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Showing 1 to 12 of 1754 entries
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[Observations on kidney diseases].

Kangogaku zasshi

Oshio E, Kawano K, Yasuoka F, Ushimado M, Kawano T.
PMID: 4974127
Kangogaku Zasshi. 1968 Dec;32(13):92-6.

No abstract available.

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Oshio E, Kawano K, Yasuoka F, et al. [Observations on kidney diseases]. Kangogaku Zasshi. 1968;32(13):92-6
Oshio, E., Kawano, K., Yasuoka, F., Ushimado, M., & Kawano, T. (1968). [Observations on kidney diseases]. Kangogaku zasshi, 32(13), 92-6.
Oshio, E, et al. "[Observations on kidney diseases]." Kangogaku zasshi vol. 32,13 (1968): 92-6.
Oshio E, Kawano K, Yasuoka F, Ushimado M, Kawano T. [Observations on kidney diseases]. Kangogaku Zasshi. 1968 Dec;32(13):92-6. Japanese. PMID: 4974127.
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[Orientation at the time of hospital admission. 2. Orientation of patients to ease their anxiety].

Kangogaku zasshi

Kamimura M, Ishinabe K, Katada M, Kinoshita Y, Kuwano T.
PMID: 4995178
Kangogaku Zasshi. 1971 Apr;35(4):15-8.

No abstract available.

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Kamimura M, Ishinabe K, Katada M, et al. [Orientation at the time of hospital admission. 2. Orientation of patients to ease their anxiety]. Kangogaku Zasshi. 1971;35(4):15-8
Kamimura, M., Ishinabe, K., Katada, M., Kinoshita, Y., & Kuwano, T. (1971). [Orientation at the time of hospital admission. 2. Orientation of patients to ease their anxiety]. Kangogaku zasshi, 35(4), 15-8.
Kamimura, M, et al. "[Orientation at the time of hospital admission. 2. Orientation of patients to ease their anxiety]." Kangogaku zasshi vol. 35,4 (1971): 15-8.
Kamimura M, Ishinabe K, Katada M, Kinoshita Y, Kuwano T. [Orientation at the time of hospital admission. 2. Orientation of patients to ease their anxiety]. Kangogaku Zasshi. 1971 Apr;35(4):15-8. Japanese. PMID: 4995178.
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[Sex and clinical medicine. 6. Therapeutic relationship and sex].

Kangogaku zasshi

Kawano T.
PMID: 6772859
Kangogaku Zasshi. 1980 Jul;44(7):757-60.

No abstract available.

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Kawano T. [Sex and clinical medicine. 6. Therapeutic relationship and sex]. Kangogaku Zasshi. 1980;44(7):757-60
Kawano, T. (1980). [Sex and clinical medicine. 6. Therapeutic relationship and sex]. Kangogaku zasshi, 44(7), 757-60.
Kawano, T. "[Sex and clinical medicine. 6. Therapeutic relationship and sex]." Kangogaku zasshi vol. 44,7 (1980): 757-60.
Kawano T. [Sex and clinical medicine. 6. Therapeutic relationship and sex]. Kangogaku Zasshi. 1980 Jul;44(7):757-60. Japanese. PMID: 6772859.
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Colon Mucosa Exhibits Loss of Ectopic MUC5AC Expression in Patients with Ulcerative Colitis Treated with Oral Tacrolimus.

ISRN gastroenterology

Mizoshita T, Tanida S, Tsukamoto H, Ozeki K, Katano T, Ebi M, Mori Y, Kataoka H, Kamiya T, Joh T.
PMID: 23691335
ISRN Gastroenterol. 2013 Apr 09;2013:304894. doi: 10.1155/2013/304894. Print 2013.

Background. Tacrolimus (FK506) is effective for patients with ulcerative colitis (UC). However, there are few reports on tacrolimus therapy (TT) with respect to the relationship with endoscopic and clinicopathologic findings. Methods. Thirty patients with moderate/severe active UC refractory to...

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Mizoshita T, Tanida S, Tsukamoto H, et al. Colon Mucosa Exhibits Loss of Ectopic MUC5AC Expression in Patients with Ulcerative Colitis Treated with Oral Tacrolimus. ISRN Gastroenterol. 2013;2013:304894doi: 10.1155/2013/304894.
Mizoshita, T., Tanida, S., Tsukamoto, H., Ozeki, K., Katano, T., Ebi, M., Mori, Y., Kataoka, H., Kamiya, T., & Joh, T. (2013). Colon Mucosa Exhibits Loss of Ectopic MUC5AC Expression in Patients with Ulcerative Colitis Treated with Oral Tacrolimus. ISRN gastroenterology, 2013304894. https://doi.org/10.1155/2013/304894
Mizoshita, Tsutomu, et al. "Colon Mucosa Exhibits Loss of Ectopic MUC5AC Expression in Patients with Ulcerative Colitis Treated with Oral Tacrolimus." ISRN gastroenterology vol. 2013 (2013): 304894. doi: https://doi.org/10.1155/2013/304894
Mizoshita T, Tanida S, Tsukamoto H, Ozeki K, Katano T, Ebi M, Mori Y, Kataoka H, Kamiya T, Joh T. Colon Mucosa Exhibits Loss of Ectopic MUC5AC Expression in Patients with Ulcerative Colitis Treated with Oral Tacrolimus. ISRN Gastroenterol. 2013 Apr 09;2013:304894. doi: 10.1155/2013/304894. Print 2013. PMID: 23691335; PMCID: PMC3649514.
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Type VII and XVII Collagen mRNA Expressions in Regenerated Epidermal Laminae in Chronic Equine Laminitis.

Journal of equine science

Kuwano A, Hasegawa T, Arai K.
PMID: 24833961
J Equine Sci. 2008;19(4):103-7. doi: 10.1294/jes.19.103. Epub 2009 Jan 09.

To confirm ability forming the basement membrane of the regenerated laminar epidermis (rLE) in chronic laminitis, expression of type VII and type XVII collagen mRNAs in the rLE was studied applying sequences of two type of murine collagens. On...

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Kuwano A, Hasegawa T, Arai K. Type VII and XVII Collagen mRNA Expressions in Regenerated Epidermal Laminae in Chronic Equine Laminitis. J Equine Sci. 2009;19(4):103-7doi: 10.1294/jes.19.103.
Kuwano, A., Hasegawa, T., & Arai, K. (2008). Type VII and XVII Collagen mRNA Expressions in Regenerated Epidermal Laminae in Chronic Equine Laminitis. Journal of equine science, 19(4), 103-7. https://doi.org/10.1294/jes.19.103
Kuwano, Atsutoshi, et al. "Type VII and XVII Collagen mRNA Expressions in Regenerated Epidermal Laminae in Chronic Equine Laminitis." Journal of equine science vol. 19,4 (2008): 103-7. doi: https://doi.org/10.1294/jes.19.103
Kuwano A, Hasegawa T, Arai K. Type VII and XVII Collagen mRNA Expressions in Regenerated Epidermal Laminae in Chronic Equine Laminitis. J Equine Sci. 2008;19(4):103-7. doi: 10.1294/jes.19.103. Epub 2009 Jan 09. PMID: 24833961; PMCID: PMC4013949.
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Transcatheter embolization for hemoptysis associated with anomalous systemic artery in a patient with scimitar syndrome.

SpringerPlus

Yamakawa H, Shimizu K, Michimoto K, Kameoka Y, Kang R, Yoshida J, Yamada M, Yoshida M, Ishikawa T, Takagi M, Kuwano K.
PMID: 26301169
Springerplus. 2015 Aug 14;4:422. doi: 10.1186/s40064-015-1219-9. eCollection 2015.

BACKGROUND: Scimitar syndrome can present with a wide clinical spectrum of symptoms either early in the neonatal period or later in life.CASE DESCRIPTION: We report a case of a 62-year-old woman with anomalous systemic arterial supply to the basal...

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Yamakawa H, Shimizu K, Michimoto K, et al. Transcatheter embolization for hemoptysis associated with anomalous systemic artery in a patient with scimitar syndrome. Springerplus. 2015;4:422doi: 10.1186/s40064-015-1219-9.
Yamakawa, H., Shimizu, K., Michimoto, K., Kameoka, Y., Kang, R., Yoshida, J., Yamada, M., Yoshida, M., Ishikawa, T., Takagi, M., & Kuwano, K. (2015). Transcatheter embolization for hemoptysis associated with anomalous systemic artery in a patient with scimitar syndrome. SpringerPlus, 4422. https://doi.org/10.1186/s40064-015-1219-9
Yamakawa, Hideaki, et al. "Transcatheter embolization for hemoptysis associated with anomalous systemic artery in a patient with scimitar syndrome." SpringerPlus vol. 4 (2015): 422. doi: https://doi.org/10.1186/s40064-015-1219-9
Yamakawa H, Shimizu K, Michimoto K, Kameoka Y, Kang R, Yoshida J, Yamada M, Yoshida M, Ishikawa T, Takagi M, Kuwano K. Transcatheter embolization for hemoptysis associated with anomalous systemic artery in a patient with scimitar syndrome. Springerplus. 2015 Aug 14;4:422. doi: 10.1186/s40064-015-1219-9. eCollection 2015. PMID: 26301169; PMCID: PMC4536247.
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Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion.

Biology open

Kawano S, Torisu T, Esaki M, Torisu K, Matsuno Y, Kitazono T.
PMID: 28970230
Biol Open. 2017 Nov 15;6(11):1644-1653. doi: 10.1242/bio.027458.

Adhesion of cells to the extracellular matrix (ECM) via focal adhesions (FAs) is crucial for cell survival, migration, and differentiation. Although the regulation of FAs, including by integrins and the ECM, is important to cell behavior, how FAs are...

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Kawano S, Torisu T, Esaki M, et al. Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion. Biol Open. 2017;6(11):1644-1653doi: 10.1242/bio.027458.
Kawano, S., Torisu, T., Esaki, M., Torisu, K., Matsuno, Y., & Kitazono, T. (2017). Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion. Biology open, 6(11), 1644-1653. https://doi.org/10.1242/bio.027458
Kawano, Shinichi, et al. "Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion." Biology open vol. 6,11 (2017): 1644-1653. doi: https://doi.org/10.1242/bio.027458
Kawano S, Torisu T, Esaki M, Torisu K, Matsuno Y, Kitazono T. Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion. Biol Open. 2017 Nov 15;6(11):1644-1653. doi: 10.1242/bio.027458. PMID: 28970230; PMCID: PMC5703610.
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Tailoring particle translocation via dielectrophoresis in pore channels.

Scientific reports

Tanaka S, Tsutsui M, Theodore H, Yuhui H, Arima A, Tsuji T, Doi K, Kawano S, Taniguchi M, Kawai T.
PMID: 27527126
Sci Rep. 2016 Aug 16;6:31670. doi: 10.1038/srep31670.

Understanding and controlling electrophoretic motions of nanoscopic objects in fluidic channels are a central challenge in developing nanopore technology for molecular analyses. Although progress has been made in slowing the translocation velocity to meet the requirement for electrical detections...

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Tanaka S, Tsutsui M, Theodore H, et al. Tailoring particle translocation via dielectrophoresis in pore channels. Sci Rep. 2016;6:31670doi: 10.1038/srep31670.
Tanaka, S., Tsutsui, M., Theodore, H., Yuhui, H., Arima, A., Tsuji, T., Doi, K., Kawano, S., Taniguchi, M., & Kawai, T. (2016). Tailoring particle translocation via dielectrophoresis in pore channels. Scientific reports, 631670. https://doi.org/10.1038/srep31670
Tanaka, Shoji, et al. "Tailoring particle translocation via dielectrophoresis in pore channels." Scientific reports vol. 6 (2016): 31670. doi: https://doi.org/10.1038/srep31670
Tanaka S, Tsutsui M, Theodore H, Yuhui H, Arima A, Tsuji T, Doi K, Kawano S, Taniguchi M, Kawai T. Tailoring particle translocation via dielectrophoresis in pore channels. Sci Rep. 2016 Aug 16;6:31670. doi: 10.1038/srep31670. PMID: 27527126; PMCID: PMC4985646.
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Successful hepatectomy for hepatic abscess with chronic granulomatous disease: a case report.

Surgical case reports

Muranushi R, Suzuki M, Araki K, Kubo N, Otake S, Nishida Y, Ishige T, Arakawa H, Kuwano H, Shirabe K.
PMID: 28447322
Surg Case Rep. 2017 Dec;3(1):57. doi: 10.1186/s40792-017-0333-z. Epub 2017 Apr 26.

BACKGROUND: Chronic granulomatous disease (CGD), a rare inherited disorder, is characterized by impaired ability of phagocytic cells to kill certain bacteria and fungi. Although liver abscess is a common manifestation of CGD, its optimal management in these patients is...

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Muranushi R, Suzuki M, Araki K, et al. Successful hepatectomy for hepatic abscess with chronic granulomatous disease: a case report. Surg Case Rep. 2017;3(1):57doi: 10.1186/s40792-017-0333-z.
Muranushi, R., Suzuki, M., Araki, K., Kubo, N., Otake, S., Nishida, Y., Ishige, T., Arakawa, H., Kuwano, H., & Shirabe, K. (2017). Successful hepatectomy for hepatic abscess with chronic granulomatous disease: a case report. Surgical case reports, 3(1), 57. https://doi.org/10.1186/s40792-017-0333-z
Muranushi, Ryo, et al. "Successful hepatectomy for hepatic abscess with chronic granulomatous disease: a case report." Surgical case reports vol. 3,1 (2017): 57. doi: https://doi.org/10.1186/s40792-017-0333-z
Muranushi R, Suzuki M, Araki K, Kubo N, Otake S, Nishida Y, Ishige T, Arakawa H, Kuwano H, Shirabe K. Successful hepatectomy for hepatic abscess with chronic granulomatous disease: a case report. Surg Case Rep. 2017 Dec;3(1):57. doi: 10.1186/s40792-017-0333-z. Epub 2017 Apr 26. PMID: 28447322; PMCID: PMC5406309.
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Reducing Cytoplasmic Polyamine Oxidase Activity in Arabidopsis Increases Salt and Drought Tolerance by Reducing Reactive Oxygen Species Production and Increasing Defense Gene Expression.

Frontiers in plant science

Sagor GH, Zhang S, Kojima S, Simm S, Berberich T, Kusano T.
PMID: 26973665
Front Plant Sci. 2016 Feb 29;7:214. doi: 10.3389/fpls.2016.00214. eCollection 2016.

The link between polyamine oxidases (PAOs), which function in polyamine catabolism, and stress responses remains elusive. Here, we address this issue using Arabidopsis pao mutants in which the expression of the five PAO genes is knocked-out or knocked-down. As...

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Sagor GH, Zhang S, Kojima S, et al. Reducing Cytoplasmic Polyamine Oxidase Activity in Arabidopsis Increases Salt and Drought Tolerance by Reducing Reactive Oxygen Species Production and Increasing Defense Gene Expression. Front Plant Sci. 2016;7:214doi: 10.3389/fpls.2016.00214.
Sagor, G. H., Zhang, S., Kojima, S., Simm, S., Berberich, T., & Kusano, T. (2016). Reducing Cytoplasmic Polyamine Oxidase Activity in Arabidopsis Increases Salt and Drought Tolerance by Reducing Reactive Oxygen Species Production and Increasing Defense Gene Expression. Frontiers in plant science, 7214. https://doi.org/10.3389/fpls.2016.00214
Sagor, G H M, et al. "Reducing Cytoplasmic Polyamine Oxidase Activity in Arabidopsis Increases Salt and Drought Tolerance by Reducing Reactive Oxygen Species Production and Increasing Defense Gene Expression." Frontiers in plant science vol. 7 (2016): 214. doi: https://doi.org/10.3389/fpls.2016.00214
Sagor GH, Zhang S, Kojima S, Simm S, Berberich T, Kusano T. Reducing Cytoplasmic Polyamine Oxidase Activity in Arabidopsis Increases Salt and Drought Tolerance by Reducing Reactive Oxygen Species Production and Increasing Defense Gene Expression. Front Plant Sci. 2016 Feb 29;7:214. doi: 10.3389/fpls.2016.00214. eCollection 2016. PMID: 26973665; PMCID: PMC4770033.
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Unexpectedly long intravenous and intracardiac extension of a small-sized pulmonary pleomorphic carcinoma.

Journal of thoracic disease

Ishii A, Kimura T, Moritani K, Cui D, Tanaka S, Kawano H, Ikeda E.
PMID: 25590004
J Thorac Dis. 2014 Dec;6(12):E259-63. doi: 10.3978/j.issn.2072-1439.2014.10.25.

Pleomorphic carcinoma of the lung is one of the uncommon histological types of lung cancers, which shows an aggressive behavior. Intravenous extension (not metastasis or direct invasion) of the tumor into the heart is a rare complication of lung...

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Ishii A, Kimura T, Moritani K, et al. Unexpectedly long intravenous and intracardiac extension of a small-sized pulmonary pleomorphic carcinoma. J Thorac Dis. 2014;6(12):E259-63doi: 10.3978/j.issn.2072-1439.2014.10.25.
Ishii, A., Kimura, T., Moritani, K., Cui, D., Tanaka, S., Kawano, H., & Ikeda, E. (2014). Unexpectedly long intravenous and intracardiac extension of a small-sized pulmonary pleomorphic carcinoma. Journal of thoracic disease, 6(12), E259-63. https://doi.org/10.3978/j.issn.2072-1439.2014.10.25
Ishii, Aya, et al. "Unexpectedly long intravenous and intracardiac extension of a small-sized pulmonary pleomorphic carcinoma." Journal of thoracic disease vol. 6,12 (2014): E259-63. doi: https://doi.org/10.3978/j.issn.2072-1439.2014.10.25
Ishii A, Kimura T, Moritani K, Cui D, Tanaka S, Kawano H, Ikeda E. Unexpectedly long intravenous and intracardiac extension of a small-sized pulmonary pleomorphic carcinoma. J Thorac Dis. 2014 Dec;6(12):E259-63. doi: 10.3978/j.issn.2072-1439.2014.10.25. PMID: 25590004; PMCID: PMC4283330.
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Angiotensin II suppresses osteoblastic differentiation and mineralized nodule formation via AT1 receptor in ROS17/2.8 cells.

Archives of medical science : AMS

Nakai K, Kawato T, Morita T, Yamazaki Y, Tanaka H, Tonogi M, Oki H, Maeno M.
PMID: 26170858
Arch Med Sci. 2015 Jun 19;11(3):628-37. doi: 10.5114/aoms.2015.52369.

INTRODUCTION: Angiotensin II (Ang II) not only regulates systemic blood pressure through a vasoconstrictive effect, but also promotes bone resorption. We recently reported that Ang II (10(-6) M) stimulated the production of matrix metalloproteinases via the AT1 receptor in...

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Nakai K, Kawato T, Morita T, et al. Angiotensin II suppresses osteoblastic differentiation and mineralized nodule formation via AT1 receptor in ROS17/2.8 cells. Arch Med Sci. 2015;11(3):628-37doi: 10.5114/aoms.2015.52369.
Nakai, K., Kawato, T., Morita, T., Yamazaki, Y., Tanaka, H., Tonogi, M., Oki, H., & Maeno, M. (2015). Angiotensin II suppresses osteoblastic differentiation and mineralized nodule formation via AT1 receptor in ROS17/2.8 cells. Archives of medical science : AMS, 11(3), 628-37. https://doi.org/10.5114/aoms.2015.52369
Nakai, Kumiko, et al. "Angiotensin II suppresses osteoblastic differentiation and mineralized nodule formation via AT1 receptor in ROS17/2.8 cells." Archives of medical science : AMS vol. 11,3 (2015): 628-37. doi: https://doi.org/10.5114/aoms.2015.52369
Nakai K, Kawato T, Morita T, Yamazaki Y, Tanaka H, Tonogi M, Oki H, Maeno M. Angiotensin II suppresses osteoblastic differentiation and mineralized nodule formation via AT1 receptor in ROS17/2.8 cells. Arch Med Sci. 2015 Jun 19;11(3):628-37. doi: 10.5114/aoms.2015.52369. PMID: 26170858; PMCID: PMC4495158.
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Showing 1 to 12 of 1754 entries