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Testosterone increases circulating dehydroepiandrosterone sulfate levels in the male rhesus macaque.

Frontiers in endocrinology

Sorwell KG, Kohama SG, Urbanski HF.
PMID: 25009533
Front Endocrinol (Lausanne). 2014 Jun 25;5:101. doi: 10.3389/fendo.2014.00101. eCollection 2014.

The adrenal steroid dehydroepiandrosterone (DHEA) and its sulfate (DHEAS) are two of the most abundant hormones in the human circulation. Furthermore, they are released in a circadian pattern and show a marked age-associated decline. Adult levels of DHEA and...

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Sorwell KG, Kohama SG, Urbanski HF. Testosterone increases circulating dehydroepiandrosterone sulfate levels in the male rhesus macaque. Front Endocrinol (Lausanne). 2014;5:101doi: 10.3389/fendo.2014.00101.
Sorwell, K. G., Kohama, S. G., & Urbanski, H. F. (2014). Testosterone increases circulating dehydroepiandrosterone sulfate levels in the male rhesus macaque. Frontiers in endocrinology, 5101. https://doi.org/10.3389/fendo.2014.00101
Sorwell, Krystina G, et al. "Testosterone increases circulating dehydroepiandrosterone sulfate levels in the male rhesus macaque." Frontiers in endocrinology vol. 5 (2014): 101. doi: https://doi.org/10.3389/fendo.2014.00101
Sorwell KG, Kohama SG, Urbanski HF. Testosterone increases circulating dehydroepiandrosterone sulfate levels in the male rhesus macaque. Front Endocrinol (Lausanne). 2014 Jun 25;5:101. doi: 10.3389/fendo.2014.00101. eCollection 2014. PMID: 25009533; PMCID: PMC4070064.
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Farnesol-like endogenous sesquiterpenoids in vertebrates: the probable but overlooked functional "inbrome" anti-aging counterpart of juvenile hormone of insects?.

Frontiers in endocrinology

De Loof A, Marchal E, Rivera-Perez C, Noriega FG, Schoofs L.
PMID: 25610425
Front Endocrinol (Lausanne). 2015 Jan 06;5:222. doi: 10.3389/fendo.2014.00222. eCollection 2014.

Literature on the question whether the juvenile stage of vertebrates is hormonally regulated is scarce. It seems to be intuitively assumed that this stage of development is automated, and does not require any specific hormone(s). Such reasoning mimics the...

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De Loof A, Marchal E, Rivera-Perez C, et al. Farnesol-like endogenous sesquiterpenoids in vertebrates: the probable but overlooked functional "inbrome" anti-aging counterpart of juvenile hormone of insects?. Front Endocrinol (Lausanne). 2015;5:222doi: 10.3389/fendo.2014.00222.
De Loof, A., Marchal, E., Rivera-Perez, C., Noriega, F. G., & Schoofs, L. (2014). Farnesol-like endogenous sesquiterpenoids in vertebrates: the probable but overlooked functional "inbrome" anti-aging counterpart of juvenile hormone of insects?. Frontiers in endocrinology, 5222. https://doi.org/10.3389/fendo.2014.00222
De Loof, Arnold, et al. "Farnesol-like endogenous sesquiterpenoids in vertebrates: the probable but overlooked functional "inbrome" anti-aging counterpart of juvenile hormone of insects?." Frontiers in endocrinology vol. 5 (2014): 222. doi: https://doi.org/10.3389/fendo.2014.00222
De Loof A, Marchal E, Rivera-Perez C, Noriega FG, Schoofs L. Farnesol-like endogenous sesquiterpenoids in vertebrates: the probable but overlooked functional "inbrome" anti-aging counterpart of juvenile hormone of insects?. Front Endocrinol (Lausanne). 2015 Jan 06;5:222. doi: 10.3389/fendo.2014.00222. eCollection 2014. PMID: 25610425; PMCID: PMC4285131.
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Impact of Maternal Melatonin Suppression on Amount and Functionality of Brown Adipose Tissue (BAT) in the Newborn Sheep.

Frontiers in endocrinology

Seron-Ferre M, Reynolds H, Mendez NA, Mondaca M, Valenzuela F, Ebensperger R, Valenzuela GJ, Herrera EA, Llanos AJ, Torres-Farfan C.
PMID: 25610428
Front Endocrinol (Lausanne). 2015 Jan 06;5:232. doi: 10.3389/fendo.2014.00232. eCollection 2014.

In human and sheep newborns, brown adipose tissue (BAT) accrued during fetal development is used for newborn thermogenesis. Here, we explored the role of maternal melatonin during gestation on the amount and functionality of BAT in the neonate. We...

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Seron-Ferre M, Reynolds H, Mendez NA, et al. Impact of Maternal Melatonin Suppression on Amount and Functionality of Brown Adipose Tissue (BAT) in the Newborn Sheep. Front Endocrinol (Lausanne). 2015;5:232doi: 10.3389/fendo.2014.00232.
Seron-Ferre, M., Reynolds, H., Mendez, N. A., Mondaca, M., Valenzuela, F., Ebensperger, R., Valenzuela, G. J., Herrera, E. A., Llanos, A. J., & Torres-Farfan, C. (2014). Impact of Maternal Melatonin Suppression on Amount and Functionality of Brown Adipose Tissue (BAT) in the Newborn Sheep. Frontiers in endocrinology, 5232. https://doi.org/10.3389/fendo.2014.00232
Seron-Ferre, Maria, et al. "Impact of Maternal Melatonin Suppression on Amount and Functionality of Brown Adipose Tissue (BAT) in the Newborn Sheep." Frontiers in endocrinology vol. 5 (2014): 232. doi: https://doi.org/10.3389/fendo.2014.00232
Seron-Ferre M, Reynolds H, Mendez NA, Mondaca M, Valenzuela F, Ebensperger R, Valenzuela GJ, Herrera EA, Llanos AJ, Torres-Farfan C. Impact of Maternal Melatonin Suppression on Amount and Functionality of Brown Adipose Tissue (BAT) in the Newborn Sheep. Front Endocrinol (Lausanne). 2015 Jan 06;5:232. doi: 10.3389/fendo.2014.00232. eCollection 2014. PMID: 25610428; PMCID: PMC4285176.
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Genes and podocytes - new insights into mechanisms of podocytopathy.

Frontiers in endocrinology

Bierzynska A, Soderquest K, Koziell A.
PMID: 25667580
Front Endocrinol (Lausanne). 2015 Jan 23;5:226. doi: 10.3389/fendo.2014.00226. eCollection 2014.

After decades of primarily morphological study, positional cloning of the NPHS1 gene was the landmark event that established aberrant podocyte genetics as a pivotal cause of malfunction of the glomerular filter. This ended any uncertainty whether genetic mutation plays...

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Bierzynska A, Soderquest K, Koziell A. Genes and podocytes - new insights into mechanisms of podocytopathy. Front Endocrinol (Lausanne). 2015;5:226doi: 10.3389/fendo.2014.00226.
Bierzynska, A., Soderquest, K., & Koziell, A. (2014). Genes and podocytes - new insights into mechanisms of podocytopathy. Frontiers in endocrinology, 5226. https://doi.org/10.3389/fendo.2014.00226
Bierzynska, Agnieszka, et al. "Genes and podocytes - new insights into mechanisms of podocytopathy." Frontiers in endocrinology vol. 5 (2014): 226. doi: https://doi.org/10.3389/fendo.2014.00226
Bierzynska A, Soderquest K, Koziell A. Genes and podocytes - new insights into mechanisms of podocytopathy. Front Endocrinol (Lausanne). 2015 Jan 23;5:226. doi: 10.3389/fendo.2014.00226. eCollection 2014. PMID: 25667580; PMCID: PMC4304234.
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Spherical Oligo-Silicic Acid SOSA Disclosed as Possible Endogenous Digitalis-Like Factor.

Frontiers in endocrinology

Kerek F, Voicu VA.
PMID: 25667581
Front Endocrinol (Lausanne). 2015 Jan 23;5:233. doi: 10.3389/fendo.2014.00233. eCollection 2014.

The Na(+)/K(+)-ATPase is a membrane ion-transporter protein, specifically inhibited by digitalis glycosides used in cardiac therapy. The existence in mammals of some endogenous digitalis-like factors (EDLFs) as presumed ATPase ligands is generally accepted. But the chemical structure of these...

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Kerek F, Voicu VA. Spherical Oligo-Silicic Acid SOSA Disclosed as Possible Endogenous Digitalis-Like Factor. Front Endocrinol (Lausanne). 2015;5:233doi: 10.3389/fendo.2014.00233.
Kerek, F., & Voicu, V. A. (2014). Spherical Oligo-Silicic Acid SOSA Disclosed as Possible Endogenous Digitalis-Like Factor. Frontiers in endocrinology, 5233. https://doi.org/10.3389/fendo.2014.00233
Kerek, Franz, and Voicu, Victor A. "Spherical Oligo-Silicic Acid SOSA Disclosed as Possible Endogenous Digitalis-Like Factor." Frontiers in endocrinology vol. 5 (2014): 233. doi: https://doi.org/10.3389/fendo.2014.00233
Kerek F, Voicu VA. Spherical Oligo-Silicic Acid SOSA Disclosed as Possible Endogenous Digitalis-Like Factor. Front Endocrinol (Lausanne). 2015 Jan 23;5:233. doi: 10.3389/fendo.2014.00233. eCollection 2014. PMID: 25667581; PMCID: PMC4304351.
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O-GlcNAcylation and Inflammation: A Vast Territory to Explore.

Frontiers in endocrinology

Baudoin L, Issad T.
PMID: 25620956
Front Endocrinol (Lausanne). 2015 Jan 09;5:235. doi: 10.3389/fendo.2014.00235. eCollection 2014.

O-GlcNAcylation is a reversible post-translational modification that regulates the activities of cytosolic and nuclear proteins according to glucose availability. This modification appears to participate in several hyperglycemia-associated complications. An important feature of metabolic diseases such as diabetes and obesity...

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Baudoin L, Issad T. O-GlcNAcylation and Inflammation: A Vast Territory to Explore. Front Endocrinol (Lausanne). 2015;5:235doi: 10.3389/fendo.2014.00235.
Baudoin, L., & Issad, T. (2014). O-GlcNAcylation and Inflammation: A Vast Territory to Explore. Frontiers in endocrinology, 5235. https://doi.org/10.3389/fendo.2014.00235
Baudoin, Léa, and Issad, Tarik. "O-GlcNAcylation and Inflammation: A Vast Territory to Explore." Frontiers in endocrinology vol. 5 (2014): 235. doi: https://doi.org/10.3389/fendo.2014.00235
Baudoin L, Issad T. O-GlcNAcylation and Inflammation: A Vast Territory to Explore. Front Endocrinol (Lausanne). 2015 Jan 09;5:235. doi: 10.3389/fendo.2014.00235. eCollection 2014. PMID: 25620956; PMCID: PMC4288382.
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Adrenocortical zonation, renewal, and remodeling.

Frontiers in endocrinology

Pihlajoki M, Dörner J, Cochran RS, Heikinheimo M, Wilson DB.
PMID: 25798129
Front Endocrinol (Lausanne). 2015 Mar 05;6:27. doi: 10.3389/fendo.2015.00027. eCollection 2015.

The adrenal cortex is divided into concentric zones. In humans the major cortical zones are the zona glomerulosa, zona fasciculata, and zona reticularis. The adrenal cortex is a dynamic organ in which senescent cells are replaced by newly differentiated...

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Pihlajoki M, Dörner J, Cochran RS, et al. Adrenocortical zonation, renewal, and remodeling. Front Endocrinol (Lausanne). 2015;6:27doi: 10.3389/fendo.2015.00027.
Pihlajoki, M., Dörner, J., Cochran, R. S., Heikinheimo, M., & Wilson, D. B. (2015). Adrenocortical zonation, renewal, and remodeling. Frontiers in endocrinology, 627. https://doi.org/10.3389/fendo.2015.00027
Pihlajoki, Marjut, et al. "Adrenocortical zonation, renewal, and remodeling." Frontiers in endocrinology vol. 6 (2015): 27. doi: https://doi.org/10.3389/fendo.2015.00027
Pihlajoki M, Dörner J, Cochran RS, Heikinheimo M, Wilson DB. Adrenocortical zonation, renewal, and remodeling. Front Endocrinol (Lausanne). 2015 Mar 05;6:27. doi: 10.3389/fendo.2015.00027. eCollection 2015. PMID: 25798129; PMCID: PMC4350438.
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High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones.

Frontiers in endocrinology

Palmisano BT, Stafford JM, Pendergast JS.
PMID: 28352249
Front Endocrinol (Lausanne). 2017 Mar 14;8:44. doi: 10.3389/fendo.2017.00044. eCollection 2017.

Obesity in women is increased by the loss of circulating estrogen after menopause. Shift work, which disrupts circadian rhythms, also increases the risk for obesity. It is not known whether ovarian hormones interact with the circadian system to protect...

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Palmisano BT, Stafford JM, Pendergast JS. High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones. Front Endocrinol (Lausanne). 2017;8:44doi: 10.3389/fendo.2017.00044.
Palmisano, B. T., Stafford, J. M., & Pendergast, J. S. (2017). High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones. Frontiers in endocrinology, 844. https://doi.org/10.3389/fendo.2017.00044
Palmisano, Brian T, et al. "High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones." Frontiers in endocrinology vol. 8 (2017): 44. doi: https://doi.org/10.3389/fendo.2017.00044
Palmisano BT, Stafford JM, Pendergast JS. High-Fat Feeding Does Not Disrupt Daily Rhythms in Female Mice because of Protection by Ovarian Hormones. Front Endocrinol (Lausanne). 2017 Mar 14;8:44. doi: 10.3389/fendo.2017.00044. eCollection 2017. PMID: 28352249; PMCID: PMC5348546.
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Effects of Neuroendocrine CB1 Activity on Adult Leydig Cells.

Frontiers in endocrinology

Cobellis G, Meccariello R, Chianese R, Chioccarelli T, Fasano S, Pierantoni R.
PMID: 27375550
Front Endocrinol (Lausanne). 2016 Jun 03;7:47. doi: 10.3389/fendo.2016.00047. eCollection 2016.

Endocannabinoids control male reproduction acting at central and local level via cannabinoid receptors. The cannabinoid receptor CB1 has been characterized in the testis, in somatic and germ cells of mammalian and non-mammalian animal models, and its activity related to...

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Cobellis G, Meccariello R, Chianese R, et al. Effects of Neuroendocrine CB1 Activity on Adult Leydig Cells. Front Endocrinol (Lausanne). 2016;7:47doi: 10.3389/fendo.2016.00047.
Cobellis, G., Meccariello, R., Chianese, R., Chioccarelli, T., Fasano, S., & Pierantoni, R. (2016). Effects of Neuroendocrine CB1 Activity on Adult Leydig Cells. Frontiers in endocrinology, 747. https://doi.org/10.3389/fendo.2016.00047
Cobellis, Gilda, et al. "Effects of Neuroendocrine CB1 Activity on Adult Leydig Cells." Frontiers in endocrinology vol. 7 (2016): 47. doi: https://doi.org/10.3389/fendo.2016.00047
Cobellis G, Meccariello R, Chianese R, Chioccarelli T, Fasano S, Pierantoni R. Effects of Neuroendocrine CB1 Activity on Adult Leydig Cells. Front Endocrinol (Lausanne). 2016 Jun 03;7:47. doi: 10.3389/fendo.2016.00047. eCollection 2016. PMID: 27375550; PMCID: PMC4891325.
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Molecular Modeling of Structures and Interaction of Human Corticotropin-Releasing Factor (CRF) Binding Protein and CRF Type-2 Receptor.

Frontiers in endocrinology

Slater PG, Gutierrez-Maldonado SE, Gysling K, Lagos CF.
PMID: 29515519
Front Endocrinol (Lausanne). 2018 Feb 20;9:43. doi: 10.3389/fendo.2018.00043. eCollection 2018.

The corticotropin-releasing factor (CRF) system is a key mediator of the stress response and addictive behavior. The CRF system includes four peptides: The CRF system includes four peptides: CRF, urocortins I-III, CRF binding protein (CRF-BP) that binds CRF with...

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Slater PG, Gutierrez-Maldonado SE, Gysling K, et al. Molecular Modeling of Structures and Interaction of Human Corticotropin-Releasing Factor (CRF) Binding Protein and CRF Type-2 Receptor. Front Endocrinol (Lausanne). 2018;9:43doi: 10.3389/fendo.2018.00043.
Slater, P. G., Gutierrez-Maldonado, S. E., Gysling, K., & Lagos, C. F. (2018). Molecular Modeling of Structures and Interaction of Human Corticotropin-Releasing Factor (CRF) Binding Protein and CRF Type-2 Receptor. Frontiers in endocrinology, 943. https://doi.org/10.3389/fendo.2018.00043
Slater, Paula G, et al. "Molecular Modeling of Structures and Interaction of Human Corticotropin-Releasing Factor (CRF) Binding Protein and CRF Type-2 Receptor." Frontiers in endocrinology vol. 9 (2018): 43. doi: https://doi.org/10.3389/fendo.2018.00043
Slater PG, Gutierrez-Maldonado SE, Gysling K, Lagos CF. Molecular Modeling of Structures and Interaction of Human Corticotropin-Releasing Factor (CRF) Binding Protein and CRF Type-2 Receptor. Front Endocrinol (Lausanne). 2018 Feb 20;9:43. doi: 10.3389/fendo.2018.00043. eCollection 2018. PMID: 29515519; PMCID: PMC5826306.
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Estrogen Receptor β in Melanoma: From Molecular Insights to Potential Clinical Utility.

Frontiers in endocrinology

Marzagalli M, Montagnani Marelli M, Casati L, Fontana F, Moretti RM, Limonta P.
PMID: 27833586
Front Endocrinol (Lausanne). 2016 Oct 26;7:140. doi: 10.3389/fendo.2016.00140. eCollection 2016.

Cutaneous melanoma is an aggressive tumor; its incidence has been reported to increase fast in the past decades. Melanoma is a heterogeneous tumor, with most patients harboring mutations in the BRAF or NRAS oncogenes, leading to the overactivation of...

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Marzagalli M, Montagnani Marelli M, Casati L, et al. Estrogen Receptor β in Melanoma: From Molecular Insights to Potential Clinical Utility. Front Endocrinol (Lausanne). 2016;7:140doi: 10.3389/fendo.2016.00140.
Marzagalli, M., Montagnani Marelli, M., Casati, L., Fontana, F., Moretti, R. M., & Limonta, P. (2016). Estrogen Receptor β in Melanoma: From Molecular Insights to Potential Clinical Utility. Frontiers in endocrinology, 7140. https://doi.org/10.3389/fendo.2016.00140
Marzagalli, Monica, et al. "Estrogen Receptor β in Melanoma: From Molecular Insights to Potential Clinical Utility." Frontiers in endocrinology vol. 7 (2016): 140. doi: https://doi.org/10.3389/fendo.2016.00140
Marzagalli M, Montagnani Marelli M, Casati L, Fontana F, Moretti RM, Limonta P. Estrogen Receptor β in Melanoma: From Molecular Insights to Potential Clinical Utility. Front Endocrinol (Lausanne). 2016 Oct 26;7:140. doi: 10.3389/fendo.2016.00140. eCollection 2016. PMID: 27833586; PMCID: PMC5080294.
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Endocrine FGFs: Evolution, Physiology, Pathophysiology, and Pharmacotherapy.

Frontiers in endocrinology

Itoh N, Ohta H, Konishi M.
PMID: 26483756
Front Endocrinol (Lausanne). 2015 Sep 29;6:154. doi: 10.3389/fendo.2015.00154. eCollection 2015.

The human fibroblast growth factor (FGF) family comprises 22 structurally related polypeptides that play crucial roles in neuronal functions, development, and metabolism. FGFs are classified as intracrine, paracrine, and endocrine FGFs based on their action mechanisms. Paracrine and endocrine...

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Itoh N, Ohta H, Konishi M. Endocrine FGFs: Evolution, Physiology, Pathophysiology, and Pharmacotherapy. Front Endocrinol (Lausanne). 2015;6:154doi: 10.3389/fendo.2015.00154.
Itoh, N., Ohta, H., & Konishi, M. (2015). Endocrine FGFs: Evolution, Physiology, Pathophysiology, and Pharmacotherapy. Frontiers in endocrinology, 6154. https://doi.org/10.3389/fendo.2015.00154
Itoh, Nobuyuki, et al. "Endocrine FGFs: Evolution, Physiology, Pathophysiology, and Pharmacotherapy." Frontiers in endocrinology vol. 6 (2015): 154. doi: https://doi.org/10.3389/fendo.2015.00154
Itoh N, Ohta H, Konishi M. Endocrine FGFs: Evolution, Physiology, Pathophysiology, and Pharmacotherapy. Front Endocrinol (Lausanne). 2015 Sep 29;6:154. doi: 10.3389/fendo.2015.00154. eCollection 2015. PMID: 26483756; PMCID: PMC4586497.
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Showing 13 to 24 of 6108 entries