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Computers, teachers, peers: Science learning partners

Science education

Hsi S, Linn MC.
GSID: bPFs4v3F0xYJ
MC Linn, S Hsi - 2000 - taylorfrancis.com

Linn and Hsi show how computers, teachers, and peers can serve as learning partners--helping students build on their ideas and become lifelong science learners. They invite …

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Linn, M. C., & Hsi, S. (2000). Computers, teachers, peers: Science learning partners. Routledge.
Linn, Marcia C., and Sherry Hsi. Computers, teachers, peers: Science learning partners. Routledge, 2000.
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Importance of Including Non-European Populations in Large Human Genetic Studies to Enhance Precision Medicine

Science and underrepresentation

Hui D, Ju D, Wonkam A.
GSID: BNeRO23M0SkJ
D Ju, D Hui, DA Hammond, A Wonkam… - … Data Science, 2022 - annualreviews.org

One goal of genomic medicine is to uncover an individual's genetic risk for disease, which generally requires data connecting genotype to phenotype, as done in genome-wide association studies (GWAS). While there may be clinical promise to employing prediction tools...

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Ju, D., Hui, D., Hammond, D. A., Wonkam, A., & Tishkoff, S. A. (2022). Importance of Including Non-European Populations in Large Human Genetic Studies to Enhance Precision Medicine. Annual Review of Biomedical Data Science, 5.
Ju, Dan, et al. "Importance of Including Non-European Populations in Large Human Genetic Studies to Enhance Precision Medicine." Annual Review of Biomedical Data Science 5 (2022).
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Weakening density dependence from climate change and agricultural intensification triggers pest outbreaks: a 37-year observation of cotton bollworms.

Ecology and evolution

Ouyang F, Hui C, Ge S, Men XY, Zhao ZH, Shi PJ, Zhang YS, Li BL.
PMID: 25535553
Ecol Evol. 2014 Sep;4(17):3362-74. doi: 10.1002/ece3.1190. Epub 2014 Aug 12.

Understanding drivers of population fluctuation, especially for agricultural pests, is central to the provision of agro-ecosystem services. Here, we examine the role of endogenous density dependence and exogenous factors of climate and human activity in regulating the 37-year population...

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Ouyang F, Hui C, Ge S, et al. Weakening density dependence from climate change and agricultural intensification triggers pest outbreaks: a 37-year observation of cotton bollworms. Ecol Evol. 2014;4(17):3362-74doi: 10.1002/ece3.1190.
Ouyang, F., Hui, C., Ge, S., Men, X. Y., Zhao, Z. H., Shi, P. J., Zhang, Y. S., & Li, B. L. (2014). Weakening density dependence from climate change and agricultural intensification triggers pest outbreaks: a 37-year observation of cotton bollworms. Ecology and evolution, 4(17), 3362-74. https://doi.org/10.1002/ece3.1190
Ouyang, Fang, et al. "Weakening density dependence from climate change and agricultural intensification triggers pest outbreaks: a 37-year observation of cotton bollworms." Ecology and evolution vol. 4,17 (2014): 3362-74. doi: https://doi.org/10.1002/ece3.1190
Ouyang F, Hui C, Ge S, Men XY, Zhao ZH, Shi PJ, Zhang YS, Li BL. Weakening density dependence from climate change and agricultural intensification triggers pest outbreaks: a 37-year observation of cotton bollworms. Ecol Evol. 2014 Sep;4(17):3362-74. doi: 10.1002/ece3.1190. Epub 2014 Aug 12. PMID: 25535553; PMCID: PMC4228611.
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A novel lncRNA, LL22NC03-N64E9.1, represses KLF2 transcription through binding with EZH2 in colorectal cancer.

Oncotarget

Lian Y, Yan C, Ding J, Xia R, Ma Z, Hui B, Ji H, Zhou J, Wang K.
PMID: 28938648
Oncotarget. 2017 Jul 31;8(35):59435-59445. doi: 10.18632/oncotarget.19738. eCollection 2017 Aug 29.

Long noncoding RNAs (lncRNA) have been implicated in variety human cancer but their mechanisms of function are mainly undocumented. In the present study, we investigated lncRNAs alteration that contributed to colorectal cancer (CRC) by utilizing TCGA RNA sequencing data...

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Lian Y, Yan C, Ding J, et al. A novel lncRNA, LL22NC03-N64E9.1, represses KLF2 transcription through binding with EZH2 in colorectal cancer. Oncotarget. 2017;8(35):59435-59445doi: 10.18632/oncotarget.19738.
Lian, Y., Yan, C., Ding, J., Xia, R., Ma, Z., Hui, B., Ji, H., Zhou, J., & Wang, K. (2017). A novel lncRNA, LL22NC03-N64E9.1, represses KLF2 transcription through binding with EZH2 in colorectal cancer. Oncotarget, 8(35), 59435-59445. https://doi.org/10.18632/oncotarget.19738
Lian, Yifan, et al. "A novel lncRNA, LL22NC03-N64E9.1, represses KLF2 transcription through binding with EZH2 in colorectal cancer." Oncotarget vol. 8,35 (2017): 59435-59445. doi: https://doi.org/10.18632/oncotarget.19738
Lian Y, Yan C, Ding J, Xia R, Ma Z, Hui B, Ji H, Zhou J, Wang K. A novel lncRNA, LL22NC03-N64E9.1, represses KLF2 transcription through binding with EZH2 in colorectal cancer. Oncotarget. 2017 Jul 31;8(35):59435-59445. doi: 10.18632/oncotarget.19738. eCollection 2017 Aug 29. PMID: 28938648; PMCID: PMC5601744.
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Ethanol-Induced Neurodegeneration and Glial Activation in the Developing Brain.

Brain sciences

Saito M, Chakraborty G, Hui M, Masiello K, Saito M.
PMID: 27537918
Brain Sci. 2016 Aug 16;6(3). doi: 10.3390/brainsci6030031.

Ethanol induces neurodegeneration in the developing brain, which may partially explain the long-lasting adverse effects of prenatal ethanol exposure in fetal alcohol spectrum disorders (FASD). While animal models of FASD show that ethanol-induced neurodegeneration is associated with glial activation,...

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Saito M, Chakraborty G, Hui M, et al. Ethanol-Induced Neurodegeneration and Glial Activation in the Developing Brain. Brain Sci. 2016;6(3)doi: 10.3390/brainsci6030031.
Saito, M., Chakraborty, G., Hui, M., Masiello, K., & Saito, M. (2016). Ethanol-Induced Neurodegeneration and Glial Activation in the Developing Brain. Brain sciences, 6(3), . https://doi.org/10.3390/brainsci6030031
Saito, Mariko, et al. "Ethanol-Induced Neurodegeneration and Glial Activation in the Developing Brain." Brain sciences vol. 6,3 (2016). doi: https://doi.org/10.3390/brainsci6030031
Saito M, Chakraborty G, Hui M, Masiello K, Saito M. Ethanol-Induced Neurodegeneration and Glial Activation in the Developing Brain. Brain Sci. 2016 Aug 16;6(3). doi: 10.3390/brainsci6030031. PMID: 27537918; PMCID: PMC5039460.
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Layer Number Dependence of Li(+) Intercalation on Few-Layer Graphene and Electrochemical Imaging of Its Solid-Electrolyte Interphase Evolution.

ACS nano

Hui J, Burgess M, Zhang J, Rodríguez-López J.
PMID: 26943950
ACS Nano. 2016 Apr 26;10(4):4248-57. doi: 10.1021/acsnano.5b07692. Epub 2016 Mar 14.

A fundamental question facing electrodes made out of few layers of graphene (FLG) is if they display chemical properties that are different to their bulk graphite counterpart. Here, we show evidence that suggests that lithium ion intercalation on FLG,...

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Hui J, Burgess M, Zhang J, et al. Layer Number Dependence of Li(+) Intercalation on Few-Layer Graphene and Electrochemical Imaging of Its Solid-Electrolyte Interphase Evolution. ACS Nano. 2016;10(4):4248-57doi: 10.1021/acsnano.5b07692.
Hui, J., Burgess, M., Zhang, J., & Rodríguez-López, J. (2016). Layer Number Dependence of Li(+) Intercalation on Few-Layer Graphene and Electrochemical Imaging of Its Solid-Electrolyte Interphase Evolution. ACS nano, 10(4), 4248-57. https://doi.org/10.1021/acsnano.5b07692
Hui, Jingshu, et al. "Layer Number Dependence of Li(+) Intercalation on Few-Layer Graphene and Electrochemical Imaging of Its Solid-Electrolyte Interphase Evolution." ACS nano vol. 10,4 (2016): 4248-57. doi: https://doi.org/10.1021/acsnano.5b07692
Hui J, Burgess M, Zhang J, Rodríguez-López J. Layer Number Dependence of Li(+) Intercalation on Few-Layer Graphene and Electrochemical Imaging of Its Solid-Electrolyte Interphase Evolution. ACS Nano. 2016 Apr 26;10(4):4248-57. doi: 10.1021/acsnano.5b07692. Epub 2016 Mar 14. PMID: 26943950.
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The role and significance of VEGFR2.

OncoTargets and therapy

Zhu P, Hu C, Hui K, Jiang X.
PMID: 28919780
Onco Targets Ther. 2017 Sep 01;10:4315-4319. doi: 10.2147/OTT.S142085. eCollection 2017.

Tumor development is closely related to angiogenesis, and VEGFR2 plays an important role in tumor angiogenesis. It is broadly expressed in the blood vessels, especially in the microvessels of tumor tissues. Furthermore, VEGFR2 is detected on the surface of...

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Zhu P, Hu C, Hui K, et al. The role and significance of VEGFR2. Onco Targets Ther. 2017;10:4315-4319doi: 10.2147/OTT.S142085.
Zhu, P., Hu, C., Hui, K., & Jiang, X. (2017). The role and significance of VEGFR2. OncoTargets and therapy, 104315-4319. https://doi.org/10.2147/OTT.S142085
Zhu, Panrong, et al. "The role and significance of VEGFR2." OncoTargets and therapy vol. 10 (2017): 4315-4319. doi: https://doi.org/10.2147/OTT.S142085
Zhu P, Hu C, Hui K, Jiang X. The role and significance of VEGFR2. Onco Targets Ther. 2017 Sep 01;10:4315-4319. doi: 10.2147/OTT.S142085. eCollection 2017. PMID: 28919780; PMCID: PMC5590762.
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Clinical Experience of the Klippel-Trenaunay Syndrome.

Archives of plastic surgery

Sung HM, Chung HY, Lee SJ, Lee JM, Huh S, Lee JW, Choi KY, Yang JD, Cho BC.
PMID: 26430625
Arch Plast Surg. 2015 Sep;42(5):552-8. doi: 10.5999/aps.2015.42.5.552. Epub 2015 Sep 15.

BACKGROUND: The Klippel-Trenaunay syndrome (KTS) is characterized by three clinical features, namely cutaneous capillary malformations, venous malformations, and soft tissue and/or bony hypertrophy of the extremities. The varied manifestations are attributed to the unpredictable clinical nature and prognosis of...

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Sung HM, Chung HY, Lee SJ, et al. Clinical Experience of the Klippel-Trenaunay Syndrome. Arch Plast Surg. 2015;42(5):552-8doi: 10.5999/aps.2015.42.5.552.
Sung, H. M., Chung, H. Y., Lee, S. J., Lee, J. M., Huh, S., Lee, J. W., Choi, K. Y., Yang, J. D., & Cho, B. C. (2015). Clinical Experience of the Klippel-Trenaunay Syndrome. Archives of plastic surgery, 42(5), 552-8. https://doi.org/10.5999/aps.2015.42.5.552
Sung, Hyung Min, et al. "Clinical Experience of the Klippel-Trenaunay Syndrome." Archives of plastic surgery vol. 42,5 (2015): 552-8. doi: https://doi.org/10.5999/aps.2015.42.5.552
Sung HM, Chung HY, Lee SJ, Lee JM, Huh S, Lee JW, Choi KY, Yang JD, Cho BC. Clinical Experience of the Klippel-Trenaunay Syndrome. Arch Plast Surg. 2015 Sep;42(5):552-8. doi: 10.5999/aps.2015.42.5.552. Epub 2015 Sep 15. PMID: 26430625; PMCID: PMC4579165.
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Chlorophyll and carbohydrate metabolism in developing silique and seed are prerequisite to seed oil content of Brassica napus L.

Botanical studies

Hua S, Chen ZH, Zhang Y, Yu H, Lin B, Zhang D.
PMID: 28510961
Bot Stud. 2014 Dec;55(1):34. doi: 10.1186/1999-3110-55-34. Epub 2014 Mar 19.

BACKGROUND: Although the seed oil content in canola is a crucial quality determining trait, the regulatory mechanisms of its formation are not fully discovered. This study compared the silique and seed physiological characteristics including fresh and dry weight, seed...

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Hua S, Chen ZH, Zhang Y, et al. Chlorophyll and carbohydrate metabolism in developing silique and seed are prerequisite to seed oil content of Brassica napus L. Bot Stud. 2014;55(1):34doi: 10.1186/1999-3110-55-34.
Hua, S., Chen, Z. H., Zhang, Y., Yu, H., Lin, B., & Zhang, D. (2014). Chlorophyll and carbohydrate metabolism in developing silique and seed are prerequisite to seed oil content of Brassica napus L. Botanical studies, 55(1), 34. https://doi.org/10.1186/1999-3110-55-34
Hua, Shuijin, et al. "Chlorophyll and carbohydrate metabolism in developing silique and seed are prerequisite to seed oil content of Brassica napus L." Botanical studies vol. 55,1 (2014): 34. doi: https://doi.org/10.1186/1999-3110-55-34
Hua S, Chen ZH, Zhang Y, Yu H, Lin B, Zhang D. Chlorophyll and carbohydrate metabolism in developing silique and seed are prerequisite to seed oil content of Brassica napus L. Bot Stud. 2014 Dec;55(1):34. doi: 10.1186/1999-3110-55-34. Epub 2014 Mar 19. PMID: 28510961; PMCID: PMC5432831.
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Identification of hub genes and pathways associated with hepatocellular carcinoma based on network strategy.

Experimental and therapeutic medicine

Liu J, Hua P, Hui L, Zhang LL, Hu Z, Zhu YW.
PMID: 27703495
Exp Ther Med. 2016 Oct;12(4):2109-2119. doi: 10.3892/etm.2016.3599. Epub 2016 Aug 12.

The objective of this study was to identify hub genes and pathways associated with hepatocellular carcinoma (HCC) by centrality analysis of a co-expression network. A co-expression network based on differentially expressed (DE) genes of HCC was constructed using the...

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Liu J, Hua P, Hui L, et al. Identification of hub genes and pathways associated with hepatocellular carcinoma based on network strategy. Exp Ther Med. 2016;12(4):2109-2119doi: 10.3892/etm.2016.3599.
Liu, J., Hua, P., Hui, L., Zhang, L. L., Hu, Z., & Zhu, Y. W. (2016). Identification of hub genes and pathways associated with hepatocellular carcinoma based on network strategy. Experimental and therapeutic medicine, 12(4), 2109-2119. https://doi.org/10.3892/etm.2016.3599
Liu, Jun, et al. "Identification of hub genes and pathways associated with hepatocellular carcinoma based on network strategy." Experimental and therapeutic medicine vol. 12,4 (2016): 2109-2119. doi: https://doi.org/10.3892/etm.2016.3599
Liu J, Hua P, Hui L, Zhang LL, Hu Z, Zhu YW. Identification of hub genes and pathways associated with hepatocellular carcinoma based on network strategy. Exp Ther Med. 2016 Oct;12(4):2109-2119. doi: 10.3892/etm.2016.3599. Epub 2016 Aug 12. PMID: 27703495; PMCID: PMC5039750.
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The Prevalence of Vitiligo: A Meta-Analysis.

PloS one

Zhang Y, Cai Y, Shi M, Jiang S, Cui S, Wu Y, Gao XH, Chen HD.
PMID: 27673680
PLoS One. 2016 Sep 27;11(9):e0163806. doi: 10.1371/journal.pone.0163806. eCollection 2016.

OBJECTIVE: To conduct a meta-analysis assessing the prevalence of vitiligo.METHODS: Literatures that reported prevalence rates of vitiligo were identified using EMBASE, PubMed, the Cochrane Library, China National Knowledge Infrastructure (CNKI), Wanfang database and Weipu database for the period from...

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Zhang Y, Cai Y, Shi M, et al. The Prevalence of Vitiligo: A Meta-Analysis. PLoS One. 2016;11(9):e0163806doi: 10.1371/journal.pone.0163806.
Zhang, Y., Cai, Y., Shi, M., Jiang, S., Cui, S., Wu, Y., Gao, X. H., & Chen, H. D. (2016). The Prevalence of Vitiligo: A Meta-Analysis. PloS one, 11(9), e0163806. https://doi.org/10.1371/journal.pone.0163806
Zhang, Yuhui, et al. "The Prevalence of Vitiligo: A Meta-Analysis." PloS one vol. 11,9 (2016): e0163806. doi: https://doi.org/10.1371/journal.pone.0163806
Zhang Y, Cai Y, Shi M, Jiang S, Cui S, Wu Y, Gao XH, Chen HD. The Prevalence of Vitiligo: A Meta-Analysis. PLoS One. 2016 Sep 27;11(9):e0163806. doi: 10.1371/journal.pone.0163806. eCollection 2016. PMID: 27673680; PMCID: PMC5038943.
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Vascular endothelial growth factor polymorphisms and lung cancer risk.

International journal of clinical and experimental medicine

Fan J, Zhang W, Lei C, Qiao B, Liu Q, Chen Q, Jiao H, Jiang L, Cui S, Chen J.
PMID: 26131266
Int J Clin Exp Med. 2015 Apr 15;8(4):6406-11. eCollection 2015.

The VEGF polymorphisms has been implicated in the susceptibility to lung cancer, but the results are not conclusive. The aim of this study is to investigate the association between the VEGF polymorphisms and the risk of lung cancer by...

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Fan J, Zhang W, Lei C, et al. Vascular endothelial growth factor polymorphisms and lung cancer risk. Int J Clin Exp Med. 2015;8(4):6406-11
Fan, J., Zhang, W., Lei, C., Qiao, B., Liu, Q., Chen, Q., Jiao, H., Jiang, L., Cui, S., & Chen, J. (2015). Vascular endothelial growth factor polymorphisms and lung cancer risk. International journal of clinical and experimental medicine, 8(4), 6406-11.
Fan, Junli, et al. "Vascular endothelial growth factor polymorphisms and lung cancer risk." International journal of clinical and experimental medicine vol. 8,4 (2015): 6406-11.
Fan J, Zhang W, Lei C, Qiao B, Liu Q, Chen Q, Jiao H, Jiang L, Cui S, Chen J. Vascular endothelial growth factor polymorphisms and lung cancer risk. Int J Clin Exp Med. 2015 Apr 15;8(4):6406-11. eCollection 2015. PMID: 26131266; PMCID: PMC4483952.
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Showing 1 to 12 of 2768 entries