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Diversification of Rosaceae since the Late Cretaceous based on plastid phylogenomics

STEM and diversification

Chase MW, Jin JJ.
GSID: vwZmWRfIMkgJ
SD Zhang, JJ Jin, SY Chen, MW Chase… - New …, 2017 - Wiley Online Library

… Our results indicate the stem age of Rosaceae to be during the Albian period of the Early Cretaceous (106.50 Ma with 95% HPD: 106.50–106.50 Ma). The crown age of Rosaceae …

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Zhang, S. D., Jin, J. J., Chen, S. Y., Chase, M. W., Soltis, D. E., Li, H. T., ... & Yi, T. S. (2017). Diversification of Rosaceae since the Late Cretaceous based on plastid phylogenomics. New Phytologist, 214(3), 1355-1367.
Zhang, Shu‐Dong, et al. "Diversification of Rosaceae since the Late Cretaceous based on plastid phylogenomics." New Phytologist 214.3 (2017): 1355-1367.
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J-aggregation induced low bandgap anthracene-based conjugated molecule for solution-processed solar cells.

Chemical communications (Cambridge, England)

Shin J, Kang NS, Kim KH, Lee TW, Jin JI, Kim M, Lee K, Ju BK, Hong JM, Choi DH.
PMID: 22801585
Chem Commun (Camb). 2012 Sep 04;48(68):8490-2. doi: 10.1039/c2cc34109c. Epub 2012 Jul 17.

A new anthracene-based X-shaped conjugated molecule, HBTATHT, was synthesized. Thin film transistors based on unannealed HBTATHT showed a carrier mobility of 0.15 cm(2) V(-1) s(-1) (I(on/off) = 7.9 × 10(6)). Further, a solution processed solar cell made of HBTATHT...

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Shin J, Kang NS, Kim KH, et al. J-aggregation induced low bandgap anthracene-based conjugated molecule for solution-processed solar cells. Chem Commun (Camb). 2012;48(68):8490-2doi: 10.1039/c2cc34109c.
Shin, J., Kang, N. S., Kim, K. H., Lee, T. W., Jin, J. I., Kim, M., Lee, K., Ju, B. K., Hong, J. M., & Choi, D. H. (2012). J-aggregation induced low bandgap anthracene-based conjugated molecule for solution-processed solar cells. Chemical communications (Cambridge, England), 48(68), 8490-2. https://doi.org/10.1039/c2cc34109c
Shin, Jicheol, et al. "J-aggregation induced low bandgap anthracene-based conjugated molecule for solution-processed solar cells." Chemical communications (Cambridge, England) vol. 48,68 (2012): 8490-2. doi: https://doi.org/10.1039/c2cc34109c
Shin J, Kang NS, Kim KH, Lee TW, Jin JI, Kim M, Lee K, Ju BK, Hong JM, Choi DH. J-aggregation induced low bandgap anthracene-based conjugated molecule for solution-processed solar cells. Chem Commun (Camb). 2012 Sep 04;48(68):8490-2. doi: 10.1039/c2cc34109c. Epub 2012 Jul 17. PMID: 22801585.
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Different Origins of Newcastle Disease Virus Hemagglutinin-Neuraminidase Protein Modulate the Replication Efficiency and Pathogenicity of the Virus.

Frontiers in microbiology

Jin JH, Cheng JL, He ZR, Ren YC, Yu XH, Song Y, Yang HM, Yang YL, Liu T, Zhang GZ.
PMID: 28878757
Front Microbiol. 2017 Aug 23;8:1607. doi: 10.3389/fmicb.2017.01607. eCollection 2017.

To investigate the exact effects of different origins of Newcastle disease virus (NDV) hemagglutinin-neuraminidase (HN) protein to the biological characteristics of the virus, we systematically studied the correlation between the HN protein and NDV virulence by exchanging the HN...

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Jin JH, Cheng JL, He ZR, et al. Different Origins of Newcastle Disease Virus Hemagglutinin-Neuraminidase Protein Modulate the Replication Efficiency and Pathogenicity of the Virus. Front Microbiol. 2017;8:1607doi: 10.3389/fmicb.2017.01607.
Jin, J. H., Cheng, J. L., He, Z. R., Ren, Y. C., Yu, X. H., Song, Y., Yang, H. M., Yang, Y. L., Liu, T., & Zhang, G. Z. (2017). Different Origins of Newcastle Disease Virus Hemagglutinin-Neuraminidase Protein Modulate the Replication Efficiency and Pathogenicity of the Virus. Frontiers in microbiology, 81607. https://doi.org/10.3389/fmicb.2017.01607
Jin, Ji-Hui, et al. "Different Origins of Newcastle Disease Virus Hemagglutinin-Neuraminidase Protein Modulate the Replication Efficiency and Pathogenicity of the Virus." Frontiers in microbiology vol. 8 (2017): 1607. doi: https://doi.org/10.3389/fmicb.2017.01607
Jin JH, Cheng JL, He ZR, Ren YC, Yu XH, Song Y, Yang HM, Yang YL, Liu T, Zhang GZ. Different Origins of Newcastle Disease Virus Hemagglutinin-Neuraminidase Protein Modulate the Replication Efficiency and Pathogenicity of the Virus. Front Microbiol. 2017 Aug 23;8:1607. doi: 10.3389/fmicb.2017.01607. eCollection 2017. PMID: 28878757; PMCID: PMC5572326.
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Genome-wide association study for acute otitis media in children identifies FNDC1 as disease contributing gene.

Nature communications

van Ingen G, Li J, Goedegebure A, Pandey R, Li YR, March ME, Jaddoe VW, Bakay M, Mentch FD, Thomas K, Wei Z, Chang X, Hain HS, Uitterlinden AG, Moll HA, van Duijn CM, Rivadeneira F, Raat H, Baatenburg de Jong RJ, Sleiman PM, van der Schroeff MP, Hakonarson H.
PMID: 27677580
Nat Commun. 2016 Sep 28;7:12792. doi: 10.1038/ncomms12792.

Acute otitis media (AOM) is among the most common pediatric diseases, and the most frequent reason for antibiotic treatment in children. Risk of AOM is dependent on environmental and host factors, as well as a significant genetic component. We...

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van Ingen G, Li J, Goedegebure A, et al. Genome-wide association study for acute otitis media in children identifies FNDC1 as disease contributing gene. Nat Commun. 2016;7:12792doi: 10.1038/ncomms12792.
van Ingen, G., Li, J., Goedegebure, A., Pandey, R., Li, Y. R., March, M. E., Jaddoe, V. W., Bakay, M., Mentch, F. D., Thomas, K., Wei, Z., Chang, X., Hain, H. S., Uitterlinden, A. G., Moll, H. A., van Duijn, C. M., Rivadeneira, F., Raat, H., Baatenburg de Jong, R. J., Sleiman, P. M., van der Schroeff, M. P., & Hakonarson, H. (2016). Genome-wide association study for acute otitis media in children identifies FNDC1 as disease contributing gene. Nature communications, 712792. https://doi.org/10.1038/ncomms12792
van Ingen, Gijs, et al. "Genome-wide association study for acute otitis media in children identifies FNDC1 as disease contributing gene." Nature communications vol. 7 (2016): 12792. doi: https://doi.org/10.1038/ncomms12792
van Ingen G, Li J, Goedegebure A, Pandey R, Li YR, March ME, Jaddoe VW, Bakay M, Mentch FD, Thomas K, Wei Z, Chang X, Hain HS, Uitterlinden AG, Moll HA, van Duijn CM, Rivadeneira F, Raat H, Baatenburg de Jong RJ, Sleiman PM, van der Schroeff MP, Hakonarson H. Genome-wide association study for acute otitis media in children identifies FNDC1 as disease contributing gene. Nat Commun. 2016 Sep 28;7:12792. doi: 10.1038/ncomms12792. PMID: 27677580; PMCID: PMC5052699.
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Roles of the Polymerase-Associated Protein Genes in Newcastle Disease Virus Virulence.

Frontiers in microbiology

Yu XH, Cheng JL, Xue J, Jin JH, Song Y, Zhao J, Zhang GZ.
PMID: 28220114
Front Microbiol. 2017 Feb 06;8:161. doi: 10.3389/fmicb.2017.00161. eCollection 2017.

The virulence of Newcastle disease virus varies greatly and is determined by multiple genetic factors. In this study, we systematically evaluated the roles of the polymerase-associated (NP, P and L) protein genes in genotype VII NDV virulence after confirming...

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Yu XH, Cheng JL, Xue J, et al. Roles of the Polymerase-Associated Protein Genes in Newcastle Disease Virus Virulence. Front Microbiol. 2017;8:161doi: 10.3389/fmicb.2017.00161.
Yu, X. H., Cheng, J. L., Xue, J., Jin, J. H., Song, Y., Zhao, J., & Zhang, G. Z. (2017). Roles of the Polymerase-Associated Protein Genes in Newcastle Disease Virus Virulence. Frontiers in microbiology, 8161. https://doi.org/10.3389/fmicb.2017.00161
Yu, Xiao-Hui, et al. "Roles of the Polymerase-Associated Protein Genes in Newcastle Disease Virus Virulence." Frontiers in microbiology vol. 8 (2017): 161. doi: https://doi.org/10.3389/fmicb.2017.00161
Yu XH, Cheng JL, Xue J, Jin JH, Song Y, Zhao J, Zhang GZ. Roles of the Polymerase-Associated Protein Genes in Newcastle Disease Virus Virulence. Front Microbiol. 2017 Feb 06;8:161. doi: 10.3389/fmicb.2017.00161. eCollection 2017. PMID: 28220114; PMCID: PMC5292418.
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DKK4 enhances resistance to chemotherapeutics 5-Fu and YN968D1 in colorectal cancer cells.

Oncology letters

He S, Shen J, Hu N, Xu X, Li J.
PMID: 28356933
Oncol Lett. 2017 Feb;13(2):587-592. doi: 10.3892/ol.2016.5461. Epub 2016 Dec 06.

Dickkopf-related protein 4 (DKK4) is a target of the β-catenin/transcription factor 4 complex in colorectal cancer. Previous research has demonstrated that its expression level may vary and has indicated that it may have a role in the development of...

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He S, Shen J, Hu N, et al. DKK4 enhances resistance to chemotherapeutics 5-Fu and YN968D1 in colorectal cancer cells. Oncol Lett. 2016;13(2):587-592doi: 10.3892/ol.2016.5461.
He, S., Shen, J., Hu, N., Xu, X., & Li, J. (2017). DKK4 enhances resistance to chemotherapeutics 5-Fu and YN968D1 in colorectal cancer cells. Oncology letters, 13(2), 587-592. https://doi.org/10.3892/ol.2016.5461
He, Shengli, et al. "DKK4 enhances resistance to chemotherapeutics 5-Fu and YN968D1 in colorectal cancer cells." Oncology letters vol. 13,2 (2017): 587-592. doi: https://doi.org/10.3892/ol.2016.5461
He S, Shen J, Hu N, Xu X, Li J. DKK4 enhances resistance to chemotherapeutics 5-Fu and YN968D1 in colorectal cancer cells. Oncol Lett. 2017 Feb;13(2):587-592. doi: 10.3892/ol.2016.5461. Epub 2016 Dec 06. PMID: 28356933; PMCID: PMC5351290.
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Acute digital ischemia: A rare presentation of antisynthetase syndrome.

European journal of rheumatology

Chan JE, Palakodeti S, Koster MJ.
PMID: 28293456
Eur J Rheumatol. 2017 Mar;4(1):63-65. doi: 10.5152/eurjrheum.2017.16077. Epub 2017 Mar 01.

Antisynthetase syndrome (ASS) is recognized as a subgroup of idiopathic inflammatory myopathies (IIMs). It is associated with autoantibodies directed against aminoacyl-transfer ribonucleic acid (tRNA) synthetase enzymes. We report the first case of anti-PL-7/anti-SSA 52kD ASS presenting as acute digital...

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Chan JE, Palakodeti S, Koster MJ. Acute digital ischemia: A rare presentation of antisynthetase syndrome. Eur J Rheumatol. 2017;4(1):63-65doi: 10.5152/eurjrheum.2017.16077.
Chan, J. E., Palakodeti, S., & Koster, M. J. (2017). Acute digital ischemia: A rare presentation of antisynthetase syndrome. European journal of rheumatology, 4(1), 63-65. https://doi.org/10.5152/eurjrheum.2017.16077
Chan, Jin Ei, et al. "Acute digital ischemia: A rare presentation of antisynthetase syndrome." European journal of rheumatology vol. 4,1 (2017): 63-65. doi: https://doi.org/10.5152/eurjrheum.2017.16077
Chan JE, Palakodeti S, Koster MJ. Acute digital ischemia: A rare presentation of antisynthetase syndrome. Eur J Rheumatol. 2017 Mar;4(1):63-65. doi: 10.5152/eurjrheum.2017.16077. Epub 2017 Mar 01. PMID: 28293456; PMCID: PMC5335890.
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Evaluation of Surface Cleaning Procedures in Terms of Gas Sensing Properties of Spray-Deposited CNT Film: Thermal- and O₂ Plasma Treatments.

Sensors (Basel, Switzerland)

Kim JH, Song MJ, Kim KB, Jin JH, Min NK.
PMID: 28042843
Sensors (Basel). 2016 Dec 30;17(1). doi: 10.3390/s17010073.

The effect of cleaning the surface of single-walled carbon nanotube (SWNT) networks by thermal and the O₂ plasma treatments is presented in terms of NH₃ gas sensing characteristics. The goal of this work is to determine the relationship between...

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Kim JH, Song MJ, Kim KB, et al. Evaluation of Surface Cleaning Procedures in Terms of Gas Sensing Properties of Spray-Deposited CNT Film: Thermal- and O₂ Plasma Treatments. Sensors (Basel). 2016;17(1)doi: 10.3390/s17010073.
Kim, J. H., Song, M. J., Kim, K. B., Jin, J. H., & Min, N. K. (2016). Evaluation of Surface Cleaning Procedures in Terms of Gas Sensing Properties of Spray-Deposited CNT Film: Thermal- and O₂ Plasma Treatments. Sensors (Basel, Switzerland), 17(1), . https://doi.org/10.3390/s17010073
Kim, Joon Hyub, et al. "Evaluation of Surface Cleaning Procedures in Terms of Gas Sensing Properties of Spray-Deposited CNT Film: Thermal- and O₂ Plasma Treatments." Sensors (Basel, Switzerland) vol. 17,1 (2016). doi: https://doi.org/10.3390/s17010073
Kim JH, Song MJ, Kim KB, Jin JH, Min NK. Evaluation of Surface Cleaning Procedures in Terms of Gas Sensing Properties of Spray-Deposited CNT Film: Thermal- and O₂ Plasma Treatments. Sensors (Basel). 2016 Dec 30;17(1). doi: 10.3390/s17010073. PMID: 28042843; PMCID: PMC5298646.
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Application of AFP whole blood one-step rapid detection kit in screening for HCC in Qidong.

American journal of cancer research

Jin J, Zhang XY, Shi JL, Xue XF, Lu LL, Lu JH, Jiang XP, Hu JF, Duan BS, Yang CQ, Lu DR, Lu DL, Chen JG, Gao HJ.
PMID: 28670498
Am J Cancer Res. 2017 Jun 01;7(6):1384-1388. eCollection 2017.

Hepatocellular carcinoma (HCC) is a big problem in China where the Hepatitis B (HBV) infection patients are near to 120 million. Early screening and diagnosis is the key to reduce the incidence and mortality of HCC. Serum AFP detection...

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Jin J, Zhang XY, Shi JL, et al. Application of AFP whole blood one-step rapid detection kit in screening for HCC in Qidong. Am J Cancer Res. 2017;7(6):1384-1388
Jin, J., Zhang, X. Y., Shi, J. L., Xue, X. F., Lu, L. L., Lu, J. H., Jiang, X. P., Hu, J. F., Duan, B. S., Yang, C. Q., Lu, D. R., Lu, D. L., Chen, J. G., & Gao, H. J. (2017). Application of AFP whole blood one-step rapid detection kit in screening for HCC in Qidong. American journal of cancer research, 7(6), 1384-1388.
Jin, Jie, et al. "Application of AFP whole blood one-step rapid detection kit in screening for HCC in Qidong." American journal of cancer research vol. 7,6 (2017): 1384-1388.
Jin J, Zhang XY, Shi JL, Xue XF, Lu LL, Lu JH, Jiang XP, Hu JF, Duan BS, Yang CQ, Lu DR, Lu DL, Chen JG, Gao HJ. Application of AFP whole blood one-step rapid detection kit in screening for HCC in Qidong. Am J Cancer Res. 2017 Jun 01;7(6):1384-1388. eCollection 2017. PMID: 28670498; PMCID: PMC5489785.
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Allopregnanolone attenuates Aβ25-35-induced neurotoxicity in PC12 cells by reducing oxidative stress.

International journal of clinical and experimental medicine

Qian X, Cao H, Ma Q, Wang Q, He W, Qin P, Ji B, Yuan K, Yang F, Liu X, Lian Q, Li J.
PMID: 26550302
Int J Clin Exp Med. 2015 Aug 15;8(8):13610-5. eCollection 2015.

Massive accumulation of amyloid beta (Aβ) has been implicated as a pivotal event in the pathogenesis of Alzheimer's disease. The underlying mechanisms of Aβ-induced neurotoxicity include generation of reactive oxidative species (ROS), inflammation, and neurons loss. Allopregnano-lone (APα), a...

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Qian X, Cao H, Ma Q, et al. Allopregnanolone attenuates Aβ25-35-induced neurotoxicity in PC12 cells by reducing oxidative stress. Int J Clin Exp Med. 2015;8(8):13610-5
Qian, X., Cao, H., Ma, Q., Wang, Q., He, W., Qin, P., Ji, B., Yuan, K., Yang, F., Liu, X., Lian, Q., & Li, J. (2015). Allopregnanolone attenuates Aβ25-35-induced neurotoxicity in PC12 cells by reducing oxidative stress. International journal of clinical and experimental medicine, 8(8), 13610-5.
Qian, Xiaowei, et al. "Allopregnanolone attenuates Aβ25-35-induced neurotoxicity in PC12 cells by reducing oxidative stress." International journal of clinical and experimental medicine vol. 8,8 (2015): 13610-5.
Qian X, Cao H, Ma Q, Wang Q, He W, Qin P, Ji B, Yuan K, Yang F, Liu X, Lian Q, Li J. Allopregnanolone attenuates Aβ25-35-induced neurotoxicity in PC12 cells by reducing oxidative stress. Int J Clin Exp Med. 2015 Aug 15;8(8):13610-5. eCollection 2015. PMID: 26550302; PMCID: PMC4612987.
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A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling.

Scientific reports

Li D, Chang X, Connolly JJ, Tian L, Liu Y, Bhoj EJ, Robinson N, Abrams D, Li YR, Bradfield JP, Kim CE, Li J, Wang F, Snyder J, Lemma M, Hou C, Wei Z, Guo Y, Qiu H, Mentch FD, Thomas KA, Chiavacci RM, Cone R, Li B, Sleiman PA, Hakonarson H.
PMID: 28630421
Sci Rep. 2017 Jun 19;7(1):3847. doi: 10.1038/s41598-017-01674-8.

We conducted a genome-wide association study (GWAS) of anorexia nervosa (AN) using a stringently defined phenotype. Analysis of phenotypic variability led to the identification of a specific genetic risk factor that approached genome-wide significance (rs929626 in EBF1 (Early B-Cell...

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Li D, Chang X, Connolly JJ, et al. A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling. Sci Rep. 2017;7(1):3847doi: 10.1038/s41598-017-01674-8.
Li, D., Chang, X., Connolly, J. J., Tian, L., Liu, Y., Bhoj, E. J., Robinson, N., Abrams, D., Li, Y. R., Bradfield, J. P., Kim, C. E., Li, J., Wang, F., Snyder, J., Lemma, M., Hou, C., Wei, Z., Guo, Y., Qiu, H., Mentch, F. D., Thomas, K. A., Chiavacci, R. M., Cone, R., Li, B., Sleiman, P. A., & Hakonarson, H. (2017). A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling. Scientific reports, 7(1), 3847. https://doi.org/10.1038/s41598-017-01674-8
Li, Dong, et al. "A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling." Scientific reports vol. 7,1 (2017): 3847. doi: https://doi.org/10.1038/s41598-017-01674-8
Li D, Chang X, Connolly JJ, Tian L, Liu Y, Bhoj EJ, Robinson N, Abrams D, Li YR, Bradfield JP, Kim CE, Li J, Wang F, Snyder J, Lemma M, Hou C, Wei Z, Guo Y, Qiu H, Mentch FD, Thomas KA, Chiavacci RM, Cone R, Li B, Sleiman PA, Hakonarson H. A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling. Sci Rep. 2017 Jun 19;7(1):3847. doi: 10.1038/s41598-017-01674-8. PMID: 28630421; PMCID: PMC5476671.
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Quantum confinement induced ultra-high intensity interfacial radiative recombination in nanolaminates.

Nanoscale

Li J, Bi X.
PMID: 29058745
Nanoscale. 2017 Nov 02;9(42):16420-16428. doi: 10.1039/c7nr06564g.

Al

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Li J, Bi X. Quantum confinement induced ultra-high intensity interfacial radiative recombination in nanolaminates. Nanoscale. 2017;9(42):16420-16428doi: 10.1039/c7nr06564g.
Li, J., & Bi, X. (2017). Quantum confinement induced ultra-high intensity interfacial radiative recombination in nanolaminates. Nanoscale, 9(42), 16420-16428. https://doi.org/10.1039/c7nr06564g
Li, Jin, and Bi, Xiaofang. "Quantum confinement induced ultra-high intensity interfacial radiative recombination in nanolaminates." Nanoscale vol. 9,42 (2017): 16420-16428. doi: https://doi.org/10.1039/c7nr06564g
Li J, Bi X. Quantum confinement induced ultra-high intensity interfacial radiative recombination in nanolaminates. Nanoscale. 2017 Nov 02;9(42):16420-16428. doi: 10.1039/c7nr06564g. PMID: 29058745.
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Showing 1 to 12 of 2765 entries