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Elastography and Contrast-enhanced Ultrasonography in the Early Detection of Hepatocellular Carcinoma in an Experimental Model of Nonalcoholic Steatohepatitis.

Journal of clinical and experimental hepatology

Carvalho CF, Chammas MC, Souza de Oliveira CP, Cogliati B, Carrilho FJ, Cerri GG.
PMID: 25755482
J Clin Exp Hepatol. 2013 Jun;3(2):96-101. doi: 10.1016/j.jceh.2013.04.004. Epub 2013 May 15.

BACKGROUND/OBJECTIVE: The early detection of focal hepatic lesions using ultrasound scanning is challenging, and this challenge becomes even greater in the presence of diffuse parenchymal disease. This study aimed to evaluate the diagnostic performance of elastography and contrast-enhanced ultrasonography...

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Carvalho CF, Chammas MC, Souza de Oliveira CP, et al. Elastography and Contrast-enhanced Ultrasonography in the Early Detection of Hepatocellular Carcinoma in an Experimental Model of Nonalcoholic Steatohepatitis. J Clin Exp Hepatol. 2013;3(2):96-101doi: 10.1016/j.jceh.2013.04.004.
Carvalho, C. F., Chammas, M. C., Souza de Oliveira, C. P., Cogliati, B., Carrilho, F. J., & Cerri, G. G. (2013). Elastography and Contrast-enhanced Ultrasonography in the Early Detection of Hepatocellular Carcinoma in an Experimental Model of Nonalcoholic Steatohepatitis. Journal of clinical and experimental hepatology, 3(2), 96-101. https://doi.org/10.1016/j.jceh.2013.04.004
Carvalho, Cibele F, et al. "Elastography and Contrast-enhanced Ultrasonography in the Early Detection of Hepatocellular Carcinoma in an Experimental Model of Nonalcoholic Steatohepatitis." Journal of clinical and experimental hepatology vol. 3,2 (2013): 96-101. doi: https://doi.org/10.1016/j.jceh.2013.04.004
Carvalho CF, Chammas MC, Souza de Oliveira CP, Cogliati B, Carrilho FJ, Cerri GG. Elastography and Contrast-enhanced Ultrasonography in the Early Detection of Hepatocellular Carcinoma in an Experimental Model of Nonalcoholic Steatohepatitis. J Clin Exp Hepatol. 2013 Jun;3(2):96-101. doi: 10.1016/j.jceh.2013.04.004. Epub 2013 May 15. PMID: 25755482; PMCID: PMC3940387.
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Connexins and pannexins in liver damage.

EXCLI journal

Crespo Yanguas S, Willebrords J, Maes M, da Silva TC, Veloso Alves Pereira I, Cogliati B, Zaidan Dagli ML, Vinken M.
PMID: 27065778
EXCLI J. 2016 Feb 25;15:177-86. doi: 10.17179/excli2016-119. eCollection 2016.

Connexins and pannexins are key players in the control of cellular communication and thus in the maintenance of tissue homeostasis. Inherent to this function these proteins are frequently involved in pathological processes. The present paper reviews the role of...

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Crespo Yanguas S, Willebrords J, Maes M, et al. Connexins and pannexins in liver damage. EXCLI J. 2016;15:177-86doi: 10.17179/excli2016-119.
Crespo Yanguas, S., Willebrords, J., Maes, M., da Silva, T. C., Veloso Alves Pereira, I., Cogliati, B., Zaidan Dagli, M. L., & Vinken, M. (2016). Connexins and pannexins in liver damage. EXCLI journal, 15177-86. https://doi.org/10.17179/excli2016-119
Crespo Yanguas, Sara, et al. "Connexins and pannexins in liver damage." EXCLI journal vol. 15 (2016): 177-86. doi: https://doi.org/10.17179/excli2016-119
Crespo Yanguas S, Willebrords J, Maes M, da Silva TC, Veloso Alves Pereira I, Cogliati B, Zaidan Dagli ML, Vinken M. Connexins and pannexins in liver damage. EXCLI J. 2016 Feb 25;15:177-86. doi: 10.17179/excli2016-119. eCollection 2016. PMID: 27065778; PMCID: PMC4822047.
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Congenital hepatic fibrosis and polycystic kidney disease not linked to C >A mutation in exon 29 of .

JFMS open reports

Guerra JM, Daniel AGT, Cardoso NC, Grandi F, Queiroga F, Cogliati B.
PMID: 28491400
JFMS Open Rep. 2015 Dec 06;1(2):2055116915619191. doi: 10.1177/2055116915619191. eCollection 2015.

CASE SUMMARY: We describe the case of a 1-year-old male Persian cat diagnosed with congenital hepatic fibrosis (CHF) associated with renal polycystic disease and, for the first time, we have shown that there was no C >A mutation in...

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Guerra JM, Daniel AGT, Cardoso NC, et al. Congenital hepatic fibrosis and polycystic kidney disease not linked to C >A mutation in exon 29 of . JFMS Open Rep. 2015;1(2):2055116915619191doi: 10.1177/2055116915619191.
Guerra, J. M., Daniel, A. G. T., Cardoso, N. C., Grandi, F., Queiroga, F., & Cogliati, B. (2015). Congenital hepatic fibrosis and polycystic kidney disease not linked to C >A mutation in exon 29 of . JFMS open reports, 1(2), 2055116915619191. https://doi.org/10.1177/2055116915619191
Guerra, Juliana Mariotti, et al. "Congenital hepatic fibrosis and polycystic kidney disease not linked to C >A mutation in exon 29 of ." JFMS open reports vol. 1,2 (2015): 2055116915619191. doi: https://doi.org/10.1177/2055116915619191
Guerra JM, Daniel AGT, Cardoso NC, Grandi F, Queiroga F, Cogliati B. Congenital hepatic fibrosis and polycystic kidney disease not linked to C >A mutation in exon 29 of . JFMS Open Rep. 2015 Dec 06;1(2):2055116915619191. doi: 10.1177/2055116915619191. eCollection 2015. PMID: 28491400; PMCID: PMC5362006.
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MyomiRs as Markers of Insulin Resistance and Decreased Myogenesis in Skeletal Muscle of Diet-Induced Obese Mice.

Frontiers in endocrinology

Frias Fde T, de Mendonça M, Martins AR, Gindro AF, Cogliati B, Curi R, Rodrigues AC.
PMID: 27445979
Front Endocrinol (Lausanne). 2016 Jun 27;7:76. doi: 10.3389/fendo.2016.00076. eCollection 2016.

High-fat diet (HFD) feeding causes insulin resistance (IR) in skeletal muscle of mice, which affects skeletal muscle metabolism and function. The involvement of muscle-specific microRNAs in the evolution of skeletal muscle IR during 4, 8, and 12 weeks in...

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Frias Fde T, de Mendonça M, Martins AR, et al. MyomiRs as Markers of Insulin Resistance and Decreased Myogenesis in Skeletal Muscle of Diet-Induced Obese Mice. Front Endocrinol (Lausanne). 2016;7:76doi: 10.3389/fendo.2016.00076.
Frias, F. d. e. . T., de Mendonça, M., Martins, A. R., Gindro, A. F., Cogliati, B., Curi, R., & Rodrigues, A. C. (2016). MyomiRs as Markers of Insulin Resistance and Decreased Myogenesis in Skeletal Muscle of Diet-Induced Obese Mice. Frontiers in endocrinology, 776. https://doi.org/10.3389/fendo.2016.00076
Frias, Flávia de Toledo, et al. "MyomiRs as Markers of Insulin Resistance and Decreased Myogenesis in Skeletal Muscle of Diet-Induced Obese Mice." Frontiers in endocrinology vol. 7 (2016): 76. doi: https://doi.org/10.3389/fendo.2016.00076
Frias Fde T, de Mendonça M, Martins AR, Gindro AF, Cogliati B, Curi R, Rodrigues AC. MyomiRs as Markers of Insulin Resistance and Decreased Myogenesis in Skeletal Muscle of Diet-Induced Obese Mice. Front Endocrinol (Lausanne). 2016 Jun 27;7:76. doi: 10.3389/fendo.2016.00076. eCollection 2016. PMID: 27445979; PMCID: PMC4921801.
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Erratum to: Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity.

Archives of toxicology

Maes M, McGill MR, da Silva TC, Abels C, Lebofsky M, Weemhoff JL, Tiburcio T, Pereira IVA, Willebrords J, Yanguas SC, Farhood A, Beschin A, Van Ginderachter JA, Penuela S, Jaeschke H, Cogliati B, Vinken M.
PMID: 28243680
Arch Toxicol. 2017 May;91(5):2263-2264. doi: 10.1007/s00204-016-1929-y.

No abstract available.

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Maes M, McGill MR, da Silva TC, et al. Erratum to: Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity. Arch Toxicol. 2017;91(5):2263-2264doi: 10.1007/s00204-016-1929-y.
Maes, M., McGill, M. R., da Silva, T. C., Abels, C., Lebofsky, M., Weemhoff, J. L., Tiburcio, T., Pereira, I. V. A., Willebrords, J., Yanguas, S. C., Farhood, A., Beschin, A., Van Ginderachter, J. A., Penuela, S., Jaeschke, H., Cogliati, B., & Vinken, M. (2017). Erratum to: Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity. Archives of toxicology, 91(5), 2263-2264. https://doi.org/10.1007/s00204-016-1929-y
Maes, Michaël, et al. "Erratum to: Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity." Archives of toxicology vol. 91,5 (2017): 2263-2264. doi: https://doi.org/10.1007/s00204-016-1929-y
Maes M, McGill MR, da Silva TC, Abels C, Lebofsky M, Weemhoff JL, Tiburcio T, Pereira IVA, Willebrords J, Yanguas SC, Farhood A, Beschin A, Van Ginderachter JA, Penuela S, Jaeschke H, Cogliati B, Vinken M. Erratum to: Inhibition of pannexin1 channels alleviates acetaminophen-induced hepatotoxicity. Arch Toxicol. 2017 May;91(5):2263-2264. doi: 10.1007/s00204-016-1929-y. PMID: 28243680.
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[Introduce grades in medical education!].

Lakartidningen

Tagliati M.
PMID: 17212278
Lakartidningen. 2006 Dec 6-12;103(49):3956.

No abstract available.

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Tagliati M. Inför betyg på läkarutbildningen! [Introduce grades in medical education!]. Lakartidningen. 2006;103(49):3956
Tagliati, M. (2006). Inför betyg på läkarutbildningen! [Introduce grades in medical education!]. Lakartidningen, 103(49), 3956.
Tagliati, Michel. "Inför betyg på läkarutbildningen!" [Introduce grades in medical education!]. Lakartidningen vol. 103,49 (2006): 3956.
Tagliati M. Inför betyg på läkarutbildningen! [Introduce grades in medical education!]. Lakartidningen. 2006 Dec 6-12;103(49):3956. Swedish. PMID: 17212278.
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Prohibitin(g) cancer: aurilide and killing by Opa1-dependent cristae remodeling.

Chemistry & biology

Semenzato M, Cogliati S, Scorrano L.
PMID: 21276934
Chem Biol. 2011 Jan 28;18(1):8-9. doi: 10.1016/j.chembiol.2011.01.001.

Proapoptotic drugs targeting the mitochondrial Bcl-2 rheostat of apoptosis are tools to selectively kill cancer cells. Sato et al. (2011) expand the available toolkit by identifying the target of the cytotoxic natural product aurilide in the prohibitin Opa1-dependent apoptotic...

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Semenzato M, Cogliati S, Scorrano L. Prohibitin(g) cancer: aurilide and killing by Opa1-dependent cristae remodeling. Chem Biol. 2011;18(1):8-9doi: 10.1016/j.chembiol.2011.01.001.
Semenzato, M., Cogliati, S., & Scorrano, L. (2011). Prohibitin(g) cancer: aurilide and killing by Opa1-dependent cristae remodeling. Chemistry & biology, 18(1), 8-9. https://doi.org/10.1016/j.chembiol.2011.01.001
Semenzato, Martina, et al. "Prohibitin(g) cancer: aurilide and killing by Opa1-dependent cristae remodeling." Chemistry & biology vol. 18,1 (2011): 8-9. doi: https://doi.org/10.1016/j.chembiol.2011.01.001
Semenzato M, Cogliati S, Scorrano L. Prohibitin(g) cancer: aurilide and killing by Opa1-dependent cristae remodeling. Chem Biol. 2011 Jan 28;18(1):8-9. doi: 10.1016/j.chembiol.2011.01.001. PMID: 21276934.
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The hyperspectral irradiometer, a new instrument for long-term and unattended field spectroscopy measurements.

The Review of scientific instruments

Meroni M, Barducci A, Cogliati S, Castagnoli F, Rossini M, Busetto L, Migliavacca M, Cremonese E, Galvagno M, Colombo R, di Cella UM.
PMID: 21528994
Rev Sci Instrum. 2011 Apr;82(4):043106. doi: 10.1063/1.3574360.

Reliable time series of vegetation optical properties are needed to improve the modeling of the terrestrial carbon budget with remote sensing data. This paper describes the development of an automatic spectral system able to collect continuous long-term in-field spectral...

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Meroni M, Barducci A, Cogliati S, et al. The hyperspectral irradiometer, a new instrument for long-term and unattended field spectroscopy measurements. Rev Sci Instrum. 2011;82(4):043106doi: 10.1063/1.3574360.
Meroni, M., Barducci, A., Cogliati, S., Castagnoli, F., Rossini, M., Busetto, L., Migliavacca, M., Cremonese, E., Galvagno, M., Colombo, R., & di Cella, U. M. (2011). The hyperspectral irradiometer, a new instrument for long-term and unattended field spectroscopy measurements. The Review of scientific instruments, 82(4), 043106. https://doi.org/10.1063/1.3574360
Meroni, M, et al. "The hyperspectral irradiometer, a new instrument for long-term and unattended field spectroscopy measurements." The Review of scientific instruments vol. 82,4 (2011): 043106. doi: https://doi.org/10.1063/1.3574360
Meroni M, Barducci A, Cogliati S, Castagnoli F, Rossini M, Busetto L, Migliavacca M, Cremonese E, Galvagno M, Colombo R, di Cella UM. The hyperspectral irradiometer, a new instrument for long-term and unattended field spectroscopy measurements. Rev Sci Instrum. 2011 Apr;82(4):043106. doi: 10.1063/1.3574360. PMID: 21528994.
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Juxtaposition of heterochromatic and euchromatic regions by chromosomal translocation mediates a heterochromatic long-range position effect associated with a severe neurological phenotype.

Molecular cytogenetics

Finelli P, Sirchia SM, Masciadri M, Crippa M, Recalcati MP, Rusconi D, Giardino D, Monti L, Cogliati F, Faravelli F, Natacci F, Zoccante L, Bernardina BD, Russo S, Larizza L.
PMID: 22475481
Mol Cytogenet. 2012 Apr 04;5:16. doi: 10.1186/1755-8166-5-16.

BACKGROUND: The term "position effect" is used when the expression of a gene is deleteriously affected by an alteration in its chromosomal environment even though the integrity of the protein coding sequences is maintained. We describe a patient affected...

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Finelli P, Sirchia SM, Masciadri M, et al. Juxtaposition of heterochromatic and euchromatic regions by chromosomal translocation mediates a heterochromatic long-range position effect associated with a severe neurological phenotype. Mol Cytogenet. 2012;5:16doi: 10.1186/1755-8166-5-16.
Finelli, P., Sirchia, S. M., Masciadri, M., Crippa, M., Recalcati, M. P., Rusconi, D., Giardino, D., Monti, L., Cogliati, F., Faravelli, F., Natacci, F., Zoccante, L., Bernardina, B. D., Russo, S., & Larizza, L. (2012). Juxtaposition of heterochromatic and euchromatic regions by chromosomal translocation mediates a heterochromatic long-range position effect associated with a severe neurological phenotype. Molecular cytogenetics, 516. https://doi.org/10.1186/1755-8166-5-16
Finelli, Palma, et al. "Juxtaposition of heterochromatic and euchromatic regions by chromosomal translocation mediates a heterochromatic long-range position effect associated with a severe neurological phenotype." Molecular cytogenetics vol. 5 (2012): 16. doi: https://doi.org/10.1186/1755-8166-5-16
Finelli P, Sirchia SM, Masciadri M, Crippa M, Recalcati MP, Rusconi D, Giardino D, Monti L, Cogliati F, Faravelli F, Natacci F, Zoccante L, Bernardina BD, Russo S, Larizza L. Juxtaposition of heterochromatic and euchromatic regions by chromosomal translocation mediates a heterochromatic long-range position effect associated with a severe neurological phenotype. Mol Cytogenet. 2012 Apr 04;5:16. doi: 10.1186/1755-8166-5-16. PMID: 22475481; PMCID: PMC3395859.
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Effect of plant extracts on H2O2-induced inflammatory gene expression in macrophages.

Journal of inflammation research

Pomari E, Stefanon B, Colitti M.
PMID: 25075197
J Inflamm Res. 2014 Jun 24;7:103-12. doi: 10.2147/JIR.S61471. eCollection 2014.

BACKGROUND: Arctium lappa (AL), Camellia sinensis (CS), Echinacea angustifolia, Eleutherococcus senticosus, Panax ginseng (PG), and Vaccinium myrtillus (VM) are plants traditionally used in many herbal formulations for the treatment of various conditions. Although they are well known and already...

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Pomari E, Stefanon B, Colitti M. Effect of plant extracts on H2O2-induced inflammatory gene expression in macrophages. J Inflamm Res. 2014;7:103-12doi: 10.2147/JIR.S61471.
Pomari, E., Stefanon, B., & Colitti, M. (2014). Effect of plant extracts on H2O2-induced inflammatory gene expression in macrophages. Journal of inflammation research, 7103-12. https://doi.org/10.2147/JIR.S61471
Pomari, Elena, et al. "Effect of plant extracts on H2O2-induced inflammatory gene expression in macrophages." Journal of inflammation research vol. 7 (2014): 103-12. doi: https://doi.org/10.2147/JIR.S61471
Pomari E, Stefanon B, Colitti M. Effect of plant extracts on H2O2-induced inflammatory gene expression in macrophages. J Inflamm Res. 2014 Jun 24;7:103-12. doi: 10.2147/JIR.S61471. eCollection 2014. PMID: 25075197; PMCID: PMC4106015.
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Optimizing Parkinson's disease diagnosis: the role of a dual nuclear imaging algorithm.

NPJ Parkinson's disease

Langston JW, Wiley JC, Tagliati M.
PMID: 29507872
NPJ Parkinsons Dis. 2018 Feb 23;4:5. doi: 10.1038/s41531-018-0041-9. eCollection 2018.

The diagnosis of Parkinson's disease (PD) currently relies almost exclusively on the clinical judgment of an experienced neurologist, ideally a specialist in movement disorders. However, such clinical diagnosis is often incorrect in a large percentage of patients, particularly in...

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Langston JW, Wiley JC, Tagliati M. Optimizing Parkinson's disease diagnosis: the role of a dual nuclear imaging algorithm. NPJ Parkinsons Dis. 2018;4:5doi: 10.1038/s41531-018-0041-9.
Langston, J. W., Wiley, J. C., & Tagliati, M. (2018). Optimizing Parkinson's disease diagnosis: the role of a dual nuclear imaging algorithm. NPJ Parkinson's disease, 45. https://doi.org/10.1038/s41531-018-0041-9
Langston, J William, et al. "Optimizing Parkinson's disease diagnosis: the role of a dual nuclear imaging algorithm." NPJ Parkinson's disease vol. 4 (2018): 5. doi: https://doi.org/10.1038/s41531-018-0041-9
Langston JW, Wiley JC, Tagliati M. Optimizing Parkinson's disease diagnosis: the role of a dual nuclear imaging algorithm. NPJ Parkinsons Dis. 2018 Feb 23;4:5. doi: 10.1038/s41531-018-0041-9. eCollection 2018. PMID: 29507872; PMCID: PMC5824845.
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Recurrence and Familial Inheritance of Intronic .

Frontiers in neurology

Masciadri M, Ficcadenti A, Milani D, Cogliati F, Divizia MT, Larizza L, Russo S.
PMID: 30538663
Front Neurol. 2018 Nov 27;9:967. doi: 10.3389/fneur.2018.00967. eCollection 2018.

Splicing pathogenic variants account for a notable fraction of

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Masciadri M, Ficcadenti A, Milani D, et al. Recurrence and Familial Inheritance of Intronic . Front Neurol. 2018;9:967doi: 10.3389/fneur.2018.00967.
Masciadri, M., Ficcadenti, A., Milani, D., Cogliati, F., Divizia, M. T., Larizza, L., & Russo, S. (2018). Recurrence and Familial Inheritance of Intronic . Frontiers in neurology, 9967. https://doi.org/10.3389/fneur.2018.00967
Masciadri, Maura, et al. "Recurrence and Familial Inheritance of Intronic ." Frontiers in neurology vol. 9 (2018): 967. doi: https://doi.org/10.3389/fneur.2018.00967
Masciadri M, Ficcadenti A, Milani D, Cogliati F, Divizia MT, Larizza L, Russo S. Recurrence and Familial Inheritance of Intronic . Front Neurol. 2018 Nov 27;9:967. doi: 10.3389/fneur.2018.00967. eCollection 2018. PMID: 30538663; PMCID: PMC6277459.
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Showing 1 to 12 of 137 entries