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MicroRNA-17-5p mediates hypoxia-induced autophagy and inhibits apoptosis by targeting signal transducer and activator of transcription 3 in vascular smooth muscle cells.

Experimental and therapeutic medicine

Hao MX, Wang X, Jiao KL.
PMID: 28450922
Exp Ther Med. 2017 Mar;13(3):935-941. doi: 10.3892/etm.2017.4048. Epub 2017 Jan 16.

The aim of the present study was to investigate hypoxia-induced apoptosis and autophagy in vascular smooth muscle cells (VSMCs) and the underlying molecular mechanisms of microRNA (miR)-17-5p responses in an anaerobic environment. The results revealed that miR-17-5p expression was...

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Hao MX, Wang X, Jiao KL. MicroRNA-17-5p mediates hypoxia-induced autophagy and inhibits apoptosis by targeting signal transducer and activator of transcription 3 in vascular smooth muscle cells. Exp Ther Med. 2017;13(3):935-941doi: 10.3892/etm.2017.4048.
Hao, M. X., Wang, X., & Jiao, K. L. (2017). MicroRNA-17-5p mediates hypoxia-induced autophagy and inhibits apoptosis by targeting signal transducer and activator of transcription 3 in vascular smooth muscle cells. Experimental and therapeutic medicine, 13(3), 935-941. https://doi.org/10.3892/etm.2017.4048
Hao, Ming-Xiu, et al. "MicroRNA-17-5p mediates hypoxia-induced autophagy and inhibits apoptosis by targeting signal transducer and activator of transcription 3 in vascular smooth muscle cells." Experimental and therapeutic medicine vol. 13,3 (2017): 935-941. doi: https://doi.org/10.3892/etm.2017.4048
Hao MX, Wang X, Jiao KL. MicroRNA-17-5p mediates hypoxia-induced autophagy and inhibits apoptosis by targeting signal transducer and activator of transcription 3 in vascular smooth muscle cells. Exp Ther Med. 2017 Mar;13(3):935-941. doi: 10.3892/etm.2017.4048. Epub 2017 Jan 16. PMID: 28450922; PMCID: PMC5403340.
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The Close Interconnection between Mitochondrial Dynamics and Mitophagy in Cancer.

Frontiers in oncology

Bordi M, Nazio F, Campello S.
PMID: 28512624
Front Oncol. 2017 May 02;7:81. doi: 10.3389/fonc.2017.00081. eCollection 2017.

Recent decades have revealed the shape changes of mitochondria and their regulators to be main players in a plethora of physiological cell processes. Mitochondria are extremely dynamic organelles whose highly controlled morphological changes respond to specific and diverse pathophysiological...

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Bordi M, Nazio F, Campello S. The Close Interconnection between Mitochondrial Dynamics and Mitophagy in Cancer. Front Oncol. 2017;7:81doi: 10.3389/fonc.2017.00081.
Bordi, M., Nazio, F., & Campello, S. (2017). The Close Interconnection between Mitochondrial Dynamics and Mitophagy in Cancer. Frontiers in oncology, 781. https://doi.org/10.3389/fonc.2017.00081
Bordi, Matteo, et al. "The Close Interconnection between Mitochondrial Dynamics and Mitophagy in Cancer." Frontiers in oncology vol. 7 (2017): 81. doi: https://doi.org/10.3389/fonc.2017.00081
Bordi M, Nazio F, Campello S. The Close Interconnection between Mitochondrial Dynamics and Mitophagy in Cancer. Front Oncol. 2017 May 02;7:81. doi: 10.3389/fonc.2017.00081. eCollection 2017. PMID: 28512624; PMCID: PMC5411450.
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Reproductive toxicity and gender differences induced by cadmium telluride quantum dots in an invertebrate model organism.

Scientific reports

Yan SQ, Xing R, Zhou YF, Li KL, Su YY, Qiu JF, Zhang YH, Zhang KQ, He Y, Lu XP, Xu SQ.
PMID: 27669995
Sci Rep. 2016 Sep 27;6:34182. doi: 10.1038/srep34182.

Sexual glands are key sites affected by nanotoxicity, but there is no sensitive assay for measuring reproductive toxicity in animals. The aim of this study was to investigate the toxic effects of cadmium telluride quantum dots (CdTe-QDs) on gonads...

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Yan SQ, Xing R, Zhou YF, et al. Reproductive toxicity and gender differences induced by cadmium telluride quantum dots in an invertebrate model organism. Sci Rep. 2016;6:34182doi: 10.1038/srep34182.
Yan, S. Q., Xing, R., Zhou, Y. F., Li, K. L., Su, Y. Y., Qiu, J. F., Zhang, Y. H., Zhang, K. Q., He, Y., Lu, X. P., & Xu, S. Q. (2016). Reproductive toxicity and gender differences induced by cadmium telluride quantum dots in an invertebrate model organism. Scientific reports, 634182. https://doi.org/10.1038/srep34182
Yan, Si-Qi, et al. "Reproductive toxicity and gender differences induced by cadmium telluride quantum dots in an invertebrate model organism." Scientific reports vol. 6 (2016): 34182. doi: https://doi.org/10.1038/srep34182
Yan SQ, Xing R, Zhou YF, Li KL, Su YY, Qiu JF, Zhang YH, Zhang KQ, He Y, Lu XP, Xu SQ. Reproductive toxicity and gender differences induced by cadmium telluride quantum dots in an invertebrate model organism. Sci Rep. 2016 Sep 27;6:34182. doi: 10.1038/srep34182. PMID: 27669995; PMCID: PMC5037452.
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Arf-like GTPase Arl8: Moving from the periphery to the center of lysosomal biology.

Cellular logistics

Khatter D, Sindhwani A, Sharma M.
PMID: 27057420
Cell Logist. 2015 Sep 21;5(3):e1086501. doi: 10.1080/21592799.2015.1086501. eCollection 2015.

Lysosomes are dynamic organelles that not only mediate degradation of cellular substrates but also play critical roles in processes such as cholesterol homeostasis, plasma membrane repair, antigen presentation, and cell migration. The small GTPase Arl8, a member of Arf-like...

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Khatter D, Sindhwani A, Sharma M. Arf-like GTPase Arl8: Moving from the periphery to the center of lysosomal biology. Cell Logist. 2015;5(3):e1086501doi: 10.1080/21592799.2015.1086501.
Khatter, D., Sindhwani, A., & Sharma, M. (2015). Arf-like GTPase Arl8: Moving from the periphery to the center of lysosomal biology. Cellular logistics, 5(3), e1086501. https://doi.org/10.1080/21592799.2015.1086501
Khatter, Divya, et al. "Arf-like GTPase Arl8: Moving from the periphery to the center of lysosomal biology." Cellular logistics vol. 5,3 (2015): e1086501. doi: https://doi.org/10.1080/21592799.2015.1086501
Khatter D, Sindhwani A, Sharma M. Arf-like GTPase Arl8: Moving from the periphery to the center of lysosomal biology. Cell Logist. 2015 Sep 21;5(3):e1086501. doi: 10.1080/21592799.2015.1086501. eCollection 2015. PMID: 27057420; PMCID: PMC4820812.
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Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration.

Frontiers in cellular neuroscience

Edens BM, Miller N, Ma YC.
PMID: 26973461
Front Cell Neurosci. 2016 Mar 03;10:44. doi: 10.3389/fncel.2016.00044. eCollection 2016.

Selective motor neuron degeneration is a hallmark of amyotrophic lateral sclerosis (ALS). Around 10% of all cases present as familial ALS (FALS), while sporadic ALS (SALS) accounts for the remaining 90%. Diverse genetic mutations leading to FALS have been...

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Edens BM, Miller N, Ma YC. Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration. Front Cell Neurosci. 2016;10:44doi: 10.3389/fncel.2016.00044.
Edens, B. M., Miller, N., & Ma, Y. C. (2016). Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration. Frontiers in cellular neuroscience, 1044. https://doi.org/10.3389/fncel.2016.00044
Edens, Brittany M, et al. "Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration." Frontiers in cellular neuroscience vol. 10 (2016): 44. doi: https://doi.org/10.3389/fncel.2016.00044
Edens BM, Miller N, Ma YC. Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration. Front Cell Neurosci. 2016 Mar 03;10:44. doi: 10.3389/fncel.2016.00044. eCollection 2016. PMID: 26973461; PMCID: PMC4776126.
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Constitutive Negative Regulation of R Proteins in Arabidopsis also via Autophagy Related Pathway?.

Frontiers in plant science

Pečenková T, Sabol P, Kulich I, Ortmannová J, Žárský V.
PMID: 26973696
Front Plant Sci. 2016 Mar 04;7:260. doi: 10.3389/fpls.2016.00260. eCollection 2016.

Even though resistance (R) genes are among the most studied components of the plant immunity, there remain still a lot of aspects to be explained about the regulation of their function. Many gain-of-function mutants of R genes and loss-of-function...

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Pečenková T, Sabol P, Kulich I, et al. Constitutive Negative Regulation of R Proteins in Arabidopsis also via Autophagy Related Pathway?. Front Plant Sci. 2016;7:260doi: 10.3389/fpls.2016.00260.
Pečenková, T., Sabol, P., Kulich, I., Ortmannová, J., & Žárský, V. (2016). Constitutive Negative Regulation of R Proteins in Arabidopsis also via Autophagy Related Pathway?. Frontiers in plant science, 7260. https://doi.org/10.3389/fpls.2016.00260
Pečenková, Tamara, et al. "Constitutive Negative Regulation of R Proteins in Arabidopsis also via Autophagy Related Pathway?." Frontiers in plant science vol. 7 (2016): 260. doi: https://doi.org/10.3389/fpls.2016.00260
Pečenková T, Sabol P, Kulich I, Ortmannová J, Žárský V. Constitutive Negative Regulation of R Proteins in Arabidopsis also via Autophagy Related Pathway?. Front Plant Sci. 2016 Mar 04;7:260. doi: 10.3389/fpls.2016.00260. eCollection 2016. PMID: 26973696; PMCID: PMC4777726.
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Autophagy: eat thyself, sustain thyself.

Nature methods

Marx V.
PMID: 26624796
Nat Methods. 2015 Dec 01;12(12):1121-5. doi: 10.1038/nmeth.3661.

No abstract available.

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Marx V. Autophagy: eat thyself, sustain thyself. Nat Methods. 2015;12(12):1121-5doi: 10.1038/nmeth.3661.
Marx, V. (2015). Autophagy: eat thyself, sustain thyself. Nature methods, 12(12), 1121-5. https://doi.org/10.1038/nmeth.3661
Marx, Vivien. "Autophagy: eat thyself, sustain thyself." Nature methods vol. 12,12 (2015): 1121-5. doi: https://doi.org/10.1038/nmeth.3661
Marx V. Autophagy: eat thyself, sustain thyself. Nat Methods. 2015 Dec 01;12(12):1121-5. doi: 10.1038/nmeth.3661. PMID: 26624796.
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Novel diet-related mouse model of colon cancer parallels human colon cancer.

World journal of gastrointestinal oncology

Prasad AR, Prasad S, Nguyen H, Facista A, Lewis C, Zaitlin B, Bernstein H, Bernstein C.
PMID: 25024814
World J Gastrointest Oncol. 2014 Jul 15;6(7):225-43. doi: 10.4251/wjgo.v6.i7.225.

AIM: To investigate the close parallels between our novel diet-related mouse model of colon cancer and human colon cancer.METHODS: Twenty-two wild-type female mice (ages 6-8 wk) were fed the standard control diet (AIN-93G) and an additional 22 female mice...

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Prasad AR, Prasad S, Nguyen H, et al. Novel diet-related mouse model of colon cancer parallels human colon cancer. World J Gastrointest Oncol. 2014;6(7):225-43doi: 10.4251/wjgo.v6.i7.225.
Prasad, A. R., Prasad, S., Nguyen, H., Facista, A., Lewis, C., Zaitlin, B., Bernstein, H., & Bernstein, C. (2014). Novel diet-related mouse model of colon cancer parallels human colon cancer. World journal of gastrointestinal oncology, 6(7), 225-43. https://doi.org/10.4251/wjgo.v6.i7.225
Prasad, Anil R, et al. "Novel diet-related mouse model of colon cancer parallels human colon cancer." World journal of gastrointestinal oncology vol. 6,7 (2014): 225-43. doi: https://doi.org/10.4251/wjgo.v6.i7.225
Prasad AR, Prasad S, Nguyen H, Facista A, Lewis C, Zaitlin B, Bernstein H, Bernstein C. Novel diet-related mouse model of colon cancer parallels human colon cancer. World J Gastrointest Oncol. 2014 Jul 15;6(7):225-43. doi: 10.4251/wjgo.v6.i7.225. PMID: 25024814; PMCID: PMC4092339.
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Repurposing Drugs in Oncology (ReDO)-clarithromycin as an anti-cancer agent.

Ecancermedicalscience

Van Nuffel AM, Sukhatme V, Pantziarka P, Meheus L, Sukhatme VP, Bouche G.
PMID: 25729426
Ecancermedicalscience. 2015 Feb 24;9:513. doi: 10.3332/ecancer.2015.513. eCollection 2015.

Clarithromycin (CAM) is a well-known macrolide antibiotic available as a generic drug. CAM is traditionally used for many types of bacterial infections, treatment of Lyme disease and eradication of gastric infection with Helicobacter pylori. Extensive preclinical and clinical data...

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Van Nuffel AM, Sukhatme V, Pantziarka P, et al. Repurposing Drugs in Oncology (ReDO)-clarithromycin as an anti-cancer agent. Ecancermedicalscience. 2015;9:513doi: 10.3332/ecancer.2015.513.
Van Nuffel, A. M., Sukhatme, V., Pantziarka, P., Meheus, L., Sukhatme, V. P., & Bouche, G. (2015). Repurposing Drugs in Oncology (ReDO)-clarithromycin as an anti-cancer agent. Ecancermedicalscience, 9513. https://doi.org/10.3332/ecancer.2015.513
Van Nuffel, An Mt, et al. "Repurposing Drugs in Oncology (ReDO)-clarithromycin as an anti-cancer agent." Ecancermedicalscience vol. 9 (2015): 513. doi: https://doi.org/10.3332/ecancer.2015.513
Van Nuffel AM, Sukhatme V, Pantziarka P, Meheus L, Sukhatme VP, Bouche G. Repurposing Drugs in Oncology (ReDO)-clarithromycin as an anti-cancer agent. Ecancermedicalscience. 2015 Feb 24;9:513. doi: 10.3332/ecancer.2015.513. eCollection 2015. PMID: 25729426; PMCID: PMC4341996.
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Microbiome: Autophagy genes link OMVs to IBD.

Nature reviews. Microbiology

Attar N.
PMID: 27180939
Nat Rev Microbiol. 2016 Jul;14(7):403. doi: 10.1038/nrmicro.2016.76. Epub 2016 May 16.

No abstract available.

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Attar N. Microbiome: Autophagy genes link OMVs to IBD. Nat Rev Microbiol. 2016;14(7):403doi: 10.1038/nrmicro.2016.76.
Attar, N. (2016). Microbiome: Autophagy genes link OMVs to IBD. Nature reviews. Microbiology, 14(7), 403. https://doi.org/10.1038/nrmicro.2016.76
Attar, Naomi. "Microbiome: Autophagy genes link OMVs to IBD." Nature reviews. Microbiology vol. 14,7 (2016): 403. doi: https://doi.org/10.1038/nrmicro.2016.76
Attar N. Microbiome: Autophagy genes link OMVs to IBD. Nat Rev Microbiol. 2016 Jul;14(7):403. doi: 10.1038/nrmicro.2016.76. Epub 2016 May 16. PMID: 27180939.
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Ypt/Rab GTPases regulate two intersections of the secretory and the endosomal/lysosomal pathways.

Cellular logistics

Lipatova Z, Segev N.
PMID: 25610722
Cell Logist. 2014 Jul 03;4(3):e954870. doi: 10.4161/21592780.2014.954870. eCollection 2014.

A prevailing question in the Ypt/Rab field is whether these conserved GTPases are specific to cellular compartments. The established role for Ypt1 and its human homolog Rab1 is in endoplasmic reticulum (ER)-to-Golgi transport. More recently these regulators were implicated...

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Lipatova Z, Segev N. Ypt/Rab GTPases regulate two intersections of the secretory and the endosomal/lysosomal pathways. Cell Logist. 2014;4(3):e954870doi: 10.4161/21592780.2014.954870.
Lipatova, Z., & Segev, N. (2014). Ypt/Rab GTPases regulate two intersections of the secretory and the endosomal/lysosomal pathways. Cellular logistics, 4(3), e954870. https://doi.org/10.4161/21592780.2014.954870
Lipatova, Zhanna, and Segev, Nava. "Ypt/Rab GTPases regulate two intersections of the secretory and the endosomal/lysosomal pathways." Cellular logistics vol. 4,3 (2014): e954870. doi: https://doi.org/10.4161/21592780.2014.954870
Lipatova Z, Segev N. Ypt/Rab GTPases regulate two intersections of the secretory and the endosomal/lysosomal pathways. Cell Logist. 2014 Jul 03;4(3):e954870. doi: 10.4161/21592780.2014.954870. eCollection 2014. PMID: 25610722; PMCID: PMC4279804.
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Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways.

Frontiers in microbiology

Alem F, Yao K, Lane D, Calvert V, Petricoin EF, Kramer L, Hale ML, Bavari S, Panchal RG, Hakami RM.
PMID: 25762983
Front Microbiol. 2015 Feb 13;6:50. doi: 10.3389/fmicb.2015.00050. eCollection 2015.

Yersinia pestis (Yp) causes the re-emerging disease plague, and is classified by the CDC and NIAID as a highest priority (Category A) pathogen. Currently, there is no approved human vaccine available and advances in early diagnostics and effective therapeutics...

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Alem F, Yao K, Lane D, et al. Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways. Front Microbiol. 2015;6:50doi: 10.3389/fmicb.2015.00050.
Alem, F., Yao, K., Lane, D., Calvert, V., Petricoin, E. F., Kramer, L., Hale, M. L., Bavari, S., Panchal, R. G., & Hakami, R. M. (2015). Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways. Frontiers in microbiology, 650. https://doi.org/10.3389/fmicb.2015.00050
Alem, Farhang, et al. "Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways." Frontiers in microbiology vol. 6 (2015): 50. doi: https://doi.org/10.3389/fmicb.2015.00050
Alem F, Yao K, Lane D, Calvert V, Petricoin EF, Kramer L, Hale ML, Bavari S, Panchal RG, Hakami RM. Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways. Front Microbiol. 2015 Feb 13;6:50. doi: 10.3389/fmicb.2015.00050. eCollection 2015. PMID: 25762983; PMCID: PMC4327736.
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Showing 25 to 36 of 14533 entries