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Changes in health risks among the participants in the united auto workers—General Motors LifeSteps Health Promotion Program

program design

Yen L.
GSID: uT9fcMfUJwcJ
L Yen, MP Edington, T McDonald… - American Journal of …, 2001 - journals.sagepub.com

… , especially when compared with worksites with a less comprehensive program design,12 has provided evidence that development and … -one focused interaction is an important element in programming design.The UAW-GM LifeSteps Health Promotion Program, jointly devel- …

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Yen, L., Edington, M. P., McDonald, T., Hirschland, D., & Edington, D. W. (2001). Changes in health risks among the participants in the united auto workers—General Motors LifeSteps Health Promotion Program. American Journal of Health Promotion, 16(1), 7-15.
Yen, Louis, et al. "Changes in health risks among the participants in the united auto workers—General Motors LifeSteps Health Promotion Program." American Journal of Health Promotion 16.1 (2001): 7-15.
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The differential effects of job design on knowledge workers and manual workers: A quasi‐experimental field study in China

quasi-experimental designs

Yan M, Peng KZ.
GSID: COw3TFtIP7EJ
M Yan, KZ Peng, AM Francesco - Human Resource …, 2011 - Wiley Online Library

… To examine our hypotheses, a quasi-experimental field study of KWs and MWs was designed, in which their jobs are enriched by real-time manipulations. The experiment process and results are presented and discussed. Finally, we summarize our contributions and...

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Yan, M., Peng, K. Z., & Francesco, A. M. (2011). The differential effects of job design on knowledge workers and manual workers: A quasi‐experimental field study in China. Human Resource Management, 50(3), 407-424.
Yan, Ming, Kelly Z. Peng, and Anne Marie Francesco. "The differential effects of job design on knowledge workers and manual workers: A quasi‐experimental field study in China." Human Resource Management 50.3 (2011): 407-424.
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Bioorthogonal Oxime Ligation Mediated .

Chemical science

Tang L, Yin Q, Xu Y, Zhou Q, Cai K, Yen J, Dobrucki LW, Cheng J.
PMID: 26146536
Chem Sci. 2015 Apr 01;6(4):2182-2186. doi: 10.1039/C5SC00063G.

Current cancer targeting relying on specific biological interaction between cell surface antigen and respective antibody or its analogue has proven to be effective in the treatment of different cancers; however, this strategy has its own limitations, such as heterogeneity...

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Tang L, Yin Q, Xu Y, et al. Bioorthogonal Oxime Ligation Mediated . Chem Sci. 2015;6(4):2182-2186doi: 10.1039/C5SC00063G.
Tang, L., Yin, Q., Xu, Y., Zhou, Q., Cai, K., Yen, J., Dobrucki, L. W., & Cheng, J. (2015). Bioorthogonal Oxime Ligation Mediated . Chemical science, 6(4), 2182-2186. https://doi.org/10.1039/C5SC00063G
Tang, Li, et al. "Bioorthogonal Oxime Ligation Mediated ." Chemical science vol. 6,4 (2015): 2182-2186. doi: https://doi.org/10.1039/C5SC00063G
Tang L, Yin Q, Xu Y, Zhou Q, Cai K, Yen J, Dobrucki LW, Cheng J. Bioorthogonal Oxime Ligation Mediated . Chem Sci. 2015 Apr 01;6(4):2182-2186. doi: 10.1039/C5SC00063G. PMID: 26146536; PMCID: PMC4486360.
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A case-control study of the association between the .

Oncotarget

Yan M, Li J, He N, Shi X, Du S, Li B, Jin T.
PMID: 28938685
Oncotarget. 2017 Apr 07;8(35):59823-59830. doi: 10.18632/oncotarget.16946. eCollection 2017 Aug 29.

The Epidermal Growth Factor Receptor gene has been reported to be involved in the progression of gliomas which is one of the deadliest primary brain tumors in humans. To determine potential association between

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Yan M, Li J, He N, et al. A case-control study of the association between the . Oncotarget. 2017;8(35):59823-59830doi: 10.18632/oncotarget.16946.
Yan, M., Li, J., He, N., Shi, X., Du, S., Li, B., & Jin, T. (2017). A case-control study of the association between the . Oncotarget, 8(35), 59823-59830. https://doi.org/10.18632/oncotarget.16946
Yan, Mengdan, et al. "A case-control study of the association between the ." Oncotarget vol. 8,35 (2017): 59823-59830. doi: https://doi.org/10.18632/oncotarget.16946
Yan M, Li J, He N, Shi X, Du S, Li B, Jin T. A case-control study of the association between the . Oncotarget. 2017 Apr 07;8(35):59823-59830. doi: 10.18632/oncotarget.16946. eCollection 2017 Aug 29. PMID: 28938685; PMCID: PMC5601781.
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Secreted protein acidic and rich in cysteine (SPARC) induces cell migration and epithelial mesenchymal transition through WNK1/snail in non-small cell lung cancer.

Oncotarget

Hung JY, Yen MC, Jian SF, Wu CY, Chang WA, Liu KT, Hsu YL, Chong IW, Kuo PL.
PMID: 28969021
Oncotarget. 2017 Jul 22;8(38):63691-63702. doi: 10.18632/oncotarget.19475. eCollection 2017 Sep 08.

The extracellular matrix is a component of physiological microenvironment and a regulator of cellular processes such as migration and proliferation. Secreted Protein Acidic and Rich in Cysteine (SPARC/osteonectin) is an extracellular matrix-associated glycoprotein involved in the regulation of cell...

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Hung JY, Yen MC, Jian SF, et al. Secreted protein acidic and rich in cysteine (SPARC) induces cell migration and epithelial mesenchymal transition through WNK1/snail in non-small cell lung cancer. Oncotarget. 2017;8(38):63691-63702doi: 10.18632/oncotarget.19475.
Hung, J. Y., Yen, M. C., Jian, S. F., Wu, C. Y., Chang, W. A., Liu, K. T., Hsu, Y. L., Chong, I. W., & Kuo, P. L. (2017). Secreted protein acidic and rich in cysteine (SPARC) induces cell migration and epithelial mesenchymal transition through WNK1/snail in non-small cell lung cancer. Oncotarget, 8(38), 63691-63702. https://doi.org/10.18632/oncotarget.19475
Hung, Jen-Yu, et al. "Secreted protein acidic and rich in cysteine (SPARC) induces cell migration and epithelial mesenchymal transition through WNK1/snail in non-small cell lung cancer." Oncotarget vol. 8,38 (2017): 63691-63702. doi: https://doi.org/10.18632/oncotarget.19475
Hung JY, Yen MC, Jian SF, Wu CY, Chang WA, Liu KT, Hsu YL, Chong IW, Kuo PL. Secreted protein acidic and rich in cysteine (SPARC) induces cell migration and epithelial mesenchymal transition through WNK1/snail in non-small cell lung cancer. Oncotarget. 2017 Jul 22;8(38):63691-63702. doi: 10.18632/oncotarget.19475. eCollection 2017 Sep 08. PMID: 28969021; PMCID: PMC5609953.
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Correction: Genetic Architecture of Atherosclerosis in Mice: A Systems Genetics Analysis of Common Inbred Strains.

PLoS genetics

Bennett BJ, Davis RC, Civelek M, Orozco L, Wu J, Qi H, Pan C, Sevag Packard RR, Eskin E, Yan M, Kirchgessner T, Wang Z, Li X, Gregory JC, Hazen SL, Gargalovic PS, Lusis AJ.
PMID: 26934746
PLoS Genet. 2016 Mar 02;12(3):e1005913. doi: 10.1371/journal.pgen.1005913. eCollection 2016 Mar.

No abstract available.

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Bennett BJ, Davis RC, Civelek M, et al. Correction: Genetic Architecture of Atherosclerosis in Mice: A Systems Genetics Analysis of Common Inbred Strains. PLoS Genet. 2016;12(3):e1005913doi: 10.1371/journal.pgen.1005913.
Bennett, B. J., Davis, R. C., Civelek, M., Orozco, L., Wu, J., Qi, H., Pan, C., Sevag Packard, R. R., Eskin, E., Yan, M., Kirchgessner, T., Wang, Z., Li, X., Gregory, J. C., Hazen, S. L., Gargalovic, P. S., & Lusis, A. J. (2016). Correction: Genetic Architecture of Atherosclerosis in Mice: A Systems Genetics Analysis of Common Inbred Strains. PLoS genetics, 12(3), e1005913. https://doi.org/10.1371/journal.pgen.1005913
Bennett, Brian J, et al. "Correction: Genetic Architecture of Atherosclerosis in Mice: A Systems Genetics Analysis of Common Inbred Strains." PLoS genetics vol. 12,3 (2016): e1005913. doi: https://doi.org/10.1371/journal.pgen.1005913
Bennett BJ, Davis RC, Civelek M, Orozco L, Wu J, Qi H, Pan C, Sevag Packard RR, Eskin E, Yan M, Kirchgessner T, Wang Z, Li X, Gregory JC, Hazen SL, Gargalovic PS, Lusis AJ. Correction: Genetic Architecture of Atherosclerosis in Mice: A Systems Genetics Analysis of Common Inbred Strains. PLoS Genet. 2016 Mar 02;12(3):e1005913. doi: 10.1371/journal.pgen.1005913. eCollection 2016 Mar. PMID: 26934746; PMCID: PMC4775237.
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p16 controls epithelial cell growth and suppresses carcinogenesis through mechanisms that do not require RB1 function.

Oncogenesis

Sen M, Akeno N, Reece A, Miller AL, Simpson DS, Wikenheiser-Brokamp KA.
PMID: 28414317
Oncogenesis. 2017 Apr 17;6(4):e320. doi: 10.1038/oncsis.2017.5.

The p16/RB1 tumor suppressor pathway is inactivated in the vast majority, if not all, human cancers. The current paradigm is that p16 and RB1 function in a linear pathway to suppress tumorigenesis; however p16 is preferentially lost in human...

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Sen M, Akeno N, Reece A, et al. p16 controls epithelial cell growth and suppresses carcinogenesis through mechanisms that do not require RB1 function. Oncogenesis. 2017;6(4):e320doi: 10.1038/oncsis.2017.5.
Sen, M., Akeno, N., Reece, A., Miller, A. L., Simpson, D. S., & Wikenheiser-Brokamp, K. A. (2017). p16 controls epithelial cell growth and suppresses carcinogenesis through mechanisms that do not require RB1 function. Oncogenesis, 6(4), e320. https://doi.org/10.1038/oncsis.2017.5
Sen, M, et al. "p16 controls epithelial cell growth and suppresses carcinogenesis through mechanisms that do not require RB1 function." Oncogenesis vol. 6,4 (2017): e320. doi: https://doi.org/10.1038/oncsis.2017.5
Sen M, Akeno N, Reece A, Miller AL, Simpson DS, Wikenheiser-Brokamp KA. p16 controls epithelial cell growth and suppresses carcinogenesis through mechanisms that do not require RB1 function. Oncogenesis. 2017 Apr 17;6(4):e320. doi: 10.1038/oncsis.2017.5. PMID: 28414317; PMCID: PMC5520502.
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Overexpression and proliferation dependence of acyl-CoA thioesterase 11 and 13 in lung adenocarcinoma.

Oncology letters

Hung JY, Chiang SR, Liu KT, Tsai MJ, Huang MS, Shieh JM, Yen MC, Hsu YL.
PMID: 28927126
Oncol Lett. 2017 Sep;14(3):3647-3656. doi: 10.3892/ol.2017.6594. Epub 2017 Jul 18.

The metabolites of fatty acyl-Coenzyme A (CoA) and metabolic enzymes contribute to lipid biosynthesis, signal transduction, and gene transcription. Previous studies have indicated that elevated concentrations of specific free fatty acids in the plasma and overexpression of specific fatty...

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Hung JY, Chiang SR, Liu KT, et al. Overexpression and proliferation dependence of acyl-CoA thioesterase 11 and 13 in lung adenocarcinoma. Oncol Lett. 2017;14(3):3647-3656doi: 10.3892/ol.2017.6594.
Hung, J. Y., Chiang, S. R., Liu, K. T., Tsai, M. J., Huang, M. S., Shieh, J. M., Yen, M. C., & Hsu, Y. L. (2017). Overexpression and proliferation dependence of acyl-CoA thioesterase 11 and 13 in lung adenocarcinoma. Oncology letters, 14(3), 3647-3656. https://doi.org/10.3892/ol.2017.6594
Hung, Jen-Yu, et al. "Overexpression and proliferation dependence of acyl-CoA thioesterase 11 and 13 in lung adenocarcinoma." Oncology letters vol. 14,3 (2017): 3647-3656. doi: https://doi.org/10.3892/ol.2017.6594
Hung JY, Chiang SR, Liu KT, Tsai MJ, Huang MS, Shieh JM, Yen MC, Hsu YL. Overexpression and proliferation dependence of acyl-CoA thioesterase 11 and 13 in lung adenocarcinoma. Oncol Lett. 2017 Sep;14(3):3647-3656. doi: 10.3892/ol.2017.6594. Epub 2017 Jul 18. PMID: 28927126; PMCID: PMC5587920.
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Vascular endothelial growth factor and protein level in pleural effusion for differentiating malignant from benign pleural effusion.

Oncology letters

Wu DW, Chang WA, Liu KT, Yen MC, Kuo PL.
PMID: 28927127
Oncol Lett. 2017 Sep;14(3):3657-3662. doi: 10.3892/ol.2017.6631. Epub 2017 Jul 20.

Pleural effusion is associated with multiple benign and malignant conditions. Currently no biomarkers differentiate malignant pleural effusion (MPE) and benign pleural effusion (BPE) sensitively and specifically. The present study identified a novel combination of biomarkers in pleural effusion for...

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Wu DW, Chang WA, Liu KT, et al. Vascular endothelial growth factor and protein level in pleural effusion for differentiating malignant from benign pleural effusion. Oncol Lett. 2017;14(3):3657-3662doi: 10.3892/ol.2017.6631.
Wu, D. W., Chang, W. A., Liu, K. T., Yen, M. C., & Kuo, P. L. (2017). Vascular endothelial growth factor and protein level in pleural effusion for differentiating malignant from benign pleural effusion. Oncology letters, 14(3), 3657-3662. https://doi.org/10.3892/ol.2017.6631
Wu, Da-Wei, et al. "Vascular endothelial growth factor and protein level in pleural effusion for differentiating malignant from benign pleural effusion." Oncology letters vol. 14,3 (2017): 3657-3662. doi: https://doi.org/10.3892/ol.2017.6631
Wu DW, Chang WA, Liu KT, Yen MC, Kuo PL. Vascular endothelial growth factor and protein level in pleural effusion for differentiating malignant from benign pleural effusion. Oncol Lett. 2017 Sep;14(3):3657-3662. doi: 10.3892/ol.2017.6631. Epub 2017 Jul 20. PMID: 28927127; PMCID: PMC5587994.
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Equilibration Dynamics of Strongly Interacting Bosons in 2D Lattices with Disorder.

Physical review letters

Yan M, Hui HY, Rigol M, Scarola VW.
PMID: 28949694
Phys Rev Lett. 2017 Aug 18;119(7):073002. doi: 10.1103/PhysRevLett.119.073002. Epub 2017 Aug 14.

Motivated by recent optical lattice experiments [J.-y. Choi et al., Science 352, 1547 (2016)SCIEAS0036-807510.1126/science.aaf8834], we study the dynamics of strongly interacting bosons in the presence of disorder in two dimensions. We show that Gutzwiller mean-field theory (GMFT) captures the...

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Yan M, Hui HY, Rigol M, et al. Equilibration Dynamics of Strongly Interacting Bosons in 2D Lattices with Disorder. Phys Rev Lett. 2017;119(7):073002doi: 10.1103/PhysRevLett.119.073002.
Yan, M., Hui, H. Y., Rigol, M., & Scarola, V. W. (2017). Equilibration Dynamics of Strongly Interacting Bosons in 2D Lattices with Disorder. Physical review letters, 119(7), 073002. https://doi.org/10.1103/PhysRevLett.119.073002
Yan, Mi, et al. "Equilibration Dynamics of Strongly Interacting Bosons in 2D Lattices with Disorder." Physical review letters vol. 119,7 (2017): 073002. doi: https://doi.org/10.1103/PhysRevLett.119.073002
Yan M, Hui HY, Rigol M, Scarola VW. Equilibration Dynamics of Strongly Interacting Bosons in 2D Lattices with Disorder. Phys Rev Lett. 2017 Aug 18;119(7):073002. doi: 10.1103/PhysRevLett.119.073002. Epub 2017 Aug 14. PMID: 28949694.
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The Effect of Mycophenolate Mofetil on Early Wound Healing in a Rodent Model.

Transplantation direct

Willems MC, Hendriks T, Lomme RM, de Man BM, van der Vliet JA.
PMID: 27500270
Transplant Direct. 2016 May 20;2(6):e80. doi: 10.1097/TXD.0000000000000591. eCollection 2016 Jun.

BACKGROUND: Immunosuppressant agents are inevitable for solid organ recipients, but may have a negative effect on wound healing that is difficult to measure because of clinical use of a polydrug regime. The evidence on mycophenolate mofetil (MMF) is scarce...

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Willems MC, Hendriks T, Lomme RM, et al. The Effect of Mycophenolate Mofetil on Early Wound Healing in a Rodent Model. Transplant Direct. 2016;2(6):e80doi: 10.1097/TXD.0000000000000591.
Willems, M. C., Hendriks, T., Lomme, R. M., de Man, B. M., & van der Vliet, J. A. (2016). The Effect of Mycophenolate Mofetil on Early Wound Healing in a Rodent Model. Transplantation direct, 2(6), e80. https://doi.org/10.1097/TXD.0000000000000591
Willems, Martine Cm, et al. "The Effect of Mycophenolate Mofetil on Early Wound Healing in a Rodent Model." Transplantation direct vol. 2,6 (2016): e80. doi: https://doi.org/10.1097/TXD.0000000000000591
Willems MC, Hendriks T, Lomme RM, de Man BM, van der Vliet JA. The Effect of Mycophenolate Mofetil on Early Wound Healing in a Rodent Model. Transplant Direct. 2016 May 20;2(6):e80. doi: 10.1097/TXD.0000000000000591. eCollection 2016 Jun. PMID: 27500270; PMCID: PMC4946522.
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Corrigendum: Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk.

Nature genetics

Warren HR, Evangelou E, Cabrera CP, Gao H, Ren M, Mifsud B, Ntalla I, Surendran P, Liu C, Cook JP, Kraja AT, Drenos F, Loh M, Verweij N, Marten J, Karaman I, Segura Lepe MP, O'Reilly PF, Knight J, Snieder H, Kato N, He J, Tai ES, Said MA, Porteous D, Alver M, Poulter N, Farrall M, Gansevoort RT, Padmanabhan S, Mägi R, Stanton A, Connell J, Bakker SJL, Metspalu A, Shields DC, Thom S, Brown M, Sever P, Esko T, Hayward C, van der Harst P, Saleheen D, Chowdhury R, Chambers JC, Chasman DI, Chakravarti A, Newton-Cheh C, Lindgren CM, Levy D, Kooner JS, Keavney B, Tomaszewski M, Samani NJ, Howson JMM, Tobin MD, Munroe PB, Ehret GB, Wain LV, Barnes MR, Tzoulaki I, Caulfield MJ, Elliott P.
PMID: 28951623
Nat Genet. 2017 Sep 27;49(10):1558. doi: 10.1038/ng1017-1558a.

No abstract available.

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Warren HR, Evangelou E, Cabrera CP, et al. Corrigendum: Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk. Nat Genet. 2017;49(10):1558doi: 10.1038/ng1017-1558a.
Warren, H. R., Evangelou, E., Cabrera, C. P., Gao, H., Ren, M., Mifsud, B., Ntalla, I., Surendran, P., Liu, C., Cook, J. P., Kraja, A. T., Drenos, F., Loh, M., Verweij, N., Marten, J., Karaman, I., Segura Lepe, M. P., O'Reilly, P. F., Knight, J., Snieder, H., Kato, N., He, J., Tai, E. S., Said, M. A., Porteous, D., Alver, M., Poulter, N., Farrall, M., Gansevoort, R. T., Padmanabhan, S., Mägi, R., Stanton, A., Connell, J., Bakker, S. J. L., Metspalu, A., Shields, D. C., Thom, S., Brown, M., Sever, P., Esko, T., Hayward, C., van der Harst, P., Saleheen, D., Chowdhury, R., Chambers, J. C., Chasman, D. I., Chakravarti, A., Newton-Cheh, C., Lindgren, C. M., Levy, D., Kooner, J. S., Keavney, B., Tomaszewski, M., Samani, N. J., Howson, J. M. M., Tobin, M. D., Munroe, P. B., Ehret, G. B., Wain, L. V., Barnes, M. R., Tzoulaki, I., Caulfield, M. J., Elliott, P. (2017). Corrigendum: Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk. Nature genetics, 49(10), 1558. https://doi.org/10.1038/ng1017-1558a
Warren, Helen R, et al. "Corrigendum: Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk." Nature genetics vol. 49,10 (2017): 1558. doi: https://doi.org/10.1038/ng1017-1558a
Warren HR, Evangelou E, Cabrera CP, Gao H, Ren M, Mifsud B, Ntalla I, Surendran P, Liu C, Cook JP, Kraja AT, Drenos F, Loh M, Verweij N, Marten J, Karaman I, Segura Lepe MP, O'Reilly PF, Knight J, Snieder H, Kato N, He J, Tai ES, Said MA, Porteous D, Alver M, Poulter N, Farrall M, Gansevoort RT, Padmanabhan S, Mägi R, Stanton A, Connell J, Bakker SJL, Metspalu A, Shields DC, Thom S, Brown M, Sever P, Esko T, Hayward C, van der Harst P, Saleheen D, Chowdhury R, Chambers JC, Chasman DI, Chakravarti A, Newton-Cheh C, Lindgren CM, Levy D, Kooner JS, Keavney B, Tomaszewski M, Samani NJ, Howson JMM, Tobin MD, Munroe PB, Ehret GB, Wain LV, Barnes MR, Tzoulaki I, Caulfield MJ, Elliott P. Corrigendum: Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk. Nat Genet. 2017 Sep 27;49(10):1558. doi: 10.1038/ng1017-1558a. PMID: 28951623.
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Showing 1 to 12 of 3334 entries