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A novel formulation for radiotherapy-induced oral mucositis: Triamcinolone acetonide mucoadhesive film.

Journal of research in medical sciences : the official journal of Isfahan University of Medical Sciences

Pakravan F, Ghalayani P, Emami H, Isfahani MN, Noorshargh P.
PMID: 31523249
J Res Med Sci. 2019 Jul 24;24:63. doi: 10.4103/jrms.JRMS_456_18. eCollection 2019.

BACKGROUND: The main purpose of this study was to evaluate the effectiveness of triamcinolone acetonide (TA) mucoadhesive films versus placebo as a preventive and therapeutic intervention of oral mucositis (OM) induced by radiotherapy for head-and-neck cancer (HNC) patients.MATERIALS AND...

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Pakravan F, Ghalayani P, Emami H, et al. A novel formulation for radiotherapy-induced oral mucositis: Triamcinolone acetonide mucoadhesive film. J Res Med Sci. 2019;24:63doi: 10.4103/jrms.JRMS_456_18.
Pakravan, F., Ghalayani, P., Emami, H., Isfahani, M. N., & Noorshargh, P. (2019). A novel formulation for radiotherapy-induced oral mucositis: Triamcinolone acetonide mucoadhesive film. Journal of research in medical sciences : the official journal of Isfahan University of Medical Sciences, 2463. https://doi.org/10.4103/jrms.JRMS_456_18
Pakravan, Fahimeh, et al. "A novel formulation for radiotherapy-induced oral mucositis: Triamcinolone acetonide mucoadhesive film." Journal of research in medical sciences : the official journal of Isfahan University of Medical Sciences vol. 24 (2019): 63. doi: https://doi.org/10.4103/jrms.JRMS_456_18
Pakravan F, Ghalayani P, Emami H, Isfahani MN, Noorshargh P. A novel formulation for radiotherapy-induced oral mucositis: Triamcinolone acetonide mucoadhesive film. J Res Med Sci. 2019 Jul 24;24:63. doi: 10.4103/jrms.JRMS_456_18. eCollection 2019. PMID: 31523249; PMCID: PMC6669991.
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Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome.

Circulation

Sánchez-López A, Espinós-Estévez C, González-Gómez C, Gonzalo P, Andrés-Manzano MJ, Fanjul V, Riquelme-Borja R, Hamczyk MR, Macías Á, Del Campo L, Camafeita E, Vázquez J, Barkaway A, Rolas L, Nourshargh S, Dorado B, Benedicto I, Andrés V.
PMID: 34694158
Circulation. 2021 Nov 30;144(22):1777-1794. doi: 10.1161/CIRCULATIONAHA.121.055313. Epub 2021 Oct 25.

No abstract available.

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Sánchez-López A, Espinós-Estévez C, González-Gómez C, et al. Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome. Circulation. 2021;144(22):1777-1794doi: 10.1161/CIRCULATIONAHA.121.055313.
Sánchez-López, A., Espinós-Estévez, C., González-Gómez, C., Gonzalo, P., Andrés-Manzano, M. J., Fanjul, V., Riquelme-Borja, R., Hamczyk, M. R., Macías, �. �., Del Campo, L., Camafeita, E., Vázquez, J., Barkaway, A., Rolas, L., Nourshargh, S., Dorado, B., Benedicto, I., & Andrés, V. (2021). Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome. Circulation, 144(22), 1777-1794. https://doi.org/10.1161/CIRCULATIONAHA.121.055313
Sánchez-López, Amanda, et al. "Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome." Circulation vol. 144,22 (2021): 1777-1794. doi: https://doi.org/10.1161/CIRCULATIONAHA.121.055313
Sánchez-López A, Espinós-Estévez C, González-Gómez C, Gonzalo P, Andrés-Manzano MJ, Fanjul V, Riquelme-Borja R, Hamczyk MR, Macías Á, Del Campo L, Camafeita E, Vázquez J, Barkaway A, Rolas L, Nourshargh S, Dorado B, Benedicto I, Andrés V. Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome. Circulation. 2021 Nov 30;144(22):1777-1794. doi: 10.1161/CIRCULATIONAHA.121.055313. Epub 2021 Oct 25. PMID: 34694158; PMCID: PMC8614561.
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Isoprenylcysteine Carboxylmethyltransferase-Based Therapy for Hutchinson-Gilford Progeria Syndrome.

ACS central science

Marcos-Ramiro B, Gil-Ordóñez A, Marín-Ramos NI, Ortega-Nogales FJ, Balabasquer M, Gonzalo P, Khiar-Fernández N, Rolas L, Barkaway A, Nourshargh S, Andrés V, Martín-Fontecha M, López-Rodríguez ML, Ortega-Gutiérrez S.
PMID: 34471675
ACS Cent Sci. 2021 Aug 25;7(8):1300-1310. doi: 10.1021/acscentsci.0c01698. Epub 2021 Jun 27.

Hutchinson-Gilford progeria syndrome (HGPS, progeria) is a rare genetic disease characterized by premature aging and death in childhood for which there were no approved drugs for its treatment until last November, when lonafarnib obtained long-sought FDA approval. However, the...

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Marcos-Ramiro B, Gil-Ordóñez A, Marín-Ramos NI, et al. Isoprenylcysteine Carboxylmethyltransferase-Based Therapy for Hutchinson-Gilford Progeria Syndrome. ACS Cent Sci. 2021;7(8):1300-1310doi: 10.1021/acscentsci.0c01698.
Marcos-Ramiro, B., Gil-Ordóñez, A., Marín-Ramos, N. I., Ortega-Nogales, F. J., Balabasquer, M., Gonzalo, P., Khiar-Fernández, N., Rolas, L., Barkaway, A., Nourshargh, S., Andrés, V., Martín-Fontecha, M., López-Rodríguez, M. L., & Ortega-Gutiérrez, S. (2021). Isoprenylcysteine Carboxylmethyltransferase-Based Therapy for Hutchinson-Gilford Progeria Syndrome. ACS central science, 7(8), 1300-1310. https://doi.org/10.1021/acscentsci.0c01698
Marcos-Ramiro, Beatriz, et al. "Isoprenylcysteine Carboxylmethyltransferase-Based Therapy for Hutchinson-Gilford Progeria Syndrome." ACS central science vol. 7,8 (2021): 1300-1310. doi: https://doi.org/10.1021/acscentsci.0c01698
Marcos-Ramiro B, Gil-Ordóñez A, Marín-Ramos NI, Ortega-Nogales FJ, Balabasquer M, Gonzalo P, Khiar-Fernández N, Rolas L, Barkaway A, Nourshargh S, Andrés V, Martín-Fontecha M, López-Rodríguez ML, Ortega-Gutiérrez S. Isoprenylcysteine Carboxylmethyltransferase-Based Therapy for Hutchinson-Gilford Progeria Syndrome. ACS Cent Sci. 2021 Aug 25;7(8):1300-1310. doi: 10.1021/acscentsci.0c01698. Epub 2021 Jun 27. PMID: 34471675; PMCID: PMC8393201.
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Endothelial cell autophagy keeps neutrophil trafficking under control.

Autophagy

Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S.
PMID: 34720030
Autophagy. 2021 Dec;17(12):4509-4511. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31.

A defining feature of an inflammatory reaction is infiltration of neutrophils into tissues, a response that requires breaching of endothelial cells (ECs) that line the lumenal aspect of blood vessels. Dysregulated neutrophil trafficking is a hallmark of pathology, but...

Cite
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021;17(12):4509-4511doi: 10.1080/15548627.2021.1987675.
Reglero-Real, N., Pérez-Gutiérrez, L., & Nourshargh, S. (2021). Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy, 17(12), 4509-4511. https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real, Natalia, et al. "Endothelial cell autophagy keeps neutrophil trafficking under control." Autophagy vol. 17,12 (2021): 4509-4511. doi: https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021 Dec;17(12):4509-4511. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31. PMID: 34720030.
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Endothelial cell autophagy keeps neutrophil trafficking under control.

Autophagy

Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S.
PMID: 34720030
Autophagy. 2021 Dec;17(12):4509-4511. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31.

A defining feature of an inflammatory reaction is infiltration of neutrophils into tissues, a response that requires breaching of endothelial cells (ECs) that line the lumenal aspect of blood vessels. Dysregulated neutrophil trafficking is a hallmark of pathology, but...

Cite
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021;17(12):4509-4511doi: 10.1080/15548627.2021.1987675.
Reglero-Real, N., Pérez-Gutiérrez, L., & Nourshargh, S. (2021). Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy, 17(12), 4509-4511. https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real, Natalia, et al. "Endothelial cell autophagy keeps neutrophil trafficking under control." Autophagy vol. 17,12 (2021): 4509-4511. doi: https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021 Dec;17(12):4509-4511. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31. PMID: 34720030.
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Endothelial cell autophagy keeps neutrophil trafficking under control.

Autophagy

Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S.
PMID: 34720030
Autophagy. 2021 Oct 31;1-3. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31.

A defining feature of an inflammatory reaction is infiltration of neutrophils into tissues, a response that requires breaching of endothelial cells (ECs) that line the lumenal aspect of blood vessels. Dysregulated neutrophil trafficking is a hallmark of pathology, but...

Cite
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021;1-3doi: 10.1080/15548627.2021.1987675.
Reglero-Real, N., Pérez-Gutiérrez, L., & Nourshargh, S. (2021). Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy, 1-3. https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real, Natalia, et al. "Endothelial cell autophagy keeps neutrophil trafficking under control." Autophagy vol. (2021): 1-3. doi: https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021 Oct 31;1-3. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31. PMID: 34720030.
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Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome.

Circulation

Sánchez-López A, Espinós-Estévez C, González-Gómez C, Gonzalo P, Andrés-Manzano MJ, Fanjul V, Riquelme-Borja R, Hamczyk MR, Macías Á, Del Campo L, Camafeita E, Vázquez J, Barkaway A, Rolas L, Nourshargh S, Dorado B, Benedicto I, Andrés V.
PMID: 34694158
Circulation. 2021 Nov 30;144(22):1777-1794. doi: 10.1161/CIRCULATIONAHA.121.055313. Epub 2021 Oct 25.

No abstract available.

Cite
Sánchez-López A, Espinós-Estévez C, González-Gómez C, et al. Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome. Circulation. 2021;144(22):1777-1794doi: 10.1161/CIRCULATIONAHA.121.055313.
Sánchez-López, A., Espinós-Estévez, C., González-Gómez, C., Gonzalo, P., Andrés-Manzano, M. J., Fanjul, V., Riquelme-Borja, R., Hamczyk, M. R., Macías, �. �., Del Campo, L., Camafeita, E., Vázquez, J., Barkaway, A., Rolas, L., Nourshargh, S., Dorado, B., Benedicto, I., & Andrés, V. (2021). Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome. Circulation, 144(22), 1777-1794. https://doi.org/10.1161/CIRCULATIONAHA.121.055313
Sánchez-López, Amanda, et al. "Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome." Circulation vol. 144,22 (2021): 1777-1794. doi: https://doi.org/10.1161/CIRCULATIONAHA.121.055313
Sánchez-López A, Espinós-Estévez C, González-Gómez C, Gonzalo P, Andrés-Manzano MJ, Fanjul V, Riquelme-Borja R, Hamczyk MR, Macías Á, Del Campo L, Camafeita E, Vázquez J, Barkaway A, Rolas L, Nourshargh S, Dorado B, Benedicto I, Andrés V. Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome. Circulation. 2021 Nov 30;144(22):1777-1794. doi: 10.1161/CIRCULATIONAHA.121.055313. Epub 2021 Oct 25. PMID: 34694158; PMCID: PMC8614561.
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Sex-specific regulation of chemokine Cxcl5/6 controls neutrophil recruitment and tissue injury in acute inflammatory states.

Biology of sex differences

Madalli S, Beyrau M, Whiteford J, Duchene J, Singh Nandhra I, Patel NS, Motwani MP, Gilroy DW, Thiemermann C, Nourshargh S, Scotland RS.
PMID: 26617980
Biol Sex Differ. 2015 Nov 26;6:27. doi: 10.1186/s13293-015-0047-5. eCollection 2015.

BACKGROUND: Tissue infiltration by neutrophils during acute inflammatory states causes substantial tissue injury. While the magnitude of tissue neutrophil accumulation in innate immune responses is profoundly greater in males than females, fundamental aspects of the molecular mechanisms underlying these...

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Madalli S, Beyrau M, Whiteford J, et al. Sex-specific regulation of chemokine Cxcl5/6 controls neutrophil recruitment and tissue injury in acute inflammatory states. Biol Sex Differ. 2015;6:27doi: 10.1186/s13293-015-0047-5.
Madalli, S., Beyrau, M., Whiteford, J., Duchene, J., Singh Nandhra, I., Patel, N. S., Motwani, M. P., Gilroy, D. W., Thiemermann, C., Nourshargh, S., & Scotland, R. S. (2015). Sex-specific regulation of chemokine Cxcl5/6 controls neutrophil recruitment and tissue injury in acute inflammatory states. Biology of sex differences, 627. https://doi.org/10.1186/s13293-015-0047-5
Madalli, Shimona, et al. "Sex-specific regulation of chemokine Cxcl5/6 controls neutrophil recruitment and tissue injury in acute inflammatory states." Biology of sex differences vol. 6 (2015): 27. doi: https://doi.org/10.1186/s13293-015-0047-5
Madalli S, Beyrau M, Whiteford J, Duchene J, Singh Nandhra I, Patel NS, Motwani MP, Gilroy DW, Thiemermann C, Nourshargh S, Scotland RS. Sex-specific regulation of chemokine Cxcl5/6 controls neutrophil recruitment and tissue injury in acute inflammatory states. Biol Sex Differ. 2015 Nov 26;6:27. doi: 10.1186/s13293-015-0047-5. eCollection 2015. PMID: 26617980; PMCID: PMC4661984.
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Correction: Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study.

PLoS medicine

Cabrera CP, Manson J, Shepherd JM, Torrance HD, Watson D, Longhi MP, Hoti M, Patel MB, O'Dwyer M, Nourshargh S, Pennington DJ, Barnes MR, Brohi K.
PMID: 30379817
PLoS Med. 2018 Oct 31;15(10):e1002694. doi: 10.1371/journal.pmed.1002694. eCollection 2018 Oct.

[This corrects the article DOI: 10.1371/journal.pmed.1002352.].

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Cabrera CP, Manson J, Shepherd JM, et al. Correction: Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study. PLoS Med. 2018;15(10):e1002694doi: 10.1371/journal.pmed.1002694.
Cabrera, C. P., Manson, J., Shepherd, J. M., Torrance, H. D., Watson, D., Longhi, M. P., Hoti, M., Patel, M. B., O'Dwyer, M., Nourshargh, S., Pennington, D. J., Barnes, M. R., & Brohi, K. (2018). Correction: Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study. PLoS medicine, 15(10), e1002694. https://doi.org/10.1371/journal.pmed.1002694
Cabrera, Claudia P, et al. "Correction: Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study." PLoS medicine vol. 15,10 (2018): e1002694. doi: https://doi.org/10.1371/journal.pmed.1002694
Cabrera CP, Manson J, Shepherd JM, Torrance HD, Watson D, Longhi MP, Hoti M, Patel MB, O'Dwyer M, Nourshargh S, Pennington DJ, Barnes MR, Brohi K. Correction: Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study. PLoS Med. 2018 Oct 31;15(10):e1002694. doi: 10.1371/journal.pmed.1002694. eCollection 2018 Oct. PMID: 30379817; PMCID: PMC6209129.
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Endothelial cell autophagy keeps neutrophil trafficking under control.

Autophagy

Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S.
PMID: 34720030
Autophagy. 2021 Dec;17(12):4509-4511. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31.

A defining feature of an inflammatory reaction is infiltration of neutrophils into tissues, a response that requires breaching of endothelial cells (ECs) that line the lumenal aspect of blood vessels. Dysregulated neutrophil trafficking is a hallmark of pathology, but...

Cite
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021;17(12):4509-4511doi: 10.1080/15548627.2021.1987675.
Reglero-Real, N., Pérez-Gutiérrez, L., & Nourshargh, S. (2021). Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy, 17(12), 4509-4511. https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real, Natalia, et al. "Endothelial cell autophagy keeps neutrophil trafficking under control." Autophagy vol. 17,12 (2021): 4509-4511. doi: https://doi.org/10.1080/15548627.2021.1987675
Reglero-Real N, Pérez-Gutiérrez L, Nourshargh S. Endothelial cell autophagy keeps neutrophil trafficking under control. Autophagy. 2021 Dec;17(12):4509-4511. doi: 10.1080/15548627.2021.1987675. Epub 2021 Oct 31. PMID: 34720030.
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The spatiotemporal localization of JAM-C following sciatic nerve crush in adult rats.

Brain and behavior

Avari P, Huang W, Averill S, Colom B, Imhof BA, Nourshargh S, Priestley JV.
PMID: 22950044
Brain Behav. 2012 Jul;2(4):402-14. doi: 10.1002/brb3.63.

JAM-C is a junctional adhesion molecule, enriched at tight junctions on endothelial and epithelial cells, and also localized to Schwann cells at junctions between adjoining myelin end loops. The role of JAM-C following peripheral nerve injury (PNI) is currently...

Cite
Avari P, Huang W, Averill S, et al. The spatiotemporal localization of JAM-C following sciatic nerve crush in adult rats. Brain Behav. 2012;2(4):402-14doi: 10.1002/brb3.63.
Avari, P., Huang, W., Averill, S., Colom, B., Imhof, B. A., Nourshargh, S., & Priestley, J. V. (2012). The spatiotemporal localization of JAM-C following sciatic nerve crush in adult rats. Brain and behavior, 2(4), 402-14. https://doi.org/10.1002/brb3.63
Avari, Parizad, et al. "The spatiotemporal localization of JAM-C following sciatic nerve crush in adult rats." Brain and behavior vol. 2,4 (2012): 402-14. doi: https://doi.org/10.1002/brb3.63
Avari P, Huang W, Averill S, Colom B, Imhof BA, Nourshargh S, Priestley JV. The spatiotemporal localization of JAM-C following sciatic nerve crush in adult rats. Brain Behav. 2012 Jul;2(4):402-14. doi: 10.1002/brb3.63. PMID: 22950044; PMCID: PMC3432963.
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The Association Between Quality of Sleep and Health-related Quality of Life in Military and Non-military Women in Tehran, Iran.

Oman medical journal

Roustaei N, Jamali H, Jamali MR, Nourshargh P, Jamali J.
PMID: 28439383
Oman Med J. 2017 Mar;32(2):134-130. doi: 10.5001/omj.2017.22.

OBJECTIVES: Quality of sleep (QoS) in individuals is affected by their occupation and is one of the factors affecting the quality of life (QoL). Few studies have evaluated the relationship between QoS and QoL in military women personnel. The...

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Roustaei N, Jamali H, Jamali MR, et al. The Association Between Quality of Sleep and Health-related Quality of Life in Military and Non-military Women in Tehran, Iran. Oman Med J. 2017;32(2):134-130doi: 10.5001/omj.2017.22.
Roustaei, N., Jamali, H., Jamali, M. R., Nourshargh, P., & Jamali, J. (2017). The Association Between Quality of Sleep and Health-related Quality of Life in Military and Non-military Women in Tehran, Iran. Oman medical journal, 32(2), 134-130. https://doi.org/10.5001/omj.2017.22
Roustaei, Narges, et al. "The Association Between Quality of Sleep and Health-related Quality of Life in Military and Non-military Women in Tehran, Iran." Oman medical journal vol. 32,2 (2017): 134-130. doi: https://doi.org/10.5001/omj.2017.22
Roustaei N, Jamali H, Jamali MR, Nourshargh P, Jamali J. The Association Between Quality of Sleep and Health-related Quality of Life in Military and Non-military Women in Tehran, Iran. Oman Med J. 2017 Mar;32(2):134-130. doi: 10.5001/omj.2017.22. PMID: 28439383; PMCID: PMC5397081.
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