Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 8 of 8 entries
Sorted by: Best Match Show Resources per page
The microtubule-associated protein PRC1 is a potential therapeutic target for lung cancer.

Oncotarget

Hanselmann S, Wolter P, Malkmus J, Gaubatz S.
PMID: 29435157
Oncotarget. 2017 Dec 22;9(4):4985-4997. doi: 10.18632/oncotarget.23577. eCollection 2018 Jan 12.

In this study, we investigated whether proteins that are involved in cytokinesis are potential targets for therapy of lung cancer. We find that the microtubule-associated protein PRC1 (protein required for cytokinesis 1), which plays a key role in organizing...

Cite
Hanselmann S, Wolter P, Malkmus J, et al. The microtubule-associated protein PRC1 is a potential therapeutic target for lung cancer. Oncotarget. 2017;9(4):4985-4997doi: 10.18632/oncotarget.23577.
Hanselmann, S., Wolter, P., Malkmus, J., & Gaubatz, S. (2018). The microtubule-associated protein PRC1 is a potential therapeutic target for lung cancer. Oncotarget, 9(4), 4985-4997. https://doi.org/10.18632/oncotarget.23577
Hanselmann, Steffen, et al. "The microtubule-associated protein PRC1 is a potential therapeutic target for lung cancer." Oncotarget vol. 9,4 (2018): 4985-4997. doi: https://doi.org/10.18632/oncotarget.23577
Hanselmann S, Wolter P, Malkmus J, Gaubatz S. The microtubule-associated protein PRC1 is a potential therapeutic target for lung cancer. Oncotarget. 2017 Dec 22;9(4):4985-4997. doi: 10.18632/oncotarget.23577. eCollection 2018 Jan 12. PMID: 29435157; PMCID: PMC5797028.
Copy Download .nbib Format: NLM
One way to rule them all: G2/M gene regulation by oncogenic YAP1 and B-MYB.

Molecular & cellular oncology

Weinstock G, Gaubatz S.
PMID: 31692816
Mol Cell Oncol. 2019 Aug 09;6(6):e1648026. doi: 10.1080/23723556.2019.1648026. eCollection 2019.

The Hippo pathway plays a major role in regulating organ size during embryogenesis. Loss of Hippo signaling can cause cancer due to unrestricted cell proliferation. Recently we found that Yes-associated protein 1 (YAP1), the major downstream effector of Hippo,...

Cite
Weinstock G, Gaubatz S. One way to rule them all: G2/M gene regulation by oncogenic YAP1 and B-MYB. Mol Cell Oncol. 2019;6(6):e1648026doi: 10.1080/23723556.2019.1648026.
Weinstock, G., & Gaubatz, S. (2019). One way to rule them all: G2/M gene regulation by oncogenic YAP1 and B-MYB. Molecular & cellular oncology, 6(6), e1648026. https://doi.org/10.1080/23723556.2019.1648026
Weinstock, Grit, and Gaubatz, Stefan. "One way to rule them all: G2/M gene regulation by oncogenic YAP1 and B-MYB." Molecular & cellular oncology vol. 6,6 (2019): e1648026. doi: https://doi.org/10.1080/23723556.2019.1648026
Weinstock G, Gaubatz S. One way to rule them all: G2/M gene regulation by oncogenic YAP1 and B-MYB. Mol Cell Oncol. 2019 Aug 09;6(6):e1648026. doi: 10.1080/23723556.2019.1648026. eCollection 2019. PMID: 31692816; PMCID: PMC6816428.
Copy Download .nbib Format: NLM
Cytosolic enzymes from rat tissues that activate the cooked meat mutagen metabolite N-Hydroxyamino-1-methyl-6- phenylimidazo[4,5-b]pyridine (N-OH-PhIP).

The Journal of nutritional biochemistry

Dubuisson JG, Murph WS, Griffin SR, Gaubatz JW.
PMID: 11834212
J Nutr Biochem. 2001 Sep;12(9):518-528. doi: 10.1016/s0955-2863(01)00168-1.

Heterocyclic amines are formed during the cooking of foods rich in protein and can be metabolically converted into cytotoxic and mutagenic compounds. These "cooked-food mutagens" constitute a potential health hazard because DNA damage arising from dietary exposure to heterocyclic...

Cite
Dubuisson JG, Murph WS, Griffin SR, et al. Cytosolic enzymes from rat tissues that activate the cooked meat mutagen metabolite N-Hydroxyamino-1-methyl-6- phenylimidazo[4,5-b]pyridine (N-OH-PhIP). J Nutr Biochem. 2001;12(9):518-528doi: 10.1016/s0955-2863(01)00168-1.
Dubuisson, J. G., Murph, W. S., Griffin, S. R., & Gaubatz, J. W. (2001). Cytosolic enzymes from rat tissues that activate the cooked meat mutagen metabolite N-Hydroxyamino-1-methyl-6- phenylimidazo[4,5-b]pyridine (N-OH-PhIP). The Journal of nutritional biochemistry, 12(9), 518-528. https://doi.org/10.1016/s0955-2863(01)00168-1
Dubuisson, J G., et al. "Cytosolic enzymes from rat tissues that activate the cooked meat mutagen metabolite N-Hydroxyamino-1-methyl-6- phenylimidazo[4,5-b]pyridine (N-OH-PhIP)." The Journal of nutritional biochemistry vol. 12,9 (2001): 518-528. doi: https://doi.org/10.1016/s0955-2863(01)00168-1
Dubuisson JG, Murph WS, Griffin SR, Gaubatz JW. Cytosolic enzymes from rat tissues that activate the cooked meat mutagen metabolite N-Hydroxyamino-1-methyl-6- phenylimidazo[4,5-b]pyridine (N-OH-PhIP). J Nutr Biochem. 2001 Sep;12(9):518-528. doi: 10.1016/s0955-2863(01)00168-1. PMID: 11834212.
Copy Download .nbib Format: NLM
Enforcement of entailment constraints in distributed service-based business processes.

Information and software technology

Hummer W, Gaubatz P, Strembeck M, Zdun U, Dustdar S.
PMID: 27667903
Inf Softw Technol. 2013 Nov;55(11):1884-1903. doi: 10.1016/j.infsof.2013.05.001.

CONTEXT: A distributed business process is executed in a distributed computing environment. The service-oriented architecture (SOA) paradigm is a popular option for the integration of software services and execution of distributed business processes. Entailment constraints, such as mutual exclusion...

Cite
Hummer W, Gaubatz P, Strembeck M, et al. Enforcement of entailment constraints in distributed service-based business processes. Inf Softw Technol. 2013;55(11):1884-1903doi: 10.1016/j.infsof.2013.05.001.
Hummer, W., Gaubatz, P., Strembeck, M., Zdun, U., & Dustdar, S. (2013). Enforcement of entailment constraints in distributed service-based business processes. Information and software technology, 55(11), 1884-1903. https://doi.org/10.1016/j.infsof.2013.05.001
Hummer, Waldemar, et al. "Enforcement of entailment constraints in distributed service-based business processes." Information and software technology vol. 55,11 (2013): 1884-1903. doi: https://doi.org/10.1016/j.infsof.2013.05.001
Hummer W, Gaubatz P, Strembeck M, Zdun U, Dustdar S. Enforcement of entailment constraints in distributed service-based business processes. Inf Softw Technol. 2013 Nov;55(11):1884-1903. doi: 10.1016/j.infsof.2013.05.001. PMID: 27667903; PMCID: PMC4461153.
Copy Download .nbib Format: NLM
The Influence of an Obesogenic Diet on Oxysterol Metabolism in C57BL/6J Mice.

Cholesterol

Wooten JS, Wu H, Raya J, Perrard XD, Gaubatz J, Hoogeveen RC.
PMID: 24672716
Cholesterol. 2014;2014:843468. doi: 10.1155/2014/843468. Epub 2014 Feb 05.

Our current understanding of oxysterol metabolism during different disease states such as obesity and dyslipidemia is limited. Therefore, the aim of this study was to determine the effect of diet-induced obesity on the tissue distribution of various oxysterols and...

Cite
Wooten JS, Wu H, Raya J, et al. The Influence of an Obesogenic Diet on Oxysterol Metabolism in C57BL/6J Mice. Cholesterol. 2014;2014:843468doi: 10.1155/2014/843468.
Wooten, J. S., Wu, H., Raya, J., Perrard, X. D., Gaubatz, J., & Hoogeveen, R. C. (2014). The Influence of an Obesogenic Diet on Oxysterol Metabolism in C57BL/6J Mice. Cholesterol, 2014843468. https://doi.org/10.1155/2014/843468
Wooten, Joshua S, et al. "The Influence of an Obesogenic Diet on Oxysterol Metabolism in C57BL/6J Mice." Cholesterol vol. 2014 (2014): 843468. doi: https://doi.org/10.1155/2014/843468
Wooten JS, Wu H, Raya J, Perrard XD, Gaubatz J, Hoogeveen RC. The Influence of an Obesogenic Diet on Oxysterol Metabolism in C57BL/6J Mice. Cholesterol. 2014;2014:843468. doi: 10.1155/2014/843468. Epub 2014 Feb 05. PMID: 24672716; PMCID: PMC3941159.
Copy Download .nbib Format: NLM
Contralesional White Matter Alterations in Patients After Hemispherotomy.

Frontiers in human neuroscience

Gaubatz J, Prillwitz CC, Ernst L, David B, Hoppe C, Hattingen E, Weber B, Vatter H, Surges R, Elger CE, Rüber T.
PMID: 32733222
Front Hum Neurosci. 2020 Jul 07;14:262. doi: 10.3389/fnhum.2020.00262. eCollection 2020.

Cerebral lesions may cause degeneration and neuroplastic reorganization in both the ipsi- and the contralesional hemisphere, presumably creating an imbalance of primarily inhibitory interhemispheric influences produced

Cite
Gaubatz J, Prillwitz CC, Ernst L, et al. Contralesional White Matter Alterations in Patients After Hemispherotomy. Front Hum Neurosci. 2020;14:262doi: 10.3389/fnhum.2020.00262.
Gaubatz, J., Prillwitz, C. C., Ernst, L., David, B., Hoppe, C., Hattingen, E., Weber, B., Vatter, H., Surges, R., Elger, C. E., & Rüber, T. (2020). Contralesional White Matter Alterations in Patients After Hemispherotomy. Frontiers in human neuroscience, 14262. https://doi.org/10.3389/fnhum.2020.00262
Gaubatz, Jennifer, et al. "Contralesional White Matter Alterations in Patients After Hemispherotomy." Frontiers in human neuroscience vol. 14 (2020): 262. doi: https://doi.org/10.3389/fnhum.2020.00262
Gaubatz J, Prillwitz CC, Ernst L, David B, Hoppe C, Hattingen E, Weber B, Vatter H, Surges R, Elger CE, Rüber T. Contralesional White Matter Alterations in Patients After Hemispherotomy. Front Hum Neurosci. 2020 Jul 07;14:262. doi: 10.3389/fnhum.2020.00262. eCollection 2020. PMID: 32733222; PMCID: PMC7358777.
Copy Download .nbib Format: NLM
Adiabatic population transfer in a three-level system driven by delayed laser pulses.

Physical review. A, General physics

Bergmann K, Gaubatz U, Hioe FT, Kuklinski JR.
PMID: 9902079
Phys Rev A Gen Phys. 1989 Dec 01;40(11):6741-6744. doi: 10.1103/physreva.40.6741.

No abstract available.

Cite
Bergmann K, Gaubatz U, Hioe FT, et al. Adiabatic population transfer in a three-level system driven by delayed laser pulses. Phys Rev A Gen Phys. 1989;40(11):6741-6744doi: 10.1103/physreva.40.6741.
Bergmann, K., Gaubatz, U., Hioe, F. T., & Kuklinski, J. R. (1989). Adiabatic population transfer in a three-level system driven by delayed laser pulses. Physical review. A, General physics, 40(11), 6741-6744. https://doi.org/10.1103/physreva.40.6741
Bergmann, et al. "Adiabatic population transfer in a three-level system driven by delayed laser pulses." Physical review. A, General physics vol. 40,11 (1989): 6741-6744. doi: https://doi.org/10.1103/physreva.40.6741
Bergmann K, Gaubatz U, Hioe FT, Kuklinski JR. Adiabatic population transfer in a three-level system driven by delayed laser pulses. Phys Rev A Gen Phys. 1989 Dec 01;40(11):6741-6744. doi: 10.1103/physreva.40.6741. PMID: 9902079.
Copy Download .nbib Format: NLM
IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability.

Molecular cancer research : MCR

Leone M, Cazorla-Vázquez S, Ferrazzi F, Wiederstein JL, Gründl M, Weinstock G, Vergarajauregui S, Eckstein M, Krüger M, Gaubatz S, Engel FB.
PMID: 34183451
Mol Cancer Res. 2021 Oct;19(10):1712-1726. doi: 10.1158/1541-7786.MCR-20-0639. Epub 2021 Jun 28.

Controlling cell proliferation is critical for organism development, tissue homeostasis, disease, and regeneration. IQGAP3 has been shown to be required for proper cell proliferation and migration, and is associated to a number of cancers. Moreover, its expression is inversely...

Cite
Leone M, Cazorla-Vázquez S, Ferrazzi F, et al. IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability. Mol Cancer Res. 2021;19(10):1712-1726doi: 10.1158/1541-7786.MCR-20-0639.
Leone, M., Cazorla-Vázquez, S., Ferrazzi, F., Wiederstein, J. L., Gründl, M., Weinstock, G., Vergarajauregui, S., Eckstein, M., Krüger, M., Gaubatz, S., & Engel, F. B. (2021). IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability. Molecular cancer research : MCR, 19(10), 1712-1726. https://doi.org/10.1158/1541-7786.MCR-20-0639
Leone, Marina, et al. "IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability." Molecular cancer research : MCR vol. 19,10 (2021): 1712-1726. doi: https://doi.org/10.1158/1541-7786.MCR-20-0639
Leone M, Cazorla-Vázquez S, Ferrazzi F, Wiederstein JL, Gründl M, Weinstock G, Vergarajauregui S, Eckstein M, Krüger M, Gaubatz S, Engel FB. IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability. Mol Cancer Res. 2021 Oct;19(10):1712-1726. doi: 10.1158/1541-7786.MCR-20-0639. Epub 2021 Jun 28. PMID: 34183451.
Copy Download .nbib Format: NLM
Showing 1 to 8 of 8 entries