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Host response to fungal infections - how immunology and host genetics could help to identify and treat patients at risk.

Swiss medical weekly

Khanna N, Stuehler C, Lünemann A, Wójtowicz A, Bochud PY, Leibundgut-Landmann S.
PMID: 27655228
Swiss Med Wkly. 2016 Sep 21;146:w14350. doi: 10.4414/smw.2016.14350. eCollection 2016.

In spite of the ever-increasing incidence and poor outcome of invasive fungal infections in immune compromised patients, there is currently no reliable method to accurately predict the risk, to monitor the outcome and to treat these infections. Protective immunity...

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Khanna N, Stuehler C, Lünemann A, et al. Host response to fungal infections - how immunology and host genetics could help to identify and treat patients at risk. Swiss Med Wkly. 2016;146:w14350doi: 10.4414/smw.2016.14350.
Khanna, N., Stuehler, C., Lünemann, A., Wójtowicz, A., Bochud, P. Y., & Leibundgut-Landmann, S. (2016). Host response to fungal infections - how immunology and host genetics could help to identify and treat patients at risk. Swiss medical weekly, 146w14350. https://doi.org/10.4414/smw.2016.14350
Khanna, Nina, et al. "Host response to fungal infections - how immunology and host genetics could help to identify and treat patients at risk." Swiss medical weekly vol. 146 (2016): w14350. doi: https://doi.org/10.4414/smw.2016.14350
Khanna N, Stuehler C, Lünemann A, Wójtowicz A, Bochud PY, Leibundgut-Landmann S. Host response to fungal infections - how immunology and host genetics could help to identify and treat patients at risk. Swiss Med Wkly. 2016 Sep 21;146:w14350. doi: 10.4414/smw.2016.14350. eCollection 2016. PMID: 27655228.
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Mutanobactin D from the Human Microbiome: Total Synthesis, Configurational Assignment, and Biological Evaluation.

Journal of the American Chemical Society

Pultar F, Hansen ME, Wolfrum S, Böselt L, Fróis-Martins R, Bloch S, Kravina AG, Pehlivanoglu D, Schäffer C, LeibundGut-Landmann S, Riniker S, Carreira EM.
PMID: 34212720
J Am Chem Soc. 2021 Jul 14;143(27):10389-10402. doi: 10.1021/jacs.1c04825. Epub 2021 Jul 02.

Mutanobactin D is a non-ribosomal, cyclic peptide isolated from

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Pultar F, Hansen ME, Wolfrum S, et al. Mutanobactin D from the Human Microbiome: Total Synthesis, Configurational Assignment, and Biological Evaluation. J Am Chem Soc. 2021;143(27):10389-10402doi: 10.1021/jacs.1c04825.
Pultar, F., Hansen, M. E., Wolfrum, S., Böselt, L., Fróis-Martins, R., Bloch, S., Kravina, A. G., Pehlivanoglu, D., Schäffer, C., LeibundGut-Landmann, S., Riniker, S., & Carreira, E. M. (2021). Mutanobactin D from the Human Microbiome: Total Synthesis, Configurational Assignment, and Biological Evaluation. Journal of the American Chemical Society, 143(27), 10389-10402. https://doi.org/10.1021/jacs.1c04825
Pultar, Felix, et al. "Mutanobactin D from the Human Microbiome: Total Synthesis, Configurational Assignment, and Biological Evaluation." Journal of the American Chemical Society vol. 143,27 (2021): 10389-10402. doi: https://doi.org/10.1021/jacs.1c04825
Pultar F, Hansen ME, Wolfrum S, Böselt L, Fróis-Martins R, Bloch S, Kravina AG, Pehlivanoglu D, Schäffer C, LeibundGut-Landmann S, Riniker S, Carreira EM. Mutanobactin D from the Human Microbiome: Total Synthesis, Configurational Assignment, and Biological Evaluation. J Am Chem Soc. 2021 Jul 14;143(27):10389-10402. doi: 10.1021/jacs.1c04825. Epub 2021 Jul 02. PMID: 34212720.
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IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation.

Science immunology

Zwicky P, Ingelfinger F, Silva de Melo BM, Ruchti F, Schärli S, Puertas N, Lutz M, Phan TS, Kündig TM, Levesque MP, Maul JT, Schlapbach C, LeibundGut-Landmann S, Mundt S, Becher B.
PMID: 34678045
Sci Immunol. 2021 Oct 22;6(64):eabg9012. doi: 10.1126/sciimmunol.abg9012. Epub 2021 Oct 22.

[Figure: see text].

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Zwicky P, Ingelfinger F, Silva de Melo BM, et al. IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation. Sci Immunol. 2021;6(64):eabg9012doi: 10.1126/sciimmunol.abg9012.
Zwicky, P., Ingelfinger, F., Silva de Melo, B. M., Ruchti, F., Schärli, S., Puertas, N., Lutz, M., Phan, T. S., Kündig, T. M., Levesque, M. P., Maul, J. T., Schlapbach, C., LeibundGut-Landmann, S., Mundt, S., & Becher, B. (2021). IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation. Science immunology, 6(64), eabg9012. https://doi.org/10.1126/sciimmunol.abg9012
Zwicky, Pascale, et al. "IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation." Science immunology vol. 6,64 (2021): eabg9012. doi: https://doi.org/10.1126/sciimmunol.abg9012
Zwicky P, Ingelfinger F, Silva de Melo BM, Ruchti F, Schärli S, Puertas N, Lutz M, Phan TS, Kündig TM, Levesque MP, Maul JT, Schlapbach C, LeibundGut-Landmann S, Mundt S, Becher B. IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation. Sci Immunol. 2021 Oct 22;6(64):eabg9012. doi: 10.1126/sciimmunol.abg9012. Epub 2021 Oct 22. PMID: 34678045.
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Interleukin 17-Mediated Host Defense against Candida albicans.

Pathogens (Basel, Switzerland)

Sparber F, LeibundGut-Landmann S.
PMID: 26274976
Pathogens. 2015 Aug 12;4(3):606-19. doi: 10.3390/pathogens4030606.

Candida albicans is part of the normal microbiota in most healthy individuals. However, it can cause opportunistic infections if host defenses are breached, with symptoms ranging from superficial lesions to severe systemic disease. The study of rare congenital defects...

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Sparber F, LeibundGut-Landmann S. Interleukin 17-Mediated Host Defense against Candida albicans. Pathogens. 2015;4(3):606-19doi: 10.3390/pathogens4030606.
Sparber, F., & LeibundGut-Landmann, S. (2015). Interleukin 17-Mediated Host Defense against Candida albicans. Pathogens (Basel, Switzerland), 4(3), 606-19. https://doi.org/10.3390/pathogens4030606
Sparber, Florian, and LeibundGut-Landmann, Salomé. "Interleukin 17-Mediated Host Defense against Candida albicans." Pathogens (Basel, Switzerland) vol. 4,3 (2015): 606-19. doi: https://doi.org/10.3390/pathogens4030606
Sparber F, LeibundGut-Landmann S. Interleukin 17-Mediated Host Defense against Candida albicans. Pathogens. 2015 Aug 12;4(3):606-19. doi: 10.3390/pathogens4030606. PMID: 26274976; PMCID: PMC4584276.
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[No title available]

Frontiers in immunology

Guiducci E, Lemberg C, Küng N, Schraner E, Theocharides APA, LeibundGut-Landmann S.
PMID: 30038623
Front Immunol. 2018 Jul 09;9:1573. doi: 10.3389/fimmu.2018.01573. eCollection 2018.

Neutrophils are the most abundant innate immune cells and the first line of defense against many pathogenic microbes, including the human fungal pathogen

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Guiducci E, Lemberg C, Küng N, et al. . Front Immunol. 2018;9:1573doi: 10.3389/fimmu.2018.01573.
Guiducci, E., Lemberg, C., Küng, N., Schraner, E., Theocharides, A. P. A., & LeibundGut-Landmann, S. (2018). . Frontiers in immunology, 91573. https://doi.org/10.3389/fimmu.2018.01573
Guiducci, Eva, et al. "." Frontiers in immunology vol. 9 (2018): 1573. doi: https://doi.org/10.3389/fimmu.2018.01573
Guiducci E, Lemberg C, Küng N, Schraner E, Theocharides APA, LeibundGut-Landmann S. . Front Immunol. 2018 Jul 09;9:1573. doi: 10.3389/fimmu.2018.01573. eCollection 2018. PMID: 30038623; PMCID: PMC6046457.
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Host Responses to .

Frontiers in immunology

Sparber F, LeibundGut-Landmann S.
PMID: 29213272
Front Immunol. 2017 Nov 22;8:1614. doi: 10.3389/fimmu.2017.01614. eCollection 2017.

The skin of mammalian organisms is home for a myriad of microbes. Many of these commensals are thought to have beneficial effects on the host by critically contributing to immune homeostasis. Consequently, dysbiosis can have detrimental effects for the...

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Sparber F, LeibundGut-Landmann S. Host Responses to . Front Immunol. 2017;8:1614doi: 10.3389/fimmu.2017.01614.
Sparber, F., & LeibundGut-Landmann, S. (2017). Host Responses to . Frontiers in immunology, 81614. https://doi.org/10.3389/fimmu.2017.01614
Sparber, Florian, and LeibundGut-Landmann, Salomé. "Host Responses to ." Frontiers in immunology vol. 8 (2017): 1614. doi: https://doi.org/10.3389/fimmu.2017.01614
Sparber F, LeibundGut-Landmann S. Host Responses to . Front Immunol. 2017 Nov 22;8:1614. doi: 10.3389/fimmu.2017.01614. eCollection 2017. PMID: 29213272; PMCID: PMC5702624.
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Common Features of Regulatory T Cell Specialization During Th1 Responses.

Frontiers in immunology

Littringer K, Moresi C, Rakebrandt N, Zhou X, Schorer M, Dolowschiak T, Kirchner F, Rost F, Keller CW, McHugh D, LeibundGut-Landmann S, Robinson MD, Joller N.
PMID: 29951069
Front Immunol. 2018 Jun 13;9:1344. doi: 10.3389/fimmu.2018.01344. eCollection 2018.

CD4

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Littringer K, Moresi C, Rakebrandt N, et al. Common Features of Regulatory T Cell Specialization During Th1 Responses. Front Immunol. 2018;9:1344doi: 10.3389/fimmu.2018.01344.
Littringer, K., Moresi, C., Rakebrandt, N., Zhou, X., Schorer, M., Dolowschiak, T., Kirchner, F., Rost, F., Keller, C. W., McHugh, D., LeibundGut-Landmann, S., Robinson, M. D., & Joller, N. (2018). Common Features of Regulatory T Cell Specialization During Th1 Responses. Frontiers in immunology, 91344. https://doi.org/10.3389/fimmu.2018.01344
Littringer, Katharina, et al. "Common Features of Regulatory T Cell Specialization During Th1 Responses." Frontiers in immunology vol. 9 (2018): 1344. doi: https://doi.org/10.3389/fimmu.2018.01344
Littringer K, Moresi C, Rakebrandt N, Zhou X, Schorer M, Dolowschiak T, Kirchner F, Rost F, Keller CW, McHugh D, LeibundGut-Landmann S, Robinson MD, Joller N. Common Features of Regulatory T Cell Specialization During Th1 Responses. Front Immunol. 2018 Jun 13;9:1344. doi: 10.3389/fimmu.2018.01344. eCollection 2018. PMID: 29951069; PMCID: PMC6008317.
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Correction: Syk Signaling in Dendritic Cells Orchestrates Innate Resistance to Systemic Fungal Infection.

PLoS pathogens

Whitney PG, Bär E, Osorio F, Rogers NC, Schraml BU, Deddouche S, LeibundGut-Landmann S, Reis e Sousa C.
PMID: 30462735
PLoS Pathog. 2018 Nov 21;14(11):e1007366. doi: 10.1371/journal.ppat.1007366. eCollection 2018 Nov.

[This corrects the article DOI: 10.1371/journal.ppat.1004276.].

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Whitney PG, Bär E, Osorio F, et al. Correction: Syk Signaling in Dendritic Cells Orchestrates Innate Resistance to Systemic Fungal Infection. PLoS Pathog. 2018;14(11):e1007366doi: 10.1371/journal.ppat.1007366.
Whitney, P. G., Bär, E., Osorio, F., Rogers, N. C., Schraml, B. U., Deddouche, S., LeibundGut-Landmann, S., & Reis e Sousa, C. (2018). Correction: Syk Signaling in Dendritic Cells Orchestrates Innate Resistance to Systemic Fungal Infection. PLoS pathogens, 14(11), e1007366. https://doi.org/10.1371/journal.ppat.1007366
Whitney, Paul G, et al. "Correction: Syk Signaling in Dendritic Cells Orchestrates Innate Resistance to Systemic Fungal Infection." PLoS pathogens vol. 14,11 (2018): e1007366. doi: https://doi.org/10.1371/journal.ppat.1007366
Whitney PG, Bär E, Osorio F, Rogers NC, Schraml BU, Deddouche S, LeibundGut-Landmann S, Reis e Sousa C. Correction: Syk Signaling in Dendritic Cells Orchestrates Innate Resistance to Systemic Fungal Infection. PLoS Pathog. 2018 Nov 21;14(11):e1007366. doi: 10.1371/journal.ppat.1007366. eCollection 2018 Nov. PMID: 30462735; PMCID: PMC6249011.
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Modulation of the Fungal-Host Interaction by the Intra-Species Diversity of C. albicans.

Pathogens (Basel, Switzerland)

Braunsdorf C, LeibundGut-Landmann S.
PMID: 29342100
Pathogens. 2018 Jan 17;7(1). doi: 10.3390/pathogens7010011.

The incidence of human infections caused by the opportunistic fungal pathogen

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Braunsdorf C, LeibundGut-Landmann S. Modulation of the Fungal-Host Interaction by the Intra-Species Diversity of C. albicans. Pathogens. 2018;7(1)doi: 10.3390/pathogens7010011.
Braunsdorf, C., & LeibundGut-Landmann, S. (2018). Modulation of the Fungal-Host Interaction by the Intra-Species Diversity of C. albicans. Pathogens (Basel, Switzerland), 7(1), . https://doi.org/10.3390/pathogens7010011
Braunsdorf, Christina, and LeibundGut-Landmann, Salomé. "Modulation of the Fungal-Host Interaction by the Intra-Species Diversity of C. albicans." Pathogens (Basel, Switzerland) vol. 7,1 (2018). doi: https://doi.org/10.3390/pathogens7010011
Braunsdorf C, LeibundGut-Landmann S. Modulation of the Fungal-Host Interaction by the Intra-Species Diversity of C. albicans. Pathogens. 2018 Jan 17;7(1). doi: 10.3390/pathogens7010011. PMID: 29342100; PMCID: PMC5874737.
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Tissue-Resident Memory T Cells in Antifungal Immunity.

Frontiers in immunology

LeibundGut-Landmann S.
PMID: 34113356
Front Immunol. 2021 May 25;12:693055. doi: 10.3389/fimmu.2021.693055. eCollection 2021.

Fungi are an integral part of the mammalian microbiota colonizing most if not all mucosal surfaces and the skin. Maintaining stable colonization on these surfaces is critical for preventing fungal dysbiosis and infection, which in some cases can lead...

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LeibundGut-Landmann S. Tissue-Resident Memory T Cells in Antifungal Immunity. Front Immunol. 2021;12:693055doi: 10.3389/fimmu.2021.693055.
LeibundGut-Landmann, S. (2021). Tissue-Resident Memory T Cells in Antifungal Immunity. Frontiers in immunology, 12693055. https://doi.org/10.3389/fimmu.2021.693055
LeibundGut-Landmann, Salomé. "Tissue-Resident Memory T Cells in Antifungal Immunity." Frontiers in immunology vol. 12 (2021): 693055. doi: https://doi.org/10.3389/fimmu.2021.693055
LeibundGut-Landmann S. Tissue-Resident Memory T Cells in Antifungal Immunity. Front Immunol. 2021 May 25;12:693055. doi: 10.3389/fimmu.2021.693055. eCollection 2021. PMID: 34113356; PMCID: PMC8185520.
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Interleukin-17 in Antifungal Immunity.

Pathogens (Basel, Switzerland)

Sparber F, LeibundGut-Landmann S.
PMID: 31013616
Pathogens. 2019 Apr 22;8(2). doi: 10.3390/pathogens8020054.

The field of IL-17 biology has received much attention over the last decade owing to the pathogenic role of this cytokine in psoriasis and other autoinflammatory disorders and the successful implementation of IL-17-targeting therapies in patients suffering from these...

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Sparber F, LeibundGut-Landmann S. Interleukin-17 in Antifungal Immunity. Pathogens. 2019;8(2)doi: 10.3390/pathogens8020054.
Sparber, F., & LeibundGut-Landmann, S. (2019). Interleukin-17 in Antifungal Immunity. Pathogens (Basel, Switzerland), 8(2), . https://doi.org/10.3390/pathogens8020054
Sparber, Florian, and LeibundGut-Landmann, Salomé. "Interleukin-17 in Antifungal Immunity." Pathogens (Basel, Switzerland) vol. 8,2 (2019). doi: https://doi.org/10.3390/pathogens8020054
Sparber F, LeibundGut-Landmann S. Interleukin-17 in Antifungal Immunity. Pathogens. 2019 Apr 22;8(2). doi: 10.3390/pathogens8020054. PMID: 31013616; PMCID: PMC6630750.
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IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation.

Science immunology

Zwicky P, Ingelfinger F, Silva de Melo BM, Ruchti F, Schärli S, Puertas N, Lutz M, Phan TS, Kündig TM, Levesque MP, Maul JT, Schlapbach C, LeibundGut-Landmann S, Mundt S, Becher B.
PMID: 34678045
Sci Immunol. 2021 Oct 22;6(64):eabg9012. doi: 10.1126/sciimmunol.abg9012. Epub 2021 Oct 22.

[Figure: see text].

Cite
Zwicky P, Ingelfinger F, Silva de Melo BM, et al. IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation. Sci Immunol. 2021;6(64):eabg9012doi: 10.1126/sciimmunol.abg9012.
Zwicky, P., Ingelfinger, F., Silva de Melo, B. M., Ruchti, F., Schärli, S., Puertas, N., Lutz, M., Phan, T. S., Kündig, T. M., Levesque, M. P., Maul, J. T., Schlapbach, C., LeibundGut-Landmann, S., Mundt, S., & Becher, B. (2021). IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation. Science immunology, 6(64), eabg9012. https://doi.org/10.1126/sciimmunol.abg9012
Zwicky, Pascale, et al. "IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation." Science immunology vol. 6,64 (2021): eabg9012. doi: https://doi.org/10.1126/sciimmunol.abg9012
Zwicky P, Ingelfinger F, Silva de Melo BM, Ruchti F, Schärli S, Puertas N, Lutz M, Phan TS, Kündig TM, Levesque MP, Maul JT, Schlapbach C, LeibundGut-Landmann S, Mundt S, Becher B. IL-12 regulates type 3 immunity through interfollicular keratinocytes in psoriasiform inflammation. Sci Immunol. 2021 Oct 22;6(64):eabg9012. doi: 10.1126/sciimmunol.abg9012. Epub 2021 Oct 22. PMID: 34678045.
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