Epstein-Barr Virus Reactivation." Viral immunology vol. 29,8 (2016): 459-463. doi: https://doi.org/10.1089/vim.2016.0043
Kumata K, Nagata K, Matsushita M, Kuwamoto S, Kato M, Murakami I, Fukata S, Hayashi K. Thyrotropin Receptor Antibody (TRAb)-IgM Levels Are Markedly Higher Than TRAb-IgG Levels in Graves' Disease Patients and Controls, and TRAb-IgM Production Is Related to Epstein-Barr Virus Reactivation. Viral Immunol. 2016 Oct;29(8):459-463. doi: 10.1089/vim.2016.0043. Epub 2016 Aug 16. PMID: 27529807; PMCID: PMC7307640.
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Thyroid antibody-negative euthyroid Graves' ophthalmopathy.

Endocrinology, diabetes & metabolism case reports

Tabasum A, Khan I, Taylor P, Das G, Okosieme OE.
PMID: 27284451
Endocrinol Diabetes Metab Case Rep. 2016;2016:160008. doi: 10.1530/EDM-16-0008. Epub 2016 Jun 02.

UNLABELLED: TSH receptor antibodies (TRAbs) are the pathological hallmark of Graves' disease, present in nearly all patients with the disease. Euthyroid Graves' ophthalmopathy (EGO) is a well-recognized clinical entity, but its occurrence in patients with negative TRAbs is a...

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Tabasum A, Khan I, Taylor P, et al. Thyroid antibody-negative euthyroid Graves' ophthalmopathy. Endocrinol Diabetes Metab Case Rep. 2016;2016:160008doi: 10.1530/EDM-16-0008.
Tabasum, A., Khan, I., Taylor, P., Das, G., & Okosieme, O. E. (2016). Thyroid antibody-negative euthyroid Graves' ophthalmopathy. Endocrinology, diabetes & metabolism case reports, 2016160008. https://doi.org/10.1530/EDM-16-0008
Tabasum, Arshiya, et al. "Thyroid antibody-negative euthyroid Graves' ophthalmopathy." Endocrinology, diabetes & metabolism case reports vol. 2016 (2016): 160008. doi: https://doi.org/10.1530/EDM-16-0008
Tabasum A, Khan I, Taylor P, Das G, Okosieme OE. Thyroid antibody-negative euthyroid Graves' ophthalmopathy. Endocrinol Diabetes Metab Case Rep. 2016;2016:160008. doi: 10.1530/EDM-16-0008. Epub 2016 Jun 02. PMID: 27284451; PMCID: PMC4898069.
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Propylthiouracil, Perchlorate, and Thyroid-Stimulating Hormone Modulate High Concentrations of Iodide Instigated Mitochondrial Superoxide Production in the Thyroids of Metallothionein I/II Knockout Mice.

Endocrinology and metabolism (Seoul, Korea)

Duan Q, Wang T, Zhang N, Perera V, Liang X, Abeysekera IR, Yao X.
PMID: 26754589
Endocrinol Metab (Seoul). 2016 Mar;31(1):174-84. doi: 10.3803/EnM.2016.31.1.174.

BACKGROUND: Increased oxidative stress has been suggested as one of the underlying mechanisms in iodide excess-induced thyroid disease. Metallothioneins (MTs) are regarded as scavengers of reactive oxygen species (ROS) in oxidative stress. Our aim is to investigate the effects...

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Duan Q, Wang T, Zhang N, et al. Propylthiouracil, Perchlorate, and Thyroid-Stimulating Hormone Modulate High Concentrations of Iodide Instigated Mitochondrial Superoxide Production in the Thyroids of Metallothionein I/II Knockout Mice. Endocrinol Metab (Seoul). 2016;31(1):174-84doi: 10.3803/EnM.2016.31.1.174.
Duan, Q., Wang, T., Zhang, N., Perera, V., Liang, X., Abeysekera, I. R., & Yao, X. (2016). Propylthiouracil, Perchlorate, and Thyroid-Stimulating Hormone Modulate High Concentrations of Iodide Instigated Mitochondrial Superoxide Production in the Thyroids of Metallothionein I/II Knockout Mice. Endocrinology and metabolism (Seoul, Korea), 31(1), 174-84. https://doi.org/10.3803/EnM.2016.31.1.174
Duan, Qi, et al. "Propylthiouracil, Perchlorate, and Thyroid-Stimulating Hormone Modulate High Concentrations of Iodide Instigated Mitochondrial Superoxide Production in the Thyroids of Metallothionein I/II Knockout Mice." Endocrinology and metabolism (Seoul, Korea) vol. 31,1 (2016): 174-84. doi: https://doi.org/10.3803/EnM.2016.31.1.174
Duan Q, Wang T, Zhang N, Perera V, Liang X, Abeysekera IR, Yao X. Propylthiouracil, Perchlorate, and Thyroid-Stimulating Hormone Modulate High Concentrations of Iodide Instigated Mitochondrial Superoxide Production in the Thyroids of Metallothionein I/II Knockout Mice. Endocrinol Metab (Seoul). 2016 Mar;31(1):174-84. doi: 10.3803/EnM.2016.31.1.174. PMID: 26754589; PMCID: PMC4803556.
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Role of abnormal anterior pituitary hormones-growth hormone and prolactin in active systemic lupus erythematosus.

International journal of clinical and experimental medicine

Zhu X, Xu J, Li S, Huang W, Li F.
PMID: 26770558
Int J Clin Exp Med. 2015 Oct 15;8(10):19223-31. eCollection 2015.

BACKGROUND: The role of anterior pituitary hormones in systemic lupus erythematosus (SLE) remains controversial.AIMS AND OBJECTIVES: We determined the expression levels of human growth hormone (GH), prolactin (PRL), and their receptors in subjects presenting with SLE, and modulation of...

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Zhu X, Xu J, Li S, et al. Role of abnormal anterior pituitary hormones-growth hormone and prolactin in active systemic lupus erythematosus. Int J Clin Exp Med. 2015;8(10):19223-31
Zhu, X., Xu, J., Li, S., Huang, W., & Li, F. (2015). Role of abnormal anterior pituitary hormones-growth hormone and prolactin in active systemic lupus erythematosus. International journal of clinical and experimental medicine, 8(10), 19223-31.
Zhu, Xiaohua, et al. "Role of abnormal anterior pituitary hormones-growth hormone and prolactin in active systemic lupus erythematosus." International journal of clinical and experimental medicine vol. 8,10 (2015): 19223-31.
Zhu X, Xu J, Li S, Huang W, Li F. Role of abnormal anterior pituitary hormones-growth hormone and prolactin in active systemic lupus erythematosus. Int J Clin Exp Med. 2015 Oct 15;8(10):19223-31. eCollection 2015. PMID: 26770558; PMCID: PMC4694458.
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Photoperiodic and circadian bifurcation theories of depression and mania.

F1000Research

Kripke DF, Elliott JA, Welsh DK, Youngstedt SD.
PMID: 26180634
F1000Res. 2015 May 06;4:107. doi: 10.12688/f1000research.6444.1. eCollection 2015.

Seasonal effects on mood have been observed throughout much of human history.  Seasonal changes in animals and plants are largely mediated through the changing photoperiod (i.e., the photophase or duration of daylight).  We review that in mammals, daylight specifically...

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Kripke DF, Elliott JA, Welsh DK, et al. Photoperiodic and circadian bifurcation theories of depression and mania. F1000Res. 2015;4:107doi: 10.12688/f1000research.6444.1.
Kripke, D. F., Elliott, J. A., Welsh, D. K., & Youngstedt, S. D. (2015). Photoperiodic and circadian bifurcation theories of depression and mania. F1000Research, 4107. https://doi.org/10.12688/f1000research.6444.1
Kripke, Daniel F, et al. "Photoperiodic and circadian bifurcation theories of depression and mania." F1000Research vol. 4 (2015): 107. doi: https://doi.org/10.12688/f1000research.6444.1
Kripke DF, Elliott JA, Welsh DK, Youngstedt SD. Photoperiodic and circadian bifurcation theories of depression and mania. F1000Res. 2015 May 06;4:107. doi: 10.12688/f1000research.6444.1. eCollection 2015. PMID: 26180634; PMCID: PMC4490783.
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Ultrastructural changes in the parrotfish thyroid afterin vitro stimulation with bovine thyrotropin.

Fish physiology and biochemistry

Smith CJ, Gordon Grau E.
PMID: 24234667
Fish Physiol Biochem. 1986 Jun;1(3):153-62. doi: 10.1007/BF02290256.

Six groups of thyroid glands ofScarus dubius were examined and compared by electron microscopy after anin vitro culture for 4h with graded doses of bovine thyrotropin (bTSH). Five doses of bTSH were used encompassing the full range of the...

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Smith CJ, Gordon Grau E. Ultrastructural changes in the parrotfish thyroid afterin vitro stimulation with bovine thyrotropin. Fish Physiol Biochem. 1986;1(3):153-62doi: 10.1007/BF02290256.
Smith, C. J., & Gordon Grau, E. (1986). Ultrastructural changes in the parrotfish thyroid afterin vitro stimulation with bovine thyrotropin. Fish physiology and biochemistry, 1(3), 153-62. https://doi.org/10.1007/BF02290256
Smith, C J, and Gordon Grau, E. "Ultrastructural changes in the parrotfish thyroid afterin vitro stimulation with bovine thyrotropin." Fish physiology and biochemistry vol. 1,3 (1986): 153-62. doi: https://doi.org/10.1007/BF02290256
Smith CJ, Gordon Grau E. Ultrastructural changes in the parrotfish thyroid afterin vitro stimulation with bovine thyrotropin. Fish Physiol Biochem. 1986 Jun;1(3):153-62. doi: 10.1007/BF02290256. PMID: 24234667.
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A new family with an activating mutation (G431S) in the TSH receptor gene: a phenotype discussion and review of the literature.

International journal of pediatric endocrinology

Larsen CC, Karaviti LP, Seghers V, Weiss RE, Refetoff S, Dumitrescu AM.
PMID: 25873976
Int J Pediatr Endocrinol. 2014;2014(1):23. doi: 10.1186/1687-9856-2014-23. Epub 2014 Nov 17.

Germline nonautoimmune hyperthyroidism due to an activating mutation in the thyroid stimulating hormone receptor gene is an uncommon disease. To date 32 different mutations have been described. The severity of the hyperthyroid symptoms is variable and phenotype differences have...

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Larsen CC, Karaviti LP, Seghers V, et al. A new family with an activating mutation (G431S) in the TSH receptor gene: a phenotype discussion and review of the literature. Int J Pediatr Endocrinol. 2014;2014(1):23doi: 10.1186/1687-9856-2014-23.
Larsen, C. C., Karaviti, L. P., Seghers, V., Weiss, R. E., Refetoff, S., & Dumitrescu, A. M. (2014). A new family with an activating mutation (G431S) in the TSH receptor gene: a phenotype discussion and review of the literature. International journal of pediatric endocrinology, 2014(1), 23. https://doi.org/10.1186/1687-9856-2014-23
Larsen, Cæcilie C, et al. "A new family with an activating mutation (G431S) in the TSH receptor gene: a phenotype discussion and review of the literature." International journal of pediatric endocrinology vol. 2014,1 (2014): 23. doi: https://doi.org/10.1186/1687-9856-2014-23
Larsen CC, Karaviti LP, Seghers V, Weiss RE, Refetoff S, Dumitrescu AM. A new family with an activating mutation (G431S) in the TSH receptor gene: a phenotype discussion and review of the literature. Int J Pediatr Endocrinol. 2014;2014(1):23. doi: 10.1186/1687-9856-2014-23. Epub 2014 Nov 17. PMID: 25873976; PMCID: PMC4396564.
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Effect of weight loss after gastric bypass surgery on thyroid function in euthyroid people with morbid obesity.

Clinical obesity

MacCuish A, Razvi S, Syed AA.
PMID: 25586044
Clin Obes. 2012 Feb;2(1):25-8. doi: 10.1111/j.1758-8111.2012.00036.x. Epub 2012 Mar 13.

Obesity is positively associated with serum thyrotropin (TSH) concentrations at the high end of the normal range. The relationship between weight loss and thyroid function is less clear and studies to date have yielded inconsistent results. Our aim was...

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MacCuish A, Razvi S, Syed AA. Effect of weight loss after gastric bypass surgery on thyroid function in euthyroid people with morbid obesity. Clin Obes. 2012;2(1):25-8doi: 10.1111/j.1758-8111.2012.00036.x.
MacCuish, A., Razvi, S., & Syed, A. A. (2012). Effect of weight loss after gastric bypass surgery on thyroid function in euthyroid people with morbid obesity. Clinical obesity, 2(1), 25-8. https://doi.org/10.1111/j.1758-8111.2012.00036.x
MacCuish, A, et al. "Effect of weight loss after gastric bypass surgery on thyroid function in euthyroid people with morbid obesity." Clinical obesity vol. 2,1 (2012): 25-8. doi: https://doi.org/10.1111/j.1758-8111.2012.00036.x
MacCuish A, Razvi S, Syed AA. Effect of weight loss after gastric bypass surgery on thyroid function in euthyroid people with morbid obesity. Clin Obes. 2012 Feb;2(1):25-8. doi: 10.1111/j.1758-8111.2012.00036.x. Epub 2012 Mar 13. PMID: 25586044.
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Single-Cell Phenotypic Characterization of Human Pituitary GHomas and Non-Functioning Adenomas Based on Hormone Content and Calcium Responses to Hypothalamic Releasing Hormones.

Frontiers in oncology

Senovilla L, Núñez L, de Campos JM, de Luis DA, Romero E, García-Sancho J, Villalobos C.
PMID: 26106585
Front Oncol. 2015 Jun 09;5:124. doi: 10.3389/fonc.2015.00124. eCollection 2015.

Human pituitary tumors are generally benign adenomas causing considerable morbidity due to excess hormone secretion, hypopituitarism, and other tumor mass effects. Pituitary tumors are highly heterogeneous and difficult to type, often containing mixed cell phenotypes. We have used calcium...

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Senovilla L, Núñez L, de Campos JM, et al. Single-Cell Phenotypic Characterization of Human Pituitary GHomas and Non-Functioning Adenomas Based on Hormone Content and Calcium Responses to Hypothalamic Releasing Hormones. Front Oncol. 2015;5:124doi: 10.3389/fonc.2015.00124.
Senovilla, L., Núñez, L., de Campos, J. M., de Luis, D. A., Romero, E., García-Sancho, J., & Villalobos, C. (2015). Single-Cell Phenotypic Characterization of Human Pituitary GHomas and Non-Functioning Adenomas Based on Hormone Content and Calcium Responses to Hypothalamic Releasing Hormones. Frontiers in oncology, 5124. https://doi.org/10.3389/fonc.2015.00124
Senovilla, Laura, et al. "Single-Cell Phenotypic Characterization of Human Pituitary GHomas and Non-Functioning Adenomas Based on Hormone Content and Calcium Responses to Hypothalamic Releasing Hormones." Frontiers in oncology vol. 5 (2015): 124. doi: https://doi.org/10.3389/fonc.2015.00124
Senovilla L, Núñez L, de Campos JM, de Luis DA, Romero E, García-Sancho J, Villalobos C. Single-Cell Phenotypic Characterization of Human Pituitary GHomas and Non-Functioning Adenomas Based on Hormone Content and Calcium Responses to Hypothalamic Releasing Hormones. Front Oncol. 2015 Jun 09;5:124. doi: 10.3389/fonc.2015.00124. eCollection 2015. PMID: 26106585; PMCID: PMC4460876.
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Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus.

Obesity research & clinical practice

Fukagawa T, Fukagawa K, Gotoh K, Noguchi H, Yoshimatsu H.
PMID: 24351449
Obes Res Clin Pract. 2007 May;1(2):I-II. doi: 10.1016/j.orcp.2007.01.002.

Under restraint stress conditions, prepro-thyrotropin releasing hormone (TRH) 178-199 suppresses adrenocorticotropic hormone (ACTH) secretion from the rat pituitary, which indicates that prepro-TRH 178-199 is a candidate endogenous corticotropin releasing inhibitory factor (CRIF). Restraint stress also activates the release of...

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Fukagawa T, Fukagawa K, Gotoh K, et al. Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obes Res Clin Pract. 2007;1(2):I-IIdoi: 10.1016/j.orcp.2007.01.002.
Fukagawa, T., Fukagawa, K., Gotoh, K., Noguchi, H., & Yoshimatsu, H. (2007). Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obesity research & clinical practice, 1(2), I-II. https://doi.org/10.1016/j.orcp.2007.01.002
Fukagawa, Tomiyo, et al. "Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus." Obesity research & clinical practice vol. 1,2 (2007): I-II. doi: https://doi.org/10.1016/j.orcp.2007.01.002
Fukagawa T, Fukagawa K, Gotoh K, Noguchi H, Yoshimatsu H. Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obes Res Clin Pract. 2007 May;1(2):I-II. doi: 10.1016/j.orcp.2007.01.002. PMID: 24351449.
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Endoderm complexity in the mouse gastrula is revealed through the expression of spink3.

BioResearch open access

Goh HN, Rathjen PD, Familari M, Rathjen J.
PMID: 24940561
Biores Open Access. 2014 Jun 01;3(3):98-109. doi: 10.1089/biores.2014.0010.

Endoderm formation in the mammalian embryo occurs first in the blastocyst, when the primitive endoderm and pluripotent cells resolve into separate lineages, and again during gastrulation, when the definitive endoderm progenitor population emerges from the primitive streak. The formation...

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Goh HN, Rathjen PD, Familari M, et al. Endoderm complexity in the mouse gastrula is revealed through the expression of spink3. Biores Open Access. 2014;3(3):98-109doi: 10.1089/biores.2014.0010.
Goh, H. N., Rathjen, P. D., Familari, M., & Rathjen, J. (2014). Endoderm complexity in the mouse gastrula is revealed through the expression of spink3. BioResearch open access, 3(3), 98-109. https://doi.org/10.1089/biores.2014.0010
Goh, Hwee Ngee, et al. "Endoderm complexity in the mouse gastrula is revealed through the expression of spink3." BioResearch open access vol. 3,3 (2014): 98-109. doi: https://doi.org/10.1089/biores.2014.0010
Goh HN, Rathjen PD, Familari M, Rathjen J. Endoderm complexity in the mouse gastrula is revealed through the expression of spink3. Biores Open Access. 2014 Jun 01;3(3):98-109. doi: 10.1089/biores.2014.0010. PMID: 24940561; PMCID: PMC4048981.
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Showing 1 to 12 of 827 entries