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Showing 1 to 12 of 33 entries
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Modulation of Autophagy by Sorafenib: Effects on Treatment Response.

Frontiers in pharmacology

Prieto-Domínguez N, Ordóñez R, Fernández A, García-Palomo A, Muntané J, González-Gallego J, Mauriz JL.
PMID: 27375485
Front Pharmacol. 2016 Jun 08;7:151. doi: 10.3389/fphar.2016.00151. eCollection 2016.

The multikinase inhibitor sorafenib is, at present, the only drug approved for the treatment of hepatocellular carcinoma (HCC), one of the most lethal types of cancer worldwide. However, the increase in the number of sorafenib tumor resistant cells reduces...

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Prieto-Domínguez N, Ordóñez R, Fernández A, et al. Modulation of Autophagy by Sorafenib: Effects on Treatment Response. Front Pharmacol. 2016;7:151doi: 10.3389/fphar.2016.00151.
Prieto-Domínguez, N., Ordóñez, R., Fernández, A., García-Palomo, A., Muntané, J., González-Gallego, J., & Mauriz, J. L. (2016). Modulation of Autophagy by Sorafenib: Effects on Treatment Response. Frontiers in pharmacology, 7151. https://doi.org/10.3389/fphar.2016.00151
Prieto-Domínguez, Nestor, et al. "Modulation of Autophagy by Sorafenib: Effects on Treatment Response." Frontiers in pharmacology vol. 7 (2016): 151. doi: https://doi.org/10.3389/fphar.2016.00151
Prieto-Domínguez N, Ordóñez R, Fernández A, García-Palomo A, Muntané J, González-Gallego J, Mauriz JL. Modulation of Autophagy by Sorafenib: Effects on Treatment Response. Front Pharmacol. 2016 Jun 08;7:151. doi: 10.3389/fphar.2016.00151. eCollection 2016. PMID: 27375485; PMCID: PMC4896953.
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Oxidative stress and cell damage in a model of precancerous lesions and advanced hepatocellular carcinoma in rats.

Toxicology reports

Moreira AJ, Rodrigues G, Bona S, Cerski CT, Marroni CA, Mauriz JL, González-Gallego J, Marroni NP.
PMID: 28962366
Toxicol Rep. 2014 Nov 28;2:333-340. doi: 10.1016/j.toxrep.2014.11.015. eCollection 2015.

Hepatocellular carcinoma (HCC) is the third most frequent cause of cancer deaths throughout the world. This study was aimed to analyze oxidative stress and cell damage in a multistage model of liver carcinogenesis induced by diethylnitrosamine (DEN) in rats....

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Moreira AJ, Rodrigues G, Bona S, et al. Oxidative stress and cell damage in a model of precancerous lesions and advanced hepatocellular carcinoma in rats. Toxicol Rep. 2014;2:333-340doi: 10.1016/j.toxrep.2014.11.015.
Moreira, A. J., Rodrigues, G., Bona, S., Cerski, C. T., Marroni, C. A., Mauriz, J. L., González-Gallego, J., & Marroni, N. P. (2015). Oxidative stress and cell damage in a model of precancerous lesions and advanced hepatocellular carcinoma in rats. Toxicology reports, 2333-340. https://doi.org/10.1016/j.toxrep.2014.11.015
Moreira, Andrea J, et al. "Oxidative stress and cell damage in a model of precancerous lesions and advanced hepatocellular carcinoma in rats." Toxicology reports vol. 2 (2015): 333-340. doi: https://doi.org/10.1016/j.toxrep.2014.11.015
Moreira AJ, Rodrigues G, Bona S, Cerski CT, Marroni CA, Mauriz JL, González-Gallego J, Marroni NP. Oxidative stress and cell damage in a model of precancerous lesions and advanced hepatocellular carcinoma in rats. Toxicol Rep. 2014 Nov 28;2:333-340. doi: 10.1016/j.toxrep.2014.11.015. eCollection 2015. PMID: 28962366; PMCID: PMC5598147.
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Effects of a 4-week eccentric training program on the repeated bout effect in young active women.

Journal of sports science & medicine

Fernandez-Gonzalo R, Bresciani G, de Souza-Teixeira F, Hernandez-Murua JA, Jimenez-Jimenez R, Gonzalez-Gallego J, de Paz JA.
PMID: 24149560
J Sports Sci Med. 2011 Dec 01;10(4):692-9. eCollection 2011.

The aim of this study was to analyze the responses of women to the repeated bout effect (RBE) and to a short eccentric training program. Twenty-four young females were randomly assigned to a training group (TG, n = 14)...

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Fernandez-Gonzalo R, Bresciani G, de Souza-Teixeira F, et al. Effects of a 4-week eccentric training program on the repeated bout effect in young active women. J Sports Sci Med. 2011;10(4):692-9
Fernandez-Gonzalo, R., Bresciani, G., de Souza-Teixeira, F., Hernandez-Murua, J. A., Jimenez-Jimenez, R., Gonzalez-Gallego, J., & de Paz, J. A. (2011). Effects of a 4-week eccentric training program on the repeated bout effect in young active women. Journal of sports science & medicine, 10(4), 692-9.
Fernandez-Gonzalo, Rodrigo, et al. "Effects of a 4-week eccentric training program on the repeated bout effect in young active women." Journal of sports science & medicine vol. 10,4 (2011): 692-9.
Fernandez-Gonzalo R, Bresciani G, de Souza-Teixeira F, Hernandez-Murua JA, Jimenez-Jimenez R, Gonzalez-Gallego J, de Paz JA. Effects of a 4-week eccentric training program on the repeated bout effect in young active women. J Sports Sci Med. 2011 Dec 01;10(4):692-9. eCollection 2011. PMID: 24149560; PMCID: PMC3761500.
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Melatonin Attenuates Dysregulation of the Circadian Clock Pathway in Mice With CCl.

Frontiers in pharmacology

González-Fernández B, Sánchez DI, Crespo I, San-Miguel B, de Urbina JO, González-Gallego J, Tuñón MJ.
PMID: 29892224
Front Pharmacol. 2018 May 28;9:556. doi: 10.3389/fphar.2018.00556. eCollection 2018.

Dysregulation of the circadian clock machinery is a critical mechanism in the pathogenesis of fibrosis. This study aimed to investigate whether the antifibrotic effect of melatonin is associated with attenuation of circadian clock pathway disturbances in mice treated with...

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González-Fernández B, Sánchez DI, Crespo I, et al. Melatonin Attenuates Dysregulation of the Circadian Clock Pathway in Mice With CCl. Front Pharmacol. 2018;9:556doi: 10.3389/fphar.2018.00556.
González-Fernández, B., Sánchez, D. I., Crespo, I., San-Miguel, B., de Urbina, J. O., González-Gallego, J., & Tuñón, M. J. (2018). Melatonin Attenuates Dysregulation of the Circadian Clock Pathway in Mice With CCl. Frontiers in pharmacology, 9556. https://doi.org/10.3389/fphar.2018.00556
González-Fernández, Bárbara, et al. "Melatonin Attenuates Dysregulation of the Circadian Clock Pathway in Mice With CCl." Frontiers in pharmacology vol. 9 (2018): 556. doi: https://doi.org/10.3389/fphar.2018.00556
González-Fernández B, Sánchez DI, Crespo I, San-Miguel B, de Urbina JO, González-Gallego J, Tuñón MJ. Melatonin Attenuates Dysregulation of the Circadian Clock Pathway in Mice With CCl. Front Pharmacol. 2018 May 28;9:556. doi: 10.3389/fphar.2018.00556. eCollection 2018. PMID: 29892224; PMCID: PMC5985434.
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Melatonin enhances sorafenib actions in human hepatocarcinoma cells by inhibiting mTORC1/p70S6K/HIF-1α and hypoxia-mediated mitophagy.

Oncotarget

Prieto-Domínguez N, Méndez-Blanco C, Carbajo-Pescador S, Fondevila F, García-Palomo A, González-Gallego J, Mauriz JL.
PMID: 29207653
Oncotarget. 2017 Aug 24;8(53):91402-91414. doi: 10.18632/oncotarget.20592. eCollection 2017 Oct 31.

The antiangiogenic effects of sustained sorafenib treatment in hepatocellular carcinoma (HCC) lead to the occurrence of hypoxia-mediated drug resistance resulting in low therapy efficiency and negative outcomes. Combined treatments with coadjuvant compounds to target the hypoxia-inducible factor-1α (HIF-1α) represent...

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Prieto-Domínguez N, Méndez-Blanco C, Carbajo-Pescador S, et al. Melatonin enhances sorafenib actions in human hepatocarcinoma cells by inhibiting mTORC1/p70S6K/HIF-1α and hypoxia-mediated mitophagy. Oncotarget. 2017;8(53):91402-91414doi: 10.18632/oncotarget.20592.
Prieto-Domínguez, N., Méndez-Blanco, C., Carbajo-Pescador, S., Fondevila, F., García-Palomo, A., González-Gallego, J., & Mauriz, J. L. (2017). Melatonin enhances sorafenib actions in human hepatocarcinoma cells by inhibiting mTORC1/p70S6K/HIF-1α and hypoxia-mediated mitophagy. Oncotarget, 8(53), 91402-91414. https://doi.org/10.18632/oncotarget.20592
Prieto-Domínguez, Néstor, et al. "Melatonin enhances sorafenib actions in human hepatocarcinoma cells by inhibiting mTORC1/p70S6K/HIF-1α and hypoxia-mediated mitophagy." Oncotarget vol. 8,53 (2017): 91402-91414. doi: https://doi.org/10.18632/oncotarget.20592
Prieto-Domínguez N, Méndez-Blanco C, Carbajo-Pescador S, Fondevila F, García-Palomo A, González-Gallego J, Mauriz JL. Melatonin enhances sorafenib actions in human hepatocarcinoma cells by inhibiting mTORC1/p70S6K/HIF-1α and hypoxia-mediated mitophagy. Oncotarget. 2017 Aug 24;8(53):91402-91414. doi: 10.18632/oncotarget.20592. eCollection 2017 Oct 31. PMID: 29207653; PMCID: PMC5710933.
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Comparison of Flywheel and Pneumatic Training on Hypertrophy, Strength, and Power in Professional Handball Players.

Research quarterly for exercise and sport

Maroto-Izquierdo S, McBride JM, Gonzalez-Diez N, García-López D, González-Gallego J, de Paz JA.
PMID: 32669052
Res Q Exerc Sport. 2020 Jul 15;1-15. doi: 10.1080/02701367.2020.1762836. Epub 2020 Jul 15.

No abstract available.

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Maroto-Izquierdo S, McBride JM, Gonzalez-Diez N, et al. Comparison of Flywheel and Pneumatic Training on Hypertrophy, Strength, and Power in Professional Handball Players. Res Q Exerc Sport. 2020;1-15doi: 10.1080/02701367.2020.1762836.
Maroto-Izquierdo, S., McBride, J. M., Gonzalez-Diez, N., García-López, D., González-Gallego, J., & de Paz, J. A. (2020). Comparison of Flywheel and Pneumatic Training on Hypertrophy, Strength, and Power in Professional Handball Players. Research quarterly for exercise and sport, 1-15. https://doi.org/10.1080/02701367.2020.1762836
Maroto-Izquierdo, Sergio, et al. "Comparison of Flywheel and Pneumatic Training on Hypertrophy, Strength, and Power in Professional Handball Players." Research quarterly for exercise and sport vol. (2020): 1-15. doi: https://doi.org/10.1080/02701367.2020.1762836
Maroto-Izquierdo S, McBride JM, Gonzalez-Diez N, García-López D, González-Gallego J, de Paz JA. Comparison of Flywheel and Pneumatic Training on Hypertrophy, Strength, and Power in Professional Handball Players. Res Q Exerc Sport. 2020 Jul 15;1-15. doi: 10.1080/02701367.2020.1762836. Epub 2020 Jul 15. PMID: 32669052.
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Association of FOXO3 Expression with Tumor Pathogenesis, Prognosis and Clinicopathological Features in Hepatocellular Carcinoma: A Systematic Review with Meta-Analysis.

Cancers

Fondevila F, Fernández-Palanca P, Méndez-Blanco C, Payo-Serafín T, Lozano E, Marin JJG, González-Gallego J, Mauriz JL.
PMID: 34771514
Cancers (Basel). 2021 Oct 26;13(21). doi: 10.3390/cancers13215349.

Forkhead box O3 (FOXO3), an essential transcription factor related to liver disease, has been linked to cancer progression. The most frequent primary liver tumor, hepatocellular carcinoma (HCC), has an elevated mortality rate and patient outcomes remain very poor. Here,...

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Fondevila F, Fernández-Palanca P, Méndez-Blanco C, et al. Association of FOXO3 Expression with Tumor Pathogenesis, Prognosis and Clinicopathological Features in Hepatocellular Carcinoma: A Systematic Review with Meta-Analysis. Cancers (Basel). 2021;13(21)doi: 10.3390/cancers13215349.
Fondevila, F., Fernández-Palanca, P., Méndez-Blanco, C., Payo-Serafín, T., Lozano, E., Marin, J. J. G., González-Gallego, J., & Mauriz, J. L. (2021). Association of FOXO3 Expression with Tumor Pathogenesis, Prognosis and Clinicopathological Features in Hepatocellular Carcinoma: A Systematic Review with Meta-Analysis. Cancers, 13(21), . https://doi.org/10.3390/cancers13215349
Fondevila, Flavia, et al. "Association of FOXO3 Expression with Tumor Pathogenesis, Prognosis and Clinicopathological Features in Hepatocellular Carcinoma: A Systematic Review with Meta-Analysis." Cancers vol. 13,21 (2021). doi: https://doi.org/10.3390/cancers13215349
Fondevila F, Fernández-Palanca P, Méndez-Blanco C, Payo-Serafín T, Lozano E, Marin JJG, González-Gallego J, Mauriz JL. Association of FOXO3 Expression with Tumor Pathogenesis, Prognosis and Clinicopathological Features in Hepatocellular Carcinoma: A Systematic Review with Meta-Analysis. Cancers (Basel). 2021 Oct 26;13(21). doi: 10.3390/cancers13215349. PMID: 34771514; PMCID: PMC8582569.
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Sage Insights Into the Phylogeny of .

Frontiers in plant science

Rose JP, Kriebel R, Kahan L, DiNicola A, González-Gallegos JG, Celep F, Lemmon EM, Lemmon AR, Sytsma KJ, Drew BT.
PMID: 34899789
Front Plant Sci. 2021 Nov 24;12:767478. doi: 10.3389/fpls.2021.767478. eCollection 2021.

Next-generation sequencing technologies have facilitated new phylogenomic approaches to help clarify previously intractable relationships while simultaneously highlighting the pervasive nature of incongruence within and among genomes that can complicate definitive taxonomic conclusions.

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Rose JP, Kriebel R, Kahan L, et al. Sage Insights Into the Phylogeny of . Front Plant Sci. 2021;12:767478doi: 10.3389/fpls.2021.767478.
Rose, J. P., Kriebel, R., Kahan, L., DiNicola, A., González-Gallegos, J. G., Celep, F., Lemmon, E. M., Lemmon, A. R., Sytsma, K. J., & Drew, B. T. (2021). Sage Insights Into the Phylogeny of . Frontiers in plant science, 12767478. https://doi.org/10.3389/fpls.2021.767478
Rose, Jeffrey P, et al. "Sage Insights Into the Phylogeny of ." Frontiers in plant science vol. 12 (2021): 767478. doi: https://doi.org/10.3389/fpls.2021.767478
Rose JP, Kriebel R, Kahan L, DiNicola A, González-Gallegos JG, Celep F, Lemmon EM, Lemmon AR, Sytsma KJ, Drew BT. Sage Insights Into the Phylogeny of . Front Plant Sci. 2021 Nov 24;12:767478. doi: 10.3389/fpls.2021.767478. eCollection 2021. PMID: 34899789; PMCID: PMC8652245.
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Author Correction: The wide utility of rabbits as models of human diseases.

Experimental & molecular medicine

Esteves PJ, Abrantes J, Baldauf HM, BenMohamed L, Chen Y, Christensen N, González-Gallego J, Giacani L, Hu J, Kaplan G, Keppler OT, Knight KL, Kong XP, Lanning DK, Le Pendu J, de Matos AL, Liu J, Liu S, Lopes AM, Lu S, Lukehart S, Manabe YC, Neves F, McFadden G, Pan R, Peng X, de Sousa-Pereira P, Pinheiro A, Rahman M, Ruvoën-Clouet N, Subbian S, Tuñón MJ, van der Loo W, Vaine M, Via LE, Wang S, Mage R.
PMID: 31270317
Exp Mol Med. 2019 Jul 04;51(7):1. doi: 10.1038/s12276-019-0252-0.

This article was originally published under a CC BY-NC-SA License, but has now been made available under a CC BY 4.0 License.

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Esteves PJ, Abrantes J, Baldauf HM, et al. Author Correction: The wide utility of rabbits as models of human diseases. Exp Mol Med. 2019;51(7):1doi: 10.1038/s12276-019-0252-0.
Esteves, P. J., Abrantes, J., Baldauf, H. M., BenMohamed, L., Chen, Y., Christensen, N., González-Gallego, J., Giacani, L., Hu, J., Kaplan, G., Keppler, O. T., Knight, K. L., Kong, X. P., Lanning, D. K., Le Pendu, J., de Matos, A. L., Liu, J., Liu, S., Lopes, A. M., Lu, S., Lukehart, S., Manabe, Y. C., Neves, F., McFadden, G., Pan, R., Peng, X., de Sousa-Pereira, P., Pinheiro, A., Rahman, M., Ruvoën-Clouet, N., Subbian, S., Tuñón, M. J., van der Loo, W., Vaine, M., Via, L. E., Wang, S., & Mage, R. (2019). Author Correction: The wide utility of rabbits as models of human diseases. Experimental & molecular medicine, 51(7), 1. https://doi.org/10.1038/s12276-019-0252-0
Esteves, Pedro J, et al. "Author Correction: The wide utility of rabbits as models of human diseases." Experimental & molecular medicine vol. 51,7 (2019): 1. doi: https://doi.org/10.1038/s12276-019-0252-0
Esteves PJ, Abrantes J, Baldauf HM, BenMohamed L, Chen Y, Christensen N, González-Gallego J, Giacani L, Hu J, Kaplan G, Keppler OT, Knight KL, Kong XP, Lanning DK, Le Pendu J, de Matos AL, Liu J, Liu S, Lopes AM, Lu S, Lukehart S, Manabe YC, Neves F, McFadden G, Pan R, Peng X, de Sousa-Pereira P, Pinheiro A, Rahman M, Ruvoën-Clouet N, Subbian S, Tuñón MJ, van der Loo W, Vaine M, Via LE, Wang S, Mage R. Author Correction: The wide utility of rabbits as models of human diseases. Exp Mol Med. 2019 Jul 04;51(7):1. doi: 10.1038/s12276-019-0252-0. PMID: 31270317; PMCID: PMC6802666.
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Endoplasmic Reticulum Unfolded Protein Response, Aging and Exercise: An Update.

Frontiers in physiology

Estébanez B, de Paz JA, Cuevas MJ, González-Gallego J.
PMID: 30568599
Front Physiol. 2018 Dec 05;9:1744. doi: 10.3389/fphys.2018.01744. eCollection 2018.

The endoplasmic reticulum (ER) is a dynamic and multifunctional organelle responsible for protein biosynthesis, folding, assembly and modifications. Loss of protein folding regulation, which leads to unfolded or misfolded proteins accumulation inside the ER lumen, drives ER stress (ERS)...

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Estébanez B, de Paz JA, Cuevas MJ, et al. Endoplasmic Reticulum Unfolded Protein Response, Aging and Exercise: An Update. Front Physiol. 2018;9:1744doi: 10.3389/fphys.2018.01744.
Estébanez, B., de Paz, J. A., Cuevas, M. J., & González-Gallego, J. (2018). Endoplasmic Reticulum Unfolded Protein Response, Aging and Exercise: An Update. Frontiers in physiology, 91744. https://doi.org/10.3389/fphys.2018.01744
Estébanez, Brisamar, et al. "Endoplasmic Reticulum Unfolded Protein Response, Aging and Exercise: An Update." Frontiers in physiology vol. 9 (2018): 1744. doi: https://doi.org/10.3389/fphys.2018.01744
Estébanez B, de Paz JA, Cuevas MJ, González-Gallego J. Endoplasmic Reticulum Unfolded Protein Response, Aging and Exercise: An Update. Front Physiol. 2018 Dec 05;9:1744. doi: 10.3389/fphys.2018.01744. eCollection 2018. PMID: 30568599; PMCID: PMC6290262.
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[MCP-1 in urine as biomarker of renal lupus in absence of cytokines, interferon-γ and growth factors].

Reumatologia clinica

Barbado J, Vega L, González-Gallego R, Jimeno A, Ortiz De Lejarazu R, Bermejo-Martin JF.
PMID: 21794736
Reumatol Clin. 2010 Nov-Dec;6(6):296-8. doi: 10.1016/j.reuma.2009.09.015. Epub 2010 Jun 08.

OBJECTIVE: To characterize 17 immunological markers in the Urine of patients with SLE.INTRODUCTION: Lupus nephritis is an inflammatory disease affecting the renal parenchyma. Cytokines and chemokines are key immune mediators that have been related with the pathogenesis of the...

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Barbado J, Vega L, González-Gallego R, et al. MCP-1 en orina como biomarcador de lupus renal en ausencia de citoquinas, interferón-γ y factores de crecimiento. [MCP-1 in urine as biomarker of renal lupus in absence of cytokines, interferon-γ and growth factors]. Reumatol Clin. 2010;6(6):296-8doi: 10.1016/j.reuma.2009.09.015.
Barbado, J., Vega, L., González-Gallego, R., Jimeno, A., Ortiz De Lejarazu, R., & Bermejo-Martin, J. F. (2010). MCP-1 en orina como biomarcador de lupus renal en ausencia de citoquinas, interferón-γ y factores de crecimiento. [MCP-1 in urine as biomarker of renal lupus in absence of cytokines, interferon-γ and growth factors]. Reumatologia clinica, 6(6), 296-8. https://doi.org/10.1016/j.reuma.2009.09.015
Barbado, Julia, et al. "MCP-1 en orina como biomarcador de lupus renal en ausencia de citoquinas, interferón-γ y factores de crecimiento." [MCP-1 in urine as biomarker of renal lupus in absence of cytokines, interferon-γ and growth factors]. Reumatologia clinica vol. 6,6 (2010): 296-8. doi: https://doi.org/10.1016/j.reuma.2009.09.015
Barbado J, Vega L, González-Gallego R, Jimeno A, Ortiz De Lejarazu R, Bermejo-Martin JF. MCP-1 en orina como biomarcador de lupus renal en ausencia de citoquinas, interferón-γ y factores de crecimiento. [MCP-1 in urine as biomarker of renal lupus in absence of cytokines, interferon-γ and growth factors]. Reumatol Clin. 2010 Nov-Dec;6(6):296-8. doi: 10.1016/j.reuma.2009.09.015. Epub 2010 Jun 08. Spanish. PMID: 21794736.
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Sphingosine 1-Phosphate Signaling as a Target in Hepatic Fibrosis Therapy.

Frontiers in pharmacology

González-Fernández B, Sánchez DI, González-Gallego J, Tuñón MJ.
PMID: 28890699
Front Pharmacol. 2017 Aug 25;8:579. doi: 10.3389/fphar.2017.00579. eCollection 2017.

Liver fibrosis is an excess production of extracellular matrix proteins as a result of chronic liver disease which leads to cell death and organ dysfunction. The key cells involved in fibrogenesis are resident hepatic stellate cells (HSCs) which are...

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González-Fernández B, Sánchez DI, González-Gallego J, et al. Sphingosine 1-Phosphate Signaling as a Target in Hepatic Fibrosis Therapy. Front Pharmacol. 2017;8:579doi: 10.3389/fphar.2017.00579.
González-Fernández, B., Sánchez, D. I., González-Gallego, J., & Tuñón, M. J. (2017). Sphingosine 1-Phosphate Signaling as a Target in Hepatic Fibrosis Therapy. Frontiers in pharmacology, 8579. https://doi.org/10.3389/fphar.2017.00579
González-Fernández, Bárbara, et al. "Sphingosine 1-Phosphate Signaling as a Target in Hepatic Fibrosis Therapy." Frontiers in pharmacology vol. 8 (2017): 579. doi: https://doi.org/10.3389/fphar.2017.00579
González-Fernández B, Sánchez DI, González-Gallego J, Tuñón MJ. Sphingosine 1-Phosphate Signaling as a Target in Hepatic Fibrosis Therapy. Front Pharmacol. 2017 Aug 25;8:579. doi: 10.3389/fphar.2017.00579. eCollection 2017. PMID: 28890699; PMCID: PMC5574909.
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