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Sequestosome‐1 (p62) expression reveals chaperone‐assisted selective autophagy in immune‐mediated necrotizing myopathies

science campus

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GSID: gEBeAYNmtDQJ
N Fischer, C Preuße, J Radke, D Pehl… - Brain …, 2020 - Wiley Online Library

Diffuse myofiber necrosis in the context of inflammatory myopathy is the hallmark of immune‐mediated necrotizing myopathy (IMNM). We have previously shown that skeletal muscle fibers of IMNM patients may display nonrimmed vacuoles and sarcoplasmic irregularities. The …

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Fischer, N., Preuße, C., Radke, J., Pehl, D., Allenbach, Y., Schneider, U., ... & Stenzel, W. (2020). Sequestosome‐1 (p62) expression reveals chaperone‐assisted selective autophagy in immune‐mediated necrotizing myopathies. Brain Pathology, 30(2), 261-271.‏
Fischer, Norina, et al. "Sequestosome‐1 (p62) expression reveals chaperone‐assisted selective autophagy in immune‐mediated necrotizing myopathies." Brain Pathology 30.2 (2020): 261-271.‏
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Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells.

Biology of sex differences

Addis R, Campesi I, Fois M, Capobianco G, Dessole S, Fenu G, Montella A, Cattaneo MG, Vicentini LM, Franconi F.
PMID: 25535548
Biol Sex Differ. 2014 Dec 14;5(1):18. doi: 10.1186/s13293-014-0018-2. eCollection 2014.

BACKGROUND: Human umbilical endothelial cells (HUVECs) are widely used to study the endothelial physiology and pathology that might be involved in sex and gender differences detected at the cardiovascular level. This study evaluated whether HUVECs are sexually dimorphic in...

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Addis R, Campesi I, Fois M, et al. Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells. Biol Sex Differ. 2014;5(1):18doi: 10.1186/s13293-014-0018-2.
Addis, R., Campesi, I., Fois, M., Capobianco, G., Dessole, S., Fenu, G., Montella, A., Cattaneo, M. G., Vicentini, L. M., & Franconi, F. (2014). Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells. Biology of sex differences, 5(1), 18. https://doi.org/10.1186/s13293-014-0018-2
Addis, Roberta, et al. "Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells." Biology of sex differences vol. 5,1 (2014): 18. doi: https://doi.org/10.1186/s13293-014-0018-2
Addis R, Campesi I, Fois M, Capobianco G, Dessole S, Fenu G, Montella A, Cattaneo MG, Vicentini LM, Franconi F. Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells. Biol Sex Differ. 2014 Dec 14;5(1):18. doi: 10.1186/s13293-014-0018-2. eCollection 2014. PMID: 25535548; PMCID: PMC4273493.
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RNA-Seq Study of Microbially Induced Hemocyte Transcripts from Larval Heliothis virescens (Lepidoptera: Noctuidae).

Insects

Shelby KS, Popham HJ.
PMID: 26466627
Insects. 2012 Aug 14;3(3):743-62. doi: 10.3390/insects3030743.

Larvae of the tobacco budworm are major polyphagous pests throughout the Americas. Development of effective microbial biopesticides for this and related noctuid pests has been stymied by the natural resistance mediated innate immune response. Hemocytes play an early and...

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Shelby KS, Popham HJ. RNA-Seq Study of Microbially Induced Hemocyte Transcripts from Larval Heliothis virescens (Lepidoptera: Noctuidae). Insects. 2012;3(3):743-62doi: 10.3390/insects3030743.
Shelby, K. S., & Popham, H. J. (2012). RNA-Seq Study of Microbially Induced Hemocyte Transcripts from Larval Heliothis virescens (Lepidoptera: Noctuidae). Insects, 3(3), 743-62. https://doi.org/10.3390/insects3030743
Shelby, Kent S, and Popham, Holly J R. "RNA-Seq Study of Microbially Induced Hemocyte Transcripts from Larval Heliothis virescens (Lepidoptera: Noctuidae)." Insects vol. 3,3 (2012): 743-62. doi: https://doi.org/10.3390/insects3030743
Shelby KS, Popham HJ. RNA-Seq Study of Microbially Induced Hemocyte Transcripts from Larval Heliothis virescens (Lepidoptera: Noctuidae). Insects. 2012 Aug 14;3(3):743-62. doi: 10.3390/insects3030743. PMID: 26466627; PMCID: PMC4553588.
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Intracellular Ca(2+) signaling: A novel player in the canonical mTOR-controlled autophagy pathway.

Communicative & integrative biology

Decuypere JP, Paudel RC, Parys J, Bultynck G.
PMID: 24265855
Commun Integr Biol. 2013 Sep 01;6(5):e25429. doi: 10.4161/cib.25429. Epub 2013 Jun 25.

Functional intracellular Ca(2+) signaling is essential for the upregulation of the canonical mTOR-controlled autophagy pathway triggered by rapamycin or by nutrient deprivation. Moreover, modifications in the Ca(2+)-signaling machinery coincide with autophagy stimulation. This results in enhanced intracellular Ca(2+) signaling...

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Decuypere JP, Paudel RC, Parys J, et al. Intracellular Ca(2+) signaling: A novel player in the canonical mTOR-controlled autophagy pathway. Commun Integr Biol. 2013;6(5):e25429doi: 10.4161/cib.25429.
Decuypere, J. P., Paudel, R. C., Parys, J., & Bultynck, G. (2013). Intracellular Ca(2+) signaling: A novel player in the canonical mTOR-controlled autophagy pathway. Communicative & integrative biology, 6(5), e25429. https://doi.org/10.4161/cib.25429
Decuypere, Jean-Paul, et al. "Intracellular Ca(2+) signaling: A novel player in the canonical mTOR-controlled autophagy pathway." Communicative & integrative biology vol. 6,5 (2013): e25429. doi: https://doi.org/10.4161/cib.25429
Decuypere JP, Paudel RC, Parys J, Bultynck G. Intracellular Ca(2+) signaling: A novel player in the canonical mTOR-controlled autophagy pathway. Commun Integr Biol. 2013 Sep 01;6(5):e25429. doi: 10.4161/cib.25429. Epub 2013 Jun 25. PMID: 24265855; PMCID: PMC3829968.
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Cell Death in the Developing Brain after Hypoxia-Ischemia.

Frontiers in cellular neuroscience

Thornton C, Leaw B, Mallard C, Nair S, Jinnai M, Hagberg H.
PMID: 28878624
Front Cell Neurosci. 2017 Aug 23;11:248. doi: 10.3389/fncel.2017.00248. eCollection 2017.

Perinatal insults such as hypoxia-ischemia induces secondary brain injury. In order to develop the next generation of neuroprotective therapies, we urgently need to understand the underlying molecular mechanisms leading to cell death. The cell death mechanisms have been shown...

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Thornton C, Leaw B, Mallard C, et al. Cell Death in the Developing Brain after Hypoxia-Ischemia. Front Cell Neurosci. 2017;11:248doi: 10.3389/fncel.2017.00248.
Thornton, C., Leaw, B., Mallard, C., Nair, S., Jinnai, M., & Hagberg, H. (2017). Cell Death in the Developing Brain after Hypoxia-Ischemia. Frontiers in cellular neuroscience, 11248. https://doi.org/10.3389/fncel.2017.00248
Thornton, Claire, et al. "Cell Death in the Developing Brain after Hypoxia-Ischemia." Frontiers in cellular neuroscience vol. 11 (2017): 248. doi: https://doi.org/10.3389/fncel.2017.00248
Thornton C, Leaw B, Mallard C, Nair S, Jinnai M, Hagberg H. Cell Death in the Developing Brain after Hypoxia-Ischemia. Front Cell Neurosci. 2017 Aug 23;11:248. doi: 10.3389/fncel.2017.00248. eCollection 2017. PMID: 28878624; PMCID: PMC5572386.
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Reversal of intramyocellular lipid accumulation by lipophagy and a p62-mediated pathway.

Cell death discovery

Lam T, Harmancey R, Vasquez H, Gilbert B, Patel N, Hariharan V, Lee A, Covey M, Taegtmeyer H.
PMID: 27625792
Cell Death Discov. 2016 Aug 22;2:16061. doi: 10.1038/cddiscovery.2016.61. eCollection 2016.

We have previously observed the reversal of lipid droplet deposition in skeletal muscle of morbidly obese patients following bariatric surgery. We now investigated whether activation of autophagy is the mechanism underlying this observation. For this purpose, we incubated rat...

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Lam T, Harmancey R, Vasquez H, et al. Reversal of intramyocellular lipid accumulation by lipophagy and a p62-mediated pathway. Cell Death Discov. 2016;2:16061doi: 10.1038/cddiscovery.2016.61.
Lam, T., Harmancey, R., Vasquez, H., Gilbert, B., Patel, N., Hariharan, V., Lee, A., Covey, M., & Taegtmeyer, H. (2016). Reversal of intramyocellular lipid accumulation by lipophagy and a p62-mediated pathway. Cell death discovery, 216061. https://doi.org/10.1038/cddiscovery.2016.61
Lam, T, et al. "Reversal of intramyocellular lipid accumulation by lipophagy and a p62-mediated pathway." Cell death discovery vol. 2 (2016): 16061. doi: https://doi.org/10.1038/cddiscovery.2016.61
Lam T, Harmancey R, Vasquez H, Gilbert B, Patel N, Hariharan V, Lee A, Covey M, Taegtmeyer H. Reversal of intramyocellular lipid accumulation by lipophagy and a p62-mediated pathway. Cell Death Discov. 2016 Aug 22;2:16061. doi: 10.1038/cddiscovery.2016.61. eCollection 2016. PMID: 27625792; PMCID: PMC4993124.
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Targeting LAMP2 in human cerebrospinal fluid with a combination of immunopurification and high resolution parallel reaction monitoring mass spectrometry.

Clinical proteomics

Sjödin S, Öhrfelt A, Brinkmalm G, Zetterberg H, Blennow K, Brinkmalm A.
PMID: 26924951
Clin Proteomics. 2016 Feb 25;13:4. doi: 10.1186/s12014-016-9104-2. eCollection 2016.

BACKGROUND: Alzheimer's disease is the most common form of dementia. An increasing body of evidence suggests that endo-lysosomal dysfunction is a pathogenic mechanism of Alzheimer's disease. Thus there is a potential for proteins involved in the normal function of...

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Sjödin S, Öhrfelt A, Brinkmalm G, et al. Targeting LAMP2 in human cerebrospinal fluid with a combination of immunopurification and high resolution parallel reaction monitoring mass spectrometry. Clin Proteomics. 2016;13:4doi: 10.1186/s12014-016-9104-2.
Sjödin, S., Öhrfelt, A., Brinkmalm, G., Zetterberg, H., Blennow, K., & Brinkmalm, A. (2016). Targeting LAMP2 in human cerebrospinal fluid with a combination of immunopurification and high resolution parallel reaction monitoring mass spectrometry. Clinical proteomics, 134. https://doi.org/10.1186/s12014-016-9104-2
Sjödin, Simon, et al. "Targeting LAMP2 in human cerebrospinal fluid with a combination of immunopurification and high resolution parallel reaction monitoring mass spectrometry." Clinical proteomics vol. 13 (2016): 4. doi: https://doi.org/10.1186/s12014-016-9104-2
Sjödin S, Öhrfelt A, Brinkmalm G, Zetterberg H, Blennow K, Brinkmalm A. Targeting LAMP2 in human cerebrospinal fluid with a combination of immunopurification and high resolution parallel reaction monitoring mass spectrometry. Clin Proteomics. 2016 Feb 25;13:4. doi: 10.1186/s12014-016-9104-2. eCollection 2016. PMID: 26924951; PMCID: PMC4768413.
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Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion.

Biology open

Kawano S, Torisu T, Esaki M, Torisu K, Matsuno Y, Kitazono T.
PMID: 28970230
Biol Open. 2017 Nov 15;6(11):1644-1653. doi: 10.1242/bio.027458.

Adhesion of cells to the extracellular matrix (ECM) via focal adhesions (FAs) is crucial for cell survival, migration, and differentiation. Although the regulation of FAs, including by integrins and the ECM, is important to cell behavior, how FAs are...

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Kawano S, Torisu T, Esaki M, et al. Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion. Biol Open. 2017;6(11):1644-1653doi: 10.1242/bio.027458.
Kawano, S., Torisu, T., Esaki, M., Torisu, K., Matsuno, Y., & Kitazono, T. (2017). Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion. Biology open, 6(11), 1644-1653. https://doi.org/10.1242/bio.027458
Kawano, Shinichi, et al. "Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion." Biology open vol. 6,11 (2017): 1644-1653. doi: https://doi.org/10.1242/bio.027458
Kawano S, Torisu T, Esaki M, Torisu K, Matsuno Y, Kitazono T. Autophagy promotes degradation of internalized collagen and regulates distribution of focal adhesions to suppress cell adhesion. Biol Open. 2017 Nov 15;6(11):1644-1653. doi: 10.1242/bio.027458. PMID: 28970230; PMCID: PMC5703610.
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Autophagy in Ischemic Livers: A Critical Role of Sirtuin 1/Mitofusin 2 Axis in Autophagy Induction.

Toxicological research

Chun SK, Go K, Yang MJ, Zendejas I, Behrns KE, Kim JS.
PMID: 26977257
Toxicol Res. 2016 Jan;32(1):35-46. doi: 10.5487/TR.2016.32.1.035. Epub 2016 Jan 31.

No-flow ischemia occurs during cardiac arrest, hemorrhagic shock, liver resection and transplantation. Recovery of blood flow and normal physiological pH, however, irreversibly injures the liver and other tissues. Although the liver has the powerful machinery for mitochondrial quality control,...

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Chun SK, Go K, Yang MJ, et al. Autophagy in Ischemic Livers: A Critical Role of Sirtuin 1/Mitofusin 2 Axis in Autophagy Induction. Toxicol Res. 2016;32(1):35-46doi: 10.5487/TR.2016.32.1.035.
Chun, S. K., Go, K., Yang, M. J., Zendejas, I., Behrns, K. E., & Kim, J. S. (2016). Autophagy in Ischemic Livers: A Critical Role of Sirtuin 1/Mitofusin 2 Axis in Autophagy Induction. Toxicological research, 32(1), 35-46. https://doi.org/10.5487/TR.2016.32.1.035
Chun, Sung Kook, et al. "Autophagy in Ischemic Livers: A Critical Role of Sirtuin 1/Mitofusin 2 Axis in Autophagy Induction." Toxicological research vol. 32,1 (2016): 35-46. doi: https://doi.org/10.5487/TR.2016.32.1.035
Chun SK, Go K, Yang MJ, Zendejas I, Behrns KE, Kim JS. Autophagy in Ischemic Livers: A Critical Role of Sirtuin 1/Mitofusin 2 Axis in Autophagy Induction. Toxicol Res. 2016 Jan;32(1):35-46. doi: 10.5487/TR.2016.32.1.035. Epub 2016 Jan 31. PMID: 26977257; PMCID: PMC4780240.
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Plant Cell Wall-Penetrable, Redox-Responsive Silica Nanoprobe for the Imaging of Starvation-Induced Vesicle Trafficking.

Analytical chemistry

Huang XC, Inoue-Aono Y, Moriyasu Y, Hsieh PY, Tu WM, Hsiao SC, Jane WN, Hsu HY.
PMID: 27673337
Anal Chem. 2016 Oct 18;88(20):10231-10236. doi: 10.1021/acs.analchem.6b02920. Epub 2016 Oct 06.

Autophagy is a self-protection process against reactive oxygen species (ROS). The intracellular level of ROS increased when cells were cultured under nutrient starvation. Antioxidants such as glutathione and ascorbic acid play an important role in ROS removal. However, the...

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Huang XC, Inoue-Aono Y, Moriyasu Y, et al. Plant Cell Wall-Penetrable, Redox-Responsive Silica Nanoprobe for the Imaging of Starvation-Induced Vesicle Trafficking. Anal Chem. 2016;88(20):10231-10236doi: 10.1021/acs.analchem.6b02920.
Huang, X. C., Inoue-Aono, Y., Moriyasu, Y., Hsieh, P. Y., Tu, W. M., Hsiao, S. C., Jane, W. N., & Hsu, H. Y. (2016). Plant Cell Wall-Penetrable, Redox-Responsive Silica Nanoprobe for the Imaging of Starvation-Induced Vesicle Trafficking. Analytical chemistry, 88(20), 10231-10236. https://doi.org/10.1021/acs.analchem.6b02920
Huang, Xin-Chun, et al. "Plant Cell Wall-Penetrable, Redox-Responsive Silica Nanoprobe for the Imaging of Starvation-Induced Vesicle Trafficking." Analytical chemistry vol. 88,20 (2016): 10231-10236. doi: https://doi.org/10.1021/acs.analchem.6b02920
Huang XC, Inoue-Aono Y, Moriyasu Y, Hsieh PY, Tu WM, Hsiao SC, Jane WN, Hsu HY. Plant Cell Wall-Penetrable, Redox-Responsive Silica Nanoprobe for the Imaging of Starvation-Induced Vesicle Trafficking. Anal Chem. 2016 Oct 18;88(20):10231-10236. doi: 10.1021/acs.analchem.6b02920. Epub 2016 Oct 06. PMID: 27673337.
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Correction: Inflammatory Stress on Autophagy in Peripheral Blood Mononuclear Cells from Patients with Alzheimer's Disease during 24 Months of Follow-Up.

PloS one

François A, Julian A, Ragot S, Dugast E, Blanchard L, Brishoual S, Terro F, Chassaing D, Page G, Paccalin M.
PMID: 26977934
PLoS One. 2016 Mar 15;11(3):e0143933. doi: 10.1371/journal.pone.0143933. eCollection 2016.

No abstract available.

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François A, Julian A, Ragot S, et al. Correction: Inflammatory Stress on Autophagy in Peripheral Blood Mononuclear Cells from Patients with Alzheimer's Disease during 24 Months of Follow-Up. PLoS One. 2016;11(3):e0143933doi: 10.1371/journal.pone.0143933.
François, A., Julian, A., Ragot, S., Dugast, E., Blanchard, L., Brishoual, S., Terro, F., Chassaing, D., Page, G., & Paccalin, M. (2016). Correction: Inflammatory Stress on Autophagy in Peripheral Blood Mononuclear Cells from Patients with Alzheimer's Disease during 24 Months of Follow-Up. PloS one, 11(3), e0143933. https://doi.org/10.1371/journal.pone.0143933
François, Arnaud, et al. "Correction: Inflammatory Stress on Autophagy in Peripheral Blood Mononuclear Cells from Patients with Alzheimer's Disease during 24 Months of Follow-Up." PloS one vol. 11,3 (2016): e0143933. doi: https://doi.org/10.1371/journal.pone.0143933
François A, Julian A, Ragot S, Dugast E, Blanchard L, Brishoual S, Terro F, Chassaing D, Page G, Paccalin M. Correction: Inflammatory Stress on Autophagy in Peripheral Blood Mononuclear Cells from Patients with Alzheimer's Disease during 24 Months of Follow-Up. PLoS One. 2016 Mar 15;11(3):e0143933. doi: 10.1371/journal.pone.0143933. eCollection 2016. PMID: 26977934; PMCID: PMC4792419.
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Inhibition of autophagy potentiates the proliferation inhibition activity of microRNA-7 in human hepatocellular carcinoma cells.

Oncology letters

Wang Y, Wang Q, Song J.
PMID: 28927113
Oncol Lett. 2017 Sep;14(3):3566-3572. doi: 10.3892/ol.2017.6573. Epub 2017 Jul 15.

MicroRNAs (miRNAs/miRs) are important molecules that are able to regulate multiple cellular processes in cancer cells. miR-7 has been previously identified as a tumor suppressive miRNA in several types of cancer. The aim of the present study was to...

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Wang Y, Wang Q, Song J. Inhibition of autophagy potentiates the proliferation inhibition activity of microRNA-7 in human hepatocellular carcinoma cells. Oncol Lett. 2017;14(3):3566-3572doi: 10.3892/ol.2017.6573.
Wang, Y., Wang, Q., & Song, J. (2017). Inhibition of autophagy potentiates the proliferation inhibition activity of microRNA-7 in human hepatocellular carcinoma cells. Oncology letters, 14(3), 3566-3572. https://doi.org/10.3892/ol.2017.6573
Wang, Yanna, et al. "Inhibition of autophagy potentiates the proliferation inhibition activity of microRNA-7 in human hepatocellular carcinoma cells." Oncology letters vol. 14,3 (2017): 3566-3572. doi: https://doi.org/10.3892/ol.2017.6573
Wang Y, Wang Q, Song J. Inhibition of autophagy potentiates the proliferation inhibition activity of microRNA-7 in human hepatocellular carcinoma cells. Oncol Lett. 2017 Sep;14(3):3566-3572. doi: 10.3892/ol.2017.6573. Epub 2017 Jul 15. PMID: 28927113; PMCID: PMC5588049.
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