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Inhibitory Effect of Yongdamsagan-Tang Water Extract, a Traditional Herbal Formula, on Testosterone-Induced Benign Prostatic Hyperplasia in Rats.

Evidence-based complementary and alternative medicine : eCAM

Park E, Lee MY, Jeon WY, Lee N, Seo CS, Shin HK.
PMID: 27504137
Evid Based Complement Alternat Med. 2016;2016:1428923. doi: 10.1155/2016/1428923. Epub 2016 Jul 18.

Yongdamsagan-tang, a traditional herbal formula, is used widely for the treatment of inflammation and viral diseases. In this study, we investigated whether Yongdamsagan-tang water extract (YSTE) affects testosterone propionate- (TP-) induced benign prostatic hyperplasia (BPH) in a rat model....

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Park E, Lee MY, Jeon WY, et al. Inhibitory Effect of Yongdamsagan-Tang Water Extract, a Traditional Herbal Formula, on Testosterone-Induced Benign Prostatic Hyperplasia in Rats. Evid Based Complement Alternat Med. 2016;2016:1428923doi: 10.1155/2016/1428923.
Park, E., Lee, M. Y., Jeon, W. Y., Lee, N., Seo, C. S., & Shin, H. K. (2016). Inhibitory Effect of Yongdamsagan-Tang Water Extract, a Traditional Herbal Formula, on Testosterone-Induced Benign Prostatic Hyperplasia in Rats. Evidence-based complementary and alternative medicine : eCAM, 20161428923. https://doi.org/10.1155/2016/1428923
Park, Eunsook, et al. "Inhibitory Effect of Yongdamsagan-Tang Water Extract, a Traditional Herbal Formula, on Testosterone-Induced Benign Prostatic Hyperplasia in Rats." Evidence-based complementary and alternative medicine : eCAM vol. 2016 (2016): 1428923. doi: https://doi.org/10.1155/2016/1428923
Park E, Lee MY, Jeon WY, Lee N, Seo CS, Shin HK. Inhibitory Effect of Yongdamsagan-Tang Water Extract, a Traditional Herbal Formula, on Testosterone-Induced Benign Prostatic Hyperplasia in Rats. Evid Based Complement Alternat Med. 2016;2016:1428923. doi: 10.1155/2016/1428923. Epub 2016 Jul 18. PMID: 27504137; PMCID: PMC4967681.
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Clinical Research Abstracts of the British Equine Veterinary Association Congress 2015.

Equine veterinary journal

Bardell DA, Archer DC, Milner PI.
PMID: 26375860
Equine Vet J. 2015 Sep;47:6. doi: 10.1111/evj.12486_12.

REASONS FOR PERFORMING STUDY: Colic remains a life-threatening condition in the horse. Ischaemia and reperfusion following correction of small intestinal strangulation may produce oxidative stress. The ability to withstand oxidative stress depends on antioxidant levels and may be linked...

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Bardell DA, Archer DC, Milner PI. Clinical Research Abstracts of the British Equine Veterinary Association Congress 2015. Equine Vet J. 2015;47:6doi: 10.1111/evj.12486_12.
Bardell, D. A., Archer, D. C., & Milner, P. I. (2015). Clinical Research Abstracts of the British Equine Veterinary Association Congress 2015. Equine veterinary journal, 476. https://doi.org/10.1111/evj.12486_12
Bardell, D A, et al. "Clinical Research Abstracts of the British Equine Veterinary Association Congress 2015." Equine veterinary journal vol. 47 (2015): 6. doi: https://doi.org/10.1111/evj.12486_12
Bardell DA, Archer DC, Milner PI. Clinical Research Abstracts of the British Equine Veterinary Association Congress 2015. Equine Vet J. 2015 Sep;47:6. doi: 10.1111/evj.12486_12. PMID: 26375860.
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Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells.

BioResearch open access

Hachem LD, Mothe AJ, Tator CH.
PMID: 26309791
Biores Open Access. 2015 Feb 01;4(1):146-59. doi: 10.1089/biores.2014.0058. eCollection 2015.

Transplantation of neural stem/progenitor cells (NSPCs) is a promising strategy in spinal cord injury (SCI). However, poor survival of transplanted stem cells remains a major limitation of this therapy due to the hostile environment of the injured cord. Oxidative...

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Hachem LD, Mothe AJ, Tator CH. Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells. Biores Open Access. 2015;4(1):146-59doi: 10.1089/biores.2014.0058.
Hachem, L. D., Mothe, A. J., & Tator, C. H. (2015). Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells. BioResearch open access, 4(1), 146-59. https://doi.org/10.1089/biores.2014.0058
Hachem, Laureen D, et al. "Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells." BioResearch open access vol. 4,1 (2015): 146-59. doi: https://doi.org/10.1089/biores.2014.0058
Hachem LD, Mothe AJ, Tator CH. Effect of BDNF and Other Potential Survival Factors in Models of In Vitro Oxidative Stress on Adult Spinal Cord-Derived Neural Stem/Progenitor Cells. Biores Open Access. 2015 Feb 01;4(1):146-59. doi: 10.1089/biores.2014.0058. eCollection 2015. PMID: 26309791; PMCID: PMC4497651.
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Disulfide bond formation of thiols by using carbon nanotubes.

Nanoscale

Hirano A, Kameda T, Sakuraba S, Wada M, Tanaka T, Kataura H.
PMID: 28422249
Nanoscale. 2017 May 04;9(17):5389-5393. doi: 10.1039/c7nr01001j.

Clarification of the interactions between carbon nanotubes (CNTs) and proteinogenic amino acids is a key approach to understanding CNT-protein interactions. Previous studies have addressed the mechanism of the physical adsorption of amino acids onto CNTs. However, little is known...

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Hirano A, Kameda T, Sakuraba S, et al. Disulfide bond formation of thiols by using carbon nanotubes. Nanoscale. 2017;9(17):5389-5393doi: 10.1039/c7nr01001j.
Hirano, A., Kameda, T., Sakuraba, S., Wada, M., Tanaka, T., & Kataura, H. (2017). Disulfide bond formation of thiols by using carbon nanotubes. Nanoscale, 9(17), 5389-5393. https://doi.org/10.1039/c7nr01001j
Hirano, A, et al. "Disulfide bond formation of thiols by using carbon nanotubes." Nanoscale vol. 9,17 (2017): 5389-5393. doi: https://doi.org/10.1039/c7nr01001j
Hirano A, Kameda T, Sakuraba S, Wada M, Tanaka T, Kataura H. Disulfide bond formation of thiols by using carbon nanotubes. Nanoscale. 2017 May 04;9(17):5389-5393. doi: 10.1039/c7nr01001j. PMID: 28422249.
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Calcitriol promotes survival of experimental random pattern flap via activation of autophagy.

American journal of translational research

Chen L, Zhou K, Chen H, Li S, Lin D, Zhou D.
PMID: 28861155
Am J Transl Res. 2017 Aug 15;9(8):3642-3653. eCollection 2017.

Calcitriol can promote random pattern flap survival and increase autophagy activity. However, effect of autophagy on flap survival after calcitriol treatment, along with the underlying mechanism, is undefined. In this study, the "McFarlane flap" model was established in 72...

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Chen L, Zhou K, Chen H, et al. Calcitriol promotes survival of experimental random pattern flap via activation of autophagy. Am J Transl Res. 2017;9(8):3642-3653
Chen, L., Zhou, K., Chen, H., Li, S., Lin, D., & Zhou, D. (2017). Calcitriol promotes survival of experimental random pattern flap via activation of autophagy. American journal of translational research, 9(8), 3642-3653.
Chen, Long, et al. "Calcitriol promotes survival of experimental random pattern flap via activation of autophagy." American journal of translational research vol. 9,8 (2017): 3642-3653.
Chen L, Zhou K, Chen H, Li S, Lin D, Zhou D. Calcitriol promotes survival of experimental random pattern flap via activation of autophagy. Am J Transl Res. 2017 Aug 15;9(8):3642-3653. eCollection 2017. PMID: 28861155; PMCID: PMC5575178.
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ESR Spin Trapping Studies of Free Radicals Generated from Nitrofuran Derivative Analogues of Nifurtimox by Electrochemical and Trypanosoma cruzi Reduction.

Free radical research

Olea-Azar C, Rigol C, Mendizabal F, Morello A, Diego Maya J, Moncada C, Cabrera E, di Maio R, González M, Cerecetto H.
PMID: 28447529
Free Radic Res. 2003;37(9):993-1001. doi: 10.1080/10715760310001598141.

Electron spin resonance (ESR) spectra of radicals obtained from two analogues of the antiprotozoal drug nifurtimox by electrolytic and Trypanosoma cruzi reduction were analyzed. The electrochemistry of these compounds was studied using cyclic voltammetry. STO 3-21G ab initio and...

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Olea-Azar C, Rigol C, Mendizabal F, et al. ESR Spin Trapping Studies of Free Radicals Generated from Nitrofuran Derivative Analogues of Nifurtimox by Electrochemical and Trypanosoma cruzi Reduction. Free Radic Res. 2003;37(9):993-1001doi: 10.1080/10715760310001598141.
Olea-Azar, C., Rigol, C., Mendizabal, F., Morello, A., Diego Maya, J., Moncada, C., Cabrera, E., di Maio, R., González, M., & Cerecetto, H. (2003). ESR Spin Trapping Studies of Free Radicals Generated from Nitrofuran Derivative Analogues of Nifurtimox by Electrochemical and Trypanosoma cruzi Reduction. Free radical research, 37(9), 993-1001. https://doi.org/10.1080/10715760310001598141
Olea-Azar, Claudio, et al. "ESR Spin Trapping Studies of Free Radicals Generated from Nitrofuran Derivative Analogues of Nifurtimox by Electrochemical and Trypanosoma cruzi Reduction." Free radical research vol. 37,9 (2003): 993-1001. doi: https://doi.org/10.1080/10715760310001598141
Olea-Azar C, Rigol C, Mendizabal F, Morello A, Diego Maya J, Moncada C, Cabrera E, di Maio R, González M, Cerecetto H. ESR Spin Trapping Studies of Free Radicals Generated from Nitrofuran Derivative Analogues of Nifurtimox by Electrochemical and Trypanosoma cruzi Reduction. Free Radic Res. 2003;37(9):993-1001. doi: 10.1080/10715760310001598141. PMID: 28447529.
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Dosakaya Juice Assuages Development of Sucrose Induced Impaired Glucose Tolerance and Imbalance in Antioxidant Defense.

Pharmacognosy magazine

Kumar DA, Sweeya PS, Shukla S, Anusha SV, Akshara D, Madhusudana K, Tiwari AK.
PMID: 26929582
Pharmacogn Mag. 2015 Oct;11:S462-8. doi: 10.4103/0973-1296.168985.

OBJECTIVE: The objective was to explore the effect of Dosakaya (DK) (Cucumis melo var. chito) juice on sucrose induced dysglycemia and disturbances in antioxidant defense in rats.MATERIALS AND METHODS: Rats were preconditioned with DK juice before administration of sucrose...

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Kumar DA, Sweeya PS, Shukla S, et al. Dosakaya Juice Assuages Development of Sucrose Induced Impaired Glucose Tolerance and Imbalance in Antioxidant Defense. Pharmacogn Mag. 2015;11:S462-8doi: 10.4103/0973-1296.168985.
Kumar, D. A., Sweeya, P. S., Shukla, S., Anusha, S. V., Akshara, D., Madhusudana, K., & Tiwari, A. K. (2015). Dosakaya Juice Assuages Development of Sucrose Induced Impaired Glucose Tolerance and Imbalance in Antioxidant Defense. Pharmacognosy magazine, 11S462-8. https://doi.org/10.4103/0973-1296.168985
Kumar, Dommati Anand, et al. "Dosakaya Juice Assuages Development of Sucrose Induced Impaired Glucose Tolerance and Imbalance in Antioxidant Defense." Pharmacognosy magazine vol. 11 (2015): S462-8. doi: https://doi.org/10.4103/0973-1296.168985
Kumar DA, Sweeya PS, Shukla S, Anusha SV, Akshara D, Madhusudana K, Tiwari AK. Dosakaya Juice Assuages Development of Sucrose Induced Impaired Glucose Tolerance and Imbalance in Antioxidant Defense. Pharmacogn Mag. 2015 Oct;11:S462-8. doi: 10.4103/0973-1296.168985. PMID: 26929582; PMCID: PMC4745218.
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Subchronic toxicity of Nile tilapia with different exposure routes to .

Veterinary world

Abdel-Latif HMR, Khashaba AMA.
PMID: 28919690
Vet World. 2017 Aug;10(8):955-963. doi: 10.14202/vetworld.2017.955-963. Epub 2017 Aug 21.

BACKGROUND: Toxic cyanobacterial blooms (AIM: Three-week subchronic exposure experiment was carried out on Nile tilapia, to determine their effects on fish behavior, tissues, liver functions, antioxidant enzymes, and lipid peroxidation.MATERIALS AND METHODS: Fish were exposed to four main treatments;...

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Abdel-Latif HMR, Khashaba AMA. Subchronic toxicity of Nile tilapia with different exposure routes to . Vet World. 2017;10(8):955-963doi: 10.14202/vetworld.2017.955-963.
Abdel-Latif, H. M. R., & Khashaba, A. M. A. (2017). Subchronic toxicity of Nile tilapia with different exposure routes to . Veterinary world, 10(8), 955-963. https://doi.org/10.14202/vetworld.2017.955-963
Abdel-Latif, H M R, and Khashaba, A M Abou. "Subchronic toxicity of Nile tilapia with different exposure routes to ." Veterinary world vol. 10,8 (2017): 955-963. doi: https://doi.org/10.14202/vetworld.2017.955-963
Abdel-Latif HMR, Khashaba AMA. Subchronic toxicity of Nile tilapia with different exposure routes to . Vet World. 2017 Aug;10(8):955-963. doi: 10.14202/vetworld.2017.955-963. Epub 2017 Aug 21. PMID: 28919690; PMCID: PMC5591486.
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Ameliorative effect of vitamin E on trichloroethylene-induced nephrotoxicity in rats.

Journal of nephropathology

Heydari M, Ahmadizadeh M, Ahmadi Angali K.
PMID: 28975097
J Nephropathol. 2017 Jul;6(3):168-173. doi: 10.15171/jnp.2017.29. Epub 2016 Dec 20.

BACKGROUND: 1,1,2-Trichloroethylene (TCE) is an important organic solvent which is widespread in the environment. Work place exposure to TCE has been associated adverse effects in many organs including kidney. Vitamin E is an antioxidant that can overcome oxidative stress.OBJECTIVES:...

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Heydari M, Ahmadizadeh M, Ahmadi Angali K. Ameliorative effect of vitamin E on trichloroethylene-induced nephrotoxicity in rats. J Nephropathol. 2016;6(3):168-173doi: 10.15171/jnp.2017.29.
Heydari, M., Ahmadizadeh, M., & Ahmadi Angali, K. (2017). Ameliorative effect of vitamin E on trichloroethylene-induced nephrotoxicity in rats. Journal of nephropathology, 6(3), 168-173. https://doi.org/10.15171/jnp.2017.29
Heydari, Mojgan, et al. "Ameliorative effect of vitamin E on trichloroethylene-induced nephrotoxicity in rats." Journal of nephropathology vol. 6,3 (2017): 168-173. doi: https://doi.org/10.15171/jnp.2017.29
Heydari M, Ahmadizadeh M, Ahmadi Angali K. Ameliorative effect of vitamin E on trichloroethylene-induced nephrotoxicity in rats. J Nephropathol. 2017 Jul;6(3):168-173. doi: 10.15171/jnp.2017.29. Epub 2016 Dec 20. PMID: 28975097; PMCID: PMC5607979.
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Cell Signaling in Mesothelial Cells by Asbestos: Evidence for the Involvement of Oxidative Stress in the Regulation of the Epidermal Growth Factor Receptor.

Inhalation toxicology

Faux SP, Houghton CE.
PMID: 26368632
Inhal Toxicol. 2000 Jan;12:327-36. doi: 10.1080/08958378.2000.11463242.

Asbestos has been shown to stimulate the mitogen-activated protein kinase signaling cascade after autophosphoryiation of the epidermal growth factor recptor (EGF-R), an event important in regulating the response of cells to extracellular signals. In studies reported here, we have...

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Faux SP, Houghton CE. Cell Signaling in Mesothelial Cells by Asbestos: Evidence for the Involvement of Oxidative Stress in the Regulation of the Epidermal Growth Factor Receptor. Inhal Toxicol. 2000;12:327-36doi: 10.1080/08958378.2000.11463242.
Faux, S. P., & Houghton, C. E. (2000). Cell Signaling in Mesothelial Cells by Asbestos: Evidence for the Involvement of Oxidative Stress in the Regulation of the Epidermal Growth Factor Receptor. Inhalation toxicology, 12327-36. https://doi.org/10.1080/08958378.2000.11463242
Faux, S P, and Houghton, C E. "Cell Signaling in Mesothelial Cells by Asbestos: Evidence for the Involvement of Oxidative Stress in the Regulation of the Epidermal Growth Factor Receptor." Inhalation toxicology vol. 12 (2000): 327-36. doi: https://doi.org/10.1080/08958378.2000.11463242
Faux SP, Houghton CE. Cell Signaling in Mesothelial Cells by Asbestos: Evidence for the Involvement of Oxidative Stress in the Regulation of the Epidermal Growth Factor Receptor. Inhal Toxicol. 2000 Jan;12:327-36. doi: 10.1080/08958378.2000.11463242. PMID: 26368632.
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PRAS40 promotes NF-κB transcriptional activity through association with p65.

Oncogenesis

Zhu G, Qi Q, Havel JJ, Li Z, Du Y, Zhang X, Fu H.
PMID: 28945219
Oncogenesis. 2017 Sep 25;6(9):e381. doi: 10.1038/oncsis.2017.80.

PRAS40 has been shown to have a crucial role in the repression of mammalian target of rapamycin (mTOR). Nonetheless, PRAS40 appears to have an oncogenic function in cancer cells. Whether PRAS40 mediates signaling independent of mTOR inhibition in cancer...

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Zhu G, Qi Q, Havel JJ, et al. PRAS40 promotes NF-κB transcriptional activity through association with p65. Oncogenesis. 2017;6(9):e381doi: 10.1038/oncsis.2017.80.
Zhu, G., Qi, Q., Havel, J. J., Li, Z., Du, Y., Zhang, X., & Fu, H. (2017). PRAS40 promotes NF-κB transcriptional activity through association with p65. Oncogenesis, 6(9), e381. https://doi.org/10.1038/oncsis.2017.80
Zhu, G, et al. "PRAS40 promotes NF-κB transcriptional activity through association with p65." Oncogenesis vol. 6,9 (2017): e381. doi: https://doi.org/10.1038/oncsis.2017.80
Zhu G, Qi Q, Havel JJ, Li Z, Du Y, Zhang X, Fu H. PRAS40 promotes NF-κB transcriptional activity through association with p65. Oncogenesis. 2017 Sep 25;6(9):e381. doi: 10.1038/oncsis.2017.80. PMID: 28945219; PMCID: PMC5623906.
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Ameliorative Effects of Chloroform Fraction of Cocos nucifera L. Husk Fiber Against Cisplatin-induced Toxicity in Rats.

Pharmacognosy research

Adaramoye OA, Azeez AF, Ola-Davies OE.
PMID: 27034598
Pharmacognosy Res. 2016 Apr-Jun;8(2):89-96. doi: 10.4103/0974-8490.172658.

BACKGROUND: Cisplatin (Cis) is used in the treatment of solid tumors and is known to elicit serious side effects.OBJECTIVE: The present study investigated the protective effects of chloroform fraction of Cocos nucifera husk fiber (CFCN) against Cis-induced organs' damage...

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Adaramoye OA, Azeez AF, Ola-Davies OE. Ameliorative Effects of Chloroform Fraction of Cocos nucifera L. Husk Fiber Against Cisplatin-induced Toxicity in Rats. Pharmacognosy Res. 2016;8(2):89-96doi: 10.4103/0974-8490.172658.
Adaramoye, O. A., Azeez, A. F., & Ola-Davies, O. E. (2016). Ameliorative Effects of Chloroform Fraction of Cocos nucifera L. Husk Fiber Against Cisplatin-induced Toxicity in Rats. Pharmacognosy research, 8(2), 89-96. https://doi.org/10.4103/0974-8490.172658
Adaramoye, Oluwatosin Adekunle, et al. "Ameliorative Effects of Chloroform Fraction of Cocos nucifera L. Husk Fiber Against Cisplatin-induced Toxicity in Rats." Pharmacognosy research vol. 8,2 (2016): 89-96. doi: https://doi.org/10.4103/0974-8490.172658
Adaramoye OA, Azeez AF, Ola-Davies OE. Ameliorative Effects of Chloroform Fraction of Cocos nucifera L. Husk Fiber Against Cisplatin-induced Toxicity in Rats. Pharmacognosy Res. 2016 Apr-Jun;8(2):89-96. doi: 10.4103/0974-8490.172658. PMID: 27034598; PMCID: PMC4780144.
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Showing 13 to 24 of 12371 entries