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Cigarette smoke induces cell motility via platelet-activating factor accumulation in breast cancer cells: a potential mechanism for metastatic disease.

Physiological reports

Kispert S, Marentette J, McHowat J.
PMID: 25802360
Physiol Rep. 2015 Mar;3(3). doi: 10.14814/phy2.12318. Epub 2015 Mar 22.

Most cancer deaths are a result of metastasis rather than the primary tumor. Although cigarette smoking has been determined as a risk factor for several cancers, its role in metastasis has not been studied in detail. We propose that...

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Kispert S, Marentette J, McHowat J. Cigarette smoke induces cell motility via platelet-activating factor accumulation in breast cancer cells: a potential mechanism for metastatic disease. Physiol Rep. 2015;3(3)doi: 10.14814/phy2.12318.
Kispert, S., Marentette, J., & McHowat, J. (2015). Cigarette smoke induces cell motility via platelet-activating factor accumulation in breast cancer cells: a potential mechanism for metastatic disease. Physiological reports, 3(3), . https://doi.org/10.14814/phy2.12318
Kispert, Shannon, et al. "Cigarette smoke induces cell motility via platelet-activating factor accumulation in breast cancer cells: a potential mechanism for metastatic disease." Physiological reports vol. 3,3 (2015). doi: https://doi.org/10.14814/phy2.12318
Kispert S, Marentette J, McHowat J. Cigarette smoke induces cell motility via platelet-activating factor accumulation in breast cancer cells: a potential mechanism for metastatic disease. Physiol Rep. 2015 Mar;3(3). doi: 10.14814/phy2.12318. Epub 2015 Mar 22. PMID: 25802360; PMCID: PMC4393154.
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A Possible Role for Platelet-Activating Factor Receptor in Amyotrophic Lateral Sclerosis Treatment.

Frontiers in neurology

Briones MRS, Snyder AM, Ferreira RC, Neely EB, Connor JR, Broach JR.
PMID: 29472887
Front Neurol. 2018 Feb 06;9:39. doi: 10.3389/fneur.2018.00039. eCollection 2018.

Amyotrophic lateral sclerosis (ALS) is the third most prevalent neurodegenerative disease affecting upper and lower motor neurons. An important pathway that may lead to motor neuron degeneration is neuroinflammation. Cerebrospinal Fluids of ALS patients have increased levels of the...

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Briones MRS, Snyder AM, Ferreira RC, et al. A Possible Role for Platelet-Activating Factor Receptor in Amyotrophic Lateral Sclerosis Treatment. Front Neurol. 2018;9:39doi: 10.3389/fneur.2018.00039.
Briones, M. R. S., Snyder, A. M., Ferreira, R. C., Neely, E. B., Connor, J. R., & Broach, J. R. (2018). A Possible Role for Platelet-Activating Factor Receptor in Amyotrophic Lateral Sclerosis Treatment. Frontiers in neurology, 939. https://doi.org/10.3389/fneur.2018.00039
Briones, Marcelo R S, et al. "A Possible Role for Platelet-Activating Factor Receptor in Amyotrophic Lateral Sclerosis Treatment." Frontiers in neurology vol. 9 (2018): 39. doi: https://doi.org/10.3389/fneur.2018.00039
Briones MRS, Snyder AM, Ferreira RC, Neely EB, Connor JR, Broach JR. A Possible Role for Platelet-Activating Factor Receptor in Amyotrophic Lateral Sclerosis Treatment. Front Neurol. 2018 Feb 06;9:39. doi: 10.3389/fneur.2018.00039. eCollection 2018. PMID: 29472887; PMCID: PMC5810282.
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A Review on Platelet Activating Factor Inhibitors: Could a New Class of Potent Metal-Based Anti-Inflammatory Drugs Induce Anticancer Properties?.

Bioinorganic chemistry and applications

Papakonstantinou VD, Lagopati N, Tsilibary EC, Demopoulos CA, Philippopoulos AI.
PMID: 28458618
Bioinorg Chem Appl. 2017;2017:6947034. doi: 10.1155/2017/6947034. Epub 2017 Mar 28.

In this minireview, we refer to recent results as far as the Platelet Activating Factor (PAF) inhibitors are concerned. At first, results of organic compounds (natural and synthetic ones and specific and nonspecific) as inhibitors of PAF are reported....

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Papakonstantinou VD, Lagopati N, Tsilibary EC, et al. A Review on Platelet Activating Factor Inhibitors: Could a New Class of Potent Metal-Based Anti-Inflammatory Drugs Induce Anticancer Properties?. Bioinorg Chem Appl. 2017;2017:6947034doi: 10.1155/2017/6947034.
Papakonstantinou, V. D., Lagopati, N., Tsilibary, E. C., Demopoulos, C. A., & Philippopoulos, A. I. (2017). A Review on Platelet Activating Factor Inhibitors: Could a New Class of Potent Metal-Based Anti-Inflammatory Drugs Induce Anticancer Properties?. Bioinorganic chemistry and applications, 20176947034. https://doi.org/10.1155/2017/6947034
Papakonstantinou, Vasiliki D, et al. "A Review on Platelet Activating Factor Inhibitors: Could a New Class of Potent Metal-Based Anti-Inflammatory Drugs Induce Anticancer Properties?." Bioinorganic chemistry and applications vol. 2017 (2017): 6947034. doi: https://doi.org/10.1155/2017/6947034
Papakonstantinou VD, Lagopati N, Tsilibary EC, Demopoulos CA, Philippopoulos AI. A Review on Platelet Activating Factor Inhibitors: Could a New Class of Potent Metal-Based Anti-Inflammatory Drugs Induce Anticancer Properties?. Bioinorg Chem Appl. 2017;2017:6947034. doi: 10.1155/2017/6947034. Epub 2017 Mar 28. PMID: 28458618; PMCID: PMC5387815.
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Associations of platelet-activating factor acetylhydrolase gene polymorphisms with risk of ischemic stroke.

Biomedical reports

Ma Y.
PMID: 26893847
Biomed Rep. 2016 Feb;4(2):246-250. doi: 10.3892/br.2015.560. Epub 2015 Dec 21.

Platelet-activating factor acetylhydrolase (PAF-AH) has an important function in the pathogenesis of ischemic stroke. The aim of the present study was to investigate the correlation between the variation of polymorphisms (R92H and V279F) in PAF-AH and ischemic stroke. A...

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Ma Y. Associations of platelet-activating factor acetylhydrolase gene polymorphisms with risk of ischemic stroke. Biomed Rep. 2015;4(2):246-250doi: 10.3892/br.2015.560.
Ma, Y. (2016). Associations of platelet-activating factor acetylhydrolase gene polymorphisms with risk of ischemic stroke. Biomedical reports, 4(2), 246-250. https://doi.org/10.3892/br.2015.560
Ma, Yongsheng. "Associations of platelet-activating factor acetylhydrolase gene polymorphisms with risk of ischemic stroke." Biomedical reports vol. 4,2 (2016): 246-250. doi: https://doi.org/10.3892/br.2015.560
Ma Y. Associations of platelet-activating factor acetylhydrolase gene polymorphisms with risk of ischemic stroke. Biomed Rep. 2016 Feb;4(2):246-250. doi: 10.3892/br.2015.560. Epub 2015 Dec 21. PMID: 26893847; PMCID: PMC4734079.
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Platelet-activating factor synthesis and receptor-mediated signaling are downregulated in ovine newborn lungs: relevance in postnatal pulmonary adaptation and persistent pulmonary hypertension of the newborn.

Journal of developmental origins of health and disease

Renteria LS, Cruz E, Ibe BO.
PMID: 24924225
J Dev Orig Health Dis. 2013 Dec;4(6):458-69. doi: 10.1017/S2040174413000366.

Platelet-activating factor (PAF) is a phospholipid with a wide range of biological activities. We studied PAF metabolism and PAF receptor (PAFR) signaling in perinatal ovine lungs to understand PAF's role in transition of the perinatal pulmonary hemodynamics and pathophysiology...

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Renteria LS, Cruz E, Ibe BO. Platelet-activating factor synthesis and receptor-mediated signaling are downregulated in ovine newborn lungs: relevance in postnatal pulmonary adaptation and persistent pulmonary hypertension of the newborn. J Dev Orig Health Dis. 2013;4(6):458-69doi: 10.1017/S2040174413000366.
Renteria, L. S., Cruz, E., & Ibe, B. O. (2013). Platelet-activating factor synthesis and receptor-mediated signaling are downregulated in ovine newborn lungs: relevance in postnatal pulmonary adaptation and persistent pulmonary hypertension of the newborn. Journal of developmental origins of health and disease, 4(6), 458-69. https://doi.org/10.1017/S2040174413000366
Renteria, L S, et al. "Platelet-activating factor synthesis and receptor-mediated signaling are downregulated in ovine newborn lungs: relevance in postnatal pulmonary adaptation and persistent pulmonary hypertension of the newborn." Journal of developmental origins of health and disease vol. 4,6 (2013): 458-69. doi: https://doi.org/10.1017/S2040174413000366
Renteria LS, Cruz E, Ibe BO. Platelet-activating factor synthesis and receptor-mediated signaling are downregulated in ovine newborn lungs: relevance in postnatal pulmonary adaptation and persistent pulmonary hypertension of the newborn. J Dev Orig Health Dis. 2013 Dec;4(6):458-69. doi: 10.1017/S2040174413000366. PMID: 24924225.
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Low High-Density Lipoprotein and Risk of Myocardial Infarction.

Clinical Medicine Insights. Cardiology

Ramirez A, Hu PP.
PMID: 26692765
Clin Med Insights Cardiol. 2015 Dec 03;9:113-7. doi: 10.4137/CMC.S26624. eCollection 2015.

Low HDL is an independent risk factor for myocardial infarction. This paper reviews our current understanding of HDL, HDL structure and function, HDL subclasses, the relationship of low HDL with myocardial infarction, HDL targeted therapy, and clinical trials and...

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Ramirez A, Hu PP. Low High-Density Lipoprotein and Risk of Myocardial Infarction. Clin Med Insights Cardiol. 2015;9:113-7doi: 10.4137/CMC.S26624.
Ramirez, A., & Hu, P. P. (2015). Low High-Density Lipoprotein and Risk of Myocardial Infarction. Clinical Medicine Insights. Cardiology, 9113-7. https://doi.org/10.4137/CMC.S26624
Ramirez, A, and Hu, P P. "Low High-Density Lipoprotein and Risk of Myocardial Infarction." Clinical Medicine Insights. Cardiology vol. 9 (2015): 113-7. doi: https://doi.org/10.4137/CMC.S26624
Ramirez A, Hu PP. Low High-Density Lipoprotein and Risk of Myocardial Infarction. Clin Med Insights Cardiol. 2015 Dec 03;9:113-7. doi: 10.4137/CMC.S26624. eCollection 2015. PMID: 26692765; PMCID: PMC4670046.
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Tools for Investigating Functional Interactions Between Lipid-Derived Autacoids and their Receptors.

American journal of therapeutics

Lerner MR, Golovyan L, Graminski GF, Harris K, Huang L, Jayawickreme CK, Karne S, McClintock TS, Potenza MN, Roby-Shemkovitz A, Quillan M.
PMID: 11862262
Am J Ther. 1996 Apr;3(4):280-286. doi: 10.1097/00045391-199604000-00005.

A method for rapidly evaluating functional interactions between ligands and G-protein--coupled receptors has been developed. The technology is based on the ability of animals to change color by controlling the position of pigmented organelles within skin cells called melanophores....

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Lerner MR, Golovyan L, Graminski GF, et al. Tools for Investigating Functional Interactions Between Lipid-Derived Autacoids and their Receptors. Am J Ther. 1996;3(4):280-286doi: 10.1097/00045391-199604000-00005.
Lerner, M. R., Golovyan, L., Graminski, G. F., Harris, K., Huang, L., Jayawickreme, C. K., Karne, S., McClintock, T. S., Potenza, M. N., Roby-Shemkovitz, A., & Quillan, M. (1996). Tools for Investigating Functional Interactions Between Lipid-Derived Autacoids and their Receptors. American journal of therapeutics, 3(4), 280-286. https://doi.org/10.1097/00045391-199604000-00005
Lerner, Michael R., et al. "Tools for Investigating Functional Interactions Between Lipid-Derived Autacoids and their Receptors." American journal of therapeutics vol. 3,4 (1996): 280-286. doi: https://doi.org/10.1097/00045391-199604000-00005
Lerner MR, Golovyan L, Graminski GF, Harris K, Huang L, Jayawickreme CK, Karne S, McClintock TS, Potenza MN, Roby-Shemkovitz A, Quillan M. Tools for Investigating Functional Interactions Between Lipid-Derived Autacoids and their Receptors. Am J Ther. 1996 Apr;3(4):280-286. doi: 10.1097/00045391-199604000-00005. PMID: 11862262.
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Pathogenetic effects of platelet activating factor on enterogenic endotoxemia after burn.

World journal of gastroenterology

Yu PW, Xiao GX, Fu WL, Yuan JC, Zhou LX, Qin XJ.
PMID: 11819627
World J Gastroenterol. 2000 Jun;6(3):451-453. doi: 10.3748/wjg.v6.i3.451.

No abstract available.

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Yu PW, Xiao GX, Fu WL, et al. Pathogenetic effects of platelet activating factor on enterogenic endotoxemia after burn. World J Gastroenterol. 2000;6(3):451-453doi: 10.3748/wjg.v6.i3.451.
Yu, P. W., Xiao, G. X., Fu, W. L., Yuan, J. C., Zhou, L. X., & Qin, X. J. (2000). Pathogenetic effects of platelet activating factor on enterogenic endotoxemia after burn. World journal of gastroenterology, 6(3), 451-453. https://doi.org/10.3748/wjg.v6.i3.451
Yu, Pei-Wu, et al. "Pathogenetic effects of platelet activating factor on enterogenic endotoxemia after burn." World journal of gastroenterology vol. 6,3 (2000): 451-453. doi: https://doi.org/10.3748/wjg.v6.i3.451
Yu PW, Xiao GX, Fu WL, Yuan JC, Zhou LX, Qin XJ. Pathogenetic effects of platelet activating factor on enterogenic endotoxemia after burn. World J Gastroenterol. 2000 Jun;6(3):451-453. doi: 10.3748/wjg.v6.i3.451. PMID: 11819627; PMCID: PMC4688781.
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Production of platelet-activating factor during focal cerebral ischemia and reperfusion in the rat.

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association

Umemura A, Yamada K, Mabe H, Nagai H.
PMID: 17895040
J Stroke Cerebrovasc Dis. 1997 Oct-Nov;6(6):394-7. doi: 10.1016/s1052-3057(97)80040-7.

Platelet-activating factor (PAF) is a phospholipid mediator implicated in a diverse range of pathological processes. Beneficial effects of PAF antagonists have been shown in various models of central nervous system ischemia. In this study, we evaluated the production of...

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Umemura A, Yamada K, Mabe H, et al. Production of platelet-activating factor during focal cerebral ischemia and reperfusion in the rat. J Stroke Cerebrovasc Dis. 1997;6(6):394-7doi: 10.1016/s1052-3057(97)80040-7.
Umemura, A., Yamada, K., Mabe, H., & Nagai, H. (1997). Production of platelet-activating factor during focal cerebral ischemia and reperfusion in the rat. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association, 6(6), 394-7. https://doi.org/10.1016/s1052-3057(97)80040-7
Umemura, A, et al. "Production of platelet-activating factor during focal cerebral ischemia and reperfusion in the rat." Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association vol. 6,6 (1997): 394-7. doi: https://doi.org/10.1016/s1052-3057(97)80040-7
Umemura A, Yamada K, Mabe H, Nagai H. Production of platelet-activating factor during focal cerebral ischemia and reperfusion in the rat. J Stroke Cerebrovasc Dis. 1997 Oct-Nov;6(6):394-7. doi: 10.1016/s1052-3057(97)80040-7. PMID: 17895040.
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Secreted phosphatase activities in trypanosomatid parasites of plants modulated by platelet-activating factor.

Phytopathology

Dutra PM, Dias FA, Santos MA, Rodrigues CO, Romeiro A, Attias M, De Souza W, Lopes AH, Meyer-Fernandes JR.
PMID: 18943854
Phytopathology. 2001 Apr;91(4):408-14. doi: 10.1094/PHYTO.2001.91.4.408.

ABSTRACT The secreted phosphatase activities of two trypanosomatid parasites were characterized and compared with supernatants of living cells. The plant parasite Phytomonas françai and the phytophagous hemipteran parasite Herpetomonas sp. hydrolyzed p-nitrophenylphosphate at a rate of 15.54 and 6.51...

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Dutra PM, Dias FA, Santos MA, et al. Secreted phosphatase activities in trypanosomatid parasites of plants modulated by platelet-activating factor. Phytopathology. 2001;91(4):408-14doi: 10.1094/PHYTO.2001.91.4.408.
Dutra, P. M., Dias, F. A., Santos, M. A., Rodrigues, C. O., Romeiro, A., Attias, M., De Souza, W., Lopes, A. H., & Meyer-Fernandes, J. R. (2001). Secreted phosphatase activities in trypanosomatid parasites of plants modulated by platelet-activating factor. Phytopathology, 91(4), 408-14. https://doi.org/10.1094/PHYTO.2001.91.4.408
Dutra, P M, et al. "Secreted phosphatase activities in trypanosomatid parasites of plants modulated by platelet-activating factor." Phytopathology vol. 91,4 (2001): 408-14. doi: https://doi.org/10.1094/PHYTO.2001.91.4.408
Dutra PM, Dias FA, Santos MA, Rodrigues CO, Romeiro A, Attias M, De Souza W, Lopes AH, Meyer-Fernandes JR. Secreted phosphatase activities in trypanosomatid parasites of plants modulated by platelet-activating factor. Phytopathology. 2001 Apr;91(4):408-14. doi: 10.1094/PHYTO.2001.91.4.408. PMID: 18943854.
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Effect of Fluticasone propionate Aqueous Nasal Spray Treatment on Platelet Activating Factor and Eicosanoid Production By nasal Mucosa in Patients with A house Dust Mite Allergy.

Mediators of inflammation

Garrelds IM, de Graaf-In 't Veld T, Jansen AP, van Wijk RG, Zijlstra FJ.
PMID: 18475584
Mediators Inflamm. 1994;3(5):381-5. doi: 10.1155/S0962935194000530.

The relationship between the release of platelet activating factor (PAF), leukotriene C(4)/D(4)/E(E) (LTC(4)/D(4)/E(4)) and prostaglandin D(2) (PGD(2)) from nasal mucosa in vivo was examined in 24 rhinitis patients allergic to the house dust mite (HDM). During a double blind...

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Garrelds IM, de Graaf-In 't Veld T, Jansen AP, et al. Effect of Fluticasone propionate Aqueous Nasal Spray Treatment on Platelet Activating Factor and Eicosanoid Production By nasal Mucosa in Patients with A house Dust Mite Allergy. Mediators Inflamm. 1994;3(5):381-5doi: 10.1155/S0962935194000530.
Garrelds, I. M., de Graaf-In 't Veld, T., Jansen, A. P., van Wijk, R. G., & Zijlstra, F. J. (1994). Effect of Fluticasone propionate Aqueous Nasal Spray Treatment on Platelet Activating Factor and Eicosanoid Production By nasal Mucosa in Patients with A house Dust Mite Allergy. Mediators of inflammation, 3(5), 381-5. https://doi.org/10.1155/S0962935194000530
Garrelds, I M, et al. "Effect of Fluticasone propionate Aqueous Nasal Spray Treatment on Platelet Activating Factor and Eicosanoid Production By nasal Mucosa in Patients with A house Dust Mite Allergy." Mediators of inflammation vol. 3,5 (1994): 381-5. doi: https://doi.org/10.1155/S0962935194000530
Garrelds IM, de Graaf-In 't Veld T, Jansen AP, van Wijk RG, Zijlstra FJ. Effect of Fluticasone propionate Aqueous Nasal Spray Treatment on Platelet Activating Factor and Eicosanoid Production By nasal Mucosa in Patients with A house Dust Mite Allergy. Mediators Inflamm. 1994;3(5):381-5. doi: 10.1155/S0962935194000530. PMID: 18475584; PMCID: PMC2365576.
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The effect of lysolecithin on prostanoid and platelet-activating factor formation by human gall-bladder mucosal cells.

Mediators of inflammation

Nag MK, Deshpande YG, Beck D, Li A, Kaminski DL.
PMID: 18475621
Mediators Inflamm. 1995;4(2):90-4. doi: 10.1155/S0962935195000147.

It has been demonstrated that lysolecithin (lysophosphatidyl choline, LPC) produces experimental cholecystitis in cats mediated by arachidonic acid metabolites. LPC is a cytolytic agent that has been postulated as a contributing factor in the development of cholecystitis in humans....

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Nag MK, Deshpande YG, Beck D, et al. The effect of lysolecithin on prostanoid and platelet-activating factor formation by human gall-bladder mucosal cells. Mediators Inflamm. 1995;4(2):90-4doi: 10.1155/S0962935195000147.
Nag, M. K., Deshpande, Y. G., Beck, D., Li, A., & Kaminski, D. L. (1995). The effect of lysolecithin on prostanoid and platelet-activating factor formation by human gall-bladder mucosal cells. Mediators of inflammation, 4(2), 90-4. https://doi.org/10.1155/S0962935195000147
Nag, M K, et al. "The effect of lysolecithin on prostanoid and platelet-activating factor formation by human gall-bladder mucosal cells." Mediators of inflammation vol. 4,2 (1995): 90-4. doi: https://doi.org/10.1155/S0962935195000147
Nag MK, Deshpande YG, Beck D, Li A, Kaminski DL. The effect of lysolecithin on prostanoid and platelet-activating factor formation by human gall-bladder mucosal cells. Mediators Inflamm. 1995;4(2):90-4. doi: 10.1155/S0962935195000147. PMID: 18475621; PMCID: PMC2365627.
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Showing 1 to 12 of 366 entries